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1 gastrointestinal tract perforation, 21 acute necrotizing pancreatitis).
2  higher mortality when compared with sterile necrotizing pancreatitis.
3 scopic approaches for patients with infected necrotizing pancreatitis.
4 ea of intra-parenchymal necrosis, indicating necrotizing pancreatitis.
5 g catheter drainage for (suspected) infected necrotizing pancreatitis.
6 for success of catheter drainage in infected necrotizing pancreatitis.
7 lure; this makes the pancreas susceptible to necrotizing pancreatitis.
8  The injection of cerulein resulted in acute necrotizing pancreatitis.
9 imicrobial use in patients with severe acute necrotizing pancreatitis.
10 ys (range, 20-300 days) after onset of acute necrotizing pancreatitis.
11 nd distant organ injury in a murine model of necrotizing pancreatitis.
12 terial or antifungal agents in patients with necrotizing pancreatitis.
13 lied to the entire spectrum of patients with necrotizing pancreatitis.
14 nduced multisystem organ failure or to acute necrotizing pancreatitis.
15 1, are central events in the pathogenesis of necrotizing pancreatitis.
16 s in the pancreas and at 12 hours in lung in necrotizing pancreatitis.
17 entral event in the progression to fulminant necrotizing pancreatitis.
18 ungs from rats with mild edematous or severe necrotizing pancreatitis.
19 e occurred in only one half of patients with necrotizing pancreatitis.
20 o detect the presence of sterile or infected necrotizing pancreatitis.
21 necrosis and assist medical therapy in acute necrotizing pancreatitis.
22 ajor complications in patients with infected necrotizing pancreatitis.
23 d to complications in patients with infected necrotizing pancreatitis.
24 d patients with clinically severe, confirmed necrotizing pancreatitis: 50 received meropenem and 50 r
25 ed successfully to manage most patients with necrotizing pancreatitis, although some will eventually
26               Patients had signs of infected necrotizing pancreatitis and an indication for intervent
27    Biliary stricture occurs frequently after necrotizing pancreatitis and is associated with splanchn
28  antibiotics can reduce mortality from acute necrotizing pancreatitis (ANP).
29 s responsible for the progression of mild to necrotizing pancreatitis are poorly understood.
30 ative approaches to the debridement of acute necrotizing pancreatitis are preferred to open surgical
31       At least 30% of patients with infected necrotizing pancreatitis are successfully treated with c
32         Between 1997 and 2013, patients with necrotizing pancreatitis at the Liverpool Pancreas Cente
33 who underwent uniform surgical treatment for necrotizing pancreatitis at the Massachusetts General Ho
34 l step-up approach in patients with infected necrotizing pancreatitis based on favorable short-term o
35 into interstitial edematous pancreatitis and necrotizing pancreatitis, (c) distinguish an early phase
36 ndoscopic transluminal approach for infected necrotizing pancreatitis, compared with minimally invasi
37               In patients with sterile acute necrotizing pancreatitis, conservative nonsurgical manag
38              Mortality after debridement for necrotizing pancreatitis continues to be inordinately hi
39    We identified a total of 89 patients with necrotizing pancreatitis, diagnosed by computed tomograp
40 s is a well-known complication of chronic or necrotizing pancreatitis due to proteolytic enzymatic di
41                    In patients with infected necrotizing pancreatitis, endoscopic necrosectomy reduce
42 otics should be given only in the setting of necrotizing pancreatitis, especially if patients are rec
43 is study was to examine a rat model of acute necrotizing pancreatitis for changes in NGF expression.
44                                Patients with necrotizing pancreatitis from May 1992 to January 1996 w
45 ins a role in the modern management of acute necrotizing pancreatitis in cases not amenable to less i
46 n vivo in the course of taurocholate-induced necrotizing pancreatitis in rats and in vitro in rat pan
47 athologic changes during the course of acute necrotizing pancreatitis in rats induced by the intraper
48 aurocholate into the pancreatic duct induced necrotizing pancreatitis in the head of pancreas and lig
49 t syndrome (DPDS) is a complication of acute necrotizing pancreatitis in the neck and body of the pan
50 e step-up approach for treatment of infected necrotizing pancreatitis in the real life displays a cli
51                     The development of acute necrotizing pancreatitis in this model leads to a signif
52 ority trial involving patients with infected necrotizing pancreatitis, in which we compared immediate
53                                     Infected necrotizing pancreatitis is a highly morbid disease with
54                                     Infected necrotizing pancreatitis is a potentially lethal disease
55                         Colon involvement in necrotizing pancreatitis is associated with substantial
56                     SUMMARY/BACKGROUND DATA: Necrotizing pancreatitis is characterized by a profound
57                         Colon involvement in necrotizing pancreatitis is common; clinical deteriorati
58  walled-off pancreatic necrosis (WOPN) after necrotizing pancreatitis is limited.
59                     In patients with severe, necrotizing pancreatitis, it is common to administer ear
60 everity of illness, studies of patients with necrotizing pancreatitis must stratify for organ failure
61                         Biliary stricture in necrotizing pancreatitis (NP) has not been systematicall
62 that the development of colon involvement in necrotizing pancreatitis (NP) negatively affects morbidi
63                                       Severe necrotizing pancreatitis occurs in young female mice fed
64  course of the disease for all patients with necrotizing pancreatitis, regardless of the status of in
65                  Most patients with infected necrotizing pancreatitis require necrosectomy.
66 mild pancreatitis, 20% develop severe and/or necrotizing pancreatitis, requiring advanced medical and
67    When operative intervention is needed for necrotizing pancreatitis, this should start with the end
68 ticipants, we found the step-up approach for necrotizing pancreatitis to be superior to open necrosec
69             There were 99 (9%) patients with necrotizing pancreatitis treated, with an overall death
70 ollected from inbred rats after induction of necrotizing pancreatitis; trypsinogen activation peptide
71 8 sterile and 3 infected) after severe acute necrotizing pancreatitis underwent attempted endoscopic
72 l), a surgical step-up approach for infected necrotizing pancreatitis was found to reduce the composi
73                                        Acute necrotizing pancreatitis was induced by i.p. administrat
74                                              Necrotizing pancreatitis was induced in transgenic (-/-)
75 criteria, patients with acute cholangitis or necrotizing pancreatitis were excluded.
76  November 1996, 64 consecutive patients with necrotizing pancreatitis were treated with necrosectomy
77    Retrospective review of 167 patients with necrotizing pancreatitis who required intervention and w
78 atients with confirmed or suspected infected necrotizing pancreatitis who required intervention from
79                          Treatment of severe necrotizing pancreatitis with monoclonal antibodies agai
80              Mice lacking NOS have a severe, necrotizing pancreatitis, with elevated pancreatic enzym
81            A reliable prediction of infected necrotizing pancreatitis would enable an early identific