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1 glucose metabolism prior to detectable optic nerve degeneration.
2 iency, a disease characterized by peripheral nerve degeneration.
3 on in the retina, but had no effect on optic nerve degeneration.
4  prevent RGC death in eyes with severe optic nerve degeneration.
5 n, but it may be useful as an index of optic nerve degeneration.
6 s of the immune system in glaucomatous optic nerve degeneration.
7 nglion cells that lead to glaucomatous optic nerve degeneration.
8 suggests a role for these molecules in early nerve degeneration.
9 s of the immune system in glaucomatous optic nerve degeneration.
10 cial settings may reflect premature auditory nerve degeneration.
11 t of metabolic disease-associated peripheral nerve degeneration.
12 nvasive techniques for diagnosing peripheral nerve degeneration.
13 T1DM demonstrate early immune activation and nerve degeneration.
14 risk of developing POAG, also modulate optic nerve degeneration among POAG patients, underscoring the
15  aqueous humor drainage, elevated IOP, optic nerve degeneration and blindness.
16 ucoma is characterized by irreversible optic nerve degeneration and is the most frequent cause of irr
17 yl-tRNA synthetase (GlyRS), cause peripheral nerve degeneration and lead to CMT disease type 2D.
18 al ganglion cells (RGCs), resulting in optic nerve degeneration and progressive bilateral central vis
19 ng surgery to avoid nerve injury and monitor nerve degeneration and regeneration after injury.
20 ied the pathological changes associated with nerve degeneration and regeneration in skin and proposed
21 and draw critical parallels to mechanisms of nerve degeneration and regeneration in the CNS and in th
22  irreversible neurotoxicity characterized by nerve degeneration and regeneration.
23 specific receptor CNTFRalpha induced sensory nerve degeneration and retarded regeneration in normal c
24 racterized by acute, severe pain, peripheral nerve degeneration, and autonomic dysfunction after inte
25 on's disease, peripheral nerve injury, optic nerve degeneration, and diabetic neuropathy.
26    Decreased corneal sensitivity and corneal nerve degeneration are observed in WFS.
27 -dependent progression pattern, with sensory nerve degeneration as the early skin nerve pathology.
28  activation of macrophages during peripheral nerve degeneration as well as a role for macrophages in
29 e (NDUFA1), we attempted rescue of the optic nerve degeneration associated with Leber hereditary opti
30 f parameters sensitively detected peripheral nerve degeneration at 1-month and 2-month post-injury, w
31 a disease characterized by progressive optic nerve degeneration, can be prevented through timely diag
32 or cognition may reflect underlying cochlear nerve degeneration (CND).
33 ers display a similar pattern of sympathetic nerve degeneration, correlating with the severity of NAF
34 ocular pressure (IOP) characterized by optic nerve degeneration, cupping of the optic disc, and loss
35                      PNI is characterized by nerve degeneration distal to the site of nerve injury re
36 al role of Sirt6 in preventing RGC and optic nerve degeneration during aging and glaucoma, setting th
37 n RGCs led to progressive RGC loss and optic nerve degeneration during aging, despite normal intraocu
38 ia musculorum mouse, exhibiting rapid spinal nerve degeneration, dystonic movements, and severe ataxi
39 ted to retinal ganglion cell (RGC) and optic-nerve degeneration, for which only mutations in TMEM126A
40 through defects in vascularization and optic nerve degeneration, genetic correlation studies have yie
41 SV-1 KOS-63 strain shows acute keratitis and nerve degeneration in a dose-dependent fashion, demonstr
42 ld be used to sensitively monitor peripheral nerve degeneration in ALS mouse models and ALS autopsy m
43                          However, studies of nerve degeneration in animals-and subsequent breakthroug
44 an degeneration slow (WldS) transgene delays nerve degeneration in both events, indicating a common m
45                                        Optic nerve degeneration in diseases such as glaucoma and mult
46 r afferent synapses and progressive cochlear nerve degeneration in noise-exposed ears with recovered
47 WT mice, highlighting their necessity during nerve degeneration in the peripheral nervous system.
48                                        Optic nerve degeneration is a feature common to diseases with
49              These observations suggest that nerve degeneration is not a prerequisite of vincristine-
50 ased release of phosphoNF-H, a biomarker for nerve degeneration, is significantly reduced by inhibiti
51 y retinal ganglion cell (RGC) loss and optic nerve degeneration leading to irreversible blindness.
52 etected in adult spinal cord, which is where nerve degeneration mainly occurs in humans with HSP-SPG4
53  infiltration and demyelination of the optic nerve, degeneration of retinal ganglion cell bodies had
54 n-derived neurotrophic factor (BDNF) prevent nerve degeneration on the long term?
55                            PCD did not cause nerve degeneration or fibrous encapsulation when implant
56 ntage of the world's population suffers from nerve degeneration or peripheral nerve damage.
57                                   Peripheral nerve degeneration or small fibre neuropathy is generall
58                                       Distal nerve degeneration refers to the process of disintegrati
59 manifests as characteristic cupping or optic nerve degeneration, resulting in visual field loss in pa
60 n disease characterized by progressive optic nerve degeneration that results in irreversible blindnes
61 g a novel role for these cells in peripheral nerve degeneration that spans genotypes.SIGNIFICANCE STA
62                  With pressure-induced optic nerve degeneration, the level of Myoc/Tigr mRNA decrease
63 cated in RGC death during glaucomatous optic nerve degeneration, this study was conducted to determin
64                 Wild-type DED mice exhibited nerve degeneration throughout the corneal epithelium, wh
65  was also associated with 2 markers of optic nerve degeneration (vertical cup:disk ratio and minimal
66 for 3-7 wk, a stage when little RGC or optic nerve degeneration was observed.
67 B p65 activation, on TNF-alpha-induced optic nerve degeneration were determined by counting the numbe
68 al nerves likely mirror different aspects of nerve degeneration, where only intraepidermal nerves app
69 or elucidating the molecular events of early nerve degeneration which might be common to neurodegener
70 ne system in all forms of glaucomatous optic nerve degeneration will facilitate the development of ef