ライフサイエンスコーパス: neural plasticity

1 eraging the stability of ECoG interfaces and neural plasticity.

2 4,5)P(2) that couples calcium responses with neural plasticity.

3 y in pre-mRNA quality control is involved in neural plasticity.

4 o provide an important platform for studying neural plasticity.

5 oligodendrocytes, which may facilitate rapid neural plasticity.

6 ndrites of many types of neuron and regulate neural plasticity.

7 gulating the molecular mechanisms underlying neural plasticity.

8 model system with which to investigate adult neural plasticity.

9 taneous recovery, the hallmarks of long-term neural plasticity.

10 tant neural locus of reconsolidation-related neural plasticity.

11 regulate key events in physiology, including neural plasticity.

12 Recovery from stroke engages mechanisms of neural plasticity.

13 TGF-beta has also been implicated in adult neural plasticity.

14 s diverse functions in development and adult neural plasticity.

15 ological processes ranging from evolution to neural plasticity.

16 ) in the central nervous system and regulate neural plasticity.

17 rapidly altered during experience-dependent neural plasticity.

18 may play a nonproteolytic role in regulating neural plasticity.

19 eceptors because of their potential roles in neural plasticity.

20 cularly cellular and molecular mechanisms of neural plasticity.

21 olarized, suggesting that it plays a role in neural plasticity.

22 model system with which to investigate adult neural plasticity.

23 hat myelin genesis might also be involved in neural plasticity.

24 arget of rapamycin (mTOR), are implicated in neural plasticity.

25 a dynamic and flexible model of compensatory neural plasticity.

26 neurons involves glutamatergic and GABAergic neural plasticity.

27 ssion, and could possibly do so by promoting neural plasticity.

28 example of competitive, experience-dependent neural plasticity.

29 s anchored these results to previous work in neural plasticity.

30 ated electrical synapses is a common form of neural plasticity.

31 potentially deterministic factors affecting neural plasticity.

32 nto sleep, suggesting a long-lasting form of neural plasticity.

33 oal of identifying novel factors involved in neural plasticity.

34 indicates the involvement of this pathway in neural plasticity.

35 eceptor (AMPAR) trafficking is important for neural plasticity.

36 trix metalloproteinases (MMPs) as markers of neural plasticity.

37 tion can be viewed as a form of drug-induced neural plasticity.

38 ent gene regulation that may be critical for neural plasticity.

39 contribute to elucidate how alcohol disrupts neural plasticity.

40 -amblyopic eye could be a model for residual neural plasticity.

41 ng protein (CREB) has been shown to regulate neural plasticity.

42 s exhibit experience-dependent developmental neural plasticity.

43 lucidating the mechanisms and time course of neural plasticity.

44 transmitters is a critical step in long-term neural plasticity.

45 proposed as a mediator of activity-dependent neural plasticity.

46 p190 by integrin and Src, both implicated in neural plasticity.

47 ain are one of many fundamental processes of neural plasticity.

48 defects in several common in vitro models of neural plasticity.

49 receptor types may be important elements in neural plasticity.

50 cated in central roles in activity-dependent neural plasticity.

51 ith amblyopia retain a significant degree of neural plasticity.

52 h Gp1 mGluR mediates protein translation and neural plasticity.

53 that VNS-tone pairing can direct therapeutic neural plasticity.

54 tional and social processing and the role of neural plasticity.

55 target genes involved in stress response and neural plasticity.

56 central nervous system (CNS) homeostasis and neural plasticity.

57 -studied model system for activity-dependent neural plasticity.

58 oral output control, motivational drive, and neural plasticity.

59 eins such as brevican that are implicated in neural plasticity.

60 ding synapse strength, circuit function, and neural plasticity.

61 ight into myelination-centered mechanisms of neural plasticity.

62 ritical mediators of transcription-dependent neural plasticity.

63 n neuronal development and activity-mediated neural plasticity.

64 s ostensibly adhere to Hebbian principles of neural plasticity.

65 echanism can underlie such stable changes in neural plasticity?

66 ectrical microstimulation is known to induce neural plasticity [10, 11], and caudate microstimulation

67 The different forms of neural plasticity [7-9] underlying these changes have be

68 an be correlated with cellular mechanisms of neural plasticity, a novel mechanism that may explain th

69 tion, which shows that Htt has a key role in neural plasticity after injury.

70 hy subjects and modulates pathophysiological neural plasticity after sensorimotor loss, but the mecha

71 These animals also exhibited abnormal neural plasticity after training, which may be a mechani

72 g a protease with functions in apoptosis and neural plasticity, alters specific social behaviors in m

73 The role of synaptophysin in neural plasticity and ageing should be further examined.

74 ic restriction (CR) has SSRI-like effects on neural plasticity and anxiety-related behavior.

75 tor kinase B (TrkB), have important roles in neural plasticity and are required for antidepressant ef

76 ion in a number of cellular processes (e.g., neural plasticity and circadian rhythms).

77 supported that younger patients have better neural plasticity and clinical recovery than do older pa

78 ng through WAVE-1 complexes is essential for neural plasticity and cognitive behavior.

79 omeostatic signaling is thought to constrain neural plasticity and contribute to the stability of neu

80 asis for understanding Arc's contribution to neural plasticity and disease.

81 plications of such epigenetic regulation for neural plasticity and disorders.

82 s appear to be involved in the regulation of neural plasticity and emotion.

83 en cellular metabolism, gene regulation, and neural plasticity and establish a link between acetyl-Co

84 hat makes it one of the master regulators of neural plasticity and excitability.

85 f functionally linked proteins that regulate neural plasticity and glutamate-mediated synaptic activi

86 zed and have emerged as a major regulator of neural plasticity and higher brain functioning.

87 sex steroid hormones play in mediating adult neural plasticity and in neuroprotection.

88 ether the impaired synthesis of DHA affected neural plasticity and inflammatory status in the adult b

89 eceptor signalling stimulates post-ischaemic neural plasticity and intranasal treatment with C3a rece

90 n (PA) has been proposed as a tool to induce neural plasticity and is used to help neglect rehabilita

91 This process crucially depends on ongoing neural plasticity and keen sensitivity to environmental

92 generated, adult-born neurons contribute to neural plasticity and learning.

93 Thus, dysbindin is essential for adaptive neural plasticity and may link altered homeostatic signa

94 prominent molecular devices for controlling neural plasticity and memory formation.

95 ctive means of restoring a high capacity for neural plasticity and of promoting recovery from the eff

96 and provides insight into the mechanisms of neural plasticity and peripheral regeneration in humans.

97 nvolvement of sleep in protein synthesis and neural plasticity and point to a novel role for sleep in

98 These results reveal a capacity for neural plasticity and recovery that is larger than anyth

99 and with treatment effects in the setting of neural plasticity and recovery.

100 an be pharmacologically modulated to improve neural plasticity and regeneration.

101 novel rehabilitation therapies that enhance neural plasticity and sculpt motor recovery.

102 medication exposure has been shown to alter neural plasticity and shift sensitive periods in percept

103 ulating neural plasticity; low doses enhance neural plasticity and spatial memory behavior, whereas c

104 l activity during sleep mediates large-scale neural plasticity and stabilizes kinematics during early

105 of a significant number of genes related to neural plasticity and stress, as well as the dynamic reg

106 Here, I discuss a new perspective on neural plasticity and suggest how plasticity might be ta

107 Transcripts implicated in neural plasticity and survival are enriched in ventral t

108 nscription factor implicated respectively in neural plasticity and the specific expression of neurona

109 rotonin reuptake inhibitors (SSRI's) enhance neural plasticity and their ability to enhance fear exti

110 ts play distinct roles in activity-dependent neural plasticity and thus can be incorporated along wit

111 been shown to be accompanied by substantial neural plasticity and to be shaped by previous and curre

112 a key role in mediating experience-dependent neural plasticity and, thus, creates a link between the

113 channels (CaVs) drive synaptic transmission, neural plasticity, and cardiac contraction.

114 hways associated with cellular regeneration, neural plasticity, and development.

115 em cell biology, developmental neurobiology, neural plasticity, and disease mechanisms.

116 patients, such as impaired memory capacity, neural plasticity, and neurogenic processes.

117 death and neurogenesis, experience-dependent neural plasticity, and sexual differentiation.

118 ychiatric disorders derive from pathological neural plasticity, and studying the mechanisms that unde

119 and current clinical treatments that harness neural plasticity, and we offer perspectives on future d

120 ounting evidence has highlighted the immense neural plasticity apparent in the developing brain.

121 Studies of neural plasticity are also important to understand the n

122 nd the beneficial trophic effects of BDNF on neural plasticity are critical components for drug respo

123 g, but the contribution of these isoforms to neural plasticity are not well understood.

124 orphology and receptor content that underlie neural plasticity are poorly understood.

125 d its receptor and may have implications for neural plasticity arising from such experiences.

126 al prefrontal cortex could reflect change in neural plasticity as a consequence of an intense stimula

127 Studies on neural plasticity associated with brain-machine interfac

128 RGS9 levels contribute to the behavioral and neural plasticity associated with chronic opiate adminis

129 addiction shares striking commonalities with neural plasticity associated with natural reward learnin

130 These findings provide strong evidence for neural plasticity at the level of large-scale networks s

131 A mechanism of neural plasticity based on protein dissociation rather t

132 Our findings reveal the neural plasticity-based mechanism for ketamine-mediated

133 ent, It also has implications for studies of neural plasticity because a stable baseline permits iden

134 ion (LTP) is an experience-dependent form of neural plasticity believed to involve mechanisms that un

135 Chromatin is a critical regulator of neural plasticity, but basic principles of chromatin fun

136 lloproteinases that cleave lecticans mediate neural plasticity by altering the structure of ECM aggre

137 Our data advance current understanding of neural plasticity by determining the differential effect

138 t a possible mechanism by which ACh augments neural plasticity by directing activity to populations o

139 modulated by experience, which could mediate neural plasticity by optimizing the performance of the c

140 g of the activity-dependent central auditory neural plasticity changes that must certainly generate t

141 3000, including many not typically linked to neural plasticity, compared with <300 following HDACi ad

142 A large body of evidence suggests that neural plasticity contributes to learning and disease.

143 The locus of the neural plasticity could be a subpopulation of directiona

144 es different brain structures and degrees of neural plasticity dependent upon task requirements.

145 osis protease with newly discovered roles in neural plasticity, disrupts attention in mice while pres

146 Neural plasticity due to hearing loss results in tonotop

147 ependent to NMDAR-independent mechanisms for neural plasticity during aging is associated with better

148 Neural plasticity during development appears to result i

149 suggest that mPFC neurons may exhibit rapid neural plasticity during novel experiences; however, dir

150 nthesis in the amygdala may be essential for neural plasticity during this form of associative learni

151 periods are temporary windows of heightened neural plasticity early in development.

152 coding strategies facilitated by underlying neural plasticity enable the adult brain to learn from v

153 osed and are under investigation to modulate neural plasticity, enhance it when it plays an adaptive

154 Neural plasticity following brain injury illustrates the

155 estigate in vivo functional consequences and neural plasticity following cell death as well as apopto

156 future behavior-based therapies that harness neural plasticity for recovery.

157 ctions, perhaps by regulating the expression neural-plasticity genes such as brain derived neurotroph

158 functional impact of activated microglia on neural plasticity has so far been elusive.

159 Studies of experience-dependent neural plasticity have largely focused on individual syn

160 s a critical modulator of blood pressure and neural plasticity; however, the mechanism by which TNFal

161 eful changes in afferent input that modulate neural plasticity impact on behavioral markers of perfor

162 is that VNS paired with experience can drive neural plasticity in a controlled and therapeutic direct

163 terminations is essential for understanding neural plasticity in any pathological condition.

164 provides biological evidence for system-wide neural plasticity in auditory experts that facilitates a

165 onatal isolation stress alters bidirectional neural plasticity in BLA-DG synapses, which may help to

166 n (PA) has been proposed as a tool to induce neural plasticity in both healthy participants and patie

167 Significance statement: We investigated neural plasticity in dorsal striatum from rats that were

168 rent input have been evaluated as drivers of neural plasticity in healthy subjects and in small group

169 viability of metabolic mechanisms supporting neural plasticity in hippocampal slices from 24 to 30 mo

170 r mental disorders, is a potent modulator of neural plasticity in humans and has been linked to defic

171 ults are beginning to emerge from studies of neural plasticity in humans.

172 ating the cascade of events underlying adult neural plasticity in ITC.

173 galese Finches, the degree of behavioral and neural plasticity in juvenile and adult birds may be les

174 BDNF is involved in many forms of adult neural plasticity in other systems and is present in the

175 Given the history of NO in mediating neural plasticity in other systems, the results point to

176 ic, treatments that maximise what remains of neural plasticity in patients with progressive multiple

177 icacy of therapeutic interventions promoting neural plasticity in primates are not well understood.

178 Instead, the neural plasticity in sensorimotor areas is sensitive to

179 ervous system development and contributes to neural plasticity in the adult brain.

180 in demyelinating diseases and for increasing neural plasticity in the adult CNS.

181 tion in demyelinating diseases and increased neural plasticity in the adult CNS.

182 valuable opportunity not only for exploring neural plasticity in the adult human brain but also for

183 Neural plasticity in the auditory cortex of trained mice

184 An attractive approach to stimulate neural plasticity in the brain is to transplant stem cel

185 Physical exercise promotes neural plasticity in the brain of healthy subjects and m

186 or studies related to sensory processing and neural plasticity in the brain.

187 mals and provides a mechanism for continuous neural plasticity in the brain.

188 t neural pathways may limit the induction of neural plasticity in the cerebellum and thereby limit th

189 This work furthers our understanding of neural plasticity in the context of adult vision loss.

190 ntribution to corticobulbar excitability and neural plasticity in the depressor anguli oris M1.

191 nnectivity, and they provide a mechanism for neural plasticity in the developing and adult nervous sy

192 nges induced by antidepressants may regulate neural plasticity in the diseased brain, providing sympt

193 strates large-scale and seemingly ubiquitous neural plasticity in the ground squirrel brain during to

194 oral estrogen could improve memory and alter neural plasticity in the hippocampus and neocortex of mi

195 uromodulatory receptors, interact to produce neural plasticity in the LA and behavioral fear conditio

196 on of a Pavlovian fear memory and associated neural plasticity in the LA.

197 levels is an essential step in understanding neural plasticity in the mature animal.

198 tive in cocaine sensitization and associated neural plasticity in the mPFC and NAc core.

199 odor attraction correlate with bidirectional neural plasticity in the mushroom body, the associative

200 bic pathway and activate common mechanism of neural plasticity in the nucleus accumbens.

201 istically with theta oscillations to promote neural plasticity in the service of learning and memory.

202 Neural plasticity in the song control system of seasonal

203 MPA)) glutamate receptors; the potential for neural plasticity in this pathway is suggested by its ca

204 that endogenous opioids during mating induce neural plasticity in VTA dopamine neurons that appear cr

205 rb mechanisms relevant to activity-dependent neural plasticity, in which neuronal activity activates

206 They are implicated in many forms of neural plasticity including hippocampal long-term potent

207 he mammalian brain exhibits diverse types of neural plasticity, including activity-dependent neurogen

208 cAMP signaling is involved in many forms of neural plasticity, including hypersensitivity of nocicep

209 ted gene expression mediates many aspects of neural plasticity, including long-term memory.

210 to synapses is essential to several forms of neural plasticity, including long-term potentiation (LTP

211 It is proposed that AAE modulates neural plasticity induced by high-frequency hearing loss

212 Neural plasticity induced by stroke can mediate positive

213 , suggesting that this LC-mediated olfactory neural plasticity, induced under anesthesia, can store a

214 inase II (CaMKII), an important modulator of neural plasticity, interacts with syntaxin-1A to regulat

215 line intake that is optimal for the types of neural plasticity involved in cognitive function.

216 Neural plasticity is crucial for understanding the exper

217 rtant future direction for understanding how neural plasticity is expressed in brain disorders.

218 This neural plasticity is largely mediated by morphological a

219 operties during the developmental epoch when neural plasticity is most pronounced.

220 ation of glutamatergic neurotransmission and neural plasticity is not well understood.

221 One dramatic example of neural plasticity is ongoing neurogenesis in the adult b

222 However, the role of orexin signaling in neural plasticity is poorly understood.

223 Finally, functional neural plasticity is possible even when a cortical lesio

224 ently described, compelling example of adult neural plasticity is the effect of patching one eye for

225 This neural plasticity is the physical basis of associative m

226 An often-overlooked aspect of neural plasticity is the plasticity of neuronal composit

227 issue concerning visual-experience-dependent neural plasticity is whether experience is required only

228 lay an important biphasic role in modulating neural plasticity; low doses enhance neural plasticity a

229 Disruptions in neurodevelopment or neural plasticity may act alone or in combination to bri

230 damage, these findings suggest that adaptive neural plasticity may be enhanced using behavioral manip

231 symptoms, the current findings suggest that neural plasticity may contribute to smaller corpus callo

232 , we review the advances in basic studies of neural plasticity mechanisms in developing and adult ner

233 R1 and mGluR5, elicits translation-dependent neural plasticity mechanisms that are crucial to animal

234 Addictive drugs usurp neural plasticity mechanisms that normally serve reward-

235 Experience alters cortical networks through neural plasticity mechanisms.

236 It is widely held that this type of neural plasticity might involve mechanisms like long-ter

237 en the hypothesis that nucleus basalis gates neural plasticity necessary for instrumental learning.

238 functions such as RNA and protein synthesis, neural plasticity, neurotransmission, and metabolism.

239 However, neither neural plasticity nor a new dimension of sensory experie

240 in the establishment of acquisition-relevant neural plasticity, not simply in the expression of the l

241 ng pathways may contribute to the changes in neural plasticity observed during brain development.

242 ategic and nonsensory factors and implicated neural plasticity occurring in both low- and high-level

243 Within the amygdala and cerebellum, neural plasticity occurs because of convergence of these

244 inal study, we investigated the auditory and neural plasticity of musical learning in 111 young child

245 afferent inputs on cortical excitability and neural plasticity often used transcranial magnetic stimu

246 f this finding for discerning the effects of neural plasticity over and above normal brain maturation

247 mates may be appropriate for preservation of neural plasticity over their longer life span and is rel

248 cruiting the CaMKIIalpha-BDNF-CREB-dependent neural plasticity pathways.

249 Memory and its underlying neural plasticity play important roles in sensory discri

250 Neural plasticity plays a critical role in learning, mem

251 ormation of new memories by interfering with neural plasticity processes in the adult brain.

252 factor (BDNF) is a key positive regulator of neural plasticity, promoting, for example, the actions o

253 Precisely targeted neural plasticity provides a new avenue for the treatmen

254 f the initiation of developmental windows of neural plasticity; pubertal hormones may trigger the ope

255 bernanthalog was found to promote structural neural plasticity, reduce alcohol- and heroin-seeking be

256 Both DARPP-32 and neural plasticity regulator activity-regulated cytoskele

257 ulture, ketamine increased expression of the neural plasticity-related protein Arc, and this was prev

258 tical role of orexin signaling in the VTA in neural plasticity relevant to addiction.

259 However, rTMS-induced neural plasticity remains insufficiently understood at t

260 Neural plasticity represents a crucial mechanism of the

261 Although extensive research on neural plasticity resulting from hearing deprivation has

262 ocused nearly exclusively on the analysis of neural plasticity resulting from paired stimuli.

263 y implanted human subjects and suggests that neural plasticity resulting from previous deafness and d

264 ily attended, suggesting that the underlying neural plasticity selectively engages when stimuli are b

265 challenging patients and athletes to promote neural plasticity, skilled performance, and recovery.

266 refore, seems to orchestrate two features of neural plasticity-somatic disinhibition and compartmenta

267 jor active zone protein presumed to regulate neural plasticity, specifically in the synaptic plasma m

268 thesis can modify the brain inflammatory and neural plasticity status, supporting the view that DHA i

269 Our results provide direct evidence for neural plasticity that compensates for the deficiency in

270 vity-dependent spinal stimulation can induce neural plasticity that improves behavioral recovery afte

271 se, such as cocaine, cause stable changes in neural plasticity that in turn drive long-term changes i

272 g protein (CREB) is a critical integrator of neural plasticity that is responsive in a brain region-s

273 vation (MD) is a model of activity-dependent neural plasticity that is restricted to an early critica

274 Homeostatic scaling is a non-Hebbian form of neural plasticity that maintains neuronal excitability a

275 Kindling is a model of the neural plasticity that occurs following stimulation to t

276 ssible and impossible images and demonstrate neural plasticity that predicts behavioral priming for s

277 These results reveal a novel form of neural plasticity, that epileptogenic stimulation can se

278 ther in the hippocampus, through homeostatic neural plasticity, the olfactory bulb or the hypothalamu

279 h of which is subject to or an expression of neural plasticity-the capacity of neurons to change thei

280 Through neural plasticity, this extensive movement training shou

281 ng development and is an integral feature of neural plasticity throughout life.

282 thought to interface with the mechanisms of neural plasticity to achieve stable yet flexible neural

283 y input and motor output and (2) controlling neural plasticity to achieve the desired behavior of the

284 findings, and hampered the ability to relate neural plasticity to behavior.

285 tial therapeutic application in manipulating neural plasticity to treat a variety of conditions, incl

286 doxical roles for REST/NRSF in neurogenesis, neural plasticity, tumour suppression and cancer progres

287 se that Notch plays an important role in the neural plasticity underlying consolidated memory.

288 minance plasticity in visual cortex, and the neural plasticity underlying learning and memory.

289 omise expression of proteins involved in the neural plasticity underlying learning.

290 abilization molecules, appear to mediate the neural plasticity underlying specific forms of long-term

291 is demonstrated by improved performance and neural plasticity underlying that improvement after slee

292 Adult neurogenesis is thought to provide neural plasticity used in forming and storing new memori

293 te mechanisms underlying this nonassociative neural plasticity using in vivo and in vitro preparation

294 een shown to support cognitive functions and neural plasticity, we generated CD3zeta(-/-) mice in whi

295 C3a receptor signalling in ischaemia-induced neural plasticity, we subjected C3a receptor-deficient m

296 Val66Met SNP alters SSRI-induced changes in neural plasticity, we used wild-type (BDNF(Val/Val)) mic

297 ese functions are often tied to processes of neural plasticity whether in the hippocampus, through ho

298 fines a temperature-driven model of dramatic neural plasticity, which provides a unique opportunity t

299 , we used the monocular deprivation model of neural plasticity, which shares many common mechanisms w

300 ation by PKC represents a novel mechanism of neural plasticity with potentially significant implicati