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1  leukotriene (cysLT) levels were measured in nasal lavage fluid.
2                 IL-8 levels were measured in nasal lavage fluid and serum on randomization, day 8, an
3 ction through analysis of cytokine levels in nasal lavage fluid and stimulated peripheral blood monon
4 tion of macrophage inflammatory protein 2 in nasal lavage fluid and subsequent recruitment of neutrop
5 ncreased levels of inflammatory cytokines in nasal lavage fluid, and confirmed olfactory dysfunction
6  antibodies was measured in tissue extracts, nasal lavage fluid, and sera by using multiplex bead arr
7                   Viral load was measured in nasal lavage fluid at day 3, 6 and 14.
8                                              Nasal lavage fluid cells were analyzed for inflammatory
9 er baseline concentrations of NGF protein in nasal lavage fluids, compared with control subjects.
10 the mouse, human CD49d(+) PMNs isolated from nasal lavage fluid during a viral respiratory tract infe
11 +) PMN frequency was significantly higher in nasal lavage fluid during acute respiratory symptoms in
12 e assessed, the inflammatory cell content in nasal lavage fluids estimated, and the activation patter
13                We collected nasal tissue and nasal lavage fluid from patients with CRS and control su
14 tase protein was also elevated in NPs and in nasal lavage fluids from patients with CRSwNP.
15  = .6) but resulted in a greater decrease in nasal lavage fluid IL-8 levels by day 15 (P = .03).
16                                              Nasal lavage fluid levels of interleukin (IL)-6, tumor n
17                                              Nasal lavage fluid myeloperoxidase (another neutrophil m
18               We analysed cytokine levels in nasal lavage fluid (NLF) in 59 subjects (46 with asthma)
19  We sought to compare MP types and levels in nasal lavage fluids (NLFs) from controls and patients wi
20                          Ethmoid tissues and nasal lavage fluids (NLFs) were obtained from control pa
21  and measured the total leukocyte content of nasal lavage fluid obtained from 10 min to 4 h after the
22 cell differential counts were evaluated from nasal lavage fluid obtained from wild-type and IL-17A-de
23 ergen significantly increased NGF protein in nasal lavage fluids of subjects with allergic rhinitis,
24  histologic assessment, cytokine analysis of nasal lavage fluid, olfactory behavioral tests, and gene
25 nts with acute bronchiolitis was elevated in nasal lavage fluid on both Days 1-2 (p = 0.014) and Days
26   We found that IL-6 and IFN-alpha levels in nasal lavage fluids peaked early (day 2) and correlated
27  IL-8, IFN-alpha, TGF-beta, and TNF-alpha in nasal lavage fluid, plasma, and serum obtained serially
28 ning cytokine and chemokine levels in serial nasal lavage fluid samples from 15 volunteers experiment
29  higher level of NTHI 2019 per milliliter of nasal lavage fluid than chinchillas colonized with predo
30 y response (measured by the albumin level in nasal-lavage fluid) to nasal provocation.
31     The percentage of leukocytes observed in nasal lavage fluid was significantly increased 12, 24, 4
32 ores, and concentrations of IL-6 and IL-8 in nasal lavage fluids were compared between treatment grou
33         The concentrations of NGF protein in nasal lavage fluids were not affected by provocation wit
34                        Sinonasal tissues and nasal lavage fluids were obtained from control patients