ライフサイエンスコーパス: neoplasia

1 .41) compared with veterans with no baseline neoplasia.

2 ith corresponding spectral signatures of the neoplasia.

3 during development, tissue regeneration and neoplasia.

4 infections and to treat human papillomavirus neoplasia.

5 d PD-L2, in invasive ocular surface squamous neoplasia.

6 d essentially cures Apc(1322T) mice of their neoplasia.

7 res and their risk for metachronous advanced neoplasia.

8 olonoscopy on detection of proximal serrated neoplasia.

9 of these patients for metachronous advanced neoplasia.

10 fective in improving detection of colorectal neoplasia.

11 press ETV1 develop prostatic intraepithelial neoplasia.

12 fects frequently observed in human sebaceous neoplasia.

13 in wound healing, and potentially to prevent neoplasia.

14 cells resulted in prostatic intraepithelial neoplasia.

15 not increase risk for metachronous advanced neoplasia.

16 isk of synchronous and metachronous advanced neoplasia.

17 de NMD and promoting BCL2 overexpression and neoplasia.

18 10 wk after dosing, well before evidence of neoplasia.

19 to modulate gene expression in early colonic neoplasia.

20 for refractory proctitis, and 20% for rectal neoplasia.

21 cally refractory disease or to treat colonic neoplasia.

22 e genetic models of clonal hematopoiesis and neoplasia.

23 or initiation in a mouse model of intestinal neoplasia.

24 ppression and stochastic effects, leading to neoplasia.

25 is sufficient for the development of T cell neoplasia.

26 otective immunity and limit autoimmunity and neoplasia.

27 ticular, the development of parasite-induced neoplasia.

28 a mechanism by which its deficiency promotes neoplasia.

29 n, suggesting these mutations play a role in neoplasia.

30 biomarkers for early detection of pancreatic neoplasia.

31 significantly associated with progression to neoplasia.

32 generally considered a risk factor for colon neoplasia.

33 translate into higher detection of advanced neoplasia.

34 ly used across the spectrum of hepatobiliary neoplasia.

35 tion of how single mutant cells give rise to neoplasia.

36 is linked to elevated IL-6 concomitant with neoplasia.

37 , recurring bacterial infections, or de novo neoplasia.

38 s found to have at least 1 incident advanced neoplasia.

39 body (18)F-FDG PET and CT scans did not show neoplasia.

40 nsistent, early microbial changes in colonic neoplasia.

41 ry outcome was diagnostic yield for advanced neoplasia.

42 ty, and the microbiota in colitis-associated neoplasia.

43 tumor suppressor mechanism in numerous human neoplasias.

44 y, resulting in growth inhibition of several neoplasias.

45 tiation inducers (eg, retinoids), in myeloid neoplasias.

46 and some enlarged pancreatic intraepithelial neoplasias.

47 RAS, premalignant pancreatic intraepithelial neoplasia 1 (PanIN1) lesions rarely become fully maligna

48 of ADM as well as pancreatic intraepithelial neoplasia-1 (PanIN-1), PanIN-2, and PanIN-3 and delayed

49 low-risk patients with metachronous advanced neoplasia (14.6%) (relative risk for high-risk categoriz

50 who were found to have metachronous advanced neoplasia (17.6%) did not differ significantly from the

51 BTC precursor lesion, intraductal papillary neoplasia (18.3%).

52 t cervical lesions (cervical intraepithelial neoplasia 2+ [CIN2+]) is an effective way to prevent cer

53 es of death were renal disease (102, 23.4%), neoplasia (37, 8.5%) and mass-associated disorder (35, 8

54 their role in the earliest stages of colonic neoplasia, 75% of the loci harboring methylation changes

55 aracteristics of FIT for advanced colorectal neoplasia (ACN) in patients with CKD, we used FIT to pro

56 l prognostic factors for advanced colorectal neoplasia (aCRN, high-grade dysplasia, or CRC) in patien

57 Large polyps, advanced neoplasia (advanced adenomas and cancer), and invasive c

58 We investigated a disseminated neoplasia affecting geographically distant populations o

59 lt to treat recurrent or residual colorectal neoplasia after previous endoscopic therapy.

60 elial lesions (ASIL) or anal intraepithelial neoplasia (AIN) are precancerous lesions.

61 on anal HPV infection, anal intraepithelial neoplasia (AIN), and anal cancer among people living wit

62 High-grade anal intraepithelial neoplasia (AIN2/3; HGAIN) is highly prevalent in human i

63 quent endoscopy, the odds for progression to neoplasia also increased greatly (odds ratio, 9.28; 95%

64 like mucosal inflammation, preneoplasia, and neoplasia, although suggested by several studies, remain

65 and later development of colorectal advanced neoplasia (AN).

66 nset of high-grade prostatic intraepithelial neoplasia and accelerated prostate tumors in comparison

67 shed technique for staging of neuroendocrine neoplasia and determining the suitability of patients fo

68 ogenous and exogenous virus and is linked to neoplasia and immunosuppression in koalas.

69 hemoattraction to pancreatic intraepithelial neoplasia and increased abdominal hypersensitivity cause

70 at the severity of prostatic intraepithelial neoplasia and inflammation development gradually increas

71 027), but was limited to open procedures for neoplasia and inflammatory bowel disease patients.

72 uidelines effectively stratified the risk of neoplasia and malignancy.

73 resentations, including benign and malignant neoplasia and neurodevelopmental disorders.

74 tly increased detection of proximal serrated neoplasia and other polyp types compared with standard c

75 mice, tissue atrophy was a hallmark of both neoplasia and pancreatitis models in the absence of Ddr1

76 trial, 10 patients with known neuroendocrine neoplasia and positive for uptake on (68)Ga-DOTA-octreot

77 veloped high-grade prostatic intraepithelial neoplasia and prostate cancer.

78 the molecular and cellular features of early neoplasia and the dynamics of the interactions of early

79 of HIF1A developed more advanced pancreatic neoplasias and PDACs with more invasion and metastasis,

80 apeutic responses against carcinogen-induced neoplasias and transplantable tumors.

81 cerous lesions (endometrioid intraepithelial neoplasia) and well-differentiated endometrioid adenocar

82 omas, 1 low-grade intraepithelial pancreatic neoplasia, and 1 case of polycystic kidney disease.

83 for conjunctival melanocytic intraepithelial neoplasia, and 81% for WHO classification system.

84 to coordinate fibrogenesis in tissue injury, neoplasia, and aging.

85 cancer, which is the second most common male neoplasia, and benign prostatic hyperplasia (BPH), which

86 xic T cells, attenuated growth of pancreatic neoplasia, and conferred survival advantage to PDA-beari

87 ) for any neoplasm 6 mm or greater, advanced neoplasia, and CRC for mt-sDNA were 54.2%, 22.7%, and 1.

88 n traditionally viewed as unique features of neoplasia, and have been used as biomarkers and therapeu

89 ologic processes that regulate inflammation, neoplasia, and infection.

90 ation of kidney cysts, renal intraepithelial neoplasia, and invasive papillary renal carcinoma.

91 AM, conjunctival melanocytic intraepithelial neoplasia, and the WHO 4th edition classification of con

92 induced acinar cell dedifferentiation, early neoplasia, and throughout PDA progression.

93 a context of intraductal papillary mucinous neoplasia, and vascular resections were independently as

94 itotic activity, axonal disruption, vascular neoplasia, and with several brain metabolites including

95 n in the distal colon of multiple intestinal neoplasia (Apc(min/+) ) mice; tumor formation is depende

96 nd postmenopausal women with intraepithelial neoplasia are discussed in the Clinical Considerations s

97 One-fourth of colorectal neoplasias are missed during screening colonoscopies; th

98 neoplasms, while pancreatic intraepithelial neoplasias are rarely detected.

99 ls are increased in cervical intraepithelial neoplasias as well as cervical carcinomas, consistent wi

100 aplasia-associated prostatic intraepithelial neoplasia at a low frequency.

101 3).ConclusionThe detection rates of advanced neoplasia at CT colonography screening were greater than

102 nal joint model to estimate risk of advanced neoplasia at each subsequent examination after baseline,

103 for prior surveillance, the risk of advanced neoplasia at each subsequent examination was not signifi

104 ce developed more pancreatic intraepithelial neoplasia, at a faster rate, than KC mice, and had more

105 Viral infections at any time and malignant neoplasia beyond 2 years were less frequent with mTORi-C

106 tection of early Barrett's esophagus-related neoplasia (BORN) is a challenge.

107 nting recurrence from breast intraepithelial neoplasia but have a lower toxicity than the standard do

108 RB loss occurs commonly in neoplasia but its contributions to advanced cancer have

109 anscription factor activity in hematological neoplasia, but do not confirm the putative role of TET2

110 ed in stroma across all stages of pancreatic neoplasia, but epithelial Tlr7 expression is relatively

111 Drak was necessary for glial neoplasia, but not for normal glial proliferation and de

112 ns are indicative of tissue inflammation and neoplasia, but whether these alterations contribute to d

113 iates protection from pathogens and controls neoplasias, but can also cause autoimmunity.

114 d estimated cumulative incidence of advanced neoplasia by Kaplan-Meier curves.

115 eloid leukemia (CML)-like myeloproliferative neoplasia by repressing an inhibitory mechanism of prese

116 the progression of prostate intraepithelial neoplasia, by promoting cell proliferation, micro-invasi

117 The biology of neoplasia caused by germline mutations has led to paradi

118 relationship between blastic plasmacytoid DC neoplasia cells and healthy DCs; and circulating progeni

119 L) is a hematopoietic stem cell (HSC)-driven neoplasia characterized by expression of the constitutiv

120 stiocytosis (LCH) is an inflammatory myeloid neoplasia characterized by granulomatous lesions contain

121 and bone represent a heterogeneous group of neoplasias characterized by a wide variety of genetic ab

122 yield of high-grade cervical intraepithelial neoplasia (CIN) and the influence on biopsy and treatmen

123 logically confirmed cervical intraepithelial neoplasia (CIN) grade 2 or higher regardless of HIV stat

124 We analyzed data on cervical intraepithelial neoplasia (CIN) grades 2-3 and adenocarcinoma in situ (d

125 ial lesion (SIL) or cervical intraepithelial neoplasia (CIN) prevalence, incidence, progression, or r

126 he population-based cervical intraepithelial neoplasia (CIN) trends when adjusting for changes in cer

127 Comparison of conjunctival intraepithelial neoplasia (CIN) vs SCC revealed SCC with greater diffuse

128 o ascertain whether cervical intraepithelial neoplasia (CIN) will regress or progress.

129 oma virus (HPV) +/- cervical intraepithelial neoplasia (CIN), or cervical cancer.

130 HPV) infection, and cervical intraepithelial neoplasia (CIN); however, causal inference remains uncer

131 h disease severity (cervical intraepithelial neoplasia [CIN] 3, 17.9% [+/-7.2] vs CIN2, 11.6% [+/-6.5

132 (PVL) on high-grade cervical intraepithelial neoplasia (CIN2+) detection at follow-up after CIN manag

133 rall, 28 high-grade cervical intraepithelial neoplasia (CIN3) cases were detected.

134 stic lesions called cervical intraepithelial neoplasias (CINs) need be treated to prevent cervical ca

135 o ascertain whether cervical intraepithelial neoplasias (CINs) regress or progress.

136 Pancreatic neoplasia continues to be extensively studied in surgica

137 s with CRC and 10 persons without colorectal neoplasia (control samples) and a second series of 81 pa

138 as, as well as 24 persons without colorectal neoplasia (control samples).

139 s, as well as 129 persons without colorectal neoplasia (control samples); 72 FIT samples from a third

140 ociated with an increased risk of colorectal neoplasia (CRN) or extracolonic cancers has not been rob

141 (PIPs) have an increased risk of colorectal neoplasia (CRN).

142 d to determine the 10-year risk for advanced neoplasia (defined as adenomas >=10mm, adenomas with vil

143 he TFF1 knockout (KO) mouse model of gastric neoplasia, demonstrated phosphorylation of STAT3 in the

144 rizes current evidence for FIT in colorectal neoplasia detection and the comparative effectiveness of

145 n the intention-to-screen analysis, advanced neoplasia detection rates were comparable among the cont

146 Outcome measures were participation, neoplasia detection, and adverse events.

147 In contrast, small intestinal neoplasia development significantly correlated with age

148 One lncRNA, Colorectal Neoplasia Differentially Expressed (CRNDE), was selected

149 ng oncoprotein critical for the evolution of neoplasia driven by Apc mutation.

150 ere, we show that pancreatic intraepithelial neoplasia driven by oncogenic mutant KrasG12D progressed

151 erhans cell histiocytosis (LCH) is a myeloid neoplasia, driven by sporadic activating mutations in th

152 of a CADe system in detection of colorectal neoplasias during real-time colonoscopy.

153 ulated that metformin would enhance the anti-neoplasia effect of BPTES by suppressing malignant cells

154 esearch in high-germline risk for pancreatic neoplasia, elucidating early ontogeny in BRCA1-mutation

155 prostatic susceptibility to hormone-induced neoplasia from early-life exposure to low-dose bisphenol

156 histological images to differentiate tongue neoplasia from normal tissue, and then correlate these d

157 0.695, and discriminate patients with early neoplasia from patients without BE with AuROC values ran

158 s to discriminate patients with BE and early neoplasia from patients without BE, using findings from

159 n cancer, and in rodent models of intestinal neoplasia, FXR knockout increases the size and number of

160 e cervical disease (cervical intraepithelial neoplasia grade 2 or 3, adenocarcinoma in situ, invasive

161 tivity of detecting cervical intraepithelial neoplasia grade 2 or greater in HIV-negative women using

162 ormative to predict cervical intraepithelial neoplasia grade 2 or greater.

163 or the detection of cervical intraepithelial neoplasia grade 2 or higher (CIN2+).

164 ocarcinoma in situ (cervical intraepithelial neoplasia grade 2 or higher [CIN2+]) in 2008.

165 elial lesion (HSIL) and anal intraepithelial neoplasia grade 2 or more severe diagnoses (HSIL/AIN2+),

166 tered, diagnoses of cervical intraepithelial neoplasia grade 2 or worse (CIN2+) or grade 3 or worse (

167 PV 16/18-associated cervical intraepithelial neoplasia grade 2 or worse (CIN2+).

168 wart diagnoses, and cervical intraepithelial neoplasia grade 2+ (CIN2+) to summarise the most recent

169 nd invasive cancer (cervical intraepithelial neoplasia grade 2+ [CIN2+]) compared to cytology, but no

170 and vaginal disease (vaginal intraepithelial neoplasia grade 2/3, vaginal cancer) related to HPV 31,

171 oma), vulvar disease (vulvar intraepithelial neoplasia grade 2/3, vulvar cancer), and vaginal disease

172 omen diagnosed with cervical intraepithelial neoplasia grade 3 (CIN3).

173 confirmed CIN2+ or cervical intraepithelial neoplasia grade 3 or worse associated with HPV 16/18 cer

174 CI, 3.64 to 12.25) for anal intraepithelial neoplasia grade 3, 4.97 (95% CI, 3.26 to 7.57) for vulva

175 I, 9.69 to 19.25) for vulvar intraepithelial neoplasia grade 3, 86.08 (95% CI, 11.98 to 618.08) for v

176 10.50 to 62.69) for vaginal intraepithelial neoplasia grade 3, and 5.51 (95% CI, 1.22 to 24.84) for

177 in patients with pancreatic intraepithelial neoplasias grade 3 and PDAC lesions relative to matched

178 d HPV 16/18 to find cervical intraepithelial neoplasia (grade >/=2 [CIN2+] or grade >/=3 [CIN3+]).

179 changes in rates of cervical intraepithelial neoplasia grades 2, 3 and adenocarcinoma in situ (CIN2+)

180 We analyzed data on cervical intraepithelial neoplasia grades 2-3 and adenocarcinoma in situ (CIN2+)

181 datory reporting of cervical intraepithelial neoplasia grades 2/3 and adenocarcinoma in situ (cervica

182 on of gene fusions as a driving mechanism in neoplasia has led to development of U.S. Food and Drug A

183 esented high-grade prostatic intraepithelial neoplasia (HG-PIN) and hyperproliferation, while Pten si

184 enotypes-and high-grade anal intraepithelial neoplasia (HGAIN) in men who have sex with men (MSM) who

185 ed from high-grade prostatic intraepithelial neoplasia (HGPIN), a pre-malignant intraductal prolifera

186 l significance of UDD in predicting advanced neoplasia [high grade dysplasia or invasive carcinoma (H

187 memory, they also can cause autoimmunity or neoplasia if misdirected or dysregulated.

188 fied the optimal site for biopsy of detected neoplasia in 97% and 92% of cases (data sets 4 and 5, re

189 een testicular microlithiasis and testicular neoplasia in a large geographically diverse pediatric po

190 ubjects with IBD undergoing surveillance for neoplasia in Australia (23 with Crohn's colitis, 29 with

191 n images of rigorously confirmed early-stage neoplasia in BE and nondysplastic BE, derived from 669 p

192 ies on histological detection of endoluminal neoplasia in biopsy specimens.

193 scoring of images that contained early-stage neoplasia in data sets 2-5 were delineated in detail for

194 th those from the BE experts in all detected neoplasia in data sets 4 and 5.

195 colorectal cancer is the fourth most common neoplasia in Europe with 28.2 new cases per 100,000 inha

196 egradation of Pten, lung adenocarcinoma, and neoplasia in mouse prostate with aberrantly high levels

197 ce, to improve endoscopic detection of early neoplasia in patients with Barrett's esophagus (BE).

198 mputer-aided system for primary detection of neoplasia in patients with BE.

199 either ICMT or NOTCH1 accelerates pancreatic neoplasia in Pdx1-Cre;LSL-Kras(G12D) mice, suggesting th

200 tified on a per genome basis, with germ cell neoplasia in situ possessing the least (median 1, range

201 icular microlithiasis and primary testicular neoplasia in this pediatric population.

202 ance of PGRB relative to that of PGRA drives neoplasia in vivo by stimulating cell cycling.

203 as-mTORC2 interaction impaired Ras-dependent neoplasia in vivo.

204 ogression of oncogenic RAS-driven melanocyte neoplasia in zebrafish.

205 tirely surrounded pancreatic intraepithelial neoplasias in KPC/Cdh11(+/+) mice and incompletely in KP

206 umor Analysis (IOTA) Assessment of Different Neoplasias in the Adnexa model system, a risk prediction

207 Ocular surface squamous neoplasia includes a spectrum of diseases from dysplasia

208 communication between the immune system and neoplasia includes an increasingly complex cellular micr

209 ncluded detection of any adenoma or advanced neoplasia (including CRC) and screening-related harms (i

210 carcinogens, are risk factors for colorectal neoplasia, including polyps, the precursor for colorecta

211 rveillance mechanism that detects a range of neoplasia-inducing processes.

212 enome Atlas (TCGA) and the Genomics Evidence Neoplasia Information Exchange (GENIE) breast cancer gen

213 ROUND & AIMS: Intraductal papillary mucinous neoplasias (IPMNs) are precancerous cystic lesions that

214 Thyroid neoplasia is common and requires appropriate clinical wo

215 ce an inflammatory insult towards epithelial neoplasia is less understood.

216 MYC, its function in normal development and neoplasia is poorly defined.

217 inal surgery for the treatment of colorectal neoplasia is typically carried out using electrocautery

218 Whether they mediate beta-catenin-dependent neoplasia is unclear.

219 cy with magnesium defect, EBV infection, and neoplasia) is a complex primary immunological deficiency

220 Neoplasia manifested as transplantable CD8+ lymphoma or

221 in a specific research area, like pancreatic neoplasia, may yield informative insights for researcher

222 veractive RET can lead to multiple endocrine neoplasia (MEN) syndromes.

223 crossed with the Kras(G12D/+); Ptf1a(Cre/+) neoplasia model and subjected to cerulein-induced experi

224 n discriminating between patients with early neoplasia (n = 94) vs no BE.

225 se-related proteins, with causative roles in neoplasia, neurodegeneration, hepatosteatosis, and other

226 nogenital warts and cervical intraepithelial neoplasia of grade 2 or 3 and cases of cervical cancer a

227 nogenital warts and cervical intraepithelial neoplasia of grade 2 or 3 and cases of cervical cancer a

228 ent qHPV infection, cervical intraepithelial neoplasia of grade 2 or higher [CIN2+], or genital warts

229 ecurrent high-grade cervical intraepithelial neoplasia of grade two or worse (CIN2+) and compared it

230 es quantifying the architectural features of neoplasia on a microscopic scale, with the spectral sign

231 sment narrowed the differential diagnosis to neoplasia or infection.

232 < .001) or HIV-positive individuals without neoplasia or opportunistic infection (1.2%; P < .001).

233 irus (HIV)-infected patients with underlying neoplasia or opportunistic infections, including tubercu

234 lymphomas and HIV-positive patients without neoplasia or opportunistic infections.

235 surgical outcomes of ocular surface squamous neoplasia (OSSN) following wide excisional biopsy with a

236 omas at baseline have a low risk of advanced neoplasia over 10 years.

237 sitive association between proviral load and neoplasia (P = 0.009).

238 es of preinvasive pancreatic intraepithelial neoplasia (PanIN) in the KrasLSL-G12D/+ Trp53LSL-R172H/+

239 to fibrogenesis, pancreatic intraepithelial neoplasia (PanIN) lesion growth, and generation of an im

240 samples of human pancreatic intraepithelial neoplasia (PanIN), along with several biochemical approa

241 for inflammation, pancreatic intraepithelial neoplasia (PanIN), and PDAC.

242 he progression of pancreatic intraepithelial neoplasia (PanIN), and resulted in the accumulation of (

243 rmal pancreas and pancreatic intraepithelial neoplasia (PanIN)- and PDAC-bearing pancreas from both h

244 ts progression to pancreatic intraepithelial neoplasia (PanIN).

245 ion of high-grade pancreatic intraepithelial neoplasias (PanIN-3), generally regarded as the non-inva

246 ion of high-grade pancreatic intraepithelial neoplasias (PanIN-3s), generally regarded as the noninva

247 iate precancerous pancreatic intraepithelial neoplasia (PanINs) when induced in mouse acinar cells.

248 features of human pancreatic intraepithelial neoplasia (PanINs), the precursor to pancreatic ductal a

249 duce formation of pancreatic intraepithelial neoplasia (PanINs)-a precursor of PDAC.

250 es (TC) and early pancreatic intraepithelial neoplasia (PanINs).

251 metaplasia (ADM), pancreatic intraepithelial neoplasias (PanINs) and ultimately pancreatic ductal ade

252 Further, prostatic intraepithelial neoplasia (PIN) progressed to carcinoma in rats given ne

253 ce and severity of prostatic intraepithelial neoplasia (PIN), a premalignant lesion.

254 ated in high-grade prostatic intraepithelial neoplasia, primary prostate carcinoma, and metastases.

255 ish model of oncogenic RAS-driven melanocyte neoplasia progression.

256 related species at the clade level revealed neoplasia rates as low as 3.1% and 1.8%, while infectiou

257 ed in a significantly lower rate of cervical neoplasia recurrence over 24 months.

258 us histology or high-grade dysplasia) and no neoplasia, respectively (log-rank P = .10).

259 idered at low risk for metachronous advanced neoplasia, resulting in an inappropriately long surveill

260 was strongly associated with progression to neoplasia; risk for progression increased greatly when a

261 of post-treatment effects for any colorectal neoplasia (RR: 1.01; 95% CI: 0.80, 1.28; P = 0.94), and

262 imately linked to several diseases including neoplasia, sepsis and autoimmune diseases(1,2).

263 mily during early pancreatic intraepithelial neoplasia stage I (PanIN-I) of pancreatic cancer.

264 oncogenic pathways implicated in pancreatic neoplasia, such as MYC, KRAS, VEGFA, and BRD4 Specifical

265 and cohesin mutations are common in myeloid neoplasia, suggesting a critical role in hematopoiesis.

266 ore accurate in discriminating BE with early neoplasia than GERD frequency and duration alone (AuROC,

267 tion, occur more frequently in early colonic neoplasia than previously believed, and identify epigeno

268 Melanoma is a malignant neoplasia that is highly resistant to chemotherapy and r

269 al and environmental insults associated with neoplasia that jeopardize proteostasis within the endopl

270 sis and diagnosis of follicular cell-derived neoplasia tissues and FNA biopsies.

271 Multiple endocrine neoplasia type 2 (MEN 2) syndrome is an autosomal domina

272 h different phenotypes of multiple endocrine neoplasia type 2 as well as sporadic medullary thyroid c

273 the following phenotypes: multiple endocrine neoplasia type 2A (MEN 2A) and multiple endocrine neopla

274 asia type 2A (MEN 2A) and multiple endocrine neoplasia type 2B (MEN 2B) syndromes.

275 ng surgery is feasible in multiple endocrine neoplasia type 2B and affords a good chance for normal a

276 Multiple endocrine neoplasia type 2B is a rare syndrome caused mainly by Me

277 pe and natural history of multiple endocrine neoplasia type 2B, to increase awareness and improve det

278 on the natural history of multiple endocrine neoplasia type 2B.

279 BACKGROUND & AIMS: The multiple endocrine neoplasia, type 1 (MEN1) locus encodes the nuclear prote

280 ve nature of STAT5B(N642H) in driving T-cell neoplasia upon hematopoietic expression in transgenic mi

281 The cumulative 10-year incidence of advanced neoplasia was 6.3% (95% CI 4.1%-8.5%) and 4.1% (95% CI 2

282 those who extended treatment, any colorectal neoplasia was found in 118 (36%) participants assigned t

283 The cumulative 10-year incidence of advanced neoplasia was highest among those with baseline CRC (43.

284 ssess microbiome changes prior to colorectal neoplasia, we investigated samples from 100 Lynch syndro

285 To dissect the events leading to neoplasia, we used transmission electron microscopy (TEM

286 graphy, overall detection rates for advanced neoplasia were 2.7% and 5.0%, respectively (P < .001); c

287 surgery, and three cases with advanced stage neoplasia were also treated with chemotherapy.

288 ients at high risk for metachronous advanced neoplasia were defined as patients with polyps that had

289 sitive predictive values (PPVs) for advanced neoplasia were determined weekly, quarterly, and yearly.

290 No differences in OS or second neoplasias were observed in in both trials.

291 All cancers (except EBV-related neoplasia) were recorded.

292 Plasticity of neoplasia, whereby cancer cells attain stem-cell-like pr

293 diseases, including inherited disorders and neoplasia, which arise from rare failures at different N

294 ic lesions called pancreatic intraepithelial neoplasias, which are challenging to detect by any curre

295 lve the recurrence of breast intraepithelial neoplasia with a limited toxicity, which provides a new

296 The system detected neoplasia with high accuracy and near-perfect delineatio

297 ) classification system, is used to identify neoplasias with invasion of the submucosa that require e

298 esions (high-grade prostatic intraepithelial neoplasia) with striking nuclear atypia and invasive, po

299 colonoscopy findings associate with advanced neoplasia within 10 years.

300 with Epstein-Barr virus (EBV) infection and neoplasia (XMEN), a disease that has a broad range of cl