ライフサイエンスコーパス: neuroendocrine cell

1 ing the effect of FTY-720 on the survival of neuroendocrine cells.

2 s onto identified preautonomic, but not onto neuroendocrine cells.

3 transporter, from single vesicles in living neuroendocrine cells.

4 teps during dense-core vesicle exocytosis in neuroendocrine cells.

5 ndamental regulatory organization of diverse neuroendocrine cells.

6 nd promoted liposome fusion and secretion in neuroendocrine cells.

7 n but had no effect on ciliated or pulmonary neuroendocrine cells.

8 on PVN and peri-PVN interneurons and not on neuroendocrine cells.

9 to Golgi membranes and synaptic vesicles in neuroendocrine cells.

10 ranin family of pro-hormone cargo of DCGs in neuroendocrine cells.

11 sponsible for pulsatile hormone release from neuroendocrine cells.

12 ially by ciliated cells, with an increase in neuroendocrine cells.

13 e receptor complex on the plasma membrane of neuroendocrine cells.

14 exocytosis of large dense-core vesicles from neuroendocrine cells.

15 s to all epithelial cell lineages, including neuroendocrine cells.

16 and activated UCHL1, a marker for pulmonary neuroendocrine cells.

17 tion and altered morphology of the pulmonary neuroendocrine cells.

18 reted by islet delta-cells and by extraislet neuroendocrine cells.

19 OR export from intracellular compartments in neuroendocrine cells.

20 pendent exocytosis of dense-core vesicles in neuroendocrine cells.

21 cassette in PAC1-mediated Ca2+ signaling in neuroendocrine cells.

22 secretory vesicle-plasma membrane fusion in neuroendocrine cells.

23 ing until eggs filling the uterus deform the neuroendocrine cells.

24 factor-binding protein) on SG maturation in neuroendocrine cells.

25 rotransmitters and hormones from neurons and neuroendocrine cells.

26 ations resulting from IA-2 knockout in other neuroendocrine cells.

27 lates the differentiation or function of the neuroendocrine cells.

28 h possible intracellular roles for SNAP25 in neuroendocrine cells.

29 ely unaltered by actin disruption, unlike in neuroendocrine cells.

30 cally by repressing IIS ligand expression in neuroendocrine cells.

31 ssical routes for secretion of proteins from neuroendocrine cells.

32 gulation of this process in both neurons and neuroendocrine cells.

33 tide in embryonic and adult murine pulmonary neuroendocrine cells.

34 ressed selectively in central and peripheral neuroendocrine cells.

35 of exocytosis and endocytosis in neurons and neuroendocrine cells.

36 ndent dense-core vesicle (DCV) exocytosis in neuroendocrine cells.

37 ne confers such a pro-secretory phenotype on neuroendocrine cells.

38 r gene expression to different sets of islet neuroendocrine cells.

39 e-stage tumors are composed predominantly of neuroendocrine cells.

40 n urine bombesin-like peptide and numbers of neuroendocrine cells.

41 for secretory vesicle biogenesis in neuronal/neuroendocrine cells.

42 integral component of secretory granules in neuroendocrine cells.

43 ane PAM serve as indicators of granule pH in neuroendocrine cells.

44 d to secretory granules in transfected AtT20 neuroendocrine cells.

45 dense-core vesicle exocytosis in vertebrate neuroendocrine cells.

46 tic hormone (AKH) secretion from specialized neuroendocrine cells.

47 e enzyme does not require features unique to neuroendocrine cells.

48 n-containing proteins by RNA interference in neuroendocrine cells.

49 regulate Ca(2+)-dependent DCV exocytosis in neuroendocrine cells.

50 rane thickness, nerve fibers, and epithelial neuroendocrine cells.

51 ration in isolated vesicles from neurons and neuroendocrine cells.

52 s neuritogenesis, upon GPCR-Gs activation in neuroendocrine cells.

53 nt with autonomic and C fibers and pulmonary neuroendocrine cells.

54 eta) were previously found to be enriched in neuroendocrine cells.

55 ia three discrete cAMP sensors identified in neuroendocrine cells.

56 disease, as well as the function of gastric neuroendocrine cells.

57 SCLC is thought to derive from pulmonary neuroendocrine cells.

58 rentiation and activating Uchl1, a marker of neuroendocrine cells.

59 in various cell types, notably in nerve and neuroendocrine cells [1, 2].

60 e pixels per mm(2), P = .02), and epithelial neuroendocrine cells (4.9/mm(2) [25th-75th IQR, 0-16.4/m

61 During differentiation, neuroendocrine cells acquire highly amplified capacities

62 Neuroendocrine cells act as oxygen sensors in animals fr

63 lower incidence of hyperplasia of pulmonary neuroendocrine cells and aggressive liver, bile duct, st

64 (NEN) are rare neoplasms that originate from neuroendocrine cells and are characterized by the potent

65 uman alpha-synuclein in mammalian kidney and neuroendocrine cells and assessing ER-to-Golgi transport

66 are the likely progenitor of both pulmonary neuroendocrine cells and CFTR-rich ionocytes.

67 d the associated regulated secretion in both neuroendocrine cells and chromogranin A-expressing COS7

68 Pulmonary neuroendocrine cells and clusters of these cells termed

69 DeltaL116 mutant proteins are mistargeted in neuroendocrine cells and form SDS-resistant aggregates,

70 nins regulate secretory vesicle formation in neuroendocrine cells and granin-derived peptides are co-

71 ed nerve terminals, neuromuscular junctions, neuroendocrine cells and hippocampal neurons, but not in

72 ed nerve terminals, neuromuscular junctions, neuroendocrine cells and in hippocampal neurons.

73 ally required for the formation of pulmonary neuroendocrine cells and is a marker for human small cel

74 to sites of dense core vesicle exocytosis in neuroendocrine cells and is required for Ca(2+)-triggere

75 cumulation of fusion-incompetent DCVs in BON neuroendocrine cells and lysosomal degradation (crinopha

76 We now demonstrate hyperplasia of both neuroendocrine cells and mast cells in lungs of baboons

77 Carcinoid tumors arise from neuroendocrine cells and may develop in almost any organ

78 Calcium-regulated exocytosis in neuroendocrine cells and neurons is accompanied by the r

79 nd in specialized secretory cells, including neuroendocrine cells and neurons.

80 complex formation and enhance exocytosis in neuroendocrine cells and neurons.

81 integral membrane glycoprotein expressed in neuroendocrine cells and neurons.

82 tored in secretory granules in endocrine and neuroendocrine cells and released in response to extrace

83 of this study demonstrate a role for ERAD in neuroendocrine cells and serve as a clinical example of

84 th the secretion of catecholamines from PC12 neuroendocrine cells and the steady-state accumulation o

85 on of a distinctive class of Pnmt-expressing neuroendocrine cells and their descendants (i.e. Pnmt(+)

86 ture of lung cancer and increased numbers of neuroendocrine cells and their peptides have been descri

87 tary cells and pituitary tumors and in other neuroendocrine cells and tumors including those of the a

88 l and neoplastic human pituitaries and other neuroendocrine cells and tumors.

89 to a specialized subset of epithelial cells (neuroendocrine cells) and sensory C fibers distributed t

90 : an increase in secretory peptide levels by neuroendocrine cells, and an increase in the number of c

91 Ca(2+) sensors for exocytosis in neurons and neuroendocrine cells, and are thus likely to regulate pi

92 oves the description of ionocytes, pulmonary neuroendocrine cells, and brush cells and identifies a r

93 Hsc70, decreasing SNAP-25 levels in cultured neuroendocrine cells, and inhibited both spontaneous and

94 tained from heterologous expression systems, neuroendocrine cells, and reconstituted systems, which r

95 of the parathyroid glands, enteropancreatic neuroendocrine cells, and the anterior pituitary gland.

96 Secretoneurin is produced in neuroendocrine cells, and the myocardium and circulating

97 Using CRISPR/Cas9, we generated HID-1 KO rat neuroendocrine cells, and we show that the absence of HI

98 The dense core vesicles (DCVs) of neuroendocrine cells are a rich source of bioactive mole

99 Factors involved in the migration of these neuroendocrine cells are as yet unresolved.

100 am may explain why lung cancers arising from neuroendocrine cells are highly metastatic.

101 Secretory granules of neuroendocrine cells are inositol 1,4,5-trisphosphate (I

102 Dense-core vesicles (DCVs) in neuroendocrine cells are maintained at an acidic pH, whi

103 Synaptic terminals and neuroendocrine cells are packed with secretory vesicles,

104 ain PC family members expressed in mammalian neuroendocrine cells are PC2 and PC1/3.

105 n of dense core secretory granules (DCGs) of neuroendocrine cells are poorly understood.

106 Neuroendocrine cells are specialized to produce, maintai

107 These neuroendocrine cells are uniquely identifiable and amena

108 nerves, PNECs, as prototype tissue-resident neuroendocrine cells, are at the center of a neuro-immun

109 more ubiquitously expressed and is found in neuroendocrine cells as well as in brain.

110 essential for the development of normal lung neuroendocrine cells as well as other endocrine and neur

111 tained from genetically modified neurons and neuroendocrine cells, as well as from reconstituted syst

112 ence of incomplete exocytosis in neurons and neuroendocrine cells, as well as recent work probing the

113 Our evidence indicates that neuroendocrine cells assemble molecularly heterogeneous

114 and coordinately regulate NE uptake in PC12 neuroendocrine cells at least in part by stimulating the

115 the hypothalamus during the period when many neuroendocrine cells become differentiated from the neur

116 er NALP, and chromogranin A) that define gut neuroendocrine cell behavior.

117 triking sensitivity to hypoxia in a range of neuroendocrine cells, being progressively inhibited from

118 of cholecystokinin and serotonin (5-HT) from neuroendocrine cells; both act peripherally, cholecystok

119 ctions in sensory neurons is also present in neuroendocrine cells but has no detectable function in t

120 CR-2 in sensory neurons, is expressed in the neuroendocrine cells but has no detectable role in egg l

121 (CHGA) in the regulated secretory pathway in neuroendocrine cells, but the mechanism by which CHGA en

122 nsmitter release both in central neurons and neuroendocrine cells, but the prevalence of this mechani

123 secretory vesicle exocytosis in neurons and neuroendocrine cells, but the underlying mechanism remai

124 ate that link metabolism and excitability in neuroendocrine cells, but their role in nonglucosensing

125 inhibited GABA neurons, including identified neuroendocrine cells, by activating GIRK conductances an

126 s, likely via direct signaling to neoplastic neuroendocrine cells capable of trophic influences.

127 neuroendocrine lung malignancies (large-cell neuroendocrine cell carcinoma, LCNEC, and small-cell lun

128 iate cells (prostate stem cell antigen), and neuroendocrine cells (CD56).

129 mmonly associated with secretory granules of neuroendocrine cells, chromogranins have also been found

130 ore, a significant increase in the number of neuroendocrine cell clusters was observed in the lungs o

131 f the Kalirin and Trio isoforms expressed in neuroendocrine cells colocalize with immature granules.

132 Increased pulmonary neuroendocrine cells containing bombesin-like peptide im

133 es have been proposed to generate a class of neuroendocrine cells containing gonadotropin-releasing h

134 o storage of regulated secretory granules in neuroendocrine cells, contributes to blood pressure home

135 ata expand our understanding of how a single neuroendocrine cell coordinates an organism-wide behavio

136 tions and contained both mucin-secreting and neuroendocrine cells, demonstrating that the crypt conta

137 sis to permeable cells (platelets, mast, and neuroendocrine cells) dependent on putative Ca(2+)-bindi

138 e in hematopoietic, inner ear, and pulmonary neuroendocrine cell development and governs cell process

139 Dividing neuroendocrine cells differentiate into a neuronal-like

140 -helix protein DIMM, a critical regulator of neuroendocrine cell differentiation, controls secretory

141 tes of exocytosis in hippocampal neurons and neuroendocrine cells differs markedly.

142 Nerve-responsive neuroendocrine cells exerted trophic influences and pote

143 t prostate basal cells, but not secretory or neuroendocrine cells, express p63.

144 e showed 55% double labeling of periurethral neuroendocrine cells expressing both serotonin and YFP,

145 In neuroendocrine cells expressing hASH1, such as a pulmona

146 vity, we show that activation of neurons (or neuroendocrine cells) expressing the neuropeptide allato

147 Rb is specifically required for restricting neuroendocrine cell fate despite functional compensation

148 viding evidence that pocket proteins inhibit neuroendocrine cell fate while being required for differ

149 s), the storage compartment in endocrine and neuroendocrine cells for hormones and neuropeptides.

150 We isolated preneoplastic neuroendocrine cells from a mouse model of SCLC and foun

151 rigin hypothesis, favoring the derivation of neuroendocrine cells from the neural crest, with the sec

152 of stress within four different categories (neuroendocrine, cell function, body condition and immune

153 amics of CAPS at sites of exocytosis in live neuroendocrine cells has not been determined.

154 leased from dense-core secretory granules of neuroendocrine cells, have been implicated as playing mu

155 In neuroendocrine cells, hormones and neuropeptides stored

156 GABA synapses on hypothalamic magnocellular neuroendocrine cells; however, retrograde endocannabinoi

157 Only lung neuroendocrine cell hyperplasia and a subtle neuropeptid

158 defined etiology comprising the two diseases neuroendocrine cell hyperplasia of infancy (NEHI) and pu

159 fluid (BALF) would distinguish children with neuroendocrine cell hyperplasia of infancy (NEHI), surfa

160 of several different forms of ILD, including neuroendocrine cell hyperplasia of infancy and ILD, due

161 Two distinctive entities, neuroendocrine cell hyperplasia of infancy and pulmonary

162 els of neprilysin increases inflammation and neuroendocrine cell hyperplasia, which may predispose to

163 yperplasia, and diffuse idiopathic pulmonary neuroendocrine cell hyperplasia.

164 phages 4 weeks after radiation and pulmonary neuroendocrine cells hyperplasia 6 weeks after radiation

165 king down CAPS1, Rbcn3alpha, or Rbcn3beta in neuroendocrine cells impaired rates of DCV reacidificati

166 a dominant negative Cdc42 construct in human neuroendocrine cells impaired the release process by com

167 s were mimicked by ablations of serotonergic neuroendocrine cells, implicating humoral release of ser

168 of a major fraction of neural crest-derived neuroendocrine cells in both the human and murine prosta

169 ng peptide (GRP) is synthesized by pulmonary neuroendocrine cells in inflammatory lung diseases, such

170 antly increased numbers of type II cells and neuroendocrine cells in neuroepithelial bodies.

171 Evidence was found for basal, luminal, and neuroendocrine cells in prostatic tubules regenerated fr

172 econstruction of the distribution pattern of neuroendocrine cells in the human fetal prostate indicat

173 likely that 5-HT2A receptors are present on neuroendocrine cells in the hypothalamic paraventricular

174 s but a reduced number of solitary pulmonary neuroendocrine cells in the neonatal lung.

175 Hyperoxia increased the number of neuroendocrine cells in the peripheral lung, which was p

176 gs provide further support for a key role of neuroendocrine cells in the reparative process of airway

177 A small population of neuroendocrine cells in the rostral hypothalamus and bas

178 tal trophoblasts, renal collecting ducts and neuroendocrine cells in the stomach and colon.

179 nd an epithelial progenitor can give rise to neuroendocrine cells in vivo.

180 dent ERK->Egr-1/Zif268 signaling in cultured neuroendocrine cells; in D1 medium spiny neurons of NAc

181 ion, have been identified in gastric mucosal neuroendocrine cells including parathyroid hormone-like

182 in associated with the secretory granules of neuroendocrine cells, including pancreatic beta-cells.

183 Neuroendocrine cells, including those in the gut, have a

184 se and inhibits calcium-triggered release in neuroendocrine cells, indicating a previously unrecogniz

185 I1 display abnormal development of pulmonary neuroendocrine cells, indicating that GFI1 is important

186 ibuting to the appropriate entrance of these neuroendocrine cells into the brain, and thus represent

187 lecular and cellular properties of all major neuroendocrine cells, irrespective of the secretory pept

188 The histogenesis of prostatic neuroendocrine cells is controversial: a stem cell hypot

189 eature of the regulated secretory pathway in neuroendocrine cells is lumenal pH, which decreases betw

190 Ca(2+)-triggered exocytosis in neurons and neuroendocrine cells is regulated by the Ca(2+)-binding

191 Exocytosis from neurons and neuroendocrine cells is tightly regulated by intracellul

192 required for proper development of pulmonary neuroendocrine cells, is essential for the survival of a

193 sicles in neurones and secretory vesicles in neuroendocrine cells, is likely to be involved in vesicl

194 220, which is a substrate of PKD proteins in neuroendocrine cells, is localized in the ends of the pr

195 l membrane protein of dense-core granules in neuroendocrine cells, is phosphorylated in a Ca(2+)-sens

196 In neuroendocrine cells, it elicits a robust, albeit transi

197 However, central synapses and neuroendocrine cells lacking these synaptotagmins still

198 to glycines, is expressed in AtT20 cells, a neuroendocrine cell line endogenously expressing PC1, bo

199 H is transiently expressed in GH4C1 cells, a neuroendocrine cell line lacking PC1, under pulse-chase

200 The neuroendocrine cell line PC12, derived from rat pheochro

201 PC12 cells, a model neuroendocrine cell line, express multiple isoforms of t

202 Downregulation of CGA expression in a neuroendocrine cell line, PC12, by antisense RNAs led to

203 nhibitor reduced the expression of ASCL1 and neuroendocrine cell lineage genes.

204 inal differentiation and maturation of these neuroendocrine cell lineages.

205 mmunofluorescence microscopy in neuronal and neuroendocrine cell lines revealed that membrane-associa

206 Unlike the neuroendocrine cell lines widely used to study trafficki

207 argeting of alpha(2C)-adrenoreceptors in two neuroendocrine cell lines with the targeting in three ep

208 By using different neuroendocrine cell lines, with or without endogenous ex

209 otropin-releasing hormone (GnRH) neurons are neuroendocrine cells, located in the hypothalamus, that

210 In mammalian neuroendocrine cells, loss of AP-3 dysregulates exocytos

211 cells, together with ciliated and pulmonary neuroendocrine cells, make up the epithelium of the bron

212 tibody to calcitonin gene-related peptide, a neuroendocrine cell marker, was used to identify the loc

213 with regard to the expression of AR and the neuroendocrine cell markers Synaptophysin and Chromogran

214 The uv1 neuroendocrine cells mechanically sense passage of eggs

215 leasing peptide (GRP), secreted by pulmonary neuroendocrine cells, mediates oxidant-induced lung inju

216 ctor for the regulation of DCG biogenesis in neuroendocrine cells, mediating the formation of functio

217 te smoking on the gene expression profile of neuroendocrine cells, microarray analysis with TaqMan co

218 spike clustering displayed by magnocellular neuroendocrine cells (MNCs) of the supraoptic (SON) and

219 In neuroendocrine cells, MYC activates Notch to dedifferent

220 These data suggest that in neuroendocrine cells myosin XVA may have a role in secre

221 Prostatic neuroendocrine cells (NE) are an integral part of prosta

222 Both cAMP and ERK are necessary for neuroendocrine cell neuritogenesis, and pituitary adenyl

223 Secretion of hormones and peptides by neuroendocrine cells occurs through fast and slow modes

224 hydroxylating monooxygenase, is defective in neuroendocrine cells of Ctr1A mutant larvae.

225 xpression of Prox1 in three novel sites: the neuroendocrine cells of the adrenal medulla, megakaryocy

226 UCHL1 expression was evident only in neuroendocrine cells of the airway epithelium in nonsmok

227 The neuroendocrine cells of the brain receive more extensive

228 NPYR-1 is expressed in neuroendocrine cells of the central nervous system and c

229 ecently that magnocellular and parvocellular neuroendocrine cells of the hypothalamic paraventricular

230 We show that in neuroendocrine cells, PC6B is localized to a paranuclear

231 In neuroendocrine cells, PI3K-C2alpha is present on a subpo

232 is induced by hormonal therapy, suggest that neuroendocrine cells play an important role in promoting

233 n many organs, but its function in pulmonary neuroendocrine cell (PNEC) differentiation has not been

234 ls, free alveolar macrophages, and pulmonary neuroendocrine cells (PNEC).

235 ribution and abundance of solitary pulmonary neuroendocrine cells (PNECs) and neuroepithelial bodies

236 Pulmonary neuroendocrine cells (PNECs) are proposed to be the firs

237 Pulmonary neuroendocrine cells (PNECs) are rare airway epithelial

238 Pulmonary neuroendocrine cells (PNECs) are the only innervated air

239 ptor (Robo) genes are expressed in pulmonary neuroendocrine cells (PNECs), a rare, innervated epithel

240 % of lung progenitor cells to form pulmonary neuroendocrine cells (PNECs), putative precursors to sma

241 - carotid body glomus cells, and 'pulmonary neuroendocrine cells' (PNECs) - are obscure.

242 AT3 activation, a decrease in the neoplastic neuroendocrine cell population, and impaired PanIN progr

243 This region is the source of many neuroendocrine cells, primarily located in the rostral s

244 tegrated physiological systems requires that neuroendocrine cells remain plastic to dramatically alte

245 activated by secretory stimuli in neural and neuroendocrine cells remains unknown.

246 pendent exocytosis of dense-core vesicles in neuroendocrine cells requires a priming step during whic

247 Since CGRP-expressing neuroendocrine cells reside in bronchiolar SCPC niches,

248 ng to the plasma membrane in melanocytes and neuroendocrine cells, respectively, was altered by 4 h a

249 ressed cells and signals to activate central neuroendocrine cell(s).

250 In neuroendocrine cells, SdpII colocalizes with dynamin, co

251 In response to physiological stimuli, neuroendocrine cells secrete neurotransmitters through a

252 es augmented neural stimulation of pulmonary neuroendocrine cell secretion.

253 ecretory pathway, which suggests that EC and neuroendocrine cells share common protein targeting reco

254 Live-cell imaging of wild-type AKH neuroendocrine cells shows heightened excitability under

255 Of 11 genes considered to be neuroendocrine cell specific, only ubiquitin carboxyl-te

256 In neuroendocrine cells, splice variants of the SNARE prote

257 nger than that of exocytic vesicle fusion in neuroendocrine cells, suggesting that additional regulat

258 was found on some epithelial basal cells and neuroendocrine cells, suggesting that these cells are ta

259 In neuroendocrine cells, synaptosome-associated protein of

260 olecules mediating the exocytosis of MSGs in neuroendocrine cells, syntaxin 1, SNAP-25, and VAMP2, we

261 otropin-releasing hormone (GnRH) neurons are neuroendocrine cells that are born in the nasal placode

262 rkel cells, a nonproliferative population of neuroendocrine cells that arise from epidermis.

263 beta-cells are neuroendocrine cells that can take up and metabolize the

264 a heterogeneous group of tumors arising from neuroendocrine cells that includes typical carcinoid, at

265 a region of the brain that gives rise to the neuroendocrine cells that innervate the ring gland, we c

266 onooxygenase (PAM) is an enzyme expressed by neuroendocrine cells that participates in hormone matura

267 the corpus cardiacum (CC) cells, a group of neuroendocrine cells that produce the adipokinetic hormo

268 For the hypothalamic neuroendocrine cells that release vasopressin and oxytoc

269 Like many neuroendocrine cells, the coupling between insulin-secre

270 Ca(2+) enters pituitary and pancreatic neuroendocrine cells through dihydropyridine-sensitive c

271 ory factors, and IA-2 is widely expressed in neuroendocrine cells throughout the body.

272 They are expressed together or separately in neuroendocrine cells throughout the brain and dispersed

273 ion, can promote a differentiation defect of neuroendocrine cells thus enhancing the ability of tumor

274 cialized secretory organelles in neurons and neuroendocrine cells to make them available for regulate

275 y be part of a more general process allowing neuroendocrine cells to originate in nonneuroectodermall

276 ell as the G-protein G alpha(o), function in neuroendocrine cells to promote release of neurotransmit

277 This could be a common mechanism used by neuroendocrine cells to regulate independently the secre

278 se that KIN-29/SIK acts in nuclei of sensory neuroendocrine cells to transduce low cellular energy ch

279 inates expression of FMRFa in that subset of neuroendocrine cells (Tv neurons) which provide the syst

280 SLMV purification scheme on a series of non-neuroendocrine cell types including the mouse fibroblast

281 K2 is dispensable for tumorigenesis in these neuroendocrine cell types.

282 '-UTR variant system in 2 different neuronal/neuroendocrine cell types.

283 ion of GABA synaptic inputs to magnocellular neuroendocrine cells under different physiological condi

284 sing SV40 large T antigen in their prostatic neuroendocrine cells, under the control of transcription

285 lipid rafts organize exocytotic pathways in neuroendocrine cells, we examined the association of pro

286 In experiments with rat neuroendocrine cells, we find that a few basic amino aci

287 In mast cells, as in neuroendocrine cells, we show that SgIII is complexed wi

288 synaptic terminals and from several types of neuroendocrine cells, we studied its involvement in oxyt

289 re the role of AP-1 in copper homeostasis in neuroendocrine cells, we used corticotrope tumor cells i

290 aV2.3 subunits are expressed in neuronal and neuroendocrine cells where they are believed to form nat

291 subunit are expressed mainly in neurons and neuroendocrine cells, whereas those containing the Cav1.

292 supraoptic nucleus, consisting primarily of neuroendocrine cells, which are persistently and commonl

293 pletion suppresses neurite outgrowth of PC12 neuroendocrine cells, which can be rescued by codepletio

294 mbrane protein mainly expressed in brain and neuroendocrine cells, which is a downstream target of th

295 In neuroendocrine cells, whose dense core granules are stri

296 is report shows that secretory proteins from neuroendocrine cells will activate the NF-kappaB pathway

297 distinguished in the secretory response from neuroendocrine cells with slow ATP-dependent priming ste

298 ses the same fusion machinery as neurons and neuroendocrine cells, with an additional requirement for

299 pro-hormone secretory proteins released from neuroendocrine cells, with effects on control of blood p

300 ed within the regulated secretory pathway of neuroendocrine cells yielding five TRH peptides and seve