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1 ered in disease models, in part, by reducing neutrophil infiltration.
2 ere pancreatic injury including necrosis and neutrophil infiltration.
3 to worsened intestinal damage and increased neutrophil infiltration.
4 of disease, including AHR and macrophage and neutrophil infiltration.
5 l catheterization, rarely showed evidence of neutrophil infiltration.
6 uced apoptosis, reactive oxygen species, and neutrophil infiltration.
7 ammatory disorders that are characterized by neutrophil infiltration.
8 late phase that is associated with enhanced neutrophil infiltration.
9 ing both proinflammatory cytokine levels and neutrophil infiltration.
10 of increased vascular permeability and early neutrophil infiltration.
11 ce of membrane attack complex activation and neutrophil infiltration.
12 d CD8(+) T cells, and reduces macrophage and neutrophil infiltration.
13 dendritic cells and significantly suppressed neutrophil infiltration.
14 cted animals through 24 h, as was peritoneal neutrophil infiltration.
15 tion model, where they caused a reduction in neutrophil infiltration.
16 nd systemically, but was not associated with neutrophil infiltration.
17 mor foci due to tumor development-associated neutrophil infiltration.
18 ntitumor immune response by inducing massive neutrophil infiltration.
19 levels, reduced hepatic necrosis, and lower neutrophil infiltration.
20 e of epithelial erosions, corneal edema, and neutrophil infiltration.
21 n adipose tissue endothelial cells to induce neutrophil infiltration.
22 eroxidase (MPO) assay was used to quantitate neutrophil infiltration.
23 elated with activation of mTOR signaling and neutrophil infiltration.
24 as interleukin-6 and bronchoalveolar lavage neutrophil infiltration.
25 h Th17-induced pathology, nor did it prevent neutrophil infiltration.
26 id TM may be caused by the early increase in neutrophil infiltration.
27 ogical findings of epidermal hyperplasia and neutrophil infiltration.
28 o model of acute lung injury associated with neutrophil infiltration.
29 ed in preterm birth and are characterized by neutrophil infiltration.
30 chemokine secretion from lung, but impairing neutrophil infiltration.
31 red wine significantly reduced post-ischemic neutrophil infiltration.
32 through a mechanism that involved increased neutrophil infiltration.
33 epidermal hyperproliferation, and increased neutrophil infiltration.
34 interleukin-1beta (IL-1beta), which sustains neutrophil infiltration.
35 s for use in inflammatory diseases involving neutrophil infiltration.
36 neutrophil [7/4] antigen, indicating reduced neutrophil infiltration.
37 tion, decreased epidermal proliferation, and neutrophil infiltration.
38 s, which did not affect cytokine release and neutrophil infiltration.
40 elayed corneal healing (13.2%) and increased neutrophil infiltration (54.1%) by day 4 in WT mice, whe
41 mokine ligand 1 (CXCL1), a key chemokine for neutrophil infiltration (a hallmark of NASH), is highly
42 onal location of the bacterial infection and neutrophil infiltration, a diffuse optical tomography re
43 Unexpectedly, myocardial infarct size and neutrophil infiltration/activity 2 days after AMI were a
48 lts from airway damage, mucosal dysfunction, neutrophil infiltration, airway coagulopathy with cast f
50 kin inflammation characterized by T cell and neutrophil infiltration and a Th17-biased cytokine respo
53 d persistent increased ACE2 activity reduces neutrophil infiltration and compromises host defense, le
56 ong inflammatory responses in the lungs with neutrophil infiltration and edema, further confirmed as
57 found that consumption of an HFD resulted in neutrophil infiltration and enhanced MPO expression and
59 ented the onset of meningitis and suppressed neutrophil infiltration and glial cell migration in the
60 inoculum (1 x 10(7) CFU) of BG2 caused less neutrophil infiltration and greater burdens in peritonea
61 wed significantly higher levels of pulmonary neutrophil infiltration and higher amounts of pulmonary
62 nd responded to the infection with extensive neutrophil infiltration and histopathological changes in
63 of either IL-17A or IL-22 reduced T cell and neutrophil infiltration and host defense peptide elabora
64 aneous periodontitis that featured excessive neutrophil infiltration and IL-17 expression; disease wa
65 sive pulmonary inflammation characterized by neutrophil infiltration and IL-17 response with increase
66 NDV-3 induced increases in CD3+ T-cell and neutrophil infiltration and IL-17A, IL-22, and host defe
67 the adult epicardium reduced injury-induced neutrophil infiltration and improved cardiac function.
68 trans expression of SsE(M28) in GAS reduced neutrophil infiltration and increased skin invasion in s
70 -selectin plays an important role in hepatic neutrophil infiltration and injury induced by chronic-bi
71 ive effect on anti-MHC induced IL-8-mediated neutrophil infiltration and innate immune responses that
72 oth acute lung injury groups as evidenced by neutrophil infiltration and levels of cytokines in bronc
75 s involved, we discovered that ACE2 inhibits neutrophil infiltration and lung inflammation by limitin
76 These effects were associated with reduced neutrophil infiltration and macrophage accumulation and
77 atment significantly reduced lymphocytic and neutrophil infiltration and mast cells degranulation (p
78 ced thrombus burden, with significantly less neutrophil infiltration and more proresolving monocytes
79 ly larger infarcts concordant with increased neutrophil infiltration and myocyte apoptosis compared w
80 etwork of inflammatory mediators that induce neutrophil infiltration and NET formation responsible fo
81 and exacerbated stroke injury by aggravating neutrophil infiltration and neurodegeneration in vivo.
82 k disassembly was accompanied by decrease in neutrophil infiltration and neutrophil extracellular tra
84 lar surface tissues, including elevated Gr1+ neutrophil infiltration and proinflammatory cytokines/ch
85 with a 54% decrease (p < 0.05) in pulmonary neutrophil infiltration and reduced alveolar wall thicke
86 also resolved E. coli infections by limiting neutrophil infiltration and stimulating bacterial phagoc
87 ate the importance of MFG-E8 in ameliorating neutrophil infiltration and suggest MFG-E8 as a novel th
88 Administration of DNase I to mice reduced neutrophil infiltration and tissue damage in the inflame
91 ibrosis, 0.86 for bilirubinostasis, 0.60 for neutrophil infiltration, and 0.46 for megamitochondria.
92 icant reduction in MMP-9/-3, less peripheral neutrophil infiltration, and a preservation of tight jun
93 less intense cytokine responses, diminished neutrophil infiltration, and accelerated uroepithelial r
94 o and triggered multiple cytokine responses, neutrophil infiltration, and acute inflammatory histopat
95 ED1 monocytes and macrophages, and pulmonary neutrophil infiltration, and also enhanced the accumulat
96 NPCT; reduced lung inflammation and injury, neutrophil infiltration, and bacterial invasion; and imp
99 eduction of microvascular hyperpermeability, neutrophil infiltration, and endothelial adhesion molecu
100 molecule-1 expression, reduced intima/media neutrophil infiltration, and increased DHCR24 and HO-1 m
101 exacerbated hepatocyte necrosis, apoptosis, neutrophil infiltration, and inflammatory cytokine gener
102 farct, demyelination, P-selectin expression, neutrophil infiltration, and microthrombi formation.
103 ed significantly higher cytokine production, neutrophil infiltration, and more rapid fungal clearance
104 xhibited significantly reduced infarct size, neutrophil infiltration, and myocyte apoptosis compared
106 otin-dUTP nick end-labeling)-positive cells, neutrophil infiltration, and proinflammatory mRNAs as we
107 layed significantly attenuated inflammation, neutrophil infiltration, and reduced severity of oviduct
108 rine model of sterile kidney injury, reduced neutrophil infiltration, and serum creatinine levels wer
109 ansaminases, histological signs of necrosis, neutrophil infiltration, and serum levels of interleukin
110 opic signs of colon inflammation, macrophage/neutrophil infiltration, and the expression of proinflam
111 an increase in plasma nucleosomes, abundant neutrophil infiltration, and the presence of citrullinat
113 assessing necrosis, platelet deposition, and neutrophil infiltration, and the status of steatosis aft
114 oviducts and uterine horns exhibited reduced neutrophil infiltration, and this reduction persisted af
115 and sepsis-induced ALI abolished lung edema, neutrophil infiltration, and tissue damage, thereby reve
116 istones, suppressed intrarenal inflammation, neutrophil infiltration, and tubular cell necrosis and i
118 Serum interleukin-8 (IL-8) levels and tumor neutrophil infiltration are associated with worse progno
120 vented chronic-binge ethanol-induced hepatic neutrophil infiltration as well as elevation of serum tr
121 d mice exhibited significantly more airspace neutrophil infiltration at 6 hours following P. aerugino
123 lity, which could be associated with reduced neutrophil infiltration at the infectious foci, increase
125 ctant-1 in the systemic circulation enhanced neutrophil infiltration, BBB permeability, and injury.
128 bone destruction, with significantly reduced neutrophil infiltration but considerably more macrophage
129 at IL-36gamma specifically induces transient neutrophil infiltration but does not impact monocyte and
130 ltured endothelial cells while reducing lung neutrophil infiltration by 40% in a mouse model of LPS-i
131 accumulated into the liver, and blockage of neutrophil infiltration by anti-granulocyte receptor 1 d
132 and induced models of colitis by regulating neutrophil infiltration, colitogenic CD4(+) T cell activ
133 is and apoptosis in the parenchyma and fewer neutrophil infiltration compared to CS liver tissues.
134 o-inflammatory cytokine responses and reduce neutrophil infiltration compared with a PLY mutant.
135 cted STAT1/IL-13 DKO mice also had increased neutrophil infiltration compared with that of RSV-infect
137 e dismutase, nitric oxide, heme oxygenase-1, neutrophil infiltration, cysteamine, mucin, hydrogen sul
138 the immune response, including reduction of neutrophil infiltration, decreased T cell cytokine produ
139 ased mortality that correlated with elevated neutrophil infiltration, diminished numbers of mature ol
140 her levels of reactive oxygen species (ROS), neutrophil infiltration, dityrosine and 4-HNE, as well a
142 th high HIC index values (0.88-0.96) induced neutrophil infiltration, elevation of pro-inflammatory c
143 ase was associated with increased intestinal neutrophil infiltration, epithelial injury, and permeabi
144 increased epidermal thickness, mast cell and neutrophil infiltration, expression of inflammatory medi
145 damage, 4-hydroxynanoneal adduct formation, neutrophil infiltration, fibrosis, and microvascular pru
147 le in the disease have long been recognized, neutrophil infiltration has also been assessed in many c
148 nted oxidative stress, NF-kappaB activation, neutrophil infiltration, hepatocyte apoptosis, and liver
149 ysteine also prevented NF-kappaB activation, neutrophil infiltration, hepatocyte apoptosis, and liver
150 neys, cytokine and chemokine storm, striking neutrophil infiltration, histological abnormalities in b
153 205 reduced disease activity index score and neutrophil infiltration in a mouse dextran sodium sulfat
154 iphenylmethane (4MDM) promoted resolution of neutrophil infiltration in a murine cigarette smoke-indu
155 n of EC Notch signaling inhibited peritoneal neutrophil infiltration in an ovarian carcinoma mouse mo
156 indices, paw thickness, cartilage damage and neutrophil infiltration in both CIA and CAIA models.
158 e treatment inhibited hepatic macrophage and neutrophil infiltration in CCl4 -induced hepatitis and s
161 recruited type 2 macrophages, and prevented neutrophil infiltration in diabetic wounded corneas.
162 utic N-acetylcysteine lowers gastric mucosal neutrophil infiltration in H. pylori-infected Le(b)-expr
164 appaB activation established a link to favor neutrophil infiltration in inducing liver damage during
165 immunostaining showed a dramatic decrease of neutrophil infiltration in infected lung tissues for lin
166 gic improvement (defined as the composite of neutrophil infiltration in less than 5% of crypts and no
167 rophil recruitment based on the reduction of neutrophil infiltration in mice in which the overall sul
171 tic target for the restriction of pathogenic neutrophil infiltration in TH17-mediated autoimmune dise
175 crophages and IL-17A produced in situ drives neutrophil infiltration in the epidermis and dermis of t
176 ke skin model, we show that KO mice had less neutrophil infiltration in the epidermis than controls,
177 ng to either insufficient MDSCs or excessive neutrophil infiltration in the fetomaternal interface ma
178 , UFP ingested mice developed macrophage and neutrophil infiltration in the intestinal villi, accompa
179 e-blood levels of QSOX1 and PLBD1 related to neutrophil infiltration in the ischemic myocardium in an
180 Anxa2(-/-) mice develop pulmonary edema and neutrophil infiltration in the lung parenchyma in respon
182 APC, more interleukin-6, and show increased neutrophil infiltration in the lungs compared with WT co
184 male OVA-specific TH17 cells increased acute neutrophil infiltration in the lungs of OVA-challenged r
185 ation of pro-inflammatory cytokines, reduced neutrophil infiltration in the lungs, and diminished hep
187 sed alveolar bone loss and a lower degree of neutrophil infiltration in the periodontium than vehicle
188 rization, fibrin depositions, macrophage and neutrophil infiltration in the placenta did not differ b
189 munohistochemistry revealed comparable early neutrophil infiltration in the renal cortex from P2X(7)(
191 ction in active glomerular proliferation and neutrophil infiltration in three of five patients, consi
192 ast cell-derived TNF, as we observed reduced neutrophil infiltration in W(sh)/W(sh) mice reconstitute
193 lation by CL 316,243 promotes adipose tissue neutrophil infiltration in wild type and P-selectin-null
195 o WT LTx, with enhanced hepatic necrosis and neutrophil infiltration, indicating a protective role of
196 tor GM6001 reduced the early BBB leakage and neutrophil infiltration, indicating that OPC-derived MMP
197 Ps (NPs-CD11b) into the tumor is mediated by neutrophil infiltration induced by photosensitization (P
198 effectively prevented cPLA2alpha induction, neutrophil infiltration into adipose tissue (likely invo
199 2 receptor and is characterized by extensive neutrophil infiltration into different organs, including
207 lung inflammation, as evidenced by a reduced neutrophil infiltration into the airways, with diminishe
208 1(-/-)) exhibited a significant reduction in neutrophil infiltration into the brain after TBI as comp
210 S3(fl/fl) mice is characterized by extensive neutrophil infiltration into the cerebellum and brainste
211 iorates atypical EAE development by reducing neutrophil infiltration into the cerebellum/brainstem.
212 ed keratinocyte activation and inhibition of neutrophil infiltration into the dermis, demonstrating t
214 l, Cj-P1 induced more C. jejuni invasion and neutrophil infiltration into the Il10(-/-) mouse colon t
216 ngth accompanied by prominent macrophage and neutrophil infiltration into the intestinal villi, admin
218 -type activated PC and significantly reduced neutrophil infiltration into the lungs of septic mice.
219 way wall thickness as well as eosinophil and neutrophil infiltration into the respiratory airway.
223 the tumor associated molecules that regulate neutrophil infiltration into tumors may provide new and
225 enous myeloperoxidase infusion revealed that neutrophil infiltration is a prerequisite for myocardial
226 ted with diabetic wounds, while reduction of neutrophil infiltration is associated with enhanced heal
227 Collectively, these data show that decidual neutrophil infiltration is not essential for the inducti
228 tigated the role of VEGF165b in brain edema, neutrophil infiltration, ischemic brain damage, and neur
229 eover, while the absence of IL-1R1 prevented neutrophil infiltration, it did not protect from acantho
230 rrier that results in edema, hemorrhage, and neutrophil infiltration, leading to exacerbated lung inf
231 ion (STAT)6 ameliorated alpha-Galcer-induced neutrophil infiltration, liver injury, and hepatitis.
232 ion including epithelial damage, congestion, neutrophil infiltration, loss of mucin from goblet cells
233 flammation signature was linked to increased neutrophil infiltration, more cell death and greater par
234 a), neutrophil chemoattractants (MIP-2, KC), neutrophil infiltration (MPO activity), lipid peroxidati
235 33, IL-17alpha, IL-2, MIP-2, and MCP-1), and neutrophil infiltration (myeloperoxidase staining)).
236 ministration of recombinant IL-17A increased neutrophil infiltration, NET formation, and liver necros
237 se PAR1 is expressed at sites where abundant neutrophil infiltration occurs, we hypothesized that neu
241 posome treatment, translating into a massive neutrophil infiltration of the peritoneum capable of neu
244 classical hallmark of acute inflammation is neutrophil infiltration of tissues, a multistep process
245 only develop fatty liver without significant neutrophil infiltration or elevation of chemokines.
246 ssed AHR and failed to dampen macrophage and neutrophil infiltration or inflammatory cytokine product
247 erestingly, mice with neutropenia, defective neutrophil infiltration or lacking elastase were protect
248 NASH progression in HFD-fed mice by inducing neutrophil infiltration, oxidative stress, and stress ki
249 y lymph (p < 0.01 at 24 and 48 hr), alveolar neutrophil infiltration (p = 0.04), and pulmonary myelop
250 s are the main cause of ALI, leading to lung neutrophil infiltration, permeability increases, deterio
251 nalyzed for morphological injury, apoptosis, neutrophil infiltration, proinflammatory mRNAs, and TGF-
252 Passive immunization with 2B11 increased neutrophil infiltration, reduced skin invasion, and prot
253 vestigated whether NLRP3 knockdown decreases neutrophil infiltration, reduces brain edema, and improv
254 gulated 10-fold, whereas expression of other neutrophil infiltration-related adhesion molecules (e.g.
257 on in vivo abrogated alcohol-induced hepatic neutrophil infiltration, resident immune cell activation
260 to establish a functional link between lung neutrophil infiltration, secretion of chemokines by canc
261 rthermore, platelet depletion abrogated lung neutrophil infiltration, suggesting a sequential partici
264 e using photosensitization rapidly activates neutrophil infiltration that mediates delivery of nanoth
265 ranscription of c-Myb and elevated levels of neutrophil infiltration, thereby alleviating infectious
269 jury, selective deletion of EC MIF decreases neutrophil infiltration to the bronchoalveolar lavage an
272 hibitor of IKK-2, resulted in suppression of neutrophil infiltration to the lung tissues and reductio
274 /W(sh) mice, respond to IL-33 treatment with neutrophil infiltration to the peritoneum, whereas other
275 roduction by gammadelta T cells and ILC3 and neutrophil infiltration to the site of infection, were g
277 CO92 Deltapgm infections, with an increased neutrophil infiltration to the spleen 5 days postinfecti
279 matory state in endothelial cells, promoting neutrophil infiltration, tumor cell adhesion, and metast
281 reflow injury, promoting repair via limiting neutrophil infiltration, up-regulating Ki67, and Roof pl
282 e destruction, bacterial invasion, increased neutrophil infiltration, upregulation of cytokines inclu
287 nd chemokines was the same as LPS alone, but neutrophil infiltration was inhibited, likely by interru
288 D36(-/-) mice, in which infarcts were small, neutrophil infiltration was large and similar to that of
292 eutrophil recruitment to the brain, and that neutrophil infiltration was required for parenchymal tis
293 CSF3R expression, a neutrophil signature and neutrophil infiltration were associated with a type 1 im
296 ithelial permeability, muscle thickness, and neutrophil infiltration were studied in colon tissues an
297 male mice, which had hepatic lymphocyte and neutrophil infiltration, were treated by vancomycin, pol
298 nts, albuminuria, serum urea, and glomerular neutrophil infiltration when compared with WT littermate
299 ase of inflammation-(d1), 15-epi-LXA4 primes neutrophil infiltration with a robust increase of Ccl2 a