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1 s to be triggered at the periphery of the HR necrotic lesion.
2 , inhibited tumor growth and led to smaller, necrotic lesions.
3 s, Pst DC3000 induced accelerated coalescing necrotic lesions.
4 lated in capillaries and neurons surrounding necrotic lesions.
5 to causes MAPKKKalpha-dependent formation of necrotic lesions.
6 restricted to myocytes surrounding piecemeal necrotic lesions.
7 e infection was associated with larger, more necrotic lesions.
8 ulated factors involved in the production of necrotic lesions.
9 aic virus, or tobacco etch virus resulted in necrotic lesions.
10 Ls resulted only in various degrees of small necrotic lesions.
11 that this feature was essential in repairing necrotic lesions.
12 y during the formation of sorgoleone-induced necrotic lesions.
13 , pMLC) to squeeze and finally eliminate the necrotic lesions.
14  Arabidopsis is characterized by spontaneous necrotic lesions, accumulation of reactive oxygen specie
15 ficking of monocyte-derived macrophages into necrotic lesions after APAP overdose.
16 sis and treatment can prevent progression of necrotic lesions and bone collapse.
17                                              Necrotic lesions and necrotic cell death characterize se
18 th lower blood urea nitrogen levels, reduced necrotic lesions, and higher survival rates.
19 ion with the wild-type GAS isolate generated necrotic lesions, and in some animals the GAS disseminat
20 in aconitase activity, stunting, spontaneous necrotic lesions, and increased resistance to paraquat.
21 eins, and manganite [Mn(III)] accumulated in necrotic lesions apparently through low Mn sequestration
22 tion process and coincided with formation of necrotic lesions approximately 5 days after inoculation.
23 wth and sporadic development of chlorotic or necrotic lesions at inoculation sites.
24 ed approaches are an important complement to necrotic lesion-based approaches and should be used in c
25 ed-9 specifically abrogated the formation of necrotic lesions, but not other symptoms, in tomato leav
26 es of seedlings showed significantly smaller necrotic lesions compared with nonprimed plants, coincid
27 ed with ex vivo porcine cardiac tissues with necrotic lesions created by different ablation durations
28 tact with living rice epidermal cells before necrotic lesions develop.
29 patocyte proliferation; however, how hepatic necrotic lesions during acute or chronic liver diseases
30 ves developed abnormally and exhibited large necrotic lesions even when supplied with sucrose.
31 nt, speckle, is characterized by spontaneous necrotic lesion formation on leaves, root, and stems, si
32 rapid weight loss, bacteremia, and abdominal necrotic lesion formation.
33 shift within the lobule in parallel with the necrotic lesion in these animals.
34 pregulate p50 transcription and induce local necrotic lesions in an A. tumefaciens infiltration assay
35 uronal cell loss, demyelination, gliosis and necrotic lesions in post-mortem material.
36 oduced high levels of ethylene and developed necrotic lesions in response to an acute O3 exposure tha
37        We used C3HeB/FeJ mice, which develop necrotic lesions in response to Mycobacterium tuberculos
38 protein gene PR-1a and developed spontaneous necrotic lesions in the absence of any pathogen.
39  treatment groups showed significantly fewer necrotic lesions in the cerebral cortex, caudate nucleus
40 gene completely lack chlorophyll and develop necrotic lesions in the light.
41     The appearance of multiple granulomatous-necrotic lesions in the liver correlates with a marked i
42                          This study revealed necrotic lesions in the peripheries of Ahr-/- fetal live
43 infection and contribute to the formation of necrotic lesions in tuberculosis.
44 rometry (GC/MS) resulted in the formation of necrotic lesions, indicating that the compound caused se
45 c, but not normoglycemic, animals showed pan-necrotic lesions ('infarction') after 4 h of recirculati
46 red delivery of antifungals to hyphae within necrotic lesions is thought to contribute to therapeutic
47 dence of muscle regeneration due to previous necrotic lesions likely caused by earlier SINV infection
48 t Slo was not necessary for the formation of necrotic lesions, nor was it necessary for escape from t
49 sion, MoMFs play a key role in repairing the necrotic lesions, not only by removing necrotic tissues,
50 ase in Opn transcription was demonstrated in necrotic lesions of both DCC-susceptible C3H/He and B6.C
51 onse to B. cinerea, measured as expansion of necrotic lesions on leaves and accumulation of the antim
52  These promoter::p50 constructs induce local necrotic lesions on NN tobacco plants in an Agrobacteriu
53 he ex vivo study, it successfully identified necrotic lesions on porcine cardiac tissue, distinguishi
54 he PPO-silenced plants developed spontaneous necrotic lesions on their leaves in the absence of patho
55 ct plants, but they can colonize and develop necrotic lesions on wounded leaves and stems.
56 h muscle actin-expressing HSCs to facilitate necrotic lesion resolution.
57           Histopathologic examination of the necrotic lesion revealed bacteria throughout inflamed su
58  was more likely than the wild type to yield necrotic lesions, suggesting that mucoidy contributes to
59 o, causing disease symptoms characterized by necrotic lesions surrounded by chlorosis.
60  to be strongly reduced in its production of necrotic lesion symptoms.
61 ) and csrRS(-) bacteria yielded larger, more necrotic lesions than did either strain at twice the ino
62 es and was characterized by an initial focal necrotic lesion that rapidly progressed to invasion of t
63 d growth alterations and their leaves showed necrotic lesions that appeared similar to lesions charac
64 D mice verified that they do not exhibit the necrotic lesions that are usually associated with SCD.
65 , in a developmentally programmed manner, of necrotic lesions that expand to kill leaves cell autonom
66 e mice, young B10.T(6R) mice did not develop necrotic lesions throughout the liver.
67 ys enhanced stress responses and spontaneous necrotic lesions under drought conditions in the absence
68 d by a hypersensitive response that included necrotic lesions, up-regulated ROS-generating and -scave
69 seedlings silenced for Sl2/3-MMP expression, necrotic lesions were observed at the base of the hypoco
70 drug-tolerant persister population only when necrotic lesions were present.
71                                        Focal necrotic lesions were seen in all cases at MR, and five
72                                          The necrotic lesions were seen in the grey matter at 100 Gy
73 ption factor 9+ (SOX9+) hepatocytes near the necrotic lesions, which acted as a barrier from further
74 e vascular changes that precede the onset of necrotic lesions with the thalamus of the pyrithiamine-i
75 ith PB or betaNF shifted the location of the necrotic lesion within the lobule from zone 2, as observ