ライフサイエンスコーパス: neuronal marker

1 -stained with antibody to beta(III) Tubulin (neuronal marker).

2 c (SMC marker), Kit (ICC marker) and Pgp9.5 (neuronal marker).

3 ease in newborn cells expressing an immature neuronal marker.

4 function of this popular activity-dependent neuronal marker.

5 ively in migrating neurons, and Hu, an early neuronal marker.

6 hus preceding that detected by using the pan-neuronal marker.

7 t at sites of plasticity, and TuJ1, an early neuronal marker.

8 hydroxyephedrine (HED), an established heart neuronal marker.

9 Elav has long served as the canonical neuronal marker.

10 o decompressive craniectomy, we used NeuN, a neuronal marker.

11 aining was co-localized with that of NeuN, a neuronal marker.

12 ching as evaluated by immunocytochemistry of neuronal markers.

13 rs develop persistent deficits in brain 5-HT neuronal markers.

14 ion of graft-derived cells expressing mature neuronal markers.

15 erentiated regions Sox1 co-labeled only with neuronal markers.

16 sitions within the DG and expressed immature neuronal markers.

17 ion of GFP-labeled cells to express immature neuronal markers.

18 neuronal counts or other immunohistochemical neuronal markers.

19 beled cells that expressed each of the eight neuronal markers.

20 rogenitor vimentin and nestin but not mature neuronal markers.

21 the neocortex and midbrain expressed mature neuronal markers.

22 U-labeled cells in the neostriatum expressed neuronal markers.

23 and represses induction of immunophenotypic neuronal markers.

24 uronal differentiation and expression of pan-neuronal markers.

25 immature, and they express only a subset of neuronal markers.

26 neurite extension and enhanced expression of neuronal markers.

27 and micro- and macroglia, and attenuation of neuronal markers.

28 egions of the spinal cord that express motor neuronal markers.

29 o staged by the expression of early and late neuronal markers.

30 ith the expression of active, differentiated neuronal markers.

31 s and decreased the expression of subsequent neuronal markers.

32 ffness of 700 Pa maximizes expression of pan-neuronal markers.

33 characterized by the expression of multiple neuronal markers.

34 of Pax3 expression and the absence of mature neuronal markers.

35 STAT3 reversed SPARC-mediated expression of neuronal markers.

36 ression of Notch signaling and expression of neuronal markers.

37 e cells were never colabeled with any of the neuronal markers.

38 Most contained one or more neuronal markers; a few were positive for A2B5 and/or GF

39 ir polarization and enrichment with specific neuronal markers, accompanied by a resurgence of spontan

40 d between brain volume and other putative MR neuronal markers also indicate that atrophy reflects axo

41 l dispersion was examined both with an early neuronal marker and through the focal application of DiI

42 SP showed increased immunoreactivity for the neuronal markers and a decrease in Notch1 expression and

43 rate of neurogenesis, based on expression of neuronal markers and cRNA microarray analyses.

44 Sling cells label with a number of neuronal markers and display electrophysiological proper

45 hologies, expressed retinal subtype-specific neuronal markers and displayed neuron-like physiological

46 mentation significantly enhanced cholinergic neuronal markers and facilitated induction of hippocampa

47 Both populations express neuronal markers and have extensive dendritic arborizati

48 gonadal steroids, resulting in masculinized neuronal markers and male sexual behavior in female rats

49 , no precocious expression of differentiated neuronal markers and no interaction of REST with miR-21.

50 neurons were identified based on morphology, neuronal markers and polarity of projection.

51 Many of the newborn cells express neuronal markers and show functional phenotypes relevant

52 However, the expression levels of neuronal markers and sodium channel function analyses in

53 expressed proliferation markers and immature neuronal markers and that lacked evidence of DNA damage

54 tion of precursor cells that express general neuronal markers and the granule cell marker RU49, but f

55 Improved methods for detecting neuronal markers and the retrograde tracer Fluoro-Gold (

56 tive for microtubule-associated protein 2 (a neuronal marker) and glial fibrillary acidic protein (an

57 biomarkers TUBB3 (melanocyte development and neuronal marker) and its upstream molecule FIG4 (phospha

58 eased the proportions of both beta-tubulin- (neuronal marker) and O4- (oligodendroglial marker) immun

59 lt tissues, characterization of an embryonic neuronal marker, and measurement of cellular proliferati

60 rotein gene product 9.5 (PGP 9.5), a general neuronal marker, and vasoactive intestinal peptide (VIP)

61 ave rise to myofibroblasts, cells expressing neuronal markers, and cells of uncharacterized phenotype

62 on of a neuronal morphology, upregulation of neuronal markers, and establishment of outward rectifyin

63 5) neurons ectopically expressed subcerebral neuronal markers, and extended their axons into subcereb

64 ion of the p21(WAF1) promoter, expression of neuronal markers, and neurite extension.

65 hese cells in the interstitial space express neuronal markers, and their number correlates with plasm

66 and contain cells immunopositive for the pan-neuronal markers ANNA-1 and PGP9.5, the inhibitory neuro

67 Immunohistochemical staining with the pan-neuronal marker, ANNA-1, revealed that the transplanted

68 By E15, cells expressing neuronal markers are also PSA-NCAM immunoreactive and be

69 it is not known whether all cells expressing neuronal markers are capable of firing action potentials

70 nd sAPPalpha, but cells expressing GABAergic neuronal markers are overrepresented.

71 antly upregulated gene expression of several neuronal markers as well as TRPV1.

72 ression of neuronal subtype-specific and pan-neuronal markers, as well as into Schwann cells and sate

73 Despite the lack of effect of CP13 on neuronal markers associated with Huntington's disease pa

74 However, some cells expressing neuronal markers at E11.5 or E12.5 did not exhibit an ac

75 of neural crest-derived cells expresses pan-neuronal markers at early stages of ENS development (at

76 Of the cells colabeled with BrdU and a neuronal marker, at least half had an inhibitory phenoty

77 scence-activated cell sorting, expressed the neuronal marker beta III tubulin.

78 and vascular tissue that did not express the neuronal marker beta-III tubulin, but was positive for F

79 at the lesion border expressed the immature neuronal marker beta-tubulin, although a small percentag

80 re were confirmed by immunostaining with pan-neuronal marker, beta-III tubulin and two populations we

81 l differentiation media, a peak level of the neuronal marker, beta-tubulin III, was observed on vmIPN

82 d the neural progenitor marker nestin or the neuronal markers beta3-tubulin, syntaxin, and VC1.1 show

83 fferentiate into neurons and express the pan-neuronal markers, beta3-tubulin (Tuj1) and Hu showing th

84 ignificant increase in the expression of pan-neuronal markers (betaIII-tubulin, PGP 9.5) as well as m

85 rentiated hNSCs showed higher percentages of neuronal markers, better hNSC engraftment, and improved

86 ally homogenous population and that specific neuronal markers (Brn3 and neurofilament) can partly dis

87 Here we show that the neuronal marker BRN3A/POU4F1 is expressed abundantly at

88 ury requires not only BrdU uptake and mature neuronal markers but also evidence showing absence of ap

89 Immediately after birth, neurons express pan-neuronal markers but do not transcribe terminal differen

90 teristics of granule neurons and expressed a neuronal marker by the 3-week time point.

91 a activation was assessed by quantifying the neuronal marker c-fos.

92 Further, fibers expressing the sensory neuronal markers calcitonin gene-related protein and sub

93 e neurochemically characterized by using six neuronal markers: calcitonin gene-related peptide (CGRP)

94 iation, and concomitant up-regulation of the neuronal markers calretinin and calbindin, as assessed b

95 these cells were neurons and coexpressed the neuronal markers class IIIbeta-tubulin or neuronal nucle

96 pha-tubulin immunostaining, making these two neuronal markers complementary to each other and, theref

97 tion factors and, importantly, a plethora of neuronal markers, consistent with ectodermal differentia

98 ion and ectopically expressed the early-born neuronal marker CTIP2.

99 ded proliferating progenitors expressing the neuronal marker Dcx (Doublecortin) in the SVZ and the DG

100 ression and observed increased expression of neuronal markers Dcx, Map2, and Tubb3.

101 eurons; thus, expression of the dopaminergic neuronal markers, dopamine, tyrosine hydroxylase, and do

102 labeled with BrdUrd coexpressed the immature neuronal markers doublecortin and proliferating cell nuc

103 GFR2/Flk-1 was colocalized with the immature neuronal marker, doublecortin (Dcx).

104 E14.5), and expression of Tuj1, an important neuronal marker during development, was down-regulated.

105 intenance, as Abl mutant photoreceptors lose neuronal markers during late pupal stages but do not re-

106 neurons, as evidenced by coexpression of mDA neuronal markers (e.g., TH, Pitx3, Nurr1, and Lmx1b) and

107 Subsequent to expression of the neuronal marker elav, dVDUP1 is up-regulated to varying

108 euronal morphologies and expressing multiple neuronal markers, even after downregulation of the exoge

109 sion proteins characteristic of EFT activate neuronal marker expression to confer a neural phenotype

110 f Tp53 rescues the decrease in proneural and neuronal marker expression, which is thus an off-target

111 ly regulate RA-induced neurite outgrowth and neuronal marker expression.

112 n and the disease-specific loss of autonomic neuronal marker expression.

113 CGE lineage, because of the lack of specific neuronal markers for these interneuron subtypes.

114 The expression patterns of the neuronal marker FOS were used to assess neurological act

115 Loss of N-acetylaspartate, a putative neuronal marker, from gray matter preceded that observed

116 ses revealed a marked reduction in GABAergic neuronal markers (GAD65, GAD67, VGAT) and increased pro-

117 cells, as determined by an overexpression of neuronal marker genes.

118 or in association with RA express the caudal neuronal marker Hoxc4.

119 ydroxylase expression in addition to the pan-neuronal marker Hu in nonneural ectoderm.

120 nsory ganglia, whereas expression of the pan-neuronal marker Hu is largely unperturbed; GABAergic and

121 Here, Dlx3, Dlx5, Pax6 and the pan-neuronal marker Hu serve as molecular labels to follow t

122 Immunostaining for active caspase-3 and the neuronal marker Hu specifically confirms the presence of

123 birth (P1), all CARTp-IR cells expressed the neuronal marker Hu.

124 between the muscle layers and expressed the neuronal marker Hu.

125 differentiate into neurons that express the neuronal markers Hu or calretinin.

126 e ENS of the colon were quantified using pan-neuronal marker, Hu, and neuronal nitric oxide synthase

127 fferentiation) and reduced the expression of neuronal markers, Hu antigen, and MAP2.

128 croscopic analysis and staining with the pan-neuronal marker HuC confirmed that this loss of dopamine

129 tion, and many newborn cells coexpressed the neuronal marker HuC/D at 30 days, suggesting they had di

130 GFAP and absence of co-localization with the neuronal marker HuC/D.

131 were also immunopositive for the nociceptive neuronal markers IB4, TRPV1, CGRP, and substance P.

132 owing: 1) extraction, purification, and NeuN neuronal marker immunotagging of nuclei from adult human

133 rcentage of BrdUrd-labeled cells expressed a neuronal marker in the dentate gyrus than in either neoc

134 Notably, the loss of subcortical cholinergic neuronal markers in aged monkeys was nearly completely r

135 lts suggest that SPARC induces expression of neuronal markers in medulloblastoma cells through its in

136 peptide colocalizes with both astrocytic and neuronal markers in mouse nRT, and investigate the role

137 strated significant reductions in functional neuronal markers in subcortical brain regions in primate

138 ciculation and loss of expression of several neuronal markers in the embryonic peripheral and central

139 n promotes the expression of ectopic sensory neuronal markers in the lateral ectoderm, suggesting a r

140 These results indicate that changes in neuronal markers in the spinal cord can persist after ap

141 dU) incorporation and expression of immature neuronal markers in the subgranular zone (SGZ) of the hi

142 Here we sought to identify reliable efferent neuronal markers in the vestibular periphery of turtle,

143 oration of BrdUrd and expression of immature neuronal markers in two neuroproliferative regions: the

144 es the number of dividing cells that express neuronal markers in vitro.

145 thors measured N-acetylaspartate (a putative neuronal marker) in the right and left thalamus of 17 ma

146 BrdU-positive cells expressed NeuN, a mature neuronal marker, in regions of cortex undergoing targete

147 the STN neurochemical organization based on neuronal markers including parvalbumin (PV), calretinin

148 aracterized using antibodies against several neuronal markers, including those expressed by sensory n

149 Neuronal markers; including PGP9.5, GAP-43, acetylated t

150 (1)H MRS revealed age-dependent decreases in neuronal markers, increases in glial markers, but no det

151 nstrated that both excitatory and inhibitory neuronal markers labeled subpopulations of E2-BSA-FITC b

152 vealed that Wnt/beta-catenin antagonists and neuronal markers localized to a particular cell populati

153 (AD) is marked by cholinergic hypofunction, neuronal marker loss, and decreased nicotinic acetylchol

154 e in vitro observation that induction of the neuronal marker MAP-2 in metastatic melanoma cells is ac

155 luciferase-positive cells also expressed the neuronal marker MAP-2.

156 Decreased expression of the neuronal marker MAP2 and synaptic markers PSD95 and syna

157 res were double-labeled against GABA and the neuronal marker MAP2, and the percentage of neurons that

158 orrespondingly, the expression of the mature neuronal markers Map2 a/b and neurofilament was increase

159 region, newly generated cells expressed the neuronal marker microtubule-associated protein-2, or neu

160 orated within the inner retina expressed the neuronal markers microtubule associated protein (MAP)-2

161 ry indicated that PR-ir colocalizes with the neuronal marker, microtubule associated protein-2, but n

162 h vimentin, and Olig2 was coexpressed with a neuronal marker, microtubule-associated protein 2.

163 in the lesioned brain with some labeled with neuronal marker mitogen-activated protein 2 and decorate

164 mber or volume correlated with levels of the neuronal marker N-acetyl aspartate (NAA).

165 ients showed reduced AN activation and lower neuronal marker N-acetylaspartate in prefrontal and pari

166 onal neurohistopathology and decrease of the neuronal marker N-acetylaspartate measured by HRMAS 1HMR

167 eurons, as revealed by the expression of the neuronal marker N-tubulin, is prevented by Xiro3 express

168 nd expression of a terminally differentiated neuronal marker, N-tubulin, was diminished upon loss of

169 tion factors, Xath3 and MyT1, as well as the neuronal markers, N-tubulin and elrC.

170 ransit amplifying cell marker MASH1, nor the neuronal marker NCAM.

171 S100B, GFAP and oligodendrocyte marker O4), neuronal markers (nestin and B-III-tubulin) and fibrobla

172 proliferating cell nuclear antigen [PCNA]), neuronal markers (nestin, neuron-specific class III beta

173 oliferating cell nuclear antigen [PCNA]); of neuronal markers (nestin, neuron-specific class III beta

174 of cells that stained positive for both the neuronal marker NeuN and 5-bromo-2'-deoxyuridine (BrdU;

175 the cell proliferation marker BrdU with the neuronal marker NeuN and peptides revealed a marked stim

176 oform TASK-1 was mainly colocalized with the neuronal marker NeuN in hippocampal pyramidal neurons an

177 Double staining studies using the neuronal marker NeuN indicated that >90% of rAAV-transdu

178 calizing the bradykinin B2 receptor with the neuronal marker NeuN or the astrocytic marker glial fibr

179 x2(+) precursors acquired positivity for the neuronal marker NeuN over time and integrated into cellu

180 onal populations and dual labelling with the neuronal marker NeuN revealed 28.5+/-1.9% of NeuN labell

181 he 67-kDa isoform of GAD (GAD67), and 7) the neuronal marker NeuN was performed in mice expressing gr

182 ile 88 +/- 2% of SNc neurons labelled by the neuronal marker NeuN were co-labelled for the catecholam

183 Using the neuronal marker NeuN, almost every neuron in LS (> 90%)

184 soma) and exhibited immunoreactivity to the neuronal marker NeuN, and to glutamic acid decarboxylase

185 Klotho protein co-localized with both the neuronal marker NeuN, as well as, oligodendrocyte marker

186 entified in deeper cortical layers using the neuronal marker NeuN, showed a marked reduction in neuro

187 s labeled with BrdU (4 weeks) and the mature neuronal marker NeuN.

188 Some of these dividing cells express the neuronal marker NeuN.

189 iated with neurons, identified using the pan-neuronal marker NeuN.

190 e cells migrate into the GCL and express the neuronal markers NeuN and calbindin-D28k.

191 ns were immunolabeled with antibodies to the neuronal markers NeuN, glutamate, and calbindin-D28k, an

192 (Ad-DsRed-SP) elevated the expression of the neuronal markers NeuN, nestin, neurofilament, and MAP-2

193 oreactivity colocalized exclusively with the neuronal marker (NeuN) and did not colocalize with the s

194 hat beta-galactosidase is coexpressed with a neuronal marker (NeuN) and with parvalbumin and neuropep

195 r/CA3 border also were double-labeled with a neuronal marker (NeuN).

196 kers; doublecortin and PSA-NCAM as the early neuronal marker, NeuN to identify mature neurons, and gl

197 g2, whereas PI5P4Kbeta co-localizes with the neuronal marker, NeuN.

198 BrdU-positive VB cells coexpressed neuronal markers: NeuN, HuC/D, microtubule-associated pr

199 Pax3+ placode cells begin to express the pan-neuronal marker neurofilament while still in the ectoder

200 and/or double or triple immunostaining with neuronal markers neurofilament 200 (NF200) and choline a

201 er of 20.78 +/- 2.5 microm and expressed the neuronal markers neurofilament 200, betaIII-tubulin, pro

202 cating decreased astrocyte activation, while neuronal markers (neurofilament-M, betaIII-tubulin) and

203 Over time, CHX10(+) cells expressed neuronal markers [neurofilament, NeuN, and vesicular glu

204 cells had neuronal morphology, expressed the neuronal marker neuron specific enolase, and were incorp

205 labeled cells primarily colocalized with the neuronal marker neuron-specific nuclear protein and rare

206 ochemistry and immunohistochemistry with the neuronal marker neuronal nuclear antigen (NeuN) in rat b

207 nNOS-IR positive cells contained IR for the neuronal marker neuronal nuclear antigen (NeuN), none of

208 n, new cells in the adult mPFC stain for the neuronal marker neuronal nuclear protein, although enhan

209 roglial marker OX-42 and not with either the neuronal marker neuronal-specific nuclear protein or the

210 These cells colocalized with the neuronal marker neuronal-specific nuclear protein.

211 measured using immunohistochemistry for the neuronal markers neuronal nuclear antigen and calbindin

212 tin immunoreactivity was co-localized with a neuronal marker (neuronal specific enolase).

213 unocytochemistry was performed to detect the neuronal-markers; neuronal nuclei (NeuN), microtubule-as

214 us, a subset of which express the orexigenic neuronal marker, Neuropeptide-Y, and respond to fasting

215 istochemically with antisera against the pan-neuronal marker neurotubulin.

216 showed a HFD-induced upregulation of ATF3, a neuronal marker of injury, in lumbar dorsal root ganglia

217 uding, neurite extension and upregulation of neuronal markers of differentiation.

218 Flies at 10 degrees C show neuronal markers of elevated sleep pressure, including i

219 sing double labeling techniques with several neuronal markers of known distribution patterns served t

220 nd for the first time, expression of typical neuronal markers of NT2/D1 differentiation.

221 Western blot analysis of PGP 9.5, a generic neuronal marker, of the colonic tissues were employed to

222 rast to MASH1, fails to induce expression of neuronal markers or a neuronal morphology, but does indu

223 ivity of "neuron"-like cells (expressing pan-neuronal markers or with neuronal morphology) in the gut

224 immunohistochemistry using PGP9.5 (a general neuronal marker) or CGRP (calcitonin gene-related peptid

225 c marker), microtubule-associated protein 2 (neuronal marker), or 2',3'-cyclic mononucleotide 3'-phos

226 unoreacted with antibodies either to NeuN, a neuronal marker, or to the 67-kDa isoform of glutamic ac

227 e study groups in the marker sets cytokines, neuronal markers, or anti-cytokine autoantibodies.

228 does not affect neurogenesis, expression of neuronal markers, or initial axonal growth.

229 ns survived and expressed the proprioceptive neuronal marker parvalbumin.

230 ion analysis of cells showed the presence of neuronal markers peripherin, PGP9.5, HuD, tau, synaptic

231 skin innervation, when antibodies to the pan-neuronal marker PGP (protein gene product) 9.5 were used

232 Total nerve density, revealed by the pan-neuronal marker PGP 9.5, was also greater after estrogen

233 the stem cell marker nestin but not the pan-neuronal marker PGP 9.5.

234 estrous rats and immunostained using the pan-neuronal marker PGP9.5 and specific markers for calciton

235 nd non-peptidergic nerve fibers with the pan-neuronal marker PGP9.5, the expression was significantly

236 d to be a subset of cells that expressed the neuronal marker PGP9.5.

237 Cultures were immunostained for the pan neuronal marker, PGP9.5, and TUNEL.

238 s directed against protein gene product (pan-neuronal marker; PGP9.5), tyrosine hydroxylase (sympathe

239 efined by coexpression of the proprioceptive neuronal marker pickpocket (ppk) and the sex-determinati

240 gene fruitless (fru) and the proprioceptive neuronal marker pickpocket (ppk) in the female reproduct

241 dult-formed cells, some of which expressed a neuronal marker, present in the bulb 3 weeks later.

242 at young neurons in adult rats show a mature neuronal marker profile and activity-induced immediate e

243 rine (NE) uptake-1 site density, sympathetic neuronal marker profiles, tissue-reduced glutathione/oxi

244 oxyuridine into cells that express the early neuronal marker protein doublecortin in the subventricul

245 GSI treatment also induced neuronal marker protein expression, as shown by Western

246 ORNs also expressed the neuronal marker protein gene product (PGP) 9.5 and the c

247 Immunostaining for the pan-neuronal marker protein gene product (PGP) 9.5 revealed

248 e-labeling experiments using the nonspecific neuronal marker Protein Gene Product 9.5 were performed

249 escence, we labeled sections for the general neuronal marker, protein gene product 9.5.

250 rporation into cells that expressed immature neuronal marker proteins and increased cell number in cu

251 , we investigated the expression of immature neuronal marker proteins that signal the birth of new ne

252 of BCCAO, in the absence of focal changes of neuronal marker proteins.

253 d recovery in the levels of several striatal neuronal markers severely impaired in R6/2 mice.

254 erentiated state expressing trace amounts of neuronal markers, SH-SY5Y cells maintain spontaneous hig

255 s 14 and 18, combined with immunolabeling of neuronal markers, showed that subpopulations of central

256 e GLI3 repressor and increase in the ventral neuronal markers specified by SHH.

257 xA8 in AnxA8-depleted cells led to decreased neuronal marker staining, and normal cell growth as judg

258 osed to growth factors and low levels of pan-neuronal markers such as beta-III-tubulin.

259 s that EWS/ETS proteins induce expression of neuronal markers such as BRN3A in EFT by showing that th

260 eled cells that do not express aromatase nor neuronal markers such as Hu or NeuN in the POM and other

261 not only Neurog1 and Neurod1 but also mature neuronal markers such as neurofilament, indicating that

262 creased apoptosis, and reduced expression of neuronal markers such as neuron specific enolase and bet

263 on, we found that reprogrammed CMs expressed neuronal markers such as Tuj1, Map2, and NCAM.

264 At week 3 post-transduction, mature neuronal markers such as vGlut and synapsin were observe

265 BrdU labeling was associated with neuronal markers, such as Hu and betaIII tubulin.

266 Neuronal markers, such as neurofilament, have been detec

267 ranular layer was co-localized with an early neuronal marker, suggesting that caspase-mediated apopto

268 do proliferate after injury and can express neuronal markers, suggesting a latent neurogenic capacit

269 that circZNF827 represses expression of key neuronal markers, suggesting that this molecule negative

270 o dorsal interneurons possessing appropriate neuronal markers, synaptic proteins and functional neuro

271 ical analysis was performed to analyze three neuronal markers: synaptophysin, microtubule-associated

272 began to extend neurites, and expressed the neuronal markers tau, NeuN, neurofilament 200 kDa, and g

273 ochlea are improperly innervated and express neuronal markers that are not normally expressed in thes

274 a substantial minority express an excitatory neuronal marker, the vesicular glutamate transporter, ex

275 Although the cells aberrantly expressed neuronal markers, they retained their pigmented CE cell

276 ase marker bromodeoxyuridine (BrdU) and used neuronal markers to characterize new cells at different

277 The immature neuronal marker TUC-4 (TOAD/Ulip/CRMP-4) was used to def

278 to 4-fold increase in the expression of the neuronal markers Tuj1 and doublecortin.

279 As cells started to express the neuronal markers Tuj1 and tyrosine hydroxylase (TH) at E

280 yte-specific phenotype marker (MART-1) and a neuronal marker (Tuj1) revealed a subpopulation of melan

281 ic dividing cells, respectively, exhibited a neuronal marker (TuJ1).

282 thors measured N-acetylaspartate (a putative neuronal marker), using in vivo proton magnetic resonanc

283 age-specific markers such as TUBB3, an early neuronal marker; vWF, VEGFA, VEGFC and IL-8, endothelial

284 f neuron-specific enolase messenger RNA as a neuronal marker was also determined in these brain areas

285 s antibodies 2 days postviral injection: the neuronal marker was the transcription factor SOX1; early

286 owing that the number of cells expressing V2 neuronal markers was drastically diminished in gata2 nul

287 th antibodies against PKD1/PKD2 and PKD3 and neuronal markers, we found that PKDs were expressed in r

288 units, calcium-binding proteins, and general neuronal markers, we selectively labeled various OSN typ

289 In addition, some cells expressing an early neuronal marker were mis-localised at the ventricular su

290 Neuronal markers were also reduced up to 40%, and hippoc

291 pmental regulatory transcription factors and neuronal markers were analyzed in the alar hypothalamus

292 pmental regulatory transcription factors and neuronal markers were analyzed in the basal hypothalamus

293 pmental regulatory transcription factors and neuronal markers were analyzed in the hypothalamus of th

294 Neuronal markers were assessed by reverse-transcription

295 Subsequently, quail cells expressing neuronal markers were found in the chick ciliary ganglio

296 Cells colabeled for BrdU and five different neuronal markers were observed within the primary somato

297 -induced neurite outgrowth and expression of neuronal markers were reduced in this model.

298 udy, selective CB(1) receptor antibodies and neuronal markers were used to identify and characterise

299 arcoma family tumors (ESFTs or EFTs) express neuronal markers, which indicates they may originate fro

300 We used antibodies against the neuronal markers zn-12, acetylated tubulin, and human ne