ライフサイエンスコーパス: neurotransmission

1 d synaptic vesicle numbers, restoring normal neurotransmission.

2 te novel insights into the chemical basis of neurotransmission.

3 enesis, axonogenesis, neuronal migration and neurotransmission.

4 tein receptor (SNARE) protein in spontaneous neurotransmission.

5 s, suggesting a role in mediating excitatory neurotransmission.

6 properly balanced excitatory and inhibitory neurotransmission.

7 ith a limited role in spontaneous inhibitory neurotransmission.

8 system, with potential impact on excitatory neurotransmission.

9 stress and MDD, and may diminish excitatory neurotransmission.

10 racterized by dysregulation of glutamatergic neurotransmission.

11 synaptic vesicles to release sites, ensuring neurotransmission.

12 e on the DAT accounts for fluctuations in DA neurotransmission.

13 d with mitochondrial dysfunction and altered neurotransmission.

14 0 days of culture and is driven by glutamate neurotransmission.

15 e observed to disrupt profoundly glycinergic neurotransmission.

16 nt in neurons with a decrease in spontaneous neurotransmission.

17 ey were mediated by an increase in GABAergic neurotransmission.

18 ptoms suggest permanent effects of injury on neurotransmission.

19 ylation modulates the efficacy of inhibitory neurotransmission.

20 n nicotinic, dopaminergic, and glutamatergic neurotransmission.

21 C to evaluate ICC-IM responses to excitatory neurotransmission.

22 nhibiting cocaine-mediated enhancement of DA neurotransmission.

23 imbalance between excitatory and inhibitory neurotransmission.

24 GABA(A)Rs which are only activated by evoked neurotransmission.

25 es reporting associations with glutamatergic neurotransmission.

26 ents to regulate excitatory and dopaminergic neurotransmission.

27 from imbalances in excitatory and inhibitory neurotransmission.

28 ues, and links to GABAergic and serotonergic neurotransmission.

29 induce a long-lasting decrease in inhibitory neurotransmission.

30 and sustain the homeostatic potentiation of neurotransmission.

31 hways associated with synaptic structure and neurotransmission.

32 eric ion channels that mediate fast chemical neurotransmission.

33 and mediate the majority of fast excitatory neurotransmission.

34 d the two work together to control glutamate neurotransmission.

35 n memory storage by modulating glutamatergic neurotransmission.

36 Ca(2+) influx, leading to impaired synaptic neurotransmission.

37 ise, at least in part, from reduced dopamine neurotransmission.

38 nt to a role for glutamatergic and GABAergic neurotransmission.

39 ative strengths of excitatory and inhibitory neurotransmission.

40 tsynaptic densities, a feature of excitatory neurotransmission.

41 y play key roles in neuronal development and neurotransmission.

42 nsitive to changes in NMDA receptor-mediated neurotransmission.

43 al layer of activity-dependent modulation of neurotransmission.

44 ysiologic and pathophysiologic glutamatergic neurotransmission.

45 egation of inhibitory spontaneous and evoked neurotransmission.

46 nge from transport through cell membranes to neurotransmission.

47 1 and Slc38a2 have a dual role in regulating neurotransmission.

48 eased from presynaptic terminals to modulate neurotransmission.

49 is an exquisite model organism to understand neurotransmission.

50 on NMDA receptor signaling and glutamatergic neurotransmission.

51 sex and compared synaptic ultrastructure and neurotransmission.

52 pid-onset kinetics of endogenous cholinergic neurotransmission.

53 istic reasoning due to reduced glutamatergic neurotransmission.

54 equency occurred without an effect on evoked neurotransmission.

55 y functions, including phototransduction and neurotransmission.

56 DA(VTA) neurons via both direct and indirect neurotransmissions.

57 mily members serve as negative regulators of neurotransmission, acting directly at the level of exocy

58 ystem elicit neuroimmune responses and alter neurotransmission, affecting host neurological functions

59 In this review, we focus on the pathology of neurotransmission after TBI, reflecting on what has been

60 lls or pharmacologically blocking inhibitory neurotransmission also induced IGF1 competence.

61 sporter (DAT) is a key regulator of dopamine neurotransmission and a target of abused psychostimulant

62 ssion of genes encoding proteins involved in neurotransmission and action potential generation.

63 r this mutation display impaired striatal DA neurotransmission and altered DA-dependent behaviors tha

64 tamate receptors (AMPARs) mediate excitatory neurotransmission and are central regulators of synaptic

65 GABA and glycine mediate fast inhibitory neurotransmission and are coreleased at several synapse

66 1)R), producing an imbalance in dopaminergic neurotransmission and cell death.

67 5-HT, independent from its contributions to neurotransmission and cellular signalling, in the mediat

68 getics, leading to abnormalities in synaptic neurotransmission and cognitive function in a region-dep

69 In live mouse embryos, we observed transient neurotransmission and early vascularization of neural cr

70 sults suggest that complementary measures of neurotransmission and energy metabolism are in partial a

71 z oscillatory stimulation enhanced vHPC-mPFC neurotransmission and entrained neural activity in the v

72 2A) knock-in mouse and evaluated its enteric neurotransmission and gastrointestinal (GI) motility to

73 oupled receptors (GPCRs)-mediates inhibitory neurotransmission and has been implicated in epilepsy, p

74 tive upregulation of gene sets implicated in neurotransmission and immune responses.

75 of known relationships (e.g., glutamatergic neurotransmission and inflammation with depression, IL-3

76 cannabinoid (eCB) signaling system modulates neurotransmission and inflammation, among other physiolo

77 ecognized role in regulating corticostriatal neurotransmission and influences social preference and r

78 calcium sensor protein that is critical for neurotransmission and is therefore extensively studied.

79 y to be necessary for terminating inhibitory neurotransmission and may also produce bioactive metabol

80 avioral aberrations, suggesting that altered neurotransmission and neuroimmune responses may act in c

81 Here, we propose that the crosstalk between neurotransmission and neuroinflammation may underlie som

82 represents the best-studied link between DA neurotransmission and neuropathology.

83 4-AP restores neurotransmission and number of proprioceptive synapses

84 noise exposure or cochlear function such as neurotransmission and oxidative stress.

85 sensory response, dopamine binding, synaptic neurotransmission and plasticity, among others.

86 questions about their dynamic allocation for neurotransmission and plasticity.

87 ected aberrant NAc excitatory and inhibitory neurotransmission and reduced BLA-NAc-elicited feed-forw

88 minals corelease zinc to modulate excitatory neurotransmission and sensory responses.

89 scope of activity-dependent pH influences on neurotransmission and short-term synaptic plasticity.

90 ether beta2-adrenergic agonists improve both neurotransmission and structural integrity of the NMJ in

91 ly altered through modulation of cholinergic neurotransmission and suggest potential for this system

92 genetic risk variants implicating excitatory neurotransmission and synapse function and underscoring

93 impaired cerebellar Golgi-granule inhibitory neurotransmission and synapse number, providing a partia

94 hannels that are key mediators of excitatory neurotransmission and synaptic plasticity throughout the

95 taxin-3 is dispensable for hippocampal basal neurotransmission and synaptic plasticity, and further s

96 tion and localization to synapses as well as neurotransmission and synaptic plasticity.

97 ha2delta1 subunits promote the glutamatergic neurotransmission and synaptogenesis, as well as strongl

98 of D(3)R antagonists on mesolimbic dopamine neurotransmission and their potential utility as pharmac

99 functions in neurons to facilitate baseline neurotransmission and to enable PHP expression, properti

100 artate, which might participate in mammalian neurotransmission and/or neuromodulation.

101 receptor autoantibodies disrupt glycinergic neurotransmission, and also suggest that the pathogenic

102 lates the excitability of VTA DA neurons, DA neurotransmission, and behaviors modulated by DA.

103 hus, CUL3 is critical to neural development, neurotransmission, and excitation-inhibition (E-I) balan

104 hfinding, neurite outgrowth, synaptogenesis, neurotransmission, and many other neuronal processes are

105 n (alpha-syn or Snca) modulates dopaminergic neurotransmission, and SNCA mutations cause familial PD,

106 e-symptomatically in the cortex, implicating neurotransmission, and symptomatically in the striatum,

107 ls of receptors and ion channels involved in neurotransmission, and the critical roles of neuronal ne

108 e-induced increase in hippocampal excitatory neurotransmission, and this effect concurred with the in

109 signal transduction, conventional models of neurotransmission are based on the specific binding of n

110 eceptors activated by spontaneous and evoked neurotransmission are segregated.

111 ts implicated in ionotropic and metabotropic neurotransmission as well as activation of immune respon

112 enzymes and receptors, markers of monoamine neurotransmission as well as steroid-related transcripts

113 ton-gated cation channels that contribute to neurotransmission, as well as initiation of pain and neu

114 xCT (xCT(-/-) mice), we uncovered decreased neurotransmission at corticostriatal synapses.

115 erring Ca(2+) sensitivity during spontaneous neurotransmission at developing neuromuscular synapses i

116 eover, functional ribbon synapses and active neurotransmission at foveal cone pedicles are possibly p

117 hanced excitatory, but unaltered inhibitory, neurotransmission at intracortical synapses in mouse mod

118 n protein, NPAS2, in mediating glutamatergic neurotransmission at medium spiny neurons (MSNs) in the

119 ing force for calcium ion flux that triggers neurotransmission at presynaptic nerve terminals.

120 ssential role in determining the behavior of neurotransmission at the presynaptic terminal.

121 are key players in mediating fast inhibitory neurotransmission at these synapses.

122 s may also perturb the excitatory-inhibitory neurotransmission balance and trigger neuropsychiatric a

123 es (DEGs) in the mPFC following a reversible neurotransmission blocking technique in D1 or D2 recepto

124 Genetically silencing Purkinje cell neurotransmission blocks the formation of sharp Purkinje

125 Deletion of Gpr151 inhibits evoked neurotransmission but enhances spontaneous miniature syn

126 They regulate serotonergic neurotransmission, but it remains unclear how altered se

127 Dopamine transporter (DAT) controls dopamine neurotransmission by clearing synaptically released dopa

128 mporters (NSSs) in the SLC6 family terminate neurotransmission by coupling the thermodynamically favo

129 Cocaine-driven changes in the modulation of neurotransmission by neuromodulators are poorly understo

130 endent de novo MT nucleation, which controls neurotransmission by providing dynamic tracks for bidire

131 5-HT(3) receptor mediates fast serotonergic neurotransmission by undergoing a series of conformation

132 that the duration and strength of purinergic neurotransmission can be modulated by targeting multiple

133 a reduced synaptic vesicle pool and impaired neurotransmission compared to WT mice, while nCLCa-only

134 g growing evidence that defects in GABAergic neurotransmission contribute to early onset epileptic en

135 eceptors or biphasic GABAergic and nicotinic neurotransmission conveyed by GABA and ACh corelease, wh

136 Robust neurotransmission depends on the replenishment of synapt

137 s involved in both excitatory and inhibitory neurotransmission display altered glycans in the disease

138 ve form of presynaptic depression stabilizes neurotransmission during an extended developmental perio

139 the idea that haloperidol elevated dopamine neurotransmission (e.g., by blocking inhibitory feedback

140 imbalances between excitatory and inhibitory neurotransmission evident in Fmr1-KO mice.

141 s indicate that alcohol increases excitatory neurotransmission exclusively on NAc D1-MSNs in male rat

142 tters, stimulate synaptogenesis and synaptic neurotransmission, form part of the blood-brain barrier,

143 rosophila groups and demonstrated that acute neurotransmission from adult alpha/beta mushroom body (M

144 sion network associated with CUD involved in neurotransmission (GABA, acetylcholine, serotonin, and d

145 Disrupted serotonergic neurotransmission has long been implicated in major depr

146 We find that GABAergic neurotransmission has no detectable role, but that gluta

147 ype 2 (VMAT2), an important regulator of CNS neurotransmission, has an analogous role in the endocrin

148 social stress and dysregulated serotonergic neurotransmission have both been implicated in the etiol

149 le modulation of NDEL1 activity secondary to neurotransmission homeostasis and provide new insights i

150 suggest that sleep/wake state influences DA neurotransmission in a manner that is likely to impact a

151 nd deposition of Abeta altered glutamatergic neurotransmission in a temporally and spatially dependen

152 rt picrotoxin, a GABA(A)R antagonist, blocks neurotransmission in a use-dependent manner at rat hippo

153 ermine the physiological role of glycinergic neurotransmission in baroreflex function, identify the m

154 es revealed the alteration of metabolism and neurotransmission in different brain regions after SCI,

155 f the bacterial cell wall and their roles in neurotransmission in higher eukaryotes are well-establis

156 al-CA1 synapses, but without affecting basal neurotransmission in male mice.

157 e and/or light/dark phase impact DA terminal neurotransmission in male rats.

158 n-1), the main Ca(2+) sensor for synchronous neurotransmission in many neurons, enhances asynchronous

159 he latter of which may contribute to altered neurotransmission in mutant mice.

160 sm for the biological reduction in monoamine neurotransmission in Parkinson's patients.

161 We studied serotonergic neurotransmission in patient forebrain neurons in vitro

162 ction between dopaminergic and glutamatergic neurotransmission in PD and disclose a possible direct i

163 sed by synapses to homeostatically stabilize neurotransmission in response.SIGNIFICANCE STATEMENT Thi

164 triatal disruptions to KOR-dependent DAergic neurotransmission in RGS12-null mice are restricted to t

165 lfactory sensory experience to glutamatergic neurotransmission in the activity-dependent remodeling o

166 rent GluN2 subunits contribute to excitatory neurotransmission in the adult CNS.

167 allosteric modulator (PAM) reduced glutamate neurotransmission in the BLA slices from panic-prone rat

168 ne receptors (GlyRs) mediate fast inhibitory neurotransmission in the brain and have been recognized

169 ut sex determination.)SIGNIFICANCE STATEMENT Neurotransmission in the brain is regulated by presynapt

170 rters are essential players in glutamatergic neurotransmission in the brain, where they maintain extr

171 nels that mediate the majority of excitatory neurotransmission in the brain.

172 annels to mediate the majority of excitatory neurotransmission in the central nervous system.

173 stinct GluN2 subunits and mediate excitatory neurotransmission in the CNS.

174 fos expression and spontaneous glutamatergic neurotransmission in the dorsal bed nucleus of the stria

175 ctional roles for dopamine and noradrenaline neurotransmission in the expression of incidental memory

176 Preclinical evidence implicates cholinergic neurotransmission in the function of these systems and s

177 gnificant regulatory mechanism for nitrergic neurotransmission in the gut.

178 rculating BCAAs levels to favor serotonergic neurotransmission in the hippocampus and promote antidep

179 ance and altered modulation of glutamatergic neurotransmission in the lateral part of the dorsal stri

180 s in mammals, DCC might also tune inhibitory neurotransmission in the mammalian brain.

181 e receptors (AMPARs) mediate fast excitatory neurotransmission in the mammalian central nervous syste

182 ropic glutamate receptors mediate excitatory neurotransmission in the mammalian central nervous syste

183 via modulation of excitatory and inhibitory neurotransmission in the NAc.

184 al that estradiol potently alters inhibitory neurotransmission in the neocortex.

185 iously described plasticity in glutamatergic neurotransmission in the NTS with CH.

186 rs have an important role in fast inhibitory neurotransmission in the spinal cord and brainstem.

187 s that depend on highly regulated inhibitory neurotransmission in the thalamus.

188 amine sensor dLight1 to monitor dopaminergic neurotransmission in the ventral striatum of NF1 mice du

189 output in the nucleus accumbens (NAc) and on neurotransmission in the ventral tegmental area (VTA).

190 nels and the main drivers of fast inhibitory neurotransmission in the vertebrate nervous system(1,2).

191 patterns might determine how ICC respond to neurotransmission in these two regions of the gastrointe

192 ons in prokaryotes and eukaryotes, including neurotransmission in vertebrates.

193 olism of amino acids and vitamins related to neurotransmission, including depletion of serotonin and

194 shed sex differences in GABAergic inhibitory neurotransmission, including extrasynaptic delta subunit

195 Selective depletion of basket cell GABAergic neurotransmission increases the frequency of Purkinje ce

196 These findings establish a neurotransmission-independent role for nuclear dopamine

197 nments is critically important in monitoring neurotransmission, inflammatory responses, cardiovascula

198 Glutamate neurotransmission is a prioritized target for antipsycho

199 naptic strength, suggesting that spontaneous neurotransmission is able to communicate independently w

200 Abnormal dopamine neurotransmission is associated with several neurologica

201 Dysregulated norepinephrine (NE) neurotransmission is associated with the nonmotor sympto

202 gma that the synaptic cleft acidifies during neurotransmission is based on the corelease of neurotran

203 Abnormal neurotransmission is central to schizophrenia (SZ).

204 synaptobrevin-1 and synaptobrevin-2, evoked neurotransmission is completely abolished; however, spon

205 nal neuronal synapses.SIGNIFICANCE STATEMENT Neurotransmission is highly sensitive to the pH of the e

206 kably, despite this exuberant growth, stable neurotransmission is maintained throughout the ATI lifes

207 Fast excitatory neurotransmission is mediated by AMPA-subtype ionotropic

208 variants in the AKT1 gene influence dopamine neurotransmission is not well understood.

209 tructure in the reward system, in which GABA neurotransmission is regulated by opioid neuropeptides,

210 Neurotransmission is sustained by endocytosis and refill

211 dritic spines, suggesting that glutamatergic neurotransmission is unnecessary for synapse assembly an

212 5 signaling influences overall glutamatergic neurotransmission is warranted.

213 s long been a model synapse for the study of neurotransmission, its presynaptic AP waveform has never

214 from sensory ribbon-type synapses show that neurotransmission itself can acidify the synaptic cleft,

215 ells utilizing only native components of the neurotransmission machinery at physiological expression

216 evidence that altered prefrontal inhibitory neurotransmission may be linked to anxiety in IBS.

217 it remains unclear how altered serotonergic neurotransmission may contribute to the SSRI resistance

218 late immune cells, while dysregulation of NE neurotransmission may exacerbate disease progression, pa

219 2+) influx, those underlying the spontaneous neurotransmission may occur with or without Ca(2+) Our f

220 ion caused selective reduction of inhibitory neurotransmission measured in dentate granule cells.

221 sed TEP sensitivity to changes in excitatory neurotransmission mediated by n-methyl-d-aspartate (NMDA

222 ctivity changes involving rhythmic GABAergic neurotransmission mediates diurnal rhythmicity in metabo

223 Excitatory neurotransmission meditated by glutamate receptors inclu

224 ween asynchronous and spontaneous excitatory neurotransmission might serve as a presynaptic substrate

225 The spatiotemporal dynamics of excitatory neurotransmission must be tightly regulated to achieve e

226 ndidate marker for the integrity of synaptic neurotransmission necessary for cognitive processes is g

227 h epigenetic modulations of genes regulating neurotransmission, neurodevelopment, and immune function

228 To achieve ultrafast neurotransmission, neurons assemble synapses with highly

229 f proinflammatory cytokines on monoaminergic neurotransmission, neurotrophic factors, and measures of

230 DD.SIGNIFICANCE STATEMENT Reduced excitatory neurotransmission occurs with major depressive disorder,

231 Altered neurotransmission of gamma-aminobutyric acid (GABA) has

232 ceptors govern the electrical properties and neurotransmission of neuronal networks.

233 ted the effects of attenuating noradrenergic neurotransmission on learning under uncertainty.

234 intestinal milieu to the brain through fast neurotransmission onto neurons, including those of the v

235 igating the D(3)R's precise role in dopamine neurotransmission or how it may be exploited to modulate

236 not exhibit significant changes in CA1 basal neurotransmission or in paired-pulse ratios.

237 s revealed a significant reduction in evoked neurotransmission, our present findings show that the st

238 Ca(v)2.2/N-type, crucial for SNARE-mediated neurotransmission, particularly in the early postnatal p

239 nistration enhanced neuronal development and neurotransmission pathways.

240 These effects were not due to cholinergic neurotransmission, persisted during partial blockade of

241 Surprisingly, however, spontaneous neurotransmission persists in the absence of both Syb1 a

242 dSLC25A4-sesB, affected synapse morphology, neurotransmission, plasticity, and sleep patterns.

243 and postsynaptic components of glutamatergic neurotransmission plays a crucial role in orchestrating

244 taining to synapse structure and morphology, neurotransmission, postsynaptic signaling pathways, and

245 ver, aberrant mPFC excitatory and inhibitory neurotransmission potentially contributing to psychologi

246 CRs) that couple to G(i/o) proteins modulate neurotransmission presynaptically by inhibiting exocytos

247 ta indicate GABAergic spontaneous and evoked neurotransmission processes are partially non-overlappin

248 tome at 3 weeks related to processes such as neurotransmission, protein turnover, and DNA transcripti

249 approach for inducibly disrupting excitatory neurotransmission, providing a first-in-class optogeneti

250 es, suggest that cleft alkalinization during neurotransmission, rather than acidification, is a gener

251 isrupted in T1D, which affects glutamatergic neurotransmission related to emotional or cognitive proc

252 Furthermore, spontaneous neurotransmission remains constant in Syb1(lew/lew)Syb2

253 Whereas evoked neurotransmission requires Ca(2+) influx, those underlyi

254 Fast, precise and sustained neurotransmission requires graded Ca(2+) signals at the

255 h inhibitory (GAD65) and excitatory (VGLUT2) neurotransmission revealed a significant positive correl

256 f stellate cell versus basket cell GABAergic neurotransmission sculpts the firing properties of Purki

257 We found that dopamine neurotransmission sensitizes the locomotor behavior elic

258 nscripts associated with inhibitory synaptic neurotransmission showed minor differences between group

259 demands of neuronal activity with excitatory neurotransmission.SIGNIFICANCE STATEMENT Synaptic AMPARs

260 anx1 in suppression of facilitated glutamate neurotransmission.SIGNIFICANCE STATEMENT The postsynapti

261 we tested the hypothesis that glutamatergic neurotransmission specifically contributes to jumping to

262 PC 36 : 1/38 : 3 and amino acids involved in neurotransmission suggest potential depression-related m

263 ing PVH neurons revealed enhanced excitatory neurotransmission, suggesting that leptin acts cell auto

264 Several analogues blocked neurotransmission, suggesting that they are selective fo

265 mouse model, and exacerbated the deficits in neurotransmission, synchrony between the hippocampus and

266 The gamma aminobutyric acid (GABA) neurotransmission system has been implicated in autism s

267 mutual coupling between the neuronal and the neurotransmission systems is essential for understanding

268 ritten by a pathophysiology of the glutamate neurotransmission that affects the excitation-inhibition

269 loss reduces sites of glutamatergic sensory neurotransmission that normally encodes naturalistic tim

270 gnificant increase in excitatory presynaptic neurotransmission that occluded presynaptic but not post

271 ice exhibit reduced spontaneous dopaminergic neurotransmission that was associated with excitation/in

272 known to modulate excitatory and inhibitory neurotransmission, the balance of which is altered in AU

273 tology category queried, including GABAergic neurotransmission, the endosomal-lysosomal pathway and a

274 to neurodevelopment, synaptic integrity, and neurotransmission; the same analysis for BD was underpow

275 Amphetamine depresses neurotransmission through stimulation of astrocytes and

276 portance of sustained PV-mediated inhibitory neurotransmission throughout life and highlight the pote

277 radigm shift from monoamine to glutamatergic neurotransmission, thus making it a unique tool to inves

278 gical evidence linking reduced glutamatergic neurotransmission to impaired information sampling and t

279 th green glutamate sensors to relate quantal neurotransmission to presynaptic Ca(2+) kinetics.

280 , but defining its role in the regulation of neurotransmission under physiologically relevant conditi

281 Alterations in dopamine neurotransmission underlie some of the clinical features

282 Thus, PVH->LSv neurotransmission underlies dynamic feeding by orchestra

283 Glutamatergic neurotransmission via alpha-amino-3-hydroxy-5-methylisox

284 ploinsufficiency reduced cortical inhibitory neurotransmission via distinct mechanisms from parvalbum

285 amilial Alzheimer's disease (FAD), modulates neurotransmission via interaction of its cytoplasmic tai

286 opamine transporter (DAT) regulates dopamine neurotransmission via reuptake of dopamine released into

287 Monoamine transporters (MATs) regulate neurotransmission via the reuptake of dopamine, serotoni

288 Syb1(lew/lew)Syb2 (-/-) NMJs; and (3) evoked neurotransmission was markedly reduced in Syb2 (-/-) NMJ

289 ate's song, although their striatal dopamine neurotransmission was only slightly elevated.

290 imaging (fMRI), we explored whether altered neurotransmission was paralleled by aberrant mPFC restin

291 ycle can compensate for decreased excitatory neurotransmission when the glutamate-glutamine shuttle i

292 id (GABA) results in prolonged inhibition of neurotransmission, which is central to brain physiology(

293 timulated 5-HT neurons excitability and 5-HT neurotransmission while hindering HFD-induced anxiety.

294 metformin increased hippocampal serotonergic neurotransmission while promoting anxiolytic- and antide

295 g as a SNARE chaperone-but with no effect on neurotransmission-while another posits that alpha-syn at

296 for Ca(2+)-dependent spontaneous excitatory neurotransmission, with a limited role in spontaneous in

297 individual sleep/wake states and DA terminal neurotransmission, with higher DA uptake rate, increased

298 into the adaptive strategies that constrain neurotransmission within narrow physiological ranges whi

299 adaptive countermeasures exist that maintain neurotransmission within proper physiological ranges.

300 nd coordinated Ca(2+)-secretion coupling for neurotransmission, yet functional and anatomical propert