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1 cating that they were cholinergic as well as nitrergic.
2  enteric inhibitory neural inputs are mainly nitrergic.
3 hat the fIJP was purinergic and the sIJP was nitrergic.
4                                              Nitrergic agonists inhibited Ca(2+) transients in ICC-SS
5                                              Nitrergic and alpha2-adrenergic modulation of gastric se
6 ows functional neurodegeneration and reduces nitrergic and glycation-associated cellular stress.
7 (+) subsets localized in close apposition to nitrergic and peptidergic nerve fibers in the colon lami
8  of the intrinsic innervation; inhibition of nitrergic and VIPergic electrical field-induced relaxati
9 vival of later-born dopaminergic, GABAergic, nitrergic, and calcitonin gene-related peptide-expressin
10 a were contacted by cholinergic, adrenergic, nitrergic, and VIP-positive terminals.
11  immunoreactivity, indicating that they were nitrergic as well.
12  immunoreactivity, indicating that they were nitrergic as well.
13 lexus contained cholinergic, adrenergic, and nitrergic axons and vasoactive intestinal polypeptide-po
14                         In the second phase, nitrergic cell bodies in the ganglia are lost via apopto
15            The results demonstrated abundant nitrergic cells in the basal ganglia, amygdaloid complex
16                  In addition, low numbers of nitrergic cells were observed in the olfactory bulb, all
17                                          The nitrergic component is characterized by membrane potenti
18 ion of enteric neurons, with cholinergic and nitrergic components blocked, evoked Ca(2+) transients i
19           This is the first demonstration of nitrergic degeneration in diabetic cerebral arteries, wh
20                         In the second phase, nitrergic degeneration takes place in the cell bodies in
21 alpha7-nAChRs, induced sympathetic-dependent nitrergic dilation of porcine basilar arterial rings.
22  Thus, hyperpolarization is not required for nitrergic effects on [Ca2+]i and tension.
23 establish accurately the localization of the nitrergic elements and to assess possible interactions b
24 al brainstem region is the primary source of nitrergic fibers in the mouse visual thalamus, similar t
25 ture preparations indicated that most of the nitrergic fibers within cardiac ganglia were extrinsic.
26      Our data suggest that ER's can regulate nitrergic function by improving Nrf2/nNOS expression in
27  in the ganglia, leading to complete loss of nitrergic function.
28  purinergic component was reduced, while the nitrergic IJP was slightly increased.
29 g neurophysiology of migraine symptoms, with nitrergic-induced changes reflecting one that is relevan
30 ance in smooth muscles and may contribute to nitrergic inhibition of gastrointestinal muscles.
31 trast, basal pyloric hypotension with normal nitrergic inhibition predisposes W/W(v) mice to duodenog
32                      Slow wave amplitude and nitrergic inhibitory junction potentials were reduced wh
33 it TREK-1 channels, native SDK channels, and nitrergic inhibitory junction potentials.
34 ortance of ICC in cholinergic excitatory and nitrergic inhibitory motor neurotransmission.
35 P developed, both cholinergic excitatory and nitrergic inhibitory neural responses were intact.
36 ial cells of Cajal (ICC) act as mediators in nitrergic inhibitory neurotransmission.
37  evoked prominent cholinergic excitatory and nitrergic inhibitory responses in longitudinal muscles o
38               In this study, we examined the nitrergic innervation to the adult mouse visual thalamus
39 -d) histochemistry to identify the source of nitrergic innervation.
40 neurons indicates that: i) DR is a source of nitrergic input to trigeminal structures, and ii) NO and
41 d that the dendritic and axonal processes of nitrergic interneurons already apposed the somata and de
42             However, it is not known whether nitrergic interneurons are in this position early in lif
43                                 In contrast, nitrergic interneurons developed more rapidly.
44                     The results suggest that nitrergic interneurons play a role in refining the corti
45                Nitric oxide (NO) containing (nitrergic) interneurons are well-positioned to convey th
46 duced by l-NAME administration, suggesting a nitrergic mechanism of gastroinhibition.
47 opment of acute inflammatory hyperaemia, but nitrergic mechanisms may supervene subsequently.
48 d/or gastric hormone receptors on regulating nitrergic-mediated gastric motility remains inconclusive
49 onsistent with the idea that cholinergic and nitrergic motor neural inputs are mediated, to a large e
50 motor neurones revealed that cholinergic and nitrergic motor neurones formed close contacts with ICC-
51                  These disorders precede the nitrergic myenteric cell loss observed in the proximal c
52 e assessed, in addition to colonic and ileal nitrergic myenteric neuron quantifications and motility.
53  disorders inducing constipation and loss of nitrergic myenteric neurons in the proximal colon.
54                DOReGFP was present mostly in nitrergic myenteric neurons of colon.
55 use of optogenetics to selectively stimulate nitrergic myenteric neurons using blue light stimulation
56                               The numbers of nitrergic myenteric neurons were reduced in the proximal
57 ess the impact of these factors on autonomic nitrergic nerve and endothelial function.
58                          In the first phase, nitrergic nerve fibers lose some of their neuronal nitri
59             In the first phase, perivascular nitrergic nerve fibers remain intact while they lose the
60 ponsiveness and greatly reduces responses to nitrergic nerve stimulation.
61       NO, produced by NOS-1 in pericytes and nitrergic nerve terminals and by NOS-3 in vascular endot
62 ta2-adrenoceptors located on the neighboring nitrergic nerve terminals, resulting in NO release and v
63                                              Nitrergic nerves and interstitial cells of Cajal (ICC) h
64    Like MPOs, their activity is inhibited by nitrergic nerves and synchronized by excitatory nerves.
65              Here, we show that perivascular nitrergic nerves around the cerebral arteries degenerate
66           However, the effect of diabetes on nitrergic nerves is unknown.
67                    It has been proposed that nitrergic nerves mediate lower esophageal sphincter (LES
68                              Cholinergic and nitrergic nerves were also present in W/W(V) mice, but f
69 ut not direct stimulation of parasympathetic nitrergic nerves, caused nitrergic neurogenic dilation.
70 ng nitric oxide (NO); hence, they are called nitrergic nerves.
71 ould occur via nonadrenergic, noncholinergic nitrergic neural and/or endocrine pathways.
72 ors of excitatory cholinergic and inhibitory nitrergic neural inputs from the enteric nervous system,
73                                              Nitrergic neural inputs regulate the Ca(2+) events in IC
74 irst ICC reported to receive cholinergic and nitrergic neural inputs.
75 used loss of ICC and loss of cholinergic and nitrergic neural responses.
76 IR was coexpressed with both cholinergic and nitrergic neurochemical codes in the myenteric plexus.
77  of parasympathetic nitrergic nerves, caused nitrergic neurogenic dilation.
78 studying cellular and synaptic mechanisms of nitrergic neuromodulation in a simple pattern-generating
79 to neurons with cholinergic, calretinin, and nitrergic neuronal immunochemical codes in the myenteric
80 h in the ganglia, which is selective for the nitrergic neurones.
81                          The analysis of the nitrergic neurons (with the enzyme nitric oxide synthase
82 establish accurately the localization of the nitrergic neurons and fibers and to assess possible inte
83 rgic subpopulation and decreased the area of nitrergic neurons and vasoactive intestinal peptide (VIP
84   Glutamatergic, GABAergic, cholinergic, and nitrergic neurons are all found in the PCRt and IRt, but
85 ay mediates caspase-3 dependent apoptosis of nitrergic neurons in the MPG following CNI and that ROCK
86 rointestinal motility and reduced numbers of nitrergic neurons in TLR4(Lps-d), TLR4(-/-), and Myd88(-
87   Significant populations of cholinergic and nitrergic neurons overlapped pre-oromotor neurons, but t
88 ed total neurons and a smaller proportion of nitrergic neurons per ganglion, and reduced markers of n
89                   Each of these identifiable nitrergic neurons projects into the ipsilateral pedal ga
90  perturbations followed neuronal activation: Nitrergic neurons regulated T helper 17 (T(H)17)-like ce
91 nly two regions of the mouse brain contained nitrergic neurons that projected to the visual thalamus:
92            In contrast, less than 10% of the nitrergic neurons were activated after TNF exposure.
93 nd their colonic LMMP had reduced numbers of nitrergic neurons, reduced neuronal nitric oxide synthas
94  did not act directly on the parasympathetic nitrergic neurons.
95 trate that nitric oxide synthase-containing (nitrergic) neurons innervating the penis and gastric pyl
96 ulus responses were blocked by inhibition of nitrergic neurotransmission in proximal colon, but P2Y1
97 12 is a significant regulatory mechanism for nitrergic neurotransmission in the gut.
98 L-citrulline recycling in the maintenance of nitrergic neurotransmission in the opossum internal anal
99           The data show that cholinergic and nitrergic neurotransmission only occurs in the gastric l
100 suggesting that ICC-IM do not play a role in nitrergic neurotransmission.
101 ine (l-NNA; 100 mum) to block purinergic and nitrergic NMT to characterize non-purinergic, non-nitrer
102 timuli (4 s) were mediated by purinergic and nitrergic NMT.
103 rgic NMT to characterize non-purinergic, non-nitrergic (NNNP) inhibitory NMT and the role of VIP in t
104 halogram recordings pointed to glutamatergic/nitrergic (NOS1) and GABAergic neurons in the ventral te
105                           Dysfunction of the nitrergic pathway has been shown to cause LES hypertensi
106 f NO-containing fibers with respect to other nitrergic profiles in the somatosensory system; and 3) t
107 onkey LGN receives a significant cholinergic/nitrergic projection from the pedunculopontine tegmentum
108                   In nNOS(-/-) mice, loss of nitrergic pyloric inhibition leads to gastric stasis and
109                             In regard to the nitrergic regulation of the circulation, the fetus is cl
110  Nitric Oxide Synthase (nNOS) expression and nitrergic relaxation in gastric neuromuscular tissues ex
111 mportant role for ICC-IM in vagally-mediated nitrergic relaxation in the proximal and distal stomach.
112                 mRNA, protein expression and nitrergic relaxation of circular gastric neuromuscular s
113 We investigated the role of ICCs and SMCs in nitrergic relaxation using mice with cell-specific disru
114                                     However, nitrergic relaxation was not observed in SM/ICC-GCKO mic
115 cantly reduced Nrf2/nNOSalpha expression and nitrergic relaxation.
116 nd stimulation of intrinsic nerves activated nitrergic responses in ICC-SS.
117 atus may be the primary mechanisms mediating nitrergic responses in these muscles.
118 NOS(+) neurons and subsequent restoration of nitrergic responses.
119 g in Aglantha, possibly by means of putative nitrergic sensory neurons in its tentacles.
120 easing number of electrical stimuli to study nitrergic signaling in the colon.
121 from the combination of SMCs and ICCs blocks nitrergic signaling.
122 ults provide evidence for the existence of a nitrergic signalling system in molluscan larvae and juve
123 rstitial cells of Cajal (ICCs), might detect nitrergic signals to indirectly regulate smooth muscle t
124  of the canine gastric fundus in response to nitrergic stimulation by NO donors and electrical field
125 uction in [Ca2+]i and tension in response to nitrergic stimulation of the canine gastric fundus does
126 uanylyl cyclase, also blocked the effects of nitrergic stimulation on [Ca2+]i and tension, without af
127  reductions in [Ca2+]i and tension caused by nitrergic stimulation were blocked.
128    Block of hyperpolarization in response to nitrergic stimulation with tetrapentylammonium chloride
129 ir hippocampi were characterized by enhanced nitrergic stress associated with a decline in neuronal f
130 sis of microglia, brain immune cells promote nitrergic stress by overproducing nitric oxide (NO), lea
131 d Parkinson's disease) exhibit oxidative and nitrergic stress following initiation of neuroinflammato
132 d protein expression levels of oxidative and nitrergic stress markers were analyzed, and electrophysi
133 he neuronal areas of the general population, nitrergic subpopulation and (VIP) varicosities were redu
134 he neuronal areas of the general population, nitrergic subpopulation and (VIP) varicosities were redu
135 odenum, the HFD increased the density of the nitrergic subpopulation and decreased the area of nitrer
136           In the jejunum, the density of the nitrergic subpopulation was increased and the neuronal a
137 m, the density of the general population and nitrergic subpopulation were increased and the neuronal
138  the electrical excitability of MNCs via the nitrergic system and strengthen the importance of HCN ch
139 s a conserved pattern of organization of the nitrergic system from this primitive fish group to amnio
140 sing a neuromeric analysis, reveals that the nitrergic system of Neoceratodus shares many neuroanatom
141                          We investigated the nitrergic system of two cladistian species by means of s
142                     We have investigated the nitrergic system with neural nitric oxide synthase (NOS)
143              In spite of the transition from nitrergic to purinergic regulation, post-stimulus respon

 
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