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1 d-type (WT), primarily driven by an elevated neutrophil count.
2 n alanine transaminase level or decreases in neutrophil count.
3 vels than wild-type mice, presented elevated neutrophil count.
4 uded hyperglycaemia and fever with decreased neutrophil count.
5 s in CD4 cell count, but not by increases in neutrophil count.
6 in cholesterol levels and with reductions in neutrophil counts.
7 protein cholesterol levels and reductions in neutrophil counts.
8 or treatment normalized CXCR4 expression and neutrophil counts.
9 of cotransplanted Tregs exhibited diminished neutrophil counts.
10 t produce mature T cells but maintain normal neutrophil counts.
11 s might contribute to regulating circulating neutrophil counts.
12 had the greatest effects on regulating blood neutrophil counts.
13 h IL-23, but not IL-12, restored circulating neutrophil counts.
14 l counts, and combined sputum eosinophil and neutrophil counts.
15 in the ischemic brain along with the reduced neutrophil counts.
16 these 52 molecules correlated with BAL fluid neutrophil counts.
17 in eosinophil counts and 64% of variation in neutrophil counts.
18 also decreased platelet counts and increased neutrophil counts.
19 lls/L, p = 9.22 x 10(-95)), particularly for neutrophil count (0.174 x [0.158, 0.190]10(9) cells/L, p
20 ts (0.16 x 10(9) cells/L; P = 0.014), higher neutrophil counts (0.11 x 10(9) cells/L; P = 0.026), and
21 nce, 0.89 x 10(9) cells/L; P < 0.001), lower neutrophil counts (0.83 x 10(9) cells/L; P < 0.001), and
22 bin concentration 9 g/dL or higher, absolute neutrophil count 1 x 10(3) cells per mm(3) or higher, an
23 ase 9.6 g/L, 95% CI 7.6-11.6 g/L; p<0.0001), neutrophil counts (1.1x10(9)/L, 0.7-1.5x10(9)/L; p<0.000
25 ression of 2 of the 3 blood counts: absolute neutrophil count 1200/mm3 or less, platelet count 70,000
26 d grade 3 or 4 adverse events were decreased neutrophil count (14 [39%] of 36 patients in the gemcita
27 common grade 3 or 4 adverse events were low neutrophil count (15 [11%] preoperatively in the chemoth
28 1 [16%] in the CRT only group), low absolute neutrophil count (15 [12%] vs 24 [19%]), fatigue (26 [20
29 in the safety population (n=57) were reduced neutrophil count (15 [26%]), hypophosphatemia (11 [19%])
30 mmon grade 3-4 adverse events were decreased neutrophil count (210 [37%] in the cetuximab group vs 15
31 l neuropathy (25 [6%] vs 12 [3%]), decreased neutrophil count (22 [5%] vs 16 [4%]), and fatigue (17 [
34 1 [11%]), dyspnoea (29 [10%]), and decreased neutrophil count (28 [10%]), and with pemetrexed alone w
35 , leucopenia (22 [7%] vs 17 [6%]), decreased neutrophil count (31 [10%] vs 41 [13%]), and decreased w
36 the fixed-dose combination group), decreased neutrophil count (31 [12%] vs 27 [11%]), febrile neutrop
37 ients), leucopenia (42 [16%]), and decreased neutrophil count (40 [15%]); in the placebo plus paclita
38 te blood cell count (11600/microL), absolute neutrophil count (4100/microL), and platelet count (362
40 ide levels (38 vs 27 ppb, P = .02) and blood neutrophil counts (5.3 vs 4.0 10(9)/L, P </= .001) and s
43 r [2%]) in the nivolumab group and decreased neutrophil count (59 [28%]) in the chemotherapy group.
45 x 1000 cells/muL [IQR, 7.5, 12.9]; P = .01), neutrophil count (6.7 x 1000 cells/muL [IQR, 2.8, 9.7] v
46 t was correlated to the CD66b microparticles/neutrophil count, a surrogate of neutrophil activation a
47 ether age, FEV1 percent predicted, and blood neutrophil counts accurately predict sputum neutrophil p
48 ed granulocyte-colony stimulating factor and neutrophil counts/activity postsuboptimal immunization.
49 d not correlate with outcome, lower absolute neutrophil count after starting concurrent chemoradiothe
50 GP generation correlates closely with airway neutrophil counts after administration of proteases in v
52 For two traits, lipoprotein(a) levels and neutrophil count, aggregate tests of low-frequency and r
56 responses, we found significantly different neutrophil counts among treatments; however, phagocytic
57 bones, a markedly enlarged spleen, elevated neutrophil counts, an enlarged heart, and behavioral hyp
58 bserved dose-dependent increases in absolute neutrophil count (ANC) and absolute lymphocyte count (AL
59 caused a dose-dependent decrease in absolute neutrophil count (ANC) and reduced inflammatory biomarke
61 ine neutropenia, 82% of those whose absolute neutrophil count (ANC) decreased by > or = 75% achieved
62 n therapy for inadequate counts: an absolute neutrophil count (ANC) more than 1,000/microL and platel
63 ivariate and multivariate models of absolute neutrophil count (ANC) nadir and pharmacokinetic paramet
64 teria: positive CSF Gram stain, CSF absolute neutrophil count (ANC) of at least 1000 cells/microL, CS
65 , but achieved less than the median absolute neutrophil count (ANC) response (ANC < 2.188 x 10(9)/L [
68 blood with normalization of counts (absolute neutrophil count [ANC]> 1.5 x 10(9)/L, hemoglobin [Hgb]
69 mation (alkaline phosphatase [ALP], absolute neutrophil count [ANC], ferritin [adjusted for iron stat
70 her with laboratory tests (white cell count, neutrophil count and C-reactive protein), and the timing
74 ized by a small increase of lymphocyte B and neutrophil counts and a transient drop of total lymphocy
75 of anakinra compared with placebo on airway neutrophil counts and airway proinflammatory cytokine le
77 y response in the lung as shown by increased neutrophil counts and elevated cytokine and chemokine co
78 an cluster T1, with no differences in sputum neutrophil counts and exhaled nitric oxide and serum IgE
79 , exogenous MCP-1 dose dependently increased neutrophil counts and G-CSF concentrations in the blood.
80 rity was correlated with total leukocyte and neutrophil counts and HCT (P <0.001, P <0.001 and P = 0.
81 iglyceride, VLDL, total WBC, lymphocyte, and neutrophil counts and increase in hsCRP, total cholester
82 white blood cell, lymphocyte, monocyte, and neutrophil counts and increased adiponectin levels witho
84 sociated with reduced bronchoalveolar lavage neutrophil counts and interleukin-6 and macrophage infla
86 GER expression correlated with higher sputum neutrophil counts and more severe AHR in COPD patients.
87 ociated TBM and is characterized by high CSF neutrophil counts and Mycobacterium tuberculosis culture
88 clude that efforts should focus on improving neutrophil counts and on treating the frequent and serio
89 in alveolar air spaces, despite normal blood neutrophil counts and survival of emigrated neutrophils,
90 Blood glucose level, total leukocyte count, neutrophil count, and leukocyte labeling efficiency vari
91 (LDH) level, ferritin level, d-dimer level, neutrophil count, and neutrophil-to-lymphocyte ratio wer
92 racy of the white blood cell count, absolute neutrophil count, and platelet count at commonly used th
93 ve protein, white blood cell count, absolute neutrophil count, and procalcitonin (PCT), specifically
94 ve protein, white blood cell count, absolute neutrophil count, and procalcitonin (PCT), specifically
96 r peripheral blood eosinophil counts, sputum neutrophil counts, and combined sputum eosinophil and ne
97 older age, lower serum albumin, higher blood neutrophil counts, and greater prevalence of chronic obs
99 tween quintiles of lymphocyte, monocyte, and neutrophil counts, and increased systolic BP, diastolic
101 CXCL10, and interleukin 10 levels; increased neutrophil counts; and decreased T-helper cell type 1 re
107 a lethal HSV-2 challenge model, we show that neutrophil counts are significantly reduced at 1 and 2 d
109 gen receptor for chemokines null variant for neutrophil count) are generalizable to WBC traits in His
110 d sham groups in the mean increment in blood neutrophil count at 8 hours (6.16 x 10(9)/L and 6.15 x 1
111 this was not the case for sex, indications, neutrophil count at initiation, and concomitant drug tre
112 solation of the ST1 severe strain and higher neutrophil counts at diagnosis in two unrelated large mu
114 ose with the Duffy-null genotype (21%) had a neutrophil count below the threshold often used as a cut
115 OPD), with sputum and bronchoalveolar lavage neutrophil counts broadly correlating with disease sever
116 imulating factor (G-CSF), which can increase neutrophil counts but does not affect cytopenias other t
117 ing was associated with higher leukocyte and neutrophil counts but had a smaller effect among black a
118 rrent chemoradiotherapy exhibit a decline in neutrophil count, but the effects of neutrophils on radi
119 We conclude that IL-17A regulates blood neutrophil counts by inducing G-CSF production mainly in
120 nt inhibition or depletion, depletion of BAL neutrophil counts by more than 90% with the monoclonal a
121 obstructive lung disease, symptom duration, neutrophil count, C-reactive protein level, lactate dehy
123 openia, lung infection with grade 4 absolute neutrophil count, colon infection with grade 4 absolute
124 vement in lung function and a fall in sputum neutrophil count compared with subjects who continued to
126 atients had higher cerebrospinal fluid (CSF) neutrophil counts compared with non-TBM-IRIS patients (m
127 venous-arterial difference in F-actin-rimmed neutrophil counts completely accounted for sequestration
128 standard white blood cell count or absolute neutrophil count, continue to be investigated, but these
129 Likewise, age, asthma duration, and blood neutrophil counts correctly predicted 64% of sputum neut
132 mmon grade 3-4 adverse events reported were: neutrophil count decreased (26 [19%] of 134 in the chemo
133 nts vs none of 62 placebo-treated patients), neutrophil count decreased (five [8%] vs four [6%]), and
134 - 2.3 log10 cfu/ml (p<0.0001), respectively; neutrophil count decreased by 0.4 +/- 0.6 log10 cells/ml
137 in decreased abscess size, lower circulating neutrophil counts, decreased anemia, and improved surviv
138 ude mice are neutropenic or have near-normal neutrophil counts, depending on the prevailing intestina
143 PA syndrome revealed significantly increased neutrophil counts, erythrocyte sedimentation rates, and
145 eno levels, IgE levels, blood eosinophil and neutrophil counts, FEV1 percent predicted, and age are p
150 y predictive of time to recovery of absolute neutrophil count for both the fourth and fifth courses (
154 ficant increase (two-fold, P < 0.05) in oral neutrophil counts found in patients with periodontitis c
155 electrocardiogram QT (five [2%]), decreased neutrophil count (four [2%]), anaemia, dyspnoea, hyponat
156 y outcomes included interleukin-8 levels and neutrophil counts from nasal wash, assessed at intake an
157 count, colon infection with grade 4 absolute neutrophil count, grade 4 changed mental status, and one
159 14 days, respectively, provided an absolute neutrophil count greater than 750x10(6)/L and a platelet
160 The median time to engraftment (absolute neutrophil count > 500/mm(3) and platelets 50 000/muL) w
162 uate hepatic and renal function, an absolute neutrophil count > or = 500/microL and a platelet count
163 0/microL, 27% (95% CI, 18% to 36%); absolute neutrophil count >/=10000/microL, 18% (95% CI, 10% to 25
164 lower after CPX-351 (median days to absolute neutrophil count >/=1000: 36 vs 32; platelets >100 000:
165 ts, mortality was associated with higher CSF neutrophil counts (hazard ratio [HR], 1.10 per 10% incre
166 GSTM1) and the use of statins, obesity, high neutrophil counts, higher blood pressure, and older age.
167 A [SAA], vascular endothelial growth factor, neutrophil count, IL-1alpha, E-selectin, intercellular a
168 n drug-related adverse event was decrease in neutrophil count in 33% and 70% of the ALCL165 and ALCL2
169 dependently associated with peripheral blood neutrophil count in contacts of patients diagnosed with
170 utropenia in seven (14%) patients, decreased neutrophil count in three (6%) patients, infections in f
172 decrease in total protein concentration and neutrophil counts in alveolar fluid through bronchoalveo
173 increased absolute lymphocyte, monocyte, and neutrophil counts in blood to normal without significant
174 we examined patient outcomes and circulating neutrophil counts in cervical cancer patients treated wi
175 hibition of BAL fluid total, eosinophil, and neutrophil counts in HDM mouse asthma in vivo, as compar
176 he primary endpoint was percentage change in neutrophil counts in induced sputum 6 h after lipopolysa
181 lso used to identify changes associated with neutrophil counts in the airway.Measurements and Main Re
182 acterial counts in the lungs, with decreased neutrophil counts in the bronchoalveolar lavage fluid as
184 eutrophils and was associated with increased neutrophil counts in the sputum in response to ozone exp
186 06; 39%) had increased monocyte, CD4(+), and neutrophil counts; increased C-reactive protein and inte
188 cates that several BAL parameters, including neutrophil count, interleukin-8, alpha defensins and MMP
189 (4)/ml vs. 3.04 [2.82] x 10(4)/ml, p = 0.02) neutrophil counts, interleukin 8 (33.8 [189.8] vs. 16.9
191 d as body temperature >/= 38.2 degrees C and neutrophil count < 0.5 x 10(9)/L on the same day of the
192 de pointes and severe neutropenia (absolute neutrophil count < or =500 cells/microL) were slightly h
193 erapy drug-induced agranulocytosis (absolute neutrophil count </=0.5 x 10(9)/L [</=500/muL]) and 5170
195 ly 242 (8%) sustained an infection and had a neutrophil count <0.75 x 10(9) cells/L at any time while
197 estigator; 648 (21%) patients had at least 1 neutrophil count <0.75 x 10(9) cells/L, but only 242 (8%
199 ients with concomitant neutropenia (absolute neutrophil count <1000 cells/microL) and abdominal pain
200 d was used to identify neutropenia (absolute neutrophil count <1500 cells/mm(3)) at the time of port
202 solidation on chest radiograph, and absolute neutrophil count <5 x 109/L at presentation, which had a
203 bjects were those with neutropenia (absolute neutrophil count <500/muL) and proven/probable/presumed
205 sfusion of RBCs stored for 10 days increased neutrophil counts, macrophage inflammatory protein-2 (MI
208 hereas age, FEV1 percent predicted, or blood neutrophil counts might predict sputum neutrophil percen
209 platelet count more than 75,000 x 10(6)/L, a neutrophil count more than 1,000 x 10(6)/L, and a creati
210 7 days from prior corticosteroids; absolute neutrophil count more than 1,500/microL (500/microL if d
212 -) mice, resulting in very high platelet and neutrophil counts, more advanced myelofibrosis, and redu
214 ere decreased lymphocyte (n=3) and decreased neutrophil count (n=2); and grade 4 anaemia was reported
219 t 3 experienced significantly lower absolute neutrophil count nadirs than did younger groups (P = .02
221 ion decline was associated with increases in neutrophil counts, neutrophil elastase, and IL-1beta and
225 .22; 95% CI, 0.17-0.30), as does an absolute neutrophil count of 6750/microL or lower (LR, 0.06; 95%
226 not significantly different at the absolute neutrophil count of less than 0.5 x 109 cells/L (P = 0.5
228 ith greater risk even among individuals with neutrophil counts of 3 to 4 versus 2 to 3 x 10(9)/l.
229 ence of enzyme replacement therapy, absolute neutrophil counts of patients with ADA deficiency vary i
230 atients with acute coronary syndrome, higher neutrophil count on admission and after revascularizatio
231 itive patients in the Oxford hospitals, mean neutrophil counts on diagnosis increased from 2003, peak
232 neutropenia are cyclic neutropenia, in which neutrophil counts oscillate with a 21-day frequency, and
233 In the immediate-initiation group, average neutrophil count over follow-up was 321 cells per muL hi
235 ite cell count was associated with increased neutrophil count (P < 0.001) and low-grade peritumoral i
236 s A and B), age (P < 0.01), mGPS (P < 0.01), neutrophil count (P < 0.05), and Klintrup criteria (P <
237 1), temperature (P<.05), pulse rate (P<.05), neutrophil count (P<.05), tumor necrosis factor- alpha l
238 an 84% decrease in the circulating absolute neutrophil count (P<0.001) before elastase perfusion.
239 ein concentration (p=0.0003) and in absolute neutrophil count (p=0.024) at 24 h after treatment than
240 tis and total leukocytes counts (p < 0.001), neutrophil counts (P <0.001) and hematocrit (HCT) levels
241 F showed a significant trend toward elevated neutrophil counts (P = 0.002) in BALF and increased mort
242 adjusted homocysteine levels (P<0.0001) and neutrophil counts (P<0.0001) than the no-progression gro
243 ere anaemia (nine [15%] of 62) and decreased neutrophil count, pancytopenia, and thrombocytopenia (tw
244 decreases from baseline in hemoglobin level, neutrophil count, platelet count, and weight compared wi
246 n between grade 1 and grade 3 severity), CSF neutrophil count (r = 0.364 and P < .0001), and cytokine
251 re and treatment duration, only decreases in neutrophil count remained associated with virologic resp
257 during the first 11 days (difference between neutrophil count slopes 0.34 x 10(9)/L/day; 95% CI 0.12-
260 te its strong association with lower average neutrophil counts, the Duffy-null genotype was not assoc
261 re anaemia (seven [12%] of 59) and decreased neutrophil count (three [5%]); an additional six events
262 The median time to recovery of absolute neutrophil count to >/= 500 cells/microL was 11 days (ra
263 ic patients as 100% increase in the absolute neutrophil count to more than 5 x 10(8) cells per L, and
265 nt, AIDS, antiretroviral therapy, cirrhosis, neutrophil count, type of peg-IFN, and granulocyte colon
266 tality and biomarkers of inflammation (blood neutrophil count, urea, and creatinine concentrations) c
269 arrow was enhanced, and the peripheral blood neutrophil count was also substantially elevated in thes
271 receive filgrastim (G-CSF) from day 0 until neutrophil count was greater than 0.5 x 10(9)/L (or for
272 Compared with control, systemic absolute neutrophil count was increased more than ten-fold in gra
274 one from infection (although their absolute neutrophil count was normal), which was definitely relat
280 ctin, TNFalpha, and MPO and peripheral blood neutrophil count were higher in patients with RA than co
281 Median change in interleukin-8 levels and neutrophil counts were also not statistically significan
283 ients of WT stem cells; however, circulating neutrophil counts were higher only in Rac2(-/-) recipien
284 s with the strongest relationships to sputum neutrophil counts were IL1R1 (standardized regression co
285 that 24 hours after infection macrophage and neutrophil counts were lower in the cornea of Lum(-/-) m
286 phil counts found in IL-17RA-deficient mice, neutrophil counts were mildly increased in IL-17A-defici
287 acterial loads, cytokine concentrations, and neutrophil counts were more likely to subsequently exper
288 differences in hemoglobin concentrations and neutrophil counts were no longer significant, whereas di
292 tney U test; P = .012) and day-14 (P = .025) neutrophil counts were significantly lower in patients r
295 of depression, and female sex, but not nadir neutrophil count, were associated with moderate, severe,
296 end on the impact of treatment on peripheral neutrophil count, which has the potential to serve as an
297 ef review summarizes the regulation of blood neutrophil counts, which is in part controlled by G-CSF,
298 vel of 1,5-anhydroglucitol restored a normal neutrophil count, while administration of 1,5-anhydroglu
299 stigated associations of clinically recorded neutrophil counts with initial presentation for a range
300 to dosage-related decreases in the absolute neutrophil count, with a median decrease of 38% in the 4