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1 cytes, and to the oral bacterium Actinomyces naeslundii.
2 n response to coaggregation with Actinomyces naeslundii.
3 s by other oral streptococci and Actinomyces naeslundii.
4 340 but did not affect coaggregation with A. naeslundii.
5 y group A and group E strains of Actinomyces naeslundii.
6 ive gp340, gp340 SRCR domain peptide, and A. naeslundii.
7 ation with group C and group D strains of A. naeslundii.
8 but was required for adhesion of Actinomyces naeslundii.
9 icola, Tannerella forsythia, and Actinomyces naeslundii.
10 regations of these bacteria with Actinomyces naeslundii.
11 T. forsythia, 1.95 (1.0, 3.84), P = 0.05; A. naeslundii, 0.46 (0.25, 0.85), P = 0.01.
12 alis, Peptostreptococcus micros, Actinomyces naeslundii, Actinomyces israelii, Streptococcus sanguis,
13 nt pathway and that ureolysis can protect A. naeslundii against environmental acidification at physio
14 The presence of higher levels of Actinomyces Naeslundii (An) enhanced the effect of being Hp(+) on bo
15                      Only the Sg/Actinomyces naeslundii (An)/Fn multispecies biofilms elicited IL-6 l
16                                  Actinomyces naeslundii, an early colonizer of the oral cavity and a
17 nd it can detect as few as 10(4) cells of A. naeslundii and A. viscosus per ml.
18                       We report that both A. naeslundii and A. viscosus react with a component in the
19                The oral bacteria Actinomyces naeslundii and Actinomyces viscosus are known to contrib
20 ue cluster score (which included Actinomyces naeslundii and Eubacterium nodatum) was inversely associ
21 ze measurements showed that attachment of A. naeslundii and of S. gordonii to glass flowcells was enh
22  including Streptococcus mutans, Actinomyces naeslundii and Prevotella intermedia.
23            In contrast, type 1 fimbriated A. naeslundii and S. gordonii, which bound purified proline
24                                  Actinomyces naeslundii and Streptococcus gordonii, oral bacteria tha
25 f S. mutans, alone or mixed with Actinomyces naeslundii and Streptococcus oralis, were initially form
26  and Tannerella forsythia, while Actinomyces naeslundii and Streptococcus sanguinis were the top heal
27  biofilms: Streptococcus mutans, Actinomyces naeslundii, and Enterococcus faecalis.
28 aecalis, Streptococcus gordonii, Actinomyces naeslundii, and Lactobacillus acidophilus), in planktoni
29 S was also highly effective at S. mutans, A. naeslundii, and S. oralis biofilm removal from machine-e
30 ococcus mutans UA159, as well as Actinomyces naeslundii ATCC 12104 and Streptococcus oralis ATCC 9811
31 livarius IUOM-8, A. viscosus IUOM-62, and A. naeslundii ATCC 12104 degraded all four substrates.
32 n streptococcal biofilms than on Actinomyces naeslundii biofilms.
33 e ability of enterococci to bind Actinomyces naeslundii cells.
34 ortant role in development of S. gordonii-A. naeslundii communities in early dental plaque.
35         Therefore, urease could confer to A. naeslundii critical selective advantages over nonureolyt
36 m that the levan-synthesizing activity of A. naeslundii existed predominantly in a cell-free form, th
37 the chromosomal DNA flanking the Actinomyces naeslundii (formerly A. viscosus) T14V type 1 fimbrial s
38 e of the levanase gene of a genospecies 2 A. naeslundii, formerly Actinomyces viscosus, a portion of
39                                 Thus, the A. naeslundii FTF is more similar in primary sequence and t
40 as raised to a histidine-tagged, purified A. naeslundii FTF, and the antibody was used to localize th
41 anguis, Haemophilus aphrophilus, Actinomyces naeslundii, Fusobacterium nucleatum, and A. actinomycete
42 cidophilus, Lactobacillus casei, Actinomyces naeslundii genospecies (gsp) 1 and 2, total streptococci
43  SIgA1 and SIgA2 antibodies reactive with A. naeslundii genospecies 1 and 2 over this period.
44 nse in saliva to colonization by Actinomyces naeslundii genospecies 1 and 2 was studied in 10 human i
45 2 antibodies reactive with whole cells of A. naeslundii genospecies 1 and 2 were detected within the
46 2 antibodies reactive with whole cells of A. naeslundii genospecies 1 and 2 were normalized to the co
47 rations of SIgA1 and SIgA2 in saliva, the A. naeslundii genospecies 1- and 2-reactive SIgA1 and SIgA2
48                 The fine specificities of A. naeslundii genospecies 1- and 2-reactive SIgA1 and SIgA2
49 roscopy showed that neither S. oralis nor A. naeslundii grew when coaggregated pairwise with S. gordo
50            For 1 unit increase in log(10) A. naeslundii gsp 2 levels, there was a 60 gm decrease in b
51 gh mutans streptococci, lactobacilli, and A. naeslundii have been implicated in its initiation and pr
52 justed for confounders, were observed for A. naeslundii I, Actinomyces gerencseriae, C. gingivalis, E
53           These species included Actinomyces naeslundii II, Actinomyces israelii, Actinomyces odontol
54  was the dominant levanase and sucrase of A. naeslundii; (ii) that LevJ was inducible by growth in su
55 w arginine concentrations with or without A. naeslundii, indicating that arginine biosynthesis was es
56 n extracts from S. gordonii, but not from A. naeslundii, interfered with S. mutans BM71 colonization.
57  found in healthy oral biofilms (Actinomyces naeslundii, Lactobacillus casei, Streptococcus mitis, Ve
58 icola, Streptococcus oralis, and Actinomyces naeslundii levels.
59  contrast to previous beliefs, strains of A. naeslundii may have the potential to be significant cont
60 se expression by nitrogen availability in A. naeslundii may require a positive transcriptional activa
61                       Thus, the S. oralis-A. naeslundii pair formed a mutualistic relationship, poten
62 ite and did not coaggregate with Actinomyces naeslundii PK606.
63 mposed of Streptococcus sanguis, Actinomyces naeslundii, Porphyromonas gingivalis, and Fusobacterium
64                                  Actinomyces naeslundii, Prevotella spp., and Porphyromonas gingivali
65    Several oral streptococci, relative to A. naeslundii, produced proteases that inactivated the S. m
66               However, both S. oralis and A. naeslundii showed luxuriant, interdigitated growth when
67                                     Thus, A. naeslundii stabilizes S. gordonii expression of arginine
68 ence of Streptococcus oralis and Actinomyces naeslundii steadily formed exopolysaccharides, which all
69 lusters present in the genome of Actinomyces naeslundii strain MG-1.
70                                           A. naeslundii strains have been previously classified into
71 pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptococcus mutans, and Aggregatibacter ac
72 ibody against type 2 fimbriae of Actinomyces naeslundii T14V (anti-type-2) were much less frequent th
73   Unlike wild-type dual-species biofilms, A. naeslundii T14V and an S. oralis 34 luxS mutant did not
74 o human oral commensal bacteria, Actinomyces naeslundii T14V and Streptococcus oralis 34, is dependen
75 cate that synthesis of type 2 fimbriae in A. naeslundii T14V may involve posttranslational cleavage o
76           The type 1 fimbriae of Actinomyces naeslundii T14V mediate adhesion of this gram-positive s
77 ad significant sequence homology with the A. naeslundii T14V type 1 and A. naeslundii WVU45 type 2 fi
78   The nucleotide sequence of the Actinomyces naeslundii T14V type 2 fimbrial structural subunit gene,
79 d against the maltose binding protein and A. naeslundii T14V whole bacteria.
80 d only with the antibody directed against A. naeslundii T14V whole bacterial cells.
81 L1, Streptococcus oralis 34, and Actinomyces naeslundii T14V).
82  and function of cell surface fimbriae of A. naeslundii T14V.
83 he other five ORFs were used to transform A. naeslundii T14V.
84 ivalis, Fusobacterium nucleatum, Actinomyces naeslundii, Tannerella forsythia, and Streptococcus gord
85 ever, within 1 h after coaggregation with A. naeslundii, the expression of argC, argG, and pyrA(b) in
86 haride also failed to support adhesion of A. naeslundii, thereby establishing the essential role of b
87                   The ability of Actinomyces naeslundii to convert sucrose to extracellular homopolym
88 ch as Streptococcus gordonii and Actinomyces naeslundii to the saliva-coated tooth surface and to eac
89 fimA mutant by Western blotting with anti-A. naeslundii type 2 fimbrial antibody, but the subunit pro
90 hen the main fimbrial subunit of Actinomyces naeslundii type I fimbriae, FimA, is expressed in coryne
91 y recombinant bacteriophages carrying the A. naeslundii urease gene cluster and roughly 30 kbp of fla
92 ximal, nitrogen-regulated promoter of the A. naeslundii urease gene cluster was identified.
93 ectively utilized as a nitrogen source by A. naeslundii via a urease-dependent pathway and that ureol
94 rt fimbriae-mediated adhesion of Actinomyces naeslundii was explained by the position of Galf, which
95 le carbon and nitrogen source showed that A. naeslundii was unable to grow either in planktonic cultu
96  against Eubacterium nodatum and Actinomyces naeslundii) was inversely associated with all-cause mort
97 gingivalis, F. nucleatum, S. sanguis, and A. naeslundii were grown on smooth, acid-etched, and acid-e
98 ell adhesion to SAG and to two strains of A. naeslundii were observed when both sspA and sspB genes w
99 us sanguis and type 2 fimbriated Actinomyces naeslundii, which bound terminal sialic acid and Galbeta
100 rized the urease gene cluster of Actinomyces naeslundii, which is one of the pioneer organisms in the
101 tivity and urease-specific mRNA levels in A. naeslundii WVU45 can increase up to 50-fold during growt
102 gy with the A. naeslundii T14V type 1 and A. naeslundii WVU45 type 2 fimbrial structural subunits.
103  An internal fragment of the ureC gene of A. naeslundii WVU45 was initially amplified by PCR with deg
104 ncoding the fructosyltransferase (FTF) of A. naeslundii WVU45.
105 e encoding FTF was isolated from Actinomyces naeslundii WVU45; the deduced amino acid sequence showed

 
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