ライフサイエンスコーパス: neurone

1 ting dorsal motor nucleus of the vagus (DMV) neurones.

2 the chick embryo including developing motor neurones.

3 ent regulation of NMDARs in SNc dopaminergic neurones.

4 prolonged bursts of action potentials in VP neurones.

5 gnalling in primary cultured rat hippocampal neurones.

6 n at least 50% of dorsal root ganglion (DRG) neurones.

7 ith or without selective ablation of RVLM-C1 neurones.

8 -SAP) was used to selectively lesion RVLM-C1 neurones.

9 nm) augmented Glu- and CCh-evoked signals in neurones.

10 otor and sensory function and nodose ganglia neurones.

11 rpin polymers in both HeLa cells and primary neurones.

12 rat dorsal motor nucleus of the vagus (DMV) neurones.

13 t of GABAB activation on ITD encoding in MSO neurones.

14 contribute to the lower somatic R(in) of DHC neurones.

15 es are intrinsically more excitable than DHC neurones.

16 in); 46 M measured from RMP) observed in VHC neurones.

17 of vasopressin from paraventricular nucleus neurones.

18 glutamine, which is detected by nearby MNTB neurones.

19 s but had no effect on the responses in Sp5l neurones.

20 (V(m)) correlated with the [ATP](i) in Hcrt neurones.

21 esulted in an inhibition of activity in Hcrt neurones.

22 receptor (NK(1)R) in endosomes of myenteric neurones.

23 l efferent connections made by inhibitory SG neurones.

24 ats that were abolished by lesion of RVLM-C1 neurones.

25 chronized in onset in pairs of magnocellular neurones.

26 ibution of these receptors in CeA projection neurones.

27 between pairs of postsynaptic magnocellular neurones.

28 g was observed in a subset of Kv3.3-positive neurones.

29 of a subpopulation of gastric-projecting DMV neurones.

30 creased action potential duration in IB4 ve, neurones.

31 onged depression of the ADP in CA3 pyramidal neurones.

32 ivation on action currents in IB4 +ve and ve neurones.

33 their accepted role in cardiac myocytes and neurones.

34 he excitability of medial septal cholinergic neurones.

35 of 5-HT3 receptors on gastric vagal afferent neurones.

36 dulation of synaptic transmission in central neurones.

37 HF rats was abolished by ablation of RVLM-C1 neurones.

38 nes while Oct-2 is restricted to B-cells and neurones.

39 ulated inhibitory drive to the magnocellular neurones.

40 use Ca(2+)-dependent run-down in hippocampal neurones.

41 trophysiological properties of CA1 pyramidal neurones across the longitudinal hippocampal axis, and s

42 Two alpha subunits are present in adult neurones, alpha1, which forms most of the synaptic glyci

43 rks, here we ask if a postnatal loss of 5-HT neurones also compromises autoresuscitation.

44 uclear palsy (PSP) where nigral dopaminergic neurones also degenerate.

45 isolated rat vagal bronchopulmonary sensory neurones and also in the cough response to SO(2) inhalat

46 The neurones and astrocytes express Na(+) pumps with a high-

47 Neurones and astrocytes were identified by immunocytoche

48 he high-ouabain-affinity Na(+) pumps in both neurones and astrocytes.

49 y of gastric-projecting dorsal motor nucleus neurones and dysregulates neurotransmitter release from

50 thermal hypersensitivity of rat dorsal horn neurones and enhanced perceptual responses of human subj

51 macological responsiveness of vagal afferent neurones and fibres, although the effects of DIO on cent

52 GABAergic neurotransmission in developing VB neurones and govern a transition from slow to fast phasi

53 ession, in both Drosophila olfactory sensory neurones and in human embryonic kidney cells, together w

54 ptic transmission to pancreas-projecting DMV neurones and increase PES.

55 clear cell cultures promoted survival of rat neurones and increased axonal length in vitro, effects t

56 augmented evoked activity of rat dorsal horn neurones and increased perceptual responses of human sub

57 e expression of CB2 receptor protein by both neurones and microglia in the spinal cord was significan

58 dings from identified gastric-projecting DMV neurones and microinjection in the dorsal vagal complex

59 They communicate with neurones and other glial cells through the release of si

60 and thermally-evoked activity of rat spinal neurones and quantitative sensory testing to assess huma

61 A high density of GLP-1 containing neurones and receptors are present in brainstem vagal ci

62 ecific subtype of mainly cholinergic enteric neurones and regulates sprouting and sensitivity of noci

63 ensitivity to ATX-II might also discriminate neurones and report that 1 microm has negligible or smal

64 siologically realistic (Hodgkin-Huxley type) neurones and synapses.

65 trical excitability of medullary respiratory neurones and their central modulation of sympatho-excita

66 fferent roles in the regulation of OT and VP neurones and their distinct patterns of physiological ac

67 bout the bioelectric properties of mouse MHb neurones and their potential circadian characteristics.

68 th an excitatory function of GABA in only VP neurones and with the generation of prolonged bursts of

69 have a diffuse action (which can affect many neurones) and, unlike classical neurotransmitters, have

70 the differentiation and survival of sensory neurones, and is phosphorylated in neuroblastoma cells f

71 reliably target more specific populations of neurones, and open up the internal circuitry of the moto

72 (CCh) evoked rapid Ca(2+) transients only in neurones, and small, delayed transients in some astrocyt

73 e action potentials in VHC neurones than DHC neurones, and that this difference stems from the more d

74 diac atrium, gastric antrum/pylorus, enteric neurones, and vagal and dorsal root ganglia.

75 oss of 5-hydroxytryptamine (5-HT, serotonin) neurones are compromised in their ability to withstand e

76 eas a substantial proportion of intrinsic SG neurones are GABAergic inhibitory, their relationship to

77 renergic and gamma-amino-butyric acid (GABA) neurones are implicated in the system's regulation, and

78 dinal hippocampal axis, and suggest that VHC neurones are intrinsically more excitable than DHC neuro

79 GABAergic and glycinergic neurones are known components of these circuits but thei

80 c mouse in which certain GABAergic lamina II neurones are labelled with green fluorescent protein (GF

81 ocesses that regulate electrical activity in neurones are now an established aspect of physiology and

82 both vasopressin and oxytocin magnocellular neurones are osmoresponsive, and their increased spiking

83 ally, changes in the intrinsic properties of neurones are reversible following removal of the depolar

84 al excitability of medullary pre-sympathetic neurones are the main causal mechanism for the developme

85 c expression of fluorescent protein in motor neurones as a morphological reporter.

86 Since glutamine can be used by neurones as a precursor for glutamate and GABA synthesis

87 al recordings from gastric-projecting nodose neurones assessed the ability of glucose to modulate the

88 a(2+) signalling (fura-2) in rat hippocampal neurone-astrocyte co-cultures.

89 ral variants of green fluorescent protein in neurones, axons and motor nerve terminals, including the

90 IB4 +ve neurones became more excitable with depolarization over

91 O(2) after postnatal day 12 (P12), when 5-HT neurones become chemosensitive in vitro.

92 ntaneous activity of suprachiasmatic nucleus neurones but does not block the harmonic discharge patte

93 -dependent increase in spike frequency in VP neurones, but not in OT neurones,consistent with an exci

94 me, regulation of NMDARs in SNc dopaminergic neurones by changes in intracellular Ca(2+) at both syna

95 affects the properties of central autonomic neurones by decreasing the membrane excitability and pha

96 excitatory input to pancreas-projecting DMV neurones by decreasing the response of excitatory synapt

97 CN, prokineticin 2 (PK2), inhibited some LHb neurones by elevating the frequency of GABA release in t

98 acellular Ca(2+) homeostasis in dopaminergic neurones by limiting Ca(2+) influx through the NMDAR.

99 We propose a novel hypothesis of respiratory neurone channelopathy induced by carotid body overactivi

100 These vasopressin neurones co-express vasopressin V1 receptors.

101 pike frequency in VP neurones, but not in OT neurones,consistent with an excitatory function of GABA

102 SCN neurones contain an intracellular molecular clock that d

103 Intriguingly, some LHb neurones contain the molecular circadian clock, but it i

104 nterpolaris (Sp5I) nucleus and cardiac vagal neurones (CVNs) in the Nucleus Ambiguus (NA).

105 verexpression of HSPB8 in immortalized motor neurones decreased the accumulation of TDP-25 and TDP-35

106 ary ALS Network Identification to Cure Motor Neurone Degeneration (Euro-MOTOR)) of data from 3 Europe

107 gs show that synergistic action of glia- and neurone-derived ATP can pre-modulate efficacy of excitat

108 In 2003 the Motor Neurone Disease (MND) Association, together with The Wel

109 In motor neurone disease (MND), respiratory muscle weakness cause

110 e for neurological disorders including motor neurone disease and Parkinsons disease in addition to va

111 University, and Research in Italy; the Motor Neurone Disease Association of England, Wales, and North

112 ted that over expression of FUS causes motor neurone disease in mouse models hence mutations leading

113 ecruited through the national Scottish Motor Neurone Disease Register and were asked to complete the

114 th Amyotrophic Lateral Sclerosis (ALS, motor neurone disease) (sporadic and familial) and Parkinson's

115 ggered averaging from expiratory bulbospinal neurones (EBSNs), with a view to revealing specific conn

116 in reporter gene is expressed in vasopressin neurones (eGFP-vasopressin), we have discovered a popula

117 Here we examined the effect of 5-HT neurone elimination or 5-HT reduction on seizure risk an

118 ous whole-cell recordings from characterized neurones establish that inhibitory SG neurones receive m

119 and responsiveness of gastric-projecting DMV neurones, even in the absence of obesity, suggesting tha

120 ction potentials; (ii) the proportion of DMV neurones excited by cholecystokinin (CCK) was unaltered

121 Furthermore, the number of DMV neurones excited by thyrotrophin-releasing hormone and t

122 h whole-cell patch-clamp recordings that LHb neurones exhibit heterogeneity in their resting state, b

123 Following exposure to KCl, neurones exhibit lower input resistances and resting pot

124 First, we determined whether striatal neurones exhibit pharmacologically induced nicotinic res

125 All burster neurones exhibited an inspiratory discharge during eupno

126 tion over the range 100 to 20 mV, but IB4 ve neurones exhibited peak excitability near 55 mV, and wer

127 MNTB principal neurones express electrogenic system A glutamine transpo

128 ur previous data suggested that dopaminergic neurones expressed triheteromeric GluN1-GluN2B-GluN2D NM

129 However, in recordings of MHb neurones from mice lacking the core molecular circadian

130 Here, using NMDARs in dopaminergic neurones from postnatal day 7 (P7) rats as a model syste

131 The DMV neurones from rats exposed to high-fat diet for 12-14 we

132 trophysiological properties of CA1 pyramidal neurones from the DHC and the VHC using the whole-cell c

133 ed, the intrinsic excitability of individual neurones from the DHC and VHC has received surprisingly

134 ysiologically identified gastric vagal motor neurones (gastric-DMN) involved in the gastric accommoda

135 strate that following perinatal HFD: (i) DMV neurones had decreased excitability and input resistance

136 Moreover, we demonstrate that mouse LHb neurones have access to and can respond to visual input,

137 ferent fibres, however, as other non-sensory neurones have displayed sensitivity to capsaicin and bra

138 Patch clamp recordings from mouse DRG neurones identified a trend for larger proton-gated curr

139 1A) and 5-HT(1B) receptors in CeA projection neurones identified by microinjection of a retrograde tr

140 ve probably more mitochondria than any other neurone in the CNS.

141 mulation was observed in abnormal dysmorphic neurones in 6 cases, but not in seven FCD type IIB and 7

142 elease of vasopressin (VP) from hypothalamic neurones in brain slices.

143 ge clamping astrocytes and neighbouring MNTB neurones in brainstem slices, we show that application o

144 terocytes through P2Y(2) and nodose ganglion neurones in co-cultures through P2X(2/3)-receptors.

145 lpha immunoreactivity was low in TH positive neurones in control tissue and quantification was not po

146 Gap junctions between inhibitory neurones in cortical regions have been well documented o

147 sms can influence the membrane properties of neurones in extra-SCN sites.

148 nd regionally confined subsets of inhibitory neurones in key respiratory circuits such as those in th

149 th vagal-afferent endings and some myenteric neurones in mouse and human stomach, respectively.

150 could enable the activation of magnocellular neurones in response to acute challenges.

151 e V(m) was significantly depolarized in Hcrt neurones in sleep-deprived mice as compared with control

152 These data suggest that vasopressin neurones in the AON may be selectively involved in the c

153 have discovered a population of vasopressin neurones in the AON.

154 Neurones in the central nucleus of the amygdala (CeA) pr

155 y in the face of continually changing input, neurones in the CNS must dynamically modulate their elec

156 t Lmx1b(f/f/p) mice, which lack >99% of 5-HT neurones in the CNS, and littermate controls (Lmx1b(f/f)

157 Given the prenatal role of 5-HT neurones in the development of neural networks, here we

158 Here, we test the hypothesis that neurones in the inferior olive actively integrate glutam

159 Glutamatergic inputs to neurones in the inferior olive generate bidirectional po

160 Our results indicate that neurones in the inferior olive implement novel rules for

161 motor coordination, the mechanisms by which neurones in the inferior olive integrate synaptic inputs

162 ITDs are computed by coincidence-detecting neurones in the medial superior olive (MSO) in mammals.

163 inhibited noxious-evoked responses of spinal neurones in the model of OA pain, but not in control rat

164 ursts of IPSCs in hypothalamic magnocellular neurones in the presence of TTX, which implicated a coor

165 Neurones in the principal olivary nucleus receive monosy

166 Magnocellular vasopressin and oxytocin neurones in the rat hypothalamus project to the posterio

167 Light-responsive neurones in the rat suprachiasmatic nucleus discharge wi

168 oteasome system (UPS) occurs in dopaminergic neurones in the SN in PD and it is associated with Lewy

169 ntanyl on the synaptic TCR responses in both neurones in the spinal trigeminal interpolaris (Sp5I) nu

170 are often thought to activate all classes of neurones in the stimulated area.

171 rdings were made from gastric-projecting DMV neurones in thin brainstem slices from rats that were ex

172 rat dorsal motor nucleus of the vagus (DMV) neurones in thin brainstem slices.

173 rrent in both control and HFD gastric nodose neurones in vitro, the 5-HT response and receptor distri

174 one can modulate sensory responses of single neurones in vivo.

175 in (CCK) was unaltered but the proportion of neurones in which CCK increased excitatory glutamatergic

176 (RVLM), a major source of sympathoexcitatory neurones, increases ABP and sympathetic nerve activity.

177 affected the morphological properties of DMV neurones, increasing their size and dendritic arborizati

178 y at levels L4 and 5 (levels predominated by neurones innervating the hindpaw) rather than L3.

179 h is typically active in parallel with motor neurone input during muscular activity.

180 a trend for larger proton-gated currents in neurones lacking STOML1, which was due to a contribution

181 ractility is provided by vagal preganglionic neurones located in the dorsal motor nucleus (DVMN).

182 activates selective receptors to excite DMV neurones mainly and that the gastroinhibition observed f

183 Corticogeniculate neurones make more synapses in the lateral geniculate nu

184 edial medulla suggests that these projection neurones may act differentially in controlling autonomic

185 reas toxin sensitivity indicates that IB4 ve neurones may express Na(V)1.1 or Na(V)1.2, or both.

186 The population of burster neurones may overlap with that previously described in t

187 It is still not clear how Hcrt neurones monitor changes in energy status in animals.

188 ence the activity of medium spiny projecting neurones (MSNs) and striatal output via a disynaptic mec

189 MHb neurones (n = 230) showed heterogeneity in electrophysio

190 whole-cell voltage-clamp recordings from VB neurones of mouse thalamic slices revealed that early in

191 whole-cell voltage-clamp recordings from VB neurones of mouse thalamic slices revealed that early in

192 mRNA and protein were expressed by myenteric neurones of rat and mouse intestine.

193 ractility is provided by vagal preganglionic neurones of the dorsal motor nucleus (DVMN).

194 he response of identified gastric-projecting neurones of the dorsal motor nucleus of the vagus (DMV)

195 gic synaptic inputs (STP(GABA)) to VP and OT neurones of the hypothalamic supraoptic nucleus elicited

196 However, in thalamocortical neurones of the mouse ventrobasal (VB) thalamus, the maj

197 However, in thalamocortical neurones of the mouse ventrobasal (VB) thalamus, the maj

198 ely affording protection to the dopaminergic neurones of the nigro-striatal pathway alone.

199 43 and is upregulated in the surviving motor neurones of transgenic ALS mice and human patients.

200 stral ventrolateral medulla (RVLM) (PVN-RVLM neurones) of rats.

201 Abnormal dysmorphic neurones on proteomics analysis were comparable to aged

202 When many NO-emitting neurones or synapses are active simultaneously in a tiss

203 although the effects of DIO on central vagal neurones or vagal efferent functions have never been inv

204 ore, in isolated rat vagal pulmonary sensory neurones, perfusion of an aqueous solution of SO(2) evok

205 t neonatal mice lacking 60-70% of their 5-HT neurones (Pet-1(-/-)) would have: (1) a reduced thermoge

206 Our findings reveal that RVLM-C1 neurones play a major role in irregular breathing patter

207 Our findings suggest that RVLM-C1 neurones play a pivotal role in breathing irregularities

208 vious studies showed that cortical pyramidal neurones (PNs) have a dynamic spike threshold that funct

209 The retrogradely-labelled neurones positive for 5-HT(1B) receptor were present in

210 in order to retain stable network function, neurones possess homeostatic mechanisms which integrate

211 In these neurones, PRiMA also co-localizes with AChE immunoreacti

212 ated dorsal motor nucleus of the vagus (DMV) neurones, prior to the development of obesity.

213 Given that PVN neurones project to brainstem cardio-respiratory regions

214 rential expression of these receptors in CeA neurones projecting to the caudal dorsomedial medulla su

215 These neurones provide functionally significant parasympatheti

216 Activity of a subpopulation of DVMN neurones provides functionally significant parasympathet

217 We conclude that 5-HT neurones raise seizure threshold and decrease seizure-re

218 erized neurones establish that inhibitory SG neurones receive monosynaptic input from a subset of unm

219 Using single-neurone recording in the rat VB in vivo with local appli

220 Partial lesion (~65%) of RVLM-C1 neurones reduces AHI, respiratory variability, and respi

221 All neurones responsive to L-AP4 were also responsive to APD

222 Reduction (~65%) of RVLM-C1 neurones resulted in attenuation of irregular breathing,

223 termined that a 60-70% loss of 5-HT-positive neurones results in a ~90% loss of 5-HT from the brainst

224 e proportion (approximately 60%) of amygdala neurones retrogradely-labelled with CTb expressed 5-HT(1

225 morphological reconstructions of individual neurones revealed significant differences in the dendrit

226 tral ventrolateral medulla catecholaminergic neurones (RVLM-C1) modulate sympathetic outflow and brea

227 tral ventrolateral medulla catecholaminergic neurones (RVLM-C1) to both haemodynamic and respiratory

228 ein modulation in superior cervical ganglion neurones (SCGNs).

229 f interspike intervals, whereas unresponsive neurones seldom do.

230 Breeding Ai38 mice with various neurone selective, promoter-driven Cre recombinase mice

231 provides new evidence in rat pups that 5-HT neurones serve a physiological role in autoresuscitation

232 rrents in identified pancreas-projecting DMV neurones showed a reduced functional response in AP rats

233 Cultured nodose ganglion neurones showed no changes in response to capsaicin, cho

234 When considered as one population, MHb neurones showed significant circadian variation in their

235 MSO neurones showed strong selectivity for bilateral delays.

236 ous studies in adult animals, devoid of 5-HT neurones, showing altered thermoregulation in cold stres

237 ctrode recordings in vivo, we found that LHb neurones sluggishly respond to retinal illumination, sug

238 Patch-clamp recordings from spiny projection neurones (SPNs) and various interneurone populations dem

239 Sympathetic preganglionic neurones (SPNs) convey sympathetic activity flowing from

240 Sensory neurone subtypes (< or = 25 microm apparent diameter) ex

241 signalling in Tmem67(-/-) mutant cerebellar neurones, suggesting that increased canonical Wnt signal

242 In particular, in certain developing neurones synaptic alpha2-GABAA Rs are replaced by alpha1

243 In particular, in certain developing neurones synaptic alpha2-GABAARs are replaced by alpha1-

244 ed with urethane was also observed when DVMN neurones targeted bilaterally to express an inhibitory D

245 DMV neurones were not affected by OXT in any neurones tested, unless the brainstem slice was (a) pret

246 ed GABAergic transmission in the majority of neurones tested.

247 mEPSCs, respectively) in the majority of the neurones tested.

248 significantly more action potentials in VHC neurones than DHC neurones, and that this difference ste

249 Our hCPG comprises two neurones that antagonise each other to provide rhythmic

250 between excitatory sensory afferents and VB neurones that can lead to a reduction in sensory-evoked

251 A quantitative model of oxytocin neurones that combines a spiking model, a model of stimu

252 dritic branching pattern between DHC and VHC neurones that could, in principle, contribute to the low

253 howed also the novelty responses of dopamine neurones that decreased as learning advanced.

254 al mapping revealed that vagal preganglionic neurones that have an impact on left ventricular contrac

255 xcitability of paraventricular nucleus (PVN) neurones that project directly to the rostral ventrolate

256 itter content of a population of respiratory neurones that retains a bursting behaviour after ionotro

257 ised low-amplitude membrane oscillations, to neurones that were moderately hyperpolarised ( approxima

258 ay little role in generating SFA in PVN-RVLM neurones, their activation nevertheless does dampen exci

259 esponse of gastric vagal afferent nerves and neurones to 5-HT but attenuates the ability of glucose t

260 ers within the endoplasmic reticulum (ER) of neurones to cause the autosomal dominant dementia famili

261 iples for the contribution of inferior olive neurones to coordinated motor behaviours.

262 erin, on the evoked responses of dorsal horn neurones to electrical, mechanical and thermal stimulati

263 ng cultures of primary hippocampal pyramidal neurones to elevated concentrations (10-30 mm) of KCl.

264 racellular molecular clock that drives these neurones to exhibit pronounced day-night differences in

265 elation in the evoked activity of rat spinal neurones to human thermal pain thresholds.

266 ddressed a potential contribution of RVLM-C1 neurones to irregular breathing in this context.

267 was to determine the contribution of RVLM-C1 neurones to irregular breathing patterns in HF.

268 ributions of the medial septum's cholinergic neurones to these functions remain unknown.

269 nnels, their behavioural roles in any single neurone type are not clear.

270 tion of subthreshold computation in a single neurone type causes specific modifications to behaviour.

271 e single cell recordings suggest diverse and neurone type-specific computational functions for HCN1 c

272 ocellular vasopressin (VP) and oxytocin (OT) neurones undergo long-term synaptic plasticity to accomm

273 ctivation on several populations of striatal neurones using a combination of genetic identification,

274 The average conduction velocity to the Sp5I neurones was 0.94 +/- 0.18 mm ms(-1) indicating a mixtur

275 nly observed when this subpopulation of DVMN neurones was activated or inhibited.

276 phagocytic response of BV2 cells to stressed neurones was also reduced in the presence of DHA.

277 ritic arborization of gastric-projecting DMV neurones was increased.

278 luorescent protein (GFP) exclusively in Hcrt neurones, we examined the roles of intracellular levels

279 gration of synaptic inputs by inferior olive neurones, we find that the inhibitory component is reduc

280 ques on identified vagal pancreas-projecting neurones, we studied the effects of metabotropic glutama

281 The DMV neurones were also less responsive to superfusion with t

282 Living orexin neurones were identified by targeted expression of green

283 uN revealed 28.5+/-1.9% of NeuN labelled MVN neurones were Kv3.3-positive.

284 f the CeA whereas 5-HT(1A) receptor positive neurones were located mainly in the lateral part of the

285 Current clamp recordings from MSO neurones were made while pharmacologically isolated exci

286 The signals evoked by 3-4 muM Glu in neurones were markedly inhibited by 3-10 mum MPEP (block

287 en NTS and identified gastric-projecting DMV neurones were not affected by OXT in any neurones tested

288 d changes in morphological properties of DMV neurones were not reversed following gastric bypass surg

289 These neurones were predominantly glutamatergic, and were loca

290 in anaesthetized rats was observed when DVMN neurones were silenced.

291 Further, in neurones where L-AP4 decreased mIPSC frequency, exendin-

292 ncluding Na(V)1.7 being expressed in IB4 +ve neurones, whereas toxin sensitivity indicates that IB4 v

293 re we focused on ventrobasal thalamocortical neurones, which contribute to behaviourally relevant osc

294 right DVMN revealed that vagal preganglionic neurones, which have an impact on LV contractility, are

295 d by dorsal motor nucleus of the vagus (DMV) neurones, whose activity is finely tuned by GABAergic an

296 ry postsynaptic current (EPSC) in trigeminal neurones with a latency of 1.8 +/- 0.1 ms, jitter of 625

297 Preincubation of neurones with SP (10 nM, 5 min) desensitized Ca(2+) tran

298 the basic electrical properties of mouse MHb neurones with whole-cell patch clamp electrophysiology,

299 The hypocretin/orexin (Hcrt)-containing neurones within the lateral hypothalamus integrate nutri

300 (FGF20) supports maintenance of dopaminergic neurones within the nigrostriatal pathway.