ライフサイエンスコーパス: nasal

1 symptom scores, peak nasal inspiratory flow, nasal (0-8 hours) and serum cytokines, serum antibodies,

2 l (0.80), infero-temporal (0.87), and supero-nasal (0.81).

3 subepithelial juxtaforniceal (3/4, 75%), or nasal (1/4, 25%) bulbar conjunctival nodules, which were

4 ior of the smart polymers found suitable for nasal administration, including poloxamer, chitosan, EHE

5 of Camsap3 (tm1a/tm1a) mice revealed severe nasal airway blockage and abnormal ciliary morphologies

6 genes in the airway, through the analysis of nasal airway transcriptome data from 695 children.

7 e pancreatic insufficiency and hypoplasia of nasal alae.

8 The direct-instillation nasal allergen challenge (NAC) and the environmental exp

9 crepant group) were subjected to consecutive nasal allergen challenges (NAC) with seasonal (NAC-S) an

10 ator) and offspring's asthma with or without nasal allergies (outcomes) was analyzed by using 2-level

11 ght in puberty on offspring's asthma without nasal allergies (relative risk ratio, 2.31 [95% CI, 1.23

12 No effect on offspring's asthma with nasal allergies was found.

13 rted by WHO and other reputed organizations, nasal anatomy, mechanism and challenges of nose-to-brain

14 ta sets containing gene expression data from nasal and airway epithelial cells from children and adul

15 Nasal and cutaneous allergen challenge tests were perfor

16 High viral RNA loads in nasal and pharyngeal specimens were associated with fata

17 Stool, nasal and skin samples of 6-month-old DIABIMMUNE study p

18 Both the nasal and temporal inverted ILM flaps repaired FTMH and

19 the superior and inferior quadrants than the nasal and temporal quadrants.

20 The difference in thickness between nasal and temporal sectors and between superior and infe

21 associated with disease are present in both nasal and tracheal epithelium.

22 In these studies, we isolated primary swine nasal and tracheal respiratory epithelial cells and immo

23 onverting enzyme 2 (ACE2), within the upper (nasal) and lower (pulmonary) respiratory tracts of human

24 ample (HCV+SS; 48%, 22/46 rectal; 62%, 29/47 nasal), and contamination with blood was excluded in all

25 samples from control subjects, and in skin, nasal, and oral samples from 302 workers from different

26 Other implicating factors include nasal anterior capsule override, haptic orientation, lar

27 bulizer expected to improve OT deposition in nasal areas putatively involved in direct nose-to-brain

28 Nasal aspirates from patients with severe disease had re

29 mental conditions (n = 8 donors); and (b) in nasal aspirates of young children (<=3 years) hospitaliz

30 ith enhanced Th2 cellular responses.Methods: Nasal aspirates were collected from infants hospitalized

31 In the nasal-associated lymphoid tissue (NALT), a muted inflamm

32 sgenic mice exhibited enhanced resistance to nasal bacterial infection as the transgenic mice showed

33 Nasal biopsies were taken 24 hours after the 1st and 5th

34 as extracted from PBMCs, sorted B cells, and nasal biopsy samples for heavy chain variable gene reper

35 ontrolled, time course SLIT study, PBMCs and nasal biopsy samples were collected from 40 adults with

36 mice have a relatively short midface, short nasal bones, tall mandibular corpora, and long mandibula

37 res include small palpebral fissures, a wide nasal bridge and nose, micrognathia, and digital anomali

38 ed via recordings from EEG electrodes at the nasal bridge represent responses from the human olfactor

39 sed in COPD patients compared to controls in nasal, bronchial and small airways brushings.

40 mechanical ventilation (59%), and high flow nasal cannula (46%).

41 termine the safety and efficacy of high-flow nasal cannula after extubation in critically ill adults.

42 delivery modalities of humidified high-flow nasal cannula and noninvasive positive-pressure ventilat

43 th modes of oxygen therapy such as high-flow nasal cannula and noninvasive positive-pressure ventilat

44 with conventional oxygen therapy, high-flow nasal cannula decreased reintubation (relative risk, 0.4

45 The role of high-flow nasal cannula during and before intubation is unclear de

46 om were receiving any supplemental oxygen or nasal cannula flow, was 46% (95% CI, 40%-53%).

47 High-flow nasal cannula had no effect on the occurrence rate of pe

48 certainty evidence that the use of high-flow nasal cannula likely has no effect on severe desaturatio

49 High-flow nasal cannula may reduce ICU length of stay (moderate ce

50 with the use of either humidified high-flow nasal cannula or noninvasive positive-pressure ventilati

51 vasive ventilation (n = 285; 44%), high-flow nasal cannula oxygen (n = 55; 8%), and noninvasive venti

52 8%), and noninvasive ventilation + high-flow nasal cannula oxygen (n = 64; 10%).

53 High-flow nasal cannula reduces reintubation compared with convent

54 time to mechanical ventilation and high-flow nasal cannula use may be associated with mortality in co

55 High-flow nasal cannula use prior to intubation was not associated

56 om ICU admission to intubation nor high-flow nasal cannula use were associated with increased mortali

57 , face mask, heated and humidified high-flow nasal cannula, and noninvasive positive-pressure ventila

58 included children with any use of high-flow nasal cannula, continuous positive airway pressure, or b

59 gen modalities tested included nonhumidified nasal cannula, face mask, heated and humidified high-flo

60 to 100% with 1 L of O(2) administered via a nasal cannula.

61 ean difference, 10.3 s higher with high-flow nasal cannula; 95% CI, 11.0 s lower to 31.7 s higher), P

62 of more than 4 L/min flow; use of high-flow nasal cannula; use of non-invasive mechanical ventilatio

63 IOLs, and neodymium:yttrium-aluminum-garnet nasal capsulectomy.

64 D printing could realistically replicate the nasal cavities and the airflow passing through them from

65 io of 1.05 +/- 0.01 (mean +/- SD) and in the nasal cavities with a ratio of 1.20 +/- 0.1 (mean +/- SD

66 Following infection of ocular, oral, or nasal cavities, sensory neurons within trigeminal gangli

67 were used to print plastic 3D prints of the nasal cavities, which were also CBCT scanned and the mea

68 ymptoms, QOL scores, face scale, findings of nasal cavity and laboratory findings before start of the

69 In mammals, the nasal cavity houses a bony system supporting soft tissue

70 , it was unable to persistently colonize the nasal cavity of inoculated pigs, with all pigs clearing

71 e a suitable and fast method for replicating nasal cavity structures and for the experimental testing

72 oV-2 infected NRP1-positive cells facing the nasal cavity.

73 dial wall and a funnel-like lateral opening, nasal channels run obliquely from lateral (surface) to m

74 also show how clinically relevant and mature nasal chondrocyte-derived engineered cartilage can be as

75 However, our knowledge of how nasal colonization impacts on the lung cells, especially

76 protection against B. pertussis and prevents nasal colonization in animal models.

77 ccharide to G. parasuis virulence as well as nasal colonization in pigs.

78 ities outcompeted pathogenic bacteria during nasal colonization in vivo.

79 controlled human infection model to achieve nasal colonization with 6B serotype, we investigated the

80 ).Conclusions: Our findings demonstrate that nasal colonization with pneumococcus and microaspiration

81 < .0001) and -0.90 versus -0.31 (P = .0140); Nasal Congestion Score, -0.89 versus -0.35 (P = .0004) a

82 smell loss without significant rhinorrhea or nasal congestion suggests that SARS-CoV-2 targets the ch

83 or nasal congestion; and current runny nose, nasal congestion, fever, chest indrawing, or cyanosis.

84 ty breathing, fast breathing, runny nose, or nasal congestion; and current runny nose, nasal congesti

85 oveal choroidal thickness (CT), temporal and nasal CT, foveal avascular zone (FAZ) diameter, and vess

86 oncentrations of neutrophils to RSV-infected nasal cultures was associated with greater epithelial da

87 nal home-based self-sampling, we demonstrate nasal cytokine levels correlate and cluster according to

88 ubjects, early induction of a minimal set of nasal cytokines that discriminated high responders (G-CS

89 Responses to nasal delivery of R848 enables simple assessment of muco

90 , runny nose), HEENT findings on exam (i.e., nasal discharge, red throat), and having a flush toilet

91 ears was 54% (95% CI 44-63) in patients with nasal disease (n=98) and 34% (27-46) in patients with ex

92 cally, horizontal cupping with minimal or no nasal displacement of vessels, and superior optic nerve

93 nifestations and diagnostic tools (including nasal endoscopy and imaging) have undergone major change

94 tro, many S. epidermidis isolates stimulated nasal epithelia to produce antimicrobial peptides, killi

95 of allergens was evaluated on primary human nasal epithelial cell cultures.

96 ersely, HCMV downregulated the expression of nasal epithelial cell-related genes.

97 tory epithelial cells and immortalized swine nasal epithelial cells (siNEC) and tracheal epithelial c

98 ) were increased in this model as well as in nasal epithelial cells from asthmatics as compared with

99 These genes are co-expressed in nasal epithelial cells with genes involved in innate imm

100 o epithelial cells or freshly isolated human nasal epithelial cells with low concentrations of sphing

101 thelial allergen passage in cultured primary nasal epithelial cells.

102 Genome-wide nasal epithelial DNAm and gene expression were measured.

103 This study aimed to identify nasal epithelial DNAm differences between severe and non

104 function measures of hyperinflation, and the nasal epithelial gene-expression profile in severe COPD.

105 ned for immune mediator levels in samples of nasal epithelial lining fluid during episodes with asthm

106 This cross-sectional study used nasal epithelium collected in 2015-2018 to compare expre

107 ssociated with SARS-CoV-2 cell entry, in the nasal epithelium of children vs adults.

108 inal obstruction, bioelectric defects in the nasal epithelium, histopathologic changes in the trachea

109 and disease can be gained from studying the nasal epithelium.

110 alue of 296.46 (+/-28.85) um(P = .001) and a nasal figure of 93.63 (+/-26.86) um(P = .001).

111 ome and immune response, we collected paired nasal filters and washes from a group of 40 healthy chil

112 oassay sensitivity, our results suggest that nasal filters are a superior sampling method to characte

113 To examine whether nasal filters are superior to nasal washes as a sampling

114 In general, nasal filters were more likely to pass quality control i

115 ed the assay by dipping swabs into synthetic nasal fluid spiked with the virus, moving the swab to vi

116 Pneumococcal adherence to adult human nasal fluid was seen only by isolates expressing pilus-1

117 to quantify HCV-RNA levels within rectal and nasal fluids.

118 Nasal foam swab self-sampling at home provides a precise

119 ructures and for the experimental testing of nasal function.

120 ed which explains the application of in situ nasal gel for brain delivery of CNS acting moieties.

121 nd application of stimuli-responsive in situ nasal gel for brain drug delivery.

122 eumocytes, ileal absorptive enterocytes, and nasal goblet secretory cells.

123 temporal group (35%) compared with 5% in the nasal group (P = .04), but these were unrelated to BCVA.

124 Displacement of the nasal hemi-disc in adduction was greater at 38.5 +/- 1.7

125 ; 11.5% receiving noninvasive ventilation or nasal high-flow oxygen; and 6.7% receiving invasive mech

126 pal component analysis was performed for all nasal immune variables, and single immune variables were

127 icker RNFLs in the temporal, superotemporal, nasal, inferonasal, and inferotemporal regions (43.6% of

128 We measured total nasal symptom scores, peak nasal inspiratory flow, nasal (0-8 hours) and serum cyto

129 Ethmoid tissues and nasal lavage fluids (NLFs) were obtained from control pa

130 ECP in the nasal lavage increased after the NAC-P in the group A (P

131 A nasal lavage was collected before and after the NACs to

132 5-75%pred) ), thus lower sEV-miRNA levels in nasal lavages associated with airway obstruction.

133 miRNAs could be confirmed in EVs from human nasal lavages.

134 Nasal levels of IL-8, IL-33, sIgG(4) and sIgE could be p

135 tines were increased in macrophages, sputum, nasal lining fluid, and plasma of patients with N-ERD.

136 Metabolites in nasal lining fluid, sputum, and plasma from patients wit

137 binds to the extracellular domain of ACE2 in nasal, lung, and gut epithelial cells through its spike

138 intramuscular genioglossus electromyography, nasal mask and pneumotachograph to measure OSA severity,

139 lockage and abnormal ciliary morphologies in nasal MCCs.

140 ce of antibiotic resistance in digestive and nasal microbiological cultures.

141 , and interactions with other species in the nasal microbiome likely modulate its carriage.

142 ling to examine the relation of longitudinal nasal microbiota abundances to the risk of recurrent whe

143 We sought to examine the relation of the nasal microbiota at bronchiolitis-related hospitalizatio

144 cing and culture-based analysis of the human nasal microbiota from different age groups.

145 erial resistance in urinary, intestinal, and nasal microbiota.

146 wNP, compared to those isolated from control nasal mucosa (control-NM), in order to identify which ke

147 n, and a mean peak of 5.97x10 2 ng/ml in the nasal mucosa 2-3 days after infusion.

148 Gli3 is critical for OEC development in the nasal mucosa and subsequent GnRH-1 neuronal migration.

149 Leukocyte subsets were assessed in nasal mucosa biopsies at baseline and after treatment.

150 an viral loads in bronchoalveolar lavage and nasal mucosa compared with after the primary infection.

151 ion of IL-4, IL-21, and IL-6 was observed in nasal mucosa following intranasal allergen challenge in

152 dysregulated innate immune responses of the nasal mucosa in allergic individuals may be important in

153 Plasma cell numbers in the nasal mucosa increased during treatment (P = .02).

154 mation of olfactory ensheathing cells in the nasal mucosa, and impairs GnRH-1 neuronal migration to t

155 m lack of olfactory ensheathing cells in the nasal mucosa, moreover, we discovered that Ascl-1 is nec

156 an viral loads in bronchoalveolar lavage and nasal mucosa, respectively, as compared with viral loads

157 e patterns to HCMV, comparing infected human nasal mucosal and placental tissues, representing the vi

158 ine ligand [CCL]2, CCL3, CCL4, and CCL13) in nasal mucosal fluid, without causing systemic immune act

159 ts with allergic disorders have an increased nasal mucosal IFN and chemokine response to the viral RN

160 ty and the full cellular repertoire of human nasal mucosal tissues.

161 ion, and 21 lipid mediators were measured in nasal mucus from 109 patients with CRSwNP, 30 patients w

162 Nasal/ oropharyngeal swabs were collected and tested for

163 rity visual analog scale (VAS), 22-item Sino-Nasal Outcome Test (SNOT-22), 5-dimension EuroQoL (EQ-5D

164 uded change from baseline to week 24 in Sino-Nasal Outcome Test-22 (SNOT-22) score, University of Pen

165 uch as noninvasive ventilation and high-flow nasal oxygen may be more effective than standard oxygen

166 g experiments demonstrated that the oral and nasal papillae are innervated by the trigeminal nerve, t

167 xchange, angled directly toward the superior nasal paracentral retina.

168 The human nasal passages host a distinct community of microbes.

169 NGI through a preinstalled NPA in the right nasal passageway (Group NPA) and 55 patients to receive

170 led nasopharyngeal airway (NPA) in the right nasal passageway can facilitate NGI in anesthetized and

171 acute respiratory syndrome coronavirus 2 at nasal-pharyngeal swabbing, negative chest CT findings, a

172 individuals were positively correlated with nasal pneumococcal density (r = 0.71; P = 0.029).

173 Nasal pollen-specific IgA and IgG isotypes are potential

174 ed to compare the transcriptional profile of nasal polyp (NP) cells from patients with AERD and patie

175 type 2 immune reactions representing >80% of nasal polyp cases in Europe and the United States.

176 higher IgE levels are associated with faster nasal polyp regrowth.

177 ort-Form Health Survey (SF-36) for HRQoL and nasal polyp-related healthcare resource use questionnair

178 Chronic rhinosinusitis with nasal polyposis (CRSwNP) negatively affects health-relat

179 The cause of severe nasal polyposis in aspirin-exacerbated respiratory disea

180 Chronic rhinosinusitis (CRS) without nasal polyps (CRSsNP) is a common disease that is charac

181 Chronic rhinosinusitis with nasal polyps (CRSwNP) is characterized by IgE hyperprodu

182 Chronic rhinosinusitis with nasal polyps (CRSwNP) is generally associated with sever

183 s) obtained from chronic rhinosinusitis with nasal polyps (CRSwNP) when differentiated in an air-liqu

184 rized by asthma, chronic rhinosinusitis with nasal polyps (CRSwNP), and an intolerance of medications

185 with AERD, chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP), CRS without nasal polyps, and con

186 tinct subtype of chronic rhinosinusitis with nasal polyps (CRSwNP).

187 d into CRS without nasal polyps and CRS with nasal polyps (CRSwNP).

188 paranasal sinuses, frequently occurring with nasal polyps and allergies.

189 A and can be differentiated into CRS without nasal polyps and CRS with nasal polyps (CRSwNP).

190 MMP3 and MMP7 observed in CRS subjects with nasal polyps compared to CRS subjects without polyps.

191 In subgroup analysis, CRS with and without nasal polyps demonstrated a significant association with

192 a, endotypes for chronic rhinosinusitis with nasal polyps have been established, with type 2 immune r

193 itating local antibody production and severe nasal polyps in AERD.

194 Chronic rhinosinusitis (CRS) with nasal polyps is a common chronic condition.

195 Subjects with AERD whose nasal polyps recurred rapidly had higher IgE levels than

196 CRS) with nasal polyps (CRSwNP), CRS without nasal polyps, and controls without CRS.

197 al tissue from non-CRS and CRS subjects with nasal polyps.

198 Approximately half of patients reported nasal polyps.

199 grass specific immunoglobulins and skin and nasal provocation tests to grass pollen.

200 T of the inferior temporal quadrant/superior nasal quadrant (R2) were calculated (R1ET, R1BLT; R2ET,

201 The resulting decrease in the inner ring nasal quadrant was 40% greater for VD and 48% greater fo

202 odels achieved high accuracy in the inferior nasal (R(2) = 0.60) and superior nasal (R(2) = 0.67) sec

203 he inferior nasal (R(2) = 0.60) and superior nasal (R(2) = 0.67) sectors, moderate accuracy in inferi

204 This study shows that nasal reactivity to aeroallergens with and without posit

205 local allergic rhinitis (LAR) are defined by nasal reactivity to aeroallergens with and without posit

206 sed that sex-related size differences in the nasal region arise during puberty.

207 nose-to-brain delivery by not only enhancing nasal residence time but also minimizing the mucociliary

208 Here, we tested the hypothesis that nasal respiration underlies the emergence of high-freque

209 with ketamine-dependent HFO being driven by nasal respiration, unilateral naris blockade led to an i

210 nanoparticles were detected in olfactory and nasal respiratory epithelia and cilia in animals.

211 lication in the upper respiratory tract, the nasal respiratory epithelium in particular, of donors is

212 e air preferentially infect ferret and human nasal respiratory epithelium.

213 h some temporally incident light rays to the nasal retina pass anterior to the IOL and some are refra

214 nd profiled a subset of monthly longitudinal nasal samples from the first year after birth using shot

215 g samples from four index cases and skin and nasal samples from workers in the machine shop area were

216 the detection of the virus antigen in spiked nasal samples showing excellent recovery percentages.

217 acter was more abundant in Estonian skin and nasal samples.

218 ecific T lymphocytes, and gene expression in nasal scrapings.

219 elial eosinophils and IL-5 concentrations in nasal secretion (r = 0.69, P = .06) in allergic rhinitis

220 Nasal symptoms, Th1/Th2 cytokines in nasal secretion and serum were evaluated.

221 Nasal tissue eosinophils and IL-5 in nasal secretion increased significantly after RAC compar

222 Serum and nasal secretion samples from patients with HDM allergy (

223 th SARS-CoV-2 have high viral loads in their nasal secretions, they can silently and efficiently spre

224 ), a vitreous thickening was observed in the nasal sector of the optic disc, remnant of fetal vascula

225 or and inferior hemispheres and temporal and nasal sectors were evaluated.

226 to identify the types of tissues found in a nasal septal biopsy, i.e., hyaline cartilage and pericho

227 However, there was no difference in nasal shedding between vaccinated and control SARS-CoV-2

228 hyperpermeable choroidal vasculature on the nasal side of the fovea in the early and later phases of

229 In the first study, nasal specimens and either nasopharyngeal or oropharynge

230 avirus 2, nasopharyngeal, mid turbinate, and nasal specimens are suitable in most cases and require l

231 pressant efficacy of S-ketamine (esketamine) nasal spray in major depressive disorder (MDD), we perfo

232 Following a 4-week mometasone furoate nasal spray run-in, patients were randomized to commence

233 nalogue of single-stranded RNA) or saline by nasal spray to healthy participants without allergy (n =

234 ods of intranasal administration (a standard nasal spray, and a nebulizer expected to improve OT depo

235 inactivated influenza vaccines (IIV3) and a nasal spray, tetravalent live-attenuated influenza vacci

236 d in Europe in 2012 and is administered as a nasal spray.

237 Topical intra-nasal sprays are amongst the most commonly prescribed th

238 we have identified a new usage strategy for nasal sprays available over-the-counter, that registers

239 As such, oral steroids, topical nasal sprays or irrigation, and surgery can be utilized

240 age instructions and hence merit a change in nasal standard-of-care, this study also demonstrates how

241 ancer routinely tested for SARS-CoV-2 RNA by nasal swab and real-time polymerase chain reaction betwe

242 CRISPR assay diagnostic results obtained nasal swab samples of individuals with suspected COVID-1

243 Nasal swab samples were collected at age 3, 6, 12, 18, 2

244 Supervised oral fluid and nasal swab specimens performed similarly to clinician-co

245 es in bronchoalveolar-lavage (BAL) fluid and nasal swab specimens were assessed by polymerase chain r

246 en when they test positive for COVID-19 on a nasal swab.

247 ian-supervised self-collected mid-turbinate (nasal) swab specimens, and clinician-collected nasophary

248 ngeal swabs (NPS) to self-collected anterior nasal swabs (ANS) and straight saliva for the diagnosis

249 Trained parents collected nasal swabs 3 weeks after hospitalization and, when heal

250 ete protection in bronchoalveolar lavage and nasal swabs after SARS-CoV-2 challenge.

251 We collected nasopharyngeal or nasal swabs at enrollment and tested for SARS-CoV-2 usin

252 d COVID-19 testing utilizing easy-to-collect nasal swabs but demonstrated <100% PPA compared to PCR.

253 in viral transport media for Cepheid and dry nasal swabs for Abbott ID Now.

254 The children were examined and nasal swabs for the detection of RSV were obtained durin

255 ters researchers collected clinical data and nasal swabs from infants hospitalized for bronchiolitis.

256 Viruses from nasal swabs taken 1, 3, and 6 days postvaccination were

257 s antiviral effects, RSV RNA viral load from nasal swabs was quantified over time using reverse-trans

258 Rectal and nasal swabs were collected to quantify HCV-RNA levels wi

259 124 (12.8%) and 17 of 8100 (0.2%) stool and nasal swabs, respectively.

260 er-titer viral isolates from human and swine nasal swabs, supported the replication of isolates that

261 load in the lungs but not viral shedding in nasal swabs.

262 We applied 16S rRNA gene sequencing to all nasal swabs.

263 for birch) has been found between the Total Nasal Symptom and Medication Score (TNSMS) and the polle

264 on defined via the EAACI criteria, and total nasal symptom and medication scores as reported with the

265 The primary outcome was the total nasal symptom score area under the curve for the first 3

266 We measured total nasal symptom scores, peak nasal inspiratory flow, nasal

267 The nasal symptoms, on which benralizumab had an unremarkabl

268 Nasal symptoms, Th1/Th2 cytokines in nasal secretion and

269 s defined as sensitisation and self-reported nasal symptoms.

270 lution are more likely to report more severe nasal symptoms.

271 tic uniaxial tensile tests were performed in nasal-temporal direction immediately after preparing cor

272 se face, upslanted palpebral fissures, broad nasal tip, and wide mouth, developmental delay and/or in

273 Major organs, along with the nasal tissue and trigeminal nerve, were harvested to ass

274 Nasal tissue eosinophils and IL-5 in nasal secretion inc

275 oteases, and antiproteases was quantified in nasal tissue from non-CRS and CRS subjects with nasal po

276 ate inflammation and the protease profile in nasal tissues and plasma from control non-CRS patients a

277 We demonstrate high SARS-CoV-2 burden in nasal tissues and secretions, which coincided with effic

278 rseasonal differences in levels of serum and nasal, total, and Betula verrucosa 1-specific immunoglob

279 demonstrated that sphingosine is present in nasal, tracheal, and bronchial epithelial cells and cons

280 th severe persistent asthma and controls for nasal transcriptome profiling and applied network-based

281 escribe the establishment and use of a novel nasal turbinate organ culture to study the initial steps

282 Nasal turbinate tissue viability and physiological funct

283 Viral antigens were detected in nasal turbinate, trachea, lungs, and intestine with acut

284 each possessing NA-F144C or NA-T342A in the nasal turbinates of one or several infected ferrets, res

285 4C/T342A mutant viruses were detected in the nasal turbinates, in addition to the lungs.

286 nodal natural killer T-cell lymphoma (NKTCL; nasal type) is an aggressive malignancy with a particula

287 ion site, our findings are relevant to intra-nasal vaccines that are in-development, to mitigate the

288 esatovir resistance had significantly higher nasal viral load area under the curve relative to those

289 fection, with a prolonged period of oral and nasal viral shedding that is not accompanied by clinical

290 vered patients was observed in the titers of nasal wash antibodies, especially binding to the central

291 RNA >=1 log10PFUe/mL (ITT-IS population) in nasal wash samples.

292 hed LID/DeltaM2-2/1030s vaccine (median peak nasal wash titers: 3.1 log10 PFU/mL by immunoplaque assa

293 response profiles; daily viral detection via nasal wash; and clinical signs and symptoms.

294 virus) produced higher infectious titres in nasal washes and the trachea, but not in the lungs, supp

295 ollect upper airway epithelial lining fluid, nasal washes are poorly reproducible, not suitable for s

296 xamine whether nasal filters are superior to nasal washes as a sampling method for the characterizati

297 r antibody affinity to RSV G was observed in nasal washes from early-recovered individuals compared w

298 In contrast, nasal washes from early-recovered individuals demonstrat

299 Serum and nasal washes were collected from influenza positive and

300 culating in Brazil and detected in swabs and nasal washes.