ライフサイエンスコーパス: nerve fibre

1 neuropathy and an increase in intraepidermal nerve fibre.

2 inated C-fibres and thinly myelinated Adelta nerve fibres.

3 ion of ASIC1 channels on capsaicin-sensitive nerve fibres.

4 sm for physiological tuning of thermosensory nerve fibres.

5 n excites cutaneous group III and IV sensory nerve fibres.

6 s associated with recovery of intraepidermal nerve fibres.

7 eurons arises from myelinated or umyelinated nerve fibres.

8 signals are conveyed onto auditory afferent nerve fibres.

9 d was observed mainly along GBSM bundles and nerve fibres.

10 arge population of nociceptive-like afferent nerve fibres.

11 on (SG), stria vascularis (SV), and afferent nerve fibres.

12 re closely apposed to substance P-containing nerve fibres.

13 nduce neurotransmitter release onto auditory nerve fibres.

14 ctivates not only efferent but also afferent nerve fibres.

15 chain (smMHC) but are in close apposition to nerve fibres.

16 eneration and transmission in CNS myelinated nerve fibres.

17 nd the sound-evoked activity of the auditory nerve fibres.

18 rae of Schwann cells related to unmyelinated nerve fibres.

19 due to earlier remyelination of demyelinated nerve fibres.

20 release of transmitter from post-ganglionic nerve fibres.

21 he heart and in pain-sensitive (nociceptive) nerve fibres.

22 that activates 5-HT(3) receptors on sensory nerve fibres.

23 and ATP and consequent excitation of sensory nerve fibres.

24 anical allodynia, and loss of intraepidermal nerve fibres.

25 tect the effects of ischaemia on sympathetic nerve fibres.

26 ted ischaemic from non-ischaemic sympathetic nerve fibres.

27 al activation of nociceptive primary sensory nerve fibres.

28 d show that there are no discrete inhibitory nerve fibres.

29 ared not to be associated with noradrenaline nerve fibres.

30 e-sensitive auditory neuropathy) or auditory nerve fibres.

31 of both slowly and rapidly adapting afferent nerve fibres.

32 iated with at least one substance P-positive nerve fibre, 32% were associated with at least two, 2% w

33 s nerve biopsies revealed loss of myelinated nerve fibres (86%), increased regenerative clusters (50%

34 s have been documented in lung vagal sensory nerves fibres, a rigorous comparison of their expression

35 ents, indicating that they were dependent on nerve fibre activation.

36 The first recordings of vagal afferent nerve fibre activity were performed by Paintal in the ea

37 MPTP lesion in mice, and of lesioned sciatic nerve fibres after nerve crush in rats.

38 t two, 2% were associated with more than two nerve fibres and 1% with none.

39 irt and P2X3 receptor co-localize in bladder nerve fibres and heterologous Pirt expression significan

40 ut from low/medium spontaneous rate auditory nerve fibres and medial olivocochlear neurons.

41 Most FFA3-immunoreactive nerve fibres and nerve endings were cholinergic, colocal

42 potential amplitude, loss of intraepidermal nerve fibres and significant degeneration of myelinated

43 nglioside and Gal(beta1-3)GalNAc moieties in nerve fibres and their relationship to voltage-gated sod

44 by the activation of small diameter afferent nerve fibres and therapeutic effects on the associated v

45 egeneration, remyelination of severed spinal nerve fibres, and functional recovery.

46 oked part of the physiological repertoire of nerve fibres, and here they are interpreted in terms of

47 patially associated with HSCs and adrenergic nerve fibres, and highly express HSC maintenance genes.

48 width dependence similar to that of auditory nerve fibres, and yield significantly greater coding eff

49 In mammals, several auditory nerve fibres (ANFs) carry information from each inner ha

50 tion in sound sensitivity of mature auditory nerve fibres appears to be primed at pre-hearing ages.

51 ightly myelinated and unmyelinated autonomic nerve fibres are selectively targeted.

52 It is concluded that these sympathetic nerve fibres are sensitive to ischaemia, and that VRCs p

53 n studies and skin biopsy for intraepidermal nerve fibre assessment.

54 toreceptor distributions(3,4) and individual nerve fibre bundles(5) in the living human retina.

55 d not only by widespread loss of myelin from nerve fibres, but also by widespread inflammation in the

56 ise to V-shaped tuning functions in auditory nerve fibres, but by the level of the inferior colliculu

57 coincident activation of groups of auditory nerve fibres by broadband transient sounds, compensating

58 Activation of afferent vagus nerve fibres by endotoxin or cytokines stimulates hypoth

59 tivation of cardiac parasympathetic efferent nerve fibres by stimulation of the cervical vagus is ass

60 Sensory nerve fibres can detect changes in temperature over a re

61 re, the distribution patterns of PROKR2(Cre) nerve fibres can retrospectively predict body regions at

62 y by direct electrical stimulation of tibial nerve fibres, confirming that centrally mediated mechani

63 citability of the shortest latency (fast, F) nerve fibres, consistent with hyperpolarization.

64 automated corneal nerve analysis identifies nerve fibre damage and may act as a biomarker for neurod

65 ly CIPN stage, to assess the extent of large nerve fibre damage and to monitor long-term outcomes, wi

66 Obese participants with small nerve fibre damage had higher serum triglycerides (P = 0

67 copy (CCM) to quantify the severity of small nerve fibre damage in relation to the severity of neurop

68 Small nerve fibre damage occurs in people with severe obesity.

69 Obese patients with evidence of small nerve fibre damage, compared to those without, had signi

70 en obese participants with and without small nerve fibre damage.

71 Although multiple causes of small nerve fibre degeneration have been reported, including v

72 cochlear damage and the subsequent auditory nerve fibre degeneration.

73 Intraepidermal nerve fibre densities at the index finger were not signi

74 and vibration perception, but lower corneal nerve fibre density (20.1 +/- 0.87 vs. 24.13 +/- 0.91, P

75 rve fibre length (CNFL) (P < 0.001), corneal nerve fibre density (CNFD) (P = 0.025) and corneal nerve

76 onfocal microscopy (CCM) to quantify corneal nerve fibre density (CNFD), branch density (CNBD) and fi

77 e function, neurophysiology, intra-epidermal nerve fibre density (IENFD) and corneal confocal microsc

78 Corneal nerve fibre density (mean difference: - 5.00 no/mm(2), 9

79 quantitative sensory testing, intraepidermal nerve fibre density (thigh), computerised myometry (lowe

80 evealed a partial recovery of intraepidermal nerve fibre density [fibres/mm epidermis pre: 4.20 (2.83

81 back were immunostained and imaged to assess nerve fibre density and neuropeptide content.

82 quantitative sensory testing, intraepidermal nerve fibre density and serum neurofilament light chain

83 vealed in six of them reduced intraepidermal nerve fibre density consistent with small fibre neuropat

84 h a significant reduction in intra-epidermal nerve fibre density in plantar hindpaw skin, and produce

85 3 months, but a reduction in epidermal skin nerve fibre density manifested only at 6 months.

86 Evaluation of intraepidermal nerve fibre density showed a striking loss in patients (

87 was assessed by the return of intraepidermal nerve fibre density through regenerative regrowth.

88 Epidermal nerve fibre density was found to be profoundly reduced i

89 The intraepidermal nerve fibre density was markedly reduced with 90.5% of p

90 Nerve fibre density was not altered in the circular musc

91 Intra-epidermal nerve fibre density was significantly reduced compared t

92 d in rectal sensory fibres, and to correlate nerve fibre density with sensory abnormalities.

93 C and there is a reduction in intraepidermal nerve fibre density, comparable to that seen in herpes z

94 lead to a clear reduction in intraepidermal nerve fibre density, which was independent of electrodia

95 wed a significant decrease in intraepidermal nerve fibre density.

96 om the trunk neural tube and associated with nerve fibres, differentiate into neurons within the gut

97 Duodenal vagal afferent nerve fibre discharge was significantly increased by clo

98 A rich supply of nerve fibres displaying immunoreactivity against nitric

99 ecordings were made of afferent discharge in nerve fibres dissected from the vagus nerve, which respo

100 y drive spontaneous discharge in nociceptive nerve fibres during inflammation.

101 s kindred, and suggest that small peripheral nerve fibre dysfunction due to this mutation may have co

102 on model and to define the rate of epidermal nerve fibre (ENF) regeneration first in healthy control

103 Both types of nerve fibres expressed substance P, but only non-vessel-

104 from increased numbers of polymodal sensory nerve fibres expressing TRPV1.

105 cultured sensory neurons and intact sensory nerve fibres from TRPM8-deficient mice exhibit profoundl

106 ory testing measures of both small and large nerve fibre function (P < 0.002).

107 ) DN underwent assessment of large and small nerve fibre function, CCM, neuropathic symptoms (small f

108 to accelerate and guide the formation of new nerve-fibre growth.

109 past 5 years that extensive arborization of nerve fibres has a dominant role in regulating the funct

110 w, brain and spinal cord injuries that sever nerve fibres have resulted in a degree of incurable func

111 equency response of the innervating auditory nerve fibres However, the data supporting these concepts

112 coding on the central recipients of auditory nerve fibres, i.e. the cochlear nucleus neurons.

113 slips of a cervical vagus were dissected and nerve fibres identified that displayed discharge pattern

114 inked immunosorbent assay (ELISA) and teased nerve fibre immunohistochemistry confirmed reactivity ag

115 In rectal hypersensitivity, nerve fibres immunoreactive to TRPV1 were increased in m

116 pR(+)) stromal cells is required to maintain nerve fibres in adult bone marrow.

117 ofilament staining confirmed the presence of nerve fibres in both regions.

118 etect membrane depolarization of sympathetic nerve fibres in human patients when autonomic neuropathy

119 ests that adipocytes signal to local sensory nerve fibres in response to perturbations in lipolysis a

120 We identified nerve fibres in the outer third of the annulus fibrosus

121 -induced sweating, and prevented the loss of nerve fibres in the skin and reduction of neuropeptide c

122 s in affected skin correlated with decreased nerve fibres in the subepidermis, e.g. axon-reflex flux

123 onal membrane potential of human sympathetic nerve fibres in vivo.

124 hanisms of action include desensitization of nerve fibres (in the case of capsaicin) and postsynaptic

125 e of the epidermis, the tissue hosting small nerve fibres, in a deeply phenotyped cohort of patients

126 uation of non-myelinated, but not myelinated nerve fibres, in sensory neuropathies.

127 annel TRPA1 is expressed by primary afferent nerve fibres, in which it functions as a low-threshold s

128 rly half of bronchopulmonary nodose afferent nerve fibres, including nodose C-fibres, as detected by

129 osites along excitatory and inhibitory motor nerve fibres increased and decreased respectively, leadi

130 inhibitors prevented loss of intraepidermal nerve fibres induced by paclitaxel and provided partial

131 Additional findings have revealed that nerve fibres infiltrating peripheral tumours can release

132 application can produce a uniform epidermal nerve fibre injury that is safe and well tolerated, and

133 lnerable medium- and high-threshold-auditory nerve fibres innervate various cell types in the cochlea

134 lpha-synuclein aggregations are also seen in nerve fibres innervating the gastro-intestinal tract.

135 Immunohistochemical studies localize P2X3 to nerve fibres innervating the urinary bladder of wild-typ

136 Single nerve fibres innervating tooth pulp were isolated from f

137 cycles can distinguish whether a sympathetic nerve fibre is depolarized or not.

138 thickness (IMT) and circumpapillary retinal nerve fibre layer (cpRNFL); and OCT angiography measurem

139 Region specific and mean nerve fibre layer (NFL) thicknesses, border NFL and peri

140 Mean thickness of peripapillary retinal nerve fibre layer (pRNFL) and macular ganglion cell and

141 We measured peripapillary retinal nerve fibre layer (pRNFL) and macular ganglion cell-inne

142 GCIPL) was superior to peripapillary retinal nerve fibre layer (pRNFL) in predicting all visual outco

143 examining BMO-MRW and peripapillary retinal nerve fibre layer (pRNFL) readings acquired with Spectra

144 he correlation between peripapillary retinal nerve fibre layer (pRNFL) thickness and glaucomatous vis

145 e assessed the role of peripapillary retinal nerve fibre layer (pRNFL) thickness and macular volume i

146 lar thickness (MT) and peripapillary retinal nerve fibre layer (pRNFL) thickness were assessed.

147 Retinal nerve fibre layer (RNFL) and ganglion cell layer (GCL) t

148 l retinal thickness, circumpapillary retinal nerve fibre layer (RNFL) and inner plexiform ganglion ce

149 study to evaluate the correlation of retinal nerve fibre layer (RNFL) and macular thickness with seru

150 the analysis of the thickness of the retinal nerve fibre layer (RNFL) by optical coherence tomography

151 We analysed retinal nerve fibre layer (RNFL) defects in eyes with normal cir

152 Retinal nerve fibre layer (RNFL) is a non-invasive structural bi

153 The retinal nerve fibre layer (RNFL) is composed largely of unmyelin

154 reoscopic optic nerve head (ONH) and retinal nerve fibre layer (RNFL) photography and imaging with Sc

155 with control eyes, the peripapillary retinal nerve fibre layer (RNFL) showed thinning in MSON eyes (m

156 Retinal nerve fibre layer (RNFL) thickness has been associated w

157 The primary outcome was retinal nerve fibre layer (RNFL) thickness in the affected eye a

158 Retinal nerve fibre layer (RNFL) thickness is related to the axo

159 ve also found that, in older adults, retinal nerve fibre layer (RNFL) thickness is significantly thin

160 o corresponding localised regions of retinal nerve fibre layer (RNFL) thickness measured by optical c

161 ual field (HVF 24-2) testing, 2D OCT retinal nerve fibre layer (RNFL) thickness measurements, and 3D

162 earning (DL) algorithm for measuring retinal nerve fibre layer (RNFL) thickness on spectral-domain op

163 Changes in retinal nerve fibre layer (RNFL) thickness, macular volume and r

164 EAD) were calculated for the macular retinal nerve fibre layer (RNFL), ganglion cell inner plexiform

165 is (MS) by capturing thinning of the retinal nerve fibre layer (RNFL).

166 f the total retina (most subfields), retinal nerve fibre layer (RNFL; outer ETDRS ring), ganglion cel

167 llecting data from the peripapillary retinal nerve fibre layer (RNLF) and the macular region.

168 The relationship between nerve fibre layer anatomy and the pattern visual evoked

169 Changes were seen not only in the retinal nerve fibre layer and ganglion cell layer, but also in t

170 ociated with reduced apoptosis and increased nerve fibre layer and inner plexiform layer thicknesses.

171 oexisting anatomical changes such as retinal nerve fibre layer and macular thinning, measured using o

172 ce of retrograde degeneration of the retinal nerve fibre layer and to ascertain if such patients may

173 Although these confirm the damage to retinal nerve fibre layer beyond what is detected by standard vi

174 f the thickness of the peripapillary retinal nerve fibre layer by optical coherence tomography has be

175 to define the temporal evolution of retinal nerve fibre layer changes and to estimate sample sizes f

176 The evolution of retinal nerve fibre layer changes in the affected eye fitted wel

177 ned the visual field scotoma and the retinal nerve fibre layer defect in the corresponding area.

178 ifying optic nerve and peripapillary retinal nerve fibre layer defects, with different efficacy and l

179 onfirm that there is thinning of the retinal nerve fibre layer following both congenital and acquired

180 rst time progressive thinning of the retinal nerve fibre layer following occipital lobe/optic radiati

181 e and mild disc oedema, temporal pallor, and nerve fibre layer haemorrhages inferiorly in the left ey

182 Thinning of the peripapillary retinal nerve fibre layer has been detected in patients with opt

183 n optic neuritis, and in imaging the retinal nerve fibre layer in both optic neuritis and multiple sc

184 s applied, OCT-derived peripapillary retinal nerve fibre layer inter-eye differences of 6 mum or grea

185 ptical coherence tomography-measured retinal nerve fibre layer loss after 6 months is a suitable outc

186 cept trials of neuroprotection using retinal nerve fibre layer loss as the outcome measure.

187 ymptom onset and the rate of ongoing retinal nerve fibre layer loss decreasing thereafter.

188 ciated with the extent of concurrent retinal nerve fibre layer loss.

189 visual dysfunction beyond that explained by nerve fibre layer loss.

190 affected eyes, whereas peripapillary retinal nerve fibre layer oedema was observed in affected eyes (

191 s not related to the extent of acute retinal nerve fibre layer swelling but was significantly associa

192 mpaired axonal transport (implied by retinal nerve fibre layer swelling) are associated with visual d

193 (Rho=-0.536, p<0.001), peripapillary retinal nerve fibre layer thickness (B=0.70, p<0.001) and visual

194 inferior quadrants and total macular retinal nerve fibre layer thickness (mRNFL) and macular ganglion

195 Retinal nerve fibre layer thickness (RNFL) was reduced in compar

196 primary outcome is the peripapillary retinal nerve fibre layer thickness (RNT) in microns.

197 ssion r = 0.54, P < 0.001) was found between nerve fibre layer thickness and elapsed time since injur

198 's disease hyperspectral profile and retinal nerve fibre layer thickness in advanced aging.

199 line relationship was found between time and nerve fibre layer thickness in micrometres over a period

200 The retinal nerve fibre layer thickness may be used as a biological

201 g lesions of the occipital lobe, the retinal nerve fibre layer thickness measured by optical coherenc

202 fected and affected eyes rather than retinal nerve fibre layer thickness of the affected eye alone.

203 linically unaffected fellow eye, the retinal nerve fibre layer thickness of the affected eye was sign

204 At presentation, increased retinal nerve fibre layer thickness was associated with impaired

205 eover, when alpha-synuclein, iron or retinal nerve fibre layer thickness were added as a cofactor thi

206 Colour-contrast sensitivity and retinal nerve fibre layer thickness were measured in subgroups.

207 inically relevant parameters such as retinal nerve fibre layer thickness, fractional flow reserve, an

208 Peripapillary retinal nerve fibre layer thickness, measured by optical coheren

209 sion, visual fields, macular volume, retinal nerve fibre layer thickness, or optic nerve magnetisatio

210 Retinal nerve fibre layer thinning is usually evident within 3 m

211 therapy, degeneration of the macular retinal nerve fibre layer was reduced over 2 years.

212 f the tumour showed proliferation in retinal nerve fibre layer with normal structure of underlying re

213 ds on examination of the optic disc, retinal nerve fibre layer, and visual field.

214 Acquired RTD affected the retinal nerve fibre layer, ganglion cell and inner plexiform lay

215 ogies (eg, thinning of peripapillary retinal nerve fibre layer, inner retinal layer, and choroidal la

216 entation, yielded thicknesses of the retinal nerve fibre layer, the ganglion cell layer plus inner pl

217 e of optic neuritis, thinning of the retinal nerve fibre layer, which indicates axonal loss, is obser

218 y of 0.17 versus -0.1) and a thinner retinal nerve fibre layer.

219 imarily focused on evaluation of the retinal nerve fibre layer.

220 tection algorithms to judge axon loss in the nerve fibre layer.

221 assessing the optic nerve and peripapillary nerve fibre layer.

222 o published arcuate divisions of the retinal nerve fibre layer.

223 cular ganglion cell layer thickness, retinal nerve fibre layer/ganglion cell layer boundary to inner

224 The local nerve-fibre layer determined patch size, and quantal mye

225 ysed the position, area and thickness of the nerve-fibre layer in 60 patches of retinal myelin in 47

226 Nerve-fibre layer thickness was obtained from an atlas o

227 edominantly macular thinning (normal retinal nerve fibre-layer thickness with average macular thickne

228 while ART-naive participants showed thicker nerve fibre layers, possibly reflecting early inflammato

229 In patients with MCA stroke, corneal nerve fibre length (CNFL) (P < 0.001), corneal nerve fib

230 I [- 23.58, - 5.92], p = 0.002), and corneal nerve fibre length (mean difference: - 2.57 mm/mm(2), 95

231 Corneal nerve fibre length and branch density were lower, whilst

232 Corneal nerve fibre length was used to stratify participants wit

233 Retinal nerve fibre loss in congenital homonymous hemianopia in

234 This study was performed to assess cutaneous nerve fibre loss in conjunction with temperature and swe

235 CCM identifies small nerve fibre loss, which correlates with the severity of

236 itivity; L, cellular debris along and within nerve fibres; M, circular axonal inclusions surrounded b

237 nalyses reveal that cancer cells degrade the nerve fibre myelin sheets.

238 tailored to target specific types of cells, nerve fibres, neurotransmitters and communication pathwa

239 ade trans-synaptic degeneration of the optic nerve fibres occurs.

240 noreactivity is greatly increased in colonic nerve fibres of patients with active inflammatory bowel

241 in most neurons, including primary afferent nerve fibres of the pain pathway.

242 nerative effect of the disease itself on the nerve fibres of the retina.

243 lized facial structure that is innervated by nerve fibres of the somatosensory system.

244 VRCs of human sympathetic nerve fibres of the superficial peroneal nerve innervati

245 or 2 times the threshold for most excitable nerve fibres) of the superficial radial (SR) and ulnar (

246 We show that stimulation of hepatic nerve fibres or perfusing the liver with physiological c

247 between HbA1c and duration of diabetes with nerve fibre parameters or LC's density.

248 n target innervation and the relationship of nerve fibre pathology to sensory symptoms and signs.

249 ion is an endogenous process in the afferent nerve fibres, perhaps linked to random channel activity

250 Expression of nerve fibres positive for GDNF (p=0.001) and tyrosine ki

251 consists of sympathetic and parasympathetic nerve fibres, primarily located in the epicardial fat pa

252 work indicates that newly formed adrenergic nerve fibres promote tumour growth, but the origin of th

253 the acid-sensing ion channel (ASIC) family; nerve fibre recordings have shown ASIC2 and ASIC3 null m

254 We find that the activity of fin ray nerve fibres reflects the amplitude and velocity of fin

255 e effect of different factors on the rate of nerve fibre regeneration and investigated whether such a

256 on and the lack of target specificity during nerve fibre regrowth interfere with a good functional re

257 The recordings from motor nerve fibres required progressive dissection of the nerv

258 magnitude, whereas the firing rate of optic nerve fibres spans less than two.

259 n seen in ontogeny and preceded regenerating nerve fibres, suggesting that enhancement of blood vesse

260 n there is damage to high-threshold auditory nerve fibre synapses with cochlear inner hair cells.

261 activates CCK A receptors on vagal afferent nerve fibre terminals, which in turn initiate a vago-vag

262 Solid tumours are innervated by nerve fibres that arise from the autonomic and sensory p

263 The solid tumour microenvironment includes nerve fibres that arise from the peripheral nervous syst

264 Our finding of isolated nerve fibres that express substance P deep within diseas

265 l the extent of damage to small unmyelinated nerve fibres that go undetected by NCS.

266 infrared signals are detected by trigeminal nerve fibres that innervate specialized pit organs on th

267 A1 channels as infrared receptors on sensory nerve fibres that innervate the pit organ.

268 ls in regulating the excitability of sensory nerve fibres that mediate mechanical pain.

269 kin contains small diameter mechanoreceptive nerve fibres that signal pleasant touch.

270 en chemosensory epithelial cells and sensory nerve fibres, that conveys interoceptive signals to the

271 n the regions of the auditory and vestibular nerve fibres, the neural and abneural limbs adjacent to

272 dators by activating TRP channels on sensory nerve fibres to elicit pain and inflammation.

273 within adipose tissue communicate with local nerve fibres to modulate neurotransmitter tone, blood fl

274 bcortical strokes is due to the inability of nerve fibres to regenerate.

275 ing cold and tactile inputs from A-type skin nerve fibres to sense wetness.

276 l nerve, to determine whether differences in nerve fibre type or location affect the level of abnorma

277 ganglia (DRG) to the axon and any peripheral nerve fibre type.

278 We reviewed four newly proposed teased nerve fibre types (Types J-M): Type J, rope-like fibres;

279 Teased nerve fibre Types J-M associate with commonly seen patho

280 and transcript analysis of dermal myelinated nerve fibres using a novel platform, revealed a marked i

281 The average density of epidermal nerve fibres was greatly diminished in the calf and back

282 e labelling of both neuronal cell bodies and nerve fibres was observed in the paraventricular nucleus

283 Single pulpal nerve fibres were electrically stimulated at just above

284 Nerve fibres were electrically stimulated using single o

285 molecular architecture of dermal myelinated nerve fibres were examined using immunohistochemistry an

286 Some scattered positive neurons and nerve fibres were found in many hypothalamic nuclei incl

287 Sympathetic nerve fibres were identified by studying their response

288 t P2X2 immunoreactivity-positive neurons and nerve fibres were localized in many hypothalamic nuclei.

289 cell bodies were strongly positive, but few nerve fibres were positive.

290 nilloid receptor 1 (VR1)-containing afferent nerve fibres were present on the epicardial surface of t

291 he samples from back-pain patients, isolated nerve fibres were seen in the discal matrix.

292 mage.In seven animals, 10 single intradental nerve fibres were selected that responded to hydrostatic

293 The time constants of motor and sensory nerve fibres were studied in normal human ulnar nerves b

294 tion on the times of activation of the motor nerve fibres which were as accurate as a direct record f

295 ased spontaneous activity of single auditory nerve fibres, while concanavalin A had no effect, sugges

296 secreted from axons and reach glial cells in nerve-fibre (white-matter) tracts?

297 estigation showed focal swellings of retinal nerve fibres with neurofilament disruption.

298 on cells and central and peripheral auditory nerve fibres within the cochlea.

299 rostimulation (INMS) we stimulated groups of nerve fibres, within individual fascicles proximal to th