ライフサイエンスコーパス: neurogenic bladder

1 ment, causing cardiac failure, diplegia, and neurogenic bladder.

2 gy influence UTI susceptibility with the SCI-neurogenic bladder.

3 sponse, upper gastrointestinal symptoms, and neurogenic bladder.

4 al urachal sinus, and lipomeningocele with a neurogenic bladder.

5 pilepsy, migraine, stroke-like episodes, and neurogenic bladder.

6 pid eye movement sleep behavior, anosmia, or neurogenic bladder.

7 sis, and voiding dysfunctions as a result of neurogenic bladders.

8 iomarker for management of the patients with neurogenic bladders.

9 method was also applied on 16 patients with neurogenic bladders (10 compliant and 6 non-compliant su

10 The most common clinical findings were neurogenic bladder (100%), spastic paraplegia with vibra

11 ocedures (43% versus 15% overall), to have a neurogenic bladder (29% versus 12% overall), to have rec

12 In the management of neurogenic bladder and chronic lower urinary tract sympt

13 Neurogenic bladder and sphincter dysfunction can be mana

14 s current surgical practice in patients with neurogenic bladders and urinary incontinence.

15 Patients with diabetes mellitus, neurogenic bladder, bladder outlet obstruction, and recu

16 treatment of pediatric voiding dysfunction, neurogenic bladder, chronic lower urinary tract symptoma

17 y a description of the various causes of the neurogenic bladder, discussed in a hierarchical order st

18 For various neurogenic bladder disorders, urodynamic studies may dem

19 review of the methods available for treating neurogenic bladder disorders.

20 Neurogenic bladder dysfunction affects up to 80% of indi

21 ers the articles that have been published on neurogenic bladder dysfunction as well as on posterior u

22 Neurogenic bladder dysfunction causes urological complic

23 ting neurodegeneration on the development of neurogenic bladder dysfunction in mice with corona-virus

24 e previously characterized a murine model of neurogenic bladder dysfunction induced by a neurotropic

25 Cognitive decline and neurogenic bladder dysfunction progressed over time in m

26 s and showed differential mechanisms driving neurogenic bladder dysfunction.

27 The outlook for patients with neurogenic bladder has been transformed by a combination

28 s, were not required for colonization of the neurogenic bladder in human beings.

29 rent standard of care in which to remedy the neurogenic bladder, it is still a stop-gap measure and i

30 This model will allow elucidation of SCI-neurogenic bladder-mediated changes in host response tha

31 ves (n = 49), spina bifida (n = 21), central neurogenic bladder (n = 13), bladder exstrophy (n = 14),

32 bidity and hospitalizations in subjects with neurogenic bladder (NB) due to spinal cord injury (SCI).

33 bidity and hospitalisations in subjects with neurogenic bladder (NB) due to spinal cord injury (SCI).

34 ransplant bladder augmentation, did not have neurogenic bladders or require preoperative catheterizat

35 micturition and defecation for patients with neurogenic bladder overactivity or spinal cord lesions.

36 Neurogenic bladder predisposes to recurrent urinary trac

37 2 mutations were absent in 23 non-neurogenic neurogenic bladder probands and, of 439 families with no

38 al recessive leukodystrophy characterized by neurogenic bladder, progressive spastic gait, and periph

39 thra for evacuation (e.g. bladder exstrophy, neurogenic bladder, radiation injury, and marked urethra

40 The median age was 51 years for the onset of neurogenic bladder symptoms, 63 years for wheelchair dep

41 nmol/L), sicca complex, abnormal pupils, and neurogenic bladder; three had severe upper gastrointesti

42 onditions such as posterior urethral valves, neurogenic bladder, ureteral ectopy, or bladder exstroph