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1 lly, is reported to be rare or absent in the neonatal animal.
2 is the major form expressed in the fetal and neonatal animal.
3 bling the immature active-stem cell niche of neonatal animals.
4 and prevented paranodal complex formation in neonatal animals.
5 ation of the developing lung in utero and in neonatal animals.
6 bset found in adults is missing in fetal and neonatal animals.
7 ctions of identified skin sensory neurons in neonatal animals.
8 ld induce in the retinocollicular pathway in neonatal animals.
9 s hypothalamic GnRH release in embryonic and neonatal animals.
10 e of the major causes of immunodeficiency in neonatal animals.
11  linked to widespread neuronal cell death in neonatal animals.
12 oform is the only one expressed in the fetal/neonatal animal and showed that expression was limited t
13 in the CNS of adult as well as embryonic and neonatal animals and can differentiate into lineage-rest
14 sruption to amygdala-related networks in the neonatal animals and cortico-striatal related networks i
15 ve been documented to induce neutrophilia in neonatal animals and human infants, increase the NSP, an
16 utrophils have been consistently reported in neonatal animals and humans and contribute to their susc
17 nts of pure olfactory ensheathing cells from neonatal animals and mixed olfactory cells from both neo
18                                           In neonatal animals, apoptosis and necrosis were concentrat
19                                              Neonatal animals are generally very susceptible to infec
20                                              Neonatal animals are highly susceptible to infectious ag
21  metabolism and myelin production persist if neonatal animals are used.
22 lt animals [past postnatal day (P)90] and in neonatal animals as early as P27 formed a dense band in
23 rebral artery myocytes isolated from control neonatal animals but were absent in myocytes from Kir2.1
24  and dysmorphogenesis, which were evident in neonatal animals by anatomical and micro-computed tomogr
25 uggest that anesthetic drugs administered to neonatal animals cause widespread neuronal apoptosis and
26                                  Finally, in neonatal animals CI-AMPARs, but not CP-AMPARs, are the p
27 on arborization, while cultures from E20 and neonatal animals did not.
28 dicate that in the absence of RFC1 function, neonatal animals die due to failure of hematopoietic org
29 g, and at necropsy on PND2 both maternal and neonatal animals displayed increased liver weights, incr
30 ordial follicles by 30% when administered to neonatal animals during the time of germline cyst breakd
31    We recently demonstrated high-dose BMT in neonatal animals enables chimeric engraftment without to
32 e been at high risk of BSE infection only if neonatal animals had inadvertently ingested contaminated
33                                   Studies in neonatal animals in which this interaction can be examin
34 nduce intestinal dysfunction and diarrhea in neonatal animals, including piglets and human infants.
35        In contrast, for neurons generated in neonatal animals, input and input-output synapses appear
36 ncy of IRF-3 and IRF-7 was age dependent, as neonatal animals lacking either factor succumbed to infe
37                                    Using two neonatal animal models (rats and calves) of chronic hypo
38 intrinsic immaturity of the immune system of neonatal animals, neonates are highly sensitive to a var
39        Interestingly, in cortical neurons of neonatal animals NMDARs signal through Sos rather than R
40  quickly, that rapid turnover occurs only in neonatal animals, only in culture, or only in response t
41                                           In neonatal animals (P8-P15), mGluR-LTD is independent of p
42  the proteins in all neurons from ganglia of neonatal animals (postnatal days 0-3) and in 85-90 % of
43          Because B cell development in fetal/neonatal animals principally results in B-1 cells, these
44                                           In neonatal animals, Renshaw cells expressed small punctate
45 ation of beta-catenin in supporting cells in neonatal animals resulted in proliferation of supporting
46                                              Neonatal animals spontaneously reduce fractures, yet the
47 cardiography in all fetal lambs (n = 13) and neonatal animals studied at one and three days of life (
48                               Both adult and neonatal animals that received NanoSiO2-adjuvanted vacci
49 wo groups of retrogradely labeled neurons in neonatal animals, those neurons with axons that ascend t
50 e progress of the reformation of this map in neonatal animals under conditions that enhanced the like
51 ed, and single-cell genomic PCR studies in a neonatal animal were used to examine any relationship be
52                                              Neonatal animals were not considered as suitable vaccine
53 endelian distribution of alleles in Tgfb1-/- neonatal animals which survive prenatal lethality.
54 sraphisms are progressively resolved and the neonatal animals, while showing signs of scarring and ti
55 g strategy for enhancing vaccine efficacy in neonatal animals with maternal antibodies.
56                    The active interaction of neonatal animals with their environment has been shown t