ライフサイエンスコーパス: neurocognitive

1 sorder characterized by hypersociability and neurocognitive abnormalities.

2 A comprehensive neurocognitive account of conscious awareness will not b

3 Here we update and review a unifying neurocognitive account-the Procedural circuit Deficit Hy

4 , and VCAM-1+ endothelial cells and improved neurocognitive activity, without blocking graft-versus-l

5 roduction and microglial MHC-II and improved neurocognitive activity.

6 tributable to study drug, which were grade 2 neurocognitive adverse events comprising slowed speech a

7 in the plasma, incident diabetes mellitus or neurocognitive adverse events.

8 ve understanding of the neural mechanisms of neurocognitive aging in our own species.

9 eep apnoea, a disorder associated with major neurocognitive and cardiovascular sequelae.

10 onents enable prediction of a broad array of neurocognitive and clinical symptom variables at levels

11 locentric representations and the underlying neurocognitive and computational mechanisms; and within

12 n linked to a separate condition that causes neurocognitive and congenital anomalies.

13 This neurocognitive and immunologic ability for humans to sym

14 In order to integrate neurocognitive and metabolic parameters, we performed un

15 psychotic disorders and establish clinical, neurocognitive and neuroanatomic associations with incre

16 The A5199 INS assessed neurocognitive and neurological performance within a ran

17 he relationship between network features and neurocognitive and psychotic scores was also assessed, r

18 fferent data modalities, including clinical, neurocognitive, and neurobiological data.

19 The axes of the low-dimensional neurocognitive architecture aligned with regional differ

20 going experience can be inferred from hidden neurocognitive architecture and demonstrate that perform

21 ntly, individual variation in the underlying neurocognitive architecture is hypothesised to determine

22 ch describes the distributed and interactive neurocognitive architecture of representations and opera

23 data suggest that KCTD13 contributes to the neurocognitive aspects of patients with the BP4-BP5 dele

24 viors across a number of domains, and direct neurocognitive assessment of 158 preschool aged children

25 andardized average differences in individual neurocognitive assessment scores over the 5.6-year (rang

26 icipants with a Fontan circulation who had a neurocognitive assessment, 55% were male and the mean ag

27 concurrent lumbar puncture, phlebotomy, and neurocognitive assessment.

28 exposure, true predrug baseline imaging and neurocognitive assessments are needed.

29 al anatomical magnetic resonance imaging and neurocognitive assessments in rhesus macaques.

30 Neurocognitive assessments were performed during therapy

31 Patients underwent neurocognitive assessments, and the debris captured was

32 MI and severe CHCs partially mediated the PA-neurocognitive associations, but the mediation effects w

33 ond creation and maintenance, as well as the neurocognitive basis of social isolation and its deep co

34 This study examines the underlying neurocognitive basis of textual knowledge structure and

35 e psychosis cohort and the Penn Computerized Neurocognitive Battery in the PNC.

36 and systolic LV function; and a standardized neurocognitive battery to assess memory, executive funct

37 Scores from a neurocognitive battery were used to assess cognitive fun

38 tion was assessed with the Penn Computerized Neurocognitive Battery.

39 Measures sensitive to early neurocognitive changes associated with ARHL would help t

40 schizophrenia patients; no concurrent acute neurocognitive changes were detected by the MCCB.

41 tion study-based genetic, environmental, and neurocognitive classifiers were trained to separate 337

42 le for clarifying the aetiologies underlying neurocognitive clinical syndromes.

43 istered 6 validated sleep questionnaires and neurocognitive (Cogstate) testing pre-switch and over 18

44 The clustering of patients with neurocognitive complaints could not be completely explai

45 Understanding neurocognitive computations will require not just locali

46 Delirium is a serious acute neurocognitive condition frequently occurring for hospit

47 This is novel evidence on the neurocognitive correlates of reading proficiency, highli

48 PA was collected at baseline, and PA and neurocognitive data were obtained 7 (1-12) years and 12

49 was a correlation between lesion volume and neurocognitive decline (p = 0.0022).

50 Progressive neurocognitive decline and death were explained by activ

51 nue for large-scale preclinical screening of neurocognitive decline as a new digital biomarker, as we

52 Neurocognitive decline was assessed using standardized c

53 ight into the neural mechanisms underpinning neurocognitive decline with ARHL and its temporal sequen

54 y and other HIV-related disorders, including neurocognitive decline.

55 achnoid hemorrhage (aSAH) and contributes to neurocognitive decline.

56 type 2 diabetes, cardiovascular disease, and neurocognitive decline.

57 Radiation-induced neurocognitive decrements in immunocompetent mice can be

58 are at increased risk for language and other neurocognitive deficits compared to term controls (TC).

59 with OSAS that may help explain some of the neurocognitive deficits described in these children.

60 e to MeHg has been associated with long-term neurocognitive deficits in children that persist into ad

61 HIV-associated neurocognitive deficits include impaired speed-of-inform

62 Neurocognitive deficits manifest across multiple cogniti

63 lymphoblastic leukemia (ALL) are at risk for neurocognitive deficits that are associated with treatme

64 eral consistent findings emerge on symptoms, neurocognitive deficits, and neuroimaging parameters and

65 STATEMENT Even with antiretroviral therapy, neurocognitive deficits, including impairments in attent

66 c health conditions, impaired health status, neurocognitive deficits, or poorer socioeconomic outcome

67 of patients with bipolar disorder (BD) have neurocognitive deficits.

68 ith schizophrenia, including the presence of neurocognitive deficits; abnormalities in brain structur

69 mine the roles of the social environment and neurocognitive development in adolescents' natural resil

70 tion is likely to reflect, at least in part, neurocognitive development.

71 ated to poverty interact to shape children's neurocognitive development.

72 Since surviving patients experience severe neurocognitive disabilities, better and more effective t

73 influences the severity of HIV-1-associated neurocognitive disease, a cellular or molecular basis fo

74 HIV-associated neurocognitive disorder (HAND) is a common condition in

75 HIV-1 associated neurocognitive disorder (HAND) is characterized by neuro

76 tes with cognitive decline in HIV-associated neurocognitive disorder (HAND) patients.

77 brovascular disease (CVD) and HIV-associated neurocognitive disorder (HAND).

78 d potentially ongoing role in HIV-associated neurocognitive disorder (HAND).

79 The number of people with neurocognitive disorder is increasing, and the majority

80 Dementia (also known as major neurocognitive disorder) is defined by a significant dec

81 cannot be better explained by a pre-existing neurocognitive disorder.

82 however, the prevalence of HIV-1-associated neurocognitive disorders (HAND) is still increasing.

83 ecreased, but milder forms of HIV-associated neurocognitive disorders (HAND) persist along with motor

84 ecreased, but milder forms of HIV-associated neurocognitive disorders (HAND) persist along with motor

85 1 in the brain often leads to HIV associated neurocognitive disorders (HAND), such as encephalitis an

86 us type 1 (HIV-1) suffer from HIV-associated neurocognitive disorders (HAND).

87 ological disorders, including HIV-associated neurocognitive disorders (HAND).

88 ation of the progression of HIV-1 associated neurocognitive disorders (HAND).

89 y disease, may play a role in HIV-associated neurocognitive disorders (HAND).

90 d individuals still develop HIV-1 associated neurocognitive disorders (HAND).

91 stress and the occurrence of HIV-associated neurocognitive disorders (HAND).

92 ed or with various degrees of HIV-associated neurocognitive disorders (HAND).

93 ction, who may be at risk for HIV-Associated Neurocognitive Disorders (HAND).

94 panied with the prevalence of HIV-associated neurocognitive disorders (HANDs) and risk of comorbiditi

95 HIV-associated neurocognitive disorders (HANDs) are a frequent outcome

96 The neuropathology underlying HIV-associated neurocognitive disorders has not been well characterized

97 HIV-associated neurocognitive disorders prevail in 20-50 percent of inf

98 itive impairments, that is, HIV-1-associated neurocognitive disorders remain prevalent potentially du

99 nst latent HIV-1 infection, HIV-1 associated neurocognitive disorders, and other HIV-1 comorbidities.

100 cular disease, certain types of cancers, and neurocognitive disorders, as well as leaving them expose

101 HIV-associated neurocognitive disorders, however, continue to be a majo

102 n regions have been implicated in a range of neurocognitive disorders.

103 ons such as delirium and other perioperative neurocognitive disorders.

104 ed as a mediating factor of HIV-1 associated neurocognitive disorders.

105 promising novel targets for the treatment of neurocognitive disorders.

106 esents a novel target against HIV-associated neurocognitive disorders.

107 ving with HIV-1 suffer from mild to moderate neurocognitive disorders.

108 current copy number variants associated with neurocognitive disorders.

109 testing had severe impairment in at least 1 neurocognitive domain at the most recent evaluation.

110 Psychological studies shed light on the neurocognitive domains implicated in PD-ICBs and identif

111 Neurocognitive domains of attention (Trial Making Test P

112 on (MNC) and -1.5 standard deviations in >=2 neurocognitive domains were secondary outcomes of NCI.

113 nimal effect in subsequent years or on other neurocognitive domains.

114 Such events may underlie METH- exacerbated neurocognitive dysfunction in HIV-infected patients.

115 ts with a Fontan circulation had more marked neurocognitive dysfunction than adolescents with a Fonta

116 cts of auditory social cognitive and general neurocognitive dysfunction.

117 on framework for understanding affective and neurocognitive dysfunctions across multiple disorders, i

118 spectrum disorder (ASD) is characterized by neurocognitive dysfunctions, such as impaired social int

119 n type-9) inhibition was not associated with neurocognitive effects in a recent phase 3 randomized tr

120 oprotein cholesterol are not associated with neurocognitive effects in blacks.

121 and neuronal activity, and are sensitive to neurocognitive effects of acute and chronic alcohol expo

122 are a growing concern due to their possible neurocognitive effects, with research showing a season o

123 in adolescence is associated with long-term neurocognitive effects.

124 posure, CONV caused permanent alterations in neurocognitive end points, whereas FLASH did not induce

125 Impulsivity has been suggested as a neurocognitive endophenotype conferring risk across a nu

126 Children underwent neurocognitive evaluation at enrollment (community child

127 its 5 and 6, participants underwent detailed neurocognitive evaluation.

128 and death certificate codes, and the visit 6 neurocognitive evaluation.

129 ognitive impairment at visit 6, based on the neurocognitive evaluation.

130 close monitoring, for the increased risk of neurocognitive events in the ongoing outcome studies and

131 Despite concerns about adverse neurocognitive events raised by prior trials, pharmacolo

132 Neurological and neurocognitive events were similar among the 3 groups.

133 ll treatment-emergent adverse event rates or neurocognitive events, although cataract incidence appea

134 ation among 42% who attended a comprehensive neurocognitive examination (2011-2013).

135 ate the study of genetic, environmental, and neurocognitive factors within a longitudinal framework,

136 ome of which show trends of association with neurocognitive faculties and symptoms.

137 We then describe present neurocognitive findings that suggest that these processe

138 We present a unifying neurocognitive framework of mechanisms underlying inform

139 llations in top-down control and extend this neurocognitive framework to preschoolers.

140 e and has received ample attention in recent neurocognitive frameworks.

141 by which food insecurity is associated with neurocognitive function among women living with or at ri

142 mine the association of food insecurity with neurocognitive function among women living with or witho

143 culation performed worse in several areas of neurocognitive function compared with those with transpo

144 Drugs that rescue synapses may improve neurocognitive function in HAND.SIGNIFICANCE STATEMENT S

145 use disorder is associated with dysregulated neurocognitive function in the right inferior frontal gy

146 security was associated with domain-specific neurocognitive function in women, and HIV serostatus mod

147 that contribute to brain injury to optimize neurocognitive function is paramount.

148 Neurocognitive function was assessed by using Cogstate s

149 and 5.6% in patients with devices (p = 0.25) Neurocognitive function was similar in control subjects

150 ata provide new insight into the genetics of neurocognitive function with relevance to understanding

151 s of Daily Living (ADL) and various tests of neurocognitive function, motor performance and mood stab

152 ts a barrier to HIV remission and may affect neurocognitive function.

153 abetes, cardiovascular disease, and impaired neurocognitive function.

154 ree survival (PFS) with a goal of preserving neurocognitive function.

155 statins or ACEI/ARB have an effect on global neurocognitive function.

156 n addition, higher FW correlated with better neurocognitive functioning following 12 weeks of antipsy

157 This study aimed to investigate neurocognitive functioning in adolescents and adults wit

158 ns suggest that lithium may be beneficial to neurocognitive functioning in patients with BD and that

159 bal brain volumes were associated with worse neurocognitive functioning in several domains (P<0.05).

160 methodically test the effects of lithium on neurocognitive functioning in the largest single cohort

161 We previously reported improved neurocognitive functioning with ART initiation in 7 reso

162 comprehensive assessment of clinical scales, neurocognitive functioning, and fMRI of unexpected finan

163 everaged to produce generalizable markers of neurocognitive functioning.

164 e effect of lithium treatment across time on neurocognitive functioning.

165 f specific underlying deficits and biases in neurocognitive functions.

166 PSCK9 inhibitors, rates of musculoskeletal, neurocognitive, gastrointestinal, or other adverse event

167 s of vitamin B-12 deficiency and its role in neurocognitive health.

168 imited data on the comparative prevalence of neurocognitive impairment (NCI) in aging people living w

169 Neurocognitive impairment (NCI) is strongly associated w

170 Neurocognitive impairment (NCI) persists among women liv

171 Self-reported neurocognitive impairment (srni) in people living with h

172 Self-reported neurocognitive impairment (SRNI) in people living with h

173 ve impairment.Although overall self-reported neurocognitive impairment (SRNI) is decreasing in the Sw

174 ted as a possible modifiable risk factor for neurocognitive impairment and dementia.

175 To evaluate the association between global neurocognitive impairment and visual field variability i

176 Almost half of HIV+ participants had neurocognitive impairment at baseline before ART, based

177 IV+ adults enrolled in ACTG 5199, 55% had no neurocognitive impairment at baseline.

178 s are most susceptible to the progression of neurocognitive impairment caused by ageing in individual

179 me loss, mineralization, microangiopathy and neurocognitive impairment in survivors of childhood acut

180 Neurocognitive impairment is common in adolescents and a

181 there were significant overall reductions in neurocognitive impairment over time, especially in those

182 Neurocognitive impairment remains a common complication

183 Mild neurocognitive impairment was found in 25%, moderate in

184 eristics should be preferentially tested for neurocognitive impairment.

185 o INS to provide previously unknown rates of neurocognitive impairment.

186 ause widespread neuronal apoptosis and later neurocognitive impairment.

187 identify children at high risk of long-term neurocognitive impairment.

188 eristics should be preferentially tested for neurocognitive impairment.Although overall self-reported

189 long cART, exhibits age-related, progressive neurocognitive impairments (NCI), including alterations

190 ors are left with permanent and debilitating neurocognitive impairments as a result of this therapy,

191 Neurocognitive impairments have emerged as clinically im

192 ellar stimulation interventions for specific neurocognitive impairments.

193 In addition, CD is associated with neurocognitive impairments; smaller grey matter volume i

194 At follow-up a significant neurocognitive improvement in the global cognitive index

195 rences ranging from -7.9 to -2.2) and larger neurocognitive improvements over time (-6.0 to -2.5), al

196 nsory systems across multiple timescales and neurocognitive loci.

197 fect may represent a computationally defined neurocognitive mechanism by which the drug could enhance

198 s theory proposes that laws can be traced to neurocognitive mechanisms and ancestral selection pressu

199 developmental trajectory and the underlying neurocognitive mechanisms are still little understood.

200 documents both types of CCD prediction, the neurocognitive mechanisms giving rise to these predictio

201 However, little is known regarding the neurocognitive mechanisms linking Ptau and memory-relate

202 However, the underlying neurocognitive mechanisms of this phenomenon are unknown

203 Programming languages might 'recycle' neurocognitive mechanisms originally developed for natur

204 tion preparation and decision-making but the neurocognitive mechanisms remain unclear.

205 We argue that careful delineation of the neurocognitive mechanisms supporting human-robot interac

206 mmon currency hypothesis and shed insight on neurocognitive mechanisms underlying information-seeking

207 o the framework, to help clarify the complex neurocognitive mechanisms underlying recollection and fa

208 Here, we investigate the neurocognitive mechanisms underlying these results, test

209 emotional SFRs may be the result of complex neurocognitive mechanisms which lead to partial mimicry

210 t decisional stages underpinned by different neurocognitive mechanisms, i.e., sensitivity to unfairne

211 We propose a neurocognitive model with three core streams; face proce

212 While current dual-steam neurocognitive models of language function have coalesce

213 These findings challenge current neurocognitive models of music-evoked pleasure and highl

214 Researchers have considered 4 neurocognitive models of urgency: excessive emotion gene

215 vity in cerebrospinal fluid (CSF) as well as neurocognitive (NC) performance change in participants s

216 vity in cerebrospinal fluid (CSF) as well as neurocognitive (NC) performance change in participants s

217 ve reported altered integrity of large-scale neurocognitive networks (NCNs) in dementing disorders.

218 apply normative data from the International Neurocognitive Normative Study (INNS) to INS to provide

219 was independently associated with favorable neurocognitive outcome in propensity-adjusted analysis (

220 engers was superior with regard to long-term neurocognitive outcome.

221 fied associations between PA consistency and neurocognitive outcomes (expected mean, 50; standard dev

222 lasms (SMNs), chronic health conditions, and neurocognitive outcomes among 6,148 survivors of childho

223 the association between aerobic fitness and neurocognitive outcomes at young adult age, along with t

224 Neurocognitive outcomes beyond childhood in people with

225 There were no significant differences in neurocognitive outcomes for patients receiving IT MTX co

226 concentration was inversely associated with neurocognitive outcomes in mitochondrial (proximal) rath

227 Clinical factors associated with worse neurocognitive outcomes included more inpatient days dur

228 of CSF CA-DNA HIV was associated with worse neurocognitive outcomes including global deficit score (

229 s identified by newborn screening had better neurocognitive outcomes than those diagnosed after the m

230 gression was used to evaluate microbleeds on neurocognitive outcomes, adjusting for age at diagnosis

231 ury, and microhemorrhage was associated with neurocognitive outcomes.

232 However, PA was not associated with the neurocognitive parameters evaluated.

233 Our results revealed the underlying neurocognitive patterns associated with information inte

234 t individuals experienced in the laboratory: neurocognitive patterns linked to better performance at

235 (CSF) and associations with inflammation and neurocognitive performance during long-term ART.METHODSP

236 e, associations between driving behavior and neurocognitive performance in glaucoma are unexplored.

237 ted the hypothesis that VF loss severity and neurocognitive performance interact to influence simulat

238 In summary, although modest declines in neurocognitive performance were seen in single domains w

239 between food insecurity and domain-specific neurocognitive performance, adjusting for relevant socio

240 ly associated with greater VF loss and worse neurocognitive performance, suggesting both factors cont

241 in expected regions correlates strongly with neurocognitive performance.

242 in expected regions is highly correlated to neurocognitive performance.

243 administered to assess global and sub-domain neurocognitive performance.

244 nd their detection is associated with poorer neurocognitive performance.FUNDINGThis observational stu

245 concept is inappropriate for explanations of neurocognitive phenomena.

246 Monitoring is a complex multidimensional neurocognitive phenomenon.

247 including RLIM cause a recognizable but mild neurocognitive phenotype in hemizygous males.

248 nherited X chromosome variants in males with neurocognitive phenotypes continues to present a challen

249 g a state-of-the-art battery of self-report, neurocognitive, physiologic, digital phenotyping, psycho

250 cer who report more consistent PA have fewer neurocognitive problems and larger improvements in these

251 res, consistent PA was associated with fewer neurocognitive problems compared with consistent inactiv

252 Neurocognitive problems during anaesthesia recovery are

253 d network dysfunction that may be related to neurocognitive problems in this devastating disorder.

254 years) and 720 siblings self-reported PA and neurocognitive problems.

255 I conclude that volition is a neurocognitive process of enormous societal importance a

256 ion between pediatric anxiety and a specific neurocognitive process), and then review extant translat

257 These neurocognitive processes appear remarkably parallel for

258 ve implications for the understanding of the neurocognitive processes leading to excessive uncertaint

259 a (AHP) can provide unique insights into the neurocognitive processes of motor awareness.

260 re, we review and synthesize research on key neurocognitive processes that emerge as potential target

261 nds upon existing research by examining core neurocognitive processes that may result in reading diff

262 by integrating research on psychological and neurocognitive processes with a current understanding of

263 rience depend on the expression of different neurocognitive processes.

264 We assessed tolerance, neurocognitive progression, brain growth, NAGLU enzymati

265 ificantly underestimated the degree to which neurocognitive representations are distributed and varia

266 ctions engaging (subsets of) this area share neurocognitive resources, whereas others rely on separab

267 No significant difference was found in neurocognitive scores with either the absence or presenc

268 The adverse neurocognitive sequelae following clinical radiotherapy

269 and allowed patients to avoid the potential neurocognitive sequelae of WBRT.

270 Executive function (EF) skills are neurocognitive skills that support the reflective, top-d

271 recognition into a continuous frontotemporal neurocognitive space.

272 h the maintenance of and transitions between neurocognitive states.

273 with incomplete viral suppression and known neurocognitive status.

274 ims to synthesize the existing literature on neurocognitive, structural neuroimaging, and functional

275 bis and NTP group jointly considered; and 4) neurocognitive, structural neuroimaging, or functional n

276 We sought to identify neurocognitive subtypes and their neurofunctional signat

277 We identified three neurocognitive subtypes in the depressed group.

278 ine learning algorithm was used to delineate neurocognitive subtypes.

279 ltiple languages places major demands on our neurocognitive system, which can impact the way the brai

280 tools for building and developing models of neurocognitive systems like the brain.

281 lexico-semantic and sublexical/phonological neurocognitive systems.

282 rm more like heterosexual women on important neurocognitive tasks on which men score higher than wome

283 We administered a comprehensive neurocognitive test battery at baseline and 2 y.

284 Baseline and follow-up neurocognitive test performance was analyzed for all boy

285 g group had the highest social cognitive and neurocognitive test scores.

286 this retrospective cross-sectional analysis, neurocognitive testing and 3 T brain MRI's were obtained

287 nts (67%) with available long-term follow-up neurocognitive testing had severe impairment in at least

288 Neurocognitive testing shows that cognitive impairment i

289 , laboratory analysis, physical fitness, and neurocognitive testing were done.

290 individuals with UCDs who had comprehensive neurocognitive testing with a cumulative follow-up of 70

291 d routine clinical lab markers, computerized neurocognitive testing, and symptom self-reports.

292 ells is associated with lower performance on neurocognitive testing.

293 o examine potential group differences across neurocognitive tests [California Verbal Learning Test (C

294 s of participants; differences on social and neurocognitive tests completed outside the scanner were

295 ialysis, we performed a set of comprehensive neurocognitive tests that included the cognitive domains

296 ecords, representative surveys, computerized neurocognitive tests, and blood samples, Army STARRS and

297 standardized continuous scores on individual neurocognitive tests.

298 , socio-technical systems engineering, and a neurocognitive theory with abstract representations of g

299 Performance on eight neurocognitive variables targeting reading outcomes (eg,

300 Surprisingly, potential neurocognitive vulnerabilities contributing to atypical