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1 d-type (WT), primarily driven by an elevated neutrophil count.
2 n alanine transaminase level or decreases in neutrophil count.
3 vels than wild-type mice, presented elevated neutrophil count.
4 uded hyperglycaemia and fever with decreased neutrophil count.
5 s in CD4 cell count, but not by increases in neutrophil count.
6 in cholesterol levels and with reductions in neutrophil counts.
7 protein cholesterol levels and reductions in neutrophil counts.
8 or treatment normalized CXCR4 expression and neutrophil counts.
9 of cotransplanted Tregs exhibited diminished neutrophil counts.
10 t produce mature T cells but maintain normal neutrophil counts.
11 s might contribute to regulating circulating neutrophil counts.
12 had the greatest effects on regulating blood neutrophil counts.
13 h IL-23, but not IL-12, restored circulating neutrophil counts.
14 l counts, and combined sputum eosinophil and neutrophil counts.
15 in the ischemic brain along with the reduced neutrophil counts.
16 these 52 molecules correlated with BAL fluid neutrophil counts.
17 in eosinophil counts and 64% of variation in neutrophil counts.
18 also decreased platelet counts and increased neutrophil counts.
19 lls/L, p = 9.22 x 10(-95)), particularly for neutrophil count (0.174 x [0.158, 0.190]10(9) cells/L, p
20 ts (0.16 x 10(9) cells/L; P = 0.014), higher neutrophil counts (0.11 x 10(9) cells/L; P = 0.026), and
21 nce, 0.89 x 10(9) cells/L; P < 0.001), lower neutrophil counts (0.83 x 10(9) cells/L; P < 0.001), and
22 bin concentration 9 g/dL or higher, absolute neutrophil count 1 x 10(3) cells per mm(3) or higher, an
23 ase 9.6 g/L, 95% CI 7.6-11.6 g/L; p<0.0001), neutrophil counts (1.1x10(9)/L, 0.7-1.5x10(9)/L; p<0.000
24 anaemia (14 [9%] vs 26 [17%]), and decreased neutrophil count (11 [7%] vs 17 [11%]).
25 ression of 2 of the 3 blood counts: absolute neutrophil count 1200/mm3 or less, platelet count 70,000
26 d grade 3 or 4 adverse events were decreased neutrophil count (14 [39%] of 36 patients in the gemcita
27  common grade 3 or 4 adverse events were low neutrophil count (15 [11%] preoperatively in the chemoth
28 1 [16%] in the CRT only group), low absolute neutrophil count (15 [12%] vs 24 [19%]), fatigue (26 [20
29 in the safety population (n=57) were reduced neutrophil count (15 [26%]), hypophosphatemia (11 [19%])
30 mmon grade 3-4 adverse events were decreased neutrophil count (210 [37%] in the cetuximab group vs 15
31 l neuropathy (25 [6%] vs 12 [3%]), decreased neutrophil count (22 [5%] vs 16 [4%]), and fatigue (17 [
32 spnoea (35 [12%] of 289 patients), decreased neutrophil count (26 [9%]), and fatigue (23 [8%]).
33 t common serious adverse event was decreased neutrophil count: 26 episodes in 14 participants.
34 1 [11%]), dyspnoea (29 [10%]), and decreased neutrophil count (28 [10%]), and with pemetrexed alone w
35 , leucopenia (22 [7%] vs 17 [6%]), decreased neutrophil count (31 [10%] vs 41 [13%]), and decreased w
36 the fixed-dose combination group), decreased neutrophil count (31 [12%] vs 27 [11%]), febrile neutrop
37 ients), leucopenia (42 [16%]), and decreased neutrophil count (40 [15%]); in the placebo plus paclita
38 te blood cell count (11600/microL), absolute neutrophil count (4100/microL), and platelet count (362
39 vacizumab group), and infections with normal neutrophil count (42 events vs 53).
40 ide levels (38 vs 27 ppb, P = .02) and blood neutrophil counts (5.3 vs 4.0 10(9)/L, P </= .001) and s
41        All patients engrafted to an absolute neutrophil count 500/microL at a median of 13 days (rang
42 aemia (138 [29%] vs 47 [20%]), and decreased neutrophil count (57 [12%] vs 19 [8%]).
43 r [2%]) in the nivolumab group and decreased neutrophil count (59 [28%]) in the chemotherapy group.
44                       Adjusted HRs comparing neutrophil counts 6 to 7 versus 2 to 3 x 10(9)/l (both w
45 x 1000 cells/muL [IQR, 7.5, 12.9]; P = .01), neutrophil count (6.7 x 1000 cells/muL [IQR, 2.8, 9.7] v
46 t was correlated to the CD66b microparticles/neutrophil count, a surrogate of neutrophil activation a
47 ether age, FEV1 percent predicted, and blood neutrophil counts accurately predict sputum neutrophil p
48 ed granulocyte-colony stimulating factor and neutrophil counts/activity postsuboptimal immunization.
49 d not correlate with outcome, lower absolute neutrophil count after starting concurrent chemoradiothe
50 GP generation correlates closely with airway neutrophil counts after administration of proteases in v
51                                              Neutrophil counts after trial entry rose significantly m
52    For two traits, lipoprotein(a) levels and neutrophil count, aggregate tests of low-frequency and r
53 137.96+/-0.08 versus 142.71+/-0.07 mm Hg for neutrophil counts; all P<10(-50)).
54                                Mean baseline neutrophil counts also varied significantly by genotype:
55                             Obesity and high neutrophil counts also worsened the PM effects with or w
56  responses, we found significantly different neutrophil counts among treatments; however, phagocytic
57  bones, a markedly enlarged spleen, elevated neutrophil counts, an enlarged heart, and behavioral hyp
58 bserved dose-dependent increases in absolute neutrophil count (ANC) and absolute lymphocyte count (AL
59 caused a dose-dependent decrease in absolute neutrophil count (ANC) and reduced inflammatory biomarke
60                          The median absolute neutrophil count (ANC) at diagnosis was 0.4 x 10(9)/L, a
61 ine neutropenia, 82% of those whose absolute neutrophil count (ANC) decreased by > or = 75% achieved
62 n therapy for inadequate counts: an absolute neutrophil count (ANC) more than 1,000/microL and platel
63 ivariate and multivariate models of absolute neutrophil count (ANC) nadir and pharmacokinetic paramet
64 teria: positive CSF Gram stain, CSF absolute neutrophil count (ANC) of at least 1000 cells/microL, CS
65 , but achieved less than the median absolute neutrophil count (ANC) response (ANC < 2.188 x 10(9)/L [
66          Median time to recovery of absolute neutrophil count (ANC) was 12 days.
67 commercial G-CSF on the increase of absolute neutrophil counts (ANC).
68 blood with normalization of counts (absolute neutrophil count [ANC]> 1.5 x 10(9)/L, hemoglobin [Hgb]
69 mation (alkaline phosphatase [ALP], absolute neutrophil count [ANC], ferritin [adjusted for iron stat
70 her with laboratory tests (white cell count, neutrophil count and C-reactive protein), and the timing
71                                          The neutrophil count and leukotriene B4 concentration in bro
72                      The association between neutrophil count and microvascular obstruction is abolis
73                              The increase in neutrophil count and their priming is mediated by granul
74 ized by a small increase of lymphocyte B and neutrophil counts and a transient drop of total lymphocy
75  of anakinra compared with placebo on airway neutrophil counts and airway proinflammatory cytokine le
76                       Profound reductions in neutrophil counts and chemotaxis as well as a diminished
77 y response in the lung as shown by increased neutrophil counts and elevated cytokine and chemokine co
78 an cluster T1, with no differences in sputum neutrophil counts and exhaled nitric oxide and serum IgE
79 , exogenous MCP-1 dose dependently increased neutrophil counts and G-CSF concentrations in the blood.
80 rity was correlated with total leukocyte and neutrophil counts and HCT (P <0.001, P <0.001 and P = 0.
81 iglyceride, VLDL, total WBC, lymphocyte, and neutrophil counts and increase in hsCRP, total cholester
82  white blood cell, lymphocyte, monocyte, and neutrophil counts and increased adiponectin levels witho
83                Adverse events, including low neutrophil counts and increased levels of alanine aminot
84 sociated with reduced bronchoalveolar lavage neutrophil counts and interleukin-6 and macrophage infla
85         Black African participants had lower neutrophil counts and lower circulating concentrations o
86 GER expression correlated with higher sputum neutrophil counts and more severe AHR in COPD patients.
87 ociated TBM and is characterized by high CSF neutrophil counts and Mycobacterium tuberculosis culture
88 clude that efforts should focus on improving neutrophil counts and on treating the frequent and serio
89 in alveolar air spaces, despite normal blood neutrophil counts and survival of emigrated neutrophils,
90  Blood glucose level, total leukocyte count, neutrophil count, and leukocyte labeling efficiency vari
91  (LDH) level, ferritin level, d-dimer level, neutrophil count, and neutrophil-to-lymphocyte ratio wer
92 racy of the white blood cell count, absolute neutrophil count, and platelet count at commonly used th
93 ve protein, white blood cell count, absolute neutrophil count, and procalcitonin (PCT), specifically
94 ve protein, white blood cell count, absolute neutrophil count, and procalcitonin (PCT), specifically
95 PET/CT correlated with lung function, sputum neutrophil counts, and CF-CT scores.
96 r peripheral blood eosinophil counts, sputum neutrophil counts, and combined sputum eosinophil and ne
97 older age, lower serum albumin, higher blood neutrophil counts, and greater prevalence of chronic obs
98 y increased proinflammatory IL-1beta levels, neutrophil counts, and IL-17-shifted immunity.
99 tween quintiles of lymphocyte, monocyte, and neutrophil counts, and increased systolic BP, diastolic
100            We also measured interleukin-17A, neutrophil counts, and total protein in bronchoalveolar
101 CXCL10, and interleukin 10 levels; increased neutrophil counts; and decreased T-helper cell type 1 re
102                                              Neutrophil counts are a ubiquitous measure of inflammati
103                                        Blood neutrophil counts are determined by the differentiation
104                        In the United States, neutrophil counts are lower in black persons than in whi
105            For instance, both eosinophil and neutrophil counts are often increased in the airways of
106                                  Circulating neutrophil counts are positively regulated by CXCR2 sign
107 a lethal HSV-2 challenge model, we show that neutrophil counts are significantly reduced at 1 and 2 d
108                                              Neutrophil counts are slightly higher in Mexican-America
109 gen receptor for chemokines null variant for neutrophil count) are generalizable to WBC traits in His
110 d sham groups in the mean increment in blood neutrophil count at 8 hours (6.16 x 10(9)/L and 6.15 x 1
111  this was not the case for sex, indications, neutrophil count at initiation, and concomitant drug tre
112 solation of the ST1 severe strain and higher neutrophil counts at diagnosis in two unrelated large mu
113          Recovery occurred even though blood neutrophil counts began to rise 48 hours after the last
114 ose with the Duffy-null genotype (21%) had a neutrophil count below the threshold often used as a cut
115 OPD), with sputum and bronchoalveolar lavage neutrophil counts broadly correlating with disease sever
116 imulating factor (G-CSF), which can increase neutrophil counts but does not affect cytopenias other t
117 ing was associated with higher leukocyte and neutrophil counts but had a smaller effect among black a
118 rrent chemoradiotherapy exhibit a decline in neutrophil count, but the effects of neutrophils on radi
119      We conclude that IL-17A regulates blood neutrophil counts by inducing G-CSF production mainly in
120 nt inhibition or depletion, depletion of BAL neutrophil counts by more than 90% with the monoclonal a
121  obstructive lung disease, symptom duration, neutrophil count, C-reactive protein level, lactate dehy
122                                   Peripheral neutrophil count, C-reactive protein, and proinflammator
123 openia, lung infection with grade 4 absolute neutrophil count, colon infection with grade 4 absolute
124 vement in lung function and a fall in sputum neutrophil count compared with subjects who continued to
125 8) congenics had significantly lower exudate neutrophil counts compared with DA.
126 atients had higher cerebrospinal fluid (CSF) neutrophil counts compared with non-TBM-IRIS patients (m
127 venous-arterial difference in F-actin-rimmed neutrophil counts completely accounted for sequestration
128  standard white blood cell count or absolute neutrophil count, continue to be investigated, but these
129    Likewise, age, asthma duration, and blood neutrophil counts correctly predicted 64% of sputum neut
130                                              Neutrophil count correlated with the molecular degree of
131                              Median absolute neutrophil count decrease was 26%, and platelet decrease
132 mmon grade 3-4 adverse events reported were: neutrophil count decreased (26 [19%] of 134 in the chemo
133 nts vs none of 62 placebo-treated patients), neutrophil count decreased (five [8%] vs four [6%]), and
134 - 2.3 log10 cfu/ml (p<0.0001), respectively; neutrophil count decreased by 0.4 +/- 0.6 log10 cells/ml
135                                              Neutrophil counts decreased by 0.31 x 10(9)/L (0.09-0.54
136                                              Neutrophil counts decreased, hemoglobin and low- and hig
137 in decreased abscess size, lower circulating neutrophil counts, decreased anemia, and improved surviv
138 ude mice are neutropenic or have near-normal neutrophil counts, depending on the prevailing intestina
139                        Although pretreatment neutrophil count did not correlate with outcome, lower a
140                                Total WBC and neutrophil counts did not change.
141  and body temperature, and systemic absolute neutrophil counts did not differ between groups.
142 de significant loci were associated with low neutrophil counts during clozapine treatment.
143 PA syndrome revealed significantly increased neutrophil counts, erythrocyte sedimentation rates, and
144                         Reduced and elevated neutrophil counts, even within the normal range, are ass
145 eno levels, IgE levels, blood eosinophil and neutrophil counts, FEV1 percent predicted, and age are p
146  patients), anaemia (six, 2%), and decreased neutrophil count (five [2%]).
147                   Total white blood cell and neutrophil counts following CLP were both significantly
148 ustained leukocytosis, due to an increase in neutrophils counts, follows.
149 nic neutropenia is defined as a low absolute neutrophil count for >6 months.
150 y predictive of time to recovery of absolute neutrophil count for both the fourth and fifth courses (
151 owed us to maintain stable circulating HoxB8 neutrophil counts for several days.
152 lood samples were drawn for determination of neutrophil counts for up to 28 d after injection.
153                           Unlike the reduced neutrophil counts found in IL-17RA-deficient mice, neutr
154 ficant increase (two-fold, P < 0.05) in oral neutrophil counts found in patients with periodontitis c
155  electrocardiogram QT (five [2%]), decreased neutrophil count (four [2%]), anaemia, dyspnoea, hyponat
156 y outcomes included interleukin-8 levels and neutrophil counts from nasal wash, assessed at intake an
157 count, colon infection with grade 4 absolute neutrophil count, grade 4 changed mental status, and one
158        The median times to reach an absolute neutrophil count greater than 500/microL and platelet co
159  14 days, respectively, provided an absolute neutrophil count greater than 750x10(6)/L and a platelet
160     The median time to engraftment (absolute neutrophil count &gt; 500/mm(3) and platelets 50 000/muL) w
161 g/dose, days 1-5 and day 15 through absolute neutrophil count &gt; 500/uL]).
162 uate hepatic and renal function, an absolute neutrophil count &gt; or = 500/microL and a platelet count
163 0/microL, 27% (95% CI, 18% to 36%); absolute neutrophil count &gt;/=10000/microL, 18% (95% CI, 10% to 25
164 lower after CPX-351 (median days to absolute neutrophil count &gt;/=1000: 36 vs 32; platelets >100 000:
165 ts, mortality was associated with higher CSF neutrophil counts (hazard ratio [HR], 1.10 per 10% incre
166 GSTM1) and the use of statins, obesity, high neutrophil counts, higher blood pressure, and older age.
167 A [SAA], vascular endothelial growth factor, neutrophil count, IL-1alpha, E-selectin, intercellular a
168 n drug-related adverse event was decrease in neutrophil count in 33% and 70% of the ALCL165 and ALCL2
169 dependently associated with peripheral blood neutrophil count in contacts of patients diagnosed with
170 utropenia in seven (14%) patients, decreased neutrophil count in three (6%) patients, infections in f
171                                    Decreased neutrophil counts in alveolar air spaces, despite normal
172  decrease in total protein concentration and neutrophil counts in alveolar fluid through bronchoalveo
173 increased absolute lymphocyte, monocyte, and neutrophil counts in blood to normal without significant
174 we examined patient outcomes and circulating neutrophil counts in cervical cancer patients treated wi
175 hibition of BAL fluid total, eosinophil, and neutrophil counts in HDM mouse asthma in vivo, as compar
176 he primary endpoint was percentage change in neutrophil counts in induced sputum 6 h after lipopolysa
177 -17RA) are all required to maintain baseline neutrophil counts in mice.
178 ansferred Rag1(-/-) or WT thymocytes correct neutrophil counts in neutropenic nude mice.
179 straints by tracking the dynamics of patient neutrophil counts in response to therapy.
180 L-17F could compensate and maintain baseline neutrophil counts in the absence of IL-17A.
181 lso used to identify changes associated with neutrophil counts in the airway.Measurements and Main Re
182 acterial counts in the lungs, with decreased neutrophil counts in the bronchoalveolar lavage fluid as
183                                Bacterial and neutrophil counts in the lung were similar in control an
184 eutrophils and was associated with increased neutrophil counts in the sputum in response to ozone exp
185 nt to maintain the normal set point of blood neutrophil counts in vivo.
186 06; 39%) had increased monocyte, CD4(+), and neutrophil counts; increased C-reactive protein and inte
187             Stratification by eosinophil and neutrophil counts increases our understanding of asthma
188 cates that several BAL parameters, including neutrophil count, interleukin-8, alpha defensins and MMP
189 (4)/ml vs. 3.04 [2.82] x 10(4)/ml, p = 0.02) neutrophil counts, interleukin 8 (33.8 [189.8] vs. 16.9
190                                              Neutrophil counts less than 1.0 x 10(9) cells/L were obs
191 d as body temperature >/= 38.2 degrees C and neutrophil count &lt; 0.5 x 10(9)/L on the same day of the
192  de pointes and severe neutropenia (absolute neutrophil count &lt; or =500 cells/microL) were slightly h
193 erapy drug-induced agranulocytosis (absolute neutrophil count &lt;/=0.5 x 10(9)/L [</=500/muL]) and 5170
194 treatment discontinuation was required for a neutrophil count &lt;0.5 x 10(9) cells/L.
195 ly 242 (8%) sustained an infection and had a neutrophil count &lt;0.75 x 10(9) cells/L at any time while
196            Dose reduction was required for a neutrophil count &lt;0.75 x 10(9) cells/L, and treatment di
197 estigator; 648 (21%) patients had at least 1 neutrophil count &lt;0.75 x 10(9) cells/L, but only 242 (8%
198               The prevalence of neutropenia (neutrophil count &lt;1.5 x 10(9) cells/L) was 4.5% among bl
199 ients with concomitant neutropenia (absolute neutrophil count &lt;1000 cells/microL) and abdominal pain
200 d was used to identify neutropenia (absolute neutrophil count &lt;1500 cells/mm(3)) at the time of port
201                 A reduction in ascitic fluid neutrophil count &lt;25% of pretreatment value was consider
202 solidation on chest radiograph, and absolute neutrophil count &lt;5 x 109/L at presentation, which had a
203 bjects were those with neutropenia (absolute neutrophil count &lt;500/muL) and proven/probable/presumed
204                                     Absolute neutrophil counts &lt;1000/uL were noted in 12/106 (11%) ev
205 sfusion of RBCs stored for 10 days increased neutrophil counts, macrophage inflammatory protein-2 (MI
206                         Benign reductions in neutrophil counts may be more common at certain ages and
207                  Nine patients had increased neutrophil counts (median increase, 1350 per cubic milli
208 hereas age, FEV1 percent predicted, or blood neutrophil counts might predict sputum neutrophil percen
209 platelet count more than 75,000 x 10(6)/L, a neutrophil count more than 1,000 x 10(6)/L, and a creati
210  7 days from prior corticosteroids; absolute neutrophil count more than 1,500/microL (500/microL if d
211                  The median time to absolute neutrophil count more than 500/microL after ASCR was 13
212 -) mice, resulting in very high platelet and neutrophil counts, more advanced myelofibrosis, and redu
213                                     Absolute neutrophil counts, motor function, Morris water maze per
214 ere decreased lymphocyte (n=3) and decreased neutrophil count (n=2); and grade 4 anaemia was reported
215                             However, neither neutrophil count, Na(+) concentration, nor Na(+) : K(+)
216 s significantly associated with the absolute neutrophil count nadir (7/7 < 6/7 < 6/6, P =.02).
217                     The time to the absolute neutrophil count nadir was shorter for the (90)Y-ibritum
218 e were significant predictors of ln(absolute neutrophil count nadir; r(2) = 0.51).
219 t 3 experienced significantly lower absolute neutrophil count nadirs than did younger groups (P = .02
220                                    BAL fluid neutrophil counts negatively and positively correlated w
221 ion decline was associated with increases in neutrophil counts, neutrophil elastase, and IL-1beta and
222 xpression in mixed chimeric mice depended on neutrophil count, not iNKT cell genotype.
223                 The median time to achieve a neutrophil count of 0.5 x 10(9)/liter was 14 days (range
224 iatric patients with cancer with an absolute neutrophil count of 500/uL or greater is unclear.
225 .22; 95% CI, 0.17-0.30), as does an absolute neutrophil count of 6750/microL or lower (LR, 0.06; 95%
226  not significantly different at the absolute neutrophil count of less than 0.5 x 109 cells/L (P = 0.5
227 nts treated with tofacitinib had an absolute neutrophil count of less than 1500.
228 ith greater risk even among individuals with neutrophil counts of 3 to 4 versus 2 to 3 x 10(9)/l.
229 ence of enzyme replacement therapy, absolute neutrophil counts of patients with ADA deficiency vary i
230 atients with acute coronary syndrome, higher neutrophil count on admission and after revascularizatio
231 itive patients in the Oxford hospitals, mean neutrophil counts on diagnosis increased from 2003, peak
232 neutropenia are cyclic neutropenia, in which neutrophil counts oscillate with a 21-day frequency, and
233   In the immediate-initiation group, average neutrophil count over follow-up was 321 cells per muL hi
234 eroxia did not further increase chemokine or neutrophil counts over normoxia.
235 ite cell count was associated with increased neutrophil count (P < 0.001) and low-grade peritumoral i
236 s A and B), age (P < 0.01), mGPS (P < 0.01), neutrophil count (P < 0.05), and Klintrup criteria (P <
237 1), temperature (P<.05), pulse rate (P<.05), neutrophil count (P<.05), tumor necrosis factor- alpha l
238  an 84% decrease in the circulating absolute neutrophil count (P<0.001) before elastase perfusion.
239 ein concentration (p=0.0003) and in absolute neutrophil count (p=0.024) at 24 h after treatment than
240 tis and total leukocytes counts (p < 0.001), neutrophil counts (P <0.001) and hematocrit (HCT) levels
241 F showed a significant trend toward elevated neutrophil counts (P = 0.002) in BALF and increased mort
242  adjusted homocysteine levels (P<0.0001) and neutrophil counts (P<0.0001) than the no-progression gro
243 ere anaemia (nine [15%] of 62) and decreased neutrophil count, pancytopenia, and thrombocytopenia (tw
244 decreases from baseline in hemoglobin level, neutrophil count, platelet count, and weight compared wi
245       Maximum decreases in hemoglobin level, neutrophil count, platelet count, and weight during ther
246 n between grade 1 and grade 3 severity), CSF neutrophil count (r = 0.364 and P < .0001), and cytokine
247 lar lavage IL-8 (R(2) = 0.20, P = 0.004) and neutrophil count (R(2) = 0.21, P = 0.001).
248 ses intravenous immunoglobulins allowed full neutrophil count recovery.
249                   (18)F-FLT changes preceded neutrophil count reductions.
250 sitive women and corresponded with increased neutrophil counts relative to BV-negative women.
251 re and treatment duration, only decreases in neutrophil count remained associated with virologic resp
252                                              Neutrophil counts remained unchanged.
253 so showed decreased lymphocyte and increased neutrophil counts, respectively.
254                                              Neutrophil counts returned to normal after cessation of
255                         White blood cell and neutrophil counts, serum or plasma levels of fibrinogen,
256                                              Neutrophil counts should be carefully evaluated in prema
257 during the first 11 days (difference between neutrophil count slopes 0.34 x 10(9)/L/day; 95% CI 0.12-
258  risk factors and acute conditions affecting neutrophil counts (such as infections and cancer).
259 ent, group A had higher median leukocyte and neutrophil counts than group B (P < .001).
260 te its strong association with lower average neutrophil counts, the Duffy-null genotype was not assoc
261 re anaemia (seven [12%] of 59) and decreased neutrophil count (three [5%]); an additional six events
262      The median time to recovery of absolute neutrophil count to >/= 500 cells/microL was 11 days (ra
263 ic patients as 100% increase in the absolute neutrophil count to more than 5 x 10(8) cells per L, and
264 a, decreased lymphocyte count, and decreased neutrophil count (two [3%] each).
265 nt, AIDS, antiretroviral therapy, cirrhosis, neutrophil count, type of peg-IFN, and granulocyte colon
266 tality and biomarkers of inflammation (blood neutrophil count, urea, and creatinine concentrations) c
267              Androgen supplement can restore neutrophil counts via stabilizing AR in castrated mice,
268               The median presenting absolute neutrophil count was 100/muL.
269 arrow was enhanced, and the peripheral blood neutrophil count was also substantially elevated in thes
270           Lymphopenia was universal, but the neutrophil count was generally not affected.
271  receive filgrastim (G-CSF) from day 0 until neutrophil count was greater than 0.5 x 10(9)/L (or for
272     Compared with control, systemic absolute neutrophil count was increased more than ten-fold in gra
273 timulating factor was used when the absolute neutrophil count was less than 1,000/microL.
274  one from infection (although their absolute neutrophil count was normal), which was definitely relat
275                                 Time-updated neutrophil count was not associated with severe bacteria
276                               Reduced sputum neutrophil count was observed among the 122 patients exa
277               The total white blood cell and neutrophil count was reduced from 7610 to 6980 cells/mic
278            Nadir lymphocyte count, not nadir neutrophil count, was independently associated with mode
279            This activation, like the drop in neutrophil count, was transient.
280 ctin, TNFalpha, and MPO and peripheral blood neutrophil count were higher in patients with RA than co
281    Median change in interleukin-8 levels and neutrophil counts were also not statistically significan
282                                        Blood neutrophil counts were elevated (4.8x), whereas lymphocy
283 ients of WT stem cells; however, circulating neutrophil counts were higher only in Rac2(-/-) recipien
284 s with the strongest relationships to sputum neutrophil counts were IL1R1 (standardized regression co
285 that 24 hours after infection macrophage and neutrophil counts were lower in the cornea of Lum(-/-) m
286 phil counts found in IL-17RA-deficient mice, neutrophil counts were mildly increased in IL-17A-defici
287 acterial loads, cytokine concentrations, and neutrophil counts were more likely to subsequently exper
288 differences in hemoglobin concentrations and neutrophil counts were no longer significant, whereas di
289                   Lung edema, cytokines, and neutrophil counts were reduced after EVLP and further, s
290                         Bacterial counts and neutrophil counts were significantly higher in the circu
291        As reported in the literature, sputum neutrophil counts were significantly increased in subjec
292 tney U test; P = .012) and day-14 (P = .025) neutrophil counts were significantly lower in patients r
293       Age, FEV1 percent predicted, and blood neutrophil counts were similarly unsatisfactory for the
294                                              Neutrophil counts were strongly associated with the inci
295 of depression, and female sex, but not nadir neutrophil count, were associated with moderate, severe,
296 end on the impact of treatment on peripheral neutrophil count, which has the potential to serve as an
297 ef review summarizes the regulation of blood neutrophil counts, which is in part controlled by G-CSF,
298 vel of 1,5-anhydroglucitol restored a normal neutrophil count, while administration of 1,5-anhydroglu
299 stigated associations of clinically recorded neutrophil counts with initial presentation for a range
300  to dosage-related decreases in the absolute neutrophil count, with a median decrease of 38% in the 4

 
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