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2 his study is to determine the reliability of neurohypophyseal abnormalities on magnetic resonance ima
5 er4.3+) and mouse (Sox1+) contribute to both neurohypophyseal and a subset of adenohypophyseal cells.
11 essin (VP) cause autosomal dominant familial neurohypophyseal diabetes insipidus (adFNDI), a rare inh
17 sin parents and three children affected with neurohypophyseal diabetes insipidus, suggesting autosoma
19 m both in the cardiovascular response to the neurohypophyseal hormone arginine vasopressin (AVP) and
21 ized that combine into a single molecule the neurohypophyseal hormone oxytocin and the potent vasopre
24 tive resistant or R-type Ca2+ current in rat neurohypophyseal nerve terminals, but concentrations of
26 excretion by the kidney is regulated by the neurohypophyseal peptide hormone vasopressin through act
28 that the phylogenetic plasticity of central neurohypophyseal peptide receptor expression may contrib
31 Physiological activation of the hypothalamo-neurohypophyseal system (HNS) by dehydration results is
32 VP) release from explants of the hypothalamo-neurohypophyseal system (HNS), but the response is not s
33 cin release from explants of the hypothalamo-neurohypophyseal system (HNS), two hypotheses were teste
34 we show expression in the brain hypothalamo-neurohypophyseal system (HNS), wherein upregulation foll
35 reactive fibers, were located throughout the neurohypophyseal tract and within the posterior pituitar
36 ers were evident outside of the hypothalamic-neurohypophyseal tract, a plexus of fibers in the latera