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1 ing activity from the ventral midline to the neurogenic ectoderm.
2 ential cell fates in adjacent regions of the neurogenic ectoderm.
3 tivator also drive expression throughout the neurogenic ectoderm.
4 ted expression within ventral regions of the neurogenic ectoderm.
5 toderm within the ventral-most region of the neurogenic ectoderm.
6 e precise lines of sim expression within the neurogenic ectoderm.
7 pression in the anterior-most regions of the neurogenic ectoderm.
8 eristic of the ciliated band, which contains neurogenic ectoderm.
9 y rescues epidermal development in overlying neurogenic ectoderm.
10 oundary between the presumptive mesoderm and neurogenic ectoderm [3].
11 istinction between tissues such as mesoderm, neurogenic ectoderm and dorsal ectoderm in the developin
12 itiates the differentiation of the mesoderm, neurogenic ectoderm and dorsal ectoderm in the precellul
13 stoderm defines the boundary between ventral neurogenic ectoderm and dorsal ectoderm.
14 on of midline cells causes a failure to form neurogenic ectoderm and Ph-sim RNAi results in severely
15  in severely dorsalized embryos lacking both neurogenic ectoderm and the appendage-bearing lateral ec
16 the animal pole domain (APD), which contains neurogenic ectoderm, and can oppose Wnt and Nodal signal
17 orsal-ventral (DV) axis to specify mesoderm, neurogenic ectoderm, and dorsal ectoderm cell fates.
18 ree primary embryonic tissues: the mesoderm, neurogenic ectoderm, and dorsal ectoderm.
19  snail (sna) and partial derepression of the neurogenic ectoderm-associated target short gastrulation
20      The sea star embryo initially has a pan-neurogenic ectoderm, but the genetic mechanism that dire
21 lda drives expression within the presumptive neurogenic ectoderm, but they also demonstrate that regu
22 EGFR is activated in the ventral midline and neurogenic ectoderm by the Spitz ligand, which is proces
23  responses, but its specification of ventral neurogenic ectoderm demands a relatively high-threshold
24                             We show that the Neurogenic Ectoderm Enhancer (NEE) at vnd takes addition
25                                          The neurogenic ectoderm enhancers (NEEs) activate different
26 nd the Wnt and Delta pathways to distinguish neurogenic ectoderm from endomesoderm.
27 ct gene expression in ventral regions of the neurogenic ectoderm in response to intermediate levels o
28 epressor, Brinker, which is expressed in the neurogenic ectoderm in response to the maternal Dorsal g
29  other receptor organs that are derived from neurogenic ectoderm including neural crest (NC).
30 al specification of the mesoderm and ventral neurogenic ectoderm is highly conserved in flies and mos
31  the broad lateral domain of the presumptive neurogenic ectoderm is not understood.
32                            Surprisingly, the neurogenic ectoderm, not the ventral midline, was found
33 lation of neural-epidermal fate decisions in neurogenic ectoderm of Drosophila and somitogenesis in v
34 oundary between the presumptive mesoderm and neurogenic ectoderm of early Drosophila embryos.
35 sential for the specification of the ventral neurogenic ectoderm prior to gastrulation.
36                       We propose a model for neurogenic ectoderm specification in which gene regulati
37 o investigate transcriptional control during neurogenic ectoderm specification, we examined divergenc
38 pressing mesoderm derivatives, including the neurogenic ectoderm, stomadeum, and hindgut.
39 ulated by the Dorsal gradient in the ventral neurogenic ectoderm (vNE) of the early Drosophila embryo
40 ipe induces ectopic expression of sim in the neurogenic ectoderm where there are low levels of the Do