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1  prevented renal dysfunction, and attenuated nephrosclerosis.
2 RD among African Americans with hypertensive nephrosclerosis.
3  HIV-associated nephropathy and hypertensive nephrosclerosis.
4 rity (76%) had FSGS and 10% had hypertensive nephrosclerosis.
5 he age-related increase in the prevalence of nephrosclerosis.
6 rat models of FSGS and in human hypertensive nephrosclerosis.
7 ed TGF-beta signaling significantly modifies nephrosclerosis.
8 regulation of MMP-14, and the development of nephrosclerosis.
9 ing diagnoses of impaired renal function and nephrosclerosis-age-associated histologic changes in the
10 ar-old female with a history of hypertensive nephrosclerosis and anuria on dialysis for 8 years.
11  with other microvascular disease, including nephrosclerosis and diabetic nephropathy.
12  increased BP in black men with hypertensive nephrosclerosis and is associated with adverse clinical
13 ldosterone contributes to the development of nephrosclerosis and renal fibrosis in rodent models, but
14 ion) among participants who had hypertensive nephrosclerosis and were enrolled in the African America
15 logy (nephron number, glomerular volume, and nephrosclerosis), and short- and long-term GFR <60 or <4
16 er GFR, lower uric acid, more nephrons, less nephrosclerosis, and a lower risk of low GFR at follow-u
17 ore nephrons, larger glomerular volume, more nephrosclerosis, and higher risk of low GFR at follow-up
18 duced aortic stiffness, ameliorated diabetic nephrosclerosis, and improved renal function.
19 nuria, more nephrons, larger glomeruli, less nephrosclerosis, and lower risk of low GFR at follow-up.
20 l segmental glomerulosclerosis, hypertensive nephrosclerosis, and lupus nephritis.
21 pyramid volume and count, glomerular volume, nephrosclerosis, and nephron number were assessed by dee
22  mild to moderate essential hypertension and nephrosclerosis; angiotensin-converting enzyme because o
23 crostructural level, the histologic signs of nephrosclerosis (arteriosclerosis/arteriolosclerosis, gl
24 ects of tamoxifen in a model of hypertensive nephrosclerosis (chronic inhibition of nitric oxide synt
25                                              Nephrosclerosis, defined by globally sclerotic glomeruli
26 selection bias because kidneys with advanced nephrosclerosis from high-risk donors are typically disc
27  among 1094 black patients with hypertensive nephrosclerosis (GFR 20 to 65 ml/min per 1.73 m(2)).
28                                              Nephrosclerosis (glomerulosclerosis, interstitial fibros
29                            The prevalence of nephrosclerosis (&gt; or =2 chronic histologic abnormalitie
30 plain the strong association between age and nephrosclerosis in healthy adults.
31          In black patients with hypertensive nephrosclerosis, increased proteinuria, reduced GFR, and
32 sed risk of progressive CKD occurs only when nephrosclerosis is abnormal for age, age-based threshold
33                                  Subclinical nephrosclerosis, larger cortical nephron size, and small
34  effects in the L-NAME model of hypertensive nephrosclerosis, likely through the inhibition of TGF-be
35                      Nephron hypertrophy and nephrosclerosis may be important determinants of CKD and
36 enosis, and arteriolar hyalinosis to measure nephrosclerosis; mean glomerular volume, cortex volume p
37  diabetic nephropathy (n = 26), hypertensive nephrosclerosis (n = 7), idiopathic focal segmental glom
38                                              Nephrosclerosis, nephron size, and nephron number vary a
39 sive CKD did not differ between persons with nephrosclerosis "normal compared with young" versus "nor
40 and age-based 95th percentile thresholds for nephrosclerosis on the basis of morphometry of kidney bi
41 r a radical nephrectomy, larger nephrons and nephrosclerosis predicted progressive CKD, and IF/TA pre
42 s abnormal for age, age-based thresholds for nephrosclerosis seem to be better than a single young-ag
43 shion parallel to albuminuria that occurs in nephrosclerosis, several facets of cerebrovascular disea
44                       Optimal thresholds for nephrosclerosis that identify persons with a progressive
45 dney failure or 40% decline in eGFR) between nephrosclerosis that was "normal compared with young," "
46 gressive CKD using aged-based thresholds for nephrosclerosis versus a single young-adult threshold is
47 nefit of slowing progression of hypertensive nephrosclerosis was observed with the lower BP goal.
48  in 1094 African Americans with hypertensive nephrosclerosis, which examined the effects of two level
49 s merging of pyramids because of age-related nephrosclerosis, with loss of detectable cortical column