ライフサイエンスコーパス: nervous system

1 for structural and functional support of the nervous system.

2 ble phases of dSarm signaling in the injured nervous system.

3 tability is proposed to be widespread in the nervous system.

4 iles among various cell types of the central nervous system.

5 is the main metabolic pathway in the central nervous system.

6 ate scar-free healing in the adult mammalian nervous system.

7 are crucial steps in the development of the nervous system.

8 mediating regeneration in the adult central nervous system.

9 s critical for the normal functioning of the nervous system.

10 Nogo-A may exert its effects in the central nervous system.

11 ant adipose depots, skeletal muscle, and the nervous system.

12 r control and pain processing in the central nervous system.

13 on of this major component of the Drosophila nervous system.

14 terest in the anatomy of the mouse autonomic nervous system.

15 he gastrointestinal tract, platelets and the nervous system.

16 representation of information throughout the nervous system.

17 (NMDARs) play critical roles in the central nervous system.

18 nts showed metastatic disease in the central nervous system.

19 tes to temporal accumulation of TRA-1 in the nervous system.

20 lved in the early development of the central nervous system.

21 ed in heart rate regulation by the autonomic nervous system.

22 tigating speciation and the evolution of the nervous system.

23 d phosphor-tau, respectively, in the central nervous system.

24 omical features of the whip spiders' central nervous system.

25 gical features across different parts of the nervous system.

26 ed, given their critical role in the central nervous system.

27 lopment of autoimmune disease in the central nervous system.

28 COVID-19 poses a global threat to the entire nervous system.

29 bitory motor neurotransmitter in the enteric nervous system.

30 communication across different parts of the nervous system.

31 hood from primitive cells of the sympathetic nervous system.

32 (Syt2a) during development of the zebrafish nervous system.

33 ocytes in autoimmune diseases of the central nervous system.

34 the regenerative potential in the mammalian nervous system.

35 currently known about their function in the nervous system.

36 uidance and synaptic organisation within the nervous system.

37 tical in the development and function of the nervous system.

38 and executing experiments within the central nervous system.

39 m, gastro-intestinal system, skin as well as nervous system.

40 fast excitatory synaptic transmission in the nervous system.

41 cing all of the neurons and macroglia in the nervous system.

42 THIK-1 is strongly expressed in the central nervous system.

43 Ks) are essential for the development of the nervous system.

44 or assessing microbial interactions with the nervous system.

45 meostasis and characteristically afflict the nervous system.

46 into adulthood, leading to a highly ramified nervous system.

47 log as a suppressor of proteotoxicity in the nervous system.

48 nue toward functional restoration of injured nervous systems.

49 citatory responses in peripheral and central nervous systems.

50 the organization and evolution of vertebrate nervous systems.

51 abolism, and function of both the immune and nervous systems.

52 r targets in both the central and peripheral nervous systems.

53 , 0.5 to 0.9 per 100 000 persons for central nervous system abscesses, and 24.4 to 32.9 per 100 000 p

54 additional important function in the central nervous system: acting as a sensory network, they detect

55 he blood-brain barrier and enter the central nervous system, activating neuroinflammatory responses a

56 at have previously been proposed to modulate nervous system activity and behaviours of their hosts(2,

57 c chemoreceptor-driven levels of sympathetic nervous system activity and respiratory drive.

58 ral hemodynamics and the effect of autonomic nervous system activity on these signals has received le

59 s included fatigue, subjective muscular, and nervous system AEs.

60 which the immune system affects the central nervous system after surgical trauma.

61 ker is released immediately from the central nervous system after TBI.

62 , and shortening of long bones), and enteric nervous system (aganglionosis).

63 in-2 that is highly expressed in the central nervous system and associated with vesicles in neurons.

64 oreflex, thereby stimulating the sympathetic nervous system and causing hypertension.

65 s, our understanding of how neurons form the nervous system and enable function is very fragmentary,

66 ng the function of the sensory and autonomic nervous system and endo- and exocrine glands.

67 Recent evidence has implicated the nervous system and glial family ligands (GFLs) as potent

68 F's role in translational homeostasis in the nervous system and implicate RQC dysfunction in causing

69 l type-specific alternative exons across the nervous system and leverage ENCODE and GTEx data sets to

70 ly resulting in rapid necrosis of the entire nervous system and muscle cells throughout the entire or

71 c studies of the role of nitric oxide in the nervous system and other organs.

72 ch is clinically manifested by damage to the nervous system and retinal degeneration.

73 n signals from the peripheral to the central nervous system and that exerts its effects on neurons by

74 nal communication system between the central nervous system and the gastrointestinal tract, especiall

75 ch signal that is provided by the adrenergic nervous system, and demonstrate that cell-intrinsic adre

76 reservoirs, with an emphasis on the central nervous system, and describe relevant new work in functi

77 ALT-1 is expressed broadly in the C. elegans nervous system, and neuronal IL-17-MALT-1 signaling regu

78 Their molluscan heritage, innovative nervous system, and specialized behaviors create a uniqu

79 Neural circuits in the adult nervous system are characterized by stable, cell type-sp

80 sms affecting the development of the central nervous system are poorly understood.

81 icity, but the effects on other cells of the nervous system are unknown.

82 The extent to which nervous systems are sexually dimorphic and the cellular

83 the principal glial cells of the peripheral nervous system, are now considered to be important playe

84 is imperative for the robust function of the nervous system as a whole, we tested whether activity-de

85 hesis must be finely tuned in the developing nervous system as the final essential step of gene expre

86 ation of different structures of the central nervous system, as compared to rodents.

87 i35-Bregs suppress central nervous system autoimmune diseases by inducing infectiou

88 Adult mammalian central nervous system axons have intrinsically poor regenerativ

89 In the developing nervous system, axons navigate through complex terrains

90 Central nervous system B cells have several potential roles in m

91 holds, tone response, compliance), autonomic nervous system (baroreceptor sensitivity and effectivene

92 ncompetent sensory neurons in the peripheral nervous system but also allowed temporal tuning and prop

93 g animal Trichoplax adhaerens, which lacks a nervous system but possesses single gene homologues for

94 regeneration following injury to the central nervous system, but the mechanism by which the membrane-

95 s communicate nutritional information to the nervous system, but whether they also relay signals from

96 N-glycan alterations in the nervous system can result in different neuropathological

97 ffective interventions for adult non-central nervous system cancer patients to manage cancer-related

98 wing cranial radiotherapy to control central nervous system cancers.

99 M) is a severe leukodystrophy of the central nervous system caused by mutations in subunits of the eu

100 ing (scRNA-seq) data from 727 peripheral and nervous system cell types spanning 17 mouse organs with

101 soforms that are highly expressed in central nervous system cells.

102 ng protein of 43 kDa (TDP-43) in the central nervous system characterizes frontotemporal dementia and

103 ontribution of each somatostatin to specific nervous systems circuits and behaviors in this important

104 prior opportunistic infection of the central nervous system (CNS) (odds ratio [OR], 3.7; P < .001), i

105 In the nilotinib group, central nervous system (CNS) amyloid burden was significantly re

106 tion of the immune system within the central nervous system (CNS) and altered the paradigm for succes

107 rebrospinal fluid (CSF) protects the central nervous system (CNS) and analyzing CSF aids the diagnosi

108 ive analyses of CAG expansion in ~50 central nervous system (CNS) and peripheral postmortem tissues f

109 HIV-1 reservoirs in the central nervous system (CNS) are challenging to address due to l

110 chived tissue samples from the human central nervous system (CNS) are currently available in brain ba

111 endocrine hormone synthetized in the central nervous system (CNS) as well as enterochromaffin cells o

112 The central nervous system (CNS) barriers are crucial interfaces bet

113 Central nervous system (CNS) blood vessels contain a functional

114 igand/receptor pair has key roles in central nervous system (CNS) development, mediating axonal, and

115 isability and disease progression in central nervous system (CNS) diseases such as amyotrophic latera

116 discoveries of their involvement in central nervous system (CNS) disorders, and in particular in Alz

117 tic programs that assemble the human central nervous system (CNS) during development and maintain its

118 Loss of central nervous system (CNS) FAO did not result in gross neuroan

119 t virus dissemination throughout the central nervous system (CNS) following many neurotropic virus in

120 Inflammation in the central nervous system (CNS) has been linked to demyelination an

121 The adult human central nervous system (CNS) has very limited regenerative capab

122 the AIM2 inflammasome contributes to central nervous system (CNS) homeostasis specifically through it

123 Disease involved the central nervous system (CNS) in 71 subjects (49%).

124 udy the role of myeloid cells in the central nervous system (CNS) in the pathogenesis of multiple scl

125 apeutic value of mTOR inhibitors for central nervous system (CNS) indications.

126 Central nervous system (CNS) infections cause substantial morbid

127 ver, the transcriptomic landscape of central nervous system (CNS) innate immune cells contributing to

128 Leukocyte entry into the central nervous system (CNS) is essential for immune surveillanc

129 Over time, the central nervous system (CNS) loses function and regenerative cap

130 ived monocytes, other non-microglial central nervous system (CNS) macrophage subtypes include border-

131 owing clinical radiotherapy (RT) for central nervous system (CNS) malignancies are often long-lasting

132 repair-proficient cancers, including central nervous system (CNS) malignancies.

133 therefore a candidate of interest in central nervous system (CNS) myelination and remyelination, and

134 Central nervous system (CNS) neurodegeneration is defined by a c

135 neurons expressing PSST mRNAs in the central nervous system (CNS) of Scyliorhinus canicula using in s

136 IFNs control MuPyV infection in non-central nervous system (CNS) organs, but their relative contribu

137 ary conversion is implemented in the central nervous system (CNS) remains poorly understood.

138 r is a major impediment for targeted central nervous system (CNS) therapeutics, especially with carbo

139 in metazoans, but how they enter the central nervous system (CNS) through the blood-brain barrier (BB

140 (CSF) flow dynamics in children with central nervous system (CNS) tumors before intraventricular ther

141 hibitory neurotransmitter within the central nervous system (CNS) with fast, transsynaptic, and modul

142 nt inflammatory tissue damage in the central nervous system (CNS), and none directly promote repair.

143 d roles that these cells play in the central nervous system (CNS), and this work has been greatly fac

144 e the most abundant cell type in the central nervous system (CNS), performing complex functions in he

145 sted to be present in the cephalopod central nervous system (CNS), Scaros, Croll, and Baratte only re

146 y role in synaptic plasticity in the central nervous system (CNS).

147 curring in the persistently inflamed central nervous system (CNS).

148 a fatal demyelinating disease of the central nervous system (CNS).

149 re the resident myeloid cells in the central nervous system (CNS).

150 interactions with other cells in the central nervous system (CNS).

151 eservoirs in vivo, especially in the central nervous system (CNS).

152 highly expressed by microglia in the central nervous system (CNS).

153 chitecture and the physiology of the central nervous system (CNS).

154 on the vasculature elsewhere in the central nervous system (CNS).

155 lia, the resident macrophages of the central nervous system (CNS).

156 and during axon regeneration in the central nervous system (CNS).

157 n development and pathologies of the Central Nervous System (CNS); however, their coordinating mechan

158 ne results in diffuse central and peripheral nervous system (CNS, PNS) demyelination.

159 n with a self-contained, autonomously acting nervous system, composed of 20 neurons that fall into 14

160 DHA is a key central nervous system constituent and the precursor of several

161 Nervous systems contain sensory neurons, local neurons,

162 ssue types present in metazoans, the central nervous system contains some of the highest levels of al

163 el below 1 nM, may lead to liver and central nervous system damages in humans and animals, while exis

164 stratified tissues of the mammalian central nervous system depends upon the proper migration and pos

165 Several features of the adult nervous systems develop in a "critical period" (CP), dur

166 Genes encoding cell-surface proteins control nervous system development and are implicated in neurolo

167 se observations indicate a role of CG4266 in nervous system development and function and support the

168 hat STRAP preferably targets transcripts for nervous system development and regulates AS through pref

169 However, the mechanisms of CHD7 function in nervous system development are not well understood.

170 higher or lower in PVEC vs. ABEC identified "Nervous system development" and "Response to Stress" as

171 DARs in vertebrates, and hence their role in nervous system development, due to experimental limitati

172 eft-right axis specification; components for nervous system development, including a suite of G-prote

173 ed laminin-related protein, is essential for nervous system development.

174 though Alzheimer's disease (AD) is a central nervous system disease and type 2 diabetes MELLITUS (T2D

175 dverse events, the most common of which were nervous system disorders (four [3%]) and cardiac disorde

176 erious treatment-related adverse events were nervous system disorders (three [4%] of 68 patients and

177 1-propargyl-4-styrylpiperidines for central nervous system disorders.

178 onutrient underlie several diseases, notably nervous system disorders.

179 that aberrant expression of fusogens in the nervous system disrupts neuronal individuality, which, i

180 eful for in vivo characterization of central nervous system drug candidates, neurodegenerative diseas

181 lia results in an overall compression of the nervous system due to integrin dysregulation, which caus

182 cell phenotypes that infiltrate the central nervous system during human immunodeficiency virus (HIV)

183 (without clinical symptoms of damage to the nervous system) during the search for the cause of hepat

184 ses sympathetic neuron death and sympathetic nervous system dysfunction (dysautonomia).

185 al beta1 and beta4 spectrin show exacerbated nervous system dysfunction compared to mice lacking beta

186 In a developing nervous system, endogenous electric field (EF) influence

187 ia, the resident immune cells of the central nervous system, engulf surplus neurons and synapses.

188 The enteric nervous system (ENS) coordinates essential intestinal fu

189 The enteric nervous system (ENS) exists in close proximity to lumina

190 f two main human tau isoforms in the enteric nervous system (ENS) in CD but not in UC.

191 The enteric nervous system (ENS) is a complex network constituted of

192 equilibrium, here, we show that the enteric nervous system (ENS) plays an essential and non-redundan

193 sensory neurons in the Drosophila peripheral nervous system exhibit organ sparing at the level of arb

194 RV) emerges from influences of the autonomic nervous system, fetal body and breathing movements, and

195 Despite the relevance of the peripheral nervous system for normal sensory and motor capabilities

196 s) - key regulators of central and autonomic nervous system function - contain two putative N-glycosy

197 gnaling, altered neurotransmitter control of nervous system function, and fundamental changes in beha

198 previously implicated in IL-17 signaling or nervous system function.

199 in the deep brain would advance the study of nervous system function.

200 ly shared between the peripheral and central nervous system glia, indicating common immunological fea

201 merous cell types and regions of the central nervous system, has been difficult to study due to limit

202 eminal advances in itch circuitry within the nervous system have intersected with discoveries in immu

203 major cell type found throughout the central nervous system, have general roles in the modulation of

204 The central nervous system hosts parenchymal macrophages, known as m

205 Astrocytes control multiple processes in the nervous system in health and disease.

206 Determining the cellular content of the nervous system in terms of cell types and the rules of t

207 ed with generating numbers that describe the nervous system in terms of the volumes, surfaces, length

208 ithout hearing loss, implicating the central nervous system in the generation of hyperacusis.

209 19 is also anticipated to take a toll on the nervous system in the long term.

210 ll other studied groups, is expressed in the nervous system in the sea anemone Nematostella vectensis

211 for development of the brain and peripheral nervous system in this vertebrate embryo model.

212 lthough ammonia plays essential roles in the nervous system, in particular at glutamatergic synapses,

213 MPH-220 provides a potential nervous-system-independent option to treat spasticity an

214 The sympathetic nervous system innervates peripheral organs to regulate

215 Autonomic nervous system involvement precedes the motor features o

216 In the vertebrate nervous system, ions accumulate in diffusion-limited syn

217 te that the peripherally distributed octopus nervous system is a key site for signal processing and h

218 t these receptors' expression in the central nervous system is crucial for T cell recruitment and rea

219 The Drosophila nervous system is ensheathed by a layer of outer glial c

220 The sympathetic nervous system is essential for maintenance of cardiac f

221 The interaction of the immune system and the nervous system is known to play an important role in the

222 The developing nervous system is remarkably sensitive to environmental

223 against pathogens and tumours in the central nervous system is thought to be limited owing to the lac

224 ver, heart, skeletal muscle, and the central nervous system, its use in adipose tissue has been limit

225 We find that the nervous system learns from the relative speed difference

226 to provide a detailed description of central nervous system lesions in Leigh syndrome and their biolo

227 gene exert its effect on CAD through central nervous system-lifestyle risk factors.

228 d gene expression in, central and peripheral nervous system, liver, kidney and skeletal muscle.

229 shape and reshape many regions of the adult nervous system long after the neurodevelopmental period.

230 Gap junctions are ubiquitous throughout the nervous system, mediating critical signal transmission a

231 in cancer progression, especially in central nervous system metastases.

232 ociated mRNA for three broad tissue classes (nervous system, muscle, and intestine) and two neuronal

233 of behavior, a complete understanding of the nervous system must incorporate glial cells.

234 for the fusion of cells-are expressed in the nervous system of different species under conditions of

235 tribution of Pax6 cells in the adult central nervous system of lungfishes, the closest living relativ

236 from individual cell clusters in the central nervous system of the marine mollusk Aplysia californica

237 ogically complex cells that exist throughout nervous systems of multiple species.

238 Insect nervous systems offer unique advantages for studying int

239 iple organs, with the central and peripheral nervous system often affected.

240 s here on one direction - the effects of the nervous system on immunity.

241 cted delivery of SMN-enhancing agents to the nervous system, or stems from broader defects of the mot

242 an important role in the development of the nervous system (p = 4.29 x 10(-11) with neuroticism), as

243 become drug candidates for distinct central nervous system pathologies and possessing accentuated th

244 ronmental information processing, immune and nervous system pathways and up-regulation of genetic inf

245 in E- embryos were characterized by improper nervous system patterning of the usually carefully progr

246 Compared with the somatic nervous system, pharyngeal neurons both physically assoc

247 landmark reports have demonstrated that the nervous system plays an active role in cancer initiation

248 w cholesterol/lipid metabolism in peripheral nervous system (PNS) contributes to the pathogenesis of

249 lerosis, and Parkinson's disease, peripheral nervous system (PNS) disorders such as chemotherapy-indu

250 ) is used to describe the sensory peripheral nervous system (PNS) in the tail of a cephalochordate, A

251 ation on axon regeneration in the peripheral nervous system (PNS) is the slow rate of regrowth.

252 Previous studies using cultured peripheral nervous system (PNS) neurons have demonstrated that KIF1

253 derstanding of other cells in the peripheral nervous system (PNS) remains limited.

254 the formation and function of the peripheral nervous system (PNS).

255 ological conditions affecting the peripheral nervous system (PNS).

256 aptic activity in the central and peripheral nervous system, presynaptically and postsynaptically.

257 wann cells (SCs), the glia of the peripheral nervous system, protect injured axons by virtue of a dra

258 hosphamide, systemic cytarabine, and central nervous system radiotherapy were not used.

259 nd, given their widespread expression in the nervous system, raise the possibility of a general role

260 tion of kidney, vasculature, and sympathetic nervous system, recent experimental data suggest that im

261 d neurotrophic factor (GDNF) induces enteric nervous system regeneration in mouse models of HSCR.

262 ergic inputs originating from the peripheral nervous system regulate the inflammatory immune response

263 aging ligand for mGluR2 in different central nervous system-related conditions.

264 in neurons, especially those of the central nervous system, remains unclear.

265 assessment of the emotional and sympathetic nervous system responding to real-life social experience

266 phenotype in key compartments of the central nervous system responsible for regulating feeding behavi

267 cine seeks to decode and modulate peripheral nervous system signals to obtain therapeutic control of

268 on in distinct Cre driver lines designed for nervous system-specific recombination.

269 nerve pathway between the colon and central nervous system (spinal cord and brain) that underlies th

270 Hospitalizations for endocarditis, central nervous system/spine infections, osteomyelitis, and sept

271 expression throughout the brain and central nervous system, strongest during early development, and

272 t DRG findings may be generalizable to other nervous system structures.

273 yn has acute systemic effects on the central nervous system such that structural and resting-state fu

274 e preferentially expressed in the peripheral nervous system, suggesting that their inhibition might t

275 ic processes are an integral part of central nervous system synapses(1,2); however, the molecular mec

276 In the isolated nervous system, T neurons were inhibited during the cont

277 likely gene expression profiles for muscle, nervous system, tegument, oesophageal gland, parenchymal

278 The internalization of the central nervous system, termed neurulation in vertebrates, is a

279 ease (CMT) is a neuropathy of the peripheral nervous system that afflicts ~1:2500 people.

280 thin the extracellular matrix of the central nervous system that have generated an explosion of inter

281 malignant embryonal tumor of the sympathetic nervous system that is most commonly diagnosed in the ab

282 luorescence map for the entire hermaphrodite nervous system that resolves all neuronal identities.

283 .IMPORTANCE HSV-1 is a pathogen of the human nervous system that uses its own virus-encoded proteins

284 lay this information to areas of the central nervous system that, in turn, regulate gut physiology(4)

285 organs: disseminated skin sites, the central nervous system, the heart and large joints.

286 In many parts of the nervous system these patterns are established during dev

287 nt and function of the immune, metabolic and nervous systems through dynamic bidirectional communicat

288 ntly identified itch-sensory circuits in the nervous system to inform a major new paradigm of neuroim

289 that actively relay signals from the central nervous system to SMCs via a caveolae-dependent pathway.

290 ll transcriptomic data from the entire mouse nervous system to systematically identify cell types und

291 rodites; rather, tra-1 also acts in the male nervous system to transiently suppress a sexual dimorphi

292 er axon outgrowth and synapse formation, the nervous system transitions to a stable architecture.

293 melanomas, and radiotherapy-related central nervous system tumors, which are associated with excess

294 e tissues of both the central and peripheral nervous systems was normalized, and the neurodegeneratio

295 g amyloid and tau pathology from the central nervous system, we hypothesized that cholinergic input o

296 or expressed within the developing and adult nervous system where it mostly functions to help maintai

297 s, a daunting task in the developing central nervous system where thousands of cell types are generat

298 s, mCH is found at CAC trinucleotides in the nervous system, where it is associated with transcriptio

299 acrophages and nociceptors in the peripheral nervous system, which may contribute to the sensitizatio

300 ) pathology is confined within the autonomic nervous system with no motor features, but mouse models