ライフサイエンスコーパス: neuronal plasticity

1 onded to detoxification, probably reflecting neuronal plasticity.

2 of gene expression during activity-dependent neuronal plasticity.

3 dulating glutamatergic neurotransmission and neuronal plasticity.

4 possible point of intervention to facilitate neuronal plasticity.

5 brain regions with suspected neurogenesis or neuronal plasticity.

6 ases and phosphatases involved in modulating neuronal plasticity.

7 f Egr2/Krox20 further because of its role in neuronal plasticity.

8 ed salience of cues through learning-related neuronal plasticity.

9 one important mechanism maintaining lifelong neuronal plasticity.

10 neurotrophic factor (BDNF) as a mediator of neuronal plasticity.

11 prominent role of REM sleep in sleep-related neuronal plasticity.

12 ponse to an acute challenge, also reflecting neuronal plasticity.

13 ibits nicotinic receptor function and limits neuronal plasticity.

14 mutations in NPC1 alter Ca(2+) signaling and neuronal plasticity.

15 tical role in cortical neurons to facilitate neuronal plasticity.

16 , regulates many neural functions, including neuronal plasticity.

17 a strong link exists between sleep need and neuronal plasticity.

18 lar cascades associated with cocaine-induced neuronal plasticity.

19 ociated with brain-specific genes related to neuronal plasticity.

20 the brain that may underlie cocaine-induced neuronal plasticity.

21 in regulating AMPA receptor trafficking and neuronal plasticity.

22 nt motor learning model to study theories of neuronal plasticity.

23 ght the significance of small RNA control in neuronal plasticity.

24 rtments during activity is a key mediator of neuronal plasticity.

25 ent gene regulation controls many aspects of neuronal plasticity.

26 leus during transcription-dependent forms of neuronal plasticity.

27 nt stress may occur via mechanisms involving neuronal plasticity.

28 iced mRNA by microRNA in drug adaptation and neuronal plasticity.

29 are crucial for numerous processes including neuronal plasticity.

30 aumatic brain injury (TBI) is partly through neuronal plasticity.

31 in neurodegenerative disease, epilepsy, and neuronal plasticity.

32 regulate angiogenesis, axonal guidance, and neuronal plasticity.

33 e conjoint influence of these two factors on neuronal plasticity.

34 nel (Kcnd2) as cell type-specific markers of neuronal plasticity.

35 thway, a signaling cascade known to regulate neuronal plasticity.

36 -driven transcription programs that underlie neuronal plasticity.

37 of ion channels by phosphorylation underlies neuronal plasticity.

38 ould mediate tolerance to alcohol, a form of neuronal plasticity.

39 tiation, mitogenesis, tumor suppression, and neuronal plasticity.

40 good target for influencing neurogenesis and neuronal plasticity.

41 ynapse is thought to be a key determinant of neuronal plasticity.

42 r trafficking is important for many forms of neuronal plasticity.

43 eptor concentration, indicating some form of neuronal plasticity.

44 drites is thought to be underlying long-term neuronal plasticity.

45 in axon guidance, synaptic development, and neuronal plasticity.

46 ction and provides a model for understanding neuronal plasticity.

47 otoreceptor survival or in the regulation of neuronal plasticity.

48 hannels may play a role in calcium-dependent neuronal plasticity.

49 ggesting a role in RAS-mediated postsynaptic neuronal plasticity.

50 ffects of D(1) and D(2) receptor blockade on neuronal plasticity.

51 plays an important role in several forms of neuronal plasticity.

52 ions, including neuronal differentiation and neuronal plasticity.

53 this receptor expression is associated with neuronal plasticity.

54 ation in expression of genes associated with neuronal plasticity.

55 ressed in the CNS during events that require neuronal plasticity.

56 and activation that can explain its role in neuronal plasticity.

57 CREB-mediated transcription is important for neuronal plasticity.

58 een shown to play a role in certain forms of neuronal plasticity.

59 ty in the hippocampus that may help regulate neuronal plasticity.

60 elevation is closely associated with altered neuronal plasticity.

61 indeed interfere with functions critical for neuronal plasticity.

62 he circadian clock and processes involved in neuronal plasticity.

63 nses of MAPK signaling in activity-dependent neuronal plasticity.

64 and memory, amyloid protein processing, and neuronal plasticity.

65 ively regulating memory stabilization during neuronal plasticity.

66 critical roles in the mechanisms underlying neuronal plasticity.

67 gy field and will spread in areas related to neuronal plasticity.

68 proteins like PC4 play a significant role in neuronal plasticity.

69 rotrophic factor (BDNF) levels as a proxy of neuronal plasticity.

70 d a critical role in cognitive functions and neuronal plasticity.

71 p codon of mRNAs, including genes related to neuronal plasticity.

72 near the start of the axon that is a site of neuronal plasticity.

73 es for segregation between distinct modes of neuronal plasticity.

74 mechanisms of hormone-dependent behavior and neuronal plasticity.

75 f evidence suggest that CDKL5 is involved in neuronal plasticity.

76 ained responding, relapse-like behavior, and neuronal plasticity.

77 tinction of ethanol cues in association with neuronal plasticity.

78 versive state of withdrawal and BDNF-induced neuronal plasticity.

79 at have long term impacts on activity driven neuronal plasticity.

80 imal models, largely because of processes of neuronal plasticity.

81 g insight into signaling pathways underlying neuronal plasticity.

82 affolds, and signaling proteins required for neuronal plasticity.

83 hing an epigenetic landscape compatible with neuronal plasticity.

84 emporal sequence, induces different forms of neuronal plasticity.

85 ion of L-lactate as a signaling molecule for neuronal plasticity.

86 ptors is the pivotal event for initiation of neuronal plasticity.

87 d gene expression are critical for long-term neuronal plasticity.

88 yloid pathologic abnormalities and increased neuronal plasticity.

89 riatum is an important site for drug-induced neuronal plasticity.

90 and is expected to contribute to homeostatic neuronal plasticity.

91 ectrophysiological signatures of maladaptive neuronal plasticity; a phenomenon associated with LID.

92 Neuromodulation and neuronal plasticity act to reconfigure circuits on both

93 BC (ChABC) has striking effects on promoting neuronal plasticity after spinal cord injury (SCI), but

94 ir cerebral cortex nor indices of structural neuronal plasticity along extreme time-scales have been

95 Fear memories are acquired through neuronal plasticity, an orchestrated sequence of events

96 aviors by evaluating how the PFC affects LAT neuronal plasticity and activity that is evoked by previ

97 Thus, L1-induced variation could affect neuronal plasticity and behavior.

98 in the adult brain, a powerful mechanism for neuronal plasticity and brain repair, is altered by agin

99 tPA functions in the CNS in neuronal plasticity and cell death.

100 lays an important role in the development of neuronal plasticity and central sensitization.

101 dult neurons confirmed its essential role in neuronal plasticity and cognition.

102 synapses are essential for the induction of neuronal plasticity and cognitive processes in animals.

103 ich is normally constrained in its levels of neuronal plasticity and connectional change.

104 (tPA) and plasminogen has been implicated in neuronal plasticity and degeneration.

105 cell-intrinsic properties both contribute to neuronal plasticity and development.

106 l types, and how these changes contribute to neuronal plasticity and disease, is lacking.

107 DNA methylation, are essential regulators of neuronal plasticity and experience-driven behavioral cha

108 gger dendritic plateau potentials that drive neuronal plasticity and firing rate modulation.

109 spines are dynamic structures which regulate neuronal plasticity and have crucial roles in myriad bra

110 mpal gene expression in pathways involved in neuronal plasticity and increased levels of norepinephri

111 f cannabinoid-mediated short-term retrograde neuronal plasticity and is found in numerous brain regio

112 molecular memory and is indeed important in neuronal plasticity and learning/memory.

113 a support a direct role of Nurr1 in aberrant neuronal plasticity and LID induction, providing a poten

114 previously unknown form of steroid-mediated neuronal plasticity and may be an initial step in establ

115 cellular resilience as well as synaptic and neuronal plasticity and may influence mood and affective

116 ry roles for CRFRs in an AD-relevant form of neuronal plasticity and may link datasets documenting al

117 action of PrP(C) with laminin (Ln) modulates neuronal plasticity and memory formation.

118 logy in dendrites, a mechanism associated to neuronal plasticity and memory formation.

119 To investigate the role of this kinase in neuronal plasticity and memory, we generated transgenic

120 ntiation, which serves as a common model for neuronal plasticity and memory.

121 ion factor Mef2 regulates activity-dependent neuronal plasticity and morphology in mammals, and clock

122 of auditory brainstem circuits, to impaired neuronal plasticity and network hyperexcitability in the

123 alpha-synuclein has been implicated in both neuronal plasticity and neurodegenerative disease, but i

124 eding, the current study determined the mPFC neuronal plasticity and neuropeptide orexin signaling ar

125 m may be the result of underlying defects in neuronal plasticity and ongoing problems with synaptic s

126 ciated with odor stimulation on second order neuronal plasticity and phenotype in the olfactory syste

127 en activator (tPA), a protease implicated in neuronal plasticity and seizures, is induced in the limb

128 transcriptomes; and epigenetically modulates neuronal plasticity and stress-response signaling genes

129 gliosis, and provides beneficial effects for neuronal plasticity and survival and attenuation of micr

130 expression is critical for certain forms of neuronal plasticity and survival in the mammalian nervou

131 effects on signaling pathways that regulate neuronal plasticity and survival may be a factor in the

132 activity, neurotrophic factors implicated in neuronal plasticity and survival, and oxidative stress.

133 , GR and AKT/PI3K pathways, known to promote neuronal plasticity and survival, as well as reported to

134 spartate receptors (NMDARs) are critical for neuronal plasticity and survival, whereas their excessiv

135 c (NT) mice on signaling factors involved in neuronal plasticity and survival.

136 nd that this mechanism is likely to regulate neuronal plasticity and survival.SIGNIFICANCE STATEMENT

137 actions of BDNF and glucocorticoids promote neuronal plasticity and that disruption in either pathwa

138 that exposure to LAN influences behavior and neuronal plasticity and that this effect is likely media

139 s only weakly inhibited by GABA and shows no neuronal plasticity and the other subgroup is strongly i

140 ne knock-out models is always complicated by neuronal plasticity and the potential for restructuring

141 tions paired with rehabilitation can enhance neuronal plasticity and thereby improve functional recov

142 in syntaxin-1A causes abnormal regulation of neuronal plasticity and vesicle recycling and that the a

143 Impaired neuronal plasticity and, specifically, altered expressio

144 oglia influence postnatal development, adult neuronal plasticity, and circuit function.

145 ement therapy, protein-protein interactions, neuronal plasticity, and embryonic development.

146 naling pathways linked to synaptic activity, neuronal plasticity, and epileptogenesis.

147 odegenerative diseases are closely linked to neuronal plasticity, and induce changes in adult neuroge

148 ession of genes important for cell survival, neuronal plasticity, and neuronal remodeling.

149 ses which regulate synaptic transmission and neuronal plasticity, and possibly underlie memory format

150 thological processes, including development, neuronal plasticity, and tumor metastasis.

151 that the molecules involved in regulation of neuronal plasticity are also involved in the mode of act

152 Most forms of neuronal plasticity are associated with induction of the

153 in the brain as a regulator of synaptic and neuronal plasticity as well as its etiological role in t

154 h receptors (nAChRs) have been implicated in neuronal plasticity as well as neurodevelopmental, neuro

155 In addition, PFC stimulation blocked LAT neuronal plasticity associated with an affective conditi

156 Neurogenesis, neurite outgrowth, and neuronal plasticity associated with BDNF, glutamate, and

157 ent brain, these molecules are implicated in neuronal plasticity associated with learning and memory.

158 ene expression are thought to be involved in neuronal plasticity associated with learning and memory.

159 mmatory indices, glutamatergic function, and neuronal plasticity at the cellular and circuitry level

160 to fear-associated responses by facilitating neuronal plasticity at the neuron-matrix interface.

161 and proregenerative modulators of poststroke neuronal plasticity at various structural levels.

162 keleton is critical for neurodevelopment and neuronal plasticity, but how neurons spatially control a

163 need is affected by developmental stage and neuronal plasticity, but the underlying mechanisms remai

164 Estrogens are known to have broad effects on neuronal plasticity, but their specific role in neuronal

165 Moreover, isoflurane affected neuronal plasticity by facilitating long-term potentiati

166 dation protein (FMRP) is thought to regulate neuronal plasticity by limiting dendritic protein synthe

167 re we show that this potential mechanism for neuronal plasticity can also be exploited in a rapid and

168 ar calcium signaling, energy metabolism, and neuronal plasticity can be influenced by inducing axonal

169 Such experience-dependent neuronal plasticity can be long-lasting and is thought t

170 ks for motor function.SIGNIFICANCE STATEMENT Neuronal plasticity can facilitate recovery of function

171 This new form of hippocampal neuronal plasticity could be a cellular correlate of lea

172 Neuronal plasticity deficits underlie many of the neurob

173 Neuronal plasticity depends on plasma membrane Ca(2+) in

174 ological and pathological processes, such as neuronal plasticity, development, and viral invasion.

175 n essential substrate for the behavioral and neuronal plasticity driven by reward-related experiences

176 important implications for the regulation of neuronal plasticity during neuronal development and in r

177 nscription serves as a functional marker for neuronal plasticity during withdrawal.

178 d postsynaptic signaling pathways related to neuronal plasticity (e.g., dendritic spine formation).

179 to neocortical development promoting greater neuronal plasticity early in postnatal life preceded the

180 acy of phosphodiesterase (PDE) inhibitors as neuronal plasticity enhancers, little is known about the

181 Here, we report studies of AOB neuronal plasticity following male-male social chemosens

182 tor with implications in neuroprotection and neuronal plasticity following NMDA receptor activation.

183 imal and after a 1-week delay, and implicate neuronal plasticity from unlesioned areas of the central

184 scade, a major signaling pathway involved in neuronal plasticity, function and survival.

185 s that is essential for the transcription of neuronal plasticity genes.

186 pared to the developing visual system, where neuronal plasticity has been well characterized at multi

187 Neuronal plasticity helps animals learn from their envir

188 n is an essential component of many forms of neuronal plasticity, however, the intracellular mechanis

189 gy in maintaining neuronal stem cells and in neuronal plasticity in adult life and we discuss how the

190 at RIN1 serves as an inhibitory modulator of neuronal plasticity in aversive memory formation.

191 ors may coordinate structural and functional neuronal plasticity in CNS health and disease.

192 coding DNA, spatial genome architecture, and neuronal plasticity in development and disease.

193 LIF also induced neuronal plasticity in dorsal root ganglia neurons by in

194 iverse Ca2+ domains that may be required for neuronal plasticity in Hermissenda.

195 re provide further evidence for synaptic and neuronal plasticity in ipsi-lesional MVN neurons during

196 tivation of PAF-r signaling induces aberrant neuronal plasticity in LE and leads to chronic dysfuncti

197 g model for understanding activity-dependent neuronal plasticity in mammals.

198 contribute to the deficits of CREB-dependent neuronal plasticity in neurodegenerative diseases.

199 neuronal correlates underlying tDCS-induced neuronal plasticity in older adults and thus might help

200 describe a recently identified mechanism of neuronal plasticity in primary afferent nociceptive nerv

201 The ability to induce and study neuronal plasticity in single dendritic spines has great

202 ent for I(Kv) regulation and a novel form of neuronal plasticity in spinal cord neurons.

203 t neurogenesis and we suggest high levels of neuronal plasticity in tanycytes, SCN, and ICj.

204 e regulation of neuronal gene expression and neuronal plasticity in the absence of dopamine.

205 These findings show that BDNF-dependent neuronal plasticity in the ACC, a structure known to be

206 d inhibition as a regulator of circuit-level neuronal plasticity in the adult retina.

207 on have been shown to mediate novel forms of neuronal plasticity in the axon.

208 ized by a high level of experience-dependent neuronal plasticity in the central nervous system and se

209 , neuronal coupling represents a new form of neuronal plasticity in the DRG and contributes to pain h

210 Numerous studies have shown that neuronal plasticity in the hippocampus and neocortex is

211 The findings suggest that cross-modal neuronal plasticity in the PN is driven by excitatory fe

212 Overall, these results show seasonal neuronal plasticity in the song-control system closely a

213 drugs of abuse and/or its role in long-term neuronal plasticity in the striatum may provide a new ro

214 Reduced neuronal plasticity in the striatum, hippocampus, and ne

215 ral months of life are a critical period for neuronal plasticity in the visual cortex during which an

216 city (ODP), a paradigm of activity-dependent neuronal plasticity in the visual cortex.

217 opiate administration activates BDNF-related neuronal plasticity in the VTA that is necessary for bot

218 develop a normal baseline by which to assess neuronal plasticity in this corticostriatal system in ra

219 Techniques to induce activity-dependent neuronal plasticity in vivo allow the underlying signali

220 e induction and maintenance of some forms of neuronal plasticity, in hippocampus-dependent and hippoc

221 icits several forms of translation-dependent neuronal plasticity including epileptogenesis.

222 ivate several kinases that are important for neuronal plasticity, including Ca2+/calmodulin-dependent

223 lutamate receptors triggers various forms of neuronal plasticity, including cerebellar long-term depr

224 iew of the critical role of CREB and BDNF in neuronal plasticity, including learning and memory, the

225 (CaMKII) are required for numerous forms of neuronal plasticity, including long-term potentiation (L

226 receptor TrkB are crucial for many forms of neuronal plasticity, including structural long-term pote

227 ied numerous Mef2 target genes implicated in neuronal plasticity, including the cell-adhesion gene Fa

228 Neuronal plasticity induced by behavioral experience, as

229 oral responses to swim stress as well as the neuronal plasticity involved in long-term behavioral cha

230 ent with the hypothesis that PbTx-2-enhanced neuronal plasticity involves NMDAR-dependent signaling.

231 be important forms of short-term retrograde neuronal plasticity involving endocannabinoids (eCB) and

232 and inhibition (DSI) are forms of short-term neuronal plasticity involving postsynaptic release of an

233 ls were also observed in an in vivo model of neuronal plasticity involving Zif268 induction: the effe

234 Activity-dependent neuronal plasticity is a fundamental mechanism through w

235 Neuronal plasticity is an important feature of the devel

236 tifying the mechanisms of activity-dependent neuronal plasticity is crucial to finding therapeutic in

237 presynaptic release machinery contribute to neuronal plasticity is less clear.

238 In cats, electrophysiologically assessed neuronal plasticity is minimal until approximately 3 wee

239 Activity-induced neuronal plasticity is partly facilitated by the express

240 P).SIGNIFICANCE STATEMENT Activity-dependent neuronal plasticity is the cellular basis for learning a

241 n, an activity-dependent gene that regulates neuronal plasticity, is decreased by chronic unpredictab

242 cits, and the detection of memory underlying neuronal plasticity, is greatly impeded by a lack of pre

243 nin, an evolutionarily conserved mediator of neuronal plasticity, is responsible for this behavioral

244 mechanisms that underlie the stress-induced neuronal plasticity leading to fear and anxiety.

245 ain region-specific changes in signaling and neuronal plasticity may be critical components in develo

246 tory and/or inhibitory neurotransmission and neuronal plasticity may lead to aberrant functional conn

247 eurite extension and branching are important neuronal plasticity mechanisms that can lead to the addi

248 ensory inputs rather than the development of neuronal plasticity mechanisms.

249 st also provide insights into the underlying neuronal plasticity mechanisms.

250 eport that effects of calorie restriction on neuronal plasticity, memory and social behavior are abol

251 (tPA), an extracellular protease involved in neuronal plasticity, modulates the biochemical and behav

252 Aergic and cholinergic systems influence the neuronal plasticity necessary for repetition priming.

253 findings reveal a critical role for CRMP2 in neuronal plasticity, neural function and behavioural mod

254 the behaviors and related cellular signaling/neuronal plasticity/neuroendocrine alterations in the de

255 function in a cooperative manner to regulate neuronal plasticity, neurogenesis, and neuronal survival

256 hese data suggest that exercise may increase neuronal plasticity not only in controls, but also in su

257 ynaptic transmission, causing impairments of neuronal plasticity observed in Fmr1 knock-out mice and

258 humans and rodents suggest that significant neuronal plasticity occurs in areas surrounding the cort

259 Neuronal plasticity of the inner retina has been observe

260 of two latitudes would have an impact on the neuronal plasticity of the song-control system in songbi

261 protein phosphorylation, a critical step in neuronal plasticity, on the level and duration of membra

262 RSK has not been performed in the context of neuronal plasticity or behavior.

263 ons in neuroscience, such as those regarding neuronal plasticity or learning and memory.

264 2+)](i) is directly correlated with impaired neuronal plasticity; or (3) the previously observed age-

265 ption regulatory factors are induced in many neuronal plasticity paradigms, it is still unclear what

266 hese receptors may play an important role in neuronal plasticity related to emotional learning.

267 d signalling pathways in immune response and neuronal plasticity related to radicular/neuropathic pai

268 Neuronal plasticity relies on tightly regulated control

269 e impact of spontaneous neurotransmission on neuronal plasticity remains poorly understood.

270 SRF in astrocytes may reduce the deficits in neuronal plasticity seen in models of FASD.

271 l, which is accompanied by subtle changes in neuronal plasticity, sensory learning and memory.

272 Such lesion-induced neuronal plasticity sheds new light on potential mechani

273 morphogenetic events, such as axon guidance, neuronal plasticity, spine maturation, and synaptogenesi

274 Using in vitro models of neuronal plasticity, such as glycine-induced chemical lo

275 the pathophysiology of schizophrenia and in neuronal plasticity suggests that facilitation of NMDAR

276 aling cascades, which play critical roles in neuronal plasticity, survival, and neurite outgrowth.

277 are poorly understood, but regulators of the neuronal plasticity that accompany learning, such as neu

278 This recovery may represent a form of neuronal plasticity that compensates for the prolonged s

279 ther, these findings identify novel forms of neuronal plasticity that contribute to granule cell dysm

280 Neuronal plasticity that develops in the cortex during l

281 KII) is thought to be a critical mediator of neuronal plasticity that links transiently triggered Ca(

282 in the ability of cocaine to induce forms of neuronal plasticity that may contribute to addiction.

283 ate excitation may represent a novel form of neuronal plasticity that regulates activity and excitabi

284 This indicates a compensatory neuronal plasticity that we now show to involve the nigr

285 13), as a candidate gene for critical-period neuronal plasticity, the expression of which is regulate

286 an astrocyte-derived protease can influence neuronal plasticity through an extracellular matrix inde

287 t a novel pharmacologic strategy to regulate neuronal plasticity through an NMDA receptor and Src fam

288 present a pharmacologic strategy to regulate neuronal plasticity through NMDAR-dependent mechanisms.

289 ling genes, including S100a10 (p11), linking neuronal plasticity to the antidepressant response.

290 also depend on a slow genomic regulation of neuronal plasticity underlying behavioral activation and

291 pamine and glutamate systems involved in the neuronal plasticity underlying cocaine craving and relap

292 c) is an immediate early gene that modulates neuronal plasticity underlying learning and memory.

293 This behaviorally induced expression of neuronal plasticity uses an NMDAR-dependent, LTP-like me

294 induced, experience-dependent expression of neuronal plasticity was blocked by the NMDA(R) antagonis

295 To identify novel miRs involved in neuronal plasticity, we exposed adult mice to chronic tr

296 ts also plays a postnatal role in regulating neuronal plasticity, we modulated BMP signaling in mice

297 II isoforms have markedly different roles in neuronal plasticity, with CaMKIIalpha regulating synapti

298 in rodents and also the capacity to modulate neuronal plasticity within the medial nucleus of the amy

299 Repeated psychostimulant exposure induces neuronal plasticity within the mesolimbic dopamine syste

300 and an immediate early gene associated with neuronal plasticity, zif268, in groups of rats selected