ライフサイエンスコーパス: necrotic tissue

1 n (the time points at which cells migrate to necrotic tissues).

2 l diameter, and distance from vasculature to necrotic tissue.

3 ans of selectively responding to bacteria or necrotic tissue.

4 was the sole microbial isolate from debrided necrotic tissue.

5 localized to the microvessels bordering pan necrotic tissue.

6 agnosis of the PET-positive mass showed only necrotic tissue.

7 expressed ICAM-1 mRNA and extended into the necrotic tissue.

8 gical processes that occur within regions of necrotic tissue.

9 Histologically confirmed necrotic tissue (432 voxels) did not demonstrate any obs

10 Debridement (removal of necrotic tissue and foreign bodies from the wound) at di

11 ly significantly different from the ratio in necrotic tissue and from each other.

12 han healing infarcts, owing to resorption of necrotic tissue and maturation of scar, but infarct size

13 sequent time points revealed liquefaction of necrotic tissue and replacement with granulation tissue.

14 1 is released from activated immune cells or necrotic tissues and acts as a damage-associated molecul

15 IAIP and HMW-HA colocalized with histones in necrotic tissues and areas that displayed neutrophil ext

16 d in a number of tissues and is prevalent in necrotic tissues and granulomata.

17 ning myocytes are unable to reconstitute the necrotic tissue, and the post-infarcted heart deteriorat

18 we found that most bacteria were located in necrotic tissue as large colonies far (750 micro m) from

19 ere was one major complication: Intraluminal necrotic tissue attached to the bladder, which was remov

20 image through blood layer and differentiate necrotic tissue based on spectral characteristics.

21 g the necrotic lesions, not only by removing necrotic tissues, but also by inducing cell death-resist

22 findings to determine the characteristics of necrotic tissue by postreperfusion MCE.

23 Surgery included debridement of necrotic tissue, carpal tunnel decompression, and extern

24 may be caused by the combined effect of the necrotic tissue (cecal ligation, [CL]) and other microbi

25 ers of dystrophic pathology (area of damaged necrotic tissue, central nuclei) were reduced in benfoti

26 imal treatment relies on surgical removal of necrotic tissues covered by a split-skin graft.

27 ic cells specialize in cross-presentation of necrotic tissue-derived epitopes to directly activate cy

28 Necrotic tissue did not enhance.

29 sminogen deficiency impedes the clearance of necrotic tissue from a diseased hepatic microenvironment

30 is 100%, but reduced to 80% if targeting of necrotic tissue from previous transurethral resections o

31 e of this study was to assess the ability of necrotic tissue, in the presence or absence of low-dose

32 Defective clearance of necrotic tissue interferes with amelioration of tissue i

33 model of macrophage loss following uptake of necrotic tissue is proposed to explain macrophage deplet

34 temic antibiotics and radical debridement of necrotic tissue, lethality remains high.

35 races each individually specializing on the necrotic tissues of a different cactus species.

36 low activity on SPECT and the percentage of necrotic tissue on immunohistochemistry.

37 verdose induces massive hepatocyte necrosis, necrotic tissue releases high mobility group B1 (HMGB1)

38 Necrotic tissue releases high mobility group B1 (HMGB1),

39 stress gradient, but changed after bone and necrotic tissue resorption.

40 ction of non-enhancing fluid collections and necrotic tissues, rim-enhancing abscesses, heterogeneous

41 y a sustained calcium flux upon contact with necrotic tissue that requires sensing of the damage sign

42 In infected and necrotic tissue, the Fc-binding proteins were removed fr

43 w muscle synthesis, the human heart replaces necrotic tissue with deposition of a noncontractile scar

44 Pretreatment of apoptotic and necrotic tissue with Klenow enzyme with or without added

45 otomy and resection of this area showed only necrotic tissue with no viable melanoma cells.

46 leading to the accumulation of apoptotic and necrotic tissue within the plaque, which results in enla

47 nical improvement and there is no additional necrotic tissue within the wound.