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1 al age, low birth weight, preterm birth, and neonatal infections).
2 It can present as a maternal-neonatal infection.
3 s consistent with an environmental source of neonatal infection.
4 rain contribute to disease outcome following neonatal infection.
5 is, this pathogen remains a leading cause of neonatal infection.
6 smission, and GBS remains a leading cause of neonatal infection.
7 itis is strongly linked to preterm birth and neonatal infection.
8 le in inhibition of antibody responses after neonatal infection.
9 ) is the leading cause of invasive bacterial neonatal infections.
10 eptococcus (GBS) is the major cause of human neonatal infections.
11 preterm labor, stillbirths, fetal injury, or neonatal infections.
12 complication of group B Streptococcus (GBS) neonatal infections.
13 carbapenem resistance genes of CRKP causing neonatal infections.
14 o preterm births, stillbirths, or late-onset neonatal infections.
15 HSV-2 to generate annual numbers of incident neonatal infections.
16 ositive bacterium that is a leading cause of neonatal infections.
17 ease morbidity and mortality associated with neonatal infections.
18 s to miscarriage, preterm birth, and serious neonatal infections.
19 oup B streptococci (GBS), the major cause of neonatal infections.
20 host defenses may attenuate the virulence of neonatal infections.
21 g the most common causes of life-threatening neonatal infections.
22 ) is the foremost bacterial cause of serious neonatal infections.
23 llowed by congenital infections (126 [20%]), neonatal infections (122 [20%]), and respiratory distres
24 epsis, pneumonia and meningitis (combined as neonatal infections; 331 [30%]), and asphyxia (151 [14%]
25 ses from 372 centres, including 107 maternal-neonatal infections, 427 cases of bacteraemia, and 252 c
26 .4]) vs 4.4% [1.9-8.1]), and antibiotics for neonatal infection (74.7% [55.3-90.1] vs 96.4% [94.0-98.
30 income country provides a baseline burden of neonatal infections against which the impact of future c
32 that reported effect measures on the risk of neonatal infection among newborns exposed to maternal in
34 s, illness severity, vaccinations, and early neonatal infections among obstetric patients during the
35 sistant HSV-2 mutants can develop rapidly in neonatal infection and cause clinically significant dise
36 implicated as one of the causative agents in neonatal infection and causes a septicemia thought to be
39 Streptococcus (GBS) is the leading cause of neonatal infections and an important pathogen in pregnan
40 CRKP contributes to a considerable number of neonatal infections and leads to significant neonatal mo
42 ht represent a therapeutic tool for treating neonatal infections and support the view that breastfeed
43 gnostic methods for identifying maternal and neonatal infection, and regarding optimal prevention and
44 of more severe symptoms such as eye disease, neonatal infection, and, in rare cases, encephalitis.
45 tted, have been implicated in preterm birth, neonatal infections, and chronic lung disease of prematu
48 pathogens responsible for most of the severe neonatal infections, and the time it takes to induce pro
55 h (aRR, 1.25; 95% CI, 1.13-1.30), and having neonatal infections (aRR, 1.42; 95% CI, 1.17-1.73), any
56 regnancy may set back biological evidence of neonatal infection at birth and underline the need for a
57 longed or unregulated treatment with oxygen, neonatal infection, blood transfusion, and mechanical in
58 to term is associated with increased risk of neonatal infection, but immediate delivery is associated
59 hat developed tumors 7 to 8 months following neonatal infection by polyomavirus (PYV) wild-type strai
62 ossible differences or clinical relevance of neonatal infection caused by different biotypes or newer
63 review of studies reporting population-based neonatal infections caused by CRKP in combination with a
65 We interpret these data to indicate that neonatal infection causes significant neuronal sequestra
72 rnal antibody concentrations and the risk of neonatal infection has been investigated in US and Afric
73 Few population-level estimates of invasive neonatal infections have been reported from sub-Saharan
75 n is a viable strategy for the prevention of neonatal infections.IMPORTANCE Herpes simplex virus is a
76 mpregnate the fetus during pregnancy, on GBS neonatal infection in cellular and mouse models of hormo
79 ished that G10P[11] RVs are a major cause of neonatal infection in Vellore, India, with half of infec
80 ll important approaches to prevent and treat neonatal infections in additional to regular neonatal se
81 ts define an important role for gamma34.5 in neonatal infections in contrast to other studies indicat
82 at emerged as the leading cause of bacterial neonatal infections in Europe and North America during t
85 line treatment of serious community-acquired neonatal infections in rural Bangladesh, which has a mod
88 g or after birth results in life-threatening neonatal infections, including pneumonia, sepsis, and me
89 e role for aberrant neuronal iron storage in neonatal infection-induced disturbances in myelination a
91 e vaccine, and antibody generated by primary neonatal infection is poorly protective against reinfect
94 eumoniae is the most common pathogen causing neonatal infections, leading to high mortality worldwide
96 d early-life IL-27 on the host response in a neonatal infection model while also defining the cell an
97 is increased susceptibility and severity of neonatal infections necessitating admission to the inten
104 owel in adult mammals, but their role during neonatal infection of the small bowel is not well establ
106 We and others previously demonstrated that neonatal infection of ZIKV results in heart failure and
107 o assess the impact of suspected early-onset neonatal infection on tactile- and noxious-evoked respon
108 ne-encoded TCR clonotypes, characteristic of neonatal infection, persisted in the brain, albeit somet
109 ncluded histories of perinatal difficulties, neonatal infections, postnatal brain infections, and tra
110 ternal infection, vertical transmission, and neonatal infection published from January 1, 1960 to Mar
111 size that acute brain inflammation caused by neonatal infection reduces the bioavailability of iron r
114 s suggests that the impact of these cells on neonatal infection risk and progression may be limited.
115 KMC), and antibiotics for clinically defined neonatal infection (sepsis, pneumonia, or meningitis), w
116 expression analysis of rat brains following neonatal infection showed increased expression of kynure
120 ist, aims to improve scientific reporting of neonatal infection studies, increasing data utility and
121 proved scientific reporting of observational neonatal infection studies, to increase comparability an
123 Thus, our study challenges the notion that neonatal infection susceptibility is due to immune cell-
125 y has decreased the incidence of early-onset neonatal infection, these measures do not prevent ascend
126 on and oral antibiotic therapy for suspected neonatal infections to a basic preventive and promotive
127 further evaluated for potential relevance to neonatal infection, transplantation, and acquired immuno
130 rize the effects of maternal UFP exposure on neonatal infection, we exposed time-mated C57Bl/6n mice
132 Although few congenital malformations or neonatal infections were seen among exposed neonates, wo
133 vents that occur in the brain as a result of neonatal infection, which likely contribute to cognitive
134 tudied the induction of adaptive immunity to neonatal infection with a murine retrovirus, under condi
135 ultures, there were 37 631 incident cases of neonatal infection with at least one pathogen isolated.
137 contrast to infection of mature BALB/c mice, neonatal infection with rhinovirus promotes an IL-25-dri
139 taphylococcus (MRS) has been associated with neonatal infections, with colonization of the anovaginal
140 ith ruptured membranes close to term reduces neonatal infection without increasing other morbidity.