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1 stages of development of a simple, specific neuronal connection.
2 u pathology can spread and propagate through neuronal connections.
3 guidance mechanisms during establishment of neuronal connections.
4 e episodes are associated with remodeling of neuronal connections.
5 amaged host neurons and reconstitute damaged neuronal connections.
6 n result in highly localized modification of neuronal connections.
7 nvolvement in the growth and regeneration of neuronal connections.
8 tant in the establishment and maintenance of neuronal connections.
9 visual system, a system with well-described neuronal connections.
10 s provides additional specificity in forming neuronal connections.
11 ience depends at least in part on changes in neuronal connections.
12 ry from injuries depend on the plasticity of neuronal connections.
13 etworks of circulating soluble molecules and neuronal connections.
14 ed, although they broadly involve humoral or neuronal connections.
15 ilk component that promotes the formation of neuronal connections.
16 interpenetrated transistors that mimic real neuronal connections.
17 fully timed reorganization and refinement of neuronal connections.
18 experience plays a crucial role in sculpting neuronal connections.
19 survive after injury and to regenerate lost neuronal connections.
20 axon guidance is necessary to form accurate neuronal connections.
21 development to determine the specificity of neuronal connections.
22 orsolateral PAG (dl-PAG), which has abundant neuronal connections.
23 isms that control the development of complex neuronal connections.
24 to chemoattractant, build tissues, and make neuronal connections.
25 lls, to determine cell lineage, and to trace neuronal connections.
26 n its cleft but also modulate development of neuronal connections.
27 utes to the formation of complex patterns of neuronal connections.
28 s and glia are essential in forming specific neuronal connections.
29 age and large-scale, permanent disruption of neuronal connections.
30 alian brain that helps sculpt the pattern of neuronal connections.
32 We propose BOINC ("barcoding of individual neuronal connections"), a method for converting the prob
33 shifts in the size and signaling strength of neuronal connections, a process known as synaptic plasti
36 circumstances: during development to refine neuronal connections, after injury to clear damaged neur
37 eral different combinations to determine the neuronal connections and cyto- and chemoarchitecture of
38 ecision should make it easier to investigate neuronal connections and find further links between conn
40 y improved our understanding not only of the neuronal connections and function of the healthy brain,
41 ought to play a crucial role in formation of neuronal connections and in experience-dependent modific
42 de information about cortical processing via neuronal connections and temporal interactions of neural
43 cell division in combination with tracers of neuronal connections and time-lapse live imaging, we fou
44 h leads to synaptic dysfunction, the loss of neuronal connections and ultimately progressive neurodeg
45 ed occurrence of cellular apoptosis, reduced neuronal connection, and reduced optokinetic behavioral
47 promote maturation of neurons, refinement of neuronal connections, and assembly of appropriate functi
48 lls and superfluous connections ensures that neuronal connections are appropriately refined, while cl
55 etwork analysis reveals that the patterns of neuronal connections are organized into putative computa
58 athway and synaptological characteristics of neuronal connection between the dorsal PCRt premotor neu
59 ortance of the retinoraphe pathway, a direct neuronal connection between the retina and the dorsal ra
60 ls that form the retinohypothalamic tract, a neuronal connection between the retina and the suprachia
63 ites is crucial for the formation of precise neuronal connections, but the mechanisms underlying deve
64 growth is the first step in the formation of neuronal connections, but the pathways that regulate axo
65 rms of epilepsy are associated with aberrant neuronal connections, but the relationship between such
66 egeneration that is characterized by loss of neuronal connections, but the role of Presenilin in esta
67 roles in the establishment and remodeling of neuronal connections, but the signaling pathways involve
68 r via changes in blood gases or by brainstem neuronal connections, but their ultimate effect is invar
69 vo and may modulate structural remodeling of neuronal connections by controlling the rate of spontane
70 ia also participate in the fine sculpting of neuronal connections by pruning excess axonal projection
75 rtant in the establishment and refinement of neuronal connections during both development and regener
76 sential for the establishment of appropriate neuronal connections during development and regeneration
77 uning and neuronal cell death help to sculpt neuronal connections during development, but their mecha
83 erosis Complex (TSC) show aberrant wiring of neuronal connections formed during development which may
84 ms to exhibit satiety quiescence, suggesting neuronal connection from ASI to RIM and RIC is essential
85 ion of electron-microscopy reconstruction of neuronal connections, genetic manipulations, electrophys
86 t clear whether they will throw light on how neuronal connections give rise to cognitive processes.
87 lity of synapses to modulate the strength of neuronal connections; however, the molecular factors tha
88 or many processes, including immune defense, neuronal connection, hyphal fusion, and sexual reproduct
90 specific mutations alter the development of neuronal connections in different brain regions and whet
92 key event in the development of appropriate neuronal connections in many regions of the central nerv
94 s in the fly including proper development of neuronal connections in photoreceptor cells and axonal t
95 ostsynaptic cells helps shape the pattern of neuronal connections in response to experience, but the
97 de required to establish complex networks of neuronal connections in the brain and the mechanisms of
99 explain the mechanism for specifying diverse neuronal connections in the brain, Sperry proposed that
103 o sensory experience-dependent refinement of neuronal connections in the developing nervous system, b
111 sent only one type of functionally important neuronal connection; in particular, extrasynaptic, "wire
112 t also destroy cells and eliminate important neuronal connections, including light reception pathways
116 n is facilitated through the organization of neuronal connections into topographic maps of space.
118 The establishment of topographic maps of neuronal connections is believed to involve graded repul
121 he brain, appropriate assembly and tuning of neuronal connections is likely to require appropriate fu
123 It has been proposed that the efficacy of neuronal connections is strengthened when there is a per
124 efects include disruptions in the pattern of neuronal connections made by all three classes of R cell
125 this is consistent with the idea that these neuronal connections may encode beliefs about "what I wo
130 vide a framework for ongoing analyses of the neuronal connections of the insular cortex of the macaqu
131 estigate the neurogenesis and time course of neuronal connections of the tectorotundal pathway in chi
133 omechanics and graph theoretical analysis of neuronal connections, or connectomics, to form a neuroco
134 nt in human milk during early lactation when neuronal connections rapidly form in the infant brain.
135 ortex develop by the progressive addition of neuronal connections rather than by sculpting preliminar
139 plasticity, the activity-dependent change in neuronal connection strength, has long been considered a
142 ntrols the remodeling of initially imprecise neuronal connections through the regulation of gene expr
144 r behavior and progressive deconstruction of neuronal connections, ultimately causing age-related neu
145 debris, in addition to helping to refine CNS neuronal connections via microglia-mediated pruning of i
147 m imaging and mutations that affect specific neuronal connections, we show that AWC neurons are activ
148 dic devices to recreate discretely organized neuronal connections, we show that the spread and propag
149 dark cycle, SCN neurons remained intact, and neuronal connections were undisturbed, including photic
150 ubtypes and alter the development of defined neuronal connections which require NMDA receptor activat
151 ent composition of neuronal types and set of neuronal connections, which among other factors, determi
152 generated granule cells (DGCs) form aberrant neuronal connections with neighboring DGCs, disrupting t
153 l forebrain region that shares bidirectional neuronal connections with the hippocampus and has recent
156 rus americanus, and the development of their neuronal connections with the paired olfactory and acces
157 of primate primary visual cortex (V1) depict neuronal connections within and between different V1 lay
158 at regulate formation of precise patterns of neuronal connections within the central nervous system r
159 d in temporal processing and share extensive neuronal connections, yet it remains unclear how these s