ライフサイエンスコーパス: nasopharynx

1 nt sites of isolation (sputum > middle ear > nasopharynx).

2 ability to successfully colonise the murine nasopharynx.

3 g with reduced macrophage recruitment to the nasopharynx.

4 spite being necessary for clearance from the nasopharynx.

5 asopharynx cancer from other diseases of the nasopharynx.

6 patients there was a mass which narrowed the nasopharynx.

7 reduced ability to survive in the chinchilla nasopharynx.

8 aque variants were less able to colonize the nasopharynx.

9 icted pathogen most often found in the human nasopharynx.

10 olonize and is more rapidly cleared from the nasopharynx.

11 he 2 genes were positively associated in the nasopharynx.

12 d leads to clearance of pneumococci from the nasopharynx.

13 s in the clearance of S. pneumoniae from the nasopharynx.

14 lated >20-fold in the lung compared with the nasopharynx.

15 or maintaining meningococcal carriage in the nasopharynx.

16 impaired clearance of S. pneumoniae from the nasopharynx.

17 s (meningococcus) is a symbiont of the human nasopharynx.

18 pneumococcus better able to colonize of the nasopharynx.

19 pathogen that frequently colonizes the human nasopharynx.

20 when this bacterium was introduced into the nasopharynx.

21 se and initiates infection by colonizing the nasopharynx.

22 o appears to be required for survival in the nasopharynx.

23 commensal organisms that colonize the human nasopharynx.

24 ci, with a matching strain identified in the nasopharynx.

25 type increased bacterial colonization of the nasopharynx.

26 r clearance of S. pneumoniae from the murine nasopharynx.

27 st prevalent are the larynx, oropharynx, and nasopharynx.

28 is colonize overlapping regions of the human nasopharynx.

29 nly be carried asymptomatically in the human nasopharynx.

30 se and initiates infection by colonizing the nasopharynx.

31 intercuspal phase of jaw motion, sealing the nasopharynx.

32 of submucosal glands in the upper airway and nasopharynx.

33 ease and within different microniches of the nasopharynx.

34 se and initiates infection by colonizing the nasopharynx.

35 tively metabolizing and growing while in the nasopharynx.

36 ant in adherence and colonization within the nasopharynx.

37 ant to colonize as well as to persist in the nasopharynx.

38 ed significantly reduced colonization of the nasopharynx.

39 nflammation and S. pneumoniae disease in the nasopharynx.

40 h implications for colonization of the human nasopharynx.

41 ve infection, prevented GAS infection of the nasopharynx.

42 itively associated when they co-occur in the nasopharynx.

43 an opportunistic pathogen that colonizes the nasopharynx.

44 le preexisting commensal colonization of the nasopharynx.

45 ace and resources during colonization of the nasopharynx.

46 sease, is most commonly carried in the human nasopharynx.

47 Moraxella catarrhalis to colonize the human nasopharynx.

48 diverse pathogen whose primary niche is the nasopharynx.

49 The bacteria were eliminated from the nasopharynx 1 week earlier than that from the control co

50 ase-deficient mutant was eliminated from the nasopharynx 2 weeks earlier than the D39 parent strain.

51 ression at temperatures typical of the human nasopharynx (34 degrees C) or warmer anatomical sites du

52 ence of galactose, a main sugar of the human nasopharynx, a highly competitive microbial environment.

53 s of disease begins with colonization of the nasopharynx, a process that likely depends on bacterial

54 his organism begins with colonization of the nasopharynx, a process that probably depends on adherenc

55 umoniae adherence during colonization of the nasopharynx, a site normally devoid of glucose.

56 Although asymptomatic colonization of the nasopharynx almost invariably precedes disease, the crit

57 ease the duration of viral shedding from the nasopharynx among patients with pneumonia.

58 successful regulation of colonisation in the nasopharynx and a brisk alveolar macrophage-mediated imm

59 NTHI) is a commensal inhabitant of the human nasopharynx and a causative agent of otitis media and ot

60 cus) is a common commensal inhabitant of the nasopharynx and a frequent etiologic agent in serious di

61 neumoniae is a common colonizer of the human nasopharynx and a leading cause of bacterial pneumonia a

62 r to other childhood tumors which affect the nasopharynx and adenoidal hypertrophy.

63 low the kinetics of viral clearance from the nasopharynx and allow for viral interference with antiba

64 To persist in the human nasopharynx and as a pathogen throughout the airways, NT

65 carbon availability is distinct between the nasopharynx and bloodstream of adult humans: glucose is

66 s influenzae exclusively colonizes the human nasopharynx and can cause a variety of respiratory infec

67 oniae (the pneumococcus) colonizes the human nasopharynx and can cause invasive disease aided by the

68 Streptococcus pneumoniae (Spn) colonizes the nasopharynx and can cause pneumonia.

69 ajor human pathogen that colonises the human nasopharynx and causes a wide range of infections.

70 man pathogen that persistently colonizes the nasopharynx and causes disease when it invades the blood

71 Streptococcus pyogenes can also colonize the nasopharynx and elaborate superantigens.

72 oss of ldh decreased bacterial burden in the nasopharynx and enhanced bacteremia in mice.

73 nd chinchilla models of RSV infection of the nasopharynx and Eustachian tube.

74 -negative coccobacillus that is found in the nasopharynx and gastrointestinal tract of many wild and

75 e carrier sclA allele persisted in the mouse nasopharynx and had increased adherence to cultured epit

76 n association between viral detection in the nasopharynx and hospitalization for pneumonia.

77 c changes in diameter were often seen in the nasopharynx and in the hypopharynx of asymptomatic sleep

78 curate lesion conspicuity (especially in the nasopharynx and in the larynx), infiltration of adjacent

79 essential for colonization of the chinchilla nasopharynx and infection of the middle ear.

80 passages of bacteremic rats to colonize the nasopharynx and invade the bloodstream.

81 influenza A virus infection to colonize the nasopharynx and invade the middle ear in the chinchilla

82 of factors required for colonization of the nasopharynx and invasive disease, because of its strict

83 pneumococcus) frequently colonizes the human nasopharynx and is an important cause of pneumonia, meni

84 istent, opportunistic commensal of the human nasopharynx and is the leading cause of community-acquir

85 MRSA colonizes the nasopharynx and is thus exposed to inhalants, including

86 r, rlrA, is required for colonization of the nasopharynx and lung infection but is dispensable for sy

87 gh the anatomy and microenvironments between nasopharynx and lungs are different, a virulence factor

88 IgA(-/-) mice also cleared virus from the nasopharynx and lungs following heterosubtypic challenge

89 s is likely to be crucial for fitness in the nasopharynx and may play a role during invasive disease.

90 zation and inflammatory responses within the nasopharynx and middle ear chamber.

91 n the ability to colonize and persist in the nasopharynx and middle ear.

92 rtial response to IC received 61.2 Gy to the nasopharynx and neck, and patients with stable disease r

93 t association between virus detection in the nasopharynx and pneumonia hospitalization (odds ratio, 1

94 rcinomas of the breast, cervix, colon, lung, nasopharynx and prostate.

95 V3 antibodies, replicated efficiently in the nasopharynx and protected against challenge with wt huma

96 g the ability of S. pyogenes to colonize the nasopharynx and provides knowledge that could help lead

97 tive diplococcus that normally colonizes the nasopharynx and rarely infects the urogenital tract.

98 phylococcus aureus, a commensal of the human nasopharynx and skin, also causes invasive disease, most

99 umoniae, is required for colonization of the nasopharynx and survival and multiplication in the lung.

100 gitidis, typically a resident of the oro- or nasopharynx and the causative agent of meningococcal men

101 CFU of the SPD0420 mutant were lower in the nasopharynx and the lungs after intranasal infection, an

102 le in colonization and adherence between the nasopharynx and the lungs.

103 strain in its ability to survive in both the nasopharynx and the middle ear of the chinchilla.

104 een the high levels of carriage in the human nasopharynx and the rare incidence of disease.

105 ifference between isolates obtained from the nasopharynx and those obtained from sterile sites (blood

106 decreased its ability to colonize the mouse nasopharynx and to adhere to and be internalized by cult

107 nsal S. pneumoniae to disseminate beyond the nasopharynx and to elicit severe infections of the middl

108 A1 and the D39 parent strain to colonize the nasopharynx and to induce OM subsequent to intranasal in

109 Human bocavirus 1 (HBoV1) can persist in nasopharynx and tonsils.

110 ls in submucosal glands throughout the mouse nasopharynx and upper airways and AQP4 at the contralate

111 a volume marker present in the fluid-filled nasopharynx and upper trachea.

112 face-expressed proteins downregulated in the nasopharynx (and thus less subject to selection pressure

113 to eukaryotic cells, colonization of the rat nasopharynx, and ability to cause sepsis.

114 osphotyrosine on CpsD when cultured from the nasopharynx, and an O phenotype that phosphorylates CpsD

115 m the eyes, gastrointestinal tract, airways, nasopharynx, and female reproductive tract, thereby stro

116 lar expression on and off (both ways) in the nasopharynx, and group C strains are more likely to be n

117 primarily an obligate commensal of the human nasopharynx, and it is unclear why the bacterium has evo

118 ations into GAS in its 'normal' habitat, the nasopharynx, and its ability to either live with its hos

119 ion, decreased numbers of macrophages in the nasopharynx, and less effective clearance.

120 o reflect successful adaptation to the human nasopharynx, and might provide a paradigm for DNA repair

121 All meningococci are carried in the nasopharynx, and most genotypes are very infrequently as

122 microbial communities (such as patient skin, nasopharynx, and stool) as well as environmental biofilm

123 al sites such as the intestine, oral cavity, nasopharynx, and vagina all have associated commensal fl

124 i) is a commensal microorganism of the human nasopharynx, and yet is also an opportunistic pathogen o

125 I) lung, (II) lung and blood, (III) lung and nasopharynx,and (IV) all three tissues were identified,

126 minant SpnIII alleles expressed in the human nasopharynx are significantly different than those predi

127 tion, whereby pneumococci harvested from the nasopharynx are typically transparent, while those simul

128 ptococcus pneumoniae naturally colonizes the nasopharynx as a commensal organism and sometimes causes

129 reptococcus pneumoniae commonly inhabits the nasopharynx as a member of the commensal biofilm.

130 S. pneumoniae to colonize and persist in the nasopharynx as well as the middle ear.

131 cluding regions of the respiratory tract and nasopharynx, as well as in a subset of lung tumour cell

132 (NTHI) bacteria are commensals in the human nasopharynx, as well as pathogens associated with a spec

133 l vertebrates such as birds and mammals, the nasopharynx-associated lymphoid tissue (NALT) represents

134 In rainbow trout (Oncorhynchus mykiss), a nasopharynx-associated lymphoid tissue (NALT) was recent

135 D8+ T-cell response, including TRM cells, in nasopharynx-associated lymphoid tissue from children and

136 atory epithelium covering nasal passages and nasopharynx-associated lymphoid tissue than H5N1 viruses

137 CD8+ T cell response including TRM cells in nasopharynx-associated lymphoid tissue(NALT) from childr

138 fic T-cell response, including TRM cells, in nasopharynx-associated lymphoid tissue, demonstrating it

139 2 (but not CK10 or CK13) expression in trout nasopharynx-associated lymphoid tissue.

140 enzae (NTHi) efficiently colonizes the human nasopharynx asymptomatically but also causes respiratory

141 Nasopharynx auditory tube glands fail to develop in HED

142 ved also in colonization and invasion of the nasopharynx, biofilm formation and evasion of host immun

143 occus pneumoniae is a commensal of the human nasopharynx but can cause invasive diseases, including o

144 ay aid in the persistence of NTSp within the nasopharynx but is not involved in invasion.

145 a commensal microbe that colonizes the human nasopharynx but occasionally invades the bloodstream to

146 plicated in virulence and persistence in the nasopharynx, but its role in biofilms has not been studi

147 n micro-dissected compartments of the bovine nasopharynx by microarray.

148 Colonization of the human nasopharynx by Moraxella catarrhalis is presumed to invo

149 Colonization of the nasopharynx by S. pneumoniae precedes pulmonary and othe

150 orrelation between colonization of the human nasopharynx by Streptococcus pneumoniae and Haemophilus

151 Asymptomatic colonization of the nasopharynx by Streptococcus pneumoniae precedes pneumoc

152 osis and differential diagnosis of childhood nasopharynx cancer from other diseases of the nasopharyn

153 ribution of MR imaging features of childhood nasopharynx cancer.

154 ry of the agent came first as in stomach and nasopharynx cancers, and epidemiology has been concerned

155 ysis by the Meta-Analysis of Chemotherapy in Nasopharynx Carcinoma (MAC-NPC) collaborative group to a

156 ighlights how the bacterial ecosystem of the nasopharynx changes the nature and course of pneumococca

157 licA and igaB are important for early human nasopharynx colonization.

158 tudied to what extent the HBoV1 DNA loads in nasopharynx correlate with acute infection markers.

159 ts verified that the surface pH of the human nasopharynx could be transiently lowered to pH approxima

160 f anti-S. pneumoniae responses in the murine nasopharynx during colonization.

161 mmatory cytokine and chemokine levels in the nasopharynxes during viral URIs, and examined the differ

162 S. pneumoniae adhesion to and invasion into nasopharynx epithelia, for its ability to induce protect

163 es from the airways, gastrointestinal tract, nasopharynx, female reproductive tract and the surface o

164 e production and cellular recruitment to the nasopharynx following colonization.

165 oping neoplasms from radium treatment of the nasopharynx for adenoid hypertrophy.

166 Heavy nasopharynx foreign body load and loss of gland protecti

167 ldren with additional Gram + bacteria in the nasopharynx (Gr+/-).

168 number of NTSp isolates colonizing the human nasopharynx has been observed, but the colonization fact

169 raditionally, respiratory specimens from the nasopharynx have been considered to have the highest sen

170 s a commensal bacterial species of the human nasopharynx; however, in the context of prior or concurr

171 ion elicited a robust IL-17A response in the nasopharynx; IL-17A neutralization reduced S. pneumoniae

172 We conclude that carbon availability in the nasopharynx impacts pneumococcal biofilm formation in vi

173 found to be required for colonization of the nasopharynx in a carriage model.

174 l cells, and enhanced clearance in lungs and nasopharynx in a mouse aerosol challenge model.

175 l cells, and enhanced clearance in lungs and nasopharynx in a mouse aerosol challenge model.

176 y be expressed on the mucosal surface of the nasopharynx in bactericidal concentrations.

177 serious disease and common colonizers of the nasopharynx in children.

178 irulence and higher capacity to colonize the nasopharynx in healthy individuals.

179 unilateral pharyngeal tonsil and ipsilateral nasopharynx in one).

180 thelium of the dorsal soft palate and dorsal nasopharynx in persistently infected cattle.

181 gene on the epithelial surface of the human nasopharynx in situ and inducible transcription in epith

182 infection, but also frequently colonizes the nasopharynx in the absence of disease.

183 orrelated with their ability to colonize the nasopharynx in vivo, with colonization-deficient strains

184 s required for efficient colonization of the nasopharynx in vivo.

185 Pathogens that colonize the nasopharynx, including Staphylococcus aureus, Streptococ

186 arious neisserial co-colonizers of the human nasopharynx increased N. meningitidis switching frequenc

187 colonization experiments of murine lung and nasopharynx, indicating a role for raffinose and stachyo

188 e mutant is more attenuated than scrH in the nasopharynx, indicating SusH can substitute in this nich

189 Asymptomatic carriage in the nasopharynx is a prerequisite for disease, but variabili

190 the connection between the nasal airway and nasopharynx is completely blocked.

191 Asymptomatic colonisation of the nasopharynx is considered to be a prerequisite for these

192 f Haemophilus influenzae type b to the human nasopharynx is facilitated by Hib pili, filaments expres

193 g how NTSp colonizes and survives within the nasopharynx is important due to the increase in NTSp car

194 t Streptococcus pneumoniae colonization, the nasopharynx is sampled using a swab placed in skim milk-

195 Colonization of the nasopharynx is the initial step in all infections caused

196 The human nasopharynx is the main reservoir for Streptococcus pneu

197 propose that the default role for DC in the nasopharynx is to maintain tolerance/ignorance of the la

198 specific function in the colonization of the nasopharynx is unknown.

199 human upper respiratory tract, including the nasopharynx, is colonized by a diverse array of microorg

200 or detecting Streptococcus pneumoniae in the nasopharynx lack sensitivity.

201 e and forms highly organized biofilms in the nasopharynx, lungs, and middle ear mucosa.

202 vivo Spn and mouse transcriptomes within the nasopharynx, lungs, blood, heart, and kidneys using thre

203 Its adaptation for survival in the human nasopharynx makes the meningococcus a highly successful

204 of pneumococci of various serotypes from the nasopharynx mediated by the cytokine IL-17A.

205 can be reliably measured in the oesophagus, nasopharynx, mouth, and bladder.

206 ological features of childhood tumors of the nasopharynx must be well known.

207 the sinus (n = 5), the ear (n = 2), and the nasopharynx (n = 18); isolates were recovered from 49 ch

208 An increase in Streptococcus pneumoniae nasopharynx (NP) colonization density during a viral coi

209 The nasopharynx (NP) is a reservoir for microbes associated

210 moniae is a commensal colonizer of the human nasopharynx (NP) that causes disease after evasion of ho

211 e oral cavity (OSCC), oropharynx (OPSCC) and nasopharynx (NPC).

212 mples, external ear canal (EEC) lavages, and nasopharynx (NPH) samples from children with OME.

213 high loads of HBoV1 DNA were detected in the nasopharynx of 14 (7%) primarily asymptomatic subjects w

214 while virus selectively persisted within the nasopharynx of a subset of animals.

215 Its normal habitat is the nasopharynx of adult humans.

216 enzae, both of which frequently colonize the nasopharynx of children, were more common in females, su

217 ll drug-resistant pneumococci colonizing the nasopharynx of healthy children attending day care.

218 of M. catarrhalis strains, isolated from the nasopharynx of healthy children or middle ear effusions

219 lar methods in detecting pneumococcus in the nasopharynx of healthy individuals and to evaluate the a

220 umoniae (the pneumococcus) is carried in the nasopharynx of healthy individuals, but can spread to ot

221 CR improved detection of pneumococcus in the nasopharynx of healthy individuals.

222 of meningococcal isolates recovered from the nasopharynx of human carriers.

223 in NTHi 2019Str(R)1 was used to colonize the nasopharynx of human subjects in a study of experimental

224 Streptococcus pneumoniae colonizes at the nasopharynx of humans and is able to disseminate and cau

225 As the flora residing in the nasopharynx of infants can be in contact with ingested f

226 The nasopharynx of infants is a major ecological reservoir o

227 f the cop operon is induced in the lungs and nasopharynx of intranasally infected mice, and a copA(-)

228 number of interfering organisms found in the nasopharynx of smokers revert to normal levels after com

229 sms were recovered from the nasal cavity and nasopharynx of the animals in numbers sufficient for DNA

230 s and an isogenic TFP mutant to colonize the nasopharynx of the chinchilla.

231 ogether, our observations highlight that the nasopharynxes of children in DCCs are a melting pot of s

232 reptococcus pneumoniae cocolonization in the nasopharynxes of humans can be attributed to hydrogen pe

233 and experiments where S. aureus invaded the nasopharynxes of rats with established S. pneumoniae pop

234 or the autocthonous S. aureus colonizing the nasopharynx or conjunctiva or lid margin to be a reservo

235 te wire swab either nasally to the posterior nasopharynx or orally to the posterior oropharynx.

236 in the lungs but not for colonization in the nasopharynx or replication in the bloodstream during sep

237 ORF3 in HT29.14S (colon), HeLa (cervix), KB (nasopharynx), or LNCaP (prostate) cancer cell lines, in

238 Motion of the nasopharynx, oropharynx, and hypopharynx was characteriz

239 The nasopharynx, oropharynx, and hypopharynx were characteri

240 sites, including the anterior and posterior nasopharynx, oropharynx, and nares.

241 and 240 polymicrobial samples (obtained from nasopharynx, oropharynx, and saliva; 80 from each site)

242 infection had lower levels of virus in their nasopharynx/oropharynx than symptomatic children, but th

243 rates from 29 cancers: lip and oral cavity; nasopharynx; other pharynx; esophageal; stomach; colon a

244 M. catarrhalis to survive in the chinchilla nasopharynx over a 3-day period.

245 ); and mean change in airway diameter of the nasopharynx (P <.001) and hypopharynx (P <.001).

246 otion in the oropharynx (P =.006) and in the nasopharynx (P <.006) but not in the hypopharynx (P =.65

247 airways (including the paranasal sinuses and nasopharynx) play an important role as a silent reservoi

248 ssociated with cancer of the nasal cavities, nasopharynx, prostate, lung, and pancreas; however, thes

249 n blocks NTHI colonization of the chinchilla nasopharynx, providing the first demonstration of a role

250 and who had tissue diagnosis and MRI of the nasopharynx region.

251 LD2 is expressed in the mandible, palate and nasopharynx regions during craniofacial development at E

252 sparent, selected for in the bloodstream and nasopharynx, respectively.

253 We also characterized oral and nasopharynx samples during RTI episodes in the first hal

254 in which we collected questionnaires and 324 nasopharynx samples from 20 infants with CF and 45 age-m

255 Individuals harbouring bacteria in the nasopharynx serve as reservoirs for intrafamilial and no

256 the 148 subjects (mean age, 3.4 years), the nasopharynx showed dynamic motion in 53 (36%).

257 l NTHi challenge, colonization of the murine nasopharynx significantly dropped in animals formerly im

258 ubclinical bacterial colonization within the nasopharynx, similar to IAV.

259 following parameters between the two groups: nasopharynx SP (P <.001) and IC (P <.001); hypopharynx S

260 We found that in the murine nasopharynx, SpeB enhanced IL-1beta-mediated inflammatio

261 dispersal include IAV-induced changes in the nasopharynx, such as increased temperature (fever) and e

262 tion of mice and were unable to colonize the nasopharynx, suggesting a diminished capacity to sense o

263 e infections and were unable to colonize the nasopharynx, suggesting that the failure to produce caps

264 4 isolate during cocolonization in the mouse nasopharynx, suggesting that the locus is functional in

265 most frequently detected pathogen in either nasopharynx swab specimens, stool specimens, serum sampl

266 enzae is a human-restricted commensal of the nasopharynx that can also be associated with disease.

267 ommunities during colonization of the murine nasopharynx that display increased antibiotic resistance

268 For example, in the nasopharynx the wild-type serotype 3 strain and the caps

269 ompetence pathway during colonization of the nasopharynx, the principal ecological niche of the pneum

270 ectly within the physical environment of the nasopharynx, thereby explaining why lineage structure is

271 occasion, meningococci disseminate from the nasopharynx to cause invasive disease.

272 by the bacterium during progression from the nasopharynx to the blood.

273 Pneumococci are transported from the nasopharynx to the lungs following exposure to DEPs, lea

274 rbon caused S. pneumoniae to spread from the nasopharynx to the lungs, which is essential for subsequ

275 th the dissemination of pneumococci from the nasopharynx to the middle ear.

276 ficantly lower numbers of pneumococci in the nasopharynx, trachea, and lungs.

277 ant effects on pneumococcal infection of the nasopharynx, trachea, and lungs.

278 SK2a-expressing strains are associated with nasopharynx tropicity, and many of these strains express

279 early, complete eradication of NTHI from the nasopharynx was highly protective, reduction of the bact

280 to induce total eradication of NTHI from the nasopharynx was not equivalent among NTHI groups.

281 late moved upward as the jaw opened, but the nasopharynx was not sealed.

282 Further, bacterial colonization of the nasopharynx was prevented in mice immunized with HA-KO/P

283 ion in 12 (67%), and viral shedding from the nasopharynx was prolonged for 7 days or longer among 15

284 NTHI residing in the nasopharynx was rapidly cleared from this site, thus pre

285 C of the oral cavity, oropharynx, larynx, or nasopharynx was used.

286 M. catarrhalis to persist in the chinchilla nasopharynx were upregulated in the mesR mutant.

287 ued adherence of H. influenzae type b to the nasopharynx, where the three-stranded Hib pilus filament

288 of adult humans: glucose is absent from the nasopharynx, whereas galactose is abundant.

289 Unlike the phase variants detected in the nasopharynx, which have at least 20% of the parental amo

290 nces pneumococcal colonization of the murine nasopharynx, which in turn promotes bacterial spread to

291 ltiple pneumococcal strains can colonize the nasopharynx, which is also home to many other bacterial

292 ed within the microenvironments of the human nasopharynx, which NTHI colonizes asymptomatically, and

293 hat CTB reduces the pneumococcal load in the nasopharynx, which required activation of the caspase-1/

294 h as influenza, leads to inflammation in the nasopharynx with an increased temperature and recruitmen

295 ciation of vaccine-serotype detection in the nasopharynx with CAAP case status, among vaccinated and

296 Colonization of the nasopharynx with Streptococcus pneumoniae (Spn), althoug

297 gitidis is a frequent colonizer of the human nasopharynx, with asymptomatic carriage providing the re

298 may extend to the resident microbiota of the nasopharynx, with implications for the pathogenesis of r

299 ms by which S. pneumoniae colonize the human nasopharynx without inducing damaging host inflammation

300 nt infection or a carrier state in the human nasopharynx without overt disease symptoms but the prese