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1 ise, sepsis, cardiac surgery, or exposure to nephrotoxins.
2 ESRD risk among cohorts exposed to potential nephrotoxins.
3 ecognition of sepsis as well as avoidance of nephrotoxins.
4 unilateral ureteral obstruction, ischemia or nephrotoxins.
5 , vancomycin dose, and number of concomitant nephrotoxins.
6 ent, adjusting for the receipt of additional nephrotoxins (aHR 1.94; 95% CI, 1.48-2.97).
7  and immune disorders, oxidative stress, and nephrotoxins, among others.
8 ischemia reperfusion, sepsis, or exposure to nephrotoxins and is associated with a high rate of morta
9 ntensive care unit level of care, receipt of nephrotoxins, and hospital, IV vancomycin plus piperacil
10 osphorylation; moreover, they are evident in nephrotoxin- and ischemia/reperfusion-induced injury, di
11 , we injured human kidney organoids with the nephrotoxin cisplatin (5 muM) followed by 5 x 10(4) USC
12 patotoxin acetaminophen (APAP) or the direct nephrotoxin cisplatin.
13 dition, recent information about the classic nephrotoxins (cisplatin, amphotericin B, and aminoglycos
14 ogenase (LDH) release, when exposed to known nephrotoxins, cisplatin and aristolochic acid, which are
15                                              Nephrotoxins contribute to 20%-40% of acute kidney injur
16                         Upon introducing the nephrotoxin, Cyclosporine A, the epithelial barrier is d
17 imals were euthanized three hours after last nephrotoxin dose or at 6 weeks of life.
18         This study suggests that exposure to nephrotoxins during early postnatal nephrogenesis causes
19 I receptor blockers), avoidance of potential nephrotoxins (eg, nonsteroidal anti-inflammatory drugs),
20 lin were developed, each validated using the nephrotoxin gentamicin.
21           KNP-1 is capable of detecting both nephrotoxin-induced and ischemia-reperfusion injury-indu
22                     We used a mouse model of nephrotoxin-induced proteinuric injury to show that prot
23                      Ochratoxin A (OTA) is a nephrotoxin produced by many species in two fungal gener
24         Addition of cisplatin or doxorubicin nephrotoxins reversed the enhanced glucose and albumin u
25 idney injury, which can arise in response to nephrotoxins, sepsis, and ischemia/reperfusion, and in c
26 ting of supportive measures and avoidance of nephrotoxins significantly reduced the occurrence of mod
27 cit adaptive responses following exposure to nephrotoxins, such as cadmium.
28                      Ochratoxin A (OTA) is a nephrotoxin that contaminates grains in storage.
29         Fluoride (F) is a widely distributed nephrotoxin with exposure potentially resulting from env