ライフサイエンスコーパス: necrotic

1 ing Toll, Spz factors, and the Spz inhibitor Necrotic.

2 ular supply and >80% of the tumor tissue was necrotic.

3 es, but few of these myocytes appeared to be necrotic.

4 l, and 12 of these lesions were incompletely necrotic.

5 e DeltafakA mutant maintains larger and more necrotic abscesses, bacterial numbers are similar to tho

6 Deletion of CTSB affected apoptotic but not necrotic acinar cell death.

7 racellular proteins, such as those shed from necrotic and apoptosing cells.

8 bel-free methodology to characterize viable, necrotic and apoptotic human lymphoma U937 cells.

9 sociating meningeal arterial calcifications, necrotic and calcified areas in basal ganglia, dentato-o

10 ive' response in which meristem cells become necrotic and kill E. solidaginis hatchlings before gall

11 essing AML cells leads to cell death through necrotic and late apoptosis pathways.

12 Three subregions of glioma-the necrotic and nonenhancing tumor core, the peritumoral ed

13 Macrophages in necrotic and symptomatic atherosclerotic plaques in huma

14 average values in the whole tumor or average necrotic and viable tissues.

15 or star-shaped chlorotic lesions that become necrotic, and infested plants may be distorted.

16 f rat brain, and in human ovarian cancerous, necrotic, and normal tissues was achieved.

17 er completely (100%) or incompletely (<100%) necrotic, and performance characteristics and predictive

18 Significant lower viabilities and more necrotic/apoptotic cells were found when these cancer ce

19 ted significantly (p < 0.05) decreased acute necrotic/apoptotic injury and significantly (p < 0.05) i

20 tated discrimination between blood lakes and necrotic areas.

21 dose and form dense cellular clusters around necrotic areas.

22 marginal), 2 (partial), 3 (segmental), or 4 (necrotic) as previously defined.

23 sensus, with most (five of six) incompletely necrotic at histopathology.

24 eatment response equivocal were incompletely necrotic at histopathology.

25 athway as a key factor in the development of necrotic atherosclerotic plaques.

26 as completely (100%) or incompletely (<=99%) necrotic based on transplant histology.

27 As attenuated cytokine-induced apoptotic and necrotic beta cell death and increased beta cell viabili

28 The area of necrotic bone and the necrotic bone ratio were significa

29 ZOL sheep (2/6) showed inflammation and necrotic bone at mandibular region.

30 Necrotic bone exposure was significantly lower in the FS

31 no significant difference in empty lacunae, necrotic bone formation, osteoclast number, and surface

32 The area of necrotic bone and the necrotic bone ratio were significantly smaller in the FS

33 bone marrow-derived macrophages treated with necrotic bone showed increased extracellular signal-regu

34 In conclusion, necrotic bone stimulates macrophage-inflammatory respons

35 and lack of collagen fiber insertion in the necrotic bone were associated with impaired socket heali

36 of LIP teeth led to increased empty lacunae, necrotic bone, and osteoclast surface area in antibiotic

37 receptor mechanisms involved in sensing the necrotic bone, using a piglet model of Legg-Calve-Perthe

38 t activity was responsible for resorbing the necrotic bone, which in turn stimulated the deposition o

39 h large paravascular haemorrhages and yellow necrotic borders, involving the posterior pole but not t

40 etry (MS), we report the lipid MS profile of necrotic breast cancer with Desorption Electrospray Ioni

41 gy through presentation of self-antigen from necrotic cardiac cells to cytotoxic CD8(+) T cells.

42 Recruited monocytes clear necrotic cardiomyocytes and differentiate into cardiac m

43 Recently it was shown that PZA penetrates necrotic caseous TB lung lesions and kills nongrowing, d

44 low toxicity and the absence of apoptotic or necrotic cell death after 24 or 48 h of incubation.

45 AKI is histologically characterized by necrotic cell death and inflammation.

46 The kinase RIPK3 is required to induce necrotic cell death and is strongly induced in cells iso

47 tion of phagocytosed Shigella led to massive necrotic cell death and release of the bacteria.

48 hat RIPK1 is a key mediator of apoptotic and necrotic cell death as well as inflammatory pathways.

49 In contrast, programmed necrotic cell death causes release of immunostimulatory

50 Necrotic cell death during Mycobacterium tuberculosis (M

51 urprised to find no evidence of apoptotic or necrotic cell death during stages of peak myofiber loss,

52 Moreover, necrotic cell death in Mtb-infected macrophage cultures

53 a CD44/ITGA4 containing complex and triggers necrotic cell death in multiple myeloma cell lines.

54 sine triphosphate depletion, and the ensuing necrotic cell death in skin fibroblasts, and this effect

55 hat activate PPARalpha, leading to increased necrotic cell death in the lung which correlates with th

56 e, network analysis indicated an increase in necrotic cell death in the lungs of superinfected mice c

57 s oxidative burst, mitochondrial damage, and necrotic cell death in TSC-deficient cells in a highly s

58 gical and genetic analyses revealed that the necrotic cell death is distinct from the RIP1/3 pathway-

59 the CNS to apoptosis and a form of regulated necrotic cell death known as necroptosis that is mediate

60 Ferroptosis is an iron-dependent form of necrotic cell death marked by oxidative damage to phosph

61 ion and p38 MAPK inhibition to abrogation of necrotic cell death mediated by mitochondrial membrane p

62 orticosteroids such as dexamethasone inhibit necrotic cell death of cells infected with Mycobacterium

63 mpound 1 concentration-dependently inhibited necrotic cell death pathway activation and 2.5 mM compou

64 Ferroptosis is a regulated form of necrotic cell death that is caused by the accumulation o

65 ar NAD(+) depletion, subsequently preventing necrotic cell death that would otherwise occur due to PA

66 amage caused by UVA to mitochondria leads to necrotic cell death via adenosine triphosphate depletion

67 to I/R increased reactive oxygen species and necrotic cell death, both of which were mitigated by ATF

68 to I/R increased reactive oxygen species and necrotic cell death, both of which were mitigated by ATF

69 eases (HD) selectively induces a new form of necrotic cell death, in which endoplasmic reticulum (ER)

70 cellular molecules associated with regulated necrotic cell death, replicating the characteristics of

71 Necroptosis, a regulated form of necrotic cell death, requires the activation of the RIP3

72 nvolves a newly described form of programmed necrotic cell death, termed necroptosis.

73 ating and suppressing two regulated forms of necrotic cell death, termed pyroptosis and necroptosis,

74 iving increased generation of superoxide and necrotic cell death, which was rescued by genetic inhibi

75 Thus, Clec2d binds to histones released upon necrotic cell death, with functional consequences to inf

76 re leads to necroptosis, a regulated form of necrotic cell death.

77 ractions between autophagy and apoptotic and necrotic cell death.

78 ich then induces plasma membrane rupture and necrotic cell death.

79 ding to disruption of the inner membrane and necrotic cell death.

80 and MeHg were found to induce apoptotic and necrotic cell death.

81 todynamic injury, resulting in predominantly necrotic cell death.

82 ve oxygen species production, culminating in necrotic cell death.

83 ction of reactive oxygen species and massive necrotic cell death.

84 promoters of necroptosis, a pro-inflammatory necrotic cell death.

85 Indeed, necrotic cell debris allow macropinocytic breast and pro

86 Here, we show that consuming necrotic cell debris via macropinocytosis (necrocytosis)

87 namic relationship between proliferating and necrotic cell populations during vascularized tumor grow

88 re composite mass consisting of tumor cells, necrotic cell, or inflammatory tissues.

89 sidual disease decreased apoptotic cell- and necrotic cell-induced immunosuppressive phenotypes, bloc

90 de enables the discrimination between viable/necrotic cells and cell fragments, whereas phase informa

91 ation when released from activated immune or necrotic cells and drives the pathogenesis of various in

92 and CD8(+) cytotoxic T cells in response to necrotic cells and may thus be crucial players in exacer

93 Moreover, FHR1 bound to MDA-rich areas on necrotic cells and prevented CFH from mediating its cofa

94 We hypothesized that sensing of necrotic cells by DNGR-1 plays a determinant role in the

95 cious cycle including the uptake of infected necrotic cells by other phagocytes, Mtb growth therein,

96 pheres with an outer rim of viable cells but necrotic cells centrally.

97 We show that the content of necrotic cells enhances angiogenesis and proliferation o

98 s play a critical role in removing apoptotic/necrotic cells in inflammation and injury, a process ter

99 day 1 and were full of intact neutrophils or necrotic cells on day 2.

100 into account the balance between living and necrotic cells proved to be able to reproduce the experi

101 hus, in addition to bacterial dissemination, necrotic cells provide first a niche for bacterial repli

102 e of micro-organisms (sterile inflammation), necrotic cells release damage-associated molecular patte

103 Necrotic cells release danger signals, activating innate

104 During sterile inflammation, necrotic cells release pro-inflammatory molecules includ

105 In contrast, many neutrophils and necrotic cells were present at the edge of Deltasse muta

106 ids (layers of proliferating, quiescent, and necrotic cells).

107 pair (CCP1/2), tethering active IL-33 within necrotic cells, preventing its release, and forestalling

108 o distinguish between living, apoptotic, and necrotic cells.

109 (CD8alpha(+) DCs) and is involved in sensing necrotic cells.

110 urfaces such as the extracellular matrix and necrotic cells.

111 e LL37 and double stranded-RNA released from necrotic cells.

112 ous nucleic acids released from apoptotic or necrotic cells.

113 lows discriminating between viable cells and necrotic cells.

114 a Clec2d reporter responded to lysates from necrotic cells.

115 Encapsulated C3HeB/FeJ granulomas show necrotic centers with transcripts associated with immuno

116 F ablation was noted in 3% (95% CI: 0, 9) of necrotic CLMs with margins of at least 5 mm.

117 reinterventions for residual peripancreatic necrotic collections and other complications during the

118 red in patients with infected or symptomatic necrotic collections in the early, acute period (<2 week

119 eposition (+38.47%, p < 0.05) and diminished necrotic core (-31.39%, p < 0.05).

120 Lipid-rich necrotic core (LRNC), a high-risk coronary plaque featur

121 vo quantitation of calcification, lipid-rich necrotic core (LRNC), and matrix was assessed with stati

122 as characterized by halted expansion of the necrotic core and accumulation of macrophages along with

123 However, plaque size, necrotic core and macrophage content were similar in Mcl

124 impaired inflammation resolution, notably a necrotic core and thinning of a protective fibrous cap t

125 , including a thinner fibrous cap, increased necrotic core area, and increased intraplaque hemorrhage

126 rosclerotic plaques, characterized by bigger necrotic core areas and increased macrophage apoptosis.

127 lesions and controlling the expansion of the necrotic core by impairing efferocytosis.

128 ed with group and phase velocity (lipid-rich necrotic core content, fibrous cap structure, intraplaqu

129 The necrotic core expansion was dependent on monocyte recrui

130 celerated lesion progression, exemplified by necrotic core expansion.

131 molecule integrin alpha4 were protected from necrotic core expansion.

132 The poor definition of necrotic core facing border of FC and the neointimal pre

133 RATIONALE: Necrotic core formation during the development of athero

134 The necrotic core has long been a hallmark of the vulnerable

135 LO1-750 specifically identified necrotic core in ex vivo human coronary lesions.

136 ques, and that it does so without increasing necrotic core of plaques or causing detectable side effe

137 Furthermore, in the BVS arm, necrotic core pre-procedure was an independent determina

138 ll thickness (0.99+/-0.11 mm) and lipid-rich necrotic core prevalence (30%), as compared with low-ris

139 CI, 0.004-0.026) and with higher lipid-rich necrotic core prevalence at older age (odds ratio, 1.26;

140 Necrotic core size was also reduced in the model of shea

141 d monocyte responses were not modulated, but necrotic core size was greater, even when adjusting for

142 Both lesion and necrotic core size were significantly reduced in ApoE(-/

143 l mean carotid wall thickness and lipid-rich necrotic core) at follow-up was assessed.

144 creased monocyte-macrophage content, reduced necrotic core, attenuated inflammatory gene expression (

145 ing expert annotations of the tumor habitat (necrotic core, enhancing tumor, and FLAIR-hyperintense s

146 y lesions that are unlikely to possess large necrotic core, rendering them safe for treatment with me

147 agocytic immune cells become confined to the necrotic core, which is surrounded by an astrocytic bord

148 dvanced lesions by formation of a lipid-rich necrotic core, which may rupture and cause myocardial in

149 romotes lesion growth and establishment of a necrotic core.

150 fibrous cap thickness, along with a smaller necrotic core.

151 ding bacteria, infected cells, and a caseous necrotic core.

152 centre may become hypoxic and die, forming a necrotic core.

153 s the accumulation of apoptotic cells in the necrotic core.

154 traffics within macrophages to the tumour's necrotic core.

155 mission intensity still drops off toward the necrotic core.

156 hough the atherosclerotic plaques with large necrotic cores (independent of the degree of luminal ste

157 ed necrosis, contributes to the formation of necrotic cores in atherosclerotic plaque in animal model

158 Lipid-rich and/or necrotic cores in ipsilateral CAP were significantly lar

159 loid-specific deletion of CaMKII had smaller necrotic cores with concomitantly thicker collagen caps.

160 A distinct type of plaque containing large necrotic cores with thin fibrous caps often precipitates

161 It is proposed that the presence of large necrotic cores within the neointima may be associated wi

162 ssociated with increased atherosclerosis and necrotic cores, and a decrease in plaque collagen.

163 soluble Mer, improved efferocytosis, smaller necrotic cores, thicker fibrous caps, and increased rati

164 rmation of advanced lesions characterized by necrotic cores, to lesion regression following aggressiv

165 membranes, the internal elastica lamina, and necrotic cores.

166 ssues correlates directly with the extent of necrotic damage during F. tularensis infection.

167 ndrial depolarization, oxidative stress, and necrotic death also in B16 mouse melanoma cells.

168 trations and glucosyltransferase-independent necrotic death at higher concentrations.

169 ter of survival) induced rapid apoptotic and necrotic death in infected cells.

170 Defining the in vivo relevance of necrotic death is hampered because the molecules initiat

171 melanoma growth in C57BL/6 mice by inducing necrotic death of tumor cells, without causing liver, sp

172 en species production, eventually leading to necrotic death of U251 glioma cells but not primary astr

173 es a previously unrecognized novel regulated necrotic death pathway that involves mitochondrial homeo

174 Although other types of necrotic death such as pyroptosis and necroptosis are me

175 n in vitro and in vivo, a typical feature of necrotic death, and inhibition of mitochondrial fission

176 bligatory respiratory conditions, leading to necrotic death.

177 Strikingly, necrotic debris also render macropinocytic, but not non-

178 erile injuries is necessary for clearance of necrotic debris and for coordination of tissue regenerat

179 thermore, Simu is necessary for clearance of necrotic debris and retention of macrophages at wounds.

180 In vitro, AIM enhanced the engulfment of necrotic debris by macrophages derived from zymosan-indu

181 the scavenging of apoptotic cell bodies and necrotic debris by PDA cells.

182 nclude antigens present in apoptotic bodies, necrotic debris, exosomes or even release of non-vesicul

183 ted protein DFNA5 after Asp270 to generate a necrotic DFNA5-N fragment that targets the plasma membra

184 Occasional necrotic diapause larvae were observed which displayed i

185 Necrotic enteritis (NE) caused by Clostridium perfringen

186 Necrotic enteritis (NE) caused by Clostridium perfringen

187 ing cause of food-poisoning and causes avian necrotic enteritis, posing a significant problem to both

188 , causing autologous immune stimulation upon necrotic exposure.

189 of pro-inflammatory factors induced by tumor necrotic factor alpha.

190 pathology, including decreased inflammation, necrotic fibers and fibrosis in heterogenetic parabionts

191 c-Fos-expressing livers display necrotic foci, immune cell infiltration, and altered hep

192 d protein mu1 in limiting the induction of a necrotic form of cell death called necroptosis.

193 rotein sigma3 in limiting the induction of a necrotic form of cell death called necroptosis.

194 or morphology indicated comparatively larger necrotic fractions for (177)Lu-DOTA-JR11 despite further

195 dramatic diagnostic wounds characterised by necrotic gashes to the head and extremities.

196 he macrophage phagosome and later within the necrotic granuloma.

197 Tuberculous pneumonia, necrotic granulomatous lesions, and bacterial disseminat

198 MCs have the ability to label differentially necrotic HeLa cells from living cells.

199 CD40-induced hyperinflammatory syndrome with necrotic hepatitis and in a nonalcoholic steatohepatitis

200 our data demonstrate that gp96 released from necrotic hepatocytes aggravates immune hyperactivation a

201 hages, isolated from APAP-treated mice, with necrotic hepatocytes decreased expression of proinflamma

202 nd highly contagious disease that results in necrotic hooves and significant economic losses in agric

203 cular patterns (DAMPs) released from damaged/necrotic host cells are crucial factors in induction of

204 Necrotic human primary hepatocytes exposed to acetaminop

205 troma, but also to cancer stem-like cells in necrotic/hypoxic regions.

206 zobia and their symbiotic plant cells become necrotic immediately after rhizobia are released from in

207 in conjunction with surgical debridement of necrotic infected tissues.

208 exerts cardioprotective effects by reducing necrotic injury and edema formation via adenosine-depend

209 ite (tibia vs jaw) nor pathology (healthy vs necrotic jaw bone tissue) affected the averaged spectral

210 PnV-tumor was necrotic, lacking the histopathological characteristics

211 ed approaches are an important complement to necrotic lesion-based approaches and should be used in c

212 ficking of monocyte-derived macrophages into necrotic lesions after APAP overdose.

213 tact with living rice epidermal cells before necrotic lesions develop.

214 red delivery of antifungals to hyphae within necrotic lesions is thought to contribute to therapeutic

215 seedlings silenced for Sl2/3-MMP expression, necrotic lesions were observed at the base of the hypoco

216 drug-tolerant persister population only when necrotic lesions were present.

217 The necrotic lesions were seen in the grey matter at 100 Gy

218 d by a hypersensitive response that included necrotic lesions, up-regulated ROS-generating and -scave

219 d the formation of an advanced lesion with a necrotic lipid core, indicative of plaque vulnerability.

220 onse to necrotic liver injury and found that necrotic liver cells induced eosinophil recruitment.

221 Necrotic liver induced eosinophil IL-1beta and IL-18 sec

222 n vivo model to study the immune response to necrotic liver injury and found that necrotic liver cell

223 These mutant mice presented with severely necrotic liver parenchyma and significantly larger hypox

224 Consistently, NETs are present in necrotic lung lesions of TB patients responding poorly t

225 that have alleviated cell death, less severe necrotic lung lesions, more efficient Mtb growth control

226 lls were injected systemically, coupled with necrotic lysates, a higher number of large lesions was d

227 where MDA-MB-231 xenografts were exposed to necrotic lysates, resulted in an increase in both prolif

228 lution, leads to accumulation of secondarily necrotic macrophages and foam cells and the formation of

229 Type 3 seeds were composed of more than 90% necrotic material admixed with few macrophages and viabl

230 e and dilated bile ducts filled with caseous necrotic material were seen intra-operatively.

231 "Cloud" seeds are mostly composed of necrotic material, explaining their lack of therapeutic

232 eral miliary lesions in the lungs along with necrotic mediastinal lymphadenopathy.

233 Dying PRs exhibited a necrotic morphology, increased 8-hydroxyguanosine abunda

234 rus (CLSV; genus Aureusvirus) and red clover necrotic mosaic virus (RCNMV; genus Dianthovirus) were d

235 s of cucumber leaf spot virus and red clover necrotic mosaic virus.

236 We observed that regenerating and necrotic muscle fibers in muscle biopsy samples from DMD

237 ion in dermatomyositis, lupus nephritis, and necrotic muscle fibres in Duchenne dystrophy.

238 T1 cutoff values for oedematous versus necrotic myocardium were identified as 1251 ms and 1400

239 from survival analysis); low risk/completely necrotic (n = 7; zero relapses), intermediate risk (n =

240 ther positive, viable tumor (V) or negative, necrotic (N).

241 lso shows that cellular RNA decreases during necrotic, necroptotic, and apoptotic cell death caused b

242 sented with higher percentages of healthy or necrotic neutrophils but lower percentages of apoptotic

243 Few intact and necrotic neutrophils were detected at MGAS315 infection

244 ntages of early apoptotic and late apoptotic/necrotic neutrophils were similar.

245 defense, little is known about Mtb-infected necrotic neutrophils.

246 ribution of H(2)S-producing enzymes in human necrotic, nonnecrotic, and cavitary pulmonary tuberculos

247 hich is proliferating, hypoxic/quiescent and necrotic/nutrient-deficient).

248 (mdDCs) and murine DCs and did not have any necrotic or apoptotic effects even at high densities.

249 tosis, without altering recognition of live, necrotic, or Ig-opsonized cells.

250 ed robust Mincle expression upon exposure to necrotic osteocytes in vitro and in vivo.

251 iated molecular patterns (DAMPs) released by necrotic osteocytes via macrophage-inducible C-type lect

252 hole tumor (P = 0.0009, P = 0.02) as well as necrotic (P = 0.008, P = 0.02) and viable (P = 0.003, P

253 (18)F-FDG in the whole tumor (P = 0.001) and necrotic (P = 0.02) and viable (P = 0.0001) tissues.

254 airway epithelial cells via the apoptotic or necrotic pathway; involvement of the pyroptosis pathway

255 edian survival time, and a 26.6% increase in necrotic percentage compared to ISFIs without TUS exposu

256 red to be caused by deposition of AIM at the necrotic peritoneum in AIM (+/+) mice.

257 ogenous ROS in the form of H2O2 reversed the necrotic phenotype and restored CD95 expression on infec

258 olid tumors versus tumor heterogeneity and a necrotic phenotype, and optimal selection of tumor slice

259 ize; solid tumor vs. tumor heterogeneity and necrotic phenotype; and optimal selection of tumor slice

260 These results suggest that necrotic platelets interact with neutrophils to exacerba

261 ex vivo-formed PNAs revealed a propensity of necrotic platelets to interact with neutrophils.

262 nhibition of GPX4 in H295R cells led to high necrotic populations compared to control, while cotreatm

263 r heterogeneity, and spatial distribution of necrotic/proliferating cells.

264 nfection, a population of macrophages became necrotic, providing a niche for M. tuberculosis replicat

265 c implants and adjacent teeth with vital and necrotic pulps.

266 Strong correlations were found for the necrotic (r = 0.88) and viable fractions (r = 0.87) betw

267 Adiponectin overexpression in vivo decreased necrotic region and increased regenerating myofibers.

268 Furthermore, we successfully detected necrotic regions within these tumor spheroids based on i

269 udopalisading cells that envelop the hypoxic-necrotic regions, and mitochondrial NIX expression was r

270 , in terms of location, size and presence of necrotic regions, to determine the ideal infusion site a

271 geted and localized at both tumor stroma and necrotic regions.

272 They are also enriched in perivascular and necrotic regions.

273 osteocalcin in the osteoblasts localized in necrotic regions.

274 oyasaponins and organic acids in the central necrotic regions.

275 The necrotic RFA lesions involved multiple esophageal tissue

276 sts a paradigm shift in which targeting late necrotic-secreted factors may increase survival and enha

277 monstrated that combining an antagonist of a necrotic signal with an anticancer treatment potentiates

278 OX lines developed brown necrotic spots on the leaves that did not appear on null

279 hannels also participate as causal actors in necrotic swelling and apoptotic volume decrease.

280 l pathogenicity factors, P. ananatis induces necrotic symptoms and extensive cell death in onion tiss

281 motherapy and in a mouse model of human-like necrotic TB lung granulomas.

282 very common clinical sign - hardening of the necrotic testicle.

283 ges plays a major role in the development of necrotic, thin-capped plaques.

284 is 100%, but reduced to 80% if targeting of necrotic tissue from previous transurethral resections o

285 Defective clearance of necrotic tissue interferes with amelioration of tissue i

286 y a sustained calcium flux upon contact with necrotic tissue that requires sensing of the damage sign

287 temic antibiotics and radical debridement of necrotic tissue, lethality remains high.

288 l diameter, and distance from vasculature to necrotic tissue.

289 8.22% +/- 14.59 more Lipiodol in viable than necrotic tumor areas.

290 This suggests that apoptotic and necrotic tumor cells, via efferocytosis and IDO1, respec

291 ssociated with well-perfused, hypoxic and/or necrotic tumor compartments.

292 detected in seconds with single MS scans of necrotic tumor tissue smears, which further accelerates

293 crosis, may permit rapid characterization of necrotic tumors from tissue slices.

294 rawling of the intraductal lesions extracted necrotic tumour-like tissue which was histologically con

295 th PC but considered them as opportunists in necrotic tumour.

296 s "tassel blasting." We identified a mutant, necrotic upper tips1 (nut1), that mimics tassel blasting

297 enograft, (2) classification of these areas (necrotic/viable) to compare similar types of tissues, (3

298 screwworm flies, C. macellaria, that invade necrotic wound and feed on dead tissue.

299 pider venom biomolecules induced smaller and necrotic xenogeneic GB; spider venom activated the innat

300 ation of anti-MG1 HNPs can enlarge a tumor's necrotic zone with photothermal ablation.