ライフサイエンスコーパス: natural killer

1 ented modification of GluK2/3 with the human natural killer-1 (HNK-1) glycan, a modulator of GluK2/3

2 transient increases in proliferating (Ki67+) natural killer and CD8 T cells.

3 NKG7 function in natural killer and CD8(+) T cells was linked with their

4 were decelerated in SZ related to antitumor natural killer and CD8T cells, which may help explain co

5 strains of mice show increased lung B cell, natural killer and T cell effector responses in the lung

6 tment, together with a loss in B lymphocyte, natural killer and T cell effector responses.

7 ors and chimeric antigen receptor-expressing natural killer and T cells, the overall survival of pati

8 ine (Thr; T), which differentially influence natural killer and T-cell alloresponses.

9 number and functions of tumour-infiltrating natural-killer and CD8(+) T lymphocytes.

10 vs 13.7%; P = .03) but similar levels of B, natural killer, and CD4 T lymphocytes.

11 reased infiltration of F4/80(+) macrophages, natural killer, and CD8 T cells, displaying a central me

12 tivity; promoted the activation of CD8(+) T, natural killer, and dendritic cells in relevant tissues;

13 n in situ modify the tumor cell surface with natural killer cell (NK cell)-activating signals to achi

14 ent phagocytosis, cellular cytotoxicity, and natural killer cell activation were assessed.

15 cyte phagocytosis, complement activation and natural killer cell activation, are substantially enhanc

16 Natural killer cell deficiencies (NKDs) are an emerging

17 Human natural killer cell deficiency (NKD) arises from inborn

18 c constraint in macrometastases triggered by natural killer cell depletion suggests a dynamic interpl

19 nt SCID phenotype with a T-cell, B-cell, and natural killer cell developmental defect and hypogammagl

20 of molecules that seek to subvert T cell and natural killer cell function via a remarkable array of m

21 r type 2 inflammation, T(H)17 signaling, and natural killer cell function.

22 Here, we show that natural killer cell granule protein 7 (NKG7) is a regula

23 llular context, supporting the hypothesis of natural killer cell mechanosensitivity.

24 regulation of innate immunity in infants and natural killer cell networks in children, and additional

25 Furthermore, a shift in the natural killer cell phenotype was observed with features

26 e group 3-like ILCs was not dependent on the natural killer cell receptor (NCR1), since NCR1-deficien

27 by which CMVs evade or reprogram T cell and natural killer cell responses in vivo However, the role

28 anti-CD137 MAb that can activate T cell and natural killer cell responses to clear tumors.

29 ctor]) coupled with T-helper cell type 2 and natural killer cell signaling in children.

30 okines with an additional role of T(H)17 and natural killer cell signaling.

31 choalveolar lavage samples and repression of natural killer cell- and T cell-associated transcripts i

32 LSCs) in acute myeloid leukemia downregulate natural killer cell-activating receptor ligands to evade

33 omics, along with CRISPR/Cas9-KO cell lines, natural killer cell-killing assays, and RNA-Seq experime

34 NSG mice, pointing to the potential role of natural killer cell-mediated antibody-dependent cell cyt

35 the D1 domain attenuated Necl-5 binding and natural killer cell-mediated cytotoxicity.

36 phils without IL-5 was only seen in EOs, and natural killer cell-mediated eosinophil killing was seen

37 f DST bm1 cells than F1 cells was reduced by natural killer-cell depletion.

38 ning 64 targets deployed in T cells (CAR-T), natural killer cells (CAR-NK) or mixtures (CAR-NK/T) fro

39 D1c + myeloid DCs; neutrophils; macrophages; natural killer cells (NK); Marginal Zone-like B cells (M

40 Cytotoxic T-lymphocytes (CTLs) and natural killer cells (NKs) kill compromised cells to def

41 rapy had an increased abundance of activated natural killer cells and a newly identified CD3(-)CD68(+

42 ed in significantly decreased frequencies of natural killer cells and a trend toward reduced ILC popu

43 regulatory cells, immunosuppressive CD56(hi) natural killer cells and ablation of proinflammatory muc

44 tiple populations of myeloid cells, T cells, natural killer cells and B cells that demonstrated both

45 d with increased accumulation of T cells and natural killer cells and decreased myeloid-derived suppr

46 IFNgamma-induced pathways and activation of natural killer cells and endothelial cells.

47 crophages and dendritic cells) and lymphoid (natural killer cells and innate lymphoid cells) cell pop

48 umor-derived PGE2 blocks early activation of natural killer cells and interferes with subsequent adap

49 an endogenous RNA editing enzyme in primary natural killer cells and lymphoma cell lines.

50 ther showed exosomes were mainly taken up by natural killer cells and macrophages in the lung.

51 n CD30 on HL cells and the CD16A receptor on natural killer cells and macrophages, to induce tumor ce

52 lin-like receptors (KIRs) which are found on natural killer cells and some T cells; for the CD94:NKG2

53 Cytotoxic T lymphocytes (T) and natural killer cells are the main cytotoxic killer cells

54 CD8(+) cytotoxic T lymphocytes (CTL) and natural killer cells are the main cytotoxic killer cells

55 er cells suppress the cytotoxic functions of natural killer cells by a variety of mechanisms.

56 Natural killer cells employ a diverse arsenal of effecto

57 dritic cells, and CD8 T lymphocytes and CD57 natural killer cells in the ALNs(-) were factors associa

58 umor cells via recruitment and activation of natural killer cells in the tumor.

59 NKG2A subset of CD56 natural killer cells increased substantially and persist

60 ial cells, dendritic cells, macrophages, and natural killer cells is crucial for its protection.

61 Our results suggest that natural killer cells modified with glycan ligands to CD2

62 In contrast to viral evasion of natural killer cells or T cell recognition, the evasion

63 A combination of serum and natural killer cells provided the most effective protect

64 By contrast, natural killer cells retained partial IL-2Rbeta expressi

65 Quantification of natural killer cells spreading on surfaces coated with t

66 RNA-seq analysis of natural killer cells subjected to cellular crowding and

67 22 ligands onto NK-92MI and cytokine-induced natural killer cells to achieve tumor-specific CD22 targ

68 also enhanced the activity and maturation of natural killer cells to effectively treat anti-PD-1 resi

69 d is secreted by cytotoxic T lymphocytes and natural killer cells to help eliminate virus-infected an

70 Additionally, NKG7 expressed by natural killer cells was critical for controlling cancer

71 Moreover, natural killer cells were involved in AS by increasing t

72 lteration in NKG2A and NKG2C subsets of CD56 natural killer cells which might have a pathogenic role

73 cells, myeloid-derived suppressor cells, and natural killer cells) as well as adaptive immune cells (

74 anulocytes, monocytes, T cells, B cells, and natural killer cells) in patients having undergone HSPC

75 on by cytotoxic cells (cytotoxic T cells and natural killer cells), and interleukin 12 production by

76 Using multiple human cell types, including natural killer cells, an IL-12 indicator cell line, and

77 ty controlled by T cells (T(H)1 and CD8(+)), natural killer cells, and antigen-presenting cells; T2 C

78 cells (Tregs), gammadelta T cells, B cells, natural killer cells, and primary and induced pluripoten

79 in the tumor, activating dendritic cells and natural killer cells, and recruiting the adaptive immune

80 acts as a costimulatory receptor on T cells, natural killer cells, B cell subsets, and some dendritic

81 luding monocytes, CD4(+) and CD8(+) T cells, natural killer cells, conventional and plasmacytoid dend

82 d to respond to vaccines, there were reduced natural killer cells, elevated regulatory T cells, M2-ty

83 s are applied to other immune cells, such as natural killer cells, hematopoietic cells or induced plu

84 oles for macrophages, innate lymphoid cells, natural killer cells, innate gammadelta T cells, and oth

85 hat monocytes, neutrophils, dendritic cells, natural killer cells, innate lymphoid cells-2, and CD (c

86 induces site-specific C-to-U RNA editing in natural killer cells, lymphoma cell lines, and, to a les

87 immune cells such as primary T and B cells, natural killer cells, macrophages, and primary microglia

88 cated that CD8 T cells, but not CD4 cells or natural killer cells, mediated elimination of KPC-Par-1(

89 ent CD8 + T cells, CD56 + T cells, CD56(dim) natural killer cells, monocytes and dendritic cells were

90 he number of tumor-infiltrating CD8(+) T and natural killer cells, slowed tumor growth, and improved

91 the cytokine milieu, macrophages/monocytes, natural killer cells, T cells, and neutrophils in severe

92 smatched grafts is driven by the recipient's natural killer cells, which overwhelmingly use perforin

93 eeks of infection, for example proliferating natural killer cells, which potentially may associate wi

94 es effectively recruited and activated human natural killer cells, while vaccine-elicited RM antibodi

95 eficiency disease (SCID) affecting B, T, and natural killer cells, with an almost complete lack of an

96 ding CD8+ cytotoxic T lymphocytes (CTLs) and natural killer cells-and regulated by Runt-related (Runx

97 dvantage in controlling DENV infection using natural killer cells.

98 cells, as well as decreased ICOS+ and 4-1BB+ natural killer cells.

99 el and exhibits SOX9-dependent resistance to natural killer cells.

100 ocytosis"), especially cytotoxic T cells and natural killer cells.

101 OXP3(+)T cells, CD11c(+) dendritic cells and natural killer cells.

102 intratumoral macrophages, and activation of natural killer cells.

103 -alpha, which drives IFN-gamma production by natural killer cells.

104 e variant and the homologous FcgammaRIIIa on natural killer cells.

105 D16(+) monocytes, innate lymphoid cells, and natural killer cells.

106 e polymorphic stress molecules recognized by natural killer cells.

107 MCMV-induced natural killer cytotoxicity was dependent on MyD88 and S

108 line to identify perturbations that enhance natural killer effector functions.

109 The ligands for the natural killer group 2 (NKG2D) protein render tumor cell

110 ed frequencies of cells expressing NKp30 and natural killer group 2, member D and an increased propor

111 ell-surface expression of immune stimulatory natural killer group 2D (NKG2D) ligands on the monocytes

112 d with defective expression of the antiviral natural-killer group 2 member D (NKG2D) protein and abno

113 l2rga(-/-) zebrafish that lacks adaptive and natural killer immune cells, can engraft a wide array of

114 C6 had the lowest natural killer infiltration rate and was represented by

115 Invariant natural killer (iNKT) cells are among the first innate i

116 ndoplasmic reticulum, membrane dynamics, and Natural Killer-mediated cytotoxicity.

117 nversely correlated with perforin-expressing natural killer (NK) and CD3+ T cells.

118 Instead, subsets of natural killer (NK) and so-called "unconventional" T cel

119 Although CXCL14 recruits natural killer (NK) and T cells to the tumor microenviro

120 To analyze human natural killer (NK) cell activation by both species, we

121 Differentiation of natural killer (NK) cell activation pathways occurs alon

122 We evaluated 2 patients with natural killer (NK) cell CAEBV and studied their NK cell

123 ility complex II upregulation and persistent natural killer (NK) cell cytolytic killing.

124 We conducted a systematic evaluation of natural killer (NK) cell cytotoxicity in adult patients

125 The mechanism by which natural killer (NK) cell education results in licensed N

126 Inhibitory signaling during natural killer (NK) cell education translates into incre

127 Smoking was also associated with a reduced natural killer (NK) cell frequency in peripheral blood,

128 Natural killer (NK) cell function is regulated by inhibi

129 Following NACT, increased natural killer (NK) cell infiltration and oligoclonal ex

130 CO-expressing macrophages, and activation of natural killer (NK) cell killing through TNF-related apo

131 Here, we demonstrate a systemic reduction in natural killer (NK) cell numbers in SRalpha-tTA/Tet-O-MY

132 Several studies suggest that harnessing natural killer (NK) cell reactivity mediated through kil

133 We show that an endogenous ligand for the natural killer (NK) cell receptor NKG2D, Retinoic Acid E

134 Genetic diversity in human natural killer (NK) cell receptors is linked to resistan

135 The impact of such HLA variation on natural killer (NK) cell recognition remains unclear.

136 Current evidence suggests that dysfunctional natural killer (NK) cell responses during hepatitis C vi

137 d how soy isoflavones and metabolites impact natural killer (NK) cell signaling and function.

138 ation, increased microbial translocation and natural killer (NK) cell skewing towards an inflammatory

139 In contrast, a CD27(+) Natural killer (NK) cell subset accumulated in the lungs

140 Herein, we profiled natural killer (NK) cell subsets in the blood of 72 asym

141 maternal antibodies differed, skewed toward natural killer (NK) cell-activating antibodies.

142 iselenide-pemetrexed assemblies that combine natural killer (NK) cell-based cancer immunotherapy with

143 ous types of cancer by unleashing both T and natural killer (NK) cell-mediated antitumor responses.

144 Natural Killer (NK) cell-mediated early innate defense r

145 estinal inflammation by protecting them from natural killer (NK) cell-mediated elimination.

146 id cells (ILC2s) orchestrated suppression of natural killer (NK) cell-mediated innate antitumor immun

147 Natural killer (NK) cell-mediated killing involves the m

148 tin-1-mediated activation of macrophages and natural killer (NK) cell-mediated metastasis control.

149 the activation of an immune cell known as a natural killer (NK) cell.

150 Natural killer (NK) cells acquire effector functions thr

151 BP134(AF), engineered to effectively harness natural killer (NK) cells afforded additional improvemen

152 reatment displayed increased infiltration by natural killer (NK) cells and CD8alpha(+) T cells, and a

153 Both natural killer (NK) cells and exosomes released from the

154 These include natural killer (NK) cells and gammadelta T cells of inna

155 l populations that express CD8alpha, such as natural killer (NK) cells and gammadelta T cells.

156 that in the brain, unlike in the periphery, natural killer (NK) cells and monocytes are not involved

157 lec, and is localised predominantly on human natural killer (NK) cells and monocytes.

158 us (ECTV), and that this resistance requires natural killer (NK) cells and other immune cells.

159 In natural killer (NK) cells and other innate lymphoid cell

160 was important for production of IFN-gamma by natural killer (NK) cells and recruitment of inflammator

161 ignaling to increase IFNgamma(+) T cells and natural killer (NK) cells and reduce the percentage of m

162 gher levels of IFN-gamma production by liver natural killer (NK) cells and stronger resistance to MCM

163 t group 1 ILCs consist of circulating mature natural killer (NK) cells and tissue-resident ILC1s, the

164 Natural killer (NK) cells are a critical component of th

165 Natural killer (NK) cells are a promising cellular immun

166 Natural killer (NK) cells are a subset of innate lymphoi

167 Human natural killer (NK) cells are critical for innate defens

168 Natural killer (NK) cells are critical mediators of host

169 Natural killer (NK) cells are critical to both innate an

170 Natural killer (NK) cells are cytotoxic lymphocytes of t

171 Natural killer (NK) cells are cytotoxic lymphocytes targ

172 Natural killer (NK) cells are cytotoxic type 1 innate ly

173 Human natural killer (NK) cells are defined as CD56(+)CD3(-).

174 Although innate lymphoid cells (ILC) and natural killer (NK) cells are found throughout the oral

175 Natural killer (NK) cells are frequent in human liver an

176 Natural killer (NK) cells are important antiviral effect

177 Natural killer (NK) cells are important effector cells i

178 Natural killer (NK) cells are important effectors of inn

179 Natural killer (NK) cells are important in the immune de

180 Natural killer (NK) cells are innate cytotoxic lymphocyt

181 Natural killer (NK) cells are innate effector lymphocyte

182 Natural killer (NK) cells are innate lymphocytes capable

183 Natural killer (NK) cells are innate lymphocytes that ar

184 Natural killer (NK) cells are innate lymphocytes that ar

185 Natural killer (NK) cells are innate lymphocytes that ex

186 Natural killer (NK) cells are innate lymphoid cells bein

187 press programmed death-ligand 1 (PD-L1), and natural killer (NK) cells are involved in trophoblast im

188 Natural killer (NK) cells are key effector cells of inna

189 Natural killer (NK) cells are key innate immunity effect

190 The promise of natural killer (NK) cells as effectors in cancer cellula

191 Natural killer (NK) cells belong to the innate immune sy

192 layer system, which mimics the activation of natural killer (NK) cells by antibody-dependent cell-med

193 al/phenotypic changes induced in CD8(+) T or natural killer (NK) cells by MTEX or non-MTEX were compa

194 Natural killer (NK) cells can recognize virus-infected a

195 Natural killer (NK) cells control viral infection throug

196 Natural killer (NK) cells develop from common progenitor

197 Recently, we and others have shown that natural killer (NK) cells exhibit memory-like recall res

198 A population of Natural Killer (NK) cells expressing the activating rece

199 nstrate a strong interdependency of MPhi and natural killer (NK) cells for efficient tumor cell killi

200 Natural killer (NK) cells form immune synapses to ascert

201 , natural killer T-like cell (NKT-like), and natural killer (NK) cells from patients with BOS (n = 10

202 Natural killer (NK) cells have an important role in immu

203 Natural Killer (NK) cells have an important role in immu

204 IMPORTANCE Epidemiological studies show that natural killer (NK) cells have anti-HIV activity: they a

205 Natural killer (NK) cells have potent antitumor and anti

206 stein-Barr virus (EBV) DNA in T cells and/or natural killer (NK) cells in blood and skin lesions indu

207 red quantities, activity, and composition of natural killer (NK) cells in blood as well as expression

208 virus (ECTV; the agent of mousepox) and that natural killer (NK) cells in CL13-infected mice are redu

209 Meanwhile, the cytotoxic role played by natural killer (NK) cells in cutaneous leishmaniasis (CL

210 led to deficiencies of B cells, T cells, and natural killer (NK) cells in Rabl3 (xm/xm) mice.

211 which initiate the recruitment of protective natural killer (NK) cells in the infected trachea.

212 y T cells and effector T cells and decreased natural killer (NK) cells in these tumors.

213 on of interferon gamma (IFN-gamma)-producing natural killer (NK) cells induced a profound remodeling

214 Natural killer (NK) cells inhibit tumor development in m

215 T121 and PGT151, bind to B, activated T, and natural killer (NK) cells of HIV-infected and -uninfecte

216 Here we report that natural killer (NK) cells of the innate immune system re

217 Natural killer (NK) cells play a key role in immunity to

218 Natural killer (NK) cells play critical roles in protect

219 ocytes, which has primarily been observed in natural killer (NK) cells responding to cytomegalovirus

220 s well as DARA-induced depletion of CD38high natural killer (NK) cells resulting in crippled antibody

221 Natural killer (NK) cells that express the NKG2C recepto

222 Natural killer (NK) cells that have been modified to exp

223 xicity (ADCC) is a key effector mechanism of natural killer (NK) cells that is mediated by therapeuti

224 preventing lethal pathology, the ability of natural killer (NK) cells to act as rheostats tuning out

225 ll viability, it engaged macrophages but not natural killer (NK) cells to induce antibody-dependent c

226 py have exploited the efficient potential of natural killer (NK) cells to kill tumor cells through an

227 t a significantly higher number of activated natural killer (NK) cells was accumulated in the colonic

228 munity associated with T cells, B cells, and natural killer (NK) cells when matched sibling donors ar

229 cy have impairment in T-cell homeostasis and natural killer (NK) cells which leads to autoimmunity, r

230 ic method for functionalizing the surface of natural killer (NK) cells with a supramolecular aptamer-

231 rtant cytokines that regulate the biology of natural killer (NK) cells(1).

232 th potential to generate all ILC subsets and natural killer (NK) cells, and also permitted discrimina

233 reduction of CD8-positive (CD8(+)) T cells, natural killer (NK) cells, and NK-T cells and increased

234 s that limit antiviral responses mediated by natural killer (NK) cells, but molecular mechanisms for

235 otoxicity, as mediated by CD8(+) T cells and natural killer (NK) cells, by obstructing contact betwee

236 injection into mice activated ILC1s, but not natural killer (NK) cells, in the liver.

237 d activation of dendritic cells, T cells and natural killer (NK) cells, indicating an important role

238 ulates the development and function of human natural killer (NK) cells, innate lymphocytes important

239 tional changes at 48 and 96 hours, including natural killer (NK) cells, macrophage polarization, and

240 cells with unique immune properties, such as natural killer (NK) cells, NKT cells, gammadelta T cells

241 aluated the frequencies of T cells, B cells, natural killer (NK) cells, polymorphonuclear myeloid-der

242 ntiation, and function of CD8(+) T cells and natural killer (NK) cells, which are implicated in host

243 ctivation of several immune cells, including Natural Killer (NK) cells, which play an important role

244 lator of interleukin-15 (IL-15) signaling in natural killer (NK) cells.

245 FcgammaRs is FcgammaRIIIa/CD16a expressed by natural killer (NK) cells.

246 transitional B cells and increased cytolytic natural killer (NK) cells.

247 pecific receptor expression on monocytes and natural killer (NK) cells.

248 ing and/or function of cytotoxic T cells and natural killer (NK) cells.

249 d innate lymphoid cell populations including natural killer (NK) cells.

250 tokine for the survival and proliferation of natural killer (NK) cells.

251 lls (DCs), innate lymphoid cells (ILCs), and natural killer (NK) cells.

252 ngly, we also find that NM-Rs lack canonical natural killer (NK) cells.

253 r is present in subsets of T cells and human natural killer (NK) cells.

254 infection is characterized by activation of natural killer (NK) cells.

255 utrophils are dictated by the status of host natural killer (NK) cells.

256 d a protein complex containing the agonistic natural killer (NK) receptor NKG2D and the NK adaptor mo

257 Extranodal natural killer (NK) T-cell lymphoma (ENKTL) is a unique

258 counts of circulating Mycobacterium-reactive natural killer (NK), invariant NKT (iNKT), mucosal-assoc

259 and adhesive phenotypes contribute to human natural killer (NK)- and T-cell development as they unde

260 immune system require abrogating both T- and natural killer (NK)-cell responses, which eliminate fore

261 Once a natural killer or T cell has identified a target cell, t

262 ed receptors, such as Toll-like receptors or natural killer receptors, are commonly contrasted with d

263 of innate-like T cells, including invariant natural killer T (iNKT) and gammadelta-T cells, but thei

264 Innate T cells, including invariant natural killer T (iNKT) and mucosal-associated innate T

265 is produced by a unique subset of invariant natural killer T (iNKT) cells (NKT2) in the thymus in th

266 Invariant natural killer T (iNKT) cells acquire effector functions

267 entional CD8(+) T cells as well as invariant natural killer T (iNKT) cells and mucosal-associated inv

268 Human invariant natural killer T (iNKT) cells are a rare innate-like lym

269 Invariant natural killer T (iNKT) cells are a specialized subset o

270 Invariant natural killer T (iNKT) cells are innate-like lymphocyte

271 Invariant natural killer T (iNKT) cells are potent immune cells fo

272 Semi-invariant natural killer T (iNKT) cells are self-reactive lymphocy

273 e investigated the hypothesis that invariant natural killer T (iNKT) cells contribute to innate immun

274 Invariant natural killer T (iNKT) cells have been tested for their

275 CD1d-restricted invariant natural killer T (iNKT) cells represent a heterogeneous

276 Invariant natural killer T (iNKT) cells serve as early rapid respo

277 led >10,000 differentiating thymic invariant natural killer T (iNKT) cells using single-cell RNA sequ

278 phocytes, including lipid-reactive invariant natural killer T (iNKT) cells.

279 gh a negative regulatory function of BTLA in natural killer T (NKT) cell activation has been reported

280 expands a CD4(-)CD8(-) double-negative (DN) natural killer T (NKT) cell subpopulation that protects

281 Natural killer T (NKT) cells are a subset of T lymphocyt

282 Valpha24-invariant natural killer T (NKT) cells have shown potent anti-tumo

283 Natural killer T (NKT) cells rapidly respond to antigeni

284 cer and alpha-galactosylceramide to activate natural killer T (NKT) cells.

285 F, which is essential for the development of natural killer T cell (NKT cell) effector functions.

286 strate that the cross-talk between invariant natural killer T cells (iNKT) and CD8(+) T cells in the

287 and enhanced interferon-gamma production by natural killer T cells as well as number of viable CD4 T

288 endothelial cells, macrophages, T cells, and natural killer T cells in the CNV.

289 nterleukin 4- and interferon gamma-producing natural killer T cells in the liver and spleen and enhan

290 ceptors promote the progression of invariant natural killer T cells through the thymic maturation pro

291 these mediators in proliferating T cells and natural killer T cells, that also expressed the antimicr

292 he face of normal frequencies of circulating natural killer T cells.

293 ruitment of tumoricidal immune cells such as natural killer T cells.

294 Extranodal natural killer T-cell lymphoma (NKTCL; nasal type) is an

295 ytokines and SIRT1 were measured in blood T, natural killer T-like cell (NKT-like), and natural kille

296 Natural killer/T cell lymphoma (NKTCL) is a rare and agg

297 the genomic and transcriptional landscape of natural killer/T cell lymphoma (NKTCL), a rare form of n

298 gy of different cancers including extranodal natural killer/T-cell lymphoma (NKTL).

299 develop and test TF-targeting CAR-engineered natural killer (TF-CAR-NK) cells that co-express CD16, t

300 Uterine natural killer (uNK) cells, which are required for SA re