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1 ivated in retroviral infection but weakly in nematode infection.
2 n autophagy showed reduced susceptibility to nematode infection.
3 ponses of many oxylipins following root-knot nematode infection.
4 of the mechanism by which Rbohs promote cyst nematode infection.
5 ophages were required for host resistance to nematode infection.
6 early-induced oxylipins in tomato roots upon nematode infection.
7 thylated region in the PRSS22 gene linked to nematode infection.
8 es and attenuation of host resistance to the nematode infection.
9  and the insect immune response to parasitic nematode infection.
10 of resident MPhis during a Th2-biased tissue nematode infection.
11 s of intestinal stem cells are unaffected by nematode infection.
12 cal mediator of resistance during intestinal nematode infection.
13 abundance of a set of antioxidant genes upon nematode infection.
14 cytokinin signaling during plant response to nematode infection.
15 food intake to maintain energy stores during nematode infection.
16 omeostasis throughout evolution is parasitic nematode infection.
17 nt and differentiation of murine MMCs during nematode infection.
18 immune-effector molecule in resistance to GI nematode infection.
19 mune defense against Steinernema carpocapsae nematode infection.
20 ype 1 responses and chronic gastrointestinal nematode infection.
21 ntly intraepithelial location of MMCs during nematode infection.
22 ier function of intestinal epithelium during nematode infection.
23 ng Th cell responses during gastrointestinal nematode infection.
24 sophila larvae in the context of Steinernema nematode infection.
25 /c background to investigate the response to nematode infection.
26 in resistant than in susceptible roots after nematode infection.
27 constitutively and in inductive responses to nematode infection.
28 ine is hyper-resistant to both bacterial and nematode infections.
29 ey to viral, bacterial, oomycete, fungal and nematode infections.
30 ng anti-Wolbachia therapies against filarial nematode infections.
31 sms contributed to MPhi proliferation during nematode infections.
32 nses limit, and in some instances eliminate, nematode infections.
33 at limit the parasite burden during filarial nematode infections.
34 ra from patients with filarial or intestinal nematode infections.
35  and secondary ASCs following viral, but not nematode, infection.
36 d that ethylene regulates plant responses to nematode infection, a mechanistic understanding of how e
37               We investigated the effects of nematode infection against obesity and the associated me
38 ate that the protective mechanism of enteric nematode infection against TNBS-induced colitis involves
39 wed that LeMir expression is up-regulated by nematode infection and by wounding.
40  strain-dependent nature of entomopathogenic nematode infection and highlight the positive and negati
41 understanding of the molecular mechanisms of nematode infection and host antinematode processes will
42 es antagonize each other's virulence in both nematode infection and in vitro biofilm models.
43 ytokine in protective immunity to intestinal nematode infection and is believed to enhance Th2 immune
44 essfully protect the host against intestinal nematode infection and suggests that IL-9 can act as a p
45 receptor mutants revealed a decrease in both nematode infection and syncytium size.
46                  Strong KRP6 expression upon nematode infection and the phenotypic resemblance betwee
47                            Furthermore, both nematode infection and transient expression of Gr-VAP1 i
48 mplications for approaches to the control of nematode infections and the disease that they cause.
49 t endosymbionts in the response to parasitic nematode infections, and the influence of nematode endos
50                                              Nematode infections are an important economic constraint
51                        Gastrointestinal (GI) nematode infections are an important public health and e
52 vation of macrophages is also analyzed, with nematode infection as prototypic disease.
53 d for antifungal activity using in vitro and nematode infection assays.
54 MYB83 expression increases were conducive to nematode infection because overexpression of a noncleava
55  and CRN is not only required for successful nematode infection but is also involved in the formation
56 haracteristic changes similar to those after nematode infection but was unable to restore the impaire
57  the major line of defense against parasitic nematode infections, but the arsenal is limited and resi
58  of Th2-mediated protective immunity against nematode infection by a mechanism involving CCL2 product
59  animals were continually exposed to a mixed nematode infection by grazing.
60 that IL-13Ralpha2 plays an important role in nematode infection by limiting the availability of IL-13
61                                     Filarial nematode infections cause a substantial global disease b
62              Previously, we showed that cyst nematode infection causes a localised ROS burst in roots
63                                We found that nematode infection damages the endodermis leading to the
64  infection model to identify novel parasitic nematode infection factors and elucidate the genetic and
65 fic differences in the host response against nematode infection factors.
66 lly produced in response to gastrointestinal nematode infections fail to enhance host protection agai
67                             Gastrointestinal nematode infections generally invoke a type 2 cytokine r
68                                              Nematode infection had no effect on isoflavonoid levels.
69                                    Parasitic nematode infection has both preventive and therapeutic e
70 CLE peptides and are required for successful nematode infection; however, the receptors for nematode
71 H)1 inflammation is thought to favor chronic nematode infection, IFN-gamma was neutralized in vivo, r
72 t mice exhibit impaired Th2 immunity against nematode infection, implicating IL-25 as a key component
73  mechanisms that take place during parasitic nematode infection in insects.
74 13 (IL-13)-producing ILC2s and resistance to nematode infection in mice, which revealed that ILCs are
75 during allergic airway inflammation (AAI) or nematode infection in mice.
76 s were produced in the airways during AAI or nematode infection in mice.
77 ponse that facilitates a prompt clearance of nematode infection in SWR/J mice may have evolved to con
78 ta0.3 TobRB7 promoter, which is induced upon nematode infection in tobacco roots.
79 ons that occur at an extraintestinal site of nematode infection in which the eosinophil functions as
80                                              Nematode infection induced a STAT6-dependent increase in
81 ata show that IL-25 plays a critical role in nematode infection-induced alterations in intestinal fun
82                                    Parasitic nematode infection induces a polarized Th2 cytokine resp
83                                      Enteric nematode infection induces a smooth muscle hypercontract
84                                      Enteric nematode infection induces a strong type 2 T helper cell
85 119 GHE disease states (excluding intestinal nematode infections, iodine deficiency, and vitamin A de
86                             Gastrointestinal nematode infection is known to alter host T cell activat
87           Rapid induction of expression upon nematode infection is localized to root tips.
88 of MPhis during intestinal as well as tissue nematode infection is restricted to sites of IL-4 produc
89 ive pathogen is necessary for virulence in a nematode infection model and for efficient killing of cu
90 n silicone elastomers, and pathogenesis in a nematode infection model as well as alters fungal morpho
91  development of IL-4-expressing B cells in a nematode infection model was dependent on both T cells a
92                                Analysis of a nematode infection model, in which the parasite migrates
93 inhibit C. albicans growth in vitro and in a nematode infection model, showing therapeutic potential.
94 rasite expulsion in several gastrointestinal nematode infection models.
95 -characterized mouse model of human filarial nematode infection, nematode survival and protective imm
96           We then investigated how potential nematode infection of NPFWs could impact wasp survival a
97 ionship in ruminants, using gastrointestinal nematode infections of sheep as the model system.
98 akeri polygyrus to investigate the impact of nematode infections on malarial morbidity and antimalari
99 that in the context of experimental filarial nematode infection, optimum tissue eosinophil recruitmen
100 kinase (MAPK) signaling module, activated by nematode infection or wounding, is crucial for soybeans
101 to the anthelmintics currently used to treat nematode infection, prompting the need to develop new an
102 f these immune cells in host defense against nematode infection remains poorly defined.
103 he host mechanism of defense against enteric nematode infection remains to be fully understood, but i
104 -standing paradigm of eosinophil toxicity in nematode infection requires reevaluation, as our results
105                                       Before nematode infection, RNA blot analysis revealed 1.3-1.8-f
106  the most abundant NPFW species suggest that nematode infection significantly reduces their longevity
107      Analysis of the hologenome of the plant-nematode infection site identified metabolic pathways th
108 r show that StCLV2 is highly up-regulated at nematode infection sites and that transgenic potatoes wi
109  activation of suberin biosynthesis genes at nematode infection sites.
110             The chronic nature of intestinal nematode infections suggests that these parasites have e
111                     Therefore, during tissue nematode infection, T helper 2 (Th2) cells control the d
112 ding the insect immune response to parasitic nematode infection that consists of seven species of EPN
113                                        After nematode infection, the mRNAs declined over 48 h in resi
114                                              Nematode infection triggers plant defense responses; how
115                                              Nematode infection upregulates interleukin-4 (IL-4) and
116 weight was maintained in both strains during nematode infection via different mechanisms.
117 phenotype" is advantageous during intestinal nematode infection, we compared the responses to Heligmo
118 nferred susceptibility to drought stress and nematode infection when overexpressed.
119 e are more energy efficient during parasitic nematode infection, which may explain their ability to t
120 re we show, using separate models of pleural nematode infection with Litomosoides sigmodontis and Alt
121 laris-associated NPFWs to be the targets for nematode infection, with infection levels sometimes exce
122 sociated with the establishment of parasitic nematode infections within the gastrointestinal environm
123 ion as a therapeutic agent against parasitic nematode infection worldwide.

 
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