ライフサイエンスコーパス: necrotize

1 ge saturates and the specimen is effectively necrotized.

2 Necrotizing and crescentic GN (NCGN) with a paucity of g

3 s discussed in thrombotic microangiopathies, necrotizing and crescentic GN, acute tubular necrosis, a

4 ght patients with active, noninfectious, non-necrotizing anterior scleritis with a scleral inflammato

5 ve participants with active, autoimmune, non-necrotizing anterior scleritis with scleral inflammatory

6 icacy of gevokizumab in the treatment of non-necrotizing anterior scleritis.

7 s, including invasive aspergillosis, chronic necrotizing aspergillosis, fungal colonization, and alle

8 Necrotizing autoimmune myopathy (NAM) is characterized p

9 including rhabdomyolysis and statin-induced necrotizing autoimmune myopathy (SINAM), are rare.

10 Necrotizing autoimmune myopathy was idiopathic in half o

11 a neonate who presented with hydrocephalus, necrotizing cellulitis, systemic inflammation, and respi

12 is central to ANCA-associated vasculitis and necrotizing crescentic glomerulonephritis (NCGN).

13 gan, including the kidneys, where they cause necrotizing crescentic glomerulonephritis.

14 pothesis that endothelial NF-kappaB mediates necrotizing crescentic GN (NCGN) and provides a specific

15 ANCA-associated vasculitis features necrotizing crescentic GN (NCGN), and ANCA-activated neu

16 CA-activated phagocytes cause vasculitis and necrotizing crescentic GN (NCGN).

17 With the emergence of necrotizing/crescentic glomerulonephritis, approximately

18 tional immunizations were needed to induce a necrotizing/crescentic glomerulonephritis.

19 xperimental autoimmune glomerulonephritis or necrotizing/crescentic glomerulonephritis.

20 s like haemorrhages, diffuse cerebral edema, necrotizing encephalitis and decreased bone size (Alizar

21 we show that Parl ablation in mouse causes a necrotizing encephalomyelopathy similar to Leigh syndrom

22 liver function and Leigh syndrome (subacute necrotizing encephalomyelopathy) seen in association wit

23 ific acute encephalopathy syndromes (4 acute necrotizing encephalopathy, 1 acute infantile encephalop

24 c infarct, hemorrhage, and acute hemorrhagic necrotizing encephalopathy.

25 (17.5% [95% CI, 16.5%-18.6%]), and death or necrotizing enterocolitis (19.3% [95% CI, 18.1%-20.4%]).

26 there were increases in deaths attributed to necrotizing enterocolitis (30 [95% CI, 27 to 34] vs. 23

27 urvivors (23.3%, 19.1%, and 11.7%), death or necrotizing enterocolitis (48.1%, 37.1%, and 32.5%), and

28 in mortality (AOR, 0.98; 95% CI, 0.70-1.37), necrotizing enterocolitis (AOR, 0.88; 95% CI, 0.65-1.20)

29 utcomes included all-cause mortality, severe necrotizing enterocolitis (NEC) (Bell stage II or more),

30 Necrotizing enterocolitis (NEC) affects up to 10% of pre

31 LNT were shown to protect neonatal rats from necrotizing enterocolitis (NEC) and are good therapeutic

32 t gut microbiota alterations associated with necrotizing enterocolitis (NEC) and feeding intolerance

33 d blood cell (RBC) transfusion and anemia to necrotizing enterocolitis (NEC) are conflicting.

34 Necrotizing enterocolitis (NEC) continues to be a major

35 Necrotizing enterocolitis (NEC) has long remained a sign

36 ant manifestation of severe diseases such as necrotizing enterocolitis (NEC) in neonates or bowel wal

37 Necrotizing enterocolitis (NEC) is a devastating disease

38 Necrotizing enterocolitis (NEC) is a devastating disease

39 Necrotizing enterocolitis (NEC) is a devastating gastroi

40 Necrotizing enterocolitis (NEC) is a devastating gastroi

41 Necrotizing enterocolitis (NEC) is a devastating inflamm

42 Necrotizing enterocolitis (NEC) is a devastating intesti

43 Necrotizing enterocolitis (NEC) is a disease of neonatal

44 Necrotizing enterocolitis (NEC) is a major cause of morb

45 Necrotizing enterocolitis (NEC) is a major cause of neon

46 Necrotizing enterocolitis (NEC) is an idiopathic, inflam

47 Necrotizing enterocolitis (NEC) is an inflammatory bowel

48 Necrotizing enterocolitis (NEC) is an inflammatory disea

49 Necrotizing enterocolitis (NEC) is one of the most serio

50 Necrotizing enterocolitis (NEC) is the most common and s

51 Necrotizing enterocolitis (NEC) is the most common gastr

52 The pathophysiology of necrotizing enterocolitis (NEC) remains poorly understoo

53 t of lung disease in the setting of neonatal necrotizing enterocolitis (NEC), a life-threatening gast

54 ure and role of the intestinal leukocytes in necrotizing enterocolitis (NEC), a severe disease affect

55 y result in antibiotic resistance, fungemia, necrotizing enterocolitis (NEC), and mortality.

56 y infections, otitis media, gastroenteritis, necrotizing enterocolitis (NEC), and sudden infant death

57 lk feeding is associated with lower rates of necrotizing enterocolitis (NEC), but an understanding of

58 the widespread use of plain films to detect necrotizing enterocolitis (NEC), it is considered a time

59 reast milk (HBM) attenuates the incidence of necrotizing enterocolitis (NEC), which remains a leading

60 possible pregnancy-related risk factors for necrotizing enterocolitis (NEC)-associated deaths during

61 is supposedly associated with development of necrotizing enterocolitis (NEC).

62 bile acids (BAs) may mediate development of necrotizing enterocolitis (NEC).

63 diseases characterized by ISC loss including necrotizing enterocolitis (NEC).

64 riate analysis, SL mortality predictors were necrotizing enterocolitis (NEC; surgical odds ratio, 5.9

65 pathy of prematurity (stage 3 or higher), or necrotizing enterocolitis (stages 2-3).

66 ve been implicated as a pathogenic factor in necrotizing enterocolitis and inflammatory bowel disease

67 bowel diseases (controls) and 8 infants with necrotizing enterocolitis and measured levels of S100A8

68 premature newborns has been shown to prevent necrotizing enterocolitis and reduce all-cause mortality

69 ts, where the virus has been associated with necrotizing enterocolitis and severe and persistent diar

70 Notably, ELGAN/ELBW infants developing necrotizing enterocolitis before day 14 had higher expre

71 Infants with necrotizing enterocolitis born into midlevel hospitals (

72 infants is associated with a reduced risk of necrotizing enterocolitis but an increased risk of late-

73 ptation to small-bowel resection (SBR) after necrotizing enterocolitis expands absorptive surface are

74 Here we show that necrotizing enterocolitis in neonate mice is accompanied

75 given enterally may reduce the incidence of necrotizing enterocolitis in preterm infants.

76 diabetes, nonalcoholic fatty liver disease, necrotizing enterocolitis in very low birth weight infan

77 Late-onset invasive infection or necrotizing enterocolitis occurred in 32% of infants (19

78 Necrotizing enterocolitis occurred in 70 of 1394 infants

79 Necrotizing enterocolitis occurred less frequently in th

80 Necrotizing enterocolitis occurred more frequently in in

81 CI, 0.58-0.78) and the combined outcomes of necrotizing enterocolitis or death and severe intraventr

82 r retinopathy of prematurity and surgery for necrotizing enterocolitis or spontaneous intestinal perf

83 Interestingly, we find that necrotizing enterocolitis patients also exhibit decrease

84 Infants with necrotizing enterocolitis represent a high-risk subgroup

85 he liberal vs restrictive thresholds groups, necrotizing enterocolitis requiring surgical interventio

86 ere intraventricular hemorrhage and death or necrotizing enterocolitis was lowest among infants born

87 of the study, just 28.6% of the infants with necrotizing enterocolitis were born into high-level, hig

88 nary dysplasia, severe cerebral lesions, and necrotizing enterocolitis).

89 2, periventricular leukomalacia >grade 1, or necrotizing enterocolitis).

90 with LOS and 164 unaffected controls (no LOS/necrotizing enterocolitis).

91 prolonged the survival of newborn mice with necrotizing enterocolitis, a severe pathology in preterm

92 sed sample of 30 566 VLBW infants, 1879 with necrotizing enterocolitis, according to the level of car

93 dities before PDA closure, including sepsis, necrotizing enterocolitis, and a dependence on mechanica

94 e, confirmed or suspected late-onset sepsis, necrotizing enterocolitis, and cerebral palsy.

95 aturity, severe intraventricular hemorrhage, necrotizing enterocolitis, and chronic lung disease amon

96 s, hearing loss, bronchopulmonary dysplasia, necrotizing enterocolitis, and severe retinopathy of pre

97 om BEC on premature infants with and without necrotizing enterocolitis, and successfully provided a t

98 iventricular or intraventricular hemorrhage, necrotizing enterocolitis, aspiration, retinopathy of pr

99 rematurity, intracranial hemorrhage, sepsis, necrotizing enterocolitis, bronchopulmonary dysplasia, o

100 Necrotizing enterocolitis, defined as Bell stage 2 or gr

101 who survived more than 12 hours, were severe necrotizing enterocolitis, infection, bronchopulmonary d

102 Infections and necrotizing enterocolitis, major causes of mortality and

103 hemorrhage, bronchopulmonary dysplasia, and necrotizing enterocolitis, most therapeutic approaches h

104 nography, severe retinopathy of prematurity, necrotizing enterocolitis, or late-onset sepsis) by 36 w

105 of serious infection (sepsis or meningitis), necrotizing enterocolitis, or mortality during the first

106 ystic periventricular leukomalacia, surgical necrotizing enterocolitis, or stage 3 or greater retinop

107 de 3-4, cystic periventricular leukomalacia, necrotizing enterocolitis, retinopathy of prematurity st

108 ites of death or bronchopulmonary dysplasia, necrotizing enterocolitis, retinopathy of prematurity, a

109 It also decreases the prevalence of necrotizing enterocolitis, sepsis, and intraventricular

110 PN significantly affects risk of mortality, necrotizing enterocolitis, sepsis, chronic lung disease,

111 onary hemorrhage but not with differences in necrotizing enterocolitis, severe bronchopulmonary dyspl

112 No differences in rates of necrotizing enterocolitis, severe bronchopulmonary dyspl

113 etinopathy of prematurity requiring surgery, necrotizing enterocolitis, spontaneous intestinal perfor

114 nical complications of prematurity including necrotizing enterocolitis, systemic infections and bronc

115 ria in premature infants are associated with necrotizing enterocolitis, underscoring the critical rol

116 ntral nervous system injury decreased, while necrotizing enterocolitis-related deaths increased.

117 d incidence of neonatal bacterial sepsis and necrotizing enterocolitis.

118 h postnatal intestinal pathologies including necrotizing enterocolitis.

119 re intraventricular hemorrhage, and death or necrotizing enterocolitis.

120 ities in infants who did and did not develop necrotizing enterocolitis.

121 rain was common to all infants who developed necrotizing enterocolitis.

122 on, a process that may lead to diseases like necrotizing enterocolitis.

123 am, urinary tract, or cerebrospinal fluid or necrotizing enterocolitis.

124 ch were profoundly depleted in newborns with necrotizing enterocolitis.

125 ther this practice reduces the prevalence of necrotizing enterocolitis.

126 so a trend towards a diminished incidence of necrotizing enterocolitis.

127 achieve the rate from the best quartile for necrotizing enterocolitis.

128 Cytotoxic necrotizing factor 1 (CNF1) and hemolysin (Hly) are toxi

129 Cytotoxic necrotizing factor 1 (CNF1) is a key UPEC toxin; however

130 ore chronic comorbidities than patients with necrotizing fasciitis (20 [87.0%] vs 17 [54.8%]; P = .02

131 or patients with cellulitis vs patients with necrotizing fasciitis (3 [2-5] vs 5 [3-11]; P = .01), wh

132 slightly younger and had a higher degree of necrotizing fasciitis (56% vs 14%).

133 known as strep throat) to severely invasive necrotizing fasciitis (also known as the flesh-eating sy

134 , imaging, and Laboratory Risk Indicator for Necrotizing Fasciitis (LRINEC) score in diagnosis of nec

135 n = 23; mean [SD] age, 57.2 [17.7] years) or necrotizing fasciitis (n = 31; mean [SD] age, 54.3 [13.5

136 Necrotizing fasciitis (NF) caused by flesh-eating bacter

137 Shock frequently complicates necrotizing fasciitis (NF) caused by group A Streptococc

138 An earlier report described a human case of necrotizing fasciitis (NF) caused by mixed infection wit

139 Necrotizing fasciitis (NF) is a destructive skin and sof

140 erile site or from a wound in a patient with necrotizing fasciitis (NF) or streptococcal toxic shock

141 hort study describes mortality predictors of necrotizing fasciitis (NF).

142 than the carriage strain in a mouse model of necrotizing fasciitis and had enhanced growth ex vivo in

143 Necrotizing fasciitis and myositis are devastating infec

144 Necrotizing fasciitis and myositis caused by group A str

145 sociated with invasive infections, including necrotizing fasciitis and toxic shock syndrome.

146 itis and impetigo to severe outcomes such as necrotizing fasciitis and toxic shock syndrome.

147 mic invasive infections worldwide, including necrotizing fasciitis and toxic shock syndrome.

148 p throat") to invasive conditions, including necrotizing fasciitis and toxic shock syndrome.

149 treptococcal toxic shock syndrome (STSS) and necrotizing fasciitis are the 2 most severe invasive man

150 the reason for ICU admission in 23 patients, necrotizing fasciitis in 31 patients, and other diagnose

151 this report, we present a case of multifocal necrotizing fasciitis in a healthy adult patient, second

152 Current therapeutic regimens for necrotizing fasciitis include surgical debridement and t

153 GAS upregulates the AdcR regulon during necrotizing fasciitis infection in WT mice but not in S1

154 Periocular necrotizing fasciitis is a rare but potentially devastat

155 Necrotizing fasciitis is often confused for cellulitis a

156 As seen in these 5 cases, periocular necrotizing fasciitis may cause severe visual loss more

157 ecal diversion surgery, 2 patients developed necrotizing fasciitis of a lower extremity that required

158 terile site or from a wound in patients with necrotizing fasciitis or streptococcal toxic shock syndr

159 k thus aimed to address whether CLI improves necrotizing fasciitis outcome by modulating virulence fa

160 a pathogen that causes both invasive (e.g., necrotizing fasciitis) and noninvasive (e.g., pharyngiti

161 e.g. pharyngitis) to severely invasive (e.g. necrotizing fasciitis).

162 tis, otitis, etc.) to life-threatening (e.g. necrotizing fasciitis).

163 , amputations, acute kidney injury, perineal necrotizing fasciitis, and hyperkalemia.

164 GAS), the causative agent of pharyngitis and necrotizing fasciitis, secretes the potent cysteine prot

165 fection (streptococcal toxic shock syndrome, necrotizing fasciitis, septic shock, or GAS cellulitis w

166 h cellulitis and patients with patients with necrotizing fasciitis, Staphylococcus aureus (10 [43.5%]

167 itically ill on admission than patients with necrotizing fasciitis, they have more chronic comorbidit

168 During the invasive infection necrotizing fasciitis, this is in part due to the GAS pr

169 istered as soon as possible to patients with necrotizing fasciitis, while our in vitro studies emphas

170 the virulence phenotype in a mouse model of necrotizing fasciitis.

171 life-threatening invasive infections such as necrotizing fasciitis.

172 d to be less severely ill than patients with necrotizing fasciitis.

173 sed virulence in a nonhuman primate model of necrotizing fasciitis.

174 and increased virulence in a mouse model of necrotizing fasciitis.

175 uding streptococcal toxic shock syndrome and necrotizing fasciitis.

176 cause illnesses ranging from pharyngitis to necrotizing fasciitis.

177 rom a nonhuman primate experimental model of necrotizing fasciitis.

178 infections such as toxic shock syndrome and necrotizing fasciitis.

179 type virulence phenotype in a mouse model of necrotizing fasciitis.

180 e-threatening invasive infections, including necrotizing fasciitis.

181 irulence in animal models of pharyngitis and necrotizing fasciitis.

182 red from nonhuman primates with experimental necrotizing fasciitis.

183 od and reduced virulence in a mouse model of necrotizing fasciitis.

184 d we review literature on this rare cause of necrotizing fasciitis.

185 tions to life-threatening infections such as necrotizing fasciitis.

186 n nonhuman primate models of pharyngitis and necrotizing fasciitis.

187 Streptococcus anginosus constellatus causing necrotizing fasciitis.

188 litis and to compare them with patients with necrotizing fasciitis.

189 inflammation in the central nervous system; necrotizing focal myelitis in the cervical spinal cord;

190 en in the pituitary can cause an aggressive (necrotizing) form of hypophysitis through type IV (T-cel

191 Focal and segmental necrotizing glomerular lesions with crescents, mimicking

192 Pauci-immune focal necrotizing GN (piFNGN) is usually associated with ANCAs

193 Using an established murine model of focal necrotizing GN mediated by autoimmunity to MPO (autoimmu

194 xic effect of NET-related histone release in necrotizing GN.

195 tion on chest imaging, and poorly formed non-necrotizing granulomas on pathology.

196 hil cytoplasmic antibodies (ANCA)-associated necrotizing granulomatous vasculitis that affects small

197 (PP) or RSA59 (P) yielded similar degrees of necrotizing hepatitis and meningitis, but only RSA59 (PP

198 al mortality in conjunction with a fulminant necrotizing hepatitis.

199 BS) in 10-d-old mouse pups produces an acute necrotizing ileocolitis resembling human NEC within 24 h

200 mITT necrotizing infection clinical composite endpoint succes

201 g a previously validated composite endpoint, necrotizing infection clinical composite endpoint, defin

202 , a rare urologic emergency characterized by necrotizing infection of the external genitalia, perineu

203 istopathological examination revealed severe necrotizing inflammation in various organs, most promine

204 nephritis, and/or severe systemic multiorgan necrotizing inflammation.

205 s and calcifications, brain infarctions with necrotizing inflammatory response, infiltrate, retinal i

206 Hypoxic necrotizing lesions rendered moxifloxacin less active.

207 arterial smooth muscle cells and deposits in necrotizing lesions.

208 microbial defence uniformly develop hypoxic necrotizing lung lesions, widely observed in human TB.

209 It has a lethal canine equivalent: necrotizing meningoencephalitis.

210 onnected left pancreatic remnant after acute necrotizing mid-body necrosis, definitive surgical manag

211 ith autoimmune myopathy have a predominantly necrotizing muscle biopsy with minimal lymphocytic infil

212 s the spectrum of diseases associated with a necrotizing muscle biopsy.

213 s puerperal sepsis, neonatal infections, and necrotizing myometritis.

214 Immune-mediated necrotizing myopathies (IMNM) may be associated with eit

215 One patient with severe necrotizing myopathy died.

216 t evidence now suggests that immune-mediated necrotizing myopathy is not one disease, but can be divi

217 A decade ago, immune-mediated necrotizing myopathy was recognized as a distinct form o

218 ients with dermatomyositis, polymyositis, or necrotizing myopathy, and 0/20 (0%) age-matched healthy

219 more than 25% of the GAS genes required for necrotizing myositis encode known or putative transporte

220 significantly (P < 0.05) impaired in causing necrotizing myositis in NHPs.

221 ns that identified GAS genes contributing to necrotizing myositis in nonhuman primates (NHP), a clini

222 d SLO toxins to virulence in mouse models of necrotizing myositis, bacteremia, and skin and soft tiss

223 ernatively, statins can induce an autoimmune necrotizing myositis.

224 rred a fitness advantage in a mouse model of necrotizing myositis.

225 ins are equally impaired in ability to cause necrotizing myositis.

226 tic requirements for P. ananatis to colonize necrotized onion tissue and its capacity for tolerance t

227 sues became progressively more fibrinous and necrotizing over time.

228 Biliary stricture in necrotizing pancreatitis (NP) has not been systematicall

229 that the development of colon involvement in necrotizing pancreatitis (NP) negatively affects morbidi

230 Biliary stricture occurs frequently after necrotizing pancreatitis and is associated with splanchn

231 ative approaches to the debridement of acute necrotizing pancreatitis are preferred to open surgical

232 At least 30% of patients with infected necrotizing pancreatitis are successfully treated with c

233 Between 1997 and 2013, patients with necrotizing pancreatitis at the Liverpool Pancreas Cente

234 ins a role in the modern management of acute necrotizing pancreatitis in cases not amenable to less i

235 n vivo in the course of taurocholate-induced necrotizing pancreatitis in rats and in vitro in rat pan

236 aurocholate into the pancreatic duct induced necrotizing pancreatitis in the head of pancreas and lig

237 Infected necrotizing pancreatitis is a highly morbid disease with

238 Colon involvement in necrotizing pancreatitis is associated with substantial

239 SUMMARY/BACKGROUND DATA: Necrotizing pancreatitis is characterized by a profound

240 Colon involvement in necrotizing pancreatitis is common; clinical deteriorati

241 ticipants, we found the step-up approach for necrotizing pancreatitis to be superior to open necrosec

242 l), a surgical step-up approach for infected necrotizing pancreatitis was found to reduce the composi

243 Acute necrotizing pancreatitis was induced by i.p. administrat

244 atients with confirmed or suspected infected necrotizing pancreatitis who required intervention from

245 ndoscopic transluminal approach for infected necrotizing pancreatitis, compared with minimally invasi

246 mild pancreatitis, 20% develop severe and/or necrotizing pancreatitis, requiring advanced medical and

247 ea of intra-parenchymal necrosis, indicating necrotizing pancreatitis.

248 g catheter drainage for (suspected) infected necrotizing pancreatitis.

249 for success of catheter drainage in infected necrotizing pancreatitis.

250 higher mortality when compared with sterile necrotizing pancreatitis.

251 scopic approaches for patients with infected necrotizing pancreatitis.

252 ions, including osteomyelitis, endocarditis, necrotizing pneumonia and sepsis(1).

253 lveolar capillary destruction in hemorrhagic/necrotizing pneumonia caused by CA-MRSA and offer novel

254 revent lethal challenge in a rabbit model of necrotizing pneumonia caused by the USA300 community-ass

255 coccus aureus (MRSA) causes life-threatening necrotizing pneumonia in children.

256 ive sudden, massive loss of lung tissue from necrotizing pneumonia or acute respiratory distress synd

257 ) infection can quickly develop into severe, necrotizing pneumonia, causing over 50% mortality despit

258 th cystic fibrosis (CF) and can cause severe necrotizing pneumonia, which is often fatal.

259 USA300 is the predominant strain that causes necrotizing pneumonia.

260 ng inflammation and, in some cases, a lethal necrotizing pneumonia.

261 rgic bronchopulmonary aspergillosis, chronic necrotizing pulmonary aspergillosis, or invasive aspergi

262 -37 with extracellular actin was observed in necrotized regions of samples from oral lesions.

263 ime of conversion and progressed to a severe necrotizing rejection early despite an unaltered baselin

264 inal detachment repair, endophthalmitis, and necrotizing retinitis were excluded, as were those with

265 irst IgM test had a greater risk of incident necrotizing retinochoroiditis (hazard ratio = 4.47, 95%

266 The increased risk of late necrotizing retinochoroiditis associated with isolated f

267 al manifestation of T gondii infection, with necrotizing retinochoroiditis occurring months or years

268 The incidence of recurrent necrotizing retinochoroiditis was 10.5/100 PY.

269 ar involvement at baseline, the incidence of necrotizing retinochoroiditis was 6.4/100 PY.

270 Incident necrotizing retinochoroiditis was more common among thos

271 ut clinically apparent retinal necrosis] and necrotizing retinochoroiditis) at initial examination (b

272 inal whitening), without clinically apparent necrotizing retinochoroiditis.

273 ith focal retinal whitening only and 13 with necrotizing retinochoroiditis.

274 We present a case of hepatosplenic necrotizing sarcoid granulomatosis, a variant form of "c

275 esent as highly invasive infections, such as necrotizing skin and soft tissue infections (NSSTI).

276 erans is known to cause Buruli ulcer (BU), a necrotizing skin disease leading to extensive cutaneous

277 Buruli ulcer (BU) is a necrotizing skin disease most prevalent among West Afric

278 virulence contribution in a murine model of necrotizing skin infection is largely driven by its abil

279 lence factor that enables CA-MRSA to produce necrotizing skin infections by allowing the bacteria to

280 isystem autoimmune diseases characterized by necrotizing small- to medium-vessel vasculitis and the p

281 ing Fasciitis (LRINEC) score in diagnosis of necrotizing soft tissue infection (NSTI) in adults with

282 Necrotizing soft tissue infections (NSTIs) caused by gro

283 ococcus (GAS) patients, including sepsis and necrotizing soft tissue infections (NSTIs).

284 IG) from patients with group A Streptococcus necrotizing soft tissue infections demonstrated a negati

285 Necrotizing soft-tissue infection-associated in-hospital

286 Necrotizing soft-tissue infections (NSTI) have high morb

287 ic streptococci are frequently implicated in necrotizing soft-tissue infections (NSTIs).

288 to more severe forms of infection, including necrotizing stromal keratitis and herpes simplex encepha

289 ity and utility of this approach by rescuing necrotizing tissues and whole limbs using two murine mod

290 erium tuberculosis secretes the tuberculosis necrotizing toxin (TNT) into the cytosol of infected mac

291 n the CNS as well as clear histopathology of necrotizing vasculitis and hemorrhage in the brain.

292 ic infectious trigger of paralyzing systemic necrotizing vasculitis most severely affecting skeletal

293 Polyarteritis nodosa is a form of necrotizing vasculitis of small and medium-sized arterie

294 et vessel wall, which may participate in the necrotizing vasculitis of the kidney during this disease

295 atosis with polyangiitis (GPA) is a systemic necrotizing vasculitis that is associated with granuloma

296 Polyarteritis nodosa is a systemic necrotizing vasculitis with a pathogenesis that is poorl

297 localized in typical PDNS lesions, including necrotizing vasculitis, glomerulonephritis, granulomatou

298 vels associated with development of systemic necrotizing vasculitis.

299 ctures and evidence of glandular atrophy and necrotizing vasculitis.

300 interact with the endothelium, resulting in necrotizing vasculitis.