ライフサイエンスコーパス: nerve growth

1 roduced compensatory increases in peripheral nerve growth.

2 ensory nerve growth and promoted sympathetic nerve growth.

3 uggested enrichment in gene sets involved in nerve growth and general cell development.

4 GF-A from infected corneas repressed sensory nerve growth and promoted sympathetic nerve growth.

5 as well as their emerging use for studies of nerve growth and repair.

6 of Chn1KI/KI mice revealed stalled abducens nerve growth and selective trochlear and first cervical

7 healing [1], development [2], cell division, nerve growth, and angiogenesis [3].

8 the loss of Limk1, results in faster sciatic nerve growth, and improved recovery of some sensory and

9 Effects of polySia removal on trigeminal nerve growth behavior were determined in vivo, using exo

10 y proteins that may influence keratocyte and nerve growth cone behavior in the cornea.

11 Effects of purified GAGs on trigeminal nerve growth cone behavior were tested using in vitro ne

12 ins that may regulate keratocytes or corneal nerve growth cone immigration interact with corneal GAGs

13 The nerve growth cone is bi-directionally attracted and repe

14 Nerve growth cones (GCs) are chemical sensors that conve

15 Sensory trigeminal nerve growth cones innervate the cornea in a highly coor

16 t GAGs may direct the movement of trigeminal nerve growth cones innervating the cornea.

17 During cornea development, chick trigeminal nerve growth cones reach the cornea margin at embryonic

18 nd-binding and lattice-binding activities in nerve growth cones, and reveal that the two MT-binding a

19 le cells - fan-like keratocytes, hand-shaped nerve growth cones, polygonal fibroblasts, to name but a

20 First, to create an optic nerve growth curve from normal optic nerve sheath diamet

21 An optic nerve growth curve was created by using ONSDs measured i

22 d in regulation of neurotransmitter systems, nerve growth/death and gene expression, and subsequently

23 .849, p<0.001), alpha1-ACT (0.638, p<0.001), nerve growth factor (5.475, p<0.005) and visinin-like pr

24 e growth factor-binding protein 2 (IGFBP-2), nerve growth factor (b-NGF), platelet-derived growth fac

25 The plasma concentrations of nerve growth factor (BCAA: 4.0 +/- 1 pg/mL; low-BCAA: 5.

26 day, driven by circadian clock control of a nerve growth factor (BDNF) in the inner ear.

27 orcine, bovine, and murine sequences of beta nerve growth factor (beta-NGF).

28 solate low-affinity antibodies to human beta nerve growth factor (hbetaNGF).

29 Human nerve growth factor (hNGF) is an important pharmaceutica

30 and sympathetic neurons is promoted whether nerve growth factor (NGF) activates TrkA receptors on th

31 rch for neuroactive compounds that mimic the nerve growth factor (NGF) activity for the protection ag

32 the preservation of sufficient expression of nerve growth factor (NGF) and activation of the neurotro

33 Neurotrophins such as nerve growth factor (NGF) and brain-derived neurotrophic

34 rait loci associated with gene expression of nerve growth factor (NGF) and calneuron 1 (CALN1) genes.

35 The nerve growth factor (NGF) and glial cell line-derived ne

36 The effect of nerve growth factor (NGF) and its receptor (NGFR) in inf

37 ends, can be modulated by luminar release of nerve growth factor (NGF) and neurotrophin-3 (NT-3), res

38 Nerve growth factor (NGF) and neurotrophin-3 serve as at

39 h cone collapse and loss of actin filaments, nerve growth factor (NGF) and neurotrophin-3 still induc

40 SorCS3 binds the nerve growth factor (NGF) and platelet-derived growth fa

41 Nerve growth factor (NGF) antagonism is on the verge of

42 brain-derived neurotrophic factor (BDNF) and nerve growth factor (NGF) are crucial modulators in the

43 Levels of nerve growth factor (NGF) are elevated in inflamed tissu

44 In this paper, we identify nerve growth factor (NGF) as a binding partner for MOG a

45 In this study, we test nerve growth factor (NGF) as an understudied therapeutic

46 satetraenoic acid (12[S] or 15[S]-HETE), and nerve growth factor (NGF) as positive control.

47 aglandin E(2) (PGE(2)), bradykinin (BK), and nerve growth factor (NGF) as well as multiple kinases, i

48 in animals and humans show that blockade of nerve growth factor (NGF) attenuates both malignant and

49 TrkA activation by nerve growth factor (NGF) binding the second extracellul

50 esis of SCG neurons at a level comparable to nerve growth factor (NGF) but also doubled the neurite l

51 ated the output activity of Ngf encoding the nerve growth factor (NGF) by increasing histone H4 acety

52 d the selective translation and secretion of nerve growth factor (NGF) by PDAC cells to promote tumor

53 odels of MM to show significant induction of nerve growth factor (NGF) by the tumour-bearing bone mic

54 Elevating levels of nerve growth factor (NGF) can have pronounced effects on

55 SIGNIFICANCE STATEMENT The R100W mutation in nerve growth factor (NGF) causes Hereditary Sensory and

56 Nerve growth factor (NGF) contributes to the development

57 Although nerve growth factor (NGF) controls survival, maturation

58 Sertad1 is also induced in neurons by nerve growth factor (NGF) deprivation and Abeta (beta-am

59 the painful arthritic knee joint and whether nerve growth factor (NGF) drives this pathologic reorgan

60 ent a detailed motion analysis of retrograde nerve growth factor (NGF) endosomes in axons to show tha

61 Nerve growth factor (NGF) exerts protective effects on t

62 We show that PC12 cells stimulated with nerve growth factor (NGF) exhibit statistically signific

63 elta), two transcription factors involved in nerve growth factor (NGF) expression and downregulated s

64 timulation of the gastric epithelium induced nerve growth factor (NGF) expression, and in turn NGF ov

65 ciated haplotype blocks were intronic to the Nerve Growth Factor (NGF) gene (P=0.001, 0.001, 0.002),

66 In 2001, we initiated a clinical trial of nerve growth factor (NGF) gene therapy in AD, the first

67 ) at the cell surface, and administration of nerve growth factor (NGF) has been considered and attemp

68 The neurotrophin nerve growth factor (NGF) has been implicated as a key m

69 ng mode of Spz to Toll is similar to that of nerve growth factor (NGF) in complex with the p75 neurot

70 +) BMCs expressed neurotrophins and secreted nerve growth factor (NGF) in conditioned medium.

71 taRII-deficient pups showed a lower level of nerve growth factor (NGF) in its mRNA; however, higher l

72 n channel subfamily M, member 8 (TRPM8); and nerve growth factor (NGF) in nasal biopsy specimens.

73 s and becomes functional by the neurotrophin nerve growth factor (NGF) in native brainstem neurons.

74 on (DRG) neurons by increasing expression of nerve growth factor (NGF) in the colon wall.

75 se it is supported by decades of research on nerve growth factor (NGF) in the peripheral nervous syst

76 The endogenous nerve growth factor (NGF) in the urinary bladder regulat

77 Nerve growth factor (NGF) induces collateral branching a

78 Nerve growth factor (NGF) influences the survival and di

79 The microinjection of nerve growth factor (NGF) into the cat pontine tegmentum

80 Nerve growth factor (NGF) is a key mediator of nocicepti

81 Nerve growth factor (NGF) is a key regulator of chronic

82 Nerve growth factor (NGF) is a neuropeptide essential fo

83 Nerve growth factor (NGF) is a neurotrophin that activat

84 Nerve growth factor (NGF) is a neurotrophin that is impl

85 Nerve growth factor (NGF) is a potent survival and axon

86 Nerve growth factor (NGF) is a target-derived neurotroph

87 Nerve growth factor (NGF) is elevated in certain chronic

88 Nerve growth factor (NGF) is initially synthesized as a

89 that retrograde signaling by target-derived nerve growth factor (NGF) is necessary for soma-to-axon

90 r mechanism of TRKA activation by its ligand nerve growth factor (NGF) is still unsolved.

91 Nerve growth factor (NGF) is the neurotrophin responsibl

92 Nerve growth factor (NGF) levels are highly increased in

93 Nerve growth factor (NGF) monoclonal antibody therapy ha

94 eactivation in medium containing antibody to nerve growth factor (NGF) or delayed reactivation in med

95 We hypothesized that nerve growth factor (NGF) plays a key role in this proce

96 they innervate is regulated by the supply of nerve growth factor (NGF) produced by these tissues.

97 Here, we report that Nerve Growth Factor (NGF) promotes endocytosis of its Tr

98 Nerve growth factor (NGF) promotes growth, differentiati

99 on that leads to cell proliferation, whereas nerve growth factor (NGF) promotes sustained ERK activat

100 In sensory neurons, nerve growth factor (NGF) promotes the formation of axon

101 Nerve growth factor (NGF) protein expression and secreti

102 Activation of the high-affinity nerve growth factor (NGF) receptor Trk occurs through mu

103 Activation of the nerve growth factor (NGF) receptor trkA and tissue acido

104 The Tp53inp2 transcript interacts with the nerve growth factor (NGF) receptor TrkA, regulating TrkA

105 Notably, we identified the nerve growth factor (NGF) receptor tyrosine kinase (NTRK

106 The nerve growth factor (NGF) receptor, trkA, the tumour sup

107 tic neurons by inhibiting endocytosis of the nerve growth factor (NGF) receptor, TrkA.

108 he administration of neuregulin 1 (NRG1) and nerve growth factor (NGF) recombinant proteins.

109 Nerve growth factor (NGF) regulates many aspects of neur

110 The neurotrophin nerve growth factor (NGF) regulates neuronal growth, dif

111 Here we demonstrate that nerve growth factor (NGF) regulates the levels of IP(5)

112 performing candidate mediates high levels of nerve growth factor (NGF) secretion from astrocytes, cau

113 s promoted ADRB2-dependent PDAC development, nerve growth factor (NGF) secretion, and pancreatic nerv

114 gic deficit is associated with alteration in nerve growth factor (NGF) signaling and its relation to

115 ic neurons that was essential for retrograde nerve growth factor (NGF) signaling and neuron target ti

116 Facilitation of nerve growth factor (NGF) signaling by the p75 neurotrop

117 This study reports that Rab22 promotes nerve growth factor (NGF) signaling-dependent neurite ou

118 MO7e express TrkA, the primary receptor for Nerve Growth Factor (NGF) signaling.

119 to disrupt retrograde axonal trafficking of nerve growth factor (NGF) signals.

120 anges in living colonies of PC12 cells under nerve growth factor (NGF) stimulation for up to 7 days u

121 RK) activity and cell proliferation, whereas nerve growth factor (NGF) stimulation leads to sustained

122 ng in cholesterol-rich membrane regions upon nerve growth factor (NGF) stimulation: We argue that thi

123 Retrograde trophic signaling of nerve growth factor (NGF) supports neuronal survival and

124 Numerous studies have indicated that nerve growth factor (NGF) supports survival and phenotyp

125 n ongoing discussion is whether signaling of nerve growth factor (NGF) through its high-affinity rece

126 reported that enables intravenously injected nerve growth factor (NGF) to enter the CNS in healthy mi

127 and impairs neurite outgrowth in response to nerve growth factor (NGF) treatment.

128 egeneration in regards to local secretion of nerve growth factor (NGF) upon carious injury.

129 Nerve growth factor (NGF) was discovered because of its

130 The death of sympathetic neurons after nerve growth factor (NGF) withdrawal requires de novo ge

131 Nerve growth factor (NGF), a classical trophic factor fo

132 asthma control by stimulating expression of nerve growth factor (NGF), a neurotrophin associated wit

133 ytosis into axon growth cones is enhanced by nerve growth factor (NGF), acting locally on distal axon

134 e treatment with an antibody that sequesters nerve growth factor (NGF), administered when the pain an

135 ptase (TPS1) and mastin, neuromedin-B (NMB), nerve growth factor (NGF), and leukotriene-synthesis enz

136 x8 expression and function were regulated by nerve growth factor (NGF), and the effect of NGF was pot

137 brain derived neurotrophic factor (BDNF) and nerve growth factor (NGF), and this increase is accompan

138 MCF2L regulates a nerve growth factor (NGF), and treatment with a humanize

139 cific enolase, growth-associated protein 43, nerve growth factor (NGF), and tyrosine kinase receptor

140 ophin family of growth factors, comprised of nerve growth factor (NGF), brain derived neurotrophic fa

141 ated by hormones of the neurotrophin family: nerve growth factor (NGF), brain derived neurotrophic fa

142 free DPSC cultures and neurotrophic factors; nerve growth factor (NGF), brain-derived neurotrophic fa

143 sence of fibroblast growth factor 2 (FGF-2), nerve growth factor (NGF), brain-derived neurotropic fac

144 d protein expression increase in response to nerve growth factor (NGF), concomitant with differentiat

145 pared routes of delivery of the neurotrophin nerve growth factor (NGF), either through a multisynapti

146 ine distinct monoclonal antibodies targeting nerve growth factor (NGF), for which we compare the pred

147 (d) in culture and this can be prevented by nerve growth factor (NGF), GDNF and ARTN, but not NRTN.

148 We show that nerve growth factor (NGF), implicated in the morphogenes

149 Neurotrophins, including nerve growth factor (NGF), increase neurite outgrowth in

150 directly binds acinus, which is regulated by nerve growth factor (NGF), inhibiting its stimulatory ef

151 ociceptive and inflammatory factors, such as nerve growth factor (NGF), interleukin-6 (IL-6), and car

152 However, the prototypic neurotrophin, nerve growth factor (NGF), is not thought to be anterogr

153 a (TNFalpha), interleukin-1 beta (IL1 beta), nerve growth factor (NGF), the neuronal nuclear protein

154 Pro-nerve growth factor (NGF), the precursor of NGF, is a we

155 ne interleukin 6 (IL-6) and the neurotrophin nerve growth factor (NGF), which are intimately linked t

156 to ischemia, retinal neuronal cells express nerve growth factor (NGF), which can be proangiogenic.

157 oietic cytokine ligands of gp130 but also to nerve growth factor (NGF), which does not bind to gp130-

158 In this study we determined the number of nerve growth factor (NGF)- and interleukin-1beta (IL1bet

159 During this process, nerve growth factor (NGF)- TrkA signaling in axons commu

160 tors tropomyosin-related kinase A (TrkA) for nerve growth factor (NGF)-beta, TrkB for brain-derived n

161 We discovered that KIF1Bbeta is required for nerve growth factor (NGF)-dependent neuronal differentia

162 This affects the development of nerve growth factor (NGF)-dependent neurons including sy

163 Drp1-mediated fission is required for nerve growth factor (NGF)-induced collateral branching i

164 terference RNA methodology strongly enhanced nerve growth factor (NGF)-induced neurite outgrowth in P

165 ative mutants of ATL1 in PC-12 cells inhibit nerve growth factor (NGF)-induced neurite outgrowth.

166 s study, we investigated the effect of ES on nerve growth factor (NGF)-induced neuronal differentiati

167 Egr3 is a nerve growth factor (NGF)-induced transcriptional regula

168 Nerve growth factor (NGF)-induced transport of large ves

169 NF-alpha-TNFR1 forward signaling to suppress nerve growth factor (NGF)-mediated neurite growth, survi

170 oter under basal conditions and also enhance nerve growth factor (NGF)-mediated trkA promoter activat

171 This is reversed by inhibition of nerve growth factor (NGF)-mediated tropomyosin receptor

172 This retention is mediated by a nerve growth factor (NGF)-regulated checkpoint that dela

173 Here, using single-cell image analysis of nerve growth factor (NGF)-stimulated PC12 cells, we iden

174 ouse sympathetic neurons, the target-derived nerve growth factor (NGF)-tropomyosin-related kinase typ

175 s mediate extension of sympathetic axons via nerve growth factor (NGF).

176 onal growth in response to the neurotrophin, nerve growth factor (NGF).

177 e transmembrane receptor tyrosine kinase for nerve growth factor (NGF).

178 ll neurite extension in response to released nerve growth factor (NGF).

179 ng leptin, insulin, growth hormone (GH), and nerve growth factor (NGF).

180 , basic fibroblast growth factor (bFGF), and nerve growth factor (NGF).

181 anscription factor activation in response to nerve growth factor (NGF).

182 lated local IMPA1 translation in response to nerve growth factor (NGF).

183 the cell surface, similar to treatment with nerve growth factor (NGF).

184 p-regulation of the growth-promoting factor, nerve growth factor (NGF).

185 found to express the following receptors for nerve growth factor (NGF): neurotrophic receptor tyrosin

186 (P<0.0001) and cells positively staining for nerve growth factor (NGF; P<0.0001) in the infarcted bra

187 Significant examples include the nerve growth factor (P = 7.86 x 10(-33)), epidermal grow

188 The precursor of nerve growth factor (proNGF) has been described as a bio

189 We report that the precursor of nerve growth factor (proNGF) is overexpressed in prostat

190 pathway entails the proteins that mature pro-nerve growth factor (proNGF) to NGF and those that degra

191 ation around itself, and that high levels of nerve growth factor and axon guidance molecules are reco

192 (2) this interaction is modified by ligands nerve growth factor and beta-amyloid; (3) APP and p75(NT

193 ed in intracellular transport and sorting of nerve growth factor and brain-derived neurotrophic facto

194 ed in intracellular transport and sorting of nerve growth factor and brain-derived neurotrophic facto

195 eutralizing antibodies against brain-derived nerve growth factor and ciliary neurotrophic factor.

196 Nerve growth factor and its TrkA receptor were upregulat

197 d the AP1-mediated transcription promoted by nerve growth factor and modulated the expression of seve

198 otrophins brain derived neurotrophic factor, nerve growth factor and neurotrophin-3 in the supernatan

199 trophin 3, insulin-like growth factor 1, and nerve growth factor and of the nerve growth factor recep

200 F-A-dependent gene transcription elicited by nerve growth factor and serum.

201 on equatorial BM except for higher levels of nerve growth factor and thrombospondin-2 (TSP2) by hES-R

202 tor blockade is independent of brain-derived nerve growth factor and TrkB receptor signaling.

203 mpathetic axon outgrowth that was blocked by nerve growth factor antibodies.

204 (i) Anti-nerve growth factor antibody accelerated the reactivatio

205 e structurally related cystine-knot protein, nerve growth factor beta (NGFbeta), plays an unexpected

206 oclonal antibody, MEDI1912, selected against nerve growth factor binds with picomolar affinity, but u

207 ecal Rab7-siRNA or, indirectly, by reversing nerve growth factor deprivation in peripheral sensory ne

208 le Caspase-6 is activated in axons following nerve growth factor deprivation, microfluidic chamber ex

209 axon-specific but not whole-cell (apoptotic) nerve growth factor deprivation.

210 The present study used nerve growth factor differentiated PC12 cells (NGFDPC12

211 melanocytes and melanogenesis plus FIG4 and nerve growth factor expression, suggesting higher cellul

212 s as well as reduce mast cell activation and nerve growth factor expression.

213 d in chromatin remodeling of the RARbeta and nerve growth factor IB ( NUR77).

214 we have demonstrated increased levels of pro-nerve growth factor in cerebrospinal fluid and increased

215 Increased expression of nerve growth factor in injured or inflamed tissue is ass

216 the plasma concentration of norepinephrine, nerve growth factor in the gastric fundus muscularis ext

217 trograde model derived from experiments with nerve growth factor in the peripheral nervous system.

218 tor to p75 neurotrophin receptor, blocks pro-nerve growth factor induced apoptosis in cells expressin

219 Nerve growth factor induced mild cold sensitization, con

220 munoprecipitation experiments indicated that nerve growth factor induced the association of endogenou

221 We find that Vgf nerve growth factor inducible gene up-regulation is a co

222 anticipated, long and extensive neuritis on nerve growth factor induction.

223 which show promise in clinical trials, like nerve growth factor inhibitors and p38 kinase inhibitors

224 onsequence of the increased levels of mature nerve growth factor levels in skin, as revealed by Weste

225 n to motoneurons, depletion of increased pro-nerve growth factor levels or p75 receptor blockade.

226 Second, there was evidence of increased nerve growth factor production and dysregulation of the

227 orward signaling loop in which tumor-derived nerve growth factor promotes enteric tumor innervation,

228 lates expression of numerous genes including nerve growth factor receptor (NGFR), which becomes trans

229 asolateral expression of the apically sorted nerve growth factor receptor (NGFR, p75; extracellular a

230 he NTRK1 gene that encodes the high-affinity nerve growth factor receptor (TRKA protein).

231 hese (AG879) is a selective inhibitor of the nerve growth factor receptor and human epidermal growth

232 gy domain transcription factor RUNX1 and the nerve growth factor receptor TrkA.

233 re initially marked by the expression of the nerve growth factor receptor TrkA.

234 r hierarchical expression of CD271/p75/NGFR (nerve growth factor receptor) marks cells with enriched

235 sine phosphorylation sequences of either the nerve growth factor receptor, TrkA (tropomyosin receptor

236 We found that the high-affinity nerve growth factor receptor, TrkA, is down-regulated by

237 DHEA was previously shown to bind to the nerve growth factor receptor, tropomyosin-related kinase

238 Adult C57BL/6N and nerve growth factor receptor-deficient mice.

239 neurotrophic receptor tyrosine kinase 1 and nerve growth factor receptor.

240 otic properties via mechanisms involving the nerve growth factor receptors (tropomyosin-related kinas

241 factor 1, and nerve growth factor and of the nerve growth factor receptors, tyrosine kinase receptor

242 Ephrins (EFNs), 8 semaphorins (SEMAs), and 2 nerve growth factor receptors.

243 nalyses implicated p75 neurotrophin receptor/nerve growth factor signaling and innate immune toll-lik

244 c convergence on gene mutations that disrupt nerve growth factor signaling, upon which sympathetic an

245 sary for PP2A/B'beta-mediated enhancement of nerve growth factor signaling.

246 cells, and enhanced neurite outgrowth after nerve growth factor stimulation.

247 that this death is not due to a reduction in nerve growth factor synthesis.

248 ons of brain-derived neurotrophic factor and nerve growth factor than NCSC-SCs.

249 uction and dysregulation of the ratio of pro-nerve growth factor to mature nerve growth factor, favou

250 we report that EVT901 reduces binding of pro-nerve growth factor to p75 neurotrophin receptor, blocks

251 ers indicate that PLCbeta induced soon after nerve growth factor treatment associates with TRAX rathe

252 After nerve growth factor withdrawal, sympathetic axons derive

253 nti-calcitonin gene-related peptide and anti-nerve growth factor) are also discussed.

254 The homodimer NGF (nerve growth factor) exerts its neuronal activity upon b

255 that are also the primary sites of action of nerve growth factor, a powerful modulator of bladder ner

256 at act at receptor tyrosine kinases, such as nerve growth factor, also cause differentiation.

257 diminished levels of inflammatory cytokines, nerve growth factor, and endogenous pruritogens, such as

258 including brain-derived neurotrophic factor, nerve growth factor, and neurotrophin 3.

259 r cells express neurotrophic markers such as nerve growth factor, brain-derived neurotrophic factor,

260 ch a GM-CSF->CCL17 pathway appears critical, nerve growth factor, CGRP, and substance P all appear to

261 ted with the well established pain mediator, nerve growth factor, could also modify macrophage gene t

262 Furthermore, decreased nerve growth factor, decreased c-fos and increased sympa

263 e ratio of pro-nerve growth factor to mature nerve growth factor, favouring p75 receptor expression a

264 including brain-derived neurotrophic factor, nerve growth factor, glial cell-derived neurotrophic fac

265 Studies indicate that treatment against nerve growth factor, interleukins, and ischemic-like med

266 elevated expression of key proteins such as nerve growth factor, myelin protein zero, and brain deri

267 inistration of neurotrophic factors (such as nerve growth factor, neurotrophin3, glial-derived neurot

268 t or the inhibition of adrenergic receptors, nerve growth factor, or brain-derived neurotrophic facto

269 When treated with nerve growth factor, PC12 cells will differentiate over

270 neuroprotective treatment (recombinant human nerve growth factor, rh-NGF) predominantly targeting sec

271 of the Trk receptors, which are activated by nerve growth factor, there are only two established phos

272 cts neuroprotection by neurotrophins such as nerve growth factor, which bind to p75NTR receptors on M

273 Nerve growth factor-beta (NGF) is essential for the corr

274 ed proteins: a therapeutic IgG1-antibody and nerve growth factor-beta (NGF).

275 is was associated with reduced expression of nerve growth factor-beta, indicating less atrial nerve s

276 c acid (PA)-induced lipotoxicity (PA-LTx) in nerve growth factor-differentiated PC12 (NGFDPC12) cells

277 from cardiac synaptosomes and dopamine from nerve growth factor-differentiated PC12 cells in a conce

278 1 transactivates the Skp2 promoter through a nerve growth factor-induced clone B response element (NB

279 ereas USP21 knockdown in PC12 cells inhibits nerve growth factor-induced neurite outgrowth.

280 siRNA improved cell survival in response to nerve growth factor-induced OL apoptosis.

281 hat increased DNA methylation at the NGFI-A (nerve growth factor-induced protein A) binding site of t

282 hibitory factors (e.g. inhibin alpha and VGF nerve growth factor-inducible), 2) activation of a signa

283 only known Toll receptor ligand is the human nerve growth factor-like cystine knot protein Spatzle.

284 owed a reduction in neurite outgrowth and in nerve growth factor-mediated neuronal differentiation, t

285 t from lactate release and activation of pro-nerve growth factor-p75 receptor signalling are key comp

286 Bone is richly innervated by nerve growth factor-responsive (NGF-responsive) tropomyo

287 ays 0, 3, 7, and 12 following treatment with nerve growth factor.

288 monoclonal antibody that binds and inhibits nerve growth factor.

289 e A (TrkA) is the high-affinity receptor for nerve growth factor.

290 Reactivation can be induced by depletion of nerve growth factor; other commonly used reactivation st

291 of brain-derived neurotrophic factor (BDNF), nerve-growth factor (NGF), neurotrophin-3 (NT-3), fibrob

292 brain-derived neurotrophic factor (BDNF) and nerve-growth factor (NGF).

293 ved regions were found near color vision and nerve-growth genes, consistent with purifying selection

294 chemical and biophysical cues for increasing nerve growth has been investigated, including neurotroph

295 zation as well as an induction of peripheral nerve growth in grafted areas compared with samples not

296 ntial for implementing optogenetics to drive nerve growth in specific cell populations.

297 ter myocardial infarction, a situation where nerve growth is hindered by age-related influences and s

298 ibited skin edema, keratinocyte hyperplasia, nerve growth, leukocyte infiltration, and antihistamine-

299 m microarray of 85 extracellular epitopes of nerve growth-related proteins.

300 s, and porosity have been shown to influence nerve growth within nerve guidance scaffolds, independen