ライフサイエンスコーパス: natural killer cell

1 precursor) and lineage (B cell, T cell, and natural killer cell).

2 cells, helper T cells, effector B cells, and natural killer cells).

3 assical MHC-Ia molecules for presentation to natural killer cells.

4 sputum eosinophils, and enhanced circulating natural killer cells.

5 P) that is produced by cytotoxic T cells and natural killer cells.

6 not dividing, fibroblasts to cytotoxicity by natural killer cells.

7 ll-free virus, and develop susceptibility to natural killer cells.

8 jor forms of lymphomas arising from B, T, or natural killer cells.

9 inflammatory signaling initiated by CD160 in natural killer cells.

10 capes of lymphomas that arise from B, T, and natural killer cells.

11 D16(+) monocytes, innate lymphoid cells, and natural killer cells.

12 t alone was not able to stimulate T cells or natural killer cells.

13 he presence of T cells, dendritic cells, and natural killer cells.

14 umor-reactive cytotoxic T lymphocytes and/or natural killer cells.

15 sistance to the shear stress and attack from natural killer cells.

16 bers of monocytes, B cells, CD4 T cells, and natural killer cells.

17 ure T and B cells and jak3 in T and putative Natural Killer cells.

18 e variant and the homologous FcgammaRIIIa on natural killer cells.

19 e polymorphic stress molecules recognized by natural killer cells.

20 f total lymphocytes because of a decrease in natural killer cells.

21 presumably by regulatory T cells but also by natural killer cells.

22 ereby sensitizing melanoma cells to lysis by natural killer cells.

23 terations in CD4+ T cells, CD8+ T cells, and natural killer cells.

24 also found to be less sensitive to purified natural killer cells.

25 ot required for the development of classical natural killer cells.

26 of local dendritic cells, and activation of natural killer cells.

27 us antiviral immune responses of T cells and natural killer cells.

28 cial role in the development and activity of natural killer cells.

29 olic granules of cytotoxic T lymphocytes and natural killer cells.

30 o lysis by both antigen-specific T cells and natural killer cells.

31 dvantage in controlling DENV infection using natural killer cells.

32 cells, as well as decreased ICOS+ and 4-1BB+ natural killer cells.

33 el and exhibits SOX9-dependent resistance to natural killer cells.

34 ocytosis"), especially cytotoxic T cells and natural killer cells.

35 intratumoral macrophages, and activation of natural killer cells.

36 -alpha, which drives IFN-gamma production by natural killer cells.

37 OXP3(+)T cells, CD11c(+) dendritic cells and natural killer cells.

38 henotype of each T-cell subset, B cells, and natural killer cells.

39 and T lymphocyte attenuator (BTLA); and the natural killer cell-activating receptor CD160.

40 LSCs) in acute myeloid leukemia downregulate natural killer cell-activating receptor ligands to evade

41 ent phagocytosis, cellular cytotoxicity, and natural killer cell activation were assessed.

42 activation, IFNgamma response, CD16-mediated natural killer cell activation, and monocyte/macrophage

43 cyte phagocytosis, complement activation and natural killer cell activation, are substantially enhanc

44 Natural killer cell activities were clustered in clinica

45 ole of IL-12 and IL-15 in the enhancement of natural killer cell activity was reported.

46 munological defense, including inhibition of natural killer cell activity with ongoing lowering of Ig

47 nosuppression was an opportunity to discover natural killer cell alloreactions that eradicated acute

48 expression profiles of a receptor protein in natural killer cells among donors infected with human cy

49 Using multiple human cell types, including natural killer cells, an IL-12 indicator cell line, and

50 set involving phagosome formation as well as natural killer cell and IL-3 signaling.

51 rapy had an increased abundance of activated natural killer cells and a newly identified CD3(-)CD68(+

52 ed in significantly decreased frequencies of natural killer cells and a trend toward reduced ILC popu

53 regulatory cells, immunosuppressive CD56(hi) natural killer cells and ablation of proinflammatory muc

54 tiple populations of myeloid cells, T cells, natural killer cells and B cells that demonstrated both

55 Type I and II IFNs, as well as natural killer cells and CD8(+) T cells, were indispensi

56 Natural killer cells and cytotoxic T-lymphocytes deploy

57 45 does reduce the viability of normal T and natural killer cells and decrease activated T-cell produ

58 d with increased accumulation of T cells and natural killer cells and decreased myeloid-derived suppr

59 T correlated with an increased proportion of natural killer cells and effector memory CD4(+) and CD8(

60 IFNgamma-induced pathways and activation of natural killer cells and endothelial cells.

61 ILCs consist of conventional natural killer cells and helper-like cells (ILC1, ILC2 a

62 eatment reduced this suppressive function of natural killer cells and increased survival of mice with

63 so in mice, mimics the cytotoxic activity of natural killer cells and increases the surface area avai

64 unodeficiency characterized by lack of T and natural killer cells and infant death from infection.

65 producers of IL-22 post-PH are conventional natural killer cells and innate lymphoid cells type 1.

66 crophages and dendritic cells) and lymphoid (natural killer cells and innate lymphoid cells) cell pop

67 umor-derived PGE2 blocks early activation of natural killer cells and interferes with subsequent adap

68 After INT-747 treatment, natural killer cells and interferon-gamma expression mar

69 in MDA5(-/-) mice, but perforin induction by natural killer cells and levels of interferon gamma, int

70 s of innate lymphoid cells with conventional natural killer cells and lymphoid tissue inducer cells.

71 an endogenous RNA editing enzyme in primary natural killer cells and lymphoma cell lines.

72 otentially by suppressing IFNgamma-producing natural killer cells and M1-polarized macrophages.

73 ther showed exosomes were mainly taken up by natural killer cells and macrophages in the lung.

74 n CD30 on HL cells and the CD16A receptor on natural killer cells and macrophages, to induce tumor ce

75 r cells, and binding to Fcgamma receptors on natural killer cells and macrophages.

76 by complement deposition and accumulation of natural killer cells and monocytes/macrophages in capill

77 ng hepatic macrophages, T and B lymphocytes, natural killer cells and platelets, as well as key effec

78 eased IL-17 production by CD4(+) T cells and natural killer cells and recruited regulatory cells and

79 lin-like receptors (KIRs) which are found on natural killer cells and some T cells; for the CD94:NKG2

80 -H3 (CD276) is both an inhibitory ligand for natural killer cells and T cells and a tumor antigen tha

81 Subsets of natural killer cells and T cells selectively induced nuc

82 cytes, impairing activation and functions of natural killer cells and T cells.

83 0-dependent elimination of dendritic cell by natural killer cells and that hydrocortisone improves ou

84 of several immune cell populations, such as natural killer cells and virus-specific T cells.

85 g FcgammaRIIIa (expressed on macrophages and natural killer cells) and FcgammaRIIIb (expressed on neu

86 on by cytotoxic cells (cytotoxic T cells and natural killer cells), and interleukin 12 production by

87 increased expression of IFNgamma-inducible, natural killer cell, and T cell transcripts, but less ex

88 , treatment-induced expansion of T cells and natural killer cells, and activation of interferon-gamma

89 ty controlled by T cells (T(H)1 and CD8(+)), natural killer cells, and antigen-presenting cells; T2 C

90 e activation of dendritic cells, mast cells, natural killer cells, and CD8 T cells to mount an antitu

91 Teffs, natural killer cells, and eosinophils also responded, wi

92 challenge, including monocytes, neutrophils, natural killer cells, and eosinophils.

93 genes with correlated expression in T cells, natural killer cells, and erythroblasts.

94 ype CD4 T cells, reduced T-bet expression by natural killer cells, and expansion of blood monocytes w

95 ures tracking CD8 and CD4 T-cell activation, natural killer cells, and IFN activation associated sign

96 ted, altered in tropism, more susceptible to natural killer cells, and less pathogenic.

97 ting IFNgamma and Granzyme B CD4 T cells and natural killer cells, and lower number of FoxP3 regulato

98 sly thought, including neutrophils, B cells, Natural Killer cells, and mast cells.

99 e examine the roles of alveolar macrophages, natural killer cells, and neutrophils in antibody-mediat

100 cells (Tregs), gammadelta T cells, B cells, natural killer cells, and primary and induced pluripoten

101 in the tumor, activating dendritic cells and natural killer cells, and recruiting the adaptive immune

102 choalveolar lavage samples and repression of natural killer cell- and T cell-associated transcripts i

103 ding CD8+ cytotoxic T lymphocytes (CTLs) and natural killer cells-and regulated by Runt-related (Runx

104 Natural killer cells are a type of cytotoxic lymphocyte

105 Natural killer cells are able to directly lyse tumor cel

106 T, B, and natural killer cells are required for normal immune resp

107 Neutrophils and natural killer cells are the likely targets of these pat

108 Cytotoxic T lymphocytes (T) and natural killer cells are the main cytotoxic killer cells

109 CD8(+) cytotoxic T lymphocytes (CTL) and natural killer cells are the main cytotoxic killer cells

110 were ineffective in ADCC assays with murine natural killer cells as effectors, whereas ADCP was equi

111 cells, myeloid-derived suppressor cells, and natural killer cells) as well as adaptive immune cells (

112 Natural killer cells assess target cell health via inter

113 innate immune cells (antitumor macrophages, natural killer cells) associated with clearance of senes

114 ile, it still revealed diversity in a set of natural killer cell-associated receptors.

115 itutive and alternate macrophage, B-cell and natural killer cell-associated transcripts (NKAT), indic

116 nd, followed by MAIT, iNKT, gammadelta T and natural killer cells at the other end.

117 acts as a costimulatory receptor on T cells, natural killer cells, B cell subsets, and some dendritic

118 as immune checkpoint receptors in T cell and natural killer cell biology are just beginning to be unc

119 marily within antigen-experienced T cells or natural killer cells but less so in naive T or B cells.

120 s a distinct lineage from Th and circulating natural killer cells but shares circuitry devoted to fun

121 terestingly, tumor rejection did not involve natural killer cells but was associated instead with a m

122 er cells suppress the cytotoxic functions of natural killer cells by a variety of mechanisms.

123 The reduction of the interleukin-10 level in natural killer cells by hydrocortisone was partially dep

124 ning 64 targets deployed in T cells (CAR-T), natural killer cells (CAR-NK) or mixtures (CAR-NK/T) fro

125 infiltrating Kupffer cells, mature activated natural killer cells (CD69+), and PD-1+ effector memory

126 ) IFN-gamma-producing group 1 ILCs (ILC1 and natural killer cells), CD8(+) cytotoxic T cells (TC1), a

127 involved in the regulation of dendritic cell/natural killer cell cluster.

128 s are comprised of two subsets, conventional natural killer cells (cNK) and tissue-resident cells oft

129 pinal fluid (including B cells, T cells, and natural killer cells) (cohort 1).

130 or the observed therapeutic effects and that natural killer cells constitute a critical human effecto

131 Natural killer cells constitute potent innate lymphoid c

132 Natural killer cells controlled early infection but were

133 luding monocytes, CD4(+) and CD8(+) T cells, natural killer cells, conventional and plasmacytoid dend

134 Compared with pretreatment levels, Treg and natural killer cell counts rose >fivefold (P < .001) and

135 hydrocortisone modulates the dendritic cell/natural killer cell cross talk in the context of posttra

136 effects of hydrocortisone on dendritic cell/natural killer cell cross talk were studied in a mouse m

137 cancer cell growth, including stimulation of natural killer cell cytotoxic activity and repression of

138 Natural killer cell deficiencies (NKDs) are an emerging

139 Human natural killer cell deficiency (NKD) arises from inborn

140 elongation (RTEL1) mutation causing isolated natural killer cell deficiency and mutations in ras-asso

141 Specific natural killer cell depletion 24 hours pre-acute myocard

142 c constraint in macrometastases triggered by natural killer cell depletion suggests a dynamic interpl

143 f DST bm1 cells than F1 cells was reduced by natural killer-cell depletion.

144 In contrast, decidual natural killer cell-derived IFN-gamma reverses such TNF-

145 Natural killer cell development and maturation were arre

146 nt SCID phenotype with a T-cell, B-cell, and natural killer cell developmental defect and hypogammagl

147 Ultimately, mice depleted of natural killer cells displayed an increased neutrophil n

148 The interaction of noncytotoxic decidual natural killer cells (dNK) and extravillous trophoblasts

149 DS1 (KIR2DS1) expressed by maternal decidual natural killer cells (dNK) and the presence of its ligan

150 s of HLA-G+ EVT with sample matched decidual natural killer cells (dNK), macrophages, and CD4+ and CD

151 d KLRG1 molecules) were increased in splenic natural killer cells during Pseudomonas aeruginosa infec

152 uced T cell effector functions, and variable natural killer cell dysfunctions.

153 16) report new mechanisms of human and mouse natural killer cell education by inhibitory and activati

154 d to respond to vaccines, there were reduced natural killer cells, elevated regulatory T cells, M2-ty

155 Natural killer cells employ a diverse arsenal of effecto

156 Natural killer cells express multiple receptors for majo

157 Natural killer cells from acute myeloid leukaemia patien

158 Natural killer cell function is regulated by inhibitory

159 of molecules that seek to subvert T cell and natural killer cell function via a remarkable array of m

160 veals that RAG endonuclease activity affects natural killer cell function, demonstrating that such do

161 r type 2 inflammation, T(H)17 signaling, and natural killer cell function.

162 glycoproteins, many of which interfere with natural killer cell function.

163 tional T lymphocytes and T-regulatory cells, natural killer cells, gamma delta T cells, and other acc

164 Here, we show that natural killer cell granule protein 7 (NKG7) is a regula

165 FU treatment induced TSLP, HLA class II, and natural killer cell group 2D (NKG2D) ligand expression i

166 Immunologically, natural killer cells had an immature phenotype and impai

167 sfer of tumor-specific cytotoxic T cells and natural killer cells, have been clinically translated fo

168 s are applied to other immune cells, such as natural killer cells, hematopoietic cells or induced plu

169 of interferon-gamma (IFN-gamma) by activated natural killer cells, IL-34-Mphi and M-CSF-Mphi prevent

170 The engagement of natural killer cell immunoglobulin-like receptors (KIRs)

171 We assessed the role of systemic natural killer cells in a Pseudomonas aeruginosa mouse p

172 CAR-engineered HSCs may produce myeloid and natural killer cells in addition to T cells expressing t

173 increased activity of cytotoxic T cells and natural killer cells in BKVN and viremia samples resembl

174 ota and an increased ratio of neutrophils to natural killer cells in esophageal tissues compared with

175 ferential effects on B and T lymphocytes and natural killer cells in humans.

176 AT4 mediates IFNgamma release in T cells and natural killer cells in response to interleukin 12 (IL12

177 dritic cells, and CD8 T lymphocytes and CD57 natural killer cells in the ALNs(-) were factors associa

178 es due to extensive upregulation of APCs and natural killer cells in the blood and tumor compared wit

179 MSCs significantly decreased natural killer cells in the heart and spleen and neutrop

180 umor cells via recruitment and activation of natural killer cells in the tumor.

181 Although the role of T cells and natural killer cells in tumor immunology has been establ

182 tablishing normal numbers of CD8 T cells and natural killer cells in tumors.

183 anulocytes, monocytes, T cells, B cells, and natural killer cells) in patients having undergone HSPC

184 NKG2A subset of CD56 natural killer cells increased substantially and persist

185 ased programmed death ligand 1 expression on natural killer cells (increased from 11.9% to 61.6%, P =

186 emorrhage-induced immunosuppression, splenic natural killer cells induced an interleukin-10-dependent

187 oles for macrophages, innate lymphoid cells, natural killer cells, innate gammadelta T cells, and oth

188 Innate lymphocytes (gammadelta T cells, natural killer cells, innate lymphoid cells) are the maj

189 hat monocytes, neutrophils, dendritic cells, natural killer cells, innate lymphoid cells-2, and CD (c

190 the utility of this microfluidic assay with natural killer cells interacting with tumor cells, and o

191 ial cells, dendritic cells, macrophages, and natural killer cells is crucial for its protection.

192 cted a study to ascertain the association of natural killer cell killer immunoglobulin-like receptors

193 omics, along with CRISPR/Cas9-KO cell lines, natural killer cell-killing assays, and RNA-Seq experime

194 The function of natural killer cells, lymphocyte phenotype, and serum im

195 s of immune cells including the conventional natural killer cells, lymphoid tissue inducers, type 1,

196 induces site-specific C-to-U RNA editing in natural killer cells, lymphoma cell lines, and, to a les

197 receptor 9 (TLR9), induces the activation of natural killer cells, macrophages, and antigen presentin

198 immune cells such as primary T and B cells, natural killer cells, macrophages, and primary microglia

199 ting adenosine signaling is found to promote natural killer cell maturation and antitumor immunity an

200 llular context, supporting the hypothesis of natural killer cell mechanosensitivity.

201 cated that CD8 T cells, but not CD4 cells or natural killer cells, mediated elimination of KPC-Par-1(

202 NSG mice, pointing to the potential role of natural killer cell-mediated antibody-dependent cell cyt

203 in G1 anti-CD47 mAb induced phagocytosis and natural killer cell-mediated cytotoxicity of TCL cells t

204 the D1 domain attenuated Necl-5 binding and natural killer cell-mediated cytotoxicity.

205 phils without IL-5 was only seen in EOs, and natural killer cell-mediated eosinophil killing was seen

206 -EMR1 IgG1 was evaluated in vitro by using a natural killer cell-mediated killing assay and in vivo i

207 osylated anti-EMR1 mAb dramatically enhanced natural killer cell-mediated killing of eosinophils from

208 rate that mitophagy plays a critical role in natural killer cell memory formation following viral inf

209 ular mechanisms important to generate innate natural killer cell "memory" are poorly understood.

210 Our results suggest that natural killer cells modified with glycan ligands to CD2

211 ent CD8 + T cells, CD56 + T cells, CD56(dim) natural killer cells, monocytes and dendritic cells were

212 Other leukocyte populations, including natural killer cells, monocytes, and dendritic cells, sh

213 recruiting cytotoxic effector cells, such as natural killer cells, monocytes, and polymorphonuclear c

214 ter molecule Stimulator of Interferon Genes, natural killer cells, myeloid cells, and B cells.

215 ations of T cells, B cells, dendritic cells, natural killer cells, myeloid-derived suppressor cells,

216 her organs, including innate lymphoid cells, natural killer cells, natural killer T cells, mucosal-as

217 atory hepatic macrophage infiltration, while natural killer cells, natural killer T cells, neutrophil

218 regulation of innate immunity in infants and natural killer cell networks in children, and additional

219 lls and regulatory T cells, dendritic cells, Natural Killer cells, neutrophils, and mast cells are pr

220 ther activating or inhibitory effects during natural killer cell (NK cell) activation.

221 n in situ modify the tumor cell surface with natural killer cell (NK cell)-activating signals to achi

222 A tissue-resident population of natural killer cells (NK cells) in the liver has recentl

223 Natural killer cells (NK) are highly enriched in the hum

224 Fluorescently labeled human T cells, natural killer cells (NK), and various target cells (NAL

225 ) T-cell ratio and increased CD16(+)CD56(hi) natural killer cells (NK), CD4(+) effector memory T cell

226 D1c + myeloid DCs; neutrophils; macrophages; natural killer cells (NK); Marginal Zone-like B cells (M

227 ident memory CD8(+) T cells (TRMs), resident natural killer cells (NKRs), and tumor-associated macrop

228 Natural killer cells (NKs) are important effectors of th

229 Cytotoxic T-lymphocytes (CTLs) and natural killer cells (NKs) kill compromised cells to def

230 Natural killer cell number and status were assessed in s

231 pe with absent T cells and normal B-cell and natural killer cell numbers.

232 PMVDS stimulated both cytotoxic T cells and natural killer cells of cell-mediated immunity to provid

233 In contrast to viral evasion of natural killer cells or T cell recognition, the evasion

234 of Env-specific ADCC antibodies to activate natural killer cells (P<.001).

235 Furthermore, a shift in the natural killer cell phenotype was observed with features

236 we found that innate immune factors, such as natural killer cells, plasmacytoid dendritic cells, and

237 Because alterations of dendritic cells and natural killer cells play a central role in trauma-induc

238 We report for the first time that natural killer cells play a major role in the mice susce

239 Natural killer cells play an important role in immunity

240 Moreover, CD8+ T-cell, CD4+ T-cell and NK (natural killer) cell populations in splenocytes were ele

241 A combination of serum and natural killer cells provided the most effective protect

242 th the frequencies of intrathecal CD56bright natural killer cells (r = 0.28; P = .007).

243 Natural killer cells readily kill target cells, and educ

244 e group 3-like ILCs was not dependent on the natural killer cell receptor (NCR1), since NCR1-deficien

245 Natural killer cell receptors and other genes related to

246 ntigens, forming ligands for cytotoxic T and natural killer cell receptors.

247 mmunity are Patr-B variants that engage with natural killer cell receptors.

248 peptide complexes probably affect T-cell and natural killer cell recognition, providing a sound basis

249 nsplantation, innate immune cells, including natural killer cells, recovered with virus rebound.

250 rial from the lung vasculature, and promoted natural killer cell recruitment and activation.

251 In this setting, the role of natural killer cells remains controversial.

252 profile are due to a different proportion of natural killer cells responding in LUJV infection than t

253 by which CMVs evade or reprogram T cell and natural killer cell responses in vivo However, the role

254 anti-CD137 MAb that can activate T cell and natural killer cell responses to clear tumors.

255 might result from impaired CD8(+) T-cell and natural killer cell responses to EBV infection in these

256 Simultaneously, natural killer cell responses to inflammatory cytokines

257 pulmonary pathology, stronger and persistent natural killer cell responses, and the extended inductio

258 related to local and systemic recruitment of natural killer cells resulting in increased interferon-g

259 By contrast, natural killer cells retained partial IL-2Rbeta expressi

260 flammatory and regulatory cytokine profiles, natural killer cells showed a predominantly proinflammat

261 Natural killer cells showed reduced T-bet expression at

262 ctor]) coupled with T-helper cell type 2 and natural killer cell signaling in children.

263 okines with an additional role of T(H)17 and natural killer cell signaling.

264 he number of tumor-infiltrating CD8(+) T and natural killer cells, slowed tumor growth, and improved

265 Quantification of natural killer cells spreading on surfaces coated with t

266 RNA-seq analysis of natural killer cells subjected to cellular crowding and

267 Natural killer cell subset association with CMV endpoint

268 nduces the death of CD56(bright) NK cells, a natural killer cell subset whose expansion is correlated

269 altered composition of gammadelta T-cell and natural killer cell subsets.

270 the cytokine milieu, macrophages/monocytes, natural killer cells, T cells, and neutrophils in severe

271 roinflammatory cytokine responses by DCs and natural killer cells, Th1 development, phagocytic recept

272 cytokine-producing dendritic cells (DCs) and natural killer cells than Cd36(-/-) mice.

273 st of TLR3 and RLR to activate dendritic and natural killer cells that can kill tumor cells.

274 ccumulating evidence for the contribution of natural killer cells, the key mediators of antibody-depe

275 t that TRAIL is part of the armamentarium of natural killer cells, these observations identify a new

276 22 ligands onto NK-92MI and cytokine-induced natural killer cells to achieve tumor-specific CD22 targ

277 also enhanced the activity and maturation of natural killer cells to effectively treat anti-PD-1 resi

278 2B8T2M activates natural killer cells to enhance antibody-dependent cellu

279 d is secreted by cytotoxic T lymphocytes and natural killer cells to help eliminate virus-infected an

280 ng HGF-independent MET activity, and engaged natural killer cells to kill MET-expressing cancer cells

281 rated that anti-CD27 stimulated CD8(+) T and natural killer cells to release myeloid chemo-attractant

282 of tumoricidal effector cells, in particular natural killer cells, to the tumor stroma for antitumor

283 al immune cells, such as T helper type 1 and natural killer cells, to unleash neurotoxicity and infla

284 Depletion of natural killer cells was achieved through an IV anti-asi

285 Additionally, NKG7 expressed by natural killer cells was critical for controlling cancer

286 IFN-gamma production, mainly by natural killer cells, was associated with protection, an

287 21R expression on CD8(+) T cells, but not on natural killer cells, was required for optimal anti-ErbB

288 major histocompatibility complex class I and natural killer cells were commonly downregulated in psor

289 Moreover, natural killer cells were involved in AS by increasing t

290 the lungs, indicating that mice depleted of natural killer cells were much more susceptible to infec

291 Natural killer cells were normal in number but not "lice

292 Specifically, T, B, and natural killer cells were severely depleted in the blood

293 Blood CD16 and CD56 natural killer cells were significantly more likely to b

294 roduction by IL-12p70-mediated activation of natural killer cells, whereas miR-146a and miR-146b over

295 lteration in NKG2A and NKG2C subsets of CD56 natural killer cells which might have a pathogenic role

296 (IFN-gamma) or TNF-alpha or cocultured with natural killer cells (which have been shown to induce an

297 smatched grafts is driven by the recipient's natural killer cells, which overwhelmingly use perforin

298 eeks of infection, for example proliferating natural killer cells, which potentially may associate wi

299 es effectively recruited and activated human natural killer cells, while vaccine-elicited RM antibodi

300 eficiency disease (SCID) affecting B, T, and natural killer cells, with an almost complete lack of an