ライフサイエンスコーパス: nerve ending

1 a- or intracellular action at the frog motor nerve ending.

2 nerve, at a bend in the nerve, and at a cut nerve ending.

3 sible target sites for phorbol esters in the nerve ending.

4 to regulate cytoskeletal organization in the nerve ending.

5 s were placed within 10 microns of the motor nerve ending.

6 t all nerves within the CE terminate as free nerve endings.

7 A(1) adenosine receptors at mammalian motor nerve endings.

8 l-epidermal junction, adjacent to peripheral nerve endings.

9 layers of the colon, via different types of nerve endings.

10 ecording of coupled glomus cells and carotid nerve endings.

11 ts differentially innervate the central free nerve endings.

12 TRPA1, an excitatory ion channel on sensory nerve endings.

13 ing capsaicin-sensitive perivascular sensory nerve endings.

14 decreases in calcium currents at mouse motor nerve endings.

15 n were immunolabeled to quantitate epidermal nerve endings.

16 treatment causes RII protein to increase at nerve endings.

17 mediators and activation of afferent sensory nerve endings.

18 wnstream of calcium entry at amphibian motor nerve endings.

19 s then distributed to peripheral cholinergic nerve endings.

20 ta-ntx hinders production of ATP in poisoned nerve endings.

21 sensory nerve distally toward the peripheral nerve endings.

22 orepinephrine into presynaptic noradrenergic nerve endings.

23 d result from a decreased Ca(2+) influx into nerve endings.

24 ing ligand-gated ion channels in presynaptic nerve endings.

25 l as some intraepidermal and free myelinated nerve endings.

26 igh-affinity monoamine uptake into rat brain nerve endings.

27 lting in elevated neurotransmitter levels in nerve endings.

28 -1) or COX-2 produce hyperalgesia in sensory nerve endings.

29 at surrounds the synaptic vesicle cluster in nerve endings.

30 ter axons that appeared to terminate as free nerve endings.

31 stimulating transmitter(s) for chemosensory nerve endings.

32 al properties from their counterparts in the nerve endings.

33 rphology resembles human mechanosensory free nerve endings.

34 itatory nicotinic ACh receptors on the motor nerve endings.

35 ng specific (vanilloid) receptors on sensory nerve endings.

36 naptic opioid receptors in mammalian central nerve endings.

37 carbachol and AVRs had abundant sympathetic nerve endings.

38 epinephrine release assay in rat hippocampal nerve endings.

39 larization-induced entry of Ca2+ into intact nerve endings.

40 both intact and streptolysin-O permeabilized nerve endings.

41 Capsaicin is a specific activator of sensory nerve endings.

42 r of environmental cold in mammalian sensory nerve endings.

43 al stimuli into the depolarization of distal nerve endings.

44 horing protein for PKC epsilon within intact nerve endings.

45 ratory behavior by communicating with nearby nerve endings.

46 th neuromedin B receptors detectable on some nerve endings.

47 ent on PIEZO2 activity within the peripheral nerve endings.

48 and by its unusually high density of sensory nerve endings.

49 ring is largely mediated by nociceptive free nerve endings.

50 uroendocrine epithelial cells, and bronchial nerve endings.

51 -like immunoreactive, thin and varicose free nerve endings.

52 resides, with DC dendrites crossing several nerve endings.

53 ing the basement membrane and branching into nerve endings.

54 dulate their sensitivity directly in sensory nerve endings.

55 logical changes starting at their peripheral nerve endings.

56 loss or selective degeneration of peripheral nerve endings.

57 exclusively transduced by the intraepidermal nerve endings.

58 , where it replicated and infected the nodal nerve endings.

59 1 cannabinoid receptors localized on sensory nerve endings.

60 ctor pili muscles, Merkel cells, and sensory nerve endings.

61 TH-negative neurons that form epidermal-free nerve endings.

62 transducer in nociceptive and thermosensory nerve endings.

63 esults in synaptic abnormalities in auditory nerve endings.

64 structure-function studies of vagal afferent nerve endings.

65 transduce noxious stimuli to nearby mucosal nerve endings(3,6) but involvement of this signalling pa

66 At the same time, HSV invades nerve ending-abutting infected cells and is transported

67 ported to be released from cutaneous sensory nerve endings after hapten application, we determined wh

68 roglia in striatum and they protect dopamine nerve endings against drug-induced nerve terminal damage

69 solitary chemosensory cells, and epithelial nerve endings, all of which regenerate after amputation.

70 Substance P labeled a group of free nerve endings along the outer edge of Eimer's organ, ind

71 On stimulation, sensory nerve endings also release neuromediators into the skin,

72 Pathologic changes in the afferent nerve ending and cell body were evaluated with light and

73 ce of P2 receptor agonists at the peripheral nerve ending and is decreased by the presence of P2 anta

74 ed acetylcholine receptors associated with a nerve ending and were thus considered to form anatomic m

75 apid, transient component observed in 70% of nerve endings and a voltage-activation relationship that

76 e in the ongoing firing activity of TRPM8(+) nerve endings and an increase in basal tearing.

77 onsisting of Merkel cells and Abeta-afferent nerve endings and are localized in fingertips, whisker h

78 e in that they possess properties of central nerve endings and endocrine cells.

79 ody, regulating the excitability of afferent nerve endings and glomus cells (putative chemoreceptor c

80 Coupling between nerve endings and glomus cells was more complex, When a

81 d hypobaric hypoxia did not change the Em of nerve endings and glomus cells.

82 ion channels, which are expressed in sensory nerve endings and in skin, respond to distinct thermal t

83 ssociation with chromaffin cells, occasional nerve endings and macrophages.

84 the branching of diabetes-suppressed sensory nerve endings and regeneration in the diabetic corneas.

85 n of neurotrophin receptors (Trks) at distal nerve endings and retrograde propagation of Trk activati

86 rus 1 invades the nervous system by entering nerve endings and sustaining long-distance retrograde ax

87 firefly lantern in cells interposed between nerve endings and the light-producing photocytes.

88 nsporters 1-5, EAATs1-5) located on both the nerve endings and the surrounding glial cells.

89 escued a significant number of VAChT stained nerve endings and treatment of fish with E(2) alone exhi

90 te in mechanically sensitive ion channels in nerve endings, and hearing in mechanically sensitive ion

91 s increased pulpal NGF, sprouting of sensory nerve endings, and increased immunoreactivity for the se

92 n) stimulates abdominal sympathetic afferent nerve endings, and recently have documented increased co

93 ed into those that target the vagal afferent nerve endings, and those that target neural activity wit

94 lation of mechanoreceptors located in the BR nerve-endings, and modulation of the action potential fr

95 Cutaneous nerve endings are assumed to change due to dermatologica

96 kout mice lacking synapsin I and II, sensory nerve endings are normally developed but not stimulated

97 that control the formation and migration of nerve endings are only beginning to be unraveled.

98 Nasal trigeminal nerve endings are particularly sensitive to oxidants for

99 that Merkel cells rather than Abeta-afferent nerve endings are primary sites of tactile transduction

100 We next asked whether proprioceptive nerve endings are similarly affected in the spinal cord

101 Most TG nerve endings are subepithelial, so this colonization im

102 than in slices, suggesting that presynaptic nerve endings are the primary site of inhibition of upta

103 The effects of capsaicin on sensory nerve endings are well known; however, little is known r

104 Nerve endings arising from two classes of colorectal-pro

105 were able to exclude T cells or sympathetic nerve endings as sources of the injury-modulating catech

106 periodontal ligament had fewer thin branched nerve endings at all ages.

107 epithelial defects, and a reduced density of nerve endings at the center of the cornea.

108 Neurectomy of the intercostal nerve endings at the level of the abdominal wall is an e

109 g the symptomatic phase and after peripheral nerve endings begin to degenerate.

110 mM K+, was 246.1 pS, compared with 213 pS in nerve ending BK channels (P<0.01).

111 Whilst blockade of nerve ending BK channels markedly slowed the repolarizat

112 Activation of C-fiber nerve endings by pressure was attributable to stimulatio

113 ne inhibits ACh release from mammalian motor nerve endings by reducing Ca(2+) calcium entry through v

114 This depolarizes the sensory nerve endings by simultaneously closing M-type potassium

115 of the endogenous Ca2+ buffering capacity of nerve endings by treatment with the mitochondrial Ca2+ u

116 annels or, as is the case at amphibian motor nerve endings, by an effect downstream of Ca(2+) entry.

117 cretion whilst simultaneously decreasing the nerve ending calcium currents that promote evoked releas

118 ng that the distribution of mechanosensitive nerve endings can be inferred by visual inspection of th

119 nd indicate that TRPM3 activation in sensory nerve endings can contribute to neurogenic inflammation.

120 Although free intra-epithelial nerve endings can detect certain lipophilic irritants (e

121 crosstalk; immune cells near the peripheral nerve endings can send signals to the brain with cytokin

122 local kallikrein-kinin system in adrenergic nerve endings capable of generating enough bradykinin to

123 ion in mucosal epithelia and transmission to nerve endings, capsids of herpes simplex virus 1 (HSV-1)

124 mine release in isolated hearts, sympathetic nerve endings (cardiac synaptosomes), and PC12 cells bea

125 ls, and resident cells can overtly stimulate nerve endings, cause long-lasting changes in neuronal ex

126 synaptic depolarizing potential (SDP) in the nerve endings caused by release of an excitatory transmi

127 ble channels in the patch) for cell body and nerve ending channels were similar: 11 vs. 14 mV per e-f

128 (H(3)Rs) are present in cardiac sympathetic nerve endings (cSNE) of animals and humans, where they a

129 otor axons in SOD1(G93A) mice, Ia/II sensory nerve endings degenerate in the absence of obvious alter

130 Baroreceptor nerve endings detect acute fluctuations in arterial pres

131 Spinal afferent nerve endings did not express TH, and lacked the cylindr

132 uctural alteration of MRGPRA3(+) pruriceptor nerve endings directed toward Merkel cells in the settin

133 capsaicin, which depletes SP in the sensory nerve endings, eliminates stress-control differences in

134 d during aging in homogenate, cytosol, and a nerve ending-enriched fraction from the hippocampus.

135 staining showed that alpha7 subunit-positive nerve endings enveloped tyrosine hydroxylase-positive gl

136 faces, after which virions enter innervating nerve endings, eventually establishing lifelong infectio

137 tivity by using NE released from sympathetic nerve endings evolved early in the history of mammals.

138 llowing the activation of peripheral sensory nerve endings following damage or exposure to inflammato

139 erature-sensitive ion channels at peripheral nerve endings for transducing thermal cues into electric

140 During development, each type of nerve endings forms at different time point.

141 2+) exchange, as measured in intact isolated nerve endings from mouse cortex and in intact varicositi

142 via confocal imaging of isolated presynaptic nerve endings from rat hippocampus and neocortex.

143 9, would stimulate group III and IV afferent nerve endings from the hindlimb of the anesthetized cat.

144 s cell was stimulated, current spread to the nerve ending (GC/NE coupling) was similar in magnitude (

145 different mechanisms for oxygen sensing: the nerve endings generate action potentials in association

146 How these nerve endings grow and reach a specific area of the skin

147 Also, when nerve endings had an Em more negative than -40 mV showed

148 ed and stained to visualize single axons and nerve endings immunoreactive to calcitonin gene-related

149 al cell bodies in trigeminal ganglia (TG) to nerve ending in the noses and corneas of infected cattle

150 zed by each other; and (3) DRG neurones with nerve endings in a glycolytic muscle developed greater i

151 likely to be reached at cardiac sympathetic nerve endings in advanced congestive heart failure, prom

152 to protons and capsaicin than did those with nerve endings in an oxidative muscle.

153 One class was peptidergic neurons that had nerve endings in circular muscle, myenteric ganglia, and

154 cytes and nerve fibers showed intraepidermal nerve endings in contact with melanocytes.

155 gh many rows of myenteric ganglia and formed nerve endings in discrete anatomical layers.

156 lcitonin gene-related peptide-positive, free nerve endings in footpad epidermis.

157 t neurons allowing for bright imaging of the nerve endings in living tissues and suitable for structu

158 esent in calretinin-immunoreactive extrinsic nerve endings in mouse and human stomach.

159 nificant alterations at Ia/II proprioceptive nerve endings in muscle spindles before the symptomatic

160 However, analysis of proprioceptive nerve endings in muscles revealed early and significant

161 Furthermore, encapsulated nerve endings in Pacinian corpuscles also contain reacti

162 The first state directs virus access to nerve endings in peripheral tissue, whereas the second d

163 oteins are found on microvesicles in sensory nerve endings in peripheral tissues.

164 of rapidly adapting nodose ganglion-derived nerve endings in response to mechanical stimuli.

165 Arterial baroreceptors are mechanosensitive nerve endings in the aortic arch and carotid sinus that

166 Our aim was to identify the sensory nerve endings in the colon that arise from single colore

167 es structural plasticity of nociceptive free nerve endings in the epidermis and inflammatory hyperalg

168 e ATP is coreleased with NE from sympathetic nerve endings in the heart, we investigated whether ATP

169 pH 6.7 are ASIC(3)-like in DRG neurons with nerve endings in the hindlimb muscles, (2) a greater aci

170 amage synovial tissues and can activate free nerve endings in the joint.

171 y M2 muscarinic receptors on parasympathetic nerve endings in the lungs decrease release of acetylcho

172 phological identification of spinal afferent nerve endings in the mammalian urinary bladder.

173 and their release from meningeal trigeminal nerve endings in the mechanism of migraine, blockade of

174 fficiently transported retrogradely from the nerve endings in the nose and eye to cell bodies in the

175 ircular and longitudinal muscle cells and to nerve endings in the plexuses.

176 regulates density and maintenance of sensory nerve endings in the skin and may have important roles i

177 Touch sensitivity in animals relies on nerve endings in the skin that convert mechanical force

178 sults from the excitation of primary sensory nerve endings in the skin, but the underlying molecular

179 soma and along nonmyelinated C-fibers and at nerve endings in the skin.

180 -Kit, is required to maintain the density of nerve endings in the skin.

181 Also, the rapid adaptation of nodose nerve endings in the trachea observed during a mechanica

182 hat BC signaling stimulates adjacent sensory nerve endings in the trachea to release the neuropeptide

183 y distinct subsets of mechanically sensitive nerve endings in the trachea/bronchus.

184 ed this approach to identify spinal afferent nerve endings in the upper GI tract of mice.

185 irst detailed description of spinal afferent nerve endings in the uterus of a vertebrate.

186 ization of TREK/TRESK at the nociceptor free nerve endings in which their acute inhibition is suffici

187 Our results suggest that the free nerve endings innervating Eimer's organ are largely mech

188 n channel, was present at the vagus afferent nerve endings innervating the aortic arch to function as

189 esicles, and rates of hormone secretion from nerve endings into the blood and from dendrites into the

190 The term free nerve ending is perhaps an insufficient and inaccurate d

191 muscarinic receptors on the parasympathetic nerve endings is likely to contribute to increased acety

192 way in which it excites nociceptive sensory nerve endings is still unclear.

193 ng glial cells present at somatic peripheral nerve endings, known collectively as terminal Schwann ce

194 Sensory nerve endings located close to the lumen of the GI tract

195 Significant nerve-ending loss occurs before a decrease in cell bodie

196 a majority of PACAP-LI, VIP-LI and VAChT-LI nerve endings making putative synaptic contact onto IMG

197 ]i regulatory properties of neurohypophysial nerve endings may explain both the depletion of peptide

198 that endogenous CGRP concentrated in sensory nerve endings may regulate locally the immune response,

199 e activity represents a direct effect on the nerve ending, mediated by P2X receptors, whereas the lat

200 endent uptake into presynaptic noradrenergic nerve endings, mediated by the norepinephrine transporte

201 terminating in peripheral zones that contain nerve endings mediating distinct perceptions of innocuou

202 Ca(V)2.2 channels at peripheral nerve endings might be important therapeutic targets to

203 es (PCs) are tactile receptors composed of a nerve ending (neurite) that is encapsulated by layers of

204 The surprising coexistence under one nerve ending of separate clusters of receptors that resp

205 s correlated with a block of Ca ion entry at nerve endings of csp mutants.

206 d demonstrate KCNQ3 expression in peripheral nerve endings of cutaneous D-hair follicles.

207 Given that the nerve endings of enteric neurons terminate at the basola

208 showed intense CGRP immunoreactivity in pulp nerve endings of mutant mice, compared with a gradual de

209 Nerve endings of nociceptors (pain-sensing neurons) expr

210 ll processes to form encapsulated lanceolate nerve endings of rapidly adapting mechanoreceptors.

211 The nerve endings of rat neurohypophyses were acutely dissoc

212 IC, in several different specialized sensory nerve endings of skin, suggesting it might participate i

213 morphological differences between peripheral nerve endings of small and large fibers.

214 ation, morphology, and neurochemistry of the nerve endings of spinal afferents that actually detect t

215 The individual nerve endings of spinal afferents that innervate the uri

216 ), the morphology and location of peripheral nerve endings of spinal afferents that transduce sensory

217 , these results predict that if one depletes nerve endings of synaptic vesicles, one may see a reduct

218 e shown that the VR1 is expressed on sensory nerve endings of the heart.

219 d also in muscle tissue, probably around the nerve endings of the neuromuscular junction.

220 ct and streptolysin-O permeabilized isolated nerve endings of the rat neurohypophysis were studied.

221 by depolarizing stimuli at single, isolated nerve endings of the rat neurohypophysis.

222 tamine causes long-term toxicity to dopamine nerve endings of the striatum.

223 us system (brain, spinal cord and peripheral nerve endings) of behaving mice.

224 e direct stimulation of intraepithelial free nerve endings or indirectly through information transmis

225 eby contributing to the long-held concept of nerve endings passing freely between keratinocytes.

226 lcitonin gene-related peptide from cutaneous nerve endings plays a key role in the local immune aberr

227 volves local changes in the skin or its free nerve endings, possibly leading to peripheral neuropathy

228 sed vesicular [(3)H]glutamate content in the nerve ending preparation synaptosome; this decrease was

229 ratinocyte communication with intraepidermal nerve endings remains poorly understood.

230 interaction with the CIRL receptor on these nerve endings resulted in ionic pore formation, generati

231 TE that, in turn, activates TRPV1 on C-fiber nerve endings resulting in depolarization of nerves and

232 this setting, decreased pH(i) in sympathetic nerve endings sequentially leads to a compensatory activ

233 Secretory responses in intact nerve endings showed AA-induced secretion to be sustaine

234 was dependent on the resting [Ca2+]i and the nerve ending size, and was depletable using repetitive d

235 ushy cells of the AVCN receive huge auditory nerve endings specialized for high fidelity neural trans

236 rofilament 200 labeled a more central set of nerve endings, suggesting that these fibers function as

237 pidermal junction next to peripheral sensory nerve endings, suggesting that viral reactivation may oc

238 We previously demonstrated that nodose nerve endings supplying the trachea are exquisitely mech

239 -HT), and norepinephrine (NE) into rat brain nerve endings (synaptosomes) were evaluated.

240 polarization of guinea pig heart sympathetic nerve endings (synaptosomes) with 1 to 100 mmol/L K+ cau

241 ' UTR, is located both in cell bodies and at nerve endings (synaptosomes).

242 -HT), and norepinephrine (NE) into rat brain nerve endings (synaptosomes).

243 5-HT) and norepinephrine (NE) into rat brain nerve endings (synaptosomes).

244 y affected in the spinal cord and found that nerve endings terminating on alpha-motor neurons are aff

245 s, there are selective C or Adelta epidermal nerve endings that are pruriceptors.

246 s with specialized mechanoreceptors and free nerve endings that connect the cementum of the tooth roo

247 blood pressure by stimulating renal sensory nerve endings that contain synapsin-positive microvesicl

248 ctions of alpha-latrotoxin on neuroendocrine nerve endings that emanate from central nervous system n

249 selectively label spinal afferent (sensory) nerve endings that innervate the periosteum and marrow c

250 onal properties of identified mechanosensory nerve endings that innervate whisker follicles.

251 is function, we found BNC1 in the lanceolate nerve endings that lie adjacent to and surround the hair

252 IFICANCE STATEMENT The skin is full of small nerve endings that sense different environmental stimuli

253 TRPM8, a cation channel expressed in sensory nerve endings that serves as the primary cold sensor in

254 n PGP 9.5 immunoreactive intraepidermal fine nerve endings that were normalized after grate removal.

255 tly induced an increase in [Ca2+]i in intact nerve endings, the AA-induced secretory response was lar

256 Through activation of peripheral nerve endings, the release of neurotransmitters, hormone

257 nsmission of mechanical forces to nociceptor nerve endings thereby reducing pain.

258 te) to the skin activates underlying sensory nerve endings, thereby producing pain, inflammation and

259 By entering nerve endings, they cleave and inactivate SNARE proteins

260 ents terminate in the skin as seemingly free nerve endings, they detect sustained pressure, transient

261 2+)-dependent exocytosis in neurohypophysial nerve endings through receptor interaction and insertion

262 al stimulation to progress from the auditory nerve ending to the inferior colliculus, was significant

263 tegies that target TRPA1 channels on sensory nerve endings to achieve chemical deterrence.

264 e relocation of activated Trk receptors from nerve endings to cell bodies is required for nuclear sig

265 in Merkel cells, which drive Abeta-afferent nerve endings to fire slowly adapting impulses.

266 t least in part, by sensitizing the afferent nerve endings to ischaemia.

267 cipal neurons, colocalizing with glycinergic nerve endings to mediate fast, phasic IPSCs.

268 f pain by increasing the response of sensory nerve endings to noxious stimuli.

269 via transport of FIV vectors from peripheral nerve endings to sensory ganglia, as evidenced by HuMOR

270 ile TeNT retrograde traffics from peripheral nerve endings to the interneuronal junction, there is li

271 y causing the release of CGRP from cutaneous nerve endings, triggers mast cell release of TNF-alpha,

272 at carotid body type I cells and the apposed nerve endings use different mechanisms for oxygen sensin

273 ngly, a significantly reduced number of free nerve endings was detected in glabrous skin from SNS-gp1

274 Mechanosensitivity of the nerve endings was quantified using calibrated von Frey f

275 R) causes CGRP to be released from cutaneous nerve endings, we examined whether CGRP participates in

276 Nerve endings were also bidirectionally and capacitively

277 Substance P-like immunoreactive free nerve endings were also present in the luminal syringeal

278 The majority of spinal afferent nerve endings were CGRP-immunoreactive.

279 Most FFA3-immunoreactive nerve fibres and nerve endings were cholinergic, colocalized with protein

280 Anterograde-labeled nerve endings were dispersed throughout the periosteum a

281 s at the membrane surface in real samples of nerve endings were estimated.

282 Less commonly, nerve endings were identified in internodal strands, blo

283 tinct morphological types of spinal afferent nerve endings were identified throughout multiple anatom

284 Most nerve endings were located in detrusor muscle where the

285 entials arriving from single airway afferent nerve endings were monitored extracellularly using a gla

286 entials arriving from single airway afferent nerve endings were monitored extracellularly using a gla

287 Synaptophysin-positive nerve endings were observed in close apposition to red p

288 the cell body whereas type II is enriched at nerve endings where it is bound to two prominent A kinas

289 a6 subunits are typically found at aminergic nerve endings where they play important roles in nicotin

290 an be found in close proximity to peripheral nerve endings where, upon activation, they release a bro

291 ive effect starts after entering the sensory nerve endings, where these agents are axonally transport

292 the outer, substance P-positive set of free nerve endings, whereas several afferents differentially

293 Different morphological types of nerve endings which innervate different anatomical layer

294 , opioids, and ions excite/sensitize sensory nerve endings, which not only induces itch but further a

295 group, we blocked 5-HT3 receptors on sensory nerve endings with tropisetron (300 microg kg(-1), I.V.)

296 estruction of both sphincteric myofibers and nerve endings, with a functional incapacity of the damag

297 ly assumed to be located exclusively on free nerve endings within the nasal epithelium, requiring tha

298 s of these sensory neurons terminate as free nerve endings within the oral epithelium.

299 Schwann cells also surround mechanoreceptor nerve-endings within the Meissner's corpuscle and in hai

300 ion of RTX transiently disrupted nociceptive nerve endings, yielding reversible analgesia.