ライフサイエンスコーパス: neuroplasticity

1 l and activity of cholinergic nerves (termed neuroplasticity).

2 dulated by genetically driven variability in neuroplasticity.

3 e impairment, suggesting adaptive structural neuroplasticity.

4 conditions and are sites of stress-mediated neuroplasticity.

5 ith neuroinflammatory signaling and impaired neuroplasticity.

6 ation, indicating long-term training-related neuroplasticity.

7 -PKA-CREB and cAMP-ERK1/2-CREB signaling and neuroplasticity.

8 eptor subunit 2 (GluN2) that is critical for neuroplasticity.

9 DHA) 30 min after spinal cord injury induces neuroplasticity.

10 e effects of mucosal mediators on intestinal neuroplasticity.

11 beneficial effects in SCI via enhancement of neuroplasticity.

12 urvivors, with evidence for training-related neuroplasticity.

13 ey pathways involved in neurodevelopment and neuroplasticity.

14 nt dental implant placement can reverse this neuroplasticity.

15 ly G-member2 (ABCG2) have important roles in neuroplasticity.

16 ng neuroprotection, axonal regeneration, and neuroplasticity.

17 gh maladaptations in glutamate signaling and neuroplasticity.

18 dependent reorganization allowing for guided neuroplasticity.

19 al roles in the neuron survival, growth, and neuroplasticity.

20 manization of Foxp2 on learning and striatal neuroplasticity.

21 tiate antidepressant responses by triggering neuroplasticity.

22 ange factor, modulates PSD-95-NR2B-dependent neuroplasticity.

23 oderate disease through experience-dependent neuroplasticity.

24 e functions by the long-term manipulation of neuroplasticity.

25 vel cognition by the long-term modulation of neuroplasticity.

26 ify factors responsible for adult peripheral neuroplasticity.

27 nct sociosexual behaviors and the associated neuroplasticity.

28 ence for localized mechanisms of hippocampal neuroplasticity.

29 ng trauma through chondroitinase ABC induced neuroplasticity.

30 e and as a key signal for dependence-related neuroplasticity.

31 ue to induce long-term depression (LTD)-like neuroplasticity.

32 and function, and regulate cell survival and neuroplasticity.

33 onstrated that chronic pain causes long-term neuroplasticity.

34 h are altered by hypoxia-induced respiratory neuroplasticity.

35 y in chronic SCI following 2 weeks of spinal neuroplasticity.

36 in neuroinflammation, oxidative stress, and neuroplasticity.

37 any genes involved in synaptic signaling and neuroplasticity.

38 own to be important for experience dependent neuroplasticity.

39 re during rescue assays in mice deficient in neuroplasticity.

40 valuable model disorder to study structural neuroplasticity.

41 characterize perinatal temporal dynamics in neuroplasticity.

42 is linked with decreased activity-dependent neuroplasticity.

43 to be within the classic critical period for neuroplasticity [7, 8].

44 structure, a critical step in understanding neuroplasticity across multiple scales.

45 ne-bound astrocyte growth inhibitor to limit neuroplasticity, activity-dependent axonal sprouting, an

46 Previous studies demonstrate distinct neuroplasticity adaptations in the two medium spiny neur

47 ent-related brain potentials (ERPs) to assay neuroplasticity after auditory conditioning in chronic s

48 epresent a strategy for inducing respiratory neuroplasticity after declines in respiratory function t

49 t employ principles consistent with inducing neuroplasticity also are showing improved performance fo

50 stress history, suggesting roles for CRH in neuroplasticity and adaptation of the reward circuitry.

51 physical activity has been shown to promote neuroplasticity and an anti-inflammatory state in the ad

52 functional relationship between cholinergic neuroplasticity and ASM contractile phenotypes that oper

53 o) enhancements in hippocampal cognition and neuroplasticity and can alleviate hippocampal cognitive

54 tients with mood disorders who have impaired neuroplasticity and cognition.

55 clude increased neurogenesis, enhancement of neuroplasticity and deployment of a successful endoplasm

56 ecific genetic interactions related to brain neuroplasticity and development, and that these AKT1 eff

57 rting AKT1's role in transducing hippocampal neuroplasticity and dopaminergic processes, we found epi

58 has been implicated in experience-dependent neuroplasticity and drug-seeking behaviors.

59 immediate early genes and genes involved in neuroplasticity and epigenetic modifications.

60 ow for poststroke intervention that promotes neuroplasticity and facilitates sensorimotor recovery.

61 gnaling, and are thus essential for striatal neuroplasticity and fear and anxiety behavior.

62 at should follow early diagnosis to optimize neuroplasticity and function.

63 n energy metabolism and processes related to neuroplasticity and growth may be involved in the develo

64 n as environmental enrichment, can stimulate neuroplasticity and improve hippocampal function and per

65 is study advances our understanding of human neuroplasticity and its genetic underpinnings following

66 ntal insights into the mechanisms underlying neuroplasticity and its role in clinical care.

67 be introduced to harness a state of enhanced neuroplasticity and lead to improved longer-term clinica

68 ays of VU0409551 treatment could reverse the neuroplasticity and learning deficits, respectively, in

69 rotein (FMRP), thought to play a key role in neuroplasticity and neuronal translation, in ASD-affecte

70 that play an important role in sleep-related neuroplasticity and offline information processing.

71 hich play an important role in sleep-related neuroplasticity and offline information processing.

72 a flourishing of novel strategies to enhance neuroplasticity and promote axon regeneration following

73 ential role for tamalin in ECS-induced adult neuroplasticity and provide new insight into the pathway

74 is a novel therapeutic target for enhancing neuroplasticity and recovery after SCI.

75 Erythropoietin (EPO) increases neuroplasticity and reduces cognitive difficulties in tr

76 data suggest that chronic pain-induced PACAP neuroplasticity and signaling in spinoparabrachioamygdal

77 ry results indicate that tDCS may facilitate neuroplasticity and suggest the potential for refining r

78 may be developed incorporating knowledge of neuroplasticity and the critical ingredients of rehabili

79 However, the linkage between this neuroplasticity and the functional deficits in carpal tu

80 ulation of CaV2.1 channels to other forms of neuroplasticity and to learning and memory are not known

81 ent of newer and more precise tools to alter neuroplasticity and to treat brain-based disorders.

82 ow and why exercise and energy intake affect neuroplasticity and, conversely, how the brain regulates

83 many kynurenines are neuroactive, modulating neuroplasticity and/or exerting neurotoxic effects in pa

84 ogical and engineering strategies to augment neuroplasticity and/or functional recovery in animal mod

85 sease, cerebrovascular deregulation, altered neuroplasticity, and changes in glutamate neurotransmiss

86 ations in neurodevelopment, neurogenesis and neuroplasticity, and is involved in the mechanisms of ac

87 Despite the importance of respiratory system neuroplasticity, and its dependence on estrogen in males

88 tor-induced potentiation of BDNF expression, neuroplasticity, and memory.SIGNIFICANCE STATEMENT Some

89 B (TrkB)], promotes hippocampal synaptic and neuroplasticity, and significantly improves learning and

90 did not), with enrichment in inflammation-, neuroplasticity- and oxidative stress-related pathways.

91 e cellular mechanisms mediating this form of neuroplasticity are poorly understood.

92 review presents a novel integrative model of neuroplasticity as a multi-domain neurobiological, cogni

93 up the perspective of harnessing respiratory neuroplasticity as a therapeutic tool for the management

94 l and paresthesia, and generates maladaptive neuroplasticity as central networks attempt to compensat

95 r dTau in adult mushroom body (MB)-dependent neuroplasticity as its downregulation within alpha'beta'

96 xamining the inter-individual variability in neuroplasticity as modeled by a functional polymorphism

97 c brain regions may be the result of altered neuroplasticity associated with chronic compulsive behav

98 hin-KOR system is a central component of the neuroplasticity associated with negative reinforcement s

99 In other mice, we silenced the neuroplasticity-associated neurotrophin brain-derived ne

100 ation and requires time-dependent changes in neuroplasticity at the level of glutamatergic synapses i

101 a and macrophage functions also will enhance neuroplasticity, at and several segments below the injur

102 at enhances endogenous repair and indices of neuroplasticity, at and several segments below the injur

103 Here we develop a neuroplasticity-based computerized cognitive remediation

104 These results provide support for future neuroplasticity-based treatments that take into account

105 se the hippocampus is a key area involved in neuroplasticity, behavior, and cognition, we hypothesize

106 strata pyramidale and radiatum and assessed neuroplasticity by inducing long-term potentiation (LTP)

107 Our findings illuminate a new mechanism of neuroplasticity by which early nicotine exposure primes

108 Neuroplasticity can be fostered by exploiting four main

109 Overall, we conclude that language-dependent neuroplasticity can enhance behavioral sensitivity to dy

110 We show that beneficial neuroplasticity can occur alongside decoder adaptation,

111 showed comparable improvement, long-lasting neuroplasticity can only be detected in the tDCS group,

112 ng neural decoding and biofeedback to target neuroplasticity causally links early visual cortical pla

113 Insulin signaling is critical for neuroplasticity, cerebral metabolism as well as for syst

114 sed thalamocortical FC, thus suggesting that neuroplasticity changes are unable to compensate for tis

115 roduce opioid-induced hyperalgesia (OIH) and neuroplasticity characterized by prolongation of inflamm

116 Our findings imply that robust neuroplasticity conferred by musical training is not res

117 Furthermore, we studied whether this neuroplasticity contributed to spinal neuron sensitizati

118 is known to regulate emotional behavior and neuroplasticity, contributes to changes in amygdalar str

119 After reviewing relevant neuroplasticity deficits in depression, we survey biolog

120 neurogenesis or gliogenesis may help reverse neuroplasticity during abstinence and, thus, may help re

121 ute to initiating or maintaining respiratory neuroplasticity during chronic hypercapnia but alone do

122 ffects of androgen and estrogen treatment on neuroplasticity during photostimulation in male and fema

123 These findings demonstrate acute functional neuroplasticity during stress, with distinct and separab

124 rphism impairs the beneficial behavioral and neuroplasticity effects induced by physical exercise.

125 beneficial effects in spinal cord injury via neuroplasticity enhancement.

126 cy, indicating limited success in modulating neuroplasticity, especially in brains with neural atypic

127 Concomitantly, neurogenesis and neuroplasticity fall significantly.

128 This is the first report to address neuroplasticity features in human limbic structures conn

129 Neuroplasticity following antidepressant drug treatment

130 rol of Arc that could be targeted to harness neuroplasticity for clinical applications.

131 In conclusion, primary somatosensory cortex neuroplasticity for median nerve innervated digits in ca

132 These results highlight the utility of neuroplasticity for real-world neuroprostheses.

133 rdsong and exhibits some of the best-studied neuroplasticity found in the adult brain.

134 iological convergence between stress-related neuroplasticity, functional neurological symptoms and re

135 epigenetic repression of common Tip60 target neuroplasticity genes occurs early in multiple types of

136 Repressed neuroplasticity genes show reduced enrichment of Tip60 a

137 the pathophysiology of depression, including neuroplasticity, genetic and neural networks, the stress

138 identified, and the impact of this system on neuroplasticity has been well established.

139 Neuroplasticity has classically been understood to arise

140 Models of AIH-induced neuroplasticity have focused on motoneurons; however, mo

141 direct current stimulation to modulate human neuroplasticity have increased in research and clinical

142 direct current stimulation to modulate human neuroplasticity have increased in research and clinical

143 lt resulted in both adaptive and maladaptive neuroplasticity: i.e., sparing contextual memory, but af

144 ferentially expressed miRNAs are involved in neuroplasticity, immune function and neurorestoration.

145 pment but continue to play a central role in neuroplasticity in adulthood; and that they work not onl

146 We examined cross-modal neuroplasticity in anatomically defined subregions of He

147 These findings show cholinergic neuroplasticity in asthma driven by TrkB signaling and s

148 ic systems, which point to the importance of neuroplasticity in AT.

149 oth measures objectively indicate functional neuroplasticity in auditory perceptual learning.SIGNIFIC

150 MRI) has greatly extended the exploration of neuroplasticity in behaving animals and humans.

151 the central role for miRNAs in drug-induced neuroplasticity in brain reward systems that drive the e

152 ibility that the mechanism may also regulate neuroplasticity in circuits other than the olfactory sen

153 ine, and BDNF may be required to link D2R to neuroplasticity in cocaine addiction in females.

154 cervical spinal cord to recover respiratory neuroplasticity in disorders of respiratory insufficienc

155 ntation, pattern recognition capability, and neuroplasticity in each efferent circuit.

156 mprovement of blood flow and facilitation of neuroplasticity in elderly people, might also have a rol

157 ticolimbic regions undergoes cocaine induced neuroplasticity in female rats.

158 specific affect of D2 receptor activation on neuroplasticity in humans, because physiological effects

159 the amount of D1-like receptor activation on neuroplasticity in humans, we combined sulpiride, a sele

160 Overall, the results suggest that the neuroplasticity in MIo and SIo occurring in parallel ser

161 Hyperalgesic priming, a form of neuroplasticity in nociceptors, is a model of the transi

162 Neuroplasticity in OT-producing neurons in the paraventr

163 that electroconvulsive therapy (ECT) induces neuroplasticity in patients with severe depression, thou

164 that are believed to result from functional neuroplasticity in prefrontal and auditory cortices.

165 The ultimate utility of neuroplasticity in real-world neuroprostheses is thus un

166 their involvement in defective signaling and neuroplasticity in relationship with mood disorders is s

167 Neuroplasticity in response to adverse environmental con

168 ate preterm adolescents had reduced LTD-like neuroplasticity in response to brain stimulation that wa

169 and limbic brain regions, though associative neuroplasticity in sensory structures is increasingly ap

170 athing protocol find expression as long-term neuroplasticity in serial measurements made over 20 days

171 Neuroplasticity in the amygdala drives pain-related beha

172 ain-related behaviors.SIGNIFICANCE STATEMENT Neuroplasticity in the amygdala has emerged as an import

173 permanent and dynamically regulated, driving neuroplasticity in the brain.

174 ECT-induced neuroplasticity in the hippocampus and amygdala relates

175 ECT induces functional neuroplasticity in the hippocampus, which could represen

176 exerts a nonlinear dose-dependent effect on neuroplasticity in the human motor cortex that differs f

177 following loss of natural reward by causing neuroplasticity in the mesolimbic pathway during the nat

178 Drugs of abuse induce neuroplasticity in the natural reward pathway, specifica

179 increases expression of cellular markers of neuroplasticity in the non-injured S1 compared to TBI ra

180 Estrogen withdrawal induces OT neuroplasticity in the paraventricular nucleus of the hy

181 ion at the wrist by somatotopically distinct neuroplasticity in the primary somatosensory cortex foll

182 o characterized by functional and structural neuroplasticity in the primary somatosensory cortex of t

183 lling evidence has demonstrated considerable neuroplasticity in the respiratory control system, few s

184 t that physical activity could contribute to neuroplasticity in the RVLM.

185 the hypothesis that singing activity induces neuroplasticity in the song control system independent o

186 by TCT in ESZ are associated with structural neuroplasticity in the thalamus.

187 , our findings suggest that learning-related neuroplasticity in the VLO may be necessary for memory r

188 TA) are a key target of addictive drugs, and neuroplasticity in this region may underlie some of the

189 We examined motor cortex neuroplasticity in three groups of adolescents who were

190 plore the use of decoder adaptation to shape neuroplasticity in two scenarios relevant for real-world

191 , in vivo administration of fentanyl induces neuroplasticity in weakly IB4+ and IB4- nociceptors that

192 ose born deaf can offer unique insights into neuroplasticity, in particular in regions of superior te

193 of several susceptibility genes involved in neuroplasticity, including Bdnf, its receptor TrkB, the

194 nding attenuation of FLX-induced hippocampal neuroplasticity, including decreased hippocampal neuroge

195 genesis and expression of genes critical for neuroplasticity, including WNT pathway components and io

196 This NPY-mediated neuroplasticity indicates that resilience or vulnerabili

197 The neuroplasticity induced by Pavlovian fear conditioning h

198 finity, concentration level, and the kind of neuroplasticity induced.

199 ficity, concentration level, and the kind of neuroplasticity induced.

200 Neuroplasticity is a fundamental property of the nervous

201 ults suggest that stimulus-specific auditory neuroplasticity is abnormal in schizophrenia.

202 ions, which emerge when experience-dependent neuroplasticity is at its peak.

203 The ability of neuritin to increase neuroplasticity is confirmed in models of learning and m

204 Neuroplasticity is essential to prevent clinical worseni

205 Neuroplasticity is limited in maturity, but it is promot

206 These data indicate that PSA-NCAM-mediated neuroplasticity is necessary for antidepressant action;

207 We demonstrated that cholinergic neuroplasticity is necessary for the induction of persis

208 hapes brain development throughout life, but neuroplasticity is variable from one brain system to ano

209 e II (alphaCaMKII), a molecule implicated in neuroplasticity, is a target of CPEB and can also affect

210 opioid-primed rats), long-lasting nociceptor neuroplasticity manifested as prolongation of prostaglan

211 Neuroplasticity manifests as normalization of aberrant f

212 thophysiology suggests that an impairment of neuroplasticity may be a critical part of the developmen

213 Disrupted neuroplasticity may be an important aspect of the neural

214 extinction occurs during DCS administration, neuroplasticity may be enhanced.

215 ve motor neurons or restore functional motor neuroplasticity may extend longevity in amyotrophic late

216 Neuroplasticity may play a critical role in developing r

217 These data suggest that altered amygdala neuroplasticity may play a role the early dispositional

218 ngs are part of highly complex molecular and neuroplasticity mechanisms underlying neuroadaptation re

219 Cocaine-induced neuroplasticity mediated by histone acetylating and deac

220 call induces a limited period of hippocampal neuroplasticity mediated, in part, by S-nitrosylation of

221 Our observations argue that neuroplasticity mediating perceptual learning occurs at

222 We report on an in vitro model of nociceptor neuroplasticity mediating this opioid-induced hyperalges

223 acilitated synaptic transmission and induced neuroplasticity mimicking the LTP of EPSPs.

224 common underlying vulnerability model; 2) a neuroplasticity model in which impairment is concurrent

225 A newly discovered form of neuroplasticity, neurotransmitter switching, involves ch

226 they may play a role related to the reduced neuroplasticity observed in depression.

227 rgic inhibition also plays a crucial role in neuroplasticity of adult animals: the balance between ex

228 This study relies on knowledge regarding the neuroplasticity of dual-system components that govern ad

229 It represents a natural example of the neuroplasticity of motion adaptation.

230 Using information from research on the neuroplasticity of selective attention and on the centra

231 We hypothesize that epilepsy-related neuroplasticity of septohippocampal cholinergic neurons

232 ave major implications for understanding the neuroplasticity of the brain, which could hypothetically

233 Neuroplasticity of the C. elegans IL2 sensory neurons pr

234 -shaped effects of D1 receptor activation on neuroplasticity of the motor cortex.

235 The neuroplasticity of the mu-opioid system is of future res

236 stem or 'darkness within.' Understanding the neuroplasticity of the neurocircuitry that comprises the

237 Nicotine intake induces addiction through neuroplasticity of the reward circuitry, altering the ac

238 Rather, NT3 induced neuroplasticity of the spared corticospinal and serotone

239 Neuroplasticity of the visual cortex or higher cortical

240 Interventions that enhance or modulate neuroplasticity often reduce depressive symptoms when ap

241 exercise and cognitive challenge to enhance neuroplasticity, our ACM addresses two fundamental quest

242 ic strokes, suggesting impaired capacity for neuroplasticity over this hemisphere as a consequence of

243 s and circuit connectivity observed in these neuroplasticity paradigms, perhaps thereby contributing

244 They directly regulate inflammation and neuroplasticity pathways and also influence insulin sens

245 otrophic factor that modulates intracellular neuroplasticity pathways.

246 lar proteases have emerged as key players in neuroplasticity phenomena.

247 et (CR) therapy based on neuromodulation and neuroplasticity principles has been proposed for the tre

248 nd P60), a highly dynamic phase of postnatal neuroplasticity, profoundly impacted transcription in ea

249 The results demonstrate that RTX induces neuroplasticity, rather than structural changes in prima

250 ncrease in DNMT3a in SST+ neurons paralleled neuroplasticity reduction at 12 and 18 months.

251 elationship between connectivity changes and neuroplasticity-related gene expression profiles in the

252 and single nucleotide polymorphisms (SNP) in neuroplasticity-related genes were compared between asth

253 tion epigenetically primes the expression of neuroplasticity-related genes, which is accompanied by h

254 associated with decreased expression of the neuroplasticity-related polysialylated neural cell adhes

255 s from living neurons that interact with the neuroplasticity-relevant CS-E sulfation motif.

256 , but the mechanisms regulating this form of neuroplasticity remain unclear.

257 language impairment after stroke (aphasia), neuroplasticity research is garnering considerable atten

258 itation (pLTF), a form of spinal respiratory neuroplasticity resulting in increased phrenic nerve mot

259 ic priming, a form of maladaptive nociceptor neuroplasticity, resulting in pain chronification.

260 R-/- mice reverses their deficits in several neuroplasticity signaling pathways and improves their co

261 , decreased dendritic spine density, altered neuroplasticity signaling pathways, and cognitive defici

262 signaling in any form of respiratory system neuroplasticity.SIGNIFICANCE STATEMENT Exposure to acute

263 Diabetes may impair the capacity for neuroplasticity such that patients experience a slower a

264 rch have been associated with impairments of neuroplasticity, such as neuronal atrophy and synaptic l

265 ion of genes implicated in the mechanisms of neuroplasticity, such as the NMDA and AMPA subunits and

266 upon a structural scaffolding with intrinsic neuroplasticity that changes with development, aging, di

267 traint stress causes long-lasting changes in neuroplasticity that likely reflect pathological modific

268 a suggest a mechanism of aberrant prefrontal neuroplasticity that underlies enhanced propensity for i

269 significantly reduced capacity for cortical neuroplasticity, the key overall mechanism underlying le

270 ognitive training and neuromodulation affect neuroplasticity, their combination could promote greater

271 the hypothesis that a robust enhancement of neuroplasticity through these methods might promote the

272 ic memory and is commonly thought to rely on neuroplasticity to adapt to the ever-changing environmen

273 Synapses and circuits rely on neuroplasticity to adjust output and meet physiological

274 global back-gating capability to demonstrate neuroplasticity to capture behavioral and/or adaptation

275 ation or TrkB signaling in vivo impaired the neuroplasticity to chronic stress and the effects of the

276 mental hypothesis linking decreased amygdala neuroplasticity to early-life dispositional anxiety.

277 lso be targeted in the context of adolescent neuroplasticity to improve outcomes.

278 Recovery of function relies on augmenting neuroplasticity to potentiate sprouting and regeneration

279 us system may interact with aberrant central neuroplasticity to produce the clinical picture.

280 h particular emphasis on neuroendocrinology, neuroplasticity, transcriptomics, and epigenetics.

281 Here we demonstrate that the neuroplasticity underlying nociceptor priming requires 7

282 Spinal mGluR5 is a key mediator of neuroplasticity underlying persistent pain.

283 The second uses neuroimaging to investigate neuroplasticity, via changes in connectivity, cortical t

284 Nevertheless, hippocampal neuroplasticity was impaired in LV-IRAS-treated rats.

285 he molecular and cellular mechanisms of this neuroplasticity, we investigated the effects of olfactor

286 Since inflammation modulates neuroplasticity, we studied the impact of inflammation c

287 Neural reuse is a form of neuroplasticity whereby neural elements originally devel

288 t is highly likely that inflammation-induced neuroplasticity, which is not detectable by clinical dia

289 dministration of fentanyl induces nociceptor neuroplasticity, which persists in culture, providing ev

290 ppocampal neural circuitry provides enhanced neuroplasticity, which plays a crucial role in learning

291 ivity-regulated neurodevelopment and ongoing neuroplasticity with an arc toward appreciating neuron-g

292 e local connectome fingerprint also revealed neuroplasticity within an individual reflected as a decr

293 ered after SCI and characterized DHA-induced neuroplasticity within the adult injured spinal cord.

294 remodels cortical synapses and suggest that neuroplasticity within the healthy oPFC gates the influe

295 However, excessive functional neuroplasticity within the hippocampus may not be conduc

296 target genes that are well-known markers of neuroplasticity within the hippocampus.

297 Activity-dependent and BDNF-dependent neuroplasticity within the OFC coordinate outcome-based

298 localize the site and candidate mechanism of neuroplasticity within upstream regions of this inhibito

299 ironments, and the subsequent development of neuroplasticity within visceral pain pathways.

300 ssue of Neuron, Newbold et al. build on this neuroplasticity work using precision neuroimaging and ar