ライフサイエンスコーパス: negative staining

1 cimens, 13 with C4d-positive and 18 with C4d-negative staining.

2 ith procapsids but had a novel appearance by negative staining.

3 = 0.045), p53 (adjusted HR for positive vs. negative staining, 1.48 [CI, 1.06 to 2.08]; P = 0.022),

4 (adjusted hazard ratio [HR] for positive vs. negative staining, 1.61 [95% CI, 1.01 to 2.57]; P = 0.04

5 ner domain of actin previously determined by negative staining, also at 3.8 nm radius.

6 y identical to that determined previously by negative staining, although at a radius of 3.8 nm, sligh

7 higher risk of graft failure than those with negative staining and a significantly lower median time

8 To address these questions we have used both negative staining and cryo-EM to generate three-dimensio

9 3-D model of the mouse 16 nm AUM particle by negative staining and electron crystallography.

10 ers, however, scattered cytochrome c oxidase-negative staining and electron dense mitochondrial inclu

11 Cross-linking followed by negative staining and electron microscopy suggested a gl

12 Using negative staining and electron microscopy, full-length p

13 n microscopy (and single particle analysis), negative staining and freeze-fracture electron microscop

14 ructural basis of MLCK-actin interactions by negative staining and helical reconstruction.

15 leavage of the BMP-10 dimer model as well as negative staining and transmission electron microscopy (

16 alian heart, and analyzed their structure by negative-staining and electron microscopy.

17 tion of the probes could be monitored by the negative-staining appearance in the fluorescence microsc

18 Using a negative staining approach, maltose phosphorylase, a pho

19 The phage capsids were visualised after negative staining by transmission electron microscopy, a

20 10-20% wider than PolyQKd-33, as measured by negative staining, cryo-electron microscopy, and scannin

21 By negative staining, cryo-EM and scanning transmission EM

22 ick-freeze/deep-etch images and our previous negative staining data indicate that the head domain of

23 associated with responses to nivolumab, but negative staining did not rule out a response.

24 ent ultracentrifugation and visualization by negative staining electromicroscopy demonstrated that th

25 ent ultracentrifugation and visualization by negative staining electron microscopy demonstrated that

26 Negative staining electron microscopy of o-gp140SF162Del

27 fied to near homogeneity, and examination by negative staining electron microscopy revealed large, fl

28 Negative staining electron microscopy revealed that GTD-

29 Negative staining electron microscopy revealed that pept

30 d HMW1B by size exclusion chromatography and negative staining electron microscopy revealed that the

31 Negative staining electron microscopy showed that FhuA i

32 Negative staining electron microscopy shows that the Chl

33 We have used negative staining electron microscopy to study the effec

34 According to negative staining electron microscopy, differential scan

35 Using time-resolved negative staining electron microscopy, we show that the

36 rotease sensitivity, spectrofluorometry, and negative staining electron microscopy.

37 ions by gel filtration, CD spectroscopy, and negative-staining electron microscopy (EM).

38 Furthermore, negative-staining electron microscopy analysis and Alpha

39 ation in the presence and absence of IGF2 by negative-staining electron microscopy and hydroxyl radic

40 The active fraction was visualized by negative-staining electron microscopy as ring-like parti

41 Mutational analysis, together with negative-staining electron microscopy images, showed tha

42 Visualization of the SEC by negative-staining electron microscopy revealed an anchor

43 bdiffraction-resolution light microscopy and negative-staining electron microscopy revealed that fasc

44 end, compatible with the density observed in negative-staining electron microscopy that depended on t

45 etry (HDX MS), native mass spectrometry, and negative-staining electron microscopy to comprehensively

46 Seen by negative-staining electron microscopy, it presents as a

47 d in the presence of lipid and visualized by negative-staining electron microscopy, the purified Kv1.

48 As seen by negative-staining electron microscopy, these lipoprotein

49 Using negative-staining electron microscopy, we sought Haufen

50 Here we report cryo-electron microscopy and negative-staining electron tomography approaches to imag

51 Small-angle X-ray scattering and negative-staining EM revealed a two-lobed shape for VPS3

52 intracellular fluorescent labels as well as negative staining experiments to measure cell-induced sc

53 ; detection of MUC5AC mRNA using RT-PCR; and negative staining for CK-4, M(1) muscarinic receptor, an

54 Multivariate regression analysis identified negative staining for cytoplasmic FANCD2 as the most sig

55 -old female, both with gingival LP, also had negative staining for ER.

56 ained cells surrounded by cells with weak or negative staining for heparanase.

57 inly proprioceptive neurons, had very low or negative staining for hexokinase I.

58 The epithelial sheet showed negative staining for laminin 5 and collagen VII, but in

59 The remaining stroma showed negative staining for laminin 5, positive linear stainin

60 rescence of the skin of the proband revealed negative staining for the integrin alpha6 and markedly r

61 Electron microscopic analysis after negative staining further revealed that actin filaments

62 Following negative staining, images of the soft-landed complexes r

63 tion conditions and also validate the use of negative staining in investigations of muscle thin filam

64 pproximately 100 A wide fibers visualized by negative staining in the electron microscope, the beta-c

65 The validations by both negative staining (NS) and cryo-electron microscopy (cry

66 , which could be used alternatively to UA in negative staining (NS), in en bloc staining, and post se

67 The method is validated by using both negative-staining (NS) and cryo-electron tomography (cry

68 ning and electron microscopic examination in negative staining of aged samples of Abeta alone and Abe

69 Negative staining of capsids revealed small patches of h

70 on thin sections of fixed materials through negative staining of isolated polymers, then on to imagi

71 Negative staining of NGF, NT4, BDNF, and TrkB was noted

72 Bacterial Adherence to Hydrocarbons assays, negative staining of polysaccharides by India ink and Tr

73 Negative staining of released fibrils showed no evidence

74 Negative staining of the proteins revealed particles con

75 tron tomography (IPET) method with optimized negative-staining (OpNS) for direct 3D reconstruction of

76 The Old group primarily had negative staining, or Type I and Type II patterns of amy

77 protein, invasive SCCs exhibited a patchy or negative staining pattern.

78 at this type of glycosylation influences the negative-staining pattern of collagen fibrils.

79 Theoretical negative-staining patterns were created based upon the "

80 mparison of the two techniques suggests that negative staining preserves the structure induced by Ca2

81 form fibrils in vitro with a similar amyloid-negative staining property to those of TDP-43 pathogenic

82 riable degrees of artifacts depending on the negative staining protocol.

83 Electron microscopy with negative staining provides further evidence that SAT has

84 gh negative predictive value suggests that a negative staining result indicates that OSSN is relative

85 AI-1 from these column fractions followed by negative staining revealed 25-nm diameter complexes of a

86 Electron microscopy with negative staining revealed an extended, filamentous CHGA

87 Electron microscopy using negative staining revealed that the addition of ATP indu

88 Negative staining reveals that when Ca2+ binds to the he

89 cated variant of FHA by electron microscopy (negative staining, shadowing and scanning transmission e

90 from skinned smooth muscles and observed by negative staining show crossbridges with a 14.5-nm repea

91 Negative staining showed a mixture of straight and twist

92 ectron microscopy using rotary shadowing and negative staining showed that the type I and II receptor

93 meric filaments by electron microscopy after negative staining showed that, remarkably, EspA filament

94 Analysis of the photosynthetic apparatus by negative staining, spectroscopy, and sodium dodecyl sulf

95 We therefore used a time-resolved negative staining technique to determine the time scale

96 ordered unwinding of the DNA was observed by negative staining that appeared to progress through four

97 ron-microscopy techniques, thin sections and negative staining, that enabled answering major question

98 tissue was not corneal, as evidenced by the negative staining to cornea-specific K12 mRNA and protei

99 h 1:1 stoichiometry, [Yfh1]24.[Isu1]24 Using negative staining transmission EM and single particle an

100 Using negative staining transmission EM and single particle an

101 By immunogold labeling and negative staining we have detected in these complexes, m

102 There was negative staining with Congo red.

103 By contrast, most MMIHS tissue gave negative staining with ISH and variable results with ICC