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1 l disorder than persons not diagnosed with a neurological disorder.
2 clinically heterogeneous autosomal-recessive neurological disorder.
3 for disability worldwide and the most common neurological disorder.
4 variants in WDR37 underlie a novel syndromic neurological disorder.
5 ge, but may sometimes represent a functional neurological disorder.
6 l in understanding the long-term progressive neurological disorder.
7 years among individuals not diagnosed with a neurological disorder.
8 ssue concentration is elevated in cancer and neurological disorders.
9 ted DBS leads was validated in patients with neurological disorders.
10 AKT is implicated in neurological disorders.
11 ange the clinical paradigm for patients with neurological disorders.
12 g with the current global status of specific neurological disorders.
13 g with their application in the treatment of neurological disorders.
14 XOC2 variants are the cause of the patients' neurological disorders.
15 l therapeutic candidate for the treatment of neurological disorders.
16 pathways may provide therapeutic targets for neurological disorders.
17 eptors (FGFRs) have been detected in various neurological disorders.
18 has been associated with many metabolic and neurological disorders.
19 important drugs for treatment in a number of neurological disorders.
20 specific TRAPP subunits are associated with neurological disorders.
21 brain vascular dysfunction accompanies many neurological disorders.
22 driving proteins in neurons across a host of neurological disorders.
23 ensorimotor symptoms incompatible with other neurological disorders.
24 Its dysfunction has been linked to many neurological disorders.
25 method has recently been used to investigate neurological disorders.
26 onists exert anti-inflammatory properties in neurological disorders.
27 provide a potential new treatment option for neurological disorders.
28 h autism spectrum disorders (ASDs) and other neurological disorders.
29 ne injury underlies synaptic failure in many neurological disorders.
30 asive optogenetic tools for the treatment of neurological disorders.
31 athways and the involvement of complement in neurological disorders.
32 brainstem volumes and eight psychiatric and neurological disorders.
33 in patients with suspected COVID-19-related neurological disorders.
34 EAG channel activity is linked to cancer and neurological disorders.
35 th disorders including cancer, diabetes, and neurological disorders.
36 and developmental stages and altered in many neurological disorders.
37 lopment and has yielded notable successes in neurological disorders.
38 d density have been associated with multiple neurological disorders.
39 matched controls across 8 psychiatric and 4 neurological disorders.
40 ) tract, and it is associated with different neurological disorders.
41 rriers can result in severe and debilitating neurological disorders.
42 common feature of many neuropsychiatric and neurological disorders.
43 of the E/I ratio is associated with numerous neurological disorders.
44 drug development for mental health and other neurological disorders.
45 has been associated with the development of neurological disorders.
46 leotide repeats cause more than 40 different neurological disorders.
47 provide therapeutic benefits for a number of neurological disorders.
48 research to tackle the growing challenge of neurological disorders.
49 ex, expanding the window of vulnerability to neurological disorders.
50 in and for studying biological mechanisms of neurological disorders.
51 gnificant enrichment of neural functions and neurological disorders.
52 ntributing to the pathogenesis of cancer and neurological disorders.
53 odegenerative conditions, suggests a role in neurological disorders.
54 ng diabetes development and autoimmune-based neurological disorders.
55 play a major role in pathophysiology of many neurological disorders.
56 They degenerate in multiple neurological disorders.
57 ironment toward the goal of treating various neurological disorders.
58 t function of UCHL1 have been linked to many neurological disorders.
59 nto the clinical diagnosis and management of neurological disorders.
60 an public health due to its association with neurological disorders.
61 pany multiple diseases, including cancer and neurological disorders.
62 eural circuits and the clinical treatment of neurological disorders.
63 er recycling, a key process in several human neurological disorders.
64 he therapeutic potential of targeting AHR in neurological disorders.
65 role in the pathogenesis of psychiatric and neurological disorders.
66 ne silencing in the CNS for the treatment of neurological disorders.
67 understanding of human brain development and neurological disorders.
68 1 and may play a role in the pathogenesis of neurological disorders.
69 ncern because of its association with severe neurological disorders.
70 understanding and treating mental health and neurological disorders.
71 myotrophic lateral sclerosis (ALS) and other neurological disorders.
72 to limit it may be effective across multiple neurological disorders.
73 f behavioral indices to diagnose and monitor neurological disorders.
74 nd suggest its dysfunction may lead to human neurological disorders.
75 Aberrant metabolism is a key factor in many neurological disorders.
76 states, focusing particularly on cancer and neurological disorders.
77 erged as a novel human pathogen, linked with neurological disorders.
78 stigations of network alterations underlying neurological disorders.
79 otor system, a relevant clinical example for neurological disorders.
80 earch is valuable for advancing treatment of neurological disorders.
81 cardiovascular, metabolic, inflammatory and neurological disorders.
82 y bowel disease, cardiovascular disease, and neurological disorders.
83 amma (PPARgamma) can be anti-inflammatory in neurological disorders.
84 hological findings in HIV+ cases on ART with neurological disorders.
85 plicated in a wide variety of behavioral and neurological disorders.
86 ic changes associated with glioma, and other neurological disorders.
87 inflammation/infection and susceptibility to neurological disorders.
88 n suggested to be associated with a range of neurological disorders.
89 l and applied to animal models of a range of neurological disorders.
90 ous system development and are implicated in neurological disorders.
91 and diseases processes, including cancer and neurological disorders.
92 ompositions and contributes to metabolic and neurological disorders.
93 in particular, are increasingly the focus of neurological disorders.
94 have been associated with many metabolic and neurological disorders.
95 eat expansion is the molecular basis for ~40 neurological disorders.
96 communities have been implicated in several neurological disorders.
97 cts of early dopamine-replacement therapy in neurological disorders.
98 s immune dysregulation in the development of neurological disorders.
99 affective behaviors that could contribute to neurological disorders.
100 n linked to autoimmune diseases, cancer, and neurological disorders.
101 sible contributions by the gut microbiota to neurological disorders.
102 drug candidates for the treatment of various neurological disorders.
103 erol metabolism is implicated in a number of neurological disorders.
104 Brain zinc dysregulation is linked to many neurological disorders.
105 BDNF levels that are dysregulated in various neurological disorders.
106 rature implicating these proteins in various neurological disorders.
107 n suggested to be associated with a range of neurological disorders.
108 ing new insights into the molecular basis of neurological disorders.
109 ion balance and trigger neuropsychiatric and neurological disorders.
110 TOR) pathway is hyperactivated in cancer and neurological disorders.
111 role in synaptic dysfunction in a variety of neurological disorders.
112 dd to the growing number of TRAPP-associated neurological disorders.
113 osteopetrosis, lysosomal storage disease and neurological disorders.
118 l temporal lobe epilepsy (MTLE) is a chronic neurological disorder affecting almost 40% of adult pati
119 encephalopathy (CTE) was conceptualized as a neurological disorder affecting some active and retired
122 understanding which changes associated with neurological disorders, aging, or stress are most likely
124 ng a fundamental role in this highly complex neurological disorder and provides a road map for future
125 as cardiovascular disease, diabetes, cancer, neurological disorders and arthritis, among others, for
126 is commonly associated with psychiatric and neurological disorders and can impose a substantial burd
128 ibute to the pathogenesis of some autoimmune neurological disorders and could even contribute to neur
131 age strains are accompanied by high rates of neurological disorders and exemplify the global health b
132 oupled receptors due to their involvement in neurological disorders and important role in pain treatm
133 c morphology is a common theme among several neurological disorders and is thought to precede neurode
134 ays holds potential for treating a number of neurological disorders and is thus crucial to understand
135 Visuomotor impairments characterize numerous neurological disorders and neurogenetic syndromes, such
136 viruses, ZIKV infection can cause congenital neurological disorders and replicates efficiently in rep
137 Immunotherapy, and the National Institute of Neurological Disorders and Stroke (NINDS), NIH (R35NS105
141 ongitudinally followed National Institute of Neurological Disorders and Stroke Parkinson's Disease Bi
142 al Institute on Aging, National Institute of Neurological Disorders and Stroke, and the Alzheimer's A
143 al Institute on Aging, National Institute of Neurological Disorders and Stroke, Banner Alzheimer's Fo
145 (C2R) was approved by National Institute of Neurological Disorders and Stroke-National Institutes of
149 provides a platform for modeling inheritable neurological disorders, and advances drug screening, as
151 Microglial activation is a common feature of neurological disorders, and although in some instances t
152 ut also in human pathologies such as cancer, neurological disorders, and cardiovascular diseases.
153 n for HIV/AIDS eradication, the treatment of neurological disorders, and enhanced CAR T/NK cell immun
154 plicated in many diseases, including cancer, neurological disorders, and more recently immune-mediate
155 ng and stimulation technologies most used in neurological disorders, and provide a vision of minimall
156 the study of the physiology and aetiology of neurological disorders, and to the development of person
157 vances in prevention and management of major neurological disorders are not sufficiently effective to
159 ects neurons against Mn-induced toxicity and neurological disorders associated with dopaminergic neur
160 arget for treatment of Mn toxicity and other neurological disorders associated with dysregulation of
161 n cortical inhibition and may be involved in neurological disorders associated with inhibitory circui
162 s widely distributed and associated with the neurological disorder autosomal recessive spinocerebella
163 ve broadened the number of avenues to target neurological disorders but have also highlighted safety
164 e to resolve them underlies the pathology of neurological disorders but is also exploited in cancer t
165 earning in normal individuals and those with neurological disorders but its mechanism of action is un
166 GAD) are observed in patients with different neurological disorders, but cells producing these autoan
167 In humans, mutant aaRS cause a diversity of neurological disorders, but their molecular aetiologies
168 ved in a number of human diseases, including neurological disorders, cancer, and autoimmune diseases.
171 entified as one of the mutations causing the neurological disorder cerebellar ataxia, mental retardat
172 Likewise, mutation I364M, which causes the neurological disorder cerebellar ataxia, mental retardat
176 Alzheimer's disease (AD) is a devastating neurological disorder characterized by changes in cell-t
178 yotrophic lateral sclerosis (ALS) is a fatal neurological disorder characterized by progressive muscu
182 Familial hemiplegic migraine is an episodic neurological disorder characterized by transient sensory
183 is of the phospholipid PI(3,5)P(2) underlies neurological disorders characterized by cytoplasmic accu
186 esent a preliminary definition of functional neurological disorder-cognitive subtype, explain its pos
187 Ds), part of the wide spectrum of functional neurological disorders (conversion disorders), are commo
188 it is crucial that medications developed for neurological disorders cross into the brain in therapeut
189 .SIGNIFICANCE STATEMENT Epilepsy is a common neurological disorder defined by recurrent, unprovoked s
190 gnitive or behavioral impairment and/or late neurological disorders, degenerative tissue effects incl
191 , and 14.7% (n = 5141) were diagnosed with a neurological disorder, equivalent to a suicide rate of 4
192 FAS may in some cases represent a functional neurological disorder, even when a structural lesion is
193 fewer than half of all patients with genetic neurological disorders, exposing considerable gaps in th
195 present a predisposing factor for functional neurological disorder (FND) but previous research has yi
198 d body, psychiatry and neurology, functional neurological disorder (FND) remains poorly understood.
199 ify existing outcome measures for functional neurological disorder (FND), to inform the development o
200 regional pain syndrome (CRPS) and functional neurological disorders (FND) but until now the discussio
201 y pathogenic triggers of other developmental neurological disorders for devising targeted therapies.
202 ene are associated with a range of inherited neurological disorders, from drug-refractory lethal epil
207 ew possibilities for probing psychiatric and neurological disorders impacted by insular cortex dysfun
208 oirs, thereby reducing neuroinflammation and neurological disorders in HIV-infected individuals.
211 parasite Toxoplasma correlates with certain neurological disorders in humans and alters behavior in
215 stic feature of several neuropsychiatric and neurological disorders in which dopamine is dysregulated
216 cognitive disorders, a subtype of functional neurological disorders, in which cognitive symptoms are
217 y offer a therapeutic strategy in cancer and neurological disorders, in which pericyte dysfunction co
218 ption factor implicated as a drug target for neurological disorders including Alzheimer's and Parkins
219 function and the pathophysiology of several neurological disorders including Alzheimer's disease.
220 ic plasticity has been implicated in several neurological disorders including epilepsy, dementia and
222 tients with chronic progressive inflammatory neurological disorders including HAM/TSP, associated wit
223 BBB dysfunction, however, is evident in many neurological disorders including ischemic stroke, trauma
224 ecially associated with ophthalmological and neurological disorders, including dementia and Parkinson
225 heir dysfunction is implicated in a range of neurological disorders, including depression, epilepsy a
228 ng is a common cause of cognitive decline in neurological disorders, including HIV-associated neuroco
229 the degree of axonal damage in a variety of neurological disorders, including inflammatory, neurodeg
230 iagnosed with a variety of acute and chronic neurological disorders, including ischemic stroke, hemor
231 o be an effective treatment for a variety of neurological disorders, including Parkinson's disease an
232 human isoforms have been linked to different neurological disorders, including Parkinson's disease.
233 h is accountable for neuronal death in acute neurological disorders, including stroke and trauma, and
234 n and control are revealing how learning and neurological disorders initiate and successively change
235 ry system function may have implications for neurological disorders involving NMDA receptor dysfuncti
238 g nerve injury, chemotherapy, and in several neurological disorders is an active process driven by SA
239 ven recent reports of Kv2.1 dysregulation in neurological disorders, it is possible that alterations
241 ations in the non-coding U8 snoRNA cause the neurological disorder leukoencephalopathy with calcifica
242 nd efficacy towards the treatment of various neurological disorders like AD, PD, schizophrenia, epile
243 Chronic manganese (Mn) exposure causes the neurological disorder manganism, presenting symptoms sim
244 is a mitochondrial disease characterized by neurological disorders, metabolic abnormality and premat
245 ance to grand challenges in the treatment of neurological disorders motivate continued growth of this
246 embolism), one of whom died; (iv) peripheral neurological disorders (n = 8), seven with Guillain-Barr
247 of its synapses are associated with serious neurological disorders, numerous versions of caged GABA
248 e sequencing in a patient with a multisystem neurological disorder of both the central and peripheral
252 gs.SIGNIFICANCE STATEMENT Epilepsy, a common neurological disorder, often develops following a brain
254 and cellular events that contribute to human neurological disorders previously linked to CTCF, includ
255 onists are used by billions of patients with neurological disorders, primarily for the treatment of m
256 plication and has been genetically linked to neurological disorders ranging from Parkinson's disease
257 te vast progress, therapeutic strategies for neurological disorders remain limited, owing to a lack o
259 initially discussed the global statistics of neurological disorders reported by WHO and other reputed
262 f individuals suffering from GPCR-associated neurological disorders show drastically lowered levels o
263 ase burden attributable to mental disorders, neurological disorders, substance use disorders, and sel
264 he therapeutic potential of cannabinoids for neurological disorders such as epilepsy, multiple sclero
265 all clinical trials of cannabinoids in other neurological disorders such as Huntington's disease, att
266 le indication of the degree of impairment in neurological disorders such as stroke, Parkinson disease
267 Patients with sialidosis present various neurological disorders such as: myoclonic epilepsy and h
268 ings have implications for the management of neurological disorders, such as autism and intellectual
269 uronal development is frequently impaired in neurological disorders, such as autism and schizophrenia
270 ations of potassium levels can be related to neurological disorders, such as epilepsy and Alzheimer's
271 lled BAF complexes) in both human cancer and neurological disorders, suggesting new mechanisms and ac
272 ualized as a cognitive variant of functional neurological disorder, termed functional cognitive disor
273 rate of suicide among those with a diagnosed neurological disorder than persons not diagnosed with a
275 aumatic brain injury (TBI) is a debilitating neurological disorder that can seriously impact the pati
276 imary cause of Rett syndrome (RTT), a severe neurological disorder that is thought to result from abs
277 ations for the pathogenesis and treatment of neurological disorders that are associated with pericyte
279 identify relevant connectional biomarkers of neurological disorders that emerge early or late in the
282 red with those who were not diagnosed with a neurological disorder, those with dementia had a lower o
283 is of RNA binding proteins is compromised in neurological disorders through their reduced expression
285 ain models that reconstruct aspects of major neurological disorders under static or dynamic condition
286 ikely windows of susceptibility for multiple neurological disorders using known disease associated ge
287 s on the potential role of gut microbiota in neurological disorders, we sought to understand the caus
288 c target for drug development toward several neurological disorders, we synthesized a series of 2,3,4
289 ir brains to the Queen Square Brain Bank for Neurological Disorders were assessed for alpha-synuclein
290 broadly effective translational therapy for neurological disorders where mTOR signaling is dysregula
292 ards a molecular mechanism-based taxonomy of neurological disorders, which could help in developing b
293 Temporal lobe epilepsy (TLE) is a prevalent neurological disorder with many patients experiencing po
296 l community composition has been reported in neurological disorders with known microglial involvement
297 nvironmental risk factor for psychiatric and neurological disorders with neurodevelopmental etiologie
299 most common form of parkinsonism, a group of neurological disorders with Parkinson disease-like movem