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1 s observed in only 1 of 187 patients without necrotizing myopathy.
2 entify novel autoantibodies in patients with necrotizing myopathy.
3 rodiagnostic findings were consistent with a necrotizing myopathy.
4 ntly identified statin-associated autoimmune-necrotizing myopathy.
5 ents with dermatomyositis and the autoimmune necrotizing myopathies.
6 ated with dermatomyositis and the autoimmune necrotizing myopathies.
7  one patient with refractory immune-mediated necrotizing myopathy and two patients with diffuse cutan
8 ients with statin-associated immune-mediated necrotizing myopathy and, less commonly, in statin-unexp
9 ients with dermatomyositis, polymyositis, or necrotizing myopathy, and 0/20 (0%) age-matched healthy
10  anti-cadherin 1 antibody in immune-mediated necrotizing myopathy, antisynthetase syndrome, and contr
11                 In a subgroup, an autoimmune necrotizing myopathy develops that persists after discon
12                      One patient with severe necrotizing myopathy died.
13 ren with profiles from children with a known necrotizing myopathy (Duchenne muscular dystrophy), as w
14            The underlying immune response in necrotizing myopathy has not yet been addressed in detai
15                              Immune-mediated necrotizing myopathies (IMNM) may be associated with eit
16                              Immune-mediated necrotizing myopathy (IMNM) is considered one of the idi
17 f the first subject with the immune-mediated necrotizing myopathy (IMNM) subtype of IIM who received
18 clusion body myositis (IBM), immune-mediated necrotizing myopathy (IMNM), and ALS, we found that TDP-
19 in use is associated with an immune-mediated necrotizing myopathy (IMNM), with autoantibodies that re
20 s), antisynthetase syndrome, immune-mediated necrotizing myopathy, inclusion body myositis, polymyosi
21 s), antisynthetase syndrome, immune-mediated necrotizing myopathy, inclusion body myositis, polymyosi
22                                              Necrotizing Myopathy is a poorly studied subacute myopat
23 t evidence now suggests that immune-mediated necrotizing myopathy is not one disease, but can be divi
24                   However, the etiology of a necrotizing myopathy is often obscure, and the question
25 rs,52% Dermatomyositis, 39% Polymyositis, 9% Necrotizing Myopathy) participated.
26                Biopsies from immune-mediated necrotizing myopathy patients, who have the lowest level
27 antibodies found in patients with autoimmune necrotizing myopathies recognize signal recognition part
28  MB in juvenile DM that focuses on extent of necrotizing myopathy, severity of vasculopathy, and feat
29                   The heterogeneous group of necrotizing myopathies shows a varying amount of necroti
30                A decade ago, immune-mediated necrotizing myopathy was recognized as a distinct form o
31 ed immune response in most of the autoimmune necrotizing myopathies, which may guide therapeutic opti
32 cificity defines a subgroup of patients with necrotizing myopathy who previously were considered to b
33  that statins may trigger an immune-mediated necrotizing myopathy with many features of polymyositis.
34 in dermatomyositis and in the rare disorder, necrotizing myopathy with pipestem capillaries.