コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ry leukoplakia) or latent infection (such as nasopharyngeal carcinoma).
2 rr virus (EBV) causes Burkitt's lymphoma and nasopharyngeal carcinoma.
3 a variety of ailments, from benign rashes to nasopharyngeal carcinoma.
4 580V, is associated with IgA nephropathy and nasopharyngeal carcinoma.
5 transplant lymphoproliferative disorder, and nasopharyngeal carcinoma.
6 event or enhance treatment of EBV-associated nasopharyngeal carcinoma.
7 nd represent a promising future direction in nasopharyngeal carcinoma.
8 < .001) of this finding in the patients with nasopharyngeal carcinoma.
9 hought to be important to the development of nasopharyngeal carcinoma.
10 ality in 36% (11 of 31) of the patients with nasopharyngeal carcinoma.
11 is not found in EBV strains associated with nasopharyngeal carcinoma.
12 velopment of several malignancies, including nasopharyngeal carcinoma.
13 is the standard of care for locally advanced nasopharyngeal carcinoma.
14 ted with a variety of malignancies including nasopharyngeal carcinoma.
15 as the standard of care in locally advanced nasopharyngeal carcinoma.
16 ral forms of cancer, including lymphomas and nasopharyngeal carcinoma.
17 states exemplified by Burkitt's lymphoma and nasopharyngeal carcinoma.
18 sociated tumors such as Burkitt lymphoma and nasopharyngeal carcinoma.
19 ents with hemophilia, autoimmune disease, or nasopharyngeal carcinoma.
20 epistaxis for 2 years and was diagnosed with nasopharyngeal carcinoma.
21 th predisposition to autoimmune diseases and nasopharyngeal carcinoma.
22 ese cell types, such as Burkitt lymphoma and nasopharyngeal carcinoma.
23 all survival over chemoradiotherapy alone in nasopharyngeal carcinoma.
24 ty profile in patients with stage T(any)N3M0 nasopharyngeal carcinoma.
25 t line treatment for recurrent or metastatic nasopharyngeal carcinoma.
26 manageable toxicities in platinum-resistant nasopharyngeal carcinoma.
27 rate biomarker for the routine management of nasopharyngeal carcinoma.
28 arget volume delineation for radiotherapy in nasopharyngeal carcinoma.
29 atlas for neck target volume delineation in nasopharyngeal carcinoma.
30 cies Hodgkin and Burkitt lymphoma as well as nasopharyngeal carcinoma.
31 val in patients with locoregionally advanced nasopharyngeal carcinoma.
32 as an aetiological factor in B lymphomas and nasopharyngeal carcinoma.
33 oth lymphoid and epithelial cells, including nasopharyngeal carcinoma.
34 e development of multiple cancers, including nasopharyngeal carcinoma.
35 showed that it improves overall survival in nasopharyngeal carcinoma.
36 as Burkitt and Hodgkin lymphoma, as well as nasopharyngeal carcinoma.
37 also causes nonlymphoid malignancies such as nasopharyngeal carcinoma.
38 hogen associated with Burkitt's lymphoma and nasopharyngeal carcinoma.
39 with the development of B cell lymphomas and nasopharyngeal carcinomas.
40 viral reactivation occurs in EBV-associated nasopharyngeal carcinomas.
41 1.23-2.54), liver cancer (1.70, 1.10-2.63), nasopharyngeal carcinoma (26.05, 11.77-57.65), and lymph
42 cell lines from patients with EBV-associated nasopharyngeal carcinoma (3 out of 22, P = 0.0003), wher
43 (EBV), which is the causative agent of most nasopharyngeal carcinoma, also employs several immunosup
44 ated recent trends in the incidence rates of nasopharyngeal carcinoma among Chinese living in Los Ang
45 olleagues report these results for recurrent nasopharyngeal carcinoma, an aggressive malignancy assoc
48 expressed in the EBV-associated malignancies nasopharyngeal carcinoma and Burkitt's lymphoma, and are
50 e protein 1 (LMP1) gene has been reported in nasopharyngeal carcinoma and EBV-associated malignant ly
51 n the EBV-associated epithelial malignancies nasopharyngeal carcinoma and gastric carcinoma, and thes
52 d association with the epithelial malignancy nasopharyngeal carcinoma and has also been reported in o
53 ithin the terminal repeats is active in both nasopharyngeal carcinoma and Hodgkin's disease tissues.
54 pattern is typical of EBV gene expression in nasopharyngeal carcinoma and Hodgkin's disease, and diff
55 essed in primary biopsies from patients with nasopharyngeal carcinoma and Hodgkin's disease, where LM
56 munotherapeutic strategies to tumors such as nasopharyngeal carcinoma and Hodgkin's lymphoma that hav
58 ears; age range, 27-68 years) with untreated nasopharyngeal carcinoma and in 31 control subjects (17
59 pAIR correctly identifies every patient with nasopharyngeal carcinoma and inflammatory bowel disease
60 EBV) LMP1 protein is frequently expressed in nasopharyngeal carcinoma and is essential for the transf
61 nvolved in ATRA-induced growth inhibition of nasopharyngeal carcinoma and may suppress EMT through PI
63 chemotherapy in patients with non-metastatic nasopharyngeal carcinoma and obtained updated data for p
64 y in certain epithelial pathologies, such as nasopharyngeal carcinoma and oral hairy leukoplakia, ind
65 in-Barr virus (EBV)-associated malignancies, nasopharyngeal carcinoma and posttransplant lymphoma, ra
66 iously reported isolates from EBV-associated nasopharyngeal carcinomas and Burkitt's lymphomas occurr
67 produced complete remissions of EBV-positive nasopharyngeal carcinomas and lymphomas developing in im
68 posttransplant lymphomas, Hodgkin's disease, nasopharyngeal carcinoma, and AIDS-related lymphomas.
69 ion of tumors - including related lymphomas, nasopharyngeal carcinoma, and gastric adenocarcinoma - i
73 pment of Burkitt lymphoma, Hodgkin lymphoma, nasopharyngeal carcinoma, and other EBV-associated disea
74 onsistently been associated with the risk of nasopharyngeal carcinoma, and patients with Hodgkin lymp
75 rasts with EBV-associated Hodgkin's disease, nasopharyngeal carcinoma, and post-transplant lymphoprol
77 ithelial cells derived from gastric cancers, nasopharyngeal carcinomas, and normal oral keratinocytes
78 s endoscopy, a common investigation tool for nasopharyngeal carcinoma, are recognised as a likely cau
79 hlights recent advances in the management of nasopharyngeal carcinoma as a result of these endeavors.
80 ssociated with epithelial cell malignancies (nasopharyngeal carcinoma) as well as B-cell malignancies
82 of an EBV-based screening strategy to detect nasopharyngeal carcinoma at early stages and hopefully r
83 primary EBV-positive Hodgkin's lymphoma and nasopharyngeal carcinoma biopsies correlates with the nu
84 a herpesvirus linked to malignancies such as nasopharyngeal carcinoma, Burkitt's lymphoma, and Hodgki
85 man herpesvirus, is latently present in most nasopharyngeal carcinomas, Burkitt lymphomas, and some g
86 (37 of 62 subjects) among the patients with nasopharyngeal carcinoma but only 3% (two of 62 subjects
88 therapy in the treatment of locally advanced nasopharyngeal carcinoma, but its reproducibility was un
89 ove the outcomes for locoregionally advanced nasopharyngeal carcinoma by testing the feasibility and
90 he cell cycle, was consistently expressed in nasopharyngeal carcinomas by immunohistochemical analysi
91 iR-BART6 found in Daudi Burkitt lymphoma and nasopharyngeal carcinoma C666-1 cell lines suppressed pr
92 on in epithelial cells (including the C666-1 nasopharyngeal carcinoma cell line), although Na does no
94 originally reported but also in a number of nasopharyngeal carcinoma cell lines and a gastric carcin
96 lytic viral gene expression in EBV-positive nasopharyngeal carcinoma cells and lymphoblastoid cells,
97 llular accumulation of HIF-1alpha of hypoxic nasopharyngeal carcinoma cells and mediates the radiatio
99 s type I interferon production, and in human nasopharyngeal carcinoma cells results in almost complet
103 4 X 10(6) folate-receptor-positive KB (human nasopharyngeal carcinoma) cells into athymic mice yielde
104 izing action on KBvin (vincristine-resistant nasopharyngeal carcinoma) cells, a multidrug resistant c
105 + cells from the two donor groups, KB (human nasopharyngeal carcinoma) cells, and M07e (human megakar
106 8 x 10(6) folate receptor-positive KB (human nasopharyngeal carcinoma) cells, yielding 0.2- to 0.6-g
109 ts in the hypoxic regions of tumor formed by nasopharyngeal carcinoma CNE2 cells and breast cancer MD
110 ing Epstein-Barr virus serologic testing for nasopharyngeal carcinoma delivered every 1 to 4 years wa
112 e guidelines are in keeping with advances in nasopharyngeal carcinoma diagnosis and treatment, embody
114 al carcinoma patients and 5860 controls from nasopharyngeal carcinoma endemic areas identifies a tota
115 at are consistently EBV genome positive (eg, nasopharyngeal carcinoma, endemic Burkitt lymphoma), thi
116 ients older than 18 years with stage IIB-IVB nasopharyngeal carcinoma from 19 centres in North Americ
117 ogies ranging from oral hairy leukoplakia to nasopharyngeal carcinoma, from infectious mononucleosis
118 d with several human malignancies, including nasopharyngeal carcinoma, gastric cancer, and lymphoma.
119 he development of various cancers, including nasopharyngeal carcinoma, gastric cancer, Burkitt lympho
120 to this systematic review and meta-analysis, nasopharyngeal carcinoma has a significantly higher ADC
123 previously in EBV-associated tumors such as nasopharyngeal carcinoma, Hodgkin's disease (HD), and T/
124 , lung cancer (HR, 3.49; 95% CI, 2.08-5.88), nasopharyngeal carcinoma (HR, 2.79; 95% CI, 1.27-6.12),
125 of endemicity.Keywords: Epstein-Barr virus, nasopharyngeal carcinoma, humoral immune response, antib
126 may have implications for the development of nasopharyngeal carcinoma in high-risk populations in whi
135 d chemoradiation treatment for patients with nasopharyngeal carcinoma is feasible, and might delay th
137 , and face-to-face consultations in managing nasopharyngeal carcinoma is urgently needed amid the cur
138 V) is invariably present in undifferentiated nasopharyngeal carcinomas, is found sporadically in othe
140 analysis of global gene expression in human nasopharyngeal carcinoma KB cells grown in folate-deplet
141 of all compounds were tested versus a human nasopharyngeal carcinoma (KB) cell line to enable an est
142 ked to the development of cancers, including nasopharyngeal carcinoma, lymphomas, and gastric cancer.
143 man malignancies including Burkitt lymphoma, nasopharyngeal carcinoma, lymphoproliferative disease an
144 related central nervous system lymphomas and nasopharyngeal carcinomas) may allow novel, EBV-based ta
145 in about half of the patients with untreated nasopharyngeal carcinoma, may reflect tumor proximity to
146 ein 1 (LMP1), is detected in at least 70% of nasopharyngeal carcinoma (NPC) and all EBV-infected prei
147 can cause various human malignancies such as nasopharyngeal carcinoma (NPC) and gastric cancer (GC).
148 U, expression is frequently downregulated in nasopharyngeal carcinoma (NPC) and many other tumors due
149 urrently involved in structural variation in nasopharyngeal carcinoma (NPC) and the identification of
156 g Burkitt's lymphoma Rael cells and cultured nasopharyngeal carcinoma (NPC) cells contained nuclear S
159 ypes, including Burkitt lymphoma (BL) cells, nasopharyngeal carcinoma (NPC) cells, and lymphoblastoid
163 tion and acquired genetic alterations during nasopharyngeal carcinoma (NPC) development remains vague
164 m to assess its ability of real-time in vivo nasopharyngeal carcinoma (NPC) diagnosis and post-treatm
166 ted tumors such as Hodgkin lymphoma (HL) and nasopharyngeal carcinoma (NPC) express FasL, we determin
169 al T lymphocyte infiltrate found in cases of nasopharyngeal carcinoma (NPC) has been implicated in th
171 er, the role of Met/HGF signaling pathway in nasopharyngeal carcinoma (NPC) has not been evaluated.
172 the correlation between APLNR expression and nasopharyngeal carcinoma (NPC) has not been reported.
191 reatment of EBV-associated cancer.IMPORTANCE Nasopharyngeal carcinoma (NPC) is highly associated with
192 late-stage Epstein-Barr virus (EBV)-positive nasopharyngeal carcinoma (NPC) is incurable, and its tre
202 d with acute side effects of radiotherapy in nasopharyngeal carcinoma (NPC) remain largely unknown.
206 de polymorphisms (SNPs) in PIN1 promoter and nasopharyngeal carcinoma (NPC) risk with a small sample
209 the expression of MMP-2, MMP-9, and VEGF in nasopharyngeal carcinoma (NPC) tumors by using three NPC
210 mprehensive profile of EBV miRNAs in primary nasopharyngeal carcinoma (NPC) tumors including estimate
211 sine (cidofovir) on the EBV-associated tumor nasopharyngeal carcinoma (NPC) was evaluated in NPC xeno
212 d brain injury (RBI) model and patients with nasopharyngeal carcinoma (NPC) who received radiation th
213 eatment of Epstein-Barr virus (EBV)-positive nasopharyngeal carcinoma (NPC) with EBV-specific cytotox
214 Genetic susceptibility is likely involved in nasopharyngeal carcinoma (NPC), a cancer caused by Epste
215 also elevated in the LMP1-expressing tumor, nasopharyngeal carcinoma (NPC), a highly invasive neopla
217 n EBV-transformed lymphoblastoid cell lines, nasopharyngeal carcinoma (NPC), a subset of Hodgkin's di
219 ansplant lymphoproliferative disease (PTLD), nasopharyngeal carcinoma (NPC), and AIDS-associated lymp
220 B cell lymphomas, Hodgkin's disease (HD) and nasopharyngeal carcinoma (NPC), and by its unique abilit
221 's lymphoma, diffuse large B-cell lymphomas, nasopharyngeal carcinoma (NPC), and lymphomas that devel
222 g tumors that have latent infection, such as nasopharyngeal carcinoma (NPC), and oral hairy leukoplak
223 BV is associated with the epithelial cancer, nasopharyngeal carcinoma (NPC), and the lymphoid maligna
224 survival rates in cancer patients, including nasopharyngeal carcinoma (NPC), breast cancer and hepato
225 tumor burden and prognosis in patients with nasopharyngeal carcinoma (NPC), but data on the relation
226 cellular differentiation is a key feature of nasopharyngeal carcinoma (NPC), but it also presents as
228 identified multiple susceptibility loci for nasopharyngeal carcinoma (NPC), but the underlying biolo
229 nant diseases, including Burkitt's lymphoma, nasopharyngeal carcinoma (NPC), certain types of lymphom
230 ly linked with human B-cell malignancies and nasopharyngeal carcinoma (NPC), establishes three types
231 related to the occurrence and development of nasopharyngeal carcinoma (NPC), gastric carcinoma (GC),
232 associated with many human cancers, such as nasopharyngeal carcinoma (NPC), Hodgkin's disease, and g
233 n-Barr virus (EBV), aetiologically linked to nasopharyngeal carcinoma (NPC), is the first human virus
234 sive-stage small cell lung cancer (ES-SCLC), nasopharyngeal carcinoma (NPC), non-small cell lung canc
235 the one viral protein uniformly expressed in nasopharyngeal carcinoma (NPC), represents a prime targe
238 ide association study (GWAS) derived PRS for nasopharyngeal carcinoma (NPC), using a multi-center stu
263 Radiotherapy is the standard therapy for nasopharyngeal carcinoma (NPC); however, radioresistance
264 es, including virtually all undifferentiated nasopharyngeal carcinomas (NPC) and a portion of gastric
267 sion between 31 laser-capture-microdissected nasopharyngeal carcinomas (NPCs) and 10 normal healthy n
269 n abundance between blood and throat washes, nasopharyngeal carcinomas (NPCs) from areas of endemicit
270 us (EBV) infection promotes undifferentiated nasopharyngeal carcinomas (NPCs) in humans, but the mech
271 to explore infectious agents associated with nasopharyngeal carcinomas (NPCs), we employed our high-t
272 for an increased hazard of myeloid leukemia, nasopharyngeal carcinoma, or other upper airway tumors f
273 e effective in the treatment of EBV-positive nasopharyngeal carcinomas passaged in nude mice than eit
276 ponses were rescued in approximately half of nasopharyngeal carcinoma patients by peptide and cytokin
277 our virus, is detectable in more than 90% of nasopharyngeal carcinoma patients in endemic regions.
278 MATERIAL/METHODS: The study included 10 nasopharyngeal carcinoma patients under the age of 18 ye
280 has been found in infectious mononucleosis, nasopharyngeal carcinoma, posttransplant lymphoma, and n
281 II trial of locally recurrent or metastatic nasopharyngeal carcinoma (R/M NPC) patients, first-line
282 plication of BRISK to archival tissue from a nasopharyngeal carcinoma resulted in identification of E
283 of whole-genome sequencing of EBV-associated nasopharyngeal carcinomas revealed that structural varia
285 international multidisciplinary panel of 50 nasopharyngeal carcinoma specialists from 17 countries a
287 parts of its sequences have been reported as nasopharyngeal carcinoma susceptibility protein and medu
288 with single-cell and bulk profiles, we find nasopharyngeal carcinoma susceptibility robustly associa
289 chemotherapy in patients with non-metastatic nasopharyngeal carcinoma that completed accrual before D
290 ologically confirmed recurrent or metastatic nasopharyngeal carcinoma that had progressed after at le
292 , solid tumors such as renal cell carcinoma, nasopharyngeal carcinoma, thymic carcinoma, meduloblasto
295 cal trial, 49 patients with stage T(any)N3M0 nasopharyngeal carcinoma were enrolled and received the
297 rican Joint Committee on Cancer stage II-IVb nasopharyngeal carcinoma were treated with 70-Gy concomi
299 originally identified in cDNA libraries from nasopharyngeal carcinoma, where they are expressed at hi
300 h heavily pretreated recurrent or metastatic nasopharyngeal carcinoma, with a manageable safety profi