ライフサイエンスコーパス: necrotizing

1 Necrotizing and crescentic GN (NCGN) with a paucity of g

2 s discussed in thrombotic microangiopathies, necrotizing and crescentic GN, acute tubular necrosis, a

3 ght patients with active, noninfectious, non-necrotizing anterior scleritis with a scleral inflammato

4 ve participants with active, autoimmune, non-necrotizing anterior scleritis with scleral inflammatory

5 icacy of gevokizumab in the treatment of non-necrotizing anterior scleritis.

6 s, including invasive aspergillosis, chronic necrotizing aspergillosis, fungal colonization, and alle

7 Necrotizing autoimmune myopathy (NAM) is characterized p

8 including rhabdomyolysis and statin-induced necrotizing autoimmune myopathy (SINAM), are rare.

9 Necrotizing autoimmune myopathy was idiopathic in half o

10 a neonate who presented with hydrocephalus, necrotizing cellulitis, systemic inflammation, and respi

11 is central to ANCA-associated vasculitis and necrotizing crescentic glomerulonephritis (NCGN).

12 gan, including the kidneys, where they cause necrotizing crescentic glomerulonephritis.

13 pothesis that endothelial NF-kappaB mediates necrotizing crescentic GN (NCGN) and provides a specific

14 ANCA-associated vasculitis features necrotizing crescentic GN (NCGN), and ANCA-activated neu

15 CA-activated phagocytes cause vasculitis and necrotizing crescentic GN (NCGN).

16 With the emergence of necrotizing/crescentic glomerulonephritis, approximately

17 tional immunizations were needed to induce a necrotizing/crescentic glomerulonephritis.

18 xperimental autoimmune glomerulonephritis or necrotizing/crescentic glomerulonephritis.

19 s like haemorrhages, diffuse cerebral edema, necrotizing encephalitis and decreased bone size (Alizar

20 we show that Parl ablation in mouse causes a necrotizing encephalomyelopathy similar to Leigh syndrom

21 liver function and Leigh syndrome (subacute necrotizing encephalomyelopathy) seen in association wit

22 ific acute encephalopathy syndromes (4 acute necrotizing encephalopathy, 1 acute infantile encephalop

23 c infarct, hemorrhage, and acute hemorrhagic necrotizing encephalopathy.

24 (17.5% [95% CI, 16.5%-18.6%]), and death or necrotizing enterocolitis (19.3% [95% CI, 18.1%-20.4%]).

25 there were increases in deaths attributed to necrotizing enterocolitis (30 [95% CI, 27 to 34] vs. 23

26 urvivors (23.3%, 19.1%, and 11.7%), death or necrotizing enterocolitis (48.1%, 37.1%, and 32.5%), and

27 in mortality (AOR, 0.98; 95% CI, 0.70-1.37), necrotizing enterocolitis (AOR, 0.88; 95% CI, 0.65-1.20)

28 utcomes included all-cause mortality, severe necrotizing enterocolitis (NEC) (Bell stage II or more),

29 Necrotizing enterocolitis (NEC) affects up to 10% of pre

30 LNT were shown to protect neonatal rats from necrotizing enterocolitis (NEC) and are good therapeutic

31 t gut microbiota alterations associated with necrotizing enterocolitis (NEC) and feeding intolerance

32 d blood cell (RBC) transfusion and anemia to necrotizing enterocolitis (NEC) are conflicting.

33 Necrotizing enterocolitis (NEC) continues to be a major

34 Necrotizing enterocolitis (NEC) has long remained a sign

35 ant manifestation of severe diseases such as necrotizing enterocolitis (NEC) in neonates or bowel wal

36 Necrotizing enterocolitis (NEC) is a devastating disease

37 Necrotizing enterocolitis (NEC) is a devastating disease

38 Necrotizing enterocolitis (NEC) is a devastating gastroi

39 Necrotizing enterocolitis (NEC) is a devastating gastroi

40 Necrotizing enterocolitis (NEC) is a devastating inflamm

41 Necrotizing enterocolitis (NEC) is a devastating intesti

42 Necrotizing enterocolitis (NEC) is a disease of neonatal

43 Necrotizing enterocolitis (NEC) is a major cause of morb

44 Necrotizing enterocolitis (NEC) is a major cause of neon

45 Necrotizing enterocolitis (NEC) is an idiopathic, inflam

46 Necrotizing enterocolitis (NEC) is an inflammatory bowel

47 Necrotizing enterocolitis (NEC) is an inflammatory disea

48 Necrotizing enterocolitis (NEC) is one of the most serio

49 Necrotizing enterocolitis (NEC) is the most common and s

50 Necrotizing enterocolitis (NEC) is the most common gastr

51 The pathophysiology of necrotizing enterocolitis (NEC) remains poorly understoo

52 r is the consequence of intestinal injury in necrotizing enterocolitis (NEC) remains unknown.

53 t of lung disease in the setting of neonatal necrotizing enterocolitis (NEC), a life-threatening gast

54 ure and role of the intestinal leukocytes in necrotizing enterocolitis (NEC), a severe disease affect

55 y result in antibiotic resistance, fungemia, necrotizing enterocolitis (NEC), and mortality.

56 y infections, otitis media, gastroenteritis, necrotizing enterocolitis (NEC), and sudden infant death

57 lk feeding is associated with lower rates of necrotizing enterocolitis (NEC), but an understanding of

58 the widespread use of plain films to detect necrotizing enterocolitis (NEC), it is considered a time

59 reast milk (HBM) attenuates the incidence of necrotizing enterocolitis (NEC), which remains a leading

60 possible pregnancy-related risk factors for necrotizing enterocolitis (NEC)-associated deaths during

61 is supposedly associated with development of necrotizing enterocolitis (NEC).

62 bile acids (BAs) may mediate development of necrotizing enterocolitis (NEC).

63 diseases characterized by ISC loss including necrotizing enterocolitis (NEC).

64 riate analysis, SL mortality predictors were necrotizing enterocolitis (NEC; surgical odds ratio, 5.9

65 pathy of prematurity (stage 3 or higher), or necrotizing enterocolitis (stages 2-3).

66 ve been implicated as a pathogenic factor in necrotizing enterocolitis and inflammatory bowel disease

67 bowel diseases (controls) and 8 infants with necrotizing enterocolitis and measured levels of S100A8

68 premature newborns has been shown to prevent necrotizing enterocolitis and reduce all-cause mortality

69 ts, where the virus has been associated with necrotizing enterocolitis and severe and persistent diar

70 Notably, ELGAN/ELBW infants developing necrotizing enterocolitis before day 14 had higher expre

71 Infants with necrotizing enterocolitis born into midlevel hospitals (

72 infants is associated with a reduced risk of necrotizing enterocolitis but an increased risk of late-

73 ptation to small-bowel resection (SBR) after necrotizing enterocolitis expands absorptive surface are

74 Here we show that necrotizing enterocolitis in neonate mice is accompanied

75 given enterally may reduce the incidence of necrotizing enterocolitis in preterm infants.

76 diabetes, nonalcoholic fatty liver disease, necrotizing enterocolitis in very low birth weight infan

77 Late-onset invasive infection or necrotizing enterocolitis occurred in 32% of infants (19

78 Necrotizing enterocolitis occurred in 70 of 1394 infants

79 Necrotizing enterocolitis occurred less frequently in th

80 Necrotizing enterocolitis occurred more frequently in in

81 CI, 0.58-0.78) and the combined outcomes of necrotizing enterocolitis or death and severe intraventr

82 r retinopathy of prematurity and surgery for necrotizing enterocolitis or spontaneous intestinal perf

83 Interestingly, we find that necrotizing enterocolitis patients also exhibit decrease

84 Infants with necrotizing enterocolitis represent a high-risk subgroup

85 he liberal vs restrictive thresholds groups, necrotizing enterocolitis requiring surgical interventio

86 ere intraventricular hemorrhage and death or necrotizing enterocolitis was lowest among infants born

87 of the study, just 28.6% of the infants with necrotizing enterocolitis were born into high-level, hig

88 nary dysplasia, severe cerebral lesions, and necrotizing enterocolitis).

89 2, periventricular leukomalacia >grade 1, or necrotizing enterocolitis).

90 with LOS and 164 unaffected controls (no LOS/necrotizing enterocolitis).

91 prolonged the survival of newborn mice with necrotizing enterocolitis, a severe pathology in preterm

92 sed sample of 30 566 VLBW infants, 1879 with necrotizing enterocolitis, according to the level of car

93 dities before PDA closure, including sepsis, necrotizing enterocolitis, and a dependence on mechanica

94 e, confirmed or suspected late-onset sepsis, necrotizing enterocolitis, and cerebral palsy.

95 aturity, severe intraventricular hemorrhage, necrotizing enterocolitis, and chronic lung disease amon

96 s, hearing loss, bronchopulmonary dysplasia, necrotizing enterocolitis, and severe retinopathy of pre

97 om BEC on premature infants with and without necrotizing enterocolitis, and successfully provided a t

98 iventricular or intraventricular hemorrhage, necrotizing enterocolitis, aspiration, retinopathy of pr

99 rematurity, intracranial hemorrhage, sepsis, necrotizing enterocolitis, bronchopulmonary dysplasia, o

100 he following morbidities: late-onset sepsis, necrotizing enterocolitis, bronchopulmonary dysplasia, p

101 Necrotizing enterocolitis, defined as Bell stage 2 or gr

102 who survived more than 12 hours, were severe necrotizing enterocolitis, infection, bronchopulmonary d

103 Infections and necrotizing enterocolitis, major causes of mortality and

104 hemorrhage, bronchopulmonary dysplasia, and necrotizing enterocolitis, most therapeutic approaches h

105 nography, severe retinopathy of prematurity, necrotizing enterocolitis, or late-onset sepsis) by 36 w

106 of serious infection (sepsis or meningitis), necrotizing enterocolitis, or mortality during the first

107 ystic periventricular leukomalacia, surgical necrotizing enterocolitis, or stage 3 or greater retinop

108 de 3-4, cystic periventricular leukomalacia, necrotizing enterocolitis, retinopathy of prematurity st

109 ites of death or bronchopulmonary dysplasia, necrotizing enterocolitis, retinopathy of prematurity, a

110 It also decreases the prevalence of necrotizing enterocolitis, sepsis, and intraventricular

111 PN significantly affects risk of mortality, necrotizing enterocolitis, sepsis, chronic lung disease,

112 onary hemorrhage but not with differences in necrotizing enterocolitis, severe bronchopulmonary dyspl

113 No differences in rates of necrotizing enterocolitis, severe bronchopulmonary dyspl

114 etinopathy of prematurity requiring surgery, necrotizing enterocolitis, spontaneous intestinal perfor

115 nical complications of prematurity including necrotizing enterocolitis, systemic infections and bronc

116 The frequencies of air leaks and necrotizing enterocolitis, the duration of respiratory s

117 ria in premature infants are associated with necrotizing enterocolitis, underscoring the critical rol

118 ntral nervous system injury decreased, while necrotizing enterocolitis-related deaths increased.

119 d incidence of neonatal bacterial sepsis and necrotizing enterocolitis.

120 h postnatal intestinal pathologies including necrotizing enterocolitis.

121 re intraventricular hemorrhage, and death or necrotizing enterocolitis.

122 ities in infants who did and did not develop necrotizing enterocolitis.

123 rain was common to all infants who developed necrotizing enterocolitis.

124 on, a process that may lead to diseases like necrotizing enterocolitis.

125 am, urinary tract, or cerebrospinal fluid or necrotizing enterocolitis.

126 ch were profoundly depleted in newborns with necrotizing enterocolitis.

127 ther this practice reduces the prevalence of necrotizing enterocolitis.

128 so a trend towards a diminished incidence of necrotizing enterocolitis.

129 achieve the rate from the best quartile for necrotizing enterocolitis.

130 Cytotoxic necrotizing factor 1 (CNF1) and hemolysin (Hly) are toxi

131 Cytotoxic necrotizing factor 1 (CNF1) is a key UPEC toxin; however

132 ore chronic comorbidities than patients with necrotizing fasciitis (20 [87.0%] vs 17 [54.8%]; P = .02

133 or patients with cellulitis vs patients with necrotizing fasciitis (3 [2-5] vs 5 [3-11]; P = .01), wh

134 slightly younger and had a higher degree of necrotizing fasciitis (56% vs 14%).

135 known as strep throat) to severely invasive necrotizing fasciitis (also known as the flesh-eating sy

136 , imaging, and Laboratory Risk Indicator for Necrotizing Fasciitis (LRINEC) score in diagnosis of nec

137 n = 23; mean [SD] age, 57.2 [17.7] years) or necrotizing fasciitis (n = 31; mean [SD] age, 54.3 [13.5

138 Necrotizing fasciitis (NF) caused by flesh-eating bacter

139 Shock frequently complicates necrotizing fasciitis (NF) caused by group A Streptococc

140 An earlier report described a human case of necrotizing fasciitis (NF) caused by mixed infection wit

141 Necrotizing fasciitis (NF) is a destructive skin and sof

142 erile site or from a wound in a patient with necrotizing fasciitis (NF) or streptococcal toxic shock

143 hort study describes mortality predictors of necrotizing fasciitis (NF).

144 than the carriage strain in a mouse model of necrotizing fasciitis and had enhanced growth ex vivo in

145 Necrotizing fasciitis and myositis are devastating infec

146 Necrotizing fasciitis and myositis caused by group A str

147 sociated with invasive infections, including necrotizing fasciitis and toxic shock syndrome.

148 itis and impetigo to severe outcomes such as necrotizing fasciitis and toxic shock syndrome.

149 mic invasive infections worldwide, including necrotizing fasciitis and toxic shock syndrome.

150 p throat") to invasive conditions, including necrotizing fasciitis and toxic shock syndrome.

151 treptococcal toxic shock syndrome (STSS) and necrotizing fasciitis are the 2 most severe invasive man

152 the reason for ICU admission in 23 patients, necrotizing fasciitis in 31 patients, and other diagnose

153 this report, we present a case of multifocal necrotizing fasciitis in a healthy adult patient, second

154 Current therapeutic regimens for necrotizing fasciitis include surgical debridement and t

155 GAS upregulates the AdcR regulon during necrotizing fasciitis infection in WT mice but not in S1

156 Periocular necrotizing fasciitis is a rare but potentially devastat

157 Necrotizing fasciitis is often confused for cellulitis a

158 As seen in these 5 cases, periocular necrotizing fasciitis may cause severe visual loss more

159 ecal diversion surgery, 2 patients developed necrotizing fasciitis of a lower extremity that required

160 terile site or from a wound in patients with necrotizing fasciitis or streptococcal toxic shock syndr

161 k thus aimed to address whether CLI improves necrotizing fasciitis outcome by modulating virulence fa

162 a pathogen that causes both invasive (e.g., necrotizing fasciitis) and noninvasive (e.g., pharyngiti

163 e.g. pharyngitis) to severely invasive (e.g. necrotizing fasciitis).

164 tis, otitis, etc.) to life-threatening (e.g. necrotizing fasciitis).

165 , amputations, acute kidney injury, perineal necrotizing fasciitis, and hyperkalemia.

166 GAS), the causative agent of pharyngitis and necrotizing fasciitis, secretes the potent cysteine prot

167 fection (streptococcal toxic shock syndrome, necrotizing fasciitis, septic shock, or GAS cellulitis w

168 h cellulitis and patients with patients with necrotizing fasciitis, Staphylococcus aureus (10 [43.5%]

169 itically ill on admission than patients with necrotizing fasciitis, they have more chronic comorbidit

170 During the invasive infection necrotizing fasciitis, this is in part due to the GAS pr

171 istered as soon as possible to patients with necrotizing fasciitis, while our in vitro studies emphas

172 the virulence phenotype in a mouse model of necrotizing fasciitis.

173 life-threatening invasive infections such as necrotizing fasciitis.

174 d to be less severely ill than patients with necrotizing fasciitis.

175 sed virulence in a nonhuman primate model of necrotizing fasciitis.

176 and increased virulence in a mouse model of necrotizing fasciitis.

177 uding streptococcal toxic shock syndrome and necrotizing fasciitis.

178 cause illnesses ranging from pharyngitis to necrotizing fasciitis.

179 rom a nonhuman primate experimental model of necrotizing fasciitis.

180 infections such as toxic shock syndrome and necrotizing fasciitis.

181 type virulence phenotype in a mouse model of necrotizing fasciitis.

182 e-threatening invasive infections, including necrotizing fasciitis.

183 irulence in animal models of pharyngitis and necrotizing fasciitis.

184 red from nonhuman primates with experimental necrotizing fasciitis.

185 od and reduced virulence in a mouse model of necrotizing fasciitis.

186 d we review literature on this rare cause of necrotizing fasciitis.

187 tions to life-threatening infections such as necrotizing fasciitis.

188 n nonhuman primate models of pharyngitis and necrotizing fasciitis.

189 Streptococcus anginosus constellatus causing necrotizing fasciitis.

190 litis and to compare them with patients with necrotizing fasciitis.

191 inflammation in the central nervous system; necrotizing focal myelitis in the cervical spinal cord;

192 en in the pituitary can cause an aggressive (necrotizing) form of hypophysitis through type IV (T-cel

193 Focal and segmental necrotizing glomerular lesions with crescents, mimicking

194 Pauci-immune focal necrotizing GN (piFNGN) is usually associated with ANCAs

195 Using an established murine model of focal necrotizing GN mediated by autoimmunity to MPO (autoimmu

196 xic effect of NET-related histone release in necrotizing GN.

197 tion on chest imaging, and poorly formed non-necrotizing granulomas on pathology.

198 hil cytoplasmic antibodies (ANCA)-associated necrotizing granulomatous vasculitis that affects small

199 (PP) or RSA59 (P) yielded similar degrees of necrotizing hepatitis and meningitis, but only RSA59 (PP

200 al mortality in conjunction with a fulminant necrotizing hepatitis.

201 BS) in 10-d-old mouse pups produces an acute necrotizing ileocolitis resembling human NEC within 24 h

202 mITT necrotizing infection clinical composite endpoint succes

203 g a previously validated composite endpoint, necrotizing infection clinical composite endpoint, defin

204 , a rare urologic emergency characterized by necrotizing infection of the external genitalia, perineu

205 istopathological examination revealed severe necrotizing inflammation in various organs, most promine

206 nephritis, and/or severe systemic multiorgan necrotizing inflammation.

207 s and calcifications, brain infarctions with necrotizing inflammatory response, infiltrate, retinal i

208 Hypoxic necrotizing lesions rendered moxifloxacin less active.

209 arterial smooth muscle cells and deposits in necrotizing lesions.

210 microbial defence uniformly develop hypoxic necrotizing lung lesions, widely observed in human TB.

211 It has a lethal canine equivalent: necrotizing meningoencephalitis.

212 onnected left pancreatic remnant after acute necrotizing mid-body necrosis, definitive surgical manag

213 ith autoimmune myopathy have a predominantly necrotizing muscle biopsy with minimal lymphocytic infil

214 s the spectrum of diseases associated with a necrotizing muscle biopsy.

215 s puerperal sepsis, neonatal infections, and necrotizing myometritis.

216 Immune-mediated necrotizing myopathies (IMNM) may be associated with eit

217 One patient with severe necrotizing myopathy died.

218 t evidence now suggests that immune-mediated necrotizing myopathy is not one disease, but can be divi

219 A decade ago, immune-mediated necrotizing myopathy was recognized as a distinct form o

220 ients with dermatomyositis, polymyositis, or necrotizing myopathy, and 0/20 (0%) age-matched healthy

221 more than 25% of the GAS genes required for necrotizing myositis encode known or putative transporte

222 significantly (P < 0.05) impaired in causing necrotizing myositis in NHPs.

223 ns that identified GAS genes contributing to necrotizing myositis in nonhuman primates (NHP), a clini

224 d SLO toxins to virulence in mouse models of necrotizing myositis, bacteremia, and skin and soft tiss

225 ernatively, statins can induce an autoimmune necrotizing myositis.

226 rred a fitness advantage in a mouse model of necrotizing myositis.

227 ins are equally impaired in ability to cause necrotizing myositis.

228 sues became progressively more fibrinous and necrotizing over time.

229 Biliary stricture in necrotizing pancreatitis (NP) has not been systematicall

230 that the development of colon involvement in necrotizing pancreatitis (NP) negatively affects morbidi

231 Biliary stricture occurs frequently after necrotizing pancreatitis and is associated with splanchn

232 ative approaches to the debridement of acute necrotizing pancreatitis are preferred to open surgical

233 At least 30% of patients with infected necrotizing pancreatitis are successfully treated with c

234 Between 1997 and 2013, patients with necrotizing pancreatitis at the Liverpool Pancreas Cente

235 ins a role in the modern management of acute necrotizing pancreatitis in cases not amenable to less i

236 n vivo in the course of taurocholate-induced necrotizing pancreatitis in rats and in vitro in rat pan

237 aurocholate into the pancreatic duct induced necrotizing pancreatitis in the head of pancreas and lig

238 Infected necrotizing pancreatitis is a highly morbid disease with

239 Colon involvement in necrotizing pancreatitis is associated with substantial

240 SUMMARY/BACKGROUND DATA: Necrotizing pancreatitis is characterized by a profound

241 Colon involvement in necrotizing pancreatitis is common; clinical deteriorati

242 ticipants, we found the step-up approach for necrotizing pancreatitis to be superior to open necrosec

243 l), a surgical step-up approach for infected necrotizing pancreatitis was found to reduce the composi

244 Acute necrotizing pancreatitis was induced by i.p. administrat

245 atients with confirmed or suspected infected necrotizing pancreatitis who required intervention from

246 ndoscopic transluminal approach for infected necrotizing pancreatitis, compared with minimally invasi

247 mild pancreatitis, 20% develop severe and/or necrotizing pancreatitis, requiring advanced medical and

248 ea of intra-parenchymal necrosis, indicating necrotizing pancreatitis.

249 g catheter drainage for (suspected) infected necrotizing pancreatitis.

250 for success of catheter drainage in infected necrotizing pancreatitis.

251 higher mortality when compared with sterile necrotizing pancreatitis.

252 scopic approaches for patients with infected necrotizing pancreatitis.

253 ions, including osteomyelitis, endocarditis, necrotizing pneumonia and sepsis(1).

254 lveolar capillary destruction in hemorrhagic/necrotizing pneumonia caused by CA-MRSA and offer novel

255 revent lethal challenge in a rabbit model of necrotizing pneumonia caused by the USA300 community-ass

256 coccus aureus (MRSA) causes life-threatening necrotizing pneumonia in children.

257 ive sudden, massive loss of lung tissue from necrotizing pneumonia or acute respiratory distress synd

258 ) infection can quickly develop into severe, necrotizing pneumonia, causing over 50% mortality despit

259 th cystic fibrosis (CF) and can cause severe necrotizing pneumonia, which is often fatal.

260 USA300 is the predominant strain that causes necrotizing pneumonia.

261 ng inflammation and, in some cases, a lethal necrotizing pneumonia.

262 rgic bronchopulmonary aspergillosis, chronic necrotizing pulmonary aspergillosis, or invasive aspergi

263 ime of conversion and progressed to a severe necrotizing rejection early despite an unaltered baselin

264 inal detachment repair, endophthalmitis, and necrotizing retinitis were excluded, as were those with

265 irst IgM test had a greater risk of incident necrotizing retinochoroiditis (hazard ratio = 4.47, 95%

266 The increased risk of late necrotizing retinochoroiditis associated with isolated f

267 al manifestation of T gondii infection, with necrotizing retinochoroiditis occurring months or years

268 The incidence of recurrent necrotizing retinochoroiditis was 10.5/100 PY.

269 ar involvement at baseline, the incidence of necrotizing retinochoroiditis was 6.4/100 PY.

270 Incident necrotizing retinochoroiditis was more common among thos

271 ut clinically apparent retinal necrosis] and necrotizing retinochoroiditis) at initial examination (b

272 inal whitening), without clinically apparent necrotizing retinochoroiditis.

273 ith focal retinal whitening only and 13 with necrotizing retinochoroiditis.

274 We present a case of hepatosplenic necrotizing sarcoid granulomatosis, a variant form of "c

275 esent as highly invasive infections, such as necrotizing skin and soft tissue infections (NSSTI).

276 erans is known to cause Buruli ulcer (BU), a necrotizing skin disease leading to extensive cutaneous

277 Buruli ulcer (BU) is a necrotizing skin disease most prevalent among West Afric

278 virulence contribution in a murine model of necrotizing skin infection is largely driven by its abil

279 lence factor that enables CA-MRSA to produce necrotizing skin infections by allowing the bacteria to

280 isystem autoimmune diseases characterized by necrotizing small- to medium-vessel vasculitis and the p

281 ing Fasciitis (LRINEC) score in diagnosis of necrotizing soft tissue infection (NSTI) in adults with

282 Necrotizing soft tissue infections (NSTIs) caused by gro

283 ococcus (GAS) patients, including sepsis and necrotizing soft tissue infections (NSTIs).

284 IG) from patients with group A Streptococcus necrotizing soft tissue infections demonstrated a negati

285 Necrotizing soft-tissue infection-associated in-hospital

286 Necrotizing soft-tissue infections (NSTI) have high morb

287 ic streptococci are frequently implicated in necrotizing soft-tissue infections (NSTIs).

288 to more severe forms of infection, including necrotizing stromal keratitis and herpes simplex encepha

289 ity and utility of this approach by rescuing necrotizing tissues and whole limbs using two murine mod

290 erium tuberculosis secretes the tuberculosis necrotizing toxin (TNT) into the cytosol of infected mac

291 n the CNS as well as clear histopathology of necrotizing vasculitis and hemorrhage in the brain.

292 ic infectious trigger of paralyzing systemic necrotizing vasculitis most severely affecting skeletal

293 Polyarteritis nodosa is a form of necrotizing vasculitis of small and medium-sized arterie

294 et vessel wall, which may participate in the necrotizing vasculitis of the kidney during this disease

295 atosis with polyangiitis (GPA) is a systemic necrotizing vasculitis that is associated with granuloma

296 Polyarteritis nodosa is a systemic necrotizing vasculitis with a pathogenesis that is poorl

297 localized in typical PDNS lesions, including necrotizing vasculitis, glomerulonephritis, granulomatou

298 vels associated with development of systemic necrotizing vasculitis.

299 ctures and evidence of glandular atrophy and necrotizing vasculitis.

300 interact with the endothelium, resulting in necrotizing vasculitis.