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1 acrophages as integral components of the DRG-neurovascular unit.
2 in depends primarily on the responses of the neurovascular unit.
3 c link between circulating platelets and the neurovascular unit.
4  multiple cellular involvement affecting the neurovascular unit.
5 onal model of bidirectional signaling in the neurovascular unit.
6 ly escalating brain edema, and damage of the neurovascular unit.
7 y in neural/vascular interactions within the neurovascular unit.
8 within the central nervous system called the neurovascular unit.
9 pivotal role in dynamic signaling within the neurovascular unit.
10 d endothelial cells, which together form the neurovascular unit.
11  is a downstream substrate of tPA within the neurovascular unit.
12 t the BBB and other nonneuronal cells of the neurovascular unit.
13  affected by the acidification of the entire neurovascular unit.
14 gate how circadian biology may influence the neurovascular unit.
15 t regulate the formation and function of CNS neurovascular units.
16 ing molecules to improve the function of the neurovascular unit after injury.
17 volved in the ischaemic death process at the neurovascular unit, an improved preselection and evaluat
18 TEMENT: Pericytes are a key component of the neurovascular unit and are essential for normal BBB func
19    Given the role of APOE in maintaining the neurovascular unit and as an anti-inflammatory molecule,
20  affects the brain through compromise of the neurovascular unit and astrocyte injury.
21 s support bidirectional signaling within the neurovascular unit and astrocytes as key modulators of P
22                                              Neurovascular unit and barrier maturation rely on vascul
23 s Lama2 expression in mural cells to promote neurovascular unit and barrier maturation.
24 ator (t-PA) can modulate permeability of the neurovascular unit and exacerbate injury in ischemic str
25 er, the relationship of these changes in the neurovascular unit and impact on vision remains to be de
26 tion between different cell types within the neurovascular unit and intact blood-brain barrier (BBB)
27 he inner BRB (iBRB) results in damage to the neurovascular unit and is a principal cause of vision lo
28 e well established action of estrogen in the neurovascular unit and its potential interaction with re
29 ighly interactive processes that involve the neurovascular unit and neural stem cells.
30  should be able to target the multi-cellular neurovascular unit and the therapeutic relationships amo
31 phenotype, it plays a pathogenic role in the neurovascular unit and triggers neuronal hyperexcitabili
32 f SAC in functional preservation of ischemic neurovascular units and its therapeutic relevance in the
33  regions of brain interface at synapses, the neurovascular unit, and other sites of intercellular com
34  knowledge of the blood-retinal barrier, the neurovascular unit, and pathological and functional resp
35 mmunication between the affected brain area, neurovascular unit, and peripheral immune cells.
36 ngers are effective in all cell types of the neurovascular unit, and should still be considered as a
37 entral nervous system (e.g., microglia), the neurovascular unit, and the peripheral circulation.
38 rrier integrity in microfluidics-based human neurovascular units, and reduced mortality in a lethal i
39 od-brain barrier (BBB) and components of the neurovascular unit are an area of active research.
40 s released into circulation from the injured neurovascular unit are important prognostic tools in pat
41 etween neurons, vasculature, and glia within neurovascular units are critical for maintenance of the
42                 Central nervous system (CNS) neurovascular units are multicellular complexes consisti
43                                              Neurovascular units are the cellular and molecular inter
44         They are positioned centrally in the neurovascular unit between endothelial cells, astrocytes
45 CNS pericytes are uniquely positioned in the neurovascular unit between endothelial cells, astrocytes
46                                       At the neurovascular unit, brain endothelial cells, astrocytes,
47 ms of endocannabinoid system function at the neurovascular unit can help to unlock the therapeutic po
48  slowly, are prone to contamination by other neurovascular unit cells, and lose blood-brain barrier c
49  shedding light on the mechanisms underlying neurovascular unit communication.
50 pelling evidence indicates impairment of all neurovascular unit components including the blood-brain
51     However, the heterotypic interactions of neurovascular units, composed of neurons, pericytes, ast
52                                  The retinal neurovascular unit consists of blood vessel endothelial
53 s well documented that all components of the neurovascular unit contribute to the restrictive nature
54  issue of the JCI points to perturbations of neurovascular unit coupling caused by perivascular macro
55                        Understanding how the neurovascular unit develops and functions, and more spec
56 erived multipotent regenerative cells to the neurovascular unit during angiogenesis.
57 pressed gene modules, suggesting coordinated neurovascular unit dysregulation in AD.
58                           The concept of the neurovascular unit emphasizes the importance of cell-cel
59  Intravenously injected DAG peptide homes to neurovascular unit endothelial cells and to reactive ast
60 the blood-brain barrier (BBB), a specialized neurovascular unit evolved to maintain brain homeostasis
61 dispensable role that astrocytes play in the neurovascular unit, few studies have investigated the fu
62 e essential for maintaining integrity of the neurovascular unit following brain ischemia.
63 these various gene products normally control neurovascular unit formation and function will lend new
64 and neurovascular repair, and to protect the neurovascular unit from divergent inducers of injury and
65 f the interactions between components of the neurovascular unit has implications for various disorder
66 ese cells to the structural integrity of the neurovascular unit has not hitherto been assessed.
67 mpair the major functional properties of the neurovascular unit (i.e., cerebral vessel regulation and
68 c link between circulating platelets and the neurovascular unit in early dementia pathophysiology.
69  increasing appreciation for the role of the neurovascular unit in neurodegenerative diseases.
70 orably regulate multiple pathways within the neurovascular unit in non-neuronal cells and neurons dur
71           The exact role of pericytes in the neurovascular unit in the adult brain and during brain a
72 g depression susceptibility, implicating the neurovascular unit in the development of migraine aura.
73 s because of progressive degeneration of the neurovascular unit in the retina, a condition known as d
74                                          The neurovascular unit, including astrocytes and microglia,
75 and, SVD heritability is enriched across the neurovascular unit, including astrocytes.
76 ay, we searched for molecular markers of the neurovascular unit, including endothelial cells and astr
77 ological mechanisms that coalesce around the neurovascular unit, including neuroinflammation and dege
78                                          The neurovascular unit is a coordinated and interactional sy
79 lutes through the paracellular spaces in the neurovascular unit is a key mechanism in CNS calcificati
80 merging evidence suggests degradation of the neurovascular unit may underlie the pathogenesis of SVD.
81 physiologically detailed multi-compartmental neurovascular unit model consisting of the vascular smoo
82 multi-targeting capabilities, using in vitro neurovascular unit models and an in vivo model of AD.
83  profoundly impacts communication within the neurovascular unit-neurons, astrocytes, and arterioles-c
84              Proper formation of the complex neurovascular unit (NVU) along with the blood-brain barr
85                                          The neurovascular unit (NVU) consists of cells intrinsic to
86 ing evidence indicates that inflammation and neurovascular unit (NVU) dysfunction contribute to depre
87 r the structural-functional integrity of the neurovascular unit (NVU) in humans.
88                                          The neurovascular unit (NVU) includes multiple different cel
89                                              Neurovascular unit (NVU) is a basic unit in the brain, i
90                                          The neurovascular unit (NVU) is a dynamic center for substan
91                                          The neurovascular unit (NVU) is composed of vascular cells,
92 ithin the greater context of a multicellular neurovascular unit (NVU) that includes neurons, astrocyt
93  results in a pathological disruption of the neurovascular unit (NVU) that involves blood-brain barri
94     Cerebral ischemia induces changes in the neurovascular unit (NVU) that result in brain edema.
95                   Age-related changes to the neurovascular unit (NVU), and their consequences for cer
96                                          The neurovascular unit (NVU), comprising vascular, glial, an
97                           The concept of the neurovascular unit (NVU), formalized at the 2001 Stroke
98 ew has been replaced with the concept of the neurovascular unit (NVU), which encompasses neuronal, gl
99 in the regulation of the permeability of the neurovascular unit (NVU).
100 veral cells that are collectively called the neurovascular unit (NVU).
101 r the structural-functional integrity of the neurovascular unit (NVU).
102  the role of microglia in the biology of the neurovascular unit (NVU).
103 to be present in all major components of the neurovascular unit of both R6/2 mice and HD patients.
104                  miR-155 is expressed at the neurovascular unit of individuals with MS and of mice wi
105 evelopment and applicability of a functional neurovascular unit on a microfluidic chip as a microphys
106 a series of structures collectively known as neurovascular units, or NVU, that are composed of endoth
107         Here, we employed a multicellular 3D neurovascular unit organoid containing human brain micro
108 mposition, involving plausible genes for the neurovascular unit (P2RX5 and TRPV3) and excitatory neur
109 al for the development and physiology of CNS neurovascular units, particularly in the brain.
110                                   Within the neurovascular unit, pericytes play important roles in re
111 hether activation of this pathway within the neurovascular unit protects the brain against blood-brai
112                           The concept of the neurovascular unit provides a useful framework to invest
113 e structural and functional integrity of the neurovascular unit rather than with the regeneration of
114 ells indicates that there is no prototypical neurovascular unit replicated at all levels of the vascu
115                                Targeting the neurovascular unit should provide novel insights for eff
116 entral role for the endothelium within brain neurovascular unit signalling networks.
117                           The concept of the neurovascular unit suggests that to be successful, strok
118 ailed model of potassium flow throughout the neurovascular unit (synaptic region, astrocytes, and art
119 astrocytes, and microglia, cell types in the neurovascular unit that can secrete MMPs.
120 rain endothelial cells are components of the neurovascular unit that comprises the blood-brain barrie
121 contribution to cellular interactions at the neurovascular unit that influence the overall function o
122 human iPSC-derived tissue, we have created a neurovascular unit that recapitulates complex BBB functi
123 ynamic purinergic contacts with cells in the neurovascular unit that shape CBF in both mice and human
124    Astrocytes are critical components of the neurovascular unit that support blood-brain barrier (BBB
125 Pericytes are uniquely positioned within the neurovascular unit to serve as vital integrators, coordi
126  Astrocytes are an integral component of the neurovascular unit where they act as homeostatic regulat
127                                          The neurovascular unit, which includes neurons, glial cells,
128 totoxicity and increased permeability of the neurovascular unit with the development of cerebral edem
129 ons, the amacrine and horizontal cells, form neurovascular units with capillaries in 2 of the 3 retin
130  shows microglia actively participate in the neurovascular unit, with aberrant microglial-vascular fu
131 rocytes and Muller cells, contribute to this neurovascular unit within the brain and retina, respecti
132 scular endothelial cells, which comprise the neurovascular unit within the central nervous system (CN

 
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