ライフサイエンスコーパス: nerve conduction study

1 of the wrists with CTS had staging done with nerve conduction study.

2 questionnaire, neurological examination and nerve conduction studies.

3 erties and are complementary to conventional nerve conduction studies.

4 commonly than motor axonal polyneuropathy on nerve conduction studies.

5 ons on behavioral tests as well as normal on nerve conduction studies.

6 tained from history, neurological exams, and nerve conduction studies.

7 s precede functional anomalies detectable on nerve conduction studies.

8 upported by cerebrospinal fluid analysis and nerve conduction studies.

9 ime constant) measured by threshold tracking nerve conduction studies.

10 DPN was defined by symptoms, signs, and nerve conduction study abnormalities in >=2 nerves; CAN

11 cale scores, greater reduction of upper limb nerve conduction study amplitudes, more frequent occurre

12 targets were recorded, and a cross-sectional nerve conduction study analyzing the peroneal, tibial, m

13 ere supported on ancillary testing including nerve conduction studies and electromyography, muscle bi

14 f peripheral nerve lesions routinely involve nerve conduction studies and electromyography.

15 tibialis anterior muscle was confirmed with nerve conduction studies and immunohistochemical analysi

16 Clinical assessments included nerve conduction studies and neuroimaging.

17 and a global symptom assessment, as well as nerve conduction studies and occurrence of new plantar f

18 Nerve conduction studies and pathological analysis revea

19 Typically, in both cohorts, normal nerve conduction studies and reduced intraepidermal nerv

20 l examination, quantitative sensory testing, nerve conduction studies and skin biopsy for intraepider

21 exopathy was made after electromyography and nerve conduction studies and the etiology of radiculopat

22 Among 46 patients in whom nerve-conduction studies and electromyography were perfo

23 using composite neurological grading scales, nerve conduction studies, and assessments of fine motor

24 urological function by clinical examination, nerve conduction studies, and autonomic function tests i

25 ssessment, peripheral nerve ultrasonography, nerve conduction studies, and axonal excitability studie

26 ons in patients with suspected neuropathies, nerve conduction studies, and magnetic resonance imaging

27 n, functional and walking disability scores, nerve conduction studies, and muscle biopsies are report

28 Physical examinations, questionnaires, nerve conduction studies, and NFTs, including the Jebsen

29 ength, tendon reflexes, vibration sense, and nerve conduction studies, and reduced intraepidermal ner

30 ile capacity and performed electromyography, nerve conduction studies, and vastus lateralis biopsies

31 Clinical examinations, nerve conduction studies, and vibratory perception thres

32 Nerve conduction studies are considered essential for a

33 a lesion site precisely, where fractionated nerve conduction studies are not applicable.

34 ited, however, by problems inherent in using nerve conduction studies as a criterion standard.

35 forms (7.7% vs. 5.3%; p=0.63) with a single nerve conduction study as compared with when serial elec

36 Nerve conduction studies assessing median nerve sensory

37 l syndrome should be investigated first with nerve conduction studies but consideration should be giv

38 rical irritability; 13 patients had abnormal nerve conduction studies but four of these had long-stan

39 s of peripheral nerve cannot proceed without nerve conduction studies but particular interest has bee

40 is also disagreement about whether pudendal nerve conduction studies can be used to predict outcome

41 The EQ group scored higher in nerve conduction studies, compared to vehicle-treated db

42 Electromyography and a nerve conduction study corroborated a peripheral axonal

43 sts for the evaluation of the motor unit are nerve conduction studies/electromyography (NCS/EMG) and

44 ropsychological testing remained stable, and nerve conduction studies have shown improvement.

45 arge fiber sensory polyneuropathy on sensory nerve conduction studies in all patients associated with

46 ralysis in 10 percent; in all of the latter, nerve conduction studies indicated an axonal polyneuropa

47 Nerve conduction studies localised abnormalities to the

48 A subset of patients underwent nerve conduction studies (n = 13).

49 Nerve conduction studies (NCS) may be used at the subcli

50 mination, needle electromyography (EMG), and nerve conduction studies (NCS) were performed before and

51 ertaken in 108 type 2 diabetic patients with nerve conduction studies (NCS), HbA(1c) levels, and tota

52 Neurophysiological measures included nerve conduction studies (NCS), quantitative sensory tes

53 Peripheral neuropathy was quantified via nerve conduction studies (NCS), specifically sural senso

54 Nerve conduction study (NCS) abnormalities suggestive of

55 The nerve conduction study (NCS) and electromyography (EMG)

56 Toxicity assessment included nerve-conduction studies (NCS) and radionuclide assessme

57 l patients were followed longitudinally with nerve conduction studies (NCSs) of peripheral motor (uln

58 Nerve conduction studies, needle electromyography, singl

59 Neuroimaging, nerve conduction studies, neurological examinations, and

60 ng neuropathy symptom and disability scores, nerve conduction studies, or quantitative sensory tests.

61 cal clinical presentations, demyelination on nerve conduction studies (p = 0.0005), and difficult ide

62 luded intraepidermal nerve fiber density and nerve conduction study parameters.

63 rs were studied, using clinical examination, nerve conduction studies, perimetry, optical coherence t

64 Nerve conduction studies, quantitative sensory testing,

65 version 2 (CMTNSv2), CMTNSv2-Rasch modified, nerve conduction studies, quantitative sensory testing,

66 Nerve conduction studies, quantitative sudomotor axon te

67 magnetic stimulation and threshold tracking nerve conduction studies, respectively, although metrics

68 Nerve conduction studies revealed axonal motor neuropath

69 Nerve conduction studies revealed no evidence of neuropa

70 y skin biopsy, quantitative sensory testing, nerve conduction studies, screening of genomic DNA for m

71 Electromyography/nerve conduction studies showed evidence for a mild myop

72 Nerve conduction studies showed normal large fibre senso

73 Serial nerve conduction studies showed progressively reduced am

74 Nerve conduction studies showed severe axonal sensorimot

75 ropathy caused by an H10P MPZ mutation whose nerve conduction studies suggested severe axonal loss bu

76 h measurements may be comparable to those of nerve conduction studies, though their prognostic value

77 e prospectively recruited and underwent both nerve conduction studies to assess the split-hand index

78 s], we used quantitative sensory testing and nerve conduction studies to evaluate the function of lar

79 Nerve conduction studies were also performed, and neurop

80 Nerve conduction studies were normal, but increased jitt

81 In all cases, large fibre nerve conduction studies were normal.

82 Results of nerve conduction studies were retrieved and assessed.

83 Nerve conduction studies were suggestive of a motor pred

84 Blood examination and nerve conduction studies were unremarkable.