ライフサイエンスコーパス: nerve stimulation

1 ng anaesthesia (n = 5) instead of repetitive nerve stimulation.

2 epicardial DOR increased during sympathetic nerve stimulation.

3 anced vascular responsiveness to sympathetic nerve stimulation.

4 mpromised the muscle twitch triggered by the nerve stimulation.

5 dominant repolarizing role during repetitive nerve stimulation.

6 s and stretchable multi-electrode arrays for nerve stimulation.

7 in relocalization to synaptic membranes upon nerve stimulation.

8 sensory evoked potentials elicited by median nerve stimulation.

9 d to achieve directional fiber activation in nerve stimulation.

10 timulation but not F-EPSPs evoked by colonic nerve stimulation.

11 timulation but not F-EPSPs evoked by colonic nerve stimulation.

12 timulation but not F-EPSPs evoked by colonic nerve stimulation.

13 postsynaptic responses elicited by afferent nerve stimulation.

14 vels ([NAD(P)H]m) increase within seconds of nerve stimulation.

15 imary sensory cortex face area due to median nerve stimulation.

16 elicited by bath-applied CCh and cholinergic nerve stimulation.

17 r inhibition of cytokine production by vagus nerve stimulation.

18 but no increased activation following tibial nerve stimulation.

19 ulation, but reduced activations with tibial nerve stimulation.

20 f non-neuronal cells, induced by sympathetic nerve stimulation.

21 firing in pulmonary veins by local autonomic nerve stimulation.

22 irements significantly decreased after vagus nerve stimulation.

23 ter activity evoked in response to selective nerve stimulation.

24 es to acetylcholine, 5-hydroxytryptamine, or nerve stimulation.

25 ing the therapeutic mechanisms of trigeminal nerve stimulation.

26 to assess gamma-knife radiosurgery or vagal nerve stimulation.

27 tional inhibition of TNF production by vagus nerve stimulation.

28 recocious corneal thinning, but augmented T4 nerve stimulation.

29 cipital region with electrodes for occipital nerve stimulation.

30 exercise in response to supramaximal femoral nerve stimulation.

31 d for the soleus H-reflex elicited by tibial nerve stimulation.

32 ease of beta-NAD depends on the frequency of nerve stimulation.

33 taste buds on the rostral 1/3 of the tongue) nerve stimulation.

34 e seen with field potentials evoked by sural nerve stimulation.

35 nual therapies and transcutaneous electrical nerve stimulation.

36 scranial magnetic stimulation and peripheral nerve stimulation.

37 vered to tibialis anterior (TA) MNs by sural nerve stimulation.

38 ed by measuring force in response to femoral nerve stimulation.

39 DeltaQTsingle ) evoked by electrical femoral nerve stimulation.

40 In vehicle-treated rats, sympathetic nerve stimulation (1 to 5 Hz) evoked decreases in femora

41 rhodamine-123, we determined how repetitive nerve stimulation (100 Hz) affects Psi(m) in motor termi

42 d, ventilated dogs were elicited via sciatic nerve stimulation (50 Hz; 200 ms duration; 1 contraction

43 ponse to bilateral anterior magnetic phrenic nerve stimulation (a pressure <11 cm H2O defined dysfunc

44 Here, we report that renal nerve stimulation after ureteral obstruction is the prim

45 magnetic stimulation and external trigeminal nerve stimulation (all with regulatory clearance) were w

46 Trigeminal nerve stimulation also decreased systemic inflammation c

47 Swallowing evoked by airway afferent nerve stimulation also desensitized at a much slower rat

48 tylcholine release in the ventricle on vagal nerve stimulation and a high density of acetylcholine M2

49 trol that included transcutaneous electrical nerve stimulation and active range-of-motion exercises d

50 (n = 105) received transcutaneous electrical nerve stimulation and active range-of-motion exercises.

51 e EAA afferents to the LC, including sciatic nerve stimulation and auditory stimuli and the tonic act

52 defects to interventional techniques such as nerve stimulation and bulking agents.

53 itry more selectively than muscle stretch or nerve stimulation and can be adapted to study GTO feedba

54 therapy, cerebrospinal fluid drainage, vagal nerve stimulation and deep brain stimulation.

55 a decremental muscle response to repetitive nerve stimulation and frequently related to postsynaptic

56 paired and decreased in response to unpaired nerve stimulation and is mediated by the opposing action

57 , paired associative stimulation using ulnar nerve stimulation and PA TMS pulses over M1, a protocol

58 AS, which consisted of peripheral electrical nerve stimulation and subsequent transcranial magnetic s

59 le studies have demonstrated that peripheral nerve stimulation and targeted reinnervation can provide

60 a method of transcutaneous mechanical vagus nerve stimulation and then investigated whether this the

61 erminals is more responsive to low-frequency nerve stimulation and this is due to higher cytosolic Ca

62 hich involves repeated pairing of peripheral nerve stimulation and transcranial magnetic stimulation

63 al, twitch responses to supramaximal femoral nerve stimulation and transcranial magnetic stimulation

64 Somatosensory cortical potentials to median nerve stimulation and visual cortical potentials to reve

65 timulation (SNS) and right and/or left vagus nerve stimulation and was compared with DOR during isopr

66 ve electrical, transcutaneous, or sham vagus nerve stimulation and were followed for survival or euth

67 horacic epidural anesthesia, low-level vagal nerve stimulation, and baroreflex stimulation.

68 pies are available, including surgery, vagus nerve stimulation, and deep brain stimulation.

69 lation, namely deep brain stimulation, vagal nerve stimulation, and transcranial magnetic stimulation

70 al heat, traction, transcutaneous electrical nerve stimulation, and ultrasonography), spinal manipula

71 Less invasive versions of nerve stimulation are being researched.

72 eep brain stimulation, vagus, and trigeminal nerve stimulation are effective only in a fraction of th

73 responsiveness, and decrement on repetitive nerve stimulation are present.

74 ner if afferent volleys evoked by peripheral nerve stimulation are repeatedly associated with the pea

75 The anti-inflammatory effects of vagus nerve stimulation are well known.

76 euromodulation approaches, such as occipital nerve stimulation, are currently being actively studied

77 s, could help to explain the effect of vagus nerve stimulation as a treatment for headache disorders.

78 ssSEPs were elicited by electrical median nerve stimulation at the left and right wrist, using a s

79 tability of repeatedly delivering peripheral nerve stimulation at three time points (-30 ms, 0 ms, +5

80 enal sympathetic denervation, cervical vagal nerve stimulation, baroreflex stimulation, cutaneous sti

81 Postjunctional responses to nerve stimulation, beta-NAD and ATP were compared using

82 he amplitude of F-EPSPs evoked by splanchnic nerve stimulation but not F-EPSPs evoked by colonic nerv

83 he amplitude of F-EPSPs evoked by splanchnic nerve stimulation but not F-EPSPs evoked by colonic nerv

84 ic fast EPSPs (F-EPSPs) evoked by splanchnic nerve stimulation but not F-EPSPs evoked by colonic nerv

85 They can also sequester Ca(2+) during nerve stimulation, but it is unknown whether this limits

86 creased activations with handgrip and median nerve stimulation, but reduced activations with tibial n

87 but only reduced contractions evoked by 4 Hz nerve stimulation by approximately 40-60% (n = 4-6) and

88 In adult muscles, PSCs can sense nerve stimulation by increasing intracellular calcium an

89 l time to demonstrate that electrical sacral nerve stimulation can activate colonic enteric neurons.

90 Vagus nerve stimulation can ameliorate autoimmune diseases suc

91 t bilateral autonomic activity (SNS or vagus nerve stimulation) cause reversible shifts of apex-base

92 re, we use brief bursts of closed-loop vagus nerve stimulation (CL-VNS) delivered during rehabilitati

93 dly pairing tones with brief pulses of vagus nerve stimulation completely eliminated the physiologica

94 Percutaneous tibial nerve stimulation continues to display superiority to sh

95 anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selectively activate

96 on, transcranial magnetic stimulation, vagal nerve stimulation, deep brain stimulation, electroconvul

97 in response to supramaximal magnetic femoral nerve stimulation (DeltaQ(tw,pot)).

98 xercise in response to supra-maximal femoral nerve stimulation (DeltaQ(tw,pot)).

99 Left-sided low-level vagus nerve stimulation did not change vagal nerve activity.

100 With perivascular sensory nerve stimulation, dilatation and inhibition of sympathe

101 Transcutaneous vagus nerve stimulation dose-dependently reduced systemic tumo

102 lammatory pathway." Here, we show that vagus nerve stimulation during endotoxemia specifically attenu

103 Sympathetic nerve stimulation elicited localized Ca(2+) increases th

104 Pelvic nerve stimulation elicited polarized responses that were

105 Trigeminal nerve stimulation elicited strong synergistic coactivati

106 Earlier we showed that nerve stimulation elicits the appearance of spH fluoresc

107 on, (3) surgical approaches, including vagus nerve stimulation, epidural electrical stimulation, and

108 afferent inhibition of face M1, while facial nerve stimulation evoked LAI but not SAI.

109 y the uncoupling agent carbenoxolone reduces nerve stimulation-evoked catecholamine release in contro

110 of beta-NAD in superfusates of muscles after nerve stimulation exceed ATP by at least 30-fold; unlike

111 lease events increases with the frequency of nerve stimulation; external Ca(2+) entry is required for

112 Vagus nerve stimulation fails to control serum TNF levels in e

113 Here we show that vagus nerve stimulation fails to inhibit tumor necrosis factor

114 od and Drug Administration approval of vagus nerve stimulation for recurrent treatment-resistant depr

115 Long-term outcomes of sacral nerve stimulation for refractory OAB have been recently

116 nd muscle action potential decrement at high nerve stimulation frequencies (P < 0.05) and miniature e

117 There was no important phrenic nerve stimulation from IPL pacing.

118 the measurement of the tension generated by nerve stimulation gave evidence of any significant impai

119 Furthermore, trigeminal nerve stimulation generated sympathetically mediated low

120 ic glutamate receptors (mGluRs) by olfactory nerve stimulation generates slow (2 Hz) oscillations nea

121 t-central gyrus at 13.6-17.5 ms after median-nerve stimulation, gradually slowed down in frequency ar

122 sepsis group (eight pigs), 2) sepsis + vagus nerve stimulation group (nine pigs), and 3) control sham

123 Sacral nerve stimulation has been approved for use in treating

124 patients with depression or epilepsy, vagal nerve stimulation has been demonstrated to promote weigh

125 Sacral nerve stimulation has minimal risk and more durable long

126 owing conditioning by high-frequency sciatic nerve stimulation (HFS) at intensities recruiting C-fibe

127 icroM) inhibited contractions evoked by 4 Hz nerve stimulation in a concentration-dependent manner (I

128 determine the safety and efficacy of sacral nerve stimulation in a large population under the rigors

129 renal catecholamine secretion and splanchnic nerve stimulation in anaesthetised mice.

130 ked by laryngeal and tracheal vagal afferent nerve stimulation in anaesthetized guinea pigs.

131 Occipital nerve stimulation in cluster headache seems to offer a s

132 esponse can also be evoked by direct C-fiber nerve stimulation in infant, but not adult, mice.

133 Ca(2+) transients evoked by nerve stimulation in PDGFRalpha(+) cells showed the same

134 m patients with SSc (SScIgGs) on cholinergic nerve stimulation in rat colon tissues.

135 icating that in vivo electrical perivascular nerve stimulation in rat mesenteric small arteries cause

136 tic force in response to submaximal rates of nerve stimulation in situ producing significantly higher

137 naptic acidification in nerve termini during nerve stimulation in situ.

138 and power in response to submaximal rates of nerve stimulation in situ.

139 imary sensory cortex face area during median nerve stimulation in subjects with spinal cord injury co

140 There has been growing interest in sacral nerve stimulation in the management of both overactivity

141 Here we show that nerve stimulation in the presence of the styryl dye FM4-

142 cral neuromodulation and percutaneous tibial nerve stimulation in the treatment of men with urge inco

143 in the direction of DOR, but bilateral vagus nerve stimulation increased and reversed DOR to base-->a

144 Axial stretch and vagus nerve stimulation induced cranial displacement of the LE

145 Sympathetic nerve stimulation inhibited both contractions of distal

146 Treatment with transcutaneous vagus nerve stimulation inhibited HMGB1 levels and improved su

147 High frequency nerve stimulation inhibited peripheral spike initiation

148 Electrical vagus nerve stimulation inhibits proinflammatory cytokine prod

149 Hypoglossal nerve stimulation is a useful second-line therapy in pat

150 Transcutaneous vagus nerve stimulation is an efficacious treatment for mice w

151 incipal functions of the bladder by pudendal nerve stimulation is an exciting prospect for neurorehab

152 Trigeminal nerve stimulation is currently being evaluated as an adj

153 Hypoglossal nerve stimulation is effective in select patients with a

154 Electrical nerve stimulation is insensitive, but specific, at detec

155 Posterior tibial nerve stimulation is not covered here.

156 1) If peripheral nerve stimulation is used, optimal clinical practice sug

157 uptake after the trigeminal and hypoglossal nerves stimulation labeled the bilateral hypoglossal mot

158 sfully place percutaneous electrical phrenic nerve stimulation leads in patients on mechanical ventil

159 Nerve stimulation leads to readily detectable and reprod

160 hypothesize that left-sided low-level vagus nerve stimulation (LL-VNS) can suppress sympathetic outf

161 stimulation (via a transcutaneous electrical nerve stimulation machine to the lateral forefoot) once

162 neuromodulatory procedures such as occipital nerve stimulation may be effective for the most disabled

163 stigated 1) the feasibility of a new phrenic nerve stimulation method allowing early diaphragmatic ac

164 This suggests that vagus nerve stimulation might provide a significant therapeuti

165 Splenic nerve stimulation mimics vagal and cholinergic induction

166 vestigated whether rhythmic pulses of median nerve stimulation (MNS) could entrain brain oscillations

167 In response to mediastinal nerve stimulation, most IC neurons became excessively ac

168 Non-invasive vagus nerve stimulation (nVNS), single-transcranial magnetic s

169 onstrated a mean inspiratory lag for phrenic nerve stimulation of 23.7 ms (p < 0.001 vs null hypothes

170 enhanced the inhibitory effect of splanchnic nerve stimulation on colonic motility.

171 ect measurements of the effects of autonomic nerve stimulation on DOR.

172 pound muscle action potentials on repetitive nerve stimulation on electromyography.

173 interneurons by testing the effect of ulnar nerve stimulation on motor-evoked potentials (MEPs) elic

174 The effects of trigeminal nerve stimulation on survival rate, autonomic nervous sy

175 e that the anti-fibrillatory effect of vagus nerve stimulation on the ventricle is mediated by nitric

176 of temporary percutaneous electrical phrenic nerve stimulation on user-specified inspiratory breaths

177 Occipital nerve stimulation (ONS) is an effective treatment for me

178 ause current methods to study GTO circuitry (nerve stimulation or muscle stretch) also activate muscl

179 timulate immune cell activation, sympathetic nerve stimulation or other factors.

180 tal development and, when activated by optic nerve stimulation or visual stimuli, induce sustained de

181 positive edrophonium or abnormal repetitive nerve stimulation, or abnormal single fibre electromyogr

182 Vagus nerve stimulation paired with exposure to conditioned cu

183 These studies used peripheral nerve stimulation paired with transcranial magnetic stim

184 Vagus nerve stimulation partially or completely prevented the

185 Nerve stimulations, pharmacological block and current cl

186 Pudendal nerve stimulation (PNS) aims to maximize afferent or eff

187 ageing alters MA reactivity to perivascular nerve stimulation (PNS) and adrenoreceptor (AR) activati

188 sympathetic neurotransmission), perivascular nerve stimulation (PNS) evoked dilatation in Young but n

189 Phrenic nerve stimulation (PNS) is a common complication of card

190 t periodic diaphragm contraction via phrenic nerve stimulation (PNS) substantially reduces MV-induced

191 bition of the micturition reflex by pudendal nerve stimulation (PNS).

192 c fields powerful enough to cause Peripheral Nerve Stimulation (PNS).

193 t (up to 72%) in response to 3 Hz repetitive nerve stimulation pointed towards a neuromuscular transm

194 We have previously shown that vagal nerve stimulation prevents intestinal barrier loss in a

195 the mechanisms are poorly understood, vagal nerve stimulation prevents weight gain in response to a

196 In contrast, facial nerve stimulation produced significant LAI (P < 0.05) as

197 cral neuromodulation and percutaneous tibial nerve stimulation prove to be viable, durable options fo

198 he long-term efficacy of percutaneous tibial nerve stimulation (PTNS) in fecal incontinence (FI).

199 Percutaneous tibial nerve stimulation (PTNS) is a new ambulatory therapy for

200 ponse to bilateral anterior magnetic phrenic nerve stimulation (Ptr,stim).

201 ect disrupted emotion circuits include vagal nerve stimulation, rapid transcranial magnetic stimulati

202 ction of muscle force in situ at sub-maximal nerve stimulation rates.

203 It is unclear how vagus nerve stimulation regulates leukocyte trafficking becaus

204 Vagus nerve stimulation remains a promising, yet unproven trea

205 Pudendal sensory nerve stimulation resulted in a significant increase in

206 Pelvic nerve stimulation resulted in significant increases of c

207 Repetitive nerve stimulation revealed low amplitude of compound mus

208 tion of botulinum toxin, percutaneous tibial nerve stimulation, sacral neuromodulation, and surgical

209 pansion with fluid resuscitation, trigeminal nerve stimulation significantly attenuated sympathetic h

210 Similarly, vagus nerve stimulation significantly attenuates neutrophil su

211 Trigeminal nerve stimulation significantly increased the short-term

212 Sacral nerve stimulation significantly reduces symptoms and imp

213 oea-predominant or mixed IBS subtypes sacral nerve stimulation (SNS) alleviates IBS-specific symptoms

214 r sinus rhythm, during bilateral sympathetic nerve stimulation (SNS) and right and/or left vagus nerv

215 tudy aimed to evaluate the outcome of sacral nerve stimulation (SNS) for fecal incontinence at 5 year

216 Here, we performed bilateral sympathetic nerve stimulation (SNS) in fully innervated, Langendorff

217 In innervated hearts, sympathetic nerve stimulation (SNS) increased heart rate to a lesser

218 Sacral nerve stimulation (SNS) is an evolving treatment for con

219 : Stimulation amplitude used in sacral nerve stimulation (SNS) is at or just above the sensory

220 tudy, we performed physiological sympathetic nerve stimulation (SNS) while optically mapping cardiac

221 t viral activation of inflammation and vagal nerve stimulation, suggesting a mechanism by which tiotr

222 iac nerve abolishes TNF suppression by vagus nerve stimulation, suggesting that the cholinergic pathw

223 Left-sided low-level vagus nerve stimulation suppresses stellate ganglion nerve act

224 Together, these results establish that vagus nerve stimulation targeting the inflammatory reflex modu

225 he effects of transcutaneous auricular vagus nerve stimulation (taVNS, Cerbomed Nemos) with sham stim

226 al evidence on the feasibility of a proximal nerve stimulation technique in controlling a variety of

227 the capabilities of a non-invasive proximal nerve stimulation technique in eliciting various hand gr

228 d electrophysiological (selective electrical nerve stimulation) techniques.

229 eral low-frequency transcutaneous electrical nerve stimulation (TENS) applied on the first dorsal int

230 Transcutaneous electrical nerve stimulation (TENS) is a commonly utilized non-phar

231 amputee's phantom limb using transcutaneous nerve stimulation (TENS).

232 slight voluntary contraction and electrical nerve stimulation that each electrode recorded motor uni

233 ring normal bursting activity and antidromic nerve stimulation, the conduction delay over the length

234 During right or left vagus nerve stimulation, there was no change in the direction

235 easing cerebral perfusion, making trigeminal nerve stimulation (TNS) a promising strategy for TBI man

236 tudies have supported potential use of vagus nerve stimulation to deliver autonomic regulation therap

237 e the voltage response, enabling sympathetic nerve stimulation to increase the heart rate.

238 e no recommendation on the use of peripheral nerve stimulation to monitor degree of block in patients

239 of direct muscle responses evoked by sciatic nerve stimulation to pretransection levels over an 8-wk

240 The current study delivered median nerve stimulation to produce SEPs during a force-matchin

241 function, or with transcutaneous electrical nerve stimulation to reduce pain (Grade: conditional rec

242 ood flow (CBF) fMRI during unilateral median nerve stimulation to show that the poststimulus fMRI sig

243 SAI was tested with median nerve stimulation to the wrist preceding TMS pulse to mo

244 One example is sacral nerve stimulation to treat overactive bladder, urinary i

245 val rate at 60 minutes was 90% in trigeminal nerve stimulation treatment group whereas 0% in control

246 ed to either control, vehicle, or trigeminal nerve stimulation treatment groups.

247 Transcutaneous vagus nerve stimulation (tVNS) has been proposed to stimulate

248 Vagus nerve stimulation (up to four times daily) in RA patient

249 Sacral nerve stimulation using InterStim Therapy is a safe and

250 ifically, the authors demonstrate that vagus nerve stimulation (VNS) activates the cholinergic antiin

251 al assessed the safety and efficacy of vagal nerve stimulation (VNS) among patients with HF and a red

252 of this study was to determine whether vagus nerve stimulation (VNS) can enhance the consolidation of

253 Cervical vagal nerve stimulation (VNS) can improve left ventricular dys

254 Vagus nerve stimulation (VNS) has been shown to enhance learni

255 Vagus nerve stimulation (VNS) has been shown to exert cardiopr

256 ATEMENT Recent studies have implicated vagus nerve stimulation (VNS) in enhanced learning and memory.

257 Vagus nerve stimulation (VNS) is a bioelectronic therapy for d

258 Vagus nerve stimulation (VNS) is a common treatment for medica

259 Vagal nerve stimulation (VNS) is an alternative therapy for ep

260 ABSTRACT: Vagus nerve stimulation (VNS) is an emerging therapy for treat

261 Noninvasive, transcutaneous vagus nerve stimulation (VNS) is currently used as a treatment

262 nity, and modulation of this reflex by vagus nerve stimulation (VNS) is effective in various inflamma

263 Vagal nerve stimulation (VNS) is well established.

264 Vagus nerve stimulation (VNS) is widely used to treat drug-res

265 to investigate the effect of cervical vagus nerve stimulation (VNS) on cerebral blood flow (CBF), in

266 ot study was to evaluate the effect of Vagus Nerve Stimulation (VNS) paired with sounds in chronic ti

267 a novel strategy that uses closed-loop vagus nerve stimulation (VNS) paired with tactile rehabilitati

268 Recent research has shown that vagus nerve stimulation (VNS) paired with tones or with rehabi

269 ly shown the safety and feasibility of vagus nerve stimulation (VNS) paired with upper-limb rehabilit

270 We have previously shown that direct vagus nerve stimulation (VNS) reduces the slope of action pote

271 d cardiac response to bipolar cervical vagus nerve stimulation (VNS) reflects a dynamic interaction b

272 Vagus nerve stimulation (VNS) therapy was shown to improve per

273 gistry investigated whether adjunctive vagus nerve stimulation (VNS) with treatment as usual in depre

274 igate the effect of vagotomy (VGX) and vagus nerve stimulation (VNS), on the development and severity

275 Only vagus nerve stimulation (VNS), which continues to develop new

276 in to the same levels as implant-based vagus nerve stimulation (VNS).

277 ge animal model of progressive sepsis, vagus nerve stimulation was associated with a number of benefi

278 with either pudendal sensory nerve or pelvic nerve stimulation was examined in the female rat using c

279 Trigeminal nerve stimulation was explored as a novel resuscitation

280 The effect of facial nerve stimulation was found to be dependent on stimulati

281 Magnetic phrenic nerve stimulation was performed before the first loading

282 Percutaneous electrical phrenic nerve stimulation was used for six 2-hour sessions at 8-

283 Unilateral electrical superior laryngeal nerve stimulation was used to elicit early (R1) and late

284 e cortical, cervicomedullary, and peripheral nerve stimulation we examined in humans motor-evoked pot

285 Using optogenetic nerve stimulation, we demonstrate activity-dependent syn

286 ne an energy-efficient waveform for cochlear nerve stimulation, we used a genetic algorithm approach,

287 Responses to nerve stimulation were abolished by MRS-2500 and not obs

288 ded (i) handgrip; and (ii) median and tibial nerve stimulation were assessed using functional magneti

289 osensitive rVLM neurons evoked by splanchnic nerve stimulation were reduced by EA and then restored w

290 z; 200 ms duration) via supramaximal sciatic nerve stimulation were used to manipulate metabolic rate

291 f release, indicated by a response to single nerve stimulation, whereas Shab channels regulate repeti

292 ves during surgery involves neurophysiologic nerve stimulation, which has practical limitations.

293 They respond to an olfactory nerve stimulation with a short barrage of excitatory inp

294 xcitability by combining paired-pulse median nerve stimulation with recording somatosensory evoked po

295 6) We suggest against the use of peripheral nerve stimulation with train of four alone for monitorin

296 5) We suggest that peripheral nerve stimulation with train-of-four monitoring may be a

297 that the dose should be based on peripheral nerve stimulation with train-of-four monitoring.

298 tion frequency-dependent manner during vagus nerve stimulation, with comparable increases seen during

299 concurrent BOLD and CBF responses to median nerve stimulation, with primary signal increases in cont

300 failed, or been denied access to, occipital nerve stimulation within the UK's National Health Servic