ライフサイエンスコーパス: nephrotic syndrome

1 direct podocyte injury drives proteinuria or nephrotic syndrome.

2 contribution of PCSK9 to the dyslipidemia of nephrotic syndrome.

3 ameliorates dyslipidemia in a mouse model of nephrotic syndrome.

4 SK9 in mediating the hypercholesterolemia of nephrotic syndrome.

5 eful for predicting relapse in patients with nephrotic syndrome.

6 philia due to a factor VIII autoantibody and nephrotic syndrome.

7 KANK1), KANK2, and KANK4 in individuals with nephrotic syndrome.

8 merular capillary tuft, podocyte injury, and nephrotic syndrome.

9 everity of the acquired hypercoagulopathy of nephrotic syndrome.

10 cytes that is dysfunctional in some types of nephrotic syndrome.

11 a severity, consistent with reports in human nephrotic syndrome.

12 aminonucleoside and adriamycin rat models of nephrotic syndrome.

13 markers or therapeutic targets in persistent nephrotic syndrome.

14 and provide insight into the pathogenesis of nephrotic syndrome.

15 e in podocytes might promote pathogenesis of nephrotic syndrome.

16 deteriorating renal function or untreatable nephrotic syndrome.

17 4 (ADCK4) gene that cause steroid-resistant nephrotic syndrome.

18 ot benefit clinical outcome in children with nephrotic syndrome.

19 ed structure, the disruption of which causes nephrotic syndrome.

20 tal interstitial lung disease and congenital nephrotic syndrome.

21 glomeruli, mimicking the phenotype of human nephrotic syndrome.

22 -based consortium studying steroid-resistant nephrotic syndrome.

23 and of patients with Finnish-type congenital nephrotic syndrome.

24 bitors in children with resistant idiopathic nephrotic syndrome.

25 mulation in podocytes to cellular damage and nephrotic syndrome.

26 rarenal defects but still exhibit congenital nephrotic syndrome.

27 eading to the filtration barrier defects and nephrotic syndrome.

28 podocyte-secreted Angptl4 has a key role in nephrotic syndrome.

29 ases the risk for high-grade proteinuria and nephrotic syndrome.

30 s of proteinuria and rates of progression to nephrotic syndrome.

31 ide (LPS)-treated mice, a model of transient nephrotic syndrome.

32 mg/dl); three (30%) patients presented with nephrotic syndrome.

33 d, lead to inherited FSGS and/or the related nephrotic syndrome.

34 ltrastructural changes and steroid-sensitive nephrotic syndrome.

35 sclerosis as a cause of steroid-unresponsive nephrotic syndrome.

36 fficking plays a role in the pathogenesis of nephrotic syndrome.

37 as to the treatment of steroid-unresponsive nephrotic syndrome.

38 ocytes in a number of experimental models of nephrotic syndrome.

39 increased in patients with steroid-sensitive nephrotic syndrome.

40 ss effacement and lipopolysaccharide-induced nephrotic syndrome.

41 ch model Pierson syndrome, a rare congenital nephrotic syndrome.

42 pathophysiology and treatment mechanisms of nephrotic syndrome.

43 te gene expression during the development of nephrotic syndrome.

44 several months, but they developed a lethal nephrotic syndrome.

45 e pathogenesis of proteinuria and congenital nephrotic syndrome.

46 ted in autosomal recessive steroid-resistant nephrotic syndrome.

47 foot process (FP) effacement and LPS-induced nephrotic syndrome.

48 s are the standard of care for most forms of nephrotic syndrome.

49 lex, and this connection is disrupted in the nephrotic syndrome.

50 in-induced experimental kidney diseases with nephrotic syndrome.

51 agnosis and care of patients with congenital nephrotic syndrome.

52 ycocalyx have been correlated with models of nephrotic syndrome.

53 ns in the gene TBC1D8B in five families with nephrotic syndrome.

54 ypothesized to be the cause of the patient's nephrotic syndrome.

55 docytes increased along with the severity of nephrotic syndrome.

56 s with idiopathic membranous nephropathy and nephrotic syndrome.

57 e in children with primary steroid-resistant nephrotic syndrome.

58 ectively, in children with steroid-resistant nephrotic syndrome.

59 model in which to study the pathogenesis of nephrotic syndrome.

60 ceptors may be novel therapeutic targets for nephrotic syndrome.

61 C, leading to nodular glomerulosclerosis and nephrotic syndrome.

62 th lupus nephritis, diabetic nephropathy, or nephrotic syndrome.

63 mutation is an important cause of hereditary nephrotic syndrome.

64 th frequently relapsing or steroid-dependent nephrotic syndrome.

65 rats with puromycin aminonucleoside-induced nephrotic syndrome.

66 the urine is a common biomarker of a variety nephrotic syndromes.

67 5.7% vs 2.6%; aHR, 2.27; 95% CI, 1.32-3.89), nephrotic syndrome (1.3% vs 0.1%; aHR, 15.7; 95% CI, 2.9

68 Of children with idiopathic nephrotic syndrome, 10%-20% fail to respond to steroids

69 hose patients with available data, 92% had a nephrotic syndrome, 64% had AKI, and 67% had a documente

70 At presentation, 49% had nephrotic syndrome, 68% had renal insufficiency, and 77%

71 logy Damage Index, the greatest frequency of nephrotic syndrome (8.9%) was observed in patients in cl

72 nt in children with glucocorticoid-sensitive nephrotic syndrome, a disorder with minimal known indepe

73 have been identified as monogenic causes of nephrotic syndrome, a frequent cause of CKD.

74 Nephrotic syndrome, a malfunction of the kidney glomerul

75 gene are a major cause of steroid-resistant nephrotic syndrome, a severe human kidney disorder.

76 ed protein (CD2AP) develop renal failure and nephrotic syndrome about 4 weeks after birth and die aro

77 hallmarks of Pierson syndrome are congenital nephrotic syndrome accompanied by ocular abnormalities,

78 Patients with biopsy-proven PMN and nephrotic syndrome after 6 months of nonimmunosuppressiv

79 ranous nephropathy (MN) is a common cause of nephrotic syndrome after transplantation and is associat

80 phrotic syndrome in patients with congenital nephrotic syndrome after transplantation may lead to imp

81 rstanding of recurrent disease in congenital nephrotic syndrome after transplantation.

82 is suggested the highest TCF21 expression in nephrotic syndrome along with the urinary protein level.

83 Glomeruli isolated from mice with nephrotic syndrome also had increased expression of IL-1

84 ldren affected by sporadic steroid-resistant nephrotic syndrome and 38 patients who exhibited a simil

85 obtained from (1) the urine of children with nephrotic syndrome and carrying potentially pathogenic m

86 merulosclerosis (FSGS) is a leading cause of nephrotic syndrome and end-stage renal disease worldwide

87 pathologic lesion that frequently causes the nephrotic syndrome and ensuing renal failure.

88 /d were enrolled at first biopsy for primary nephrotic syndrome and followed.

89 ilial nephropathy in which steroid-sensitive nephrotic syndrome and glomerular deposits of fibrillar

90 erular disorder, manifests clinically with a nephrotic syndrome and has a high propensity for recurre

91 tified in a child with encephalomyopathy and nephrotic syndrome and in a younger sibling with only ne

92 on bilateral transperitoneal nephrectomy for nephrotic syndrome and laparoscopic nephrectomy for Wilm

93 The incidence of nephrotic syndrome and leukopenia was also lower in clus

94 aracterized by early-onset steroid-resistant nephrotic syndrome and microcephaly.

95 phila orthologs of human monogenic causes of nephrotic syndrome and observed conservation of the cent

96 l target for therapeutic intervention in the nephrotic syndrome and other proteinuric diseases.

97 -year-old man with cystic fibrosis developed nephrotic syndrome and progressed to end-stage renal fai

98 a clinicopathologic entity characterized by nephrotic syndrome and progression to ESRD.

99 ng anti-PLA2R antibody levels associate with nephrotic syndrome and progressive loss of kidney functi

100 CD2AP-deficient mice develop nephrotic syndrome and renal failure caused by glomerulo

101 se of a 61-year-old woman who presented with nephrotic syndrome and renal impairment.

102 ive genes have been found to cause inherited nephrotic syndromes and FSGS.

103 Two patients presented with nephrotic syndromes and one patient with an IgG4-related

104 scribe an infant with severe Leigh syndrome, nephrotic syndrome, and CoQ(10) deficiency in muscle and

105 in steroid-dependent or frequently relapsing nephrotic syndrome, and halted disease-associated growth

106 ry diseases; 2.77 (1.49-5.15) for nephritis, nephrotic syndrome, and nephrosis; 4.08 (1.38-12.08) for

107 itis), and inflammatory (glomerulonephritis, nephrotic syndrome, and osteoarthritis) disorders.

108 c approach to sepsis-associated hypotension, nephrotic syndrome, and other sight-threatening and life

109 odel with which to study the pathogenesis of nephrotic syndrome, and ROS formation may be a pathomech

110 vents; partial and complete remission of the nephrotic syndrome; and a composite of doubling of serum

111 Common causes of nephrotic syndrome are diabetic nephropathy, minimal cha

112 naling events contributing to development of nephrotic syndrome are not well defined.

113 the underlying disease, the severity of the nephrotic syndrome (as assessed by serum albumin concent

114 terations in children with sporadic forms of nephrotic syndrome associate with resistance to steroids

115 ers, previous immobility, surgery or trauma, nephrotic syndrome, associated tumor, inflammatory disea

116 nt revealed a renal failure symptomatic of a nephrotic syndrome, associated with proteinuria composed

117 subset of CD2AP(-/-) mice exhibiting severe nephrotic syndrome, associated with systemic illness.

118 nhibitors may be beneficial in patients with nephrotic syndrome-associated hypercholesterolemia.

119 k4 in zebrafish and Drosophila recapitulated nephrotic syndrome-associated phenotypes.

120 lassic pathway and the highest prevalence of nephrotic syndrome at disease onset.

121 hypercoagulopathy to identify patients with nephrotic syndrome at highest risk for thrombotic diseas

122 tion of a subset of patients with congenital nephrotic syndrome at increased risk of recurrence who a

123 ) collagen developed massive albuminuria and nephrotic syndrome, because of subepithelial deposits of

124 -LAMB2 missense mutation leads to congenital nephrotic syndrome but only mild extrarenal symptoms; th

125 orted to reduce proteinuria in patients with nephrotic syndrome, but mechanisms remain unknown.

126 e to FFAs may function in the development of nephrotic syndrome by amplifying the effects of proteinu

127 sults suggest that the R246Q mutation causes nephrotic syndrome by impairing secretion of laminin-521

128 In children, sporadic nephrotic syndrome can be related to a genetic cause, bu

129 Patients with nephrotic syndrome can develop nephrocalcinosis, which a

130 Here, we report a phenotype comprising nephrotic syndrome, cataracts, sensorineural deafness, e

131 patients with congenital red hair color and nephrotic syndrome caused by idiopathic membranous nephr

132 insufficiency syndrome and steroid-resistant nephrotic syndrome caused by loss-of-function mutations

133 It is increasingly clear that nephrotic syndrome caused by monogenic mutations is dist

134 Here, using mouse models of human nephrotic syndrome caused by mutant laminin beta2 protei

135 These data show that diseases that cause nephrotic syndrome change glomerular protein permeabilit

136 l(DeltaPod) mice develop an acute congenital nephrotic syndrome characterized by focal segmental glom

137 Congenital nephrotic syndrome (CNS), a rare entity, is known to pre

138 omal recessive SRNS presenting as congenital nephrotic syndrome (CNS).

139 Lamb2(-/-) mice abrogates the development of nephrotic syndrome, correlating with a greatly extended

140 In nephrotic syndrome, damage to the podocytes of the kidne

141 erosis (FSGS) is a common form of idiopathic nephrotic syndrome defined by the characteristic lesions

142 Requiring that the sum of nephrotic syndrome diagnosis codes exceed that of glomer

143 ests that immunomodulatory therapies used in nephrotic syndrome directly target the podocytes.

144 ding patients with congestive heart failure, nephrotic syndrome, diuretic resistance, or generalized

145 proteinuria, edema and, in steroid-resistant nephrotic syndrome, end-stage kidney disease.

146 were similar to those reported in congenital nephrotic syndrome, Finnish type, that is caused by muta

147 with frequently relapsing steroid-sensitive nephrotic syndrome (FR-SSNS).

148 , together with the zebrafish model of human nephrotic syndrome generated by plce1 knockdown, open ne

149 histomorphometry and sequencing of Mendelian nephrotic syndrome genes were performed.

150 Steroid-resistant nephrotic syndrome has a poor prognosis and often leads

151 se, a major life-threatening complication of nephrotic syndrome, has been associated with proteinuria

152 c, thrombotic microangiopathic syndromes and nephrotic syndrome have been described.

153 However, relevant pathomechanisms of nephrotic syndrome have not been studied in nephrocytes.

154 eral nephrectomy in patients with congenital nephrotic syndrome have permitted transplantation to be

155 search terms included amyloid, amyloidosis, nephrotic syndrome, heart failure preserved ejection fra

156 differential diagnosis of adult nondiabetic nephrotic syndrome; heart failure with preserved ejectio

157 is of structural variation in cases of human nephrotic syndrome identified rare heterozygous deletion

158 anous nephropathy (MN) is a leading cause of nephrotic syndrome in adults and a significant cause of

159 Membranous nephropathy is a common cause of nephrotic syndrome in adults and can be primary or secon

160 nephropathy (MN) is the most common cause of nephrotic syndrome in adults, and one-third of patients

161 d as one of the leading causes of idiopathic nephrotic syndrome in adults, particularly among African

162 phropathy (MN), a relatively common cause of nephrotic syndrome in adults, were considered idiopathic

163 nephropathy (MN) is the most common cause of nephrotic syndrome in adults, with an uncertain clinical

164 ranous nephropathy (MN) is a common cause of nephrotic syndrome in adults.

165 (MCD) is the etiology of 10%-25% of cases of nephrotic syndrome in adults.

166 tic syndrome (SSNS), the most common form of nephrotic syndrome in childhood, is considered an autoim

167 yndrome (SSNS) accounts for >80% of cases of nephrotic syndrome in childhood.

168 is the leading cause of steroid-unresponsive nephrotic syndrome in childhood.

169 Idiopathic nephrotic syndrome in children is commonly associated wi

170 In conclusion, nephrotic syndrome in children with truncating or homozy

171 d mutations in podocyte-specific genes cause nephrotic syndrome in humans.

172 phologic changes indicate the development of nephrotic syndrome in mice bearing the Asah1 podocyte-sp

173 or the alpha3beta1 ligand laminin result in nephrotic syndrome in murine models.

174 SGPL1, displayed a phenotype reminiscent of nephrotic syndrome in nephrocytes.

175 ed cataracts and sensorineural deafness, but nephrotic syndrome in only one case of skewed X-inactiva

176 ievable, would likely lessen the severity of nephrotic syndrome in patients carrying LAMB2 mutations.

177 New findings regarding recurrence of nephrotic syndrome in patients with congenital nephrotic

178 glomerular diseases and induces remission of nephrotic syndrome in patients with diverse glomerulopat

179 tients, and immune-mediated pancytopenia and nephrotic syndrome in the recipient of a double UCB tran

180 target genes, reported as possible causes of nephrotic syndrome, in a cohort of 31 children affected

181 in the epidemiology of steroid-unresponsive nephrotic syndrome, in particular focal segmental glomer

182 The main manifestations of nephrotic syndrome include proteinuria, hypoalbuminemia,

183 ity and mortality associated with protracted nephrotic syndrome, including ESRD.

184 g nephrin lead to proteinuria and congenital nephrotic syndrome, indicating that nephrin is essential

185 -brain barrier integrity was correlated with nephrotic syndrome, indicating that these effects are di

186 of podocyte disorders in cases of idiopathic nephrotic syndrome (INS) are complex and remain incomple

187 Idiopathic nephrotic syndrome (INS) describes a group of pathologie

188 described as unifying feature in idiopathic nephrotic syndromes (INS).

189 multiorgan disorder that included congenital nephrotic syndrome, interstitial lung disease, and epide

190 Nephrotic syndrome is a common disorder in adults and ch

191 In conclusion, nephrotic syndrome is associated with an increased filtr

192 It has long been recognized that nephrotic syndrome is associated with an increased risk

193 Nephrotic syndrome is associated with up-regulation of t

194 Nephrotic syndrome is characterized by increased triglyc

195 Nephrotic syndrome is characterized by massive proteinur

196 Steroid-resistant nephrotic syndrome is characterized by podocyte dysfunct

197 e type of glomerular disease that causes the nephrotic syndrome is necessary for appropriate treatmen

198 nk between proteinuria and hyperlipidemia in nephrotic syndrome is not known.

199 Steroid-unresponsive nephrotic syndrome is often characterized by persistent

200 Nephrotic syndrome is recognized by the presence of prot

201 The pathogenesis of nephrotic syndrome is unclear.

202 membranous nephropathy, a common form of the nephrotic syndrome, is an antibody-mediated autoimmune g

203 tl4 secreted from podocytes in some forms of nephrotic syndrome lacks normal sialylation.

204 is dissociation, wild-type mice with induced nephrotic syndrome maintained an intact blood-brain barr

205 that leak through the abnormal glomerulus in nephrotic syndrome may affect tubular transport by inter

206 pic correlations of patients with congenital nephrotic syndrome may not only modify our understanding

207 eatment for the initial episode of childhood nephrotic syndrome may reduce relapse rate, but whether

208 elper T-cell abnormalities in minimal-change nephrotic syndrome (MCNS), which often complicate allerg

209 ritis, type I membranoproliferative GN), and nephrotic syndrome (minimal change/FSGS, membranous neph

210 nt CGD and DM (n=1), and steroid therapy for nephrotic syndrome (n=1).

211 o heart disease, cancer, diabetes, nephritis/nephrotic syndrome/nephrosis, chronic lower respiratory

212 out of nine individuals), steroid-resistant nephrotic syndrome (nine out of 21 individuals), congeni

213 mutations profile in Brazilian children with nephrotic syndrome (NS) and to determine a genotype-phen

214 The discovery of >60 monogenic causes of nephrotic syndrome (NS) has revealed a central role for

215 Nephrotic syndrome (NS) is a genetically heterogeneous g

216 Nephrotic syndrome (NS) is associated with hyperlipidemi

217 Nephrotic syndrome (NS) is divided into steroid-sensitiv

218 Nephrotic syndrome (NS) occurs when the glomerular filtr

219 Rituximab induces nephrotic syndrome (NS) remission in two-thirds of patie

220 Nephrotic syndrome (NS) represents the association of pr

221 nefits of genetic screening of patients with nephrotic syndrome (NS) to diagnose monogenic causes, re

222 Nephrotic syndrome (NS) was the most common indication f

223 Glucocorticoids are the primary therapy for nephrotic syndrome (NS), but have serious side effects a

224 have been identified as monogenic causes of nephrotic syndrome (NS), but important knowledge gaps ex

225 Patients with nephrotic syndrome (NS), even with normal GFR, often dis

226 Emerging evidence has established primary nephrotic syndrome (NS), including focal segmental glome

227 Nephrotic syndrome (NS), the association of gross protei

228 thyroid function are common in patients with nephrotic syndrome (NS).

229 tion in kidney tubules may vary with time in nephrotic syndrome (NS).

230 ducing remissions of proteinuria, relapse of nephrotic syndrome occurred significantly more often aft

231 of steroid-dependent or frequently relapsing nephrotic syndrome of minimal change disease (MCD), mesa

232 ce of either nephrin (as in human congenital nephrotic syndrome of the Finnish type, NPHS1) or NEPH1.

233 HS1 gene encoding nephrin lead to congenital nephrotic syndrome of the Finnish type.

234 lomerular diseases such as steroid-resistant nephrotic syndrome or Alport syndrome were often the pri

235 rulonephritis codes identified children with nephrotic syndrome or biopsy-based minimal change nephro

236 as either primary or adjunctive therapy for nephrotic syndrome or other diseases treated with glucoc

237 ytopathies associated with steroid-resistant nephrotic syndrome or severe proteinuria.

238 PCSK9 and plasma lipids were studied in nephrotic syndrome patients before and after remission o

239 ckdown of kank2 in zebrafish recapitulated a nephrotic syndrome phenotype, resulting in proteinuria a

240 he model eliminated the association with the nephrotic syndrome (ratio, 0.99; 95 percent confidence i

241 nucleoside-induced injury (designed to mimic nephrotic syndrome-related injury), as determined by bot

242 relative risk 4.79; 95% CI 2.71 to 8.46) and nephrotic syndrome (relative risk 7.78; 95% CI 1.80 to 3

243 iltration, many of the disease mechanisms of nephrotic syndrome remain unresolved.

244 athogenic circulating permeability factor in nephrotic syndrome remains to be confirmed, we propose i

245 boembolic events with oral anticoagulants in nephrotic syndrome requires a careful case-by-case analy

246 Idiopathic nephrotic syndrome resistant to standard treatments rema

247 months later, the patient suddenly developed nephrotic syndrome resistant to steroid therapy 1 week a

248 D in relapse and in remission, patients with nephrotic syndrome resulting from other glomerular disea

249 Nephrotic syndrome results from a breakdown of the kidne

250 odels of glomerular injury and patients with nephrotic syndrome revealed that calpain-induced talin1

251 Steroid-dependent nephrotic syndrome (SDNS) carries a high risk of toxicit

252 cation of patients with steroid-unresponsive nephrotic syndrome, severity of disease, progression and

253 Patients with nephrotic syndrome showed a decrease in plasma cholester

254 ified >55 genes as causing steroid-resistant nephrotic syndrome (SRNS) and localized its pathogenesis

255 racterized by the combination of early-onset nephrotic syndrome (SRNS) and microcephaly with brain an

256 yme Q10 biosynthesis cause steroid-resistant nephrotic syndrome (SRNS) as part of multiorgan involvem

257 Steroid-resistant nephrotic syndrome (SRNS) causes 15% of chronic kidney d

258 Steroid-resistant nephrotic syndrome (SRNS) causes 15% of chronic kidney d

259 s reduced in patients with steroid-resistant nephrotic syndrome (SRNS) due to monogenic disorders.

260 n of single-gene causes of steroid-resistant nephrotic syndrome (SRNS) has furthered the understandin

261 Steroid-resistant nephrotic syndrome (SRNS) is a frequent cause of end-sta

262 Steroid-resistant nephrotic syndrome (SRNS) is a frequent cause of progres

263 Steroid-resistant nephrotic syndrome (SRNS) is characterized by high-range

264 Steroid-resistant nephrotic syndrome (SRNS) is the second most frequent ca

265 Steroid-resistant nephrotic syndrome (SRNS), a frequent cause of chronic k

266 Steroid-resistant nephrotic syndrome (SRNS), a heterogeneous disorder of t

267 cause autosomal recessive steroid-resistant nephrotic syndrome (SRNS).

268 novel monogenic causes of steroid-resistant nephrotic syndrome (SRNS).

269 Steroid-sensitive nephrotic syndrome (SSNS) accounts for >80% of cases of

270 cocorticoid, children with steroid-sensitive nephrotic syndrome (SSNS) have almost normal adult heigh

271 Steroid-sensitive nephrotic syndrome (SSNS), the most common form of nephr

272 trastructural changes (descriptors) from the Nephrotic Syndrome Study Network (NEPTUNE) Digital Patho

273 tasets derived from 90 black subjects in the Nephrotic Syndrome Study Network (NEPTUNE), stratified b

274 ogenic NS genes in 312 participants from the Nephrotic Syndrome Study Network and 61 putative control

275 diseases and in other human pathologies with nephrotic syndromes such as HIV and Hantavirus infection

276 lacking B7-1 are protected from LPS-induced nephrotic syndrome, suggesting a link between podocyte B

277 f rituximab, a B cell-depleting antibody, in nephrotic syndrome suggests a pathogenic role of B cells

278 treatment strategy for those cases of human nephrotic syndrome that are caused by a primary dysfunct

279 his the first report of a molecular cause of nephrotic syndrome that may resolve after therapy.

280 al pattern frequently found in patients with nephrotic syndrome that often progress to end-stage kidn

281 s central to proteinuric states, such as the nephrotic syndrome, the murine adriamycin nephrosis mode

282 ldren (9 months to 17 years) presenting with nephrotic syndrome to either 3 months of prednisolone fo

283 ong the glomerular basement membranes, and a nephrotic syndrome, two additional immunizations were ne

284 trolled trial in 31 children with idiopathic nephrotic syndrome unresponsive to the combination of ca

285 relationships among different components of nephrotic syndrome use naturally occurring pathways and

286 Nephrotic syndrome was reported in a highly-sensitized p

287 CD2AP(-/-) mice normally die by 6 weeks from nephrotic syndrome, we used mice that also express a CD2

288 n during organogenesis results in congenital nephrotic syndrome, whereas VEGF164 overexpression after

289 be single-gene defects-eg, steroid-resistant nephrotic syndrome, which is caused by podocin mutations

290 The etiology of steroid-resistant nephrotic syndrome, which manifests as FSGS, is not comp

291 therapy for patients with steroid-resistant nephrotic syndrome with ADCK4 mutations.

292 epsilon gene (PLCE1) as causing early-onset nephrotic syndrome with end-stage kidney disease.

293 Pierson syndrome is a congenital nephrotic syndrome with eye and neurologic defects cause

294 notypic variance of patients with congenital nephrotic syndrome with nephrin and podocin mutations re

295 ients with first manifestation of idiopathic nephrotic syndrome with no immune complexes can improve

296 cause Pierson syndrome, a severe congenital nephrotic syndrome with ocular and neurologic defects.

297 cause Pierson syndrome, a severe congenital nephrotic syndrome with ocular and neurologic defects.

298 Pierson syndrome is a congenital nephrotic syndrome with ocular and neurological defects

299 THSD7A antibodies into mice induced a severe nephrotic syndrome with proteinuria, weight gain, and hy

300 100 consecutive IMN patients with persistent nephrotic syndrome with rituximab.

301 anous nephropathy is a common cause of adult nephrotic syndrome, with recent evidence suggesting that