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1 bles the human capacity both cognitively and neurologically.
2               Before MRI, rats were examined neurologically.
3 -hour survivors were normal or nearly normal neurologically.
4 CP2 levels in brain is strongly suggested by neurologically abnormal phenotypes of mouse models with
5 ta4-7/Delta4-7) mice are growth retarded and neurologically abnormal.
6 od establishes the feasibility of monitoring neurologically active amine content and potentially othe
7  instrument is used for the determination of neurologically active biogenic amines, especially tyrami
8                  The higher AdoMet levels in neurologically affected patients may result from inhibit
9 nd the initial use of tetrathiomolybdate for neurologically affected patients will change our future
10                                Most improved neurologically after immunomodulatory or tumor-directed
11 sis and management of voiding dysfunction in neurologically and anatomically normal children.
12 udes a population template generated from 30 neurologically and clinically normal non-brachycephalic
13 iduals in this subgroup are less compromised neurologically and display structural brain abnormalitie
14 m to learning and memory in animals that are neurologically and genetically intact.
15 ion in brain regions of an individual with a neurologically and pathologically distinct repeat expans
16 ogical function and neuroimaging findings in neurologically asymptomatic adults with SCA and controls
17                    Patients in cohort A were neurologically asymptomatic and were not receiving corti
18                            Among 32 patients neurologically asymptomatic at diagnosis, 16 (50%) devel
19                Lorenzo's oil administered to neurologically asymptomatic boys who are less than 6 yea
20                               A total of 269 neurologically asymptomatic cancer patients were enrolle
21 cted from ischemic stroke patients (n = 39), neurologically asymptomatic controls (n = 20), and strok
22 tile range [IQR], 3-8 samples/patient) in 75 neurologically asymptomatic human immunodeficiency virus
23 d, more important, from HTLV-I seropositive, neurologically asymptomatic individuals.
24                     A relevant proportion of neurologically asymptomatic participants had abnormal CS
25               There is a high risk for adult neurologically asymptomatic patients to develop neurolog
26          Early diagnosis is important in the neurologically asymptomatic stage.
27 untless apparently uneventful anesthetics in neurologically at-risk patients.
28 component of anesthesia in neurosurgical and neurologically at-risk patients.
29 , psychiatric features are sometimes seen in neurologically based movement disorders.
30  patients with motor disability to measure 3 neurologically based subtypes of apathy: Executive, Emot
31                   Compared with kidneys from neurologically brain dead (NBD) donors, DCD kidneys had
32                        Recent work involving neurologically chimeric mice may suggest that such worri
33 om sleep in four subjects with high (>/= C3) neurologically complete spinal cord injuries while on co
34 dation can be systematically assessed in the neurologically critically ill; the majority can also be
35 red with those from a young (29 years ave.), neurologically-damaged Mexico City cohort.
36         Sacks attributed the experience to a neurologically determined disorder of body-image and bod
37 re to donate their organs and who are either neurologically devastated or imminently dying should be
38          Taken together, our findings reveal neurologically dissociable PFC and parieto-occipital sys
39  did not significantly improve survival with neurologically favorable outcome at 180 days compared wi
40  to 29 minutes of CPR (8 of 8) survived with neurologically favorable status compared with 24% (24 of
41 ted odds ratio, 1.57; 95% CI, 1.25-1.96) and neurologically favorable survival (adjusted proportion,
42 re-hospital AAM approach was associated with neurologically favorable survival (CPC 2) in OHCA.
43            ECPR was associated with improved neurologically favorable survival at all CPR durations <
44 ependently associated with decreased odds of neurologically favorable survival compared with conventi
45  CI, 1.51-2.79) was associated with improved neurologically favorable survival compared with no BCPR
46            CO-CPR was associated with better neurologically favorable survival compared with NO-CPR i
47                            The likelihood of neurologically favorable survival declines with prolonge
48    Analysis of the effect of CPR duration on neurologically favorable survival demonstrated significa
49 e survival demonstrated significantly higher neurologically favorable survival for UMN-ECPR patients
50 was associated with a progressive decline in neurologically favorable survival in both groups.
51 suscitation (ECPR) has been shown to improve neurologically favorable survival in patients with refra
52 d with return of spontaneous circulation and neurologically favorable survival to hospital discharge.
53  analysis, RB-CPR was associated with better neurologically favorable survival versus NO-CPR in all a
54 e ALPS cohort with CPR >=40 minutes, whereas neurologically favorable survival was 25% (9 of 36) for
55                       In the overall cohort, neurologically favorable survival was associated with RB
56                                              Neurologically favorable survival was significantly high
57                         Overall survival and neurologically favorable survival were 11.3% (440 of 390
58                       Similarly, the odds of neurologically favorable survival were significantly low
59 ort (357,228 patients), the adjusted odds of neurologically favorable survival were significantly low
60 ssociated with improved overall survival and neurologically favorable survival while compression-only
61                         Overall survival and neurologically favorable survival, defined as a Cerebral
62 lyzed and ranked on their ability to predict neurologically favorable survival.
63 tion markedly amplified odds of survival and neurologically favorable survival.
64 n was used to assess effectiveness of AAM on neurologically favorable survival.
65  similarly associated with decreased odds of neurologically favorable survival.
66 justed proportion, 9.5%; 95% CI, 8.28-10.69; neurologically favorable survival: adjusted proportion,
67                                There were no neurologically favorable survivors in the ALPS cohort wi
68  developed, and fewer patients deteriorating neurologically from diagnosis to discharge.
69 hat Neanderthal-derived genetic variation is neurologically functional in the contemporary population
70  also may have an impact on the treatment of neurologically handicapped children in the future.
71 aphy on diffusion spectrum imaging data from neurologically healthy adults (N = 60) was used to map f
72                                        Eight neurologically healthy adults viewed sentences that were
73                                           50 neurologically healthy adults were tested in an online e
74 t right hemisphere activation levels even in neurologically healthy adults.
75 eralized to the left hemisphere (LH) in most neurologically healthy adults.
76  with microbleed evidence of TAI compared to neurologically healthy age-matched controls.
77                           In this study, 320 neurologically healthy ageing participants (age M = 62.6
78         98 (56%, 95% CI 48-63) children were neurologically healthy before their first ever episode a
79                                  Relative to neurologically healthy comparison subjects, the patients
80 ntington's disease patients, and none of the neurologically healthy comparison subjects.
81 21 patients with Huntington's disease and 29 neurologically healthy comparison subjects.
82 ntal Disorders (Fourth Edition) matched with neurologically healthy control individuals.
83 and without anosognosia (n = 15), as well as neurologically healthy control subjects (n = 15).
84 ency of the variant was also assessed in 489 neurologically healthy controls and in publicly availabl
85 ed twenty-five patients with ALS and fifteen neurologically healthy controls in a P300 brain-computer
86 resonance imaging (fMRI) data collected from neurologically healthy controls in a separate experiment
87 rformance of patients with dlPFC lesions and neurologically healthy controls on a memory-guided sacca
88 sequencing in large cohorts of LBD cases and neurologically healthy controls to study the genetic arc
89 or levodopa, and age- and education- matched neurologically healthy controls were assessed using the
90 s from 18 autopsied patients with ALS and 12 neurologically healthy controls, as well as from wild-ty
91 patients without constructional problems and neurologically healthy controls.
92 e epilepsy with hippocampal sclerosis and 75 neurologically healthy controls.
93 iation of the APOE epsilon4 against the 2928 neurologically healthy controls.
94 ex was also very similar to that observed in neurologically healthy controls.
95 rom 106 AD patients, 51 MCI patients, and 29 neurologically healthy controls.
96  verbal and visual creativity as compared to neurologically healthy controls.
97 imately 2-fold improvement in the ability of neurologically healthy human adults to localize mechanic
98                  Across 5 weeks of practice, neurologically healthy human subjects learned either a c
99 these two functional pathways, two groups of neurologically healthy humans were tested using transcra
100 ctural and functional connectivity data from neurologically healthy individuals to elucidate lesion-a
101 thus far come from correlational research in neurologically healthy individuals.
102 ces in fornix white matter microstructure in neurologically healthy participants were related to diff
103 unctional magnetic resonance imaging to scan neurologically healthy participants while they performed
104 up was significantly higher than that in the neurologically healthy subjects (31%).
105 ease (81%) and about double that seen in the neurologically healthy subjects (41%).
106                            We investigated 6 neurologically healthy subjects and 5 PD patients: 2 wit
107  that, compared with age- and gender-matched neurologically healthy subjects, the vmPFC-lesioned pati
108 Lewy body dementia (LBD) patients, and 3,158 neurologically healthy subjects.
109 rker of cerebral function even in apparently neurologically healthy subjects.
110  dementia with Lewy bodies cases versus 2928 neurologically healthy, aged controls.
111  whole-brain MRI scans were acquired from 31 neurologically-healthy participants to empirically measu
112 rone positioning is safe and feasible in the neurologically ill patients with acute respiratory distr
113  and feasibility of prone positioning in the neurologically ill patients.
114          One hundred one patients (78%) were neurologically impaired (NI), 74 (57%) had chronic pulmo
115 ion to jejunostomy feedings is suggested for neurologically impaired after two wrap failures and a pa
116 significant differences were found comparing neurologically impaired and normally developed patients.
117 o the PrP(Sc)106 purified from the brains of neurologically impaired animals, multimeric beta-rPrP106
118  containing bla(KPC-3) in an LTCF caring for neurologically impaired children and young adults.
119         Visual field examination in young or neurologically impaired children is a challenge.
120 al perimetry cannot be performed in young or neurologically impaired children.
121 e few similar data for brain tissue from non-neurologically impaired donors.
122      Dysphagia is a frequent complication in neurologically impaired patients, which can lead to aspi
123                                A 6-month-old neurologically impaired premature female patient was adm
124 p survived for 24 hrs, but all were severely neurologically impaired.
125    Twenty-three patients in both groups were neurologically impaired.
126 why patients are not being assessed properly neurologically, in part as many doctors have limited neu
127         In the past, clinicians treated some neurologically injured patients with the sustained appli
128 turally reorganize when interrogated using a neurologically inspired electrical stimulation protocol,
129 ergency department) and 79 (40 percent) were neurologically intact (good overall capability or modera
130 h 24 ET patients (14 female; 10 male) and 28 neurologically intact (NI) human volunteers (17 female;
131                       The primary end point, neurologically intact 24-hr survival, was evaluated usin
132 ration is related to hippocampal activity in neurologically intact adults.
133 alling for 15 persons with hemiplegia and 12 neurologically intact age-matched control subjects.
134 ting), accurately simulating the behavior of neurologically intact and frontally damaged people.
135           About 53 of 60 patients (88%) were neurologically intact at discharge or follow-up.
136 with ventricular fibrillation were alive and neurologically intact at one year.
137       In conclusion, TAT-PHLPP9c may improve neurologically intact cardiac arrest survival without th
138   This novel approach was validated using, a neurologically intact cohort, a cohort with unilateral P
139 se with partial destruction of the SCN or in neurologically intact control animals.
140  chronically maintained decerebrate (CD) and neurologically intact control rats given fourth-ventricl
141                                       CD and neurologically intact control rats were injected (i.p.)
142                            CD rats, like the neurologically intact controls, decreased intraoral food
143 (taste reactivity) in CD rats, as it does in neurologically intact controls.
144      Among 67 patients, 30 (45%) survived to neurologically intact discharge (overall performance cat
145  in the conventional ACLT model of OA in the neurologically intact dog.
146  cruciate ligament left intact (n = 8) or of neurologically intact dogs that underwent ACLT (n = 7) a
147  brains resist damage and the animals emerge neurologically intact each spring.
148 st functional evidence for such efferents in neurologically intact hamsters by exploiting a phenomeno
149 ivity in response to unilateral stimulation, neurologically intact humans do not experience bilateral
150                              We used fMRI on neurologically intact humans to investigate whether or n
151 ation task that can be learned implicitly by neurologically intact individuals.
152 ted brains and in tonsils and platelets from neurologically intact individuals.
153 ute right hemisphere ischaemic stroke and 24 neurologically intact inpatients on a test of affective
154 movement kinematics has been well studied in neurologically intact nonhuman primates.
155  for 2.5 years in female squirrels that were neurologically intact or in which the hypothalamic supra
156 7.9% of patients as very unlikely to survive neurologically intact or with mild deficits (< 3%); the
157 nd stroke) as well as semantic processing in neurologically intact participants.
158  as functional imaging in stroke aphasic and neurologically intact participants.
159         A total of 100 generally healthy and neurologically intact patients scheduled for radical ret
160 score of 3 +/- 0 vs. five of seven alive and neurologically intact positive pressure ventilation pigs
161 ch of the four aforementioned brain areas of neurologically intact rats but only in the NTS of CD rat
162                             Indicating TPOA, neurologically intact rats conditioned with the odor/tas
163                                              Neurologically intact rats expecting to receive a high-v
164 file resembled that obtained previously with neurologically intact rats tested according to the same
165 n a second experiment, locomotor activity in neurologically intact rats was used.
166 ueous odor as well as each taste stimulus in neurologically intact rats.
167 ke immunoreactivity (FLI) in the striatum of neurologically intact rats.
168  connectivity and cessation likelihood among neurologically intact smokers.
169                        During PI resolution, neurologically intact subjects recruited a BF network th
170 wed "pseudoneglect", the natural tendency of neurologically intact subjects to perceive the left side
171 th spinal cord injury (SCI) when compared to neurologically intact subjects using neuromuscular elect
172                                        Using neurologically intact subjects, Experiment 2 established
173 ionally 73.0%; frequently 70.0%, P=0.06) and neurologically intact survival (rarely 21.9%, occasional
174  rates in 2015-2017 remained proportional to neurologically intact survival (~35-40%) wherever tracke
175 ermia during CPCR would increase the rate of neurologically intact survival after prolonged cardiac a
176 iocerebral resuscitation as a model for CQI, neurologically intact survival of patients with OHCA in
177 QI approach if the community does not have a neurologically intact survival rate of at least 30%.
178 crease cerebral perfusion, thereby improving neurologically intact survival rates following prolonged
179 rees C was associated with increased odds of neurologically intact survival to discharge (odds ratio,
180                          Primary outcome was neurologically intact survival to discharge.
181 ement 33 degrees C period had higher odds of neurologically intact survival to hospital discharge com
182 arge, return of spontaneous circulation, and neurologically intact survival were comparted for patien
183 c arrest survivors has been shown to improve neurologically intact survival, optimal methods to ensur
184 previously been described with high level of neurologically intact survival.
185    From the 150 patients, 129 (86%) remained neurologically intact whereas 21 (14%) developed DP-SCI.
186 ous for the Mln64 mutant allele were viable, neurologically intact, and fertile.
187 timulation of the sacral segments (S1-S2) in neurologically intact, chloralose anesthetized adult mal
188                 Here, we use genetically and neurologically intact, forced desynchronized rats to tes
189 ncy department, and 79 (39%) were discharged neurologically intact.
190 ccurs while SCN circuitry is genetically and neurologically intact.
191 mRNA, and (2) whether this effect is seen in neurologically-intact rats (in previous experiments we s
192 orms state-of-the-art methods in identifying neurologically meaningful brain connectivity substructur
193 n myocardial dysfunction and thereby improve neurologically meaningful survival in a rodent model aft
194                 This study describes a novel neurologically mutant rat exhibiting a forebrain anomaly
195 considered to be diagnostically nonspecific, neurologically nonlocalizing, and, hence, "soft." This s
196 nuous chest compression CPR produces greater neurologically normal 24-hour survival than standard ABC
197                     The primary end point of neurologically normal 24-hour survival was significantly
198                         Both patients became neurologically normal after transplantation.
199 gestation or less who had been classified as neurologically normal at 7.5-8 years.
200 on with age- and region-matched samples from neurologically normal autopsy cases (n = 22).
201 ve sensorimotor deficits resembling those of neurologically normal children with strabismus and ambly
202 5 have more severe deficits, not observed in neurologically normal children.
203 l trans octadecadienoic acid values than did neurologically normal children.
204             In 141 plasma samples (96 PD, 45 neurologically normal control [NC] individuals; 45.4% fe
205  1-related amyotrophic lateral sclerosis and neurologically normal control cases, in order to determi
206 umber of Purkinje cells in 15 HD cases and 8 neurologically normal control cases.
207 opsy from individuals with primary RLS and a neurologically normal control group.
208 two of the previously published mutations in neurologically normal Control individuals.
209  mild cognitive impairment (MCI; n = 75), or neurologically normal control subjects (NC; n = 137) at
210 sease patients and seven healthy age-matched neurologically normal control subjects during intentiona
211 that both regions are actively involved when neurologically normal control subjects name visually pre
212                        The movement speed of neurologically normal control subjects' was over 40% hig
213 r the same neural pathway was used by our 24 neurologically normal control subjects.
214  of Parkinson's disease patients (n=267) and neurologically normal controls (n=270).
215 artile range [IQR] = 1.6-6.7 hours) and from neurologically normal controls (NCs; N = 112).
216 choroid plexus, obtained at autopsy, from 18 neurologically normal controls and 14 individuals who ha
217 h 766 case patients with the disease and 750 neurologically normal controls, and replication series 2
218 42 patients with the ASDs relative to 12,544 neurologically normal controls, to find potentially drug
219  western Washington State and 644 unrelated, neurologically normal controls, we examined whether PD w
220 t and that the risk variants were present in neurologically normal controls.
221 late-onset disease and in 828 North American neurologically normal controls.
222  from 276 patients with sporadic ALS and 271 neurologically normal controls.
223 m neuropathologically confirmed PD cases and neurologically normal controls.
224 eral sclerosis (ALS) patients versus that of neurologically normal controls.
225  subtypes of frontotemporal dementia against neurologically normal controls.
226  made more navigation and safety errors than neurologically normal drivers on a RFT that placed deman
227 e PD (median Hoehn-Yahr stage = 2.0) and 152 neurologically normal elderly adults, all active and lic
228 ta accumulation (normal ageing, n = 13), and neurologically normal elderly with cortical amyloid-beta
229 = 13, age at death: 82.9 +/- 10.8 years) and neurologically normal elderly with extensive cortical am
230 eas from Alzheimer's disease cases (n = 19), neurologically normal elderly without amyloid-beta accum
231  (n = 19; age at death: 84.7 +/- 7.8 years), neurologically normal elderly without amyloid-beta accum
232  performed in vivo MRI on the brain of eight neurologically normal felines.
233 h minor neurological signs (HIV-MCMD) and 93 neurologically normal HIV-seropositive individuals (HIV-
234 V1 in one or both cerebral hemispheres of 15 neurologically normal human brains obtained at autopsy.
235 ration in Parkinson's disease, in brain from neurologically normal individuals and patients with Park
236 d representing TMEM106B CTF, whereas in most neurologically normal individuals it was absent or much
237  was obtained from the left hemisphere of 26 neurologically normal individuals ranging in age from 14
238 e H-reflex, which is absent or very small in neurologically normal individuals, is abnormally large i
239 ls with restless legs syndrome compared with neurologically normal individuals.
240 rrelate of calculation ability in a group of neurologically normal individuals.
241                      Twenty-four healthy and neurologically normal men (mean age, 29 years) reporting
242 rs assayed 5-HIAA in "leftover" CSF from 193 neurologically normal newborns and obtained family psych
243         Seventy-nine drivers with PD and 151 neurologically normal older adults underwent a battery o
244 l T2-weighted fast spin-echo MR images in 30 neurologically normal patients (60 hemispheres) were ret
245 ALS) than in lumbar spinal cord samples from neurologically normal patients.
246 dr2 is not expressed in tumors obtained from neurologically normal patients.
247  accommodative responses compared with their neurologically normal peers.
248 rability of perception focuses on studies of neurologically normal populations.
249 alization were investigated in right-handed, neurologically normal subjects (n = 100) and right-hande
250 nvestigate narrative speech activation in 18 neurologically normal subjects and 17 patients with left
251                          Six (60%) of the 10 neurologically normal subjects had abnormal PET.
252             In this study, we demonstrate in neurologically normal subjects that in addition to small
253 t from our laboratory that used this task in neurologically normal subjects, 'errors' (incongruent tr
254                                           In neurologically normal subjects, a double dissociation in
255 /CT brain acquisitions were obtained from 88 neurologically normal subjects, aged 6 mo to 18 y.
256 termine auditory perceptual thresholds in 14 neurologically normal subjects, and in 31 patients who h
257 patients with Alzheimer's disease, and eight neurologically normal subjects.
258                                              Neurologically normal survival at 24 hours after resusci
259   This study was designed to compare 24-hour neurologically normal survival between the initial appli
260                             Twenty-four-hour neurologically normal survival occurred in seven of 10 g
261 gnificantly better 24-hour postresuscitation neurologically normal survival than did the initial byst
262 o patients with cardiac arrest that improves neurologically normal survival.
263 386 white patients with sporadic ALS and 542 neurologically normal white controls (the discovery seri
264 espite their very low birth weight, all were neurologically normal with no consistent abnormalities o
265 ysis with positron emission tomography in 33 neurologically normal young adults at rest.
266 ren have neurological disabilities, many are neurologically normal, and the latter group provides us
267 d 30 subjects (age, 47 to 82 years) who were neurologically normal, of whom 16 were carriers of the A
268 vered slowly so that by 3-4 months they were neurologically normal.
269  CCS/wild-type-SOD1 dual transgenic mice are neurologically normal.
270 he neuron specific enolase promoter appeared neurologically normal.
271 e spectrum of epilepsy syndromes and in 1299 neurologically-normal controls.
272 ckle cell anemia (HbSS) are at high risk for neurologically overt cerebral infarcts associated with s
273                    These results delineate a neurologically plausible network of converging corticost
274 riable natural history of CMT with regard to neurologically produced musculoskeletal changes.
275 ade cerebral perfusion have been shown to be neurologically protective during ascending aortic replac
276             Ten out of these were considered neurologically recovered (modified Rankin scale of zero)
277 licated on energy, growth, reproduction, and neurologically related processes, which may be responsib
278 ming fundoscopic examination and identifying neurologically relevant findings on another fundoscopy s
279 ed binding sites in the promoters of several neurologically relevant genes including MECP2.
280 f primary human glial precursor cells with a neurologically relevant human herpesvirus causes profoun
281 nolamine (PE) also found in other classes of neurologically relevant lipid species.
282 fy neuronal signaling pathways responsive to neurologically relevant manganese levels, as previous da
283 ein quantitative trait locus analysis of 184 neurologically-relevant proteins, using whole genome seq
284 d mice with a very low minute volume did not neurologically respond to hypercapnia or optogenetic pho
285 cerebral infarcts associated with stroke and neurologically silent cerebral infarcts correlated with
286 n patients with untreated or locally treated neurologically stable brain metastases at baseline.
287 n reuptake inhibitor mirtazapine and remains neurologically stable, with resolution of cerebral lesio
288               In addition, unusual states of neurologically symptomatic cobalamin deficiency are bein
289                                              Neurologically symptomatic patients require carotid reva
290 ndingly, shaking mice are much less impaired neurologically than either Caspr-null or CST-null mice.
291                         As animals recovered neurologically, the VEP latencies decreased in associati
292                      Each finger corresponds neurologically to a spinal-cord segment ranging from C6
293 f postmortem samples of human cerebella from neurologically typical individuals supports the presence
294 e a counterproductive strategy, at least for neurologically typical students.
295 ys-gly (85.0 versus 54.7 microM) than the 11 neurologically unaffected patients.
296  undergone permissive hypercapnia seem to be neurologically unaffected.
297  disease (HD) patients but not in those from neurologically unremarkable controls, suggesting the rel
298         Excluding the patients who presented neurologically unresponsive or with ongoing cardiopulmon
299           Patients who remain clinically and neurologically unstable require urgent transsphenoidal s
300                           Infants who appear neurologically well and have fractures concerning for ab

 
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