ライフサイエンスコーパス: neutrophilic

1 3: COPD predominant, mixed eosinophilic and neutrophilic.

2 s follows: eosinophilic (40%), mixed (6.7%), neutrophilic (5.4%) and paucigranulocytic (47.9%).

3 Acute inflammation is neutrophilic (75% of ocular CD45 + cells) which is refle

4 Neutrophilic airway diseases, including cystic fibrosis,

5 s adjuvants developed mixed eosinophilic and neutrophilic airway inflammation and airway hyperrespons

6 utophagy in pulmonary CD11c(+) cells induces neutrophilic airway inflammation and hyperreactivity.

7 icrobiome across asthmatic patients, whereas neutrophilic airway inflammation does not.

8 Because azithromycin attenuated neutrophilic airway inflammation in a murine viral bronc

9 ces eosinophilic and endotoxin (LPS)-induced neutrophilic airway inflammation in animal models and he

10 to 500 mug/m(3) wood smoke particles caused neutrophilic airway inflammation in human volunteers, wi

11 itamin D supplementation on eosinophilic and neutrophilic airway inflammation in patients with nonato

12 In a murine model of neutrophilic airway inflammation induced by HDM and poly

13 Neutrophilic airway inflammation, disease severity, and

14 patients with severe asthma and is linked to neutrophilic airway inflammation, suggesting that these

15 nfiltrating CD4 T cells express Foxp3 during neutrophilic airway inflammation.

16 ecular patterns, innate cytokine release and neutrophilic airway inflammation.

17 ased Cldn4 expression was characteristic for neutrophilic airway inflammation.

18 tions of HDM-induced asthma characterized by neutrophilic airway inflammation.

19 mentary therapeutic agent in conditions with neutrophilic airway inflammation.

20 NF-kappaB activation in the lungs, increased neutrophilic alveolitis, and greater lung inflammation/i

21 After allergen exposure, rhinovirus-induced neutrophilic and eosinophilic airway inflammation and hy

22 ired during the allergen challenge phase for neutrophilic and eosinophilic inflammation.

23 emonstrated that Exserohilum provokes robust neutrophilic and granulomatous inflammation capable of t

24 Initial neutrophilic and histiocytic inflammation in affected ti

25 thma, while IL-8 was higher in patients with neutrophilic and mixed asthma (P<.001 for all comparison

26 IL-6 and sIL-6R levels were enriched for the neutrophilic and mixed granulocytic subtypes.

27 69.7-95.0), P=.009] for eosinophilic, mixed, neutrophilic and paucigranulocytic asthma, respectively,

28 7%, respectively) and less frequently in the neutrophilic and paucigranulocytic phenotype (25% and 21

29 of these as highly eosinophilic, 3 as highly neutrophilic, and 2 as highly atopic with relatively low

30 ammatory end points, including eosinophilic, neutrophilic, and ST2(+)CD4(+) T-cell infiltration.

31 ted by leukocyte transmigration, whereas the neutrophilic antimicrobial peptide HNP-1 is noted as a p

32 uction of chemokine production and increased neutrophilic apoptosis.

33 H2/TH17-low asthma had 2 distinct subgroups: neutrophilic asthma (45%) and pauci-inflammatory asthma

34 oup of patients with TH2/TH17-low asthma had neutrophilic asthma and increased BAL fluid IL-1alpha, I

35 ike receptor C4 (NLRC4) were associated with neutrophilic asthma and with sputum IL-1beta protein lev

36 s indeed not widely available and markers of neutrophilic asthma are still lacking.Objectives: To det

37 Eosinophilic and neutrophilic asthma endotypes are also classified by epi

38 Sixty percent of the patients with neutrophilic asthma had a pathogenic microorganism in BA

39 t and TH2-predominant asthma, which included neutrophilic asthma in 6% and 0% of patients, respective

40 Our data indicate that neutrophilic asthma in Atg5(-/-) mice is glucocorticoid

41 Neutrophilic asthma in STAT6(-/-) mice was accompanied b

42 Neutrophilic asthma is associated with airway microbiolo

43 n was amplified in bronchial/nasal mucosa of neutrophilic asthma prone to exacerbation, suggesting a

44 airway inflammation-eosinophilic, mixed, and neutrophilic asthma via different methods of house dust

45 to 4 unique phenotypes: eosinophilic asthma, neutrophilic asthma, mixed granulocytic asthma, and pauc

46 For neutrophilic asthma, the combination of nonanal, 1-propa

47 nas taxa relative abundance in patients with neutrophilic asthma.

48 lmonary disease, respiratory infections, and neutrophilic asthma.

49 otential therapeutic target in patients with neutrophilic asthma.

50 function and identify molecular markers for neutrophilic asthma.

51 ociated with more severe, steroid-refractory neutrophilic asthma.

52 the pathogenesis of corticosteroid-resistant neutrophilic asthma.

53 ol subjects and was highest in patients with neutrophilic asthma.

54 l responses can lead to pathology as seen in neutrophilic asthma.

55 for factors other than IL-17 as targets for neutrophilic asthma.

56 levels in sputum samples from patients with neutrophilic asthma.

57 role for IL-1beta in both acute and chronic neutrophilic asthma.

58 e with asthma severity, especially in severe neutrophilic asthma.

59 Casp-1, and IL-1beta was characteristic for neutrophilic asthma.

60 ith asthma and provide surrogate markers for neutrophilic asthma.

61 in IL-8 production and likely contributed to neutrophilic asthma.

62 ose compounds were found in higher levels in neutrophilic asthma.Conclusions: Our study is the first

63 The overexpression was restricted to neutrophilic asthmatics (sputum neutrophils >/= 76%), wh

64 n addition, we showed that inflammasome/Th17/neutrophilic axis cytokine-IL-1beta-may transiently impa

65 ell count and % neutrophils were elevated in neutrophilic BOS and RAS compared to stable and non-neut

66 could not detect any differences between non-neutrophilic BOS and stable patients.

67 ulated in RAS compared to stable, whereas in neutrophilic BOS IL-1beta (P<0.001), IL-1Ralpha (P<0.01)

68 hilic BOS and RAS compared to stable and non-neutrophilic BOS patients, whereas also the % eosinophil

69 ble patients, 20 patients suffering from non-neutrophilic BOS, 17 from neutrophilic BOS, and 20 from

70 suffering from non-neutrophilic BOS, 17 from neutrophilic BOS, and 20 from RAS using classic enzyme-l

71 pes (bronchiolitis obliterans syndrome [BOS]-neutrophilic BOS-restrictive allograft syndrome [RAS]) h

72 to interrogate the underlying mechanisms of neutrophilic breach of mucosal barriers.

73 Neutrophilic, but not eosinophilic, asthmatics display o

74 more patients in the C4d-positive group had neutrophilic capillaritis (54% vs. 29%, P = .035), there

75 re severe histological injury such as ALI or neutrophilic capillaritis.

76 als are expected to decrease the activity of neutrophilic CatC without affecting those of elastase-li

77 l fluid/blood glucose ratio less than 0.5, a neutrophilic cerebrospinal fluid pleocytosis (> 5 cells/

78 table population of Ly6G(+) eosinophils with neutrophilic characteristics and functions, and had incr

79 rexpression (OE) increased the production of neutrophilic chemoattractant IL-8 in the absence or pres

80 tokines; cluster 2: asthma and COPD overlap, neutrophilic; cluster 3: COPD predominant, mixed eosinop

81 , TNF-alpha, IFN-gamma) were enhanced in the neutrophilic cohorts.

82 (P = .022) and more dissimilar (P = .005) in neutrophilic compared with eosinophilic participants.

83 Neutrophilic corticosteroid-resistant asthma accounts fo

84 oid precursor cells, dysplastic bone marrow, neutrophilic cutaneous and pulmonary inflammation, chond

85 consequence of impaired recruitment of a non-neutrophilic CXCR2 positive leukocyte population.

86 atients presented with neonatal-onset fever, neutrophilic dermatitis/panniculitis, and failure to thr

87 an inflammatory skin disease that resembles neutrophilic dermatosis in humans.

88 Pyoderma gangrenosum (PG) is a rare neutrophilic dermatosis that presents with rapidly devel

89 mmune-dysregulatory disease chronic atypical neutrophilic dermatosis with lipodystrophy and elevated

90 Chronic Atypical Neutrophilic Dermatosis with Lipodystrophy and Elevated

91 Sweet syndrome (SS) is a prototypical neutrophilic dermatosis, a class of inflammatory disease

92 Resistin inhibited neutrophilic-differentiated NB4 cell migration and intra

93 ptic shock and acute kidney injury inhibited neutrophilic-differentiated NB4 cell migration.

94 howed elevated resistin levels and inhibited neutrophilic-differentiated NB4 cell migration.

95 th or without severe acute kidney injury) on neutrophilic-differentiated NB4 cells.

96 Neutrophilic differentiation is dependent on CCAAT enhan

97 Bet v 1 was digested with neutrophilic endolysosomal extracts, and the resulting f

98 d Western diet triggers focal granuloma-like neutrophilic enteritis in mice that lack one allele of G

99 ted effects of mutations in MPO encoding the neutrophilic enzyme myeloperoxidase (MPO).

100 as evidenced by salivary and serum levels of neutrophilic enzymes.

101 = 14), paucigranulocytic (n = 60), or mixed neutrophilic-eosinophilic (n = 9) asthma phenotypes.

102 Eosinophilic and mixed neutrophilic/eosinophilic inflammation were more prevale

103 Mice lacking PTX3 have exaggerated neutrophilic/eosinophilic lung inflammation, mucus produ

104 In neutrophilic Escherichia coli, internal pH is kept at ~7

105 at uric acid crystals became enmeshed in the neutrophilic extracellular traps (NETs) produced from ho

106 ons and noneosinophilic endotypes, including neutrophilic forms of disease.

107 e not associated with asthma, even in severe neutrophilic forms.

108 ongly associated with asthma, even in severe neutrophilic forms.

109 humans, such as Howell-Jolly bodies and low neutrophilic granularity, are commonplace in healthy mic

110 IFN-gamma accumulates in primary neutrophilic granules and is released upon induction of

111 Polymorphonuclear neutrophilic granulocytes (PMN) as cellular components o

112 psilon)-(carboxymethyl)lysine (CML), VCAM-1, neutrophilic granulocytes, lymphocytes, and macrophages

113 blood vessels and the numbers of infiltrated neutrophilic granulocytes, lymphocytes, and macrophages

114 ower numbers of infiltrating macrophages and neutrophilic granulocytes.

115 enhancement of transendothelial migration of neutrophilic granulocytes.

116 Neutrophilic groups were also characterized by higher pr

117 ls in 44 patients with nonatopic asthma with neutrophilic (>/=53%) and/or eosinophilic (>/=3%) airway

118 response in vivo, the first demonstration of neutrophilic IL-1beta production in response to viral lu

119 A CRS endotype with a proinflammatory neutrophilic immune signature was enriched in older pati

120 ften including non-atopic episodes driven by neutrophilic infection.

121 sis, reactive oxygen species generation, and neutrophilic infiltrate that characterize the severe ast

122 ide reduced its expression and the prominent neutrophilic infiltrate, a hallmark of the disease.

123 Propofol exposure increased neutrophilic infiltrates into the kidney and enhanced ba

124 round to spindle-shaped cells with prominent neutrophilic infiltrates.

125 In vivo, RvD4 (ng/mouse) reduced neutrophilic infiltration (~40%) and enhanced uptake of

126 d neutrophilic inflammation, with a profound neutrophilic infiltration and activation in type 2 CRSwN

127 zation of circulating RANTES decreased liver neutrophilic infiltration and attenuated HCC tumor initi

128 Neutrophilic infiltration is a leading contributor to pa

129 Cytometric methods revealed extensive neutrophilic infiltration of oral tissues in GRAKO mice;

130 cigarettes may develop unstable disease and neutrophilic infiltration of the airways, features more

131 n histological patterns of S grafts revealed neutrophilic infiltration surrounding fat accumulation.

132 tors for neutrophils (e.g. CXCL5, CXCL8) and neutrophilic infiltration was followed with much delay b

133 Furthermore, we observed neutrophilic infiltration was slightly increased in anti

134 showed severe epidermal hyperplasia, greater neutrophilic infiltration, and higher expression of Th17

135 This was associated with increased liver-neutrophilic infiltration, whereas infiltration of lymph

136 Further, more neutrophilic infiltrations, epithelial damage, and infla

137 ections were also associated with markers of neutrophilic inflammation (P < 0.001) and respiratory ad

138 s to naive TLR2(-/-) mice was sufficient for neutrophilic inflammation after rhinovirus infection, wh

139 IL-1 receptor appears to be a marker of neutrophilic inflammation and airflow obstruction in pat

140 1beta responses that drive steroid-resistant neutrophilic inflammation and airway hyperresponsiveness

141 monary adaptation evidenced by abrogation of neutrophilic inflammation and airway hyperresponsiveness

142 h for prevention and management of excessive neutrophilic inflammation and alveolar barrier dysfuncti

143 ammasome rescued zebrafish disease models of neutrophilic inflammation and anemia.

144 Protection was characterized by robust neutrophilic inflammation and compartmentalization of ba

145 IL-17A is involved in neutrophilic inflammation and corticosteroid resistance,

146 a tissue-specific role for AC in regulating neutrophilic inflammation and cytokine production.

147 f host-directed therapies in TBDM, targeting neutrophilic inflammation and diabetic complication path

148 ntagonist anakinra had protective effects on neutrophilic inflammation and emphysema.

149 IL-17A is central for neutrophilic inflammation and has been linked to COPD pa

150 complex mediated vasculitis characterized by neutrophilic inflammation and nuclear debris in post cap

151 We aimed to investigate the role of neutrophilic inflammation and PAR1 in S pneumoniae-induc

152 PGP elicits an exacerbated neutrophilic inflammation and protease imbalance that fu

153 Anakinra effectively reduced airway neutrophilic inflammation and resulted in no serious adv

154 NTHi-induced pulmonary neutrophilic inflammation and tumor-associated neutrophi

155 ype) mice demonstrated higher levels of lung neutrophilic inflammation and viral load, but lower leve

156 expected for lower AcPGP levels, markers of neutrophilic inflammation are blunted.

157 This study demonstrated a severe neutrophilic inflammation associated with severe eosinop

158 Thus, mucosal neutrophilic inflammation at the time of RSV exposure en

159 te the substantial heterogeneity in systemic neutrophilic inflammation between tumour-bearing hosts a

160 IL-17RA and IL-17RC subunits, also promotes neutrophilic inflammation but its effects on vascular ce

161 sponse to pathogenic bacteria, but excessive neutrophilic inflammation can be associated with bystand

162 osa infections coupled with robust, damaging neutrophilic inflammation characterize the chronic lung

163 scrapings from patients with CF showed that neutrophilic inflammation correlated with reduced expres

164 However, neutrophilic inflammation did not restrict infection and

165 an rhinovirus (HRV) infections are linked to neutrophilic inflammation during acute exacerbations of

166 ate that children with refractory asthma and neutrophilic inflammation had a BAL cytokine pattern con

167 4A3 expression is also increased at sites of neutrophilic inflammation in a human model of intraderma

168 This review discusses the role of neutrophilic inflammation in angiotropism and pericytic

169 tress may be useful for alleviating damaging neutrophilic inflammation in CF airways.

170 This study aimed to characterize the neutrophilic inflammation in CRSwNP and its relation to

171 in pulmonary neutrophilia and showed reduced neutrophilic inflammation in response to dexamethasone t

172 insufficient to prevent circadian control of neutrophilic inflammation in response to inhaled LPS, wi

173 caused by airway mucus obstruction precedes neutrophilic inflammation in Scnn1b-transgenic (Scnn1b-T

174 c epithelial necrosis in the pathogenesis of neutrophilic inflammation independent of bacterial infec

175 Pulmonary neutrophilic inflammation induced by CS is significantly

176 self-resolving and GC-induced resolution of neutrophilic inflammation induced by LPS in mice.

177 poptotic program that promotes resolution of neutrophilic inflammation induced by LPS.

178 Neutrophilic inflammation is a common feature of many au

179 Neutrophilic inflammation is central to disease pathogen

180 Neutrophilic inflammation is tightly regulated and subse

181 he hypothesis that asthmatic smokers develop neutrophilic inflammation of the airways propagated at l

182 Neutrophilic inflammation often persists for days despit

183 TLR4 expression did not correlate with neutrophilic inflammation or AHR.

184 ted toxin neutralization and tissue-specific neutrophilic inflammation preserve tissue integrity duri

185 ients with cystic fibrosis (CF), its role in neutrophilic inflammation remains unknown.

186 Acute gonorrhea is characterized by neutrophilic inflammation that is insufficient to clear

187 -proline (AcPGP) pathway is a novel means of neutrophilic inflammation that is pathologic in the deve

188 We used a mouse model of peritoneal neutrophilic inflammation to determine if Ang-1 could st

189 e data suggest that M. tuberculosis exploits neutrophilic inflammation to preferentially replicate at

190 In contrast to eosinophilic inflammation, neutrophilic inflammation was not promoted by beta2AR si

191 Roles for IL-1beta-induced neutrophilic inflammation were examined using IL-1beta a

192 rom infected mice, but antibody deposits and neutrophilic inflammation were not features of the lesio

193 TH17 cells and the consequent neutrophilic inflammation were poorly sustained by inhal

194 es associated with type I IFN signalling and neutrophilic inflammation were shown to correlate with d

195 Neutrophilic inflammation with prolonged neutrophil surv

196 akin to the mucous and squamous metaplasia, neutrophilic inflammation, and fibrosis seen in COPD.

197 owever, its role during S pneumoniae-induced neutrophilic inflammation, and the mechanisms for neutro

198 here are few specific therapies that reverse neutrophilic inflammation, but uncovering mechanisms reg

199 ut undergo infection-related weight loss and neutrophilic inflammation, development of anti-pseudomon

200 um eDNA in asthma was associated with airway neutrophilic inflammation, increases in soluble NET comp

201 njury and murine acute lung injury models of neutrophilic inflammation, overexpression of SEMA3F dela

202 ophage production of IL-1beta and subsequent neutrophilic inflammation, resulting in reduced metastas

203 most patients with COPD have a predominantly neutrophilic inflammation, some have an increase in eosi

204 and gammadelta-17 cells, proposed drivers of neutrophilic inflammation, were not strongly associated

205 r 4 (TLR4) to stimulate CXCL-mediated innate neutrophilic inflammation, which in turn facilitates all

206 splayed variable degrees of eosinophilic and neutrophilic inflammation, with a profound neutrophilic

207 atients with RA, which likely contributed to neutrophilic inflammation.

208 eutrophil migration throughout the course of neutrophilic inflammation.

209 iding with AEC sloughing, HMGB1 release, and neutrophilic inflammation.

210 learance of pulmonary bacteria and increased neutrophilic inflammation.

211 e and was associated with the suppression of neutrophilic inflammation.

212 of asthma in the absence of eosinophilic or neutrophilic inflammation.

213 metic) drive caspase-dependent resolution of neutrophilic inflammation.

214 severe asthma who present with TH17-mediated neutrophilic inflammation.

215 vere asthma often present with TH17-mediated neutrophilic inflammation.

216 The cytokine IL-17A is associated with neutrophilic inflammation.

217 lator La-related protein 7 (LaRP7) increased neutrophilic inflammation.

218 n primary neutrophils and in mouse models of neutrophilic inflammation.

219 haracterization of miRs that are relevant to neutrophilic inflammation.

220 eumoniae, which is associated with excessive neutrophilic inflammation.

221 alcium levels, tumor growth, metastasis, and neutrophilic inflammation.

222 cerbations that are thought to be related to neutrophilic inflammation.

223 (WT) mice in M. tuberculosis replication and neutrophilic inflammation.

224 unable to control tissue-damaging, pulmonary neutrophilic inflammation.

225 ystemic hypoxia and consequent on disordered neutrophilic inflammation.

226 but little is known about the role of ST2 in neutrophilic inflammation.

227 that are shown to be important mediators of neutrophilic inflammation; selective targeting of their

228 Synovial NK cells promote a neutrophilic inflammatory cell infiltrate, and persisten

229 f ErbB kinases have therapeutic potential in neutrophilic inflammatory disease.

230 rgic sensitization did not induce T(H) 17, a neutrophilic inflammatory response as observed in global

231 brosis (CF) is characterized by an excessive neutrophilic inflammatory response within the airway as

232 or preventing exacerbation of disease in the neutrophilic inflammatory response.

233 s, a model in which Stx2 induces a primarily neutrophilic inflammatory response.

234 AP-alpha could be a potential drug target in neutrophilic inflammatory responses to avoid exaggerated

235 CL8 accumulation correlated to the degree of neutrophilic influx in affected mucosa.

236 The system is dominated by neutrophilic, iron-oxidizing bacteria, including 'marine

237 s a recurrent activating mutation in chronic neutrophilic leukemia (CNL) and to a lesser extent in at

238 Chronic neutrophilic leukemia (CNL) is a distinct myeloprolifera

239 Chronic neutrophilic leukemia (CNL), atypical chronic myeloid le

240 -proximal mutations seen commonly in chronic neutrophilic leukemia (e.g., T618I), functionally defect

241 Blood tests showed mild anemia, neutrophilic leukocytosis and elevated inflammation mark

242 This can result in stimulation of both neutrophilic leukocytosis and the release of immature gr

243 Thus, LTB4 was the major driver of excessive neutrophilic lung inflammation in CGD mice in the early

244 pontaneous airway hyperreactivity and severe neutrophilic lung inflammation in mice.

245 d-glycosyl aesculin significantly suppressed neutrophilic lung inflammation, a hallmark of acute lung

246 In patients with neutrophilic lung inflammation, mature CatC is found in

247 g infection delays the recovery process from neutrophilic lung inflammation.

248 ng inflammation to corticosteroid-refractory neutrophilic manifestation.

249 DG172-mediated repression of genes encoding neutrophilic markers in both differentiating wild-type a

250 Mechanistically, pathogenic fungi induce neutrophilic MDSCs through the pattern recognition recep

251 Neutrophilic microbial aerobic oxidation of ferrous iron

252 enotypes have been recognized (eosinophilic, neutrophilic, mixed and paucigranulocytic).

253 criminate T2-high (eosinophilic) and T2-low (neutrophilic/mixed-inflammation) asthma and atopy are st

254 surements with clonally determined lymphoid, neutrophilic/monocytic, and/or erythroid progeny outputs

255 andida albicans induces a distinct subset of neutrophilic myeloid-derived suppressor cells (MDSCs), w

256 nts and classified as eosinophilic (n = 84), neutrophilic (n = 14), paucigranulocytic (n = 60), or mi

257 te that formyl peptide receptor 1 (FPR1) and neutrophilic NADPH oxidase (NOX2) are required for the r

258 strategies against one of the most powerful neutrophilic oxidants may provide novel insights into tr

259 Sixty percent of neutrophilic patients had a subclinical infection.

260 healthy controls; and 3) effect of the human neutrophilic peptide-1 (HNP-1) on epithelial adhesion mo

261 sed peripheral neutrophilia, which amplified neutrophilic peritoneal inflammation in X-CGD mice.

262 upregulated in eosinophilic and even more in neutrophilic phenotype.

263 isk over a 1-year period were detected among neutrophilic phenotypes.

264 over a 1-year period were observed among the neutrophilic phenotypes.

265 f alcoholic liver disease (ALD) with intense neutrophilic (polymorphonuclear; PMN) inflammation and h

266 nism that can be entirely managed by CatS in neutrophilic precursor cells.

267 inflammatory signature that corresponds to a neutrophilic proinflammatory response.

268 ine for the pathogenesis of asthma including neutrophilic pulmonary inflammation and airway hyperresp

269 skin and colocalized with IL-36gamma around neutrophilic pustules.

270 stemic inflammatory disease characterized by neutrophilic pustulosis and triggered by pro-inflammator

271 Infection-negative neutrophilic RA was associated with an increase in level

272 A subgroup of patients with noninfected neutrophilic RA was associated with systemic inflammatio

273 adelta T cells showed a partial reduction in neutrophilic recruitment and a significant decrease in I

274 sing intravital microscopy, we observed that neutrophilic recruitment to corneal infections limits P.

275 colony-stimulating factor (G-CSF)-regulated neutrophilic response and prolonged inflammation.

276 This neutrophilic response augmented bacterial clearance and

277 atracheal administration of IL-17 provoked a neutrophilic response in the airways of WT and CF animal

278 a haven for S. aureus, thereby delaying the neutrophilic response in the peritoneum by 48 hours and

279 limiting the duration and magnitude of this neutrophilic response remain poorly understood.

280 s eosinophilic airway inflammation and acute neutrophilic response to inhaled LPS challenge in volunt

281 e of humans, characterized by a dysregulated neutrophilic response to specific bacterial species with

282 C3s, and gammadelta T cells and an increased neutrophilic response without increased inflammation or

283 and STAT6-deficient mice exhibit a primarily neutrophilic response.

284 ase in regulating the amplitude of the early neutrophilic response.

285 nificantly reduced time to resolution of the neutrophilic response.

286 receptor family member Nlrp12 in modulating neutrophilic responses during lethal IAV infection.

287 Additionally, all tissue-based neutrophilic responses to fungal infections necessitate

288 he endosymbiotic bacteria, Wolbachia, induce neutrophilic responses to the human helminth pathogen On

289 ion through the regulation of CXCL1-mediated neutrophilic responses.

290 asome expression is highest in patients with neutrophilic SA.

291 ntly controlled development of TH17-mediated neutrophilic severe asthma in both acute and chronic HDM

292 parasitism to acute-phase levels and induced neutrophilic skeletal muscle inflammation.

293 odel, it has recently been demonstrated that neutrophilic skin inflammation promotes angiotropism and

294 ated MMP-8, MMP-9, and MPO suggests a common neutrophilic source and provides evidence of neutrophil

295 an alternative approach, we analyzed ex vivo neutrophilic superoxide inhibition in response to beta2

296 and relating genotypes to beta2 AR-mediated neutrophilic superoxide inhibition.

297 d with hematologic malignancies than classic neutrophilic Sweet syndrome.

298 n from eosinophilic T helper cell 2 (TH2) to neutrophilic TH17 polarity.

299 oles to collectively coordinate an effective neutrophilic transepithelial migratory response.

300 apy-resistant asthma stratified according to neutrophilic versus nonneutrophilic BAL inflammatory cel