ライフサイエンスコーパス: neuronal excitation

1 t unconjugated CGRP8-37, prevented sustained neuronal excitation.

2 synaptic currents, and synaptic response to neuronal excitation.

3 subtype of glutamate receptors that mediate neuronal excitation.

4 dynamin, ERK, and PKC suppressed persistent neuronal excitation.

5 ation of ASICs in the medulla also triggered neuronal excitation.

6 anging from epithelial HCO3 (-) secretion to neuronal excitation.

7 mouse neurons induced by forskolin requires neuronal excitation.

8 s and extrasynaptic receptors in controlling neuronal excitation.

9 conversion of light mechanical stimuli into neuronal excitation.

10 )) from internal stores and elicit prolonged neuronal excitation.

11 le in the brain by controlling the extent of neuronal excitation.

12 athways and contributes to the regulation of neuronal excitation.

13 hibits the Kir3.1/3.2 channels, resulting in neuronal excitation.

14 as mediated by the alpha7 nAChR and required neuronal excitation.

15 s inhibit Kir3 (GIRK) channels, resulting in neuronal excitation.

16 of these hormonal actions would increase VMN neuronal excitation.

17 e stability will have major consequences for neuronal excitation.

18 t transcriptional changes observed following neuronal excitation.

19 ement as the sole basis for the drug-induced neuronal excitation.

20 BA may exert depolarizing actions leading to neuronal excitation.

21 uch as 175 microm, depending on the level of neuronal excitation.

22 +) competes Ca(2+) to inhibit its influx and neuronal excitation.

23 rons promotes chronic seizures by increasing neuronal excitation.

24 urrogate marker for metabolic changes during neuronal excitation.

25 e cyclic nucleotide-gated (CNG) channels and neuronal excitation.

26 lated responding, moreover, was dominated by neuronal excitations.

27 on, which correlated positively with delayed neuronal excitations.

28 sitive feedback loop, whereby sleep loss and neuronal excitation accelerate the accumulation of Abeta

29 We report that neuronal excitation acts with ER Ca(2+) stores to induce

30 Neuronal excitation and cardiorespiratory effects follow

31 A model to link neuronal excitation and CSAD activation by a Ca2+-depend

32 emerging data point to an imbalance between neuronal excitation and inhibition in at least a subgrou

33 tribute to the receptor's role in modulating neuronal excitation and inhibition patterns, including l

34 These data suggest that neuronal excitation and inhibition, depending on the ana

35 s strongly influenced by the balance between neuronal excitation and inhibition.

36 ns in many genes affecting the ratio between neuronal excitation and inhibition.

37 tory brainstem is based on an interaction of neuronal excitation and inhibition.

38 Thus, calexcitin is implicated in neuronal excitation and plasticity.

39 (Na(+)) are major charge carriers mediating neuronal excitation and play a fundamental role in brain

40 ammatory pain hypersensitivity by increasing neuronal excitation and reducing inhibition.

41 the BAD-K(ATP) axis in metabolic control of neuronal excitation and seizure responses.

42 ed calcium channels (VGCCs) are essential to neuronal excitation and signal transduction.

43 NS implies that they serve distinct roles in neuronal excitation and signaling within the bowel.

44 hosphate (InsP(6)) levels rise and fall with neuronal excitation and silence, respectively, in the hi

45 xidant sensor in sensory neurons, initiating neuronal excitation and subsequent physiological respons

46 action in cryptochrome that alters levels of neuronal excitation, and represent a vital step forward

47 ic glutamate receptors (iGluRs), which drive neuronal excitation, and type A gamma-aminobutyric acid

48 red spontaneously active cells, and enhanced neuronal excitations associated with movement.

49 ced elevation of extracellular glutamate and neuronal excitation augmented chemoreflex-mediated press

50 al neuron excitability, facilitating greater neuronal excitation by a subsequent stimulus.

51 Here, we induced neuronal excitation by anodal transcranial direct curren

52 In sum, PVN GGS buffers neuronal excitation by apparent "over-filling" of vesicl

53 To examine the inhibition of neuronal excitation by GIRKs, we overexpressed GIRKs in

54 smitter, GABA provides the dominant mode for neuronal excitation by inducing membrane depolarization

55 hat acidosis may inhibit low [Ca2+]o-induced neuronal excitation by inhibiting the activity of the cs

56 Neuronal excitation can be substantially modulated by al

57 Neuronal excitation can induce new mRNA transcription, a

58 alter the demand for active ribosomes, like neuronal excitation, cause increased inactivation of red

59 ole treatment, which decreases glutamatergic neuronal excitation, decreases the synthesis and levels

60 yte density in the juvenile amygdala reduces neuronal excitation during play.

61 By contrast, increasing neuronal excitation during the same period in wild-type

62 X formation in the adult rat brain following neuronal excitation evoked by seizure activity in vivo.

63 rring irritant compounds because the initial neuronal excitation evoked is followed by a long-lasting

64 Neuronal excitation imposes a high demand of ATP in neur

65 Patterns of neuronal excitation in complex populations can be mapped

66 n the cerebellar cortex limits the extent of neuronal excitation in part through activation of metabo

67 iated inhibition is critical for restraining neuronal excitation in the brain, and therefore potentia

68 sistent with pyrethroids producing increased neuronal excitation in the cortex following a low-dose i

69 smitter, GABA, serves as the major source of neuronal excitation in the developing brain.

70 Therefore, we tested the hypothesis that neuronal excitation in the RP mediates tachycardia seen

71 area with the electrocatalytic fibres evoked neuronal excitation in the targeted brain region and its

72 ct stimulation of superior colliculus evoked neuronal excitation in ZIv and caused inhibition of spon

73 nal fluid, and Fra-like activity (indicating neuronal excitation) in paraventricular nucleus.

74 m increased LC cFos expression, a marker for neuronal excitation, in both No MSS and MSS mice, this i

75 Zn(2+) release occurs with neuronal excitation, including seizure events, and exert

76 y, but was less effective in attenuating the neuronal excitations induced by GLU or associated with m

77 yers, or indeed whether modulation reflected neuronal excitation, inhibition, or both.

78 romosome 21 that, when overexpressed, alters neuronal excitation-inhibition balance and increases GAD

79 ced ANK3 exon 1b isoform expression disrupts neuronal excitation-inhibition balance.

80 er spectrum, which is thought to reflect the neuronal excitation/inhibition ratio.

81 Both auditory and visual stimuli produced neuronal excitation, involving a greater than 5-fold inc

82 SIGNIFICANCE STATEMENT Dynamic regulation of neuronal excitation is a fundamental mechanism for infor

83 Neuronal excitation is generally associated with an incr

84 Neuronal excitation is required for normal brain functio

85 Neuronal excitation is unexpected in the presence of thi

86 associated with fluctuations in spontaneous neuronal excitation, less is known about state-dependent

87 The sonication did not elicit tissue-level neuronal excitation, measured by an electroencephalogram

88 However, recent data demonstrate that neuronal excitation modulates both the pial and meningea

89 In both subgroups, neuronal excitations occurred with short latencies (4-8

90 on of the ARC or vlPAG respectively leads to neuronal excitation of the both nuclei during EA.

91 al amygdala (CeA)(2); however, the source of neuronal excitation of the CeA that contributes to high-

92 cid (GABA) transporter 1 (GAT1)(1) regulates neuronal excitation of the central nervous system by cle

93 Although optogenetic approaches permit neuronal excitation or inhibition at the network level,

94 xogenous Plk2 expression or chronic elevated neuronal excitation, produces exaggerated homeostatic re

95 an attenuation of alpha activity induced by neuronal excitation related to stimulus onset.

96 rotein-synthesis-dependent implementation of neuronal excitation-repression equilibria.

97 naptic Ca2+ channels are crucial elements in neuronal excitation-secretion coupling.

98 2.1.1) isoforms II and VII are implicated in neuronal excitation, seizures, and neuropathic pain (NP)

99 Ischemia and seizure cause excessive neuronal excitation that is associated with brain acidos

100 TRPA1 channels are required for neuronal excitation, the release of inflammatory neurope

101 ite individual-particle potentials below the neuronal excitation threshold.

102 The device serves to regulate neuronal excitation through G-protein-coupled, metabotro

103 cal excitation is overwhelmed by spontaneous neuronal excitation through the modulation of alpha osci

104 S by demonstrating their inhibition produced neuronal excitation to alter basal cardiorespiratory fun

105 o control somatic Ca(2+) signals that couple neuronal excitation to gene expression.

106 ochondria as a novel regulatory mechanism in neuronal excitation-transcription coupling.

107 ium flux and glutamate uptake in response to neuronal excitation under physiological and pathological

108 The excessive neuronal excitation underlying several clinically import

109 In separate experiments, neuronal excitation was found to be suppressed by anesth

110 Golgi satellites that formed during neuronal excitation were in close association with endop

111 both calcium-signaling genes responsible for neuronal excitation, were deleted in 16 cases and duplic

112 bradycardia through TRPV1 sensitization and neuronal excitation, which may contribute to the pathoge

113 nel as a key mediator for E(2)-induced rapid neuronal excitation, which may have a broad impact on mu

114 ChRger2 enables light-induced neuronal excitation without fiber-optic implantation; th