ライフサイエンスコーパス: nicotinamide adenine dinucleotide

1 ant metabolites, such as the reduced form of nicotinamide adenine dinucleotide.

2 P-ribose)polymerase 1 activation, exhausting nicotinamide adenine dinucleotide and adenosine triphosp

3 a common intermediate in the biosynthesis of nicotinamide adenine dinucleotide and its derivatives in

4 skeletal proteins, and loss of intracellular nicotinamide adenine dinucleotide and nicotinamide adeni

5 thesis of RNA with NADH (the reduced form of nicotinamide adenine dinucleotide) and FAD (flavin adeni

6 primarily SAM (S-adenosyl methionine), NAD (nicotinamide adenine dinucleotide), and FAD (flavin aden

7 curs via a non-canonical mechanism that uses nicotinamide adenine dinucleotide as a cofactor.

8 In the presence of beta-nicotinamide adenine dinucleotide as cofactor, and ethan

9 ctate, with concomitant oxidation of reduced nicotinamide adenine dinucleotide as the final step in t

10 nicotinamide adenine dinucleotide to reduced nicotinamide adenine dinucleotide, as a result of oxidat

11 netics of the signal transducing reaction of nicotinamide adenine dinucleotide at CNTs accounted for

12 functional capillary density: 573+/-13cm/cm; nicotinamide adenine dinucleotide autofluorescence: 56+/

13 functional capillary density: 469+/-22cm/cm; nicotinamide adenine dinucleotide autofluorescence: 61+/

14 ry density: 379+/-20cm/cm;), tissue hypoxia (nicotinamide adenine dinucleotide autofluorescence: 77+/

15 beta-Nicotinamide adenine dinucleotide (beta-NAD) is a key in

16 Prior studies suggest that beta-nicotinamide adenine dinucleotide (beta-NAD) is an impor

17 show that NHDs are NAD(+) (oxidized form of nicotinamide adenine dinucleotide) binding domains that

18 erase (NAMPT) is the rate-limiting enzyme in nicotinamide adenine dinucleotide biosynthesis.

19 te-limiting enzyme in the salvage pathway of nicotinamide adenine dinucleotide biosynthesis.

20 of extracellular adenosine triphosphate and nicotinamide adenine dinucleotide, both pathways converg

21 y to the Shp promoter, which was enhanced by nicotinamide adenine dinucleotide, but not nicotinamide

22 -1) with electrochemical regeneration of the nicotinamide adenine dinucleotide cofactor.

23 cient oxidative phosphorylation, diminishing nicotinamide adenine dinucleotide concentrations and imp

24 europathy (LHON) caused by a mutation in the nicotinamide adenine dinucleotide dehydrogenase subunit

25 Fe-S center within Complex I (Ndufs1, NADH [nicotinamide adenine dinucleotide] dehydrogenase [ubiqui

26 ease in the activity of the NAD(+) (oxidized nicotinamide adenine dinucleotide)-dependent deacetylase

27 Sirtuin 1 (SIRT1), an NAD(+) (nicotinamide adenine dinucleotide)-dependent deacetylase

28 mutant mouse models to demonstrate that the nicotinamide adenine dinucleotide-dependent (NAD-depende

29 The nicotinamide adenine dinucleotide-dependent deacetylase

30 SIRT1 level, a nicotinamide adenine dinucleotide-dependent deacetylase

31 Sirtuin 1 (SIRT1) is a nicotinamide adenine dinucleotide-dependent deacetylase,

32 otide phosphate-dependent IDH1 and IDH2, and nicotinamide adenine dinucleotide-dependent IDH3) contri

33 IRT1), the most conserved mammalian oxidized nicotinamide adenine dinucleotide-dependent protein deac

34 Sirtuin (SIRT1) is a nicotinamide adenine dinucleotide-dependent protein deac

35 d the recently discovered DNA damage-induced nicotinamide adenine dinucleotide(+) depletion to underl

36 ial mapping, analysis of lactate production, nicotinamide adenine dinucleotide epifluorescence, lacta

37 ease in mitochondrial calcium content and in nicotinamide adenine dinucleotide fluorescence following

38 zoles promote neuronal survival by enhancing nicotinamide adenine dinucleotide flux in injured neuron

39 measuring the rates of production of reduced nicotinamide adenine dinucleotides from 91 potential ene

40 catenation of FeS and glycerol-dehydrogenase/nicotinamide-adenine-dinucleotide (GlDH-NAD(+)) apoenzym

41 matrix containing lactate dehydrogenase and nicotinamide adenine dinucleotide hydrate (NAD(+)).

42 llular nicotinamide adenine dinucleotide and nicotinamide adenine dinucleotide + hydrogen.

43 ontaining 1) is responsible for depletion of nicotinamide adenine dinucleotide in its oxidized form (

44 ins of NLRs are enzymes capable of degrading nicotinamide adenine dinucleotide in its oxidized form (

45 oxidase that helps to regulate intracellular nicotinamide adenine dinucleotide levels in many cell ty

46 mal redox potential, associated with reduced nicotinamide adenine dinucleotide metabolism and altered

47 ion of 2',3'-cyclic phosphate-activated beta-nicotinamide adenine dinucleotide (NAD>p) and ACA>p RNA

48 resembling human fatty liver, lowers hepatic nicotinamide adenine dinucleotide (NAD(+) ) levels drivi

49 Preclinical evidence suggests that the nicotinamide adenine dinucleotide (NAD(+) ) precursor ni

50 enzymes of redox reactions: oxidized/reduced nicotinamide adenine dinucleotide (NAD(+) and NADH) and

51 eously measure oxidized and reduced forms of nicotinamide adenine dinucleotide (NAD(+) and NADH), oxi

52 ruction program involving rapid breakdown of nicotinamide adenine dinucleotide (NAD(+)) after injury.

53 ypoxia produced PARP1-dependent depletion of nicotinamide adenine dinucleotide (NAD(+)) and inhibitio

54 s an enzyme that catalyses the hydrolysis of nicotinamide adenine dinucleotide (NAD(+)) and is a cand

55 a common intermediate in the biosynthesis of nicotinamide adenine dinucleotide (NAD(+)) and its deriv

56 ADH and its cofactor nicotinamide adenine dinucleotide (NAD(+)) are immobiliz

57 ld access a Ru-H intermediate using oxidized nicotinamide adenine dinucleotide (NAD(+)) as the H(-) s

58 anscription feedback loop produces cycles of nicotinamide adenine dinucleotide (NAD(+)) biosynthesis,

59 Here we show that nicotinamide adenine dinucleotide (NAD(+)) blocks experi

60 yl guanosine (m(7)G) cap, a non-canonical 5' nicotinamide adenine dinucleotide (NAD(+)) cap can tag c

61 ver, mammalian mRNAs can also carry a 5' end nicotinamide adenine dinucleotide (NAD(+)) cap that, in

62 al demise is due to severe, neuron-specific, nicotinamide adenine dinucleotide (NAD(+)) depletion.

63 The reduction of nicotinamide adenine dinucleotide (NAD(+)) generating a

64 critical step in the de novo biosynthesis of nicotinamide adenine dinucleotide (NAD(+)) in mammals.

65 Nicotinamide adenine dinucleotide (NAD(+)) is a coenzyme

66 Nicotinamide adenine dinucleotide (NAD(+)) is a coenzyme

67 Nicotinamide adenine dinucleotide (NAD(+)) is a critical

68 Nicotinamide adenine dinucleotide (NAD(+)) is an endogen

69 Nicotinamide adenine dinucleotide (NAD(+)) is an enzyme

70 Nicotinamide adenine dinucleotide (NAD(+)) is an essenti

71 Nicotinamide adenine dinucleotide (NAD(+)) is an essenti

72 energy stress and oxidative stress response, nicotinamide adenine dinucleotide (NAD(+)) is emerging a

73 Nicotinamide adenine dinucleotide (NAD(+)) is essential

74 Changes in nicotinamide adenine dinucleotide (NAD(+)) levels that c

75 nzymes implicated in L-tryptophan/kynurenine/nicotinamide adenine dinucleotide (NAD(+)) metabolism, t

76 by interacting with L-tryptophan/kynurenine/nicotinamide adenine dinucleotide (NAD(+)) metabolism.

77 Here, we show that exogenously applied nicotinamide adenine dinucleotide (NAD(+)) moves systemi

78 sfer of ADP-ribose from the oxidized form of nicotinamide adenine dinucleotide (NAD(+)) onto substrat

79 ich transitions to a proton-pumping Fd(red): nicotinamide adenine dinucleotide (NAD(+)) oxidoreductas

80 Nicotinamide adenine dinucleotide (NAD(+)) participates

81 Nicotinamide adenine dinucleotide (NAD(+)) participates

82 Nicotinamide adenine dinucleotide (NAD(+)) plays a criti

83 tinamide riboside (NR) is a newly discovered nicotinamide adenine dinucleotide (NAD(+)) precursor vit

84 preclinical studies showed the potential of nicotinamide adenine dinucleotide (NAD(+)) precursors to

85 sferase (NMNAT), an evolutionarily conserved nicotinamide adenine dinucleotide (NAD(+)) synthase and

86 Mitochondria require nicotinamide adenine dinucleotide (NAD(+)) to carry out

87 high lactate production, and a high ratio of nicotinamide adenine dinucleotide (NAD(+)) to its reduce

88 a PTM, in which ADP-ribosyltransferases use nicotinamide adenine dinucleotide (NAD(+)) to modify tar

89 ction in the ratio of the amount of oxidized nicotinamide adenine dinucleotide (NAD(+)) to that of it

90 Recently, cellular production of nicotinamide adenine dinucleotide (NAD(+)) via nicotinam

91 monstrate that cell-autonomous generation of nicotinamide adenine dinucleotide (NAD(+)) via the kynur

92 y, quantum dots (QDs) modified with cofactor nicotinamide adenine dinucleotide (NAD(+)) were prepared

93 Supplementation of nicotinamide adenine dinucleotide (NAD(+)) with nicotina

94 Nicotinamide (NAM) is the main precursor of nicotinamide adenine dinucleotide (NAD(+)), a coenzyme e

95 er the ADP-ribose moiety from its substrate, nicotinamide adenine dinucleotide (NAD(+)), to amino aci

96 Nicotinamide adenine dinucleotide (NAD(+))-dependent ADP

97 such factor is the sirtuin (SIRT) family of nicotinamide adenine dinucleotide (NAD(+))-dependent dea

98 resveratrol action is the activation of the nicotinamide adenine dinucleotide (NAD(+))-dependent dea

99 rients, which requires the activation of the nicotinamide adenine dinucleotide (NAD(+))-dependent dea

100 and the sirtuins as a conserved family of a nicotinamide adenine dinucleotide (NAD(+))-dependent pro

101 ependency on metabolic pathways regulated by nicotinamide adenine dinucleotide (NAD(+)).

102 d, the precursor for de novo biosynthesis of nicotinamide adenine dinucleotide (NAD(+)).

103 glucose dehydrogenase and its cofactor beta-nicotinamide adenine dinucleotide (NAD(+)).

104 washed with a high salt solution followed by nicotinamide adenine dinucleotide (NAD(+)).

105 genesis or function, via increased levels of nicotinamide adenine dinucleotide (NAD(+)).

106 that KinA is activated by a decrease in the nicotinamide adenine dinucleotide (NAD(+))/NADH ratio vi

107 Retinal levels of nicotinamide adenine dinucleotide (NAD(+), a key molecul

108 of renal recovery from injury by regulating nicotinamide adenine dinucleotide (NAD) biosynthesis.

109 A), the universal precursor of the essential nicotinamide adenine dinucleotide (NAD) coenzyme.

110 Stx2a predominantly altered the nicotinamide adenine dinucleotide (NAD) cofactor pathway

111 ic acid (QA), the precursor of the universal nicotinamide adenine dinucleotide (NAD) cofactor.

112 ng NAD synthetase 1, the final enzyme of the nicotinamide adenine dinucleotide (NAD) de novo synthesi

113 e levels and was up-regulated in response to nicotinamide adenine dinucleotide (NAD) depletion and in

114 ing liver regeneration, the concentration of nicotinamide adenine dinucleotide (NAD) falls, at least

115 The nicotinamide adenine dinucleotide (NAD) glycohydrolase C

116 Nicotinamide adenine dinucleotide (NAD) is increasingly

117 Nicotinamide adenine dinucleotide (NAD) is produced via

118 Nicotinamide adenine dinucleotide (NAD) is synthesized d

119 BACKGROUND & AIMS: The mitochondrial nicotinamide adenine dinucleotide (NAD) kinase (NADK2, a

120 ation telomerase knockout mice display lower nicotinamide adenine dinucleotide (NAD) levels, and an i

121 be potent supplements boosting intracellular nicotinamide adenine dinucleotide (NAD) levels, thus pre

122 s the major determinant of dependence on the nicotinamide adenine dinucleotide (NAD) metabolic pathwa

123 (kcatc) for Rubisco from the C4 grasses with nicotinamide adenine dinucleotide (NAD) phosphate malic

124 Nicotinamide adenine dinucleotide (NAD) provides an impo

125 n variants in HAAO or KYNU, two genes of the nicotinamide adenine dinucleotide (NAD) synthesis pathwa

126 SIRT1 and PARP1, that are each consumers of nicotinamide adenine dinucleotide (NAD), a metabolite in

127 Nicotinamide adenine dinucleotide (NAD), a ubiquitous co

128 Examples include the adduct of glutamate and nicotinamide adenine dinucleotide (NAD), fragments of NA

129 We recently reported the presence of nicotinamide adenine dinucleotide (NAD)-capped RNAs in m

130 Here, we report that the nicotinamide adenine dinucleotide (NAD)-dependent deacet

131 2 family of enzymes or sirtuins are known as nicotinamide adenine dinucleotide (NAD)-dependent deacet

132 Sirtuin 2 (SIRT2), one of the mammalian nicotinamide adenine dinucleotide (NAD)-dependent lysine

133 a class of enzymes originally identified as nicotinamide adenine dinucleotide (NAD)-dependent protei

134 olved in the conversion of nicotinamide into nicotinamide adenine dinucleotide (NAD).

135 , a recently discovered vitamin precursor of nicotinamide adenine dinucleotide (NAD).

136 (GI) and electrochemical immobilization with nicotinamide adenine dinucleotide (NAD).

137 bosyltransferases such as PARPs that utilize nicotinamide adenine dinucleotide (NAD+) as a cofactor t

138 Sirtuins are nicotinamide adenine dinucleotide (NAD+)-dependent deacy

139 in generating cellular energy in the form of nicotinamide adenine dinucleotide (NAD+).

140 cations, including the recently described 5' nicotinamide-adenine dinucleotide (NAD(+)) RNA in bacter

141 The nicotinamide-adenine dinucleotide (NAD+)-dependent deace

142 osphate [ADP]/adenosine monophosphate [AMP], nicotinamide adenine dinucleotide /NAD, nicotinamide ade

143 ly promoted ROS production by downregulating nicotinamide adenine dinucleotide(+) (NAD(+))/reduced fo

144 ed electron transfer (PCET) reaction between nicotinamide adenine dinucleotide (NADH) and a protein-b

145 ltammetric determination of cysteamine (CA), nicotinamide adenine dinucleotide (NADH) and folic acid

146 s and produce in situ chemical species (beta-nicotinamide adenine dinucleotide (NADH) and H2O2) actin

147 1 mediates reduction of the diiron center by nicotinamide adenine dinucleotide (NADH) and initiates O

148 cleic acids (AAA + NA), tryptophan residues, nicotinamide adenine dinucleotide (NADH) and vitamin A w

149 to pyruvate to form l-lactate, using reduced nicotinamide adenine dinucleotide (NADH) as the cofactor

150 le electrochemical sensing platform for beta-nicotinamide adenine dinucleotide (NADH) based on uncapp

151 Reduced nicotinamide adenine dinucleotide (NADH) can generate a

152 rts was associated with a marked decrease in nicotinamide adenine dinucleotide (NADH) fluorescence, l

153 de adenine dinucleotide phosphate (NADPH) to nicotinamide adenine dinucleotide (NADH) have been made

154 e new sensor were tested by the oxidation of nicotinamide adenine dinucleotide (NADH) in a 0.1 M Robi

155 mediators for the oxidation of reduced beta-nicotinamide adenine dinucleotide (NADH) in two polymeri

156 We propose that the reduced cofactor nicotinamide adenine dinucleotide (NADH) is a possible h

157 Nicotinamide Adenine Dinucleotide (NADH) is an important

158 l alcohol sensing, whereby the coenzyme beta-Nicotinamide adenine dinucleotide (NADH) is employed as

159 ith the intracellular application of reduced nicotinamide adenine dinucleotide (NADH), an effect that

160 ine spectral profiles of tryptophan, reduced nicotinamide adenine dinucleotide (NADH), and flavin den

161 se that is stimulated by the reduced form of nicotinamide adenine dinucleotide (NADH), suggesting a s

162 Autofluorescence, attributed to nicotinamide adenine dinucleotide (NADH), was induced by

163 ew electrode material for the development of nicotinamide adenine dinucleotide (NADH)-based biosensor

164 By combining a nicotinamide adenine dinucleotide (NADH)-consuming aceta

165 amide coenzymes, such as the reduced form of nicotinamide adenine dinucleotide (NADH).

166 e in mitochondrial matrix Ca(2+) and reduced nicotinamide adenine dinucleotide (NADH).

167 adenine dinucleotide phosphate (NADPH), and nicotinamide adenine dinucleotide (NADH).

168 We assessed the reduced/oxidized ratio of nicotinamide adenine dinucleotide (NADH/NAD(+) ratio) an

169 decreasing Complex I activity, elevating the nicotinamide adenine dinucleotide (NADH/NAD(+)) ratio an

170 intracellular lactate levels, disrupted the nicotinamide adenine dinucleotide (NADH/NAD(+)) ratio, a

171 s we measured the production rate of reduced nicotinamide adenine dinucleotides (NADH) from 91 potent

172 active oxygen species (ROS) by the phagocyte nicotinamide adenine dinucleotide (NADPH) oxidase in pat

173 The tryptophan-kynurenine-nicotinamide adenine dinucleotide (oxidized; NAD+) pathw

174 enine dinucleotide, reduced form) to NAD(+) (nicotinamide adenine dinucleotide, oxidized form).

175 Here we show that nicotinamide adenine dinucleotide phosphatase (Nox2)-ind

176 RSV alone reduced nicotinamide adenine dinucleotide phosphatase oxidase (N

177 nase 2 was reduced, whereas the NOX2 (NADPH [nicotinamide adenine dinucleotide phosphatase] oxidase s

178 phatase] oxidase subunit 2) and NOX4 (NADPH [nicotinamide adenine dinucleotide phosphatase] oxidase s

179 translocation of the cytosolic components of nicotinamide adenine dinucleotide phosphate (NAD(P)H)-ox

180 ADP by exchanging the nicotinamide moiety of nicotinamide adenine dinucleotide phosphate (NADP(+) ) f

181 e adenine dinucleotide (NAD(+) and NADH) and nicotinamide adenine dinucleotide phosphate (NADP(+) and

182 (+) and NADH), oxidized and reduced forms of nicotinamide adenine dinucleotide phosphate (NADP(+) and

183 Nicotinamide adenine dinucleotide phosphate (NADP(+)) is

184 Nicotinamide adenine dinucleotide phosphate (NADP) is a

185 oduction of reactive oxygen species (ROS) by nicotinamide adenine dinucleotide phosphate (NADPH) 2 (N

186 chemical analogue exploiting this principle, nicotinamide adenine dinucleotide phosphate (NADPH) and

187 dehyde as substrates and the reduced form of nicotinamide adenine dinucleotide phosphate (NADPH) as c

188 gulate the activity of dFB neurons through a nicotinamide adenine dinucleotide phosphate (NADPH) cofa

189 to support the synthesis and regeneration of nicotinamide adenine dinucleotide phosphate (NADPH) in p

190 Reduced nicotinamide adenine dinucleotide phosphate (NADPH) is e

191 is metabolism, reducing power in the form of nicotinamide adenine dinucleotide phosphate (NADPH) is r

192 blast function, activating integrin-mediated nicotinamide adenine dinucleotide phosphate (NADPH) oxid

193 8-mediated ROS was generated through reduced nicotinamide adenine dinucleotide phosphate (NADPH) oxid

194 Nicotinamide adenine dinucleotide phosphate (NADPH) oxid

195 neutrophils exhibited reduced (hydrogenated) nicotinamide adenine dinucleotide phosphate (NADPH) oxid

196 this study, we show that antibiotics rescue nicotinamide adenine dinucleotide phosphate (NADPH) oxid

197 ure to LPS led to up-regulated expression of nicotinamide adenine dinucleotide phosphate (NADPH) oxid

198 The leukocyte nicotinamide adenine dinucleotide phosphate (NADPH) oxid

199 sing intravital microscopy with mice lacking nicotinamide adenine dinucleotide phosphate (NADPH) oxid

200 ation of p47(phox) (an organizer subunit for nicotinamide adenine dinucleotide phosphate (NADPH) oxid

201 ng protein 9 (CARD9), but was independent of nicotinamide adenine dinucleotide phosphate (NADPH) oxid

202 because of defective activation of phagocyte nicotinamide adenine dinucleotide phosphate (NADPH) oxid

203 s in the genes that encode components of the nicotinamide adenine dinucleotide phosphate (NADPH) oxid

204 ME1 also promotes nicotinamide adenine dinucleotide phosphate (NADPH) prod

205 causing hemolytic anemia linked to impaired nicotinamide adenine dinucleotide phosphate (NADPH) prod

206 s, G6PD is upregulated and generates reduced nicotinamide adenine dinucleotide phosphate (NADPH) that

207 cofactor specificity of oxidoreductases from nicotinamide adenine dinucleotide phosphate (NADPH) to n

208 Across phyla, reduced nicotinamide adenine dinucleotide phosphate (NADPH) tran

209 the enzyme with acyl-coenzyme A and reduced nicotinamide adenine dinucleotide phosphate (NADPH), a p

210 l and cellular concentration of glutathione, nicotinamide adenine dinucleotide phosphate (NADPH), and

211 operoxidase (LPO) and myeloperoxidase (MPO), nicotinamide adenine dinucleotide phosphate (NADPH)-depe

212 vealed age-related changes in the density of nicotinamide adenine dinucleotide phosphate (NADPH)-diap

213 , which exhibited decreased steatohepatitis, nicotinamide adenine dinucleotide phosphate (NADPH)-oxid

214 icotinamide adenine dinucleotide phosphate / nicotinamide adenine dinucleotide phosphate , flavin ade

215 MP], nicotinamide adenine dinucleotide /NAD, nicotinamide adenine dinucleotide phosphate / nicotinami

216 rtas of wild-type mice or mice deficient for nicotinamide adenine dinucleotide phosphate [NAD(P)H] ox

217 ependent protein kinase II), which decreases nicotinamide adenine dinucleotide phosphate hydrogen syn

218 ore, post-R had significantly higher reduced nicotinamide adenine dinucleotide phosphate levels, redu

219 The classic phagocyte nicotinamide adenine dinucleotide phosphate oxidase (gp9

220 the STAT5/PI3K/Akt signalling axis and that nicotinamide adenine dinucleotide phosphate oxidase (NOX

221 rtate (NMDA) receptor-mediated activation of nicotinamide adenine dinucleotide phosphate oxidase (NOX

222 Defects in phagocytic nicotinamide adenine dinucleotide phosphate oxidase 2 (N

223 Activity of myocardial reduced nicotinamide adenine dinucleotide phosphate oxidase 2 (N

224 uced blood pressure, whereas the transfer of nicotinamide adenine dinucleotide phosphate oxidase 2-de

225 We investigated whether nicotinamide adenine dinucleotide phosphate oxidase 4 (N

226 at adiponectin directly decreases myocardial nicotinamide adenine dinucleotide phosphate oxidase acti

227 rated that adiponectin suppresses myocardial nicotinamide adenine dinucleotide phosphate oxidase acti

228 nulomatous disease (CGD) is due to defective nicotinamide adenine dinucleotide phosphate oxidase acti

229 the signal involves plasma membrane reduced nicotinamide adenine dinucleotide phosphate oxidase acti

230 p47(phox) subunit beyond and independent of nicotinamide adenine dinucleotide phosphate oxidase acti

231 neuroinflammation by selectively inhibiting nicotinamide adenine dinucleotide phosphate oxidase and

232 the generation of reactive oxygen species by nicotinamide adenine dinucleotide phosphate oxidase and

233 blocked agonist-initiated association of the nicotinamide adenine dinucleotide phosphate oxidase comp

234 airment through neurohormonal activation of (nicotinamide adenine dinucleotide phosphate oxidase depe

235 ing the efficacy of an ultra-low dose of the nicotinamide adenine dinucleotide phosphate oxidase inhi

236 ), TEMPOL (a general antioxidant), apocynin (nicotinamide adenine dinucleotide phosphate oxidase inhi

237 Nicotinamide adenine dinucleotide phosphate oxidase isof

238 Adult rats; wild-type/nicotinamide adenine dinucleotide phosphate oxidase subu

239 ed baroreflex sensitivity is associated with nicotinamide adenine dinucleotide phosphate oxidase subu

240 In mice, genetic deficiency of gp91 nicotinamide adenine dinucleotide phosphate oxidase subu

241 xpression in P47 and Rac-1 expression of two nicotinamide adenine dinucleotide phosphate oxidase subu

242 own experimentally to activate transmembrane nicotinamide adenine dinucleotide phosphate oxidase type

243 th P67 dominant negative mice to inhibit the nicotinamide adenine dinucleotide phosphate oxidase were

244 The NOX (nicotinamide adenine dinucleotide phosphate oxidase) fam

245 Nicotinamide adenine dinucleotide phosphate oxidase, a k

246 show that the oxidative burst, catalyzed by nicotinamide adenine dinucleotide phosphate oxidase, can

247 cleotide phosphate oxidase 4 (Nox4), a major nicotinamide adenine dinucleotide phosphate oxidase, med

248 drial electron transport chain reactions and nicotinamide adenine dinucleotide phosphate oxidase, or

249 These pathways include nicotinamide adenine dinucleotide phosphate oxidase, whi

250 uncoupled endothelial nitric oxide synthase, nicotinamide adenine dinucleotide phosphate oxidase, xan

251 Increased nicotinamide adenine dinucleotide phosphate oxidase-2 (N

252 Nitric oxide and reduced nicotinamide adenine dinucleotide phosphate oxidase-depe

253 oding adiponectin) led to reduced myocardial nicotinamide adenine dinucleotide phosphate oxidase-deri

254 tion by restoring renal blood flow, reducing nicotinamide adenine dinucleotide phosphate oxidase-deri

255 Although nicotinamide adenine dinucleotide phosphate oxidase-targ

256 n peroxide (H(2)O(2)), likely by stimulating nicotinamide adenine dinucleotide phosphate oxidase.

257 activation, implicating a crosstalk between nicotinamide adenine dinucleotide phosphate oxidases and

258 downstream effectors such as Rho kinase and nicotinamide adenine dinucleotide phosphate oxidases are

259 Dual oxidases (DUOX) are conserved reduced nicotinamide adenine dinucleotide phosphate oxidases tha

260 reased malondialdehyde, 3-nitrotyrosine, and nicotinamide adenine dinucleotide phosphate oxidases).

261 nship between two membrane transporters, the Nicotinamide adenine dinucleotide phosphate reduced form

262 tive was to investigate the possible role of nicotinamide adenine dinucleotide phosphate reduced form

263 e hypothesized that AGEs induce TACE through nicotinamide adenine dinucleotide phosphate reduced oxid

264 te to cardioprotection by generating reduced nicotinamide adenine dinucleotide phosphate to enhance b

265 of ribose and NADP(+) (the oxidized form of nicotinamide adenine dinucleotide phosphate) complexes o

266 NADPH/NADP(+) (the reduced form of NADP(+)/nicotinamide adenine dinucleotide phosphate) homeostasis

267 vascular wall include NADPH (reduced form of nicotinamide adenine dinucleotide phosphate) oxidase, xa

268 ial susceptibility to NADPH (reduced form of nicotinamide adenine dinucleotide phosphate) oxidase-dep

269 ing the cofactors adenosine triphosphate and nicotinamide adenine dinucleotide phosphate, and have be

270 derivatives in complex with PfIspC, reduced nicotinamide adenine dinucleotide phosphate, and Mg(2+)

271 ion, depleting and oxidizing glutathione and nicotinamide adenine dinucleotide phosphate, and signifi

272 osynthetic electron transport in the form of nicotinamide adenine dinucleotide phosphate, reduced are

273 , Shc expression, markers of senescence, and nicotinamide adenine dinucleotide phosphate, reduced for

274 nockout, PBL13 is able to associate with the nicotinamide adenine dinucleotide phosphate, reduced oxi

275 forms of nicotinamide adenine nucleotide and nicotinamide adenine dinucleotide phosphate, respectivel

276 The three IDH isoforms (nicotinamide adenine dinucleotide phosphate-dependent ID

277 Gain-of-function mutations in nicotinamide adenine dinucleotide phosphate-dependent is

278 Nicotinamide adenine dinucleotide phosphate-diaphorase (

279 e performed for acetylcholinesterase (AChE), nicotinamide adenine dinucleotide phosphate-diaphorase (

280 enchymal stem/stromal cell therapy decreased nicotinamide adenine dinucleotide phosphate-oxidase 2 an

281 y nicotinamide adenine dinucleotide, but not nicotinamide adenine dinucleotide phosphate.

282 ADPH, by the negatively charged hydride from nicotinamide adenine dinucleotide phosphate.

283 for G6PD enzyme activity, cellular oxidized nicotinamide adenine dinucleotide phosphate/NADPH levels

284 olism via endogenous fluorescence of reduced nicotinamide adenine dinucleotide (phosphate) (NAD(P)H)

285 FLIM of autofluorescent reduced nicotinamide adenine dinucleotide (phosphate), NAD(P)H,

286 otein adducts together with increased NADPH (nicotinamide adenine dinucleotide phosphateoxidase) acti

287 mic reticulum on electron microscopy, and 3) nicotinamide adenine dinucleotide redox potential and ad

288 tathione/oxidized glutathione (GSH/GSSG) and nicotinamide adenine dinucleotide reduced/oxidized forms

289 ubunits of the RNA polymerase, and thylakoid nicotinamide adenine dinucleotide (reduced) and cytochro

290 hain reaction) and oxidative phosphorylation nicotinamide adenine dinucleotide (reduced) dehydrogenas

291 inactivate Sirt3 because of increased NADH (nicotinamide adenine dinucleotide, reduced form) and ace

292 ence of C-SWCNT, the oxidation of NADH (beta-nicotinamide adenine dinucleotide, reduced form) and DTT

293 e of ALDH7A1 activity, which generates NADH (nicotinamide adenine dinucleotide, reduced form) from NA

294 acetate production to the formation of NADH (nicotinamide adenine dinucleotide, reduced form) that is

295 esults in the concomitant oxidation of NADH (nicotinamide adenine dinucleotide, reduced form) to NAD(

296 etabolic dependencies (fatty acid oxidation, nicotinamide adenine dinucleotide synthesis, glutamine b

297 witch, we demonstrated that depletion of the nicotinamide adenine dinucleotide synthetase (NadE) rapi

298 ms-ethanol by reducing the ratio of oxidized nicotinamide adenine dinucleotide to reduced nicotinamid

299 n catalysed by these enzymes is energized by nicotinamide adenine dinucleotide, which activates ubiqu

300 NA damage and consumes and depletes cellular nicotinamide adenine dinucleotide, which leads to mitoch